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Artykuły w czasopismach na temat "Diarrhetic shellfish poisoning"
Lloyd, Jennifer K., Jeffrey S. Duchin, Jerry Borchert, Harold Flores Quintana i Alison Robertson. "Diarrhetic Shellfish Poisoning, Washington, USA, 2011". Emerging Infectious Diseases 19, nr 8 (sierpień 2013): 1314–16. http://dx.doi.org/10.3201/eid1908.121824.
Pełny tekst źródłaMaranda, Lucie, i Yuzuru Shimizu. "Diarrhetic Shellfish Poisoning in Narragansett Bay". Estuaries 10, nr 4 (grudzień 1987): 298. http://dx.doi.org/10.2307/1351887.
Pełny tekst źródłaScoging, Anne, i M. Bahl. "Diarrhetic shellfish poisoning in the UK". Lancet 352, nr 9122 (lipiec 1998): 117. http://dx.doi.org/10.1016/s0140-6736(98)85023-x.
Pełny tekst źródłaIgboeli, Okechukwu O., i Isaac U. Asuzu. "‘Diarrhetic’ Type Shellfish Poisoning in Nigeria". Bulletin of Environmental Contamination and Toxicology 84, nr 1 (30.09.2009): 15–18. http://dx.doi.org/10.1007/s00128-009-9885-3.
Pełny tekst źródłaMadigan, Thomas L., Ken G. Lee, David J. Padula, Paul McNabb i Andrew M. Pointon. "Diarrhetic shellfish poisoning (DSP) toxins in South Australian shellfish". Harmful Algae 5, nr 2 (marzec 2006): 119–23. http://dx.doi.org/10.1016/j.hal.2004.12.005.
Pełny tekst źródłaSuárez-Gómez, Blanca, María L. Souto, Patricia G. Cruz, José J. Fernández i Manuel Norte. "New Targets in Diarrhetic Shellfish Poisoning Control". Journal of Natural Products 68, nr 4 (kwiecień 2005): 596–99. http://dx.doi.org/10.1021/np040183t.
Pełny tekst źródłaQiu, Xianxin, Longjie Zhong, Ying Gan, Kaiqi Su, Shihui Hu i Ping Wang. "A method combining a kit with the Bionic e-Eye for rapid on site detection of diarrhetic shellfish poisoning". Analytical Methods 10, nr 22 (2018): 2604–13. http://dx.doi.org/10.1039/c8ay00676h.
Pełny tekst źródłaSidari, L., P. Nichetto, S. Cok, S. Sosa, A. Tubaro, G. Honsell i R. Della Loggia. "Phytoplankton selection by mussels, and diarrhetic shellfish poisoning". Marine Biology 131, nr 1 (29.04.1998): 103–11. http://dx.doi.org/10.1007/s002270050301.
Pełny tekst źródłaGarthwaite, Ian, Kathryn M. Ross, Christopher O. Miles, Lyn R. Briggs, Neale R. Towers, Teresa Borrell i Phil Busby. "Integrated Enzyme-Linked Immunosorbent Assay Screening System for Amnesic, Neurotoxic, Diarrhetic, and Paralytic Shellfish Poisoning Toxins Found in New Zealand". Journal of AOAC INTERNATIONAL 84, nr 5 (1.09.2001): 1643–48. http://dx.doi.org/10.1093/jaoac/84.5.1643.
Pełny tekst źródłaS�chet, V., P. Safran, P. Hovgaard i T. Yasumoto. "Causative species of diarrhetic shellfish poisoning (DSP) in Norway". Marine Biology 105, nr 2 (czerwiec 1990): 269–74. http://dx.doi.org/10.1007/bf01344296.
Pełny tekst źródłaRozprawy doktorskie na temat "Diarrhetic shellfish poisoning"
Burgess, Vanessa Anne, i n/a. "Toxicology Investigations With The Pectenotoxin-2 Seco Acids". Griffith University. School of Public Health, 2003. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20030905.090222.
Pełny tekst źródłaBurgess, Vanessa Anne. "Toxicology Investigations With The Pectenotoxin-2 Seco Acids". Thesis, Griffith University, 2003. http://hdl.handle.net/10072/365382.
Pełny tekst źródłaThesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Public Health
Faculty of Health Sciences
Full Text
Windust, Anthony James. "The physiological ecology of diarrhetic shellfish poisoning (DSP) toxin production by the dinoflagellate Exuviaella lima (Ehr.) Bütschli". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ36593.pdf.
Pełny tekst źródłaMarin?, Geisi Ferreira. "Detec??o de ?cido ocadaico em cultivo de mexilh?es Perna perna (Linn?, 1758) e identifica??o do fitopl?ncton potencialmente produtor, em Maci?is, Angra dos Reis, RJ". Universidade Federal Rural do Rio de Janeiro, 2007. https://tede.ufrrj.br/jspui/handle/tede/860.
Pełny tekst źródłaCoordena??o de Aperfei?oamento de Pessoal de N?vel Superior
The okadaic acid phycotoxin AO is produced by a group of micro seaweed known as Dinoflagellated. The mussels when feeding themselves from this micro seaweed accumulate in their hepatopancreas, this toxin, trigging in the human being the Syndrome or Diarrhetic Shellfish Poisoning (DSP). The symptoms appear at around 30 minutes after the consumption of the contaminated clam; the symptoms vary among abdominal nauseas, pains, vomits and diarrhea. When the toxin ingestion happens in amounts lower than 48 μg.g-1, the above described symptoms do not develop, however, its continued consumption favors the appearing of tumors in the gastrointestinal tract due to the high carcinogenic power of AO. This study intended to detect and quantify the diarrheic toxin AO in Perna perna mussels collected between the months of May and December of the 2006 and verification of the potentially toxic micro seaweed presence, in the seasons Spring/Summer (from September until December of 2006). Since May until december of 2006, mussels (Perna perna) were collected and analyzed regarding to the presence of the Phycotoxin Ao .In order to collect the micro seaweed were used a plankton net (20μm of mesh) and Bomb Rule 2000. The identification of the dinoflagellates ones was carried through in inverted biological microscope. The detection of AO in the mussels was carried through by High Efficiency of Liquid Chromatography with Fluorimetric Detection (HPLC-FC). The chromatographic results had indicated the presence of AO toxin in all the gotten mussel samples from May until October of 2006 in low concentrations. In the analyses of phytoplankton, the diatoms were the most representative group compared with the Dinoflagellated. The species Prorocentrum micans and P. gracile examined were not pointed as toxin producers until the moment. Among the Dino-flagellated potentially toxic were found the species : Dinophysis acuminata, D. tripos, D. rotundata and D. fortii. The results indicate the necessity of elaboration and effective application of a hygienic-sanitary controlling program of the clams as well as monitoring the environment, aiming above everything the public health Safety.
A ficotoxina ?cido ocadaico (AO) ? produzida por um grupo de microalgas conhecidas como dinoflagelados. Os mexilh?es ao se alimentarem destas microalgas acumulam em seu hepatop?ncreas, esta toxina, desencadeando no ser humano a S?ndrome ou Envenenamento Diarr?ico por Moluscos - EDM. Os sintomas se apresentam em torno de 30 minutos ap?s o consumo do molusco contaminado; variando entre n?useas, dores abdominais, v?mitos e diarr?ia. Quando a ingest?o da toxina acontece em quantidades inferiores a 48 μg.g-1, os sintomas acima descritos n?o se desenvolvem, por?m, seu consumo continuado favorece o surgimento de tumores no trato gastrointestinal devido ao poder carcinog?nico do AO. Este estudo pretendeu detectar e quantificar a toxina diarr?ica AO em mexilh?es Perna perna coletados entre os meses de maio e dezembro de 2006, e a verifica??o da presen?a de microalgas potencialmente t?xicas, nas esta??es primavera/ver?o (setembro a dezembro de 2006). De maio a dezembro de 2006, mexilh?es (Perna perna) foram coletados e analisados quanto ? presen?a da ficotoxina AO. As microalgas foram coletadas entre setembro e dezembro de 2006, com aux?lio de uma rede de pl?ncton (de 20μm de malha) e Bomba Rule 2000. A identifica??o dos dinoflagelados foi realizada em microsc?pio biol?gico invertido. A detec??o do AO nos mexilh?es foi realizada por Cromatografia L?quida de Alta Efici?ncia com Detec??o Fluorim?trica (CLAE-DF). Os resultados cromatogr?ficos indicaram a presen?a da toxina AO em todas as amostras obtidas de mexilh?es, de maio a outubro de 2006, em baixas concentra??es. Nas an?lises do fitopl?ncton, as diatom?ceas foram o grupo mais representativo comparado aos dinoflagelados. As esp?cies Prorocentrum micans e P. gracile observadas n?o foram apontadas como produtoras de toxinas at? o momento. Dentre os dinoflagelados potencialmente t?xicos foram encontradas as esp?cies: Dinophysis acuminata, D. tripos, D. rotundata e D. fortii. Os resultados indicam a necessidade da elabora??o e aplica??o efetiva de um programa de controle higi?nico-sanit?rio dos moluscos assim como monitoramento do ambiente, objetivando acima de tudo a seguran?a ? sa?de p?blica.
Manita, Diana Filipa Pereira. "Bioacessibilidade in vitro das biotoxinas marinhas ácido ocadaico, dinofisistoxina-2 e seus derivados em bivalves crus e cozinhados". Master's thesis, Universidade de Lisboa, Faculdade de Medicina Veterinária, 2017. http://hdl.handle.net/10400.5/13218.
Pełny tekst źródłaAs biotoxinas marinhas mais frequentes e abundantes em Portugal são as toxinas lipofílicas, nomeadamente as toxinas do grupo do ácido ocadaico (AO), que inclui as dinofisistoxinas (DTX1 e DTX2) e os seus derivados (DTX3), responsáveis pela intoxicação diarreica (DSP diarrhetic shellfish poisoning). A investigação da presença, variabilidade e transformação de biotoxinas marinhas em moluscos bivalves é de elevada importância não só para os consumidores e produtores de moluscos bivalves, mas também para as entidades reguladoras em segurança alimentar. Até à data, a quantidade de toxinas ingeridas nos alimentos tem sido considerada igual à quantidade de toxinas disponível para a absorção pelo corpo humano após o processo digestivo. Neste estudo, avaliou-se através de um modelo de digestão in vitro estático, a fração de AO, DTX2 e das suas formas esterificadas (DTX3), libertada do alimento para os fluidos digestivos (bioacessibilidade) em amostras naturalmente contaminadas de mexilhão, berbigão, conquilha e lingueirão. As amostras de mexilhão e conquilha apresentam regularmente um perfil de toxinas composto por AO e DTX2 nas suas formas livres, enquanto que o berbigão e o lingueirão têm uma elevada capacidade de biotransformação, o que conduz a que o seu perfil de toxinas seja constituído maioritariamente por formas esterificadas (DTX3). Amostras colhidas na costa Portuguesa foram utilizadas para avaliar a bioacessibilidade destes compostos, quer na matriz crua, como após cozedura a vapor. A Bioacessibilidade do conteúdo total de toxinas variou entre as espécies analisadas. A maior percentagem de toxinas bioacessiveis foi encontrada no mexilhão (86 ± 4 %), seguido da conquilha (74 ± 10 %), do lingueirão (71 ± 10 %) e, por último, do berbigão (59 ± 10 %). As toxinas que não se libertaram da matriz alimentar foram detetadas na fração nãobioacessivel, correspondente à fração excretada. Nas amostras de mexilhão e conquilha determinou-se mais AO na fração bioacessivel do que o presente na matriz alimentar antes da digestão. Não podendo haver mais AO do que aquele que foi ingerido, e tendo sido observado paralelamente um valor muito reduzido de AO esterificado na fração bioacessivel, sugere-se uma conversão dos compostos esterificados no composto parental durante o processo digestivo. A conversão no trato digestivo dos compostos menos tóxicos (DTX3) nos seus compostos parentais mais potentes, vem justificar os casos de intoxicação após o consumo de bivalves contendo um perfil de toxinas dominado por DTX3. No entanto, nas amostras de berbigão e lingueirão não foi observada a conversão das formas esterificadas de AO, sugerindo um perfil de formas esterificadas diferente do mexilhão e conquilha. O tratamento a vapor foi avaliado nas amostras de mexilhão e berbigão, tendo-se observado um aumento da concentração das toxinas lipofílicas, mas uma redução da bioacessibilidade dos compostos face às matrizes cruas, sendo de destacar que a bioacessibilidade estimada para as amostras de berbigão foi interior a 50%. Este estudo fornece novos dados relevantes que podem melhorar e levar a estudos de avaliação de risco em segurança alimentar mais precisos sobre estas toxinas. A avaliação de risco com base exclusivamente na ocorrência das toxinas DSP em moluscos bivalves pode conduzir a uma sobrestimação da exposição e levar a medidas regulamentares mais conservadoras do que tendo em conta a quantidade de toxinas que podem ser absorvidas pelos epitélios intestinais.
ABSTRACT - In vitro bioaccessibility of the marine biotoxins okadaic acid, dinophysistoxin-2 and their derivatives in raw and steamed shellfish - The most common and abundant biotoxins in Portugal are lipophilic toxins, including okadaic acid group (OA) toxins, which includes the dinophysistoxins (DTX1 and DTX2) and its derivatives (DTX3), responsible for the human food borne illness diarrhetic shellfish poisoning (DSP). Investigating the presence, variability and processing of marine biotoxins in bivalve molluscs is highly important not only for consumers and shellfish producers but also for governmental agencies with responsibilities on food safety. To date, the amount of toxins ingested in the food has been considered equal to the amount of toxins available for uptake by the human body after the digestive process. In this study, we evaluated through a static in vitro digestion model, the fraction of OA, DTX2 and DTX3 released from the food to the digestive fluids (bioaccessibility) in naturally contaminated mussels, donax clams, cockles, and razor clams. Bioaccessibility was assessed in both raw and steamed shellfish matrices. Higher bioaccessibilty was estimated for mussels (86 ± 4 %), followed by donax clams (74 ± 10 %), razor clams (71 ± 10 %) and finally cockles (59 ± 10 %). The amount of toxins not released into the digestive juices were detected in the non-bioaccessible fraction. In mussels and donax clams higher levels of OA were determined in the digestive juices than in the food matrix. Parallel to higher amounts of OA in bioaccessible fraction was observed a reduction of its esterified forms (DTX3), suggesting that DTX3 are converted into the parental compounds during the digestion process. This gastrointestinal conversion of less potent toxins into their more toxic parental compounds can be pointed out as a reason for human food poisonings after ingestion of contaminated shellfish containing mostly DTX3, as was previously observed in Portugal. However, conversion of DTX3 into OA was not observed in cockles and razor clams, suggesting that the suite of esterified compounds in these species is different from mussels and donax clams. Steaming of shellfish matrices, simulating a cooking treatment, lead to an increase of toxins concentration but significantly reduced their bioaccessibility, which was particular evident for cockles having the release of toxins been estimated by this in vitro model to levels below 50%. This study provides relevant new data that can improve and lead to more accurate food safety risk assessment studies concerning these toxins. Risk assessment based solely on DSP toxins occurrence in seafood can conduct to an overestimation of the exposure and lead to regulatory measures more conservative than taking into account the amount of toxins that can be absorbed by the intestinal epithelia.
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Rühl, Alexander [Verfasser]. "Entwicklung und Anwendung von flüssigchromatographischen Analyseverfahren zum Nachweis von Diarrhetic Shellfish Poisoning (DSP)-Toxinen / von Alexander Rühl". 2004. http://d-nb.info/971963800/34.
Pełny tekst źródłaKsiążki na temat "Diarrhetic shellfish poisoning"
Scotland. Food Protection (Emergency Prohibitions) (Diarrhetic Shellfish Poisoning) (East Coast) (Scotland) Order 2004. Stationery Office, The, 2004.
Znajdź pełny tekst źródłaScotland. Food Protection (Emergency Prohibitions) (Diarrhetic Shellfish Poisoning) (East Coast) (Scotland) Revocation Order 2004. Stationery Office, The, 2004.
Znajdź pełny tekst źródłaScotland. Food Protection (Emergency Prohibitions) (Diarrhetic Shellfish Poisoning) (East Coast) (No. 3) (Scotland) Order 2004. Stationery Office, The, 2004.
Znajdź pełny tekst źródłaScotland. Food Protection (Emergency Prohibitions) (Diarrhetic Shellfish Poisoning) (East Coast) (No. 2) (Scotland) Order 2004. Stationery Office, The, 2004.
Znajdź pełny tekst źródłaScotland. Food Protection (Emergency Prohibitions) (Diarrhetic Shellfish Poisoning) (East Coast) (No. 3) (Scotland) Revocation Order 2004. Stationery Office, The, 2004.
Znajdź pełny tekst źródłaScotland. Food Protection (Emergency Prohibitions) (Diarrhetic Shellfish Poisoning) (East Coast) (No. 2) (Scotland) Revocation Order 2004. Stationery Office, The, 2004.
Znajdź pełny tekst źródłaCzęści książek na temat "Diarrhetic shellfish poisoning"
Raine, R., A. M. Wilson, G. Hermann i J. P. Lacaze. "A Comparison of Assay Techniques for the Analysis of Diarrhetic Shellfish Poisoning Toxins in Shellfish". W Molluscan Shellfish Safety, 205–13. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6588-7_18.
Pełny tekst źródłaQuilliam, M. A., i N. W. Ross. "Analysis of Diarrhetic Shellfish Poisoning Toxins and Metabolites in Plankton and Shellfish by Ion-Spray Liquid Chromatography—Mass Spectrometry". W ACS Symposium Series, 351–64. Washington, DC: American Chemical Society, 1996. http://dx.doi.org/10.1021/bk-1995-0619.ch018.
Pełny tekst źródłaAune, Tore, i Magne Yndestad. "Diarrhetic Shellfish Poisoning". W Algal Toxins in Seafood and Drinking Water, 87–104. Elsevier, 1993. http://dx.doi.org/10.1016/b978-0-08-091811-2.50010-1.
Pełny tekst źródła"Prediction of Aquatic Biotoxin Potential in Fish and Shellfish Harvesting Areas: Ciguatera and Diarrheic Shellfish Poisoning". W Coastal Ocean Space Utilization 3, 309–20. CRC Press, 2002. http://dx.doi.org/10.1201/9781482271669-34.
Pełny tekst źródłaStreszczenia konferencji na temat "Diarrhetic shellfish poisoning"
Renyan, Liu, Xu Daoyan i Liang Yubo. "Preparation of Monoclonal Antibody against Okadaic Acid and Development of a Sensitive ELISA for Detection Diarrhetic Shellfish Poisoning in Shellfish". W 2009 International Conference on Environmental Science and Information Application Technology, ESIAT. IEEE, 2009. http://dx.doi.org/10.1109/esiat.2009.101.
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