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Książki na temat „Dementia”

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Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 30 lipca 2024

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1

Nori, Graham, i Warner James Dr, red. Dementia: Alzheimer's and other dementias. Wyd. 2. London: Class, 2002.

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2

Mendez, Mario F. Dementia: A clinical approach. Wyd. 3. Philadelphia, PA: Butterworth-Heinemann, 2003.

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Burns, Alistair, i Raymond Levy, red. Dementia. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-6805-6.

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Edwards, Allen Jack. Dementia. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4757-9963-7.

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Krishnamoorthy, Ennapadam S., Martin J. Prince i Jeffrey L. Cummings, red. Dementia. Cambridge: Cambridge University Press, 2009. http://dx.doi.org/10.1017/cbo9780511780615.

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Maj, Mario, i Norman Sartorius, red. Dementia. Chichester, UK: John Wiley & Sons, Ltd, 2002. http://dx.doi.org/10.1002/0470861878.

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7

Mahendra, B. Dementia. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3183-1.

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8

Quinn, Joseph F. Dementia. Oxford: John Wiley & Sons Ltd, 2013. http://dx.doi.org/10.1002/9781118656082.

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9

Maj, Mario, i Norman Sartorius, red. Dementia. Chichester, UK: John Wiley & Sons, Ltd, 2000. http://dx.doi.org/10.1002/0470842350.

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10

Hickey, Ellen M., i Michelle S. Bourgeois, red. Dementia. 2nd edition. | New York: Routledge, 2018. | Revised edition of: Dementia: from diagnosis to management: a functional approach/Michelle S. Bourgeois and Ellen M. Hickey, 2009.: Routledge, 2017. http://dx.doi.org/10.4324/9781315103891.

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11

Keen, Justin. Dementia. London: Office of Health Economics, 1992.

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12

Hardman, Lizabeth. Dementia. Detroit: Lucent Books, 2009.

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13

Hardman, Lizabeth. Dementia. Detroit: Lucent Books, 2009.

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14

Stackpole, Michael A. Dementia. London: Roc, 1994.

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15

Brice, Pitt, red. Dementia. Edinburgh: Churchill Livingstone, 1987.

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16

1953-, Maj Mario, i Sartorius N, red. Dementia. Wyd. 2. Chichester: Wiley, 2002.

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17

Executive, NHS Management. Dementia. Heywood: Health Publications Unit, 1994.

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18

S, Burns Alistair, i Levy Raymond, red. Dementia. London: Chapman & Hall Medical, 1994.

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19

S, Burns Alistair, O'Brien John DM i Ames David 1954-, red. Dementia. Wyd. 3. London: Hodder Arnold, 2005.

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20

Keen, Justin. Dementia. London: Office of Health Economics, 1992.

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21

Executive, NHS Management. Dementia. [London]: NHS Management Executive, 1994.

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22

D, Bowen James, red. Dementia. Philadelphia: Saunders, 2002.

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23

Patrick, McNamara. Dementia. Santa Barbara, Calif: Praeger, 2011.

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24

Biernacki, Claire. Dementia. New York: John Wiley & Sons, Ltd., 2007.

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25

T, DeKosky Steven, red. Dementia. Philadelphia: W.B. Saunders, 2000.

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26

DM, O'Brien John, Burns Alistair S i Levy Raymond, red. Dementia. Wyd. 2. London: Arnold, 2000.

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27

J, Whitehouse Peter, red. Dementia. Philadelphia: F.A. Davis, 1992.

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28

Fernandez, Evelina. Dementia. Alexandria, VA: Alexander Street Press, 2007.

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29

1953-, Maj Mario, i Sartorius N, red. Dementia. Chichester: John Wiley, 2000.

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30

1948-, Morris John C., red. Handbook of dementing illnesses. New York: Dekker, 1994.

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31

Beller, Jerry, i Beller Health. 14 Dementia Types: Alzheimers Lewy Body Dementia Frontotemporal Dementia Vascular Dementia Huntington's Disease Other Dementias. Independently Published, 2019.

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32

Beller, Jerry, i Beller Health. 2019 Dementia Overview: Alzheimers , Lewy Body Dementia , Frontotemporal Dementia , Vascular Dementia , Huntington's Disease , Other Dementias. Independently Published, 2019.

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33

Piggott, Margaret Ann. Neurochemical pathology of dementia. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0007.

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This chapter considers the neurodegenerative disorders Alzheimer’s disease (AD), Lewy body dementias (dementia with Lewy bodies (DLB) and Parkinson’s disease dementia(PDD)), frontotemporal dementia (FTD); and also vascular dementia (VaD) which results from cerebrovascular disease. These different conditions, which give rise to dementia syndromes, each have distinct neurochemical pathologies, with important implications for treatment. As increased age is the common risk factor generally associated with dementing illnesses, neurochemical changes are set in the context of the changes which occur during ageing. A detailed understanding of the neurotransmitter function in each condition can lead to rational drug design and treatment strategies appropriate for each group of patients. Neurochemical pathology in transmitter systems underlying clinical features of these disorders are reviewed.

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34

Brookes, Alpha. Dementia Signs : Frontotemporal Dementia: Dementia Meaning. Independently Published, 2021.

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35

Gan, Li. Cellular Mechanisms of Dementia. Redaktorzy Dennis S. Charney, Eric J. Nestler, Pamela Sklar i Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0054.

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Neurodegenerative dementias, including Alzheimer’s disease (AD), Parkinson’s disease (PD) and Frontotemporal dementia (FTD), pose enormous challenges for our aging society. Genetic and mechanistic studies have revealed common molecular and cellular pathways, including imbalanced proteostasis and aberrant innate immune responses. Key pathogens in AD, PD, and FTD accumulate and spread from one brain region to another, resulting in network dysfunction and cognitive decline. These diseases are multifactorial, caused by interactions among multiple genetic, epigenetic, and environmental factors and pathways. Combination therapies that target multiple pathways may also be needed to stop or delay the dementing conditions in neurodegenerative dementias.

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36

Zahn, Roland, i Alistair Burns. Dementia disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198779803.003.0001.

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This chapter provides a brief overview of the different forms of dementia syndromes and provides a simple algorithm for initial differential diagnosis. Rapidly progressive dementias have to be excluded which require specific investigations to detect Creutzfeldt–Jakob as well as inflammatory and autoimmune diseases. A lead symptom-based approach in patients with slowly progressive cognitive and behavioural impairments without neurological symptoms is applied: progressive and primary impairments in recent memory are characteristic of typical Alzheimer’s dementia, primary behavioural changes point to the behavioural variant of frontotemporal dementia, primary impairments of language or speech are distinctive for progressive aphasias, fluctuating impairments of attention are a hallmark of Lewy body dementia, whereas primary visuospatial impairments suggest a posterior cortical atrophy. The chapter further discusses updated vascular dementia guidelines and DSM-5 revisions of defining dementia. Current diagnostic criteria for the different dementias are referenced and the role of neuroimaging is illustrated.

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37

Engdahl, Sylvia. Dementia. Greenhaven Publishing LLC, 2013.

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38

Hughes, Julian, Stephen Louw i Steven R. Sabat, red. Dementia. Oxford University Press, 2005. http://dx.doi.org/10.1093/med/9780198566151.001.0001.

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39

Dickerson, Bradford, i Alireza Atri, red. Dementia. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199928453.001.0001.

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40

Armstrong, Richard. Dementia. Redaktorzy Patrick Davey i David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0228.

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Dementia is a syndrome defined by a persistent, progressive decline in multiple cognitive functions to a degree sufficient to detrimentally impact activities of daily living and social function. The syndromic diagnosis of dementia is useful, since the general management and economic burden of these patients remains similar, irrespective of etiology. However, a more precise etiological diagnosis must be sought, since disease-specific treatment is increasingly likely to be appropriate. The term ‘mild cognitive impairment’ (MCI) refers to an objective impairment in a cognitive function (usually memory) which does not impair activities of daily living. The aim underlying the use of this term is to identify those with the early pathophysiological changes of neurodegenerative disease. Nonetheless, ‘MCI’ is not synonymous with ‘early dementia’, as a significant proportion of those with MCI will not worsen. The term should therefore be used cautiously.

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41

Hydén, Lars-Christer. Dementia. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780199391578.003.0003.

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This chapter focuses on the changing brain of individuals living with dementia and the challenges produced by these changes. It is argued that to better understand what persons with changing brains can do as storytellers, together with other persons, it is necessary to add a social psychological perspective to more traditional psychological knowledge, which often is centered on a decontextualized individual and that person’s diseased brain. The chapter discusses the brain’s role in storytelling. Storytelling is a ubiquitous and important everyday activity, but also a very complex social activity taking many different resources into account; cognitive as well as semiotic resources are used, together with a host of emotional resources, to regulate social interaction. The focus is on the limitations of the clinical understanding of the effects of brain injuries and the theoretical concepts that are needed to understand how the brains of persons with dementia are affected.

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42

Quinn, Joseph. Dementia. Wiley & Sons, Incorporated, John, 2013.

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43

Dementia. Philadelphia: W.B. Saunders Co., 2002.

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44

Widera, Eric, i Rachelle Bernacki. Dementia. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0154.

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Dementia is caused by a variety of disorders that result in a progressive loss of both cognitive and functional abilities. Despite the heterogeneity of disorders, there is a common set of problems that patients and families face living with this syndrome. Symptoms such as pain, eating difficulties, depression, and agitation are all common. As the disease progresses to the advanced stages, the different disorders share a common functional trajectory that includes persistently severe disability with complete dependence on others for basic activities of daily living. Care for individuals with dementia should involve a number of important palliative interventions. Advance care planning should occur early on in the disease process as it is anticipated that an individual will lose capacity to make medical and financial decisions at some point in their illness; specialized programmes for end-of-life care, such as hospice, should be considered for all patients with advanced dementia.

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45

Burrell, James R., i John R. Hodges. Dementia. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199658602.003.0010.

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Cognitive neurology has exploded over the last century, and especially over the last 20 years. From the distinction of dementia as a pathological entity, rather than just ‘normal’ ageing, to more sophisticated sub-classification of dementia syndromes, much has been learned, though great challenges remain. From an incredible array of worthy research studies, ten landmark papers in the field of dementia are presented in this chapter. With regard to Alzheimer’s disease, the following are discussed: the initial description of the disease, both clinically and pathologically; the development of meaningful clinical assessment measures; the early clinical manifestations and genetic causes; the precursors to symptomatic treatment; the use of neuroimaging to identify amyloid pathology in vivo; and the staging of Alzheimer’s pathology. The clinical features and genetic causes of frontotemporal dementia, an important non-Alzheimer’s primary dementia syndrome seen especially in younger patients, are also discussed.

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46

Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson i David Ledingham. Dementia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0011.

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This chapter discusses the clinical features and evidence-based pharmacological management of dementia disorders (Alzheimer’s disease (AD), vascular dementia, dementia with Lewy bodies (DLB), Parkinson’s disease dementia (PDD), frontotemporal dementia (FTD), mixed dementia, and mild cognitive impairment (MCI)).

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47

Pace, Victor, Adrian Treloar i Sharon Scott, red. Dementia. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199237807.001.0001.

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48

Ames, David, Alistair Burns i John T. O'Brien. Dementia. CRC Press, 2010. http://dx.doi.org/10.1201/b13196.

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49

Quinn, Joseph. Dementia. Wiley & Sons, Incorporated, John, 2013.

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50

Stackpole, Michael A. Dementia. Signet, 1995.

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