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Shaw, Allan. "Functional bowel disorders in anxiety disorder out patients". Thesis, London South Bank University, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.288174.
Pełny tekst źródłaArnold, Marla N. "Validating a model of risk factors associated with eating disorder risk in adolescents". Columbus, Ohio : Ohio State University, 2006. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1148575712.
Pełny tekst źródłaHommersen, Paul. "Separation Anxiety Disorder and Oppositional Defiant Disorder : perceived comorbidity between disorders resulting from ambiguous items and halo effects". Thesis, University of British Columbia, 2007. http://hdl.handle.net/2429/31331.
Pełny tekst źródłaArts, Faculty of
Psychology, Department of
Graduate
MacCallam, Jackie. "Cognitive appraisals in obsessive-compulsive disorder & other anxiety disorders". Thesis, University of Plymouth, 1997. http://hdl.handle.net/10026.1/1138.
Pełny tekst źródłaVesco, Anthony Thomas. "Examining the Course of Cyclothymic Disorder and Comparing it to Dysthymic Disorder and Other Bipolar Spectrum Disorders in Children". The Ohio State University, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=osu1364907946.
Pełny tekst źródłaStinson, Jill D., i Brittany V. Williams. "Redefining Borderline Personality Disorder: BPD, DSM-v, and Emotion Regulation Disorders". Digital Commons @ East Tennessee State University, 2013. https://dc.etsu.edu/etsu-works/7970.
Pełny tekst źródłaTaha, Ai Yun. "Exploring functional connectivity across borderline personality disorder, post traumatic stress disorder and dissociative disorder". Thesis, University College London (University of London), 2015. http://discovery.ucl.ac.uk/1471093/.
Pełny tekst źródłaGuiot, Stacey L. "Body dysmorphic disorder: insight into the somatoform disorder". Thesis, Boston University, 2012. https://hdl.handle.net/2144/12406.
Pełny tekst źródłaBody Dysmorphic Disorder, or BDD, is a prevalent disease that affects children, adolescence, and adults. Its onset is usually in late childhood/early adolescence, and the disorder frequently extends for the lifetime ofthe patient. The disorder has been in the Diagnostic and Statistical Manual ofMental Disorders since its third edition as a somatoform disorder. The primary definition of BDD centers around the fact that those suffering from the disorder have a preoccupation with an imagined defect in their physical appearance that is usually not seen from an outsider's perspective. This preoccupation results in impairment in one's social life, education, and employment atmosphere. Through various research projects, it has been discovered that BDD shares many common similarities to other disorders, including obsessive-compulsive, social anxiety, and eating disorders. Like obsessive-compulsive disorder, those with BDD have several types of obsessions and compulsions, such as mirror checking for multiple hours a day to study their defect. This can further lead into the yearning desire to obtain cosmetic surgery. Patients with BDD often suffer from anxiety and depression, which can result in a low educational level, no employment, and trouble being in any sort of relationship. These symptoms tend to be more severe in those with the non-delusional form of BDD versus the delusional form. Research via functional Magnetic Resonance Imaginf and other imaging techniques has shown that those suffering from BDD may have different brain patterns than healthy subjects, especially concerning spatial frequency. Currently there are no FDA-approved medications specifically for the treatment ofBDD, but serotonin-reuptake inhibitors often used to treat depression have shown to be successful in alleviating BDD symptoms. Cognitive behavioral therapy, exposure and response prevention, and interpersonal psychotherapy, are also implemented as alternative treatment options to pharmacological therapy. The fifth edition of the Diagnostic and Statistical Manual ofMental Disorders is expected to be released in 2013 addressing the new information that has resulted from the great amount of research that has been conducted in the past decade and a half.
Butler, Emma. "The clinical relevance of personality disorder cognitions in the eating disorders". Thesis, University of East London, 2009. http://roar.uel.ac.uk/3729/.
Pełny tekst źródłaPISCIOTTA, LIVIA. "Autism Spectrum Disorder and other Neurodevelopmental Disorders: cytogenetic and genomic approaches". Doctoral thesis, Università degli studi di Genova, 2021. http://hdl.handle.net/11567/1057765.
Pełny tekst źródłaKnipp, Diana Kathleen. "Teens' Perceptions About Attention Deficit Disorder/Attention Deficit Hyperactivity Disorder Medications and Adaptation to Attention Deficit Disorder/Attention Deficit Hyperactivity Disorder". Thesis, Tucson, Arizona : University of Arizona, 2005. http://etd.library.arizona.edu/etd/GetFileServlet?file=file:///data1/pdf/etd/azu%5Fetd%5F1312%5F1%5Fm.pdf&type=application/pdf.
Pełny tekst źródłaDavis, Sally, i sallyjjdavis@bigpond com. "Chronik disorder". RMIT University. Creative Media, 2006. http://adt.lib.rmit.edu.au/adt/public/adt-VIT20080416.092509.
Pełny tekst źródłaDavis, Sally, i sakkyjdavis@bigpond com. "Chronik disorder". RMIT University. Creative Media, 2006. http://adt.lib.rmit.edu.au/adt/public/adt-VIT20080704.102340.
Pełny tekst źródłaHolt, Jim. "Biopolar Disorder". Digital Commons @ East Tennessee State University, 2007. https://dc.etsu.edu/etsu-works/6500.
Pełny tekst źródłaStinson, Jill D., i Judith V. Becker. "Pedophilic Disorder". Digital Commons @ East Tennessee State University, 2016. https://dc.etsu.edu/etsu-works/8004.
Pełny tekst źródłaNewton, Angus William. "Charge transfer and disorder broadening in disordered transition metal alloys". Thesis, University of Liverpool, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.343931.
Pełny tekst źródłaHammel, Jacinda Celeste McGlynn F. Dudley. "Meta worry and generalized anxiety disorder". Auburn, Ala., 2006. http://repo.lib.auburn.edu/2006%20Summer/Dissertations/HAMMEL_JACINDA_58.pdf.
Pełny tekst źródłaDahal, Liza. "Functional relevance of protein disorder : why is disorder favourable?" Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/283008.
Pełny tekst źródłaDalvie, Shareefa. "Genetic analysis of bipolar disorder and alcohol use disorder". Doctoral thesis, University of Cape Town, 2015. http://hdl.handle.net/11427/16560.
Pełny tekst źródłaBackground: Mental health disorders represent a major public health problem in most countries around the world. In South Africa, the lifetime prevalence of psychiatric disorders is 30.3%, with substance-use disorders and mood disorders being the second and third most prevalent classes of lifetime disorders, respectively. Bipolar disorder (BD) has a lifetime prevalence of 1.4% and alcohol use disorder (AUD) a lifetime prevalence of 30.3%, and they are frequently comorbid. Both of these disorders have a relatively high heritability, yet the exact genetic basis of each remains unknown. Genetic variants within the hypothalamic-pituitary-adrenal (HPA)-axis and glutamatergic pathways have previously been implicated in both phenotypes. The aim of this project was to investigate the aetiology of BD and AUD, using high-throughput genomic technologies, bioinformatics, brain-imaging and environmental measures. An additional aim was to assess the genetic aetiology of BD-AUD comorbidity. Methods: For the genetic analysis underlying BD, a South African 'Afrikaner' family was investigated. Whole-genome sequencing (WGS) and whole-genome linkage analysis was performed for individuals with BD Type I (BDI) and unaffected family members using the Illumina HiSeq2000 and Affymetrix Axiom TM Genome-wide CEU 1 Array, respectively. For the AUD analysis, two groups were investigated; a South African adolescent group comprising 80 individuals with AUD and 80 controls, and a group of 8123 individuals from the Avon Longitudinal Study of Parents and Children (ALSPAC) birth cohort. The South African group of adolescents were genotyped using the Illumina Infinium iSelect custom 6000 BeadChip, childhood trauma data was obtained and brain magnetic resonance images were collected for a subset of this group. Genotype data on HPA-axis genes were obtained from a previous study for the ALSPAC cohort. The fourth group of individuals investigated in this thesis comprised 233 individuals with BD-AUD comorbidity from the Systemic Treatment Enhancement Program for BD (STEP-BD). Genotype data for genes from the glutamatergic and HPA-axis pathways were obtained from a previous study conducted on these individuals. Results: The chromosomal regions 6p25, 10p14-10p15.1, 11q23-11q25, and 13q21-22 scored the highest LOD scores for BD and the most over-represented pathway in the affected family members was the T-cell receptor signalling pathway. In the South African adolescent group, circadian rhythm genes were associated with AUD and childhood trauma predicted alcohol use in adolescence. The gene-imaging analysis identified a SNP in the glutamate receptor, ionotropic, N-methyl D-aspartate 2B (GRIN2B) gene as being associated with brain volume in the left orbitofrontal cortex and posterior cingulate. HPA-axis genes did not show an association with AUD and no significant gene x environment interactions were detected for AUD in the ALSPAC cohort. Single variants in the glutamatergic genes and HPA-axis were not associated with BD-AUD comorbidity. However, from the gene-based analysis, the glutamatergic gene PRKCI was associated with BD-AUD comorbidity. Conclusions: It appears that disruption in immune-related genes may contribute to the development of BD in an Afrikaner family. No significant gene x environment interactions were detected for adolescent AUD. The circadian pathway and childhood trauma may play a role in the development of adolescent AUD. Differential brain volume and BD-AUD comorbidity may be characterised by variation in the glutamatergic pathway. These pathways and the interactions between them should be further investigated in BD and AUD.
Stark, Claire. "Trauma, alexithymia, emotional regulation and dissociation in alcohol use disorder, substance use disorder and polysubstance disorder". Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/25755.
Pełny tekst źródłaDuffy, Colleen. "Prevalence of Undiagnosed Dissociative Disorders in an Inpatient Setting". Thesis, University of North Texas, 2000. https://digital.library.unt.edu/ark:/67531/metadc2578/.
Pełny tekst źródłaTobiassen, Linn Graham. "Eating Disorders in Obsessive-Compulsive Disorder : Prevalence and Effect on Treatment Outcome". Thesis, Norges teknisk-naturvitenskapelige universitet, Psykologisk institutt, 2013. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-25188.
Pełny tekst źródłaPäären, Aivar. "Long-Term Health Outcome of Adolescent Mood Disorders : Focus on Bipolar Disorder". Doctoral thesis, Uppsala universitet, Institutionen för neurovetenskap, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-239835.
Pełny tekst źródłaSchweitzer, Jana. "Eating disorders : the correlation of family relationships with an eating disorder continuum". PDXScholar, 1988. https://pdxscholar.library.pdx.edu/open_access_etds/3844.
Pełny tekst źródłaKenny, Lucy Margaret. "Memory processes in posttraumatic stress disorder". [New South Wales : University of New South Wales], 2006. http://www.library.unsw.edu.au/~thesis/adt-NUN/uploads/approved/adt-NUN20061110.142022/public/02whole.pdf.
Pełny tekst źródłaPezzoli, Maria Elisabetta. "Disorder and Interaction: ground state properties of the disordered Hubbard model". Doctoral thesis, SISSA, 2008. http://hdl.handle.net/20.500.11767/4178.
Pełny tekst źródłaBrohede, Sabina. "Body Dysmorphic Disorder : Capturing a prevalent but under-recognized disorder". Doctoral thesis, Linköpings universitet, Avdelningen för kliniska vetenskaper, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-133368.
Pełny tekst źródłaWilliams, Howard Mark. "Disorder in materials". Thesis, De Montfort University, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.254680.
Pełny tekst źródłaSmith, Adam. "Disorder-free localization". Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/282870.
Pełny tekst źródłaHur, Cem. "Tackling Bipolar Disorder". Thesis, Birmingham City University, 2017. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.731716.
Pełny tekst źródłaHilbert, Anja. "Binge-Eating Disorder". Elsevier, 2019. https://ul.qucosa.de/id/qucosa%3A75711.
Pełny tekst źródłaDarke, Sacha. "Crime and disorder". Thesis, University of Westminster, 2007. https://westminsterresearch.westminster.ac.uk/item/91x70/crime-and-disorder.
Pełny tekst źródłaEpps, Susan Bramlett, Robert C. Barnhart, Mary Jo Davenport i Vey M. Norquist. "Developmental Coordination Disorder". Digital Commons @ East Tennessee State University, 2003. https://dc.etsu.edu/etsu-works/2556.
Pełny tekst źródłaPack, Robert P. "Opioid Use Disorder". Digital Commons @ East Tennessee State University, 2017. https://dc.etsu.edu/etsu-works/1335.
Pełny tekst źródłaHitt, Sara Beth, i false. "Autism Spectrum Disorder". Digital Commons @ East Tennessee State University, 2016. https://dc.etsu.edu/etsu-works/4068.
Pełny tekst źródłaMoody, Lara. "Evidence of Executive Dysfunction in Co-occurring Substance Use Disorder and Major Depressive Disorder or Antisocial Personality Disorder". Thesis, Virginia Tech, 2014. http://hdl.handle.net/10919/78165.
Pełny tekst źródłaMaster of Science
Warner, Megan Beth. "Personality traits, traitedness, and disorders: towards an enhanced understanding of trait-disorder relationships". Texas A&M University, 2005. http://hdl.handle.net/1969.1/4238.
Pełny tekst źródłaCook, Laura Michele. "Elucidating the relation of hoarding to obsessive compulsive disorder and impulse control disorders". Diss., Online access via UMI:, 2007.
Znajdź pełny tekst źródłaRall, Edrich. "Cluster analysis of disorders characterized by impulsivity in patients with methamphetamine use disorder". Master's thesis, Faculty of Health Sciences, 2019. http://hdl.handle.net/11427/31204.
Pełny tekst źródłaOrtiz-Dominguez, Tania Abigail. "Migraine comorbidity in bipolar disorder". Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=116105.
Pełny tekst źródłaObjective: To explore the prevalence and clinical characteristics of BD patients with and without migraine (Study 1), and to examine the psychiatric comorbidity in patients suffering from migraine (Study 2).
Methods: 323 BD patients were studied, using SADS-L and SCID as diagnostic interviews, and ill-Migraine questionnaire to assess the presence of migraine. Statistical analyses were conducted using parametric analysis and the development of log-linear models. Additionally, 102 migraine patients were interviewed using SADS-L, and the descriptive characteristics of the sample were analyzed.
Results: For Study 1, we found that 24.5% of BD patients suffer from migraine, and it is significantly associated with BD 2, suicidal behaviour, and a variety of anxiety disorders. As well, over 70% of migraine patients showed a lifetime psychiatric diagnosis, mainly within the spheres of mood and anxiety disorders; specifically, the prevalence of BD among migraine patients was 12.7%.
Conclusions: Our study highlights the high prevalence of migraine among BD patients, and the elevated prevalence of psychiatric comorbidity among migraine sufferers. The study of this comorbidity will deepen our understanding of the mechanisms that underlie both disorders and provide a better framework for the developing of molecular techniques to further analyze the molecular physiopathology of Bipolar Disorder.
Alamian, Golnoush. "Intellectual functioning in first-episode schizophrenia, schizoaffective disorder and bipolar disorder". Thesis, University of British Columbia, 2014. http://hdl.handle.net/2429/51790.
Pełny tekst źródłaMedicine, Faculty of
Graduate
Cruddace, Susan Ann. "Attention deficits in children with reading disorder, movement disorder or both". Thesis, University of Reading, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.408093.
Pełny tekst źródłaPazesh, Samaneh. "Process-induced disorder of pharmaceutical materials : Mechanisms and quantification of disorder". Doctoral thesis, Uppsala universitet, Institutionen för farmaci, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-317801.
Pełny tekst źródłaPrane, Jada Z. "Multiple personality disorder/dissociated identity disorder : the client as actor model /". view abstract or download file of text, 1999. http://wwwlib.umi.com/cr/uoregon/fullcit?p9957569.
Pełny tekst źródłaTypescript. Includes vita and abstract. Includes bibliographical references (leaves 337-340). Also available for download via the World Wide Web; free to University of Oregon users. Address: http://wwwlib.umi.com/cr/uoregon/fullcit?p9957569.
Solhan, Marika. "Affective instability and impulsivity in borderline personality disorder". Diss., Columbia, Mo. : University of Missouri-Columbia, 2006. http://hdl.handle.net/10355/4605.
Pełny tekst źródłaThe entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from title screen of research.pdf file (viewed on August 28, 2007) Includes bibliographical references.
au, chris theunissen@health wa gov, i Christopher Theunissen. "A Multidimensional Developmental Neuropsychological Model of Borderline Personality Disorder (BPD): Examining Evidence for Impairments in Executive Function". Murdoch University, 2005. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20050602.162509.
Pełny tekst źródłaNiklasson, Lena. "22q11 deletion syndrome neuropsychological and neuropsychiatric correlates : a clinical study of 100 cases /". Göteborg : Göteborg University, Institute of Neuroscience and Physiology, Child and Adolescent Psychiatry, 2007. http://hdl.handle.net/2077/7499.
Pełny tekst źródłaMidic, Uros. "Genome-Wide Prediction of Intrinsic Disorder; Sequence Alignment of Intrinsically Disordered Proteins". Diss., Temple University Libraries, 2012. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/159800.
Pełny tekst źródłaPh.D.
Intrinsic disorder (ID) is defined as a lack of stable tertiary and/or secondary structure under physiological conditions in vitro. Intrinsically disordered proteins (IDPs) are highly abundant in nature. IDPs possess a number of crucial biological functions, being involved in regulation, recognition, signaling and control, e.g. their functional repertoire complements the functions of ordered proteins. Intrinsically disordered regions (IDRs) of IDPs have a different amino-acid composition than structured regions and proteins. This fact has been exploited for development of predictors of ID; the best predictors currently achieve around 80% per-residue accuracy. Earlier studies revealed that some IDPs are associated with various human diseases, including cancer, cardiovascular disease, amyloidoses, neurodegenerative diseases, diabetes and others. We developed a methodology for prediction and analysis of abundance of intrinsic disorder on the genome scale, which combines data from various gene and protein databases, and utilizes several ID prediction tools. We used this methodology to perform a large-scale computational analysis of the abundance of (predicted) ID in transcripts of various classes of disease-related genes. We further analyzed the relationships between ID and the occurrence of alternative splicing and Molecular Recognition Features (MoRFs) in human disease classes. An important, never before addressed issue with such genome-wide applications of ID predictors is that - for less-studied organisms - in addition to the experimentally confirmed protein sequences, there is a large number of putative sequences, which have been predicted with automated annotation procedures and lack experimental confirmation. In the human genome, these predicted sequences have significantly higher predicted disorder content. I investigated a hypothesis that this discrepancy is not correct, and that it is due to incorrectly annotated parts of the putative protein sequences that exhibit some similarities to confirmed IDRs, which lead to high predicted ID content. I developed a procedure to create synthetic nonsense peptide sequences by translation of non-coding regions of genomic sequences and translation of coding regions with incorrect codon alignment. I further trained several classifiers to discriminate between confirmed sequences and synthetic nonsense sequences, and used these predictors to estimate the abundance of incorrectly annotated regions in putative sequences, as well as to explore the link between such regions and intrinsic disorder. Sequence alignment is an essential tool in modern bioinformatics. Substitution matrices - such as the BLOSUM family - contain 20x20 parameters which are related to the evolutionary rates of amino acid substitutions. I explored various strategies for extension of sequence alignment to utilize the (predicted) disorder/structure information about the sequences being aligned. These strategies employ an extended 40 symbol alphabet which contains 20 symbols for amino acids in ordered regions and 20 symbols for amino acids in IDRs, as well as expanded 40x40 and 40x20 matrices. The new matrices exhibit significant and substantial differences in the substitution scores for IDRs and structured regions. Tests on a reference dataset show that 40x40 matrices perform worse than the standard 20x20 matrices, while 40x20 matrices - used in a scenario where ID is predicted for a query sequence but not for the target sequences - have at least comparable performance. However, I also demonstrate that the variations in performance between 20x20 and 20x40 matrices are insignificant compared to the variation in obtained matrices that occurs when the underlying algorithm for calculation of substitution matrices is changed.
Temple University--Theses
Enright, Simon James. "Obsessive-compulsive disorder: anxiety disorder or schizotype? : a questionnaire and experimental investigation". Thesis, University of Reading, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.357850.
Pełny tekst źródłaSmolkina, Milana. "Epidemiological and genetic associations between Cannabis Use Disorder and Major Depressive Disorder". Thesis, King's College London (University of London), 2019. https://kclpure.kcl.ac.uk/portal/en/theses/epidemiological-and-genetic-associations-between-cannabis-use-disorder-and-major-depressive-disorder(aae240ea-e4b3-4c30-8fc0-fba14831b3a1).html.
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