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Artykuły w czasopismach na temat "Differential display polymerase chain reaction amplification"
Kretzler, M., G. Fan, D. Rose, L. J. Arend, J. P. Briggs i L. B. Holzman. "Novel mouse embryonic renal marker gene products differentially expressed during kidney development". American Journal of Physiology-Renal Physiology 271, nr 3 (1.09.1996): F770—F777. http://dx.doi.org/10.1152/ajprenal.1996.271.3.f770.
Pełny tekst źródłaLiu, Yong-Gang, Yuan-Zhu Xiong i Chang-Yan Deng. "Isolation, Sequence Analysis and Expression Profile of a Novel Swine Gene Differentially Expressed in the Longissimus Dorsi Muscle Tissues from Landrace×Large White Cross-combination". Acta Biochimica et Biophysica Sinica 37, nr 3 (1.03.2005): 186–91. http://dx.doi.org/10.1093/abbs/37.3.186.
Pełny tekst źródłaSelisana, S. M., M. J. Yanoria, B. Quime, C. Chaipanya, G. Lu, R. Opulencia, G. L. Wang i in. "Avirulence (AVR) Gene-Based Diagnosis Complements Existing Pathogen Surveillance Tools for Effective Deployment of Resistance (R) Genes Against Rice Blast Disease". Phytopathology® 107, nr 6 (czerwiec 2017): 711–20. http://dx.doi.org/10.1094/phyto-12-16-0451-r.
Pełny tekst źródłaMahalingam, Ramamurthy, Gejiao Wang i Halina T. Knap. "Polygalacturonase and Polygalacturonase Inhibitor Protein: Gene Isolation and Transcription in Glycine max - Heterodera glycines Interactions". Molecular Plant-Microbe Interactions® 12, nr 6 (czerwiec 1999): 490–98. http://dx.doi.org/10.1094/mpmi.1999.12.6.490.
Pełny tekst źródłaReysenbach, A. L., L. J. Giver, G. S. Wickham i N. R. Pace. "Differential amplification of rRNA genes by polymerase chain reaction." Applied and Environmental Microbiology 58, nr 10 (1992): 3417–18. http://dx.doi.org/10.1128/aem.58.10.3417-3418.1992.
Pełny tekst źródłaJay, Venita, Sue MacNeill i Maria Zielenska. "MYCN Amplification in Pediatric Brain Tumors: Detection by Differential Polymerase Chain Reaction". Journal of Histotechnology 20, nr 2 (czerwiec 1997): 115–18. http://dx.doi.org/10.1179/his.1997.20.2.115.
Pełny tekst źródłaBombeccari, G. P., G. P. Guzzi, F. Pallotti i F. Spadari. "Focal Epithelial Hyperplasia: Polymerase Chain Reaction Amplification as a Differential Diagnosis Tool". American Journal of Dermatopathology 31, nr 1 (luty 2009): 98–100. http://dx.doi.org/10.1097/dad.0b013e31818ffc04.
Pełny tekst źródłaWang, Chao, Fan Meng, Yanping Huang, Nongyue He i Zhu Chen. "Design and Implementation of Polymerase Chain Reaction Device for Aptamers Selection of Tumor Cells". Journal of Nanoscience and Nanotechnology 20, nr 3 (1.03.2020): 1332–40. http://dx.doi.org/10.1166/jnn.2020.17356.
Pełny tekst źródłaChen, Yihua, Bin Wang, Song Weining i Grant Daggard. "Anchor primer associated problems in differential display reverse transcription polymerase chain reaction". Analytical Biochemistry 329, nr 1 (czerwiec 2004): 145–47. http://dx.doi.org/10.1016/j.ab.2004.02.030.
Pełny tekst źródłaGramlich, Terry L., Christine R. Fritsch, David Maurer, Mary Eberle i Ted S. Gansler. "Differential Polymerase Chain Reaction Assay of Cyclin Dl Gene Amplification in Esophageal Carcinoma". Diagnostic Molecular Pathology 3, nr 4 (grudzień 1994): 255–59. http://dx.doi.org/10.1097/00019606-199412000-00007.
Pełny tekst źródłaRozprawy doktorskie na temat "Differential display polymerase chain reaction amplification"
Mellick, Albert S. Jr, i n/a. "Tissue Specific Gene Expression Patterning and Carcinogenesis". Griffith University. School of Health Science, 2004. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20041102.114313.
Pełny tekst źródłaBalch, Signe Gyrite. "Cloning of novel macrophage-specific genes using differential-display PCR". Thesis, University of Oxford, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.312248.
Pełny tekst źródłaChen, Hong. "Identification of novel Wilms' tumor related genes by using differential display polymerase chain reaction, DD-PCR". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/mq22583.pdf.
Pełny tekst źródłaReed, Deborah G. "Characterization of the A/B regulon in tobacco (Nicotiana tabacum)". Thesis, Virginia Tech, 2003. http://hdl.handle.net/10919/10159.
Pełny tekst źródłaMaster of Science
Rae, Fiona Karen. "Identification and characterisation of genes expressed in renal cell carcinoma". Thesis, Queensland University of Technology, 2001.
Znajdź pełny tekst źródła"Identification of peroxisome proliferator-activated receptor alpha (PPARα)-dependent genes involved in peroxisome proliferator-induced short-term pleiotropic responses using fluorescent differential display technique". 2000. http://library.cuhk.edu.hk/record=b5895799.
Pełny tekst źródłaThesis (M.Phil.)--Chinese University of Hong Kong, 2000.
Includes bibliographical references (leaves 206-226).
Abstracts in English and Chinese.
Abstract --- p.i
Abstract (Chinese Version) --- p.iv
Acknowledgements --- p.vii
Table of Contents --- p.viii
List of Abbreviations --- p.xiv
List of Figures --- p.xvii
List of Tables --- p.xxiv
Chapter Chapter 1 --- Introduction --- p.1
Chapter Chapter 2 --- Literature review --- p.3
Chapter 2.1 --- Peroxisomes --- p.3
Chapter 2.2 --- Peroxisome proliferators --- p.5
Chapter 2.3 --- Human exposure pathways to peroxisome proliferators --- p.5
Chapter 2.4 --- Peroxisome proliferator-induced pleiotropic effects in rodents --- p.7
Chapter 2.4.1 --- Short-term effects --- p.7
Chapter 2.4.1.1 --- Hepatomegaly --- p.7
Chapter 2.4.2.1 --- Peroxisome proliferation --- p.8
Chapter 2.4.1.3 --- Alteration of gene transcriptions --- p.8
Chapter 2.4.2 --- Long-term effect --- p.9
Chapter 2.5 --- Mechanisms of actions of peroxisome proliferators --- p.9
Chapter 2.5.1 --- Substrate overload --- p.9
Chapter 2.5.2 --- Receptor-mediated --- p.11
Chapter 2.6 --- Peroxisome proliferator-activated receptors (PPARs) --- p.11
Chapter 2.6.1 --- Structure of PPARs --- p.11
Chapter 2.6.2 --- Tissue-specific expression of PPARs --- p.15
Chapter 2.6.3 --- Physiological functions of PPARs --- p.19
Chapter 2.6.3.1 --- PPARα --- p.19
Chapter 2.6.3.2 --- PPARγ --- p.21
Chapter 2.6.3.3 --- PPARδ --- p.23
Chapter 2.7 --- Role of PPARα involved in peroxisome proliferator-induced pleiotropic responses --- p.24
Chapter 2.7.1 --- Short-term effects --- p.24
Chapter 2.7.2 --- Long-term effect --- p.24
Chapter 2.8 --- Mechanisms of peroxisome proliferator-induced hepatocarcinogenesis --- p.25
Chapter 2.8.1 --- Oxidative stress --- p.25
Chapter 2.8.2 --- Suppression of apoptosis --- p.26
Chapter 2.8.3 --- Increased cell proliferation --- p.27
Chapter 2.9 --- Species difference to peroxisome proliferator-induced pleiotropic effects --- p.28
Chapter 2.10 --- Fluorescent differential display (FDD) --- p.32
Chapter Chapter 3 --- Objectives --- p.35
Chapter Chapter 4 --- Materials and methods --- p.37
Chapter 4.1 --- Animals and treatments --- p.37
Chapter 4.1.1 --- Materials --- p.37
Chapter 4.1.2 --- Methods --- p.37
Chapter 4.2 --- Serum triglyceride and cholesterol analyses --- p.39
Chapter 4.2.1 --- Materials --- p.41
Chapter 4.2.2 --- Methods --- p.41
Chapter 4.2.2.1 --- Serum preparation --- p.41
Chapter 4.2.2.2 --- Triglyceride determination --- p.41
Chapter 4.2.2.3 --- Cholesterol determination --- p.42
Chapter 4.3 --- Statistical analysis --- p.42
Chapter 4.4 --- Tail-genotyping --- p.42
Chapter 4.4.1 --- Materials --- p.44
Chapter 4.4.2 --- Methods. --- p.44
Chapter 4.4.2.1 --- Preparation of genomic tail DNA --- p.44
Chapter 4.4.2.2 --- PCR reaction --- p.45
Chapter 4.5 --- Total RNA isolation --- p.45
Chapter 4.5.1 --- Materials --- p.48
Chapter 4.5.2 --- Methods --- p.48
Chapter 4.6 --- DNase I treatment --- p.48
Chapter 4.6.1 --- Materials --- p.49
Chapter 4.6.2 --- Methods --- p.49
Chapter 4.7 --- Reverse transcription of mRNA and fluorescent PCR amplification --- p.50
Chapter 4.7.1 --- Materials --- p.50
Chapter 4.7.2 --- Methods --- p.53
Chapter 4.8 --- Fluorescent differential display (FDD) --- p.53
Chapter 4.8.1 --- Materials --- p.53
Chapter 4.8.2 --- Methods --- p.54
Chapter 4.9 --- Excision of differentially expressed cDNA fragments --- p.54
Chapter 4.9.1 --- Materials --- p.57
Chapter 4.9.2 --- Methods --- p.57
Chapter 4.10 --- Reamplification of differentially expressed fragments --- p.57
Chapter 4.10.1 --- Materials --- p.60
Chapter 4.10.2 --- Methods --- p.60
Chapter 4.11 --- Subcloning of reamplified cDNA fragments --- p.62
Chapter 4.11.1 --- PCR-TRAP® cloning system --- p.62
Chapter 4.11.1.1 --- Materials --- p.63
Chapter 4.11.1.2 --- Methods --- p.63
Chapter 4.11.2 --- AdvaTage´ёØ PCR cloning system --- p.65
Chapter 4.11.2.1 --- Materials --- p.65
Chapter 4.11.2.2 --- Methods --- p.66
Chapter 4.12 --- Purification of plasmid DNA from recombinant clones --- p.69
Chapter 4.12.1 --- Materials --- p.69
Chapter 4.12.2 --- Methods --- p.69
Chapter 4.13 --- DNA sequencing of differentially expressed cDNA fragments --- p.70
Chapter 4.13.1 --- CEQ 2000 Dye Terminator Cycle Sequence system --- p.71
Chapter 4.13.1.1 --- Materials --- p.71
Chapter 4.13.1.2 --- Methods --- p.71
Chapter 4.13.2 --- ABI PRISM´ёØ dRhodamine Terminator Cycle Sequencing system --- p.72
Chapter 4.13.2.1 --- Materials --- p.72
Chapter 4.13.2.2 --- Methods --- p.72
Chapter 4.13.3 --- Homology search against computer databases --- p.73
Chapter 4.14 --- Northern analysis of differentially expressed cDNA fragments --- p.73
Chapter 4.14.1 --- Formaldehyde gel electrophoresis of total RNA --- p.74
Chapter 4.14.1.1 --- Materials --- p.74
Chapter 4.14.1.2 --- Methods --- p.74
Chapter 4.14.2 --- Preparation of cDNA probes for hybridization --- p.74
Chapter 4.14.2.1 --- PCR DIG labeling --- p.75
Chapter 4.14.2.1.1 --- Materials --- p.75
Chapter 4.14.2.1.2 --- Methods --- p.75
Chapter 4.14.2.2 --- Random Prime cDNA DIG labeling --- p.75
Chapter 4.14.2.2.1 --- Materials --- p.75
Chapter 4.14.2.2.2 --- Methods --- p.76
Chapter 4.14.3 --- Purification of DNA from agarose gel --- p.77
Chapter 4.14.3.1 --- Materials --- p.77
Chapter 4.14.3.2 --- Methods --- p.78
Chapter 4.14.4 --- Hybridization --- p.78
Chapter 4.14.4.1 --- Materials --- p.78
Chapter 4.14.4.2 --- Methods --- p.73
Chapter 4.14.5 --- Synthesis of mouse GAPDH probe from normalization --- p.80
Chapter 4.14.5.1 --- Materials --- p.80
Chapter 4.14.5.2 --- Methods --- p.80
Chapter Chapter 5 --- Results --- p.82
Chapter 5.1 --- Liver morphology --- p.82
Chapter 5.2 --- Liver weight --- p.82
Chapter 5.3 --- Serum triglyceride and cholesterol levels --- p.88
Chapter 5.4 --- Confirmation of genotypes --- p.91
Chapter 5.5 --- DNase I treatment --- p.91
Chapter 5.6 --- FDD RT-PCR and band excision --- p.98
Chapter 5.7 --- Reamplification of excised cDNA fragments --- p.111
Chapter 5.8 --- Subcloning of reamplified cDNA fragments --- p.121
Chapter 5.9 --- DNA sequencing of subcloned cDNA fragments --- p.124
Chapter 5.10 --- Confirmation of the differentially expressed cDNA fragments by Northern blot analysis --- p.132
Chapter 5.11 --- Temporal expression pattern of differentially expressed genes --- p.157
Chapter 5.12 --- Tissue distribution pattern of differentially expressed genes --- p.171
Chapter Chapter 6 --- Discussions --- p.183
Chapter 6.1 --- "Lack of hepatomegaly, hypotriglyceridemia and hepatic nodule formation in PPARα (-/-) mice" --- p.184
Chapter 6.2 --- "Identification of PPARα-dependent and Wy-14,643 responsive genes" --- p.185
Chapter 6.3 --- Functional roles of the isolated cDNA fragments --- p.186
Chapter 6.3.1 --- Fragments B14 and H4 --- p.187
Chapter 6.3.2 --- Fragment H1 --- p.189
Chapter 6.3.3 --- Fragment H5 --- p.192
Chapter 6.3.4 --- Fragment H8 --- p.194
Chapter 6.4 --- Temporal expression patterns of the isolated cDNA fragments --- p.196
Chapter 6.5 --- Tissue distribution patterns of the isolated cDNA fragments --- p.197
Chapter Chapter 7 --- Conclusions --- p.200
Chapter Chapter 8 --- Future studies --- p.204
Chapter 8.1 --- Subcloning and characterization of the other differentially expressed genes --- p.204
Chapter 8.2 --- Overexpression and inhibition expression of specific genes --- p.204
Chapter 8.3 --- Generating transgenic mice with target disruption of specific gene --- p.205
References --- p.206
"Identification of differentially expressed genes in fibroblasts from human hypertrophic scars by using differential display RT-PCR technique". 1998. http://library.cuhk.edu.hk/record=b5889768.
Pełny tekst źródłaThesis (M.Phil.)--Chinese University of Hong Kong, 1998.
Includes bibliographical references (leaves 110-120).
Abstract also in Chinese.
Title --- p.i
Abstract --- p.ii
Acknowledgement --- p.iv
Abbreviations --- p.v
Abbreviation Table for Amino Acids --- p.vi
Table of Contents --- p.vii
List of Figures --- p.xii
List of Tables --- p.xv
Chapter Chapter 1 --- Introduction --- p.1
Chapter Chapter 2 --- Literature review --- p.2
Chapter Part I --- Hypertrophic Scar
Chapter 2.1 --- Definition of hypertrophic scar --- p.2
Chapter 2.2 --- Pathology --- p.2
Chapter 2.3 --- Epidemiology findings --- p.3
Chapter 2.3.1 --- Ethnicity --- p.3
Chapter 2.3.2 --- Age --- p.3
Chapter 2.3.3 --- Body location --- p.3
Chapter 2.4 --- Mechanism of cutaneous wound healing --- p.4
Chapter 2.4.1 --- Phase I - Haemostasis and inflammation --- p.4
Chapter 2.4.1.1 --- Haemostasis --- p.6
Chapter 2.4.1.2 --- Early phase of inflammation --- p.6
Chapter 2.4.1.3 --- Late phase of inflammation --- p.7
Chapter 2.4.2 --- Phase II - Re-epithelialization --- p.7
Chapter 2.4.2.1 --- Migration of epidermal keratinocytes --- p.8
Chapter 2.4.2.2 --- Migration of fibroblasts --- p.8
Chapter 2.4.2.3 --- Angiogenesis --- p.9
Chapter 2.4.3 --- Phase III - Tissue remodeling --- p.10
Chapter 2.4.3.1 --- Cell maturation and apoptosis --- p.10
Chapter 2.4.3.2 --- Exrtracellular matrix remodeling --- p.10
Chapter 2.5 --- Alteration of wound healing - Possible pathogenic factors of hypertrophic scar --- p.11
Chapter 2.5.1 --- Changes in Phase I-Inflammation --- p.13
Chapter 2.5.2 --- Changes in Phase II - Re-epithelialization/ tissue formation --- p.14
Chapter 2.5.3 --- Changes in Phase III - Tissue remodeling --- p.15
Chapter 2.6 --- The Role of fibroblasts in the formation of hypertrophic scar --- p.16
Chapter 2.6.1 --- Functions of fibroblasts in wound healing --- p.16
Chapter 2.6.2 --- Suggested aetiological role in the formation of hypertrophic scar fibroblasts --- p.16
Chapter 2.6.2.1 --- Fibroproliferation disorder --- p.18
Chapter 2.6.2.2 --- Extracellular Matrix remodeling disorder --- p.18
Chapter a) --- CoUaqen --- p.18
Chapter b) --- Proteoglycan --- p.19
Chapter 2.6.2.3 --- Other differentially expressed factors --- p.20
Chapter 2.7 --- Treatment of hypertrophic scar --- p.21
Chapter Part II --- Differential Display
Chapter 2.8 --- Current approaches for the studies of differential gene expression --- p.23
Chapter 2.9 --- Comparison amongst different approaches --- p.23
Chapter 2.10 --- The strategy of Differential Display RT-PCR (DDRT-PCR) --- p.24
Chapter 2.11 --- The application of DDRT-PCR to identify differentially expressed genes --- p.26
Chapter Chapter 3 --- Aims and Strategies --- p.27
Chapter Chapter 4 --- Methods and Materials --- p.29
Chapter 4.1 --- Materials --- p.29
Chapter 4.2 --- Clinical specimen collection --- p.31
Chapter 4.3 --- Primary explant culture --- p.31
Chapter 4.4 --- Immunohistochemical staining --- p.32
Chapter 4.5 --- Total RNA extraction --- p.32
Chapter 4.6 --- DNase I digestion --- p.33
Chapter 4.7 --- Differential display-RTPCR (DD-RTPCR) --- p.33
Chapter 4.8 --- Polyacrylamide gel electrophoresis --- p.34
Chapter 4.9 --- Reamplification of the differentially expressed fragments --- p.35
Chapter 4.10 --- Molecular cloning of the DNA fragments --- p.35
Chapter 4.11 --- Screening and miniprep of the plasmid DNA --- p.36
Chapter 4.12 --- Cycle sequencing --- p.38
Chapter 4.13 --- Data analysis --- p.38
Chapter 4.14 --- RT-PCR --- p.39
Chapter 4.15 --- Probe labeling by PCR with DIG-dUTP --- p.40
Chapter 4.16 --- Southern blotting --- p.41
Chapter Chapter5 --- p.42
Chapter 5.1 --- Clinical Specimen --- p.42
Chapter 5.2 --- Primary explant culture --- p.42
Chapter 5.3 --- The total RNA extraction from the cultured fibroblast --- p.45
Chapter 5.4 --- Differential display RT-PCR --- p.47
Chapter 5.5 --- Reamplification of the DNA fragments --- p.49
Chapter 5.6 --- Molecular cloning of the DNA fragment --- p.53
Chapter 5.7 --- DNA sequencing of the inserts --- p.58
Chapter 5.8 --- Analysis and identification of the DNA sequences --- p.62
Chapter 5.9 --- Semi-quantitative RT-PCR analysis of the differentially expressed genes --- p.76
Chapter Chapter6 --- p.87
Chapter Part I --- Validity of the Findings
Chapter 6.1 --- The Limitation of Tissue Sampling --- p.87
Chapter 6.2 --- Tissue Culture model --- p.88
Chapter 6.3 --- Differential Display RT-PCR --- p.89
Chapter 6.3.1 --- Identification of the differentially expressed genes --- p.89
Chapter 6.3.2 --- Confirmation of the differentially expressed genes --- p.91
Chapter 6.4 --- Technical difficulties and Limitations --- p.92
Chapter 6.4.1 --- Sampling --- p.92
Chapter 6.4.2 --- Primary tissue culture --- p.93
Chapter Part II --- Significance and Future Studies
Chapter 6.5 --- Down-regulation of thrombospondin 1 (TSP 1) in the hypertrophic scar fibroblasts --- p.94
Chapter 6.6 --- Biochemical and biological functions of TSP1 --- p.96
Chapter 6.6.1 --- The biochemical functions of TSP1 --- p.96
Chapter 6.6.2 --- The biochemical functions of TSP1 --- p.97
Chapter 6.7 --- The role of TSP 1 in the pathogenesis of hypertrophic scar --- p.98
Chapter 6.7.1 --- Down-regulation of TSP 1 may be responsible for the excessive microvessels in hypertrophic scar --- p.98
Chapter 6.7.2 --- Down-regulation of TSP 1 may be responsible for the failure of the apoptosis of the fibroblasts in the hypertrophic scar --- p.101
Chapter 6.8 --- Expression of TSP 1 during wound healing --- p.103
Chapter 6.9 --- Expression of TSP 1 in hypertrophic scarring --- p.107
Chapter 6.10 --- Cytochrome b561 and its biological function --- p.109
Chapter 6.11 --- Future studies --- p.108
Chapter 6.11.1 --- The expression of TSP 1 in hypertrophic scarring and normal wound healing --- p.108
Chapter 6.11.2 --- The expression of cytochrome b561 --- p.109
Chapter 6.11.3 --- A full scale study of differential display RT-PCR --- p.109
References --- p.110
Appendices --- p.121
Chapter I --- The complete mRNA sequence of thrombospondin1 precursor --- p.121
Chapter II --- The mRNA sequence of cytochrome b561 --- p.123
Książki na temat "Differential display polymerase chain reaction amplification"
Colonna-Romano, Sergio. Differential-display reverse transcription-PCR (DDRT-PCR). Berlin: Springer-Verlag, 1998.
Znajdź pełny tekst źródła(Editor), R. A. Leslie, i H. A. Robertson (Editor), red. Differential Display: A Practical Approach. Oxford University Press, USA, 2000.
Znajdź pełny tekst źródła(Editor), R. A. Leslie, i H. A. Robertson (Editor), red. Differential Display: A Practical Approach. Oxford University Press, USA, 2000.
Znajdź pełny tekst źródłaA, Leslie Ronald, i Robertson H, red. Differential display: A practical approach. Oxford: Oxford University Press, 2000.
Znajdź pełny tekst źródłaPeng, Liang, i Pardee Arthur B. 1921-, red. Differential display methods and protocols. Totowa, N.J: Humana Press, 1997.
Znajdź pełny tekst źródłaMcClelland, Michael, red. Expression Genetics: Differential Display (Biotechniques Update Series). EATON PUBLISHING, 1999.
Znajdź pełny tekst źródłaPeng, Liang, Meade Jonathan D i Pardee Arthur B. 1921-, red. Differential display methods and protocols. Wyd. 2. Totowa, N.J: Humana Press, 2005.
Znajdź pełny tekst źródłaPardee, Arthur B., Peng Liang i Jonathan Meade. Differential Display Methods and Protocols. Humana Press, 2010.
Znajdź pełny tekst źródła(Editor), Peng Liang, Jonathan Meade (Editor) i Arthur B. Pardee (Editor), red. Differential Display Methods and Protocols (Methods in Molecular Biology). Wyd. 2. Humana Press, 2005.
Znajdź pełny tekst źródłaCzęści książek na temat "Differential display polymerase chain reaction amplification"
Brandt, B., U. Vogt, C. Griwatz, F. Harms i K. S. Zänker. "Detection of Amplified Oncogenes by Differential Polymerase Chain Reaction". W Methods in DNA Amplification, 55–64. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2530-1_7.
Pełny tekst źródłaShepard, Brett D., i Michael S. Gilmore. "Identification of Virulence Genes in Enterococcus faecalis by Differential Display Polymerase Chain Reaction". W Streptococci and the Host, 777–79. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1825-3_183.
Pełny tekst źródłaAli, Manir, i John D. Isaacs. "Differential Display Reverse Transcription-Polymerase Chain Reaction to Identify Novel Biomolecules in Arthritis Research". W Arthritis Research, 329–47. Totowa, NJ: Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-402-5_23.
Pełny tekst źródłaAn, Gang, i Robert Veltr. "Differential Display Polymerase Chain Reaction Using Chemiluminescent Detection". W Luminescence Biotechnology, 203–9. CRC Press, 2001. http://dx.doi.org/10.1201/9781420041804.ch13.
Pełny tekst źródła"Differential Display Polymerase Chain Reaction Using Chemiluminescent Detection". W Luminescence Biotechnology, 225–32. CRC Press, 2001. http://dx.doi.org/10.1201/9781420041804-16.
Pełny tekst źródłaIto, Takashi, i Yoshiyuki Sakaki. "[17] Fluorescent differential display: A fast and reliable method for message display polymerase chain reaction". W Methods in Enzymology, 298–309. Elsevier, 1999. http://dx.doi.org/10.1016/s0076-6879(99)03019-0.
Pełny tekst źródłaRaporty organizacyjne na temat "Differential display polymerase chain reaction amplification"
Morrison, Mark, i Joshuah Miron. Molecular-Based Analysis of Cellulose Binding Proteins Involved with Adherence to Cellulose by Ruminococcus albus. United States Department of Agriculture, listopad 2000. http://dx.doi.org/10.32747/2000.7695844.bard.
Pełny tekst źródła