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Artykuły w czasopismach na temat "Deficiency of assets"
GUPTA, UMESH C., Y. W. JAME, C. A. CAMPBELL, A. J. LEYSHON i W. NICHOLAICHUK. "BORON TOXICITY AND DEFICIENCY: A REVIEW". Canadian Journal of Soil Science 65, nr 3 (1.08.1985): 381–409. http://dx.doi.org/10.4141/cjss85-044.
Pełny tekst źródłaTaslina Pervin i Emdadul Hoque. "Profitability measurement of selected Islamic Shariah-based private commercial banks in Bangladesh: A comparative Study". World Journal of Advanced Research and Reviews 22, nr 1 (30.04.2024): 264–76. http://dx.doi.org/10.30574/wjarr.2024.22.1.0999.
Pełny tekst źródłaUdoh, Edet J., Sunday B. Akpan i Edikan Francis Uko. "Assessment of Sustainable Livelihood Assets of Farming Households in Akwa Ibom State, Nigeria". Journal of Sustainable Development 10, nr 4 (30.07.2017): 83. http://dx.doi.org/10.5539/jsd.v10n4p83.
Pełny tekst źródłaVerma, Preeti, Pankaj Gupta i Amarjeet Singh. "A Comprehensive Survey on Novel Fault Current Limiters in Wind Energy Conversion System". SAMRIDDHI : A Journal of Physical Sciences, Engineering and Technology 12, nr 01 (30.06.2020): 45–53. http://dx.doi.org/10.18090/samriddhi.v12i01.10.
Pełny tekst źródłaPolidi, T. "Accumulated Deficiency of Housing Investments in Russia:Threats and Prospects". Voprosy Ekonomiki, nr 4 (20.04.2014): 37–55. http://dx.doi.org/10.32609/0042-8736-2014-4-37-55.
Pełny tekst źródłaHawk, Ashton, Jeffrey J. Reuer i Andrew Garofolo. "The Impact of Firm Speed Capabilities on the Decision to Partner or Go It Alone". Strategy Science 6, nr 3 (wrzesień 2021): 191–208. http://dx.doi.org/10.1287/stsc.2020.0122.
Pełny tekst źródłaTretyakov, V. D. "Scientific Methodological Approach to Optimization of Machine-Building Enterprise’s Order Portfolio Execution Plan in the Conditions of Deficiency of Circulating Assets". Bulletin of Ural Federal University. Series Economics and Management 18, nr 2 (2019): 210–31. http://dx.doi.org/10.15826/vestnik.2019.18.2.011.
Pełny tekst źródłaAslinda, Wery, Elvyrah Faisal, Asriani i Bahja. "Workshop On The Implementation Of Balanced Nutrition Messages To Prevent Anemia In Female Adolescent". Jurnal Pengabdian Masyarakat: Svasta Harena 1, nr 2 (28.02.2022): 39–42. http://dx.doi.org/10.33860/jpmsh.v1i2.491.
Pełny tekst źródłaBotes, Vida, Rory Diver i Howard Davey. "Preserving the past: an accountability study of the annual reporting practices by leading museums in USA, UK and Europe." Corporate Ownership and Control 11, nr 1 (2013): 893–906. http://dx.doi.org/10.22495/cocv11i1c11p1.
Pełny tekst źródłaShroff, Nemit. "Real Effects of PCAOB International Inspections". Accounting Review 95, nr 5 (21.10.2019): 399–433. http://dx.doi.org/10.2308/accr-52635.
Pełny tekst źródłaRozprawy doktorskie na temat "Deficiency of assets"
Kouassi, Koffi Samir Rehmann. "L'extension d 'une procédure collective". Electronic Thesis or Diss., Toulon, 2020. http://www.theses.fr/2020TOUL0130.
Pełny tekst źródłaThe extension of a collective procedure is a jurisprudential creation enshrined in the law on the protection of companies of 26 July 2005. Its objective is to artificially reconstitute the debtor's prepared assets. Thus, it will consist in extending the initial collective procedure to one or more natural or legal persons. The extension of collective proceedings is of considerable benefit to the debtor insofar as it will offer him more possibilities and means to solve his difficulties as well as possible. The same applies to the creditor who will see the debtor's assets combined with those of a third party.Its legal basis is Article L. 621-2 of the French Commercial Code. It provides that the court must characterize the existence of a Confusion of patrimony or fictitious nature before pronouncing a judgment extending collective proceedings. Case law contributes significantly to the legislative evolution of the extension of collective proceedings. Despite the codification of this procedure, case law is omnipresent. This role is explained by the fact that the legislator gives the judge the power to interpret and assess the extension of collective proceedings, on the basis of its two causes, namely confusion of assets and fictitious acts. What is the discretion left to the judge? This is the latter's interpretation of the existence of fictitious or confusing assets. Thus, therefore, in order to pronounce an extension of collective proceedings on the basis of the Confusion of Assets, the judge must necessarily characterise the existence of abnormal financial relations or the inextricable interweaving of assets, namely that of the principal debtor and that of the person or persons to whom he would like to extend the collective proceedings.The assessment of the fictitious nature of the legal person is the least frequent case but remains the most complex to determine. Judges have a heavy responsibility to detect the fictitious nature of a legal person and most often encounter the various financial arrangements established within corporate groups.The need not to look for whether the confusion of assets between two natural or legal persons has caused prejudice to the principal debtor in order to extend his collective proceedings. This legislative framework shows that judges are free to extend collective proceedings, taking into account the fact that their decision is rigorously reviewed by the Court of Cassation. In addition, in view of the divergent interests of the debtor and the creditor, but also because of the impact of decisions to extend collective proceedings on the economic life of these two categories of persons, decisions ordering the extension of collective proceedings are subject to rigorous control by the Court of Cassation.However, while there are many advantages to this procedure, the fact remains that, in terms of its practice, some reforms could improve it
Truong, Thuong. "La responsabilité des associés des sociétés commerciales". Thesis, Paris 2, 2017. http://www.theses.fr/2017PA020051.
Pełny tekst źródłaThe liability of shareholders is a notion not often referred to in coursebook. In in bonis companies, and in the case of external relationships, the shareholder personal liability could be engaged for ‘ fault detachable’. However, the principle of a commitment to personal liability on the part of shareholders in relations with third parties is contested, due to the essentially internal nature of their activity In a collective procedure, the non-liability of the parent company for the acts of their subsidiary is challenged. The development of this challenge is facilitated by powerful weapons of the repressive arsenal, weapons to be used in a highly derogatory environment of collective procedures. There is an aggravating trend in the parent company responsibility, particularly in regard to social and environmental domains. The search for a better protection of the victims pushes the legislator to legislate on isolated problematic issues, distilling the irreversible character of partial and specific solutions, and thus forcing the passage towards the establishment of a presumption of liability of the parent company for the acts of their subsidiary. However, there is a large and effective repressive arsenal, and there are avenues to limit the parent company’s liability while involving them in their subsidiary problems
Kattaru, Dattavardhan M. "Quantitative food frequency questionnaire to assess iron and energy intake of adolescents in rural India". Online version, 2009. http://www.uwstout.edu/lib/thesis/2009/2009kattarud.pdf.
Pełny tekst źródłaVerma, Nitin. "A Pilot Survey to Assess the Vitamin A Status of Children Aged 6-72 months in the Ramu Region of Papua New Guinea". Thesis, The University of Sydney, 2000. http://hdl.handle.net/2123/355.
Pełny tekst źródłaVerma, Nitin. "A Pilot Survey to Assess the Vitamin A Status of Children Aged 6-72 months in the Ramu Region of Papua New Guinea". University of Sydney. NT Clinical School, Flinders University, 2000. http://hdl.handle.net/2123/355.
Pełny tekst źródłaPike, Gillian. "Investigation of the role of global haemostasis assays and bleeding scores in the assessment and management of patients with Factor XI deficiency". Thesis, University of Manchester, 2016. https://www.research.manchester.ac.uk/portal/en/theses/investigation-of-the-role-of-global-haemostasis-assays-and-bleeding-scores-in-the-assessment-and-management-of-patients-with-factor-xi-deficiency(0ff0defc-ba60-4f91-b283-86cb3fe0c9c5).html.
Pełny tekst źródłaCORNO, ANNA ROSA. "CARENZA CONGENITA DI ANTITROMBINA E DIAGNOSI DI LABORTORIO: QUALE TEST FUNZIONALE?" Doctoral thesis, Università degli Studi di Milano, 2014. http://hdl.handle.net/2434/233996.
Pełny tekst źródłaIntroduction – Antithrombin (AT) deficiency, associated with an increased risk for venous thrombosis, is classified into type I (quantitative defect) and type II (qualitative defect). Qualitative defects may affect the reactive site (RS), the heparin binding site (HBS) of AT, or they may have a pleiotropic effect (PE). Screening tests, which measure the ability of AT, in the presence of heparin, to inhibits either thrombin (anti-IIa activity) or FXa (anti-Xa activity), are able to detect most AT deficiencies; however, few cases of discrepancies have been described (i.e. normal vs. pathological value) with the two different methods. Aim of the study was the evaluation of agreement between an anti-Xa assay and an anti-IIa assay for AT, and the evaluation of their ability in detecting AT defects. Materials and Methods – The study population consisted of the “routine and thrombophilic” group (493 patients for which AT test was required) and the “historical deficiencies” group (23 subjects with known AT deficiency and 18 relatives). Anti-Xa HemosIL Antithrombin kit (from Instrumentation Laboratory) and a home-made anti-IIa method were used to measure AT activities. A control group (n= 100) was used to determine AT reference ranges. SERPINC1 gene analysis was carried out for 21 patients (Universitair Ziekenhuis in Bruxelles). Results – The results provided by the two methods showed a high correlation (Spearman rho>0.70); however, 8 discrepant results were observed (3 in the “routine and thrombophilia” group and 5 in the “historical deficiencies” group). Gene analysis confirmed the presence of a molecular defect in 18/21 subjects, 5 of which had also descrepant AT results. In fact, normal anti-Xa AT values were obtained for Cambridge II defect (RS), whereas anti-IIa test provided normal values for a HBS defect. Both methods provided pathological AT values for 5 type I deficiencies but normal AT values for other 2 HBS defects. In the study population AT anti-Xa and AT anti-IIa sensitivity was 61.1% and 55.6%, respectively; when both tests were used, sensitivity increased to 72.2%. When the ratio between AT anti-IIa and AT anti-Xa was added, sensitivity increased to 88.9%. Conclusions – Currently avaible screening tests are not able to detect all molecular defects. However, when anti-Xa assay is carried out together with anti-IIa method, and the ratio between the results provided by both is considered, the diagnostic power is increased. Anyway, laboratory test results should be considered together with personal and familiar clinical history of the single subject under evaluation.
Tan, Mustafa Tumer. "Seismic Strengthening Of A Mid-rise Reinforced Concrete Frame Using Cfrps: An Application From Real Life". Master's thesis, METU, 2009. http://etd.lib.metu.edu.tr/upload/12610562/index.pdf.
Pełny tekst źródłamer M.S., Department Of Civil Engineering Supervisor: Prof. Dr. Gü
ney Ö
zcebe Co-Supervisor: Assoc. Prof. Dr. BariS Binici May 2009, 162 pages FRP retrofitting allows the utilization of brick infill walls as lateral load resisting elements. This practical retrofit scheme is a strong alternative to strengthen low to mid-rise deficient reinforced concrete (RC) structures in Turkey. The advantages of the FRP applications, to name a few, are the speed of construction and elimination of the need for building evacuation during construction. In this retrofit scheme, infill walls are adopted to the existing frame system by using FRP tension ties anchored the boundary frame using FRP dowels. Results of experiments have previously shown that FRP strengthened infill walls can enhance lateral load carrying capacity and reduce damage by limiting interstory drift deformations. In previous, analytical studies, a detailed mathematical model and a simplified version of the model for compression struts and tension ties was proposed and verified by comparing model estimations with test results. In this study, an existing 9-storey deficient RC building located in Antakya was chosen to design and apply a hybrid strengthening scheme with FRPs and reduced number of shear walls. Linear elastic analysis procedure was utilized (force based assessment technique) along with the rules of Mode Superposition Method for the reftrofit design. FRP retrofit scheme was employed using the simplified model and design was conducted such that life safety performance criterion is satisfied employing elastic spectrum with 10% probability of exceedance in 50 years according to the Turkish Earthquake Code 2007. Further analytical studies are performed by using Modal Pushover and Nonlinear Time-History Analyses. At the end of these nonlinear analyses, performance check is performed according to Turkish Earthquake Code 2007, using the strains resulting from the sum of yield and plastic rotations at demand in the critical sections. CFRP retrofitting works started at October 2008 and finished at December 2008 for the building mentioned in this study. Eccentric reinforced concrete shearwall installation is still being undertaken. All construction business is carried out without evacuation of the building occupants. This project is one of the first examples of its kind in Turkey. Keywords: CFRP, Carbon Fiber Reinforced Polymers, Masonry Infill Walls, Reinforced Concrete Infill Walls, Mid-Rise Deficient Structures, Turkish Earthquake Code 2007, Modal Pushover Analysis, Nonlinear Time History Analysis, Linear Elastic Building Assessment
Díez, Fernández Carmen. "USING RECOMBINANT HUMAN CARBAMOYL PHOSPHATE SYNTHETASE 1 (CPS1) FOR STUDYING THIS ENZYME'S FUNCTION, REGULATION, PATHOLOGY AND STRUCTURE". Doctoral thesis, Universitat Politècnica de València, 2015. http://hdl.handle.net/10251/52855.
Pełny tekst źródła[ES] La carbamil fosfato sintetasa 1 (CPS1), una enzima mitocondrial, cataliza la entrada del amonio en el ciclo de la urea, que convierte esta neurotoxina derivada del catabolismo de las proteínas en urea, mucho menos tóxica. El déficit de CPS1 (CPS1D) es un error innato del ciclo de la urea, una enfermedad rara autosómica recesiva, que se debe a mutaciones en el gen CPS1 (>200 mutaciones descritas) y que cursa con hiperamonemia. Hemos producido CPS1 humana recombinante (hCPS1) en un sistema de expresión de células de insecto y baculovirus, y la hemos aislado en forma activa, muy pura y en cantidad elevada. Este sistema de producción de hCPS1 permite la realización de mutagénesis dirigida y la caracterización de la enzima como catalizador (actividad, cinética) y como proteína (estabilidad, estado de agregación y composición de dominios). Hemos revelado características de la hCPS1 antes no exploradas como es la composición de dominios, la capacidad que tiene el glicerol para reemplazar al activador natural y esencial de la CPS1, N-acetil-L-glutamato (NAG), y la protección de la hCPS1 por NAG y por su análogo farmacológico N-carbamil-L-glutamato (NCG) (chaperonas químicas). Hemos utilizado este sistema para explorar los efectos en actividad, parámetros cinéticos y estabilidad/plegamiento de la enzima, y para comprobar la naturaleza patogénica de mutaciones identificadas en pacientes con CPS1D. Estos resultados, junto con los obtenidos con otras mutaciones no clínicas, han aportado información novedosa sobre tres de los dominios no catalíticos de CPS1. Las observaciones realizadas tras introducir en el dominio de tipo glutaminasa de la enzima tres mutaciones asociadas a CPS1D y un polimorfismo trivial, apoyan la contribución de este dominio no catalítico a la estabilidad y a aumentar la actividad de la enzima. Dos mutaciones introducidas en el dominio de fosforilación de bicarbonato han arrojado luz sobre el modo de unión del bicarbonato (un sustrato). Los resultados de estas mutaciones también han confirmado la contribución de este dominio para la unión de NAG, cuyo sitio de unión se encuentra en el dominio C-terminal de CPS1, bastante alejado (en la secuencia) del dominio de fosforilación de bicarbonato. Además, hemos introducido 18 mutaciones de cambio de sentido asociadas a CPS1D, las cuales están localizadas en un dominio no catalítico, central y de elevada elocuencia clínica. Estos resultados han demostrado la naturaleza patogénica de estas mutaciones, ya que en la mayoría de los casos estas mutaciones producen un mal plegamiento o/y desestabilización de la enzima. Debido a que estos resultados han puesto de manifiesto el importante papel de este dominio en la integración estructural de la proteína multidominio CPS1, lo hemos llamado Dominio Integrador. Finalmente, hemos examinado los efectos de 8 mutaciones asociadas a CPS1D, de un polimorfismo trivial y de 5 mutaciones no clínicas, todas localizadas en el dominio C-terminal de la enzima, donde se une NAG. Además, hemos reanalizado resultados anteriores con otras 4 mutaciones clínicas y 5 no clínicas afectando a este dominio. Hemos confirmado el carácter patogénico de las mutaciones clínicas, las cuales predominantemente causan una disminución en la actividad enzimática, en muchos casos debida a que la unión de NAG se encuentra obstaculizada. Unas pocas mutaciones mostraron efectos negativos en la estabilidad/plegamiento de CPS1. Nuestros análisis revelan que la activación por el NAG empieza con un movimiento de la parte final del bucle ß4-¿4 del sitio de NAG. La transmisión de la señal activadora a los dominios de fosforilación implica a la hélice ¿4 de este dominio y posiblemente se transmite a través de los bucles homólogos 1313-1332 y 778-787 (numeración de residuos) pertenecientes, respectivamente, a los dominios de fosforilación de carbamato y bicarbonato. Por ello, hemos llamado a ambos bucles Bucles de
[CAT] La carbamil fosfat sintetasa 1 (CPS1), un enzim mitocondrial, catalitza l'entrada d'amoni en el cicle de la urea, que convertix l'amoni, producte neurotòxic del catabolisme de les proteïnes, en urea, una molècula molt poc tòxica. El dèficit de CPS1 (CPS1D) és un error innat del cicle de la urea, una malaltia rara autosòmica recessiva, que es deu a mutacions en el gen CPS1 (>200 mutacions descrites) i que cursa amb hiperamonièmia. Hem produït CPS1 humana recombinant (hCPS1) en un sistema d'expressió de cèl·lules d'insecte i baculovirus, i l'hem aïllada en forma activa, molt pura i en gran quantitat. Això ha permés la cristal·lització de l'enzim per a estudis estructurals amb difracció de raios-X (treball no inclòs en esta tesi Aquest sistema de producció de hCPS1 permet la realització de mutagènesi dirigida i la caracterització de l'enzim com a catalitzador (activitat, cinètica) i com a proteïna (estabilitat, estat d'agregació i composició de dominis). Hem revelat característiques de la hCPS1 no explorades abans com és la composició de dominis, la capacitat que té el glicerol per a reemplaçar l'activador natural i essencial de CPS1, N-acetil-L-glutamat (NAG), i la protecció de la hCPS1 per NAG i pel seu anàleg farmacològic N-carbamil-L-glutamat (NCG) (xaperones químiques) . Hem utilitzat aquest sistema per a explorar els efectes en l'activitat, els paràmetres cinètics i l'estabilitat/plegament de l'enzim, i per a comprovar la naturalesa patogènica de mutacions identificades en pacients amb CPS1D. Aquestos resultats, junt amb els obtinguts amb altres mutacions no clíniques, han aportat informació nova sobre tres dels dominis no catalítics de la CPS1. Les observacions, després d'introduir tres mutacions associades a CPS1D i un polimorfisme trivial en el domini tipus glutaminasa de CPS1, recolzen la contribució d'aquest domini no catalític a l'estabilitat i a l'optimització de l'activitat enzimàtica. Dues mutacions introduïdes en el domini de fosforilació de bicarbonat han esclarit el mode d'unió de bicarbonat. Els resultats d'aquestes mutacions també han confirmat la contribució d'aquest domini per a la unió de NAG, el lloc d'unió de la qual es troba en el domini C-terminal de CPS1, prou allunyat (en la seqüència) del domini de fosforilació de bicarbonat. A més, hem introduït 18 mutacions de canvi de sentit associades a CPS1D, les quals estan localitzades en un domini no catalític, central i d'elevada eloqüència clínica. Aquestos resultats han demostrat la naturalesa patogènica d'aquestes mutacions, ja que, en la majoria dels casos produïxen un mal plegament o/i desestabilització de l'enzim. Pel fet que aquestos resultats han posat de manifest l'important paper d'aquest domini en la integració estructural de la proteïna multidomini CPS1, l'hem anomenat Domini Integrador. Finalment, hem examinat els efectes de huit mutacions associades a CPS1D, un polimorfisme trivial i cinc mutacions no clíniques, totes elles localitzades en el domini C-terminal de l'enzim, on s'unix NAG. A més, hem reanalitzat resultats anteriors amb altres quatre mutacions clíniques i cinc no clíniques que afecten aquest domini. Hem confirmat el caràcter patogènic de les mutacions clíniques, les quals predominantment causen una disminució en l'activitat enzimàtica, en molts casos pel fet que la unió de NAG es troba obstaculitzada. Unes poques mutacions van mostrar efectes negatius substancials en l'estabilitat/plegament de CPS1. Les nostres anàlisis revelen que l'activació de NAG comença amb un moviment de la part final del bucle ß4-¿4 del lloc de NAG. La transmissió del senyal activadora als dominis de fosforilació involucra l'hèlix ¿4 d'aquest domini i es transmet, possiblement, a través dels bucles homòlegs 1313-1332 i 778-787 (numeració dels residus), pertanyents, respectivament, als dominis de fosforilació de carbamato i bicarbonat. Per això, hem anomenat a ambd
Díez Fernández, C. (2015). USING RECOMBINANT HUMAN CARBAMOYL PHOSPHATE SYNTHETASE 1 (CPS1) FOR STUDYING THIS ENZYME'S FUNCTION, REGULATION, PATHOLOGY AND STRUCTURE [Tesis doctoral no publicada]. Universitat Politècnica de València. https://doi.org/10.4995/Thesis/10251/52855
TESIS
Książki na temat "Deficiency of assets"
J, Haselow Nancy, Rosen David S. 1960-, Sloan Nancy L i Helen Keller International. Vitamin A Technical Assistance Program., red. How to use the HKI food frequency method to assess community risk of vitamin A deficiency. Campinas: Helen Keller International, 1993.
Znajdź pełny tekst źródłaRosen, David S., i Nancy J. Haselow. How to Use the Hki Food Frequency Method to Assess Community Risk of Vitamin a Deficiency. Helen Keller Intl, 1997.
Znajdź pełny tekst źródłaPickering, Matthew C., i Jyoti Bakshi. Complement. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0064.
Pełny tekst źródłaPickering, Matthew C., i Jyoti Bakshi. Complement. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0064_update_001.
Pełny tekst źródłaDompere, Kofi Kissi, i Manzur Ejaz. Epistemics of Development Economics. Greenwood Publishing Group, Inc., 1995. http://dx.doi.org/10.5040/9798216187134.
Pełny tekst źródłaQuint, David. Getting What You Wish For: A Reading of the Fall. Princeton University Press, 2017. http://dx.doi.org/10.23943/princeton/9780691161914.003.0007.
Pełny tekst źródłaPotter, Nancy Nyquist. Theorizing defiance. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199663866.003.0002.
Pełny tekst źródłaMöller, Bertram, Andrea J. Levinson i Zafiris J. Daskalakis. Using the TMS-induced Motor-evoked potential to evaluate the neurophysiology of psychiatric disorders. Redaktorzy Charles M. Epstein, Eric M. Wassermann i Ulf Ziemann. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780198568926.013.0023.
Pełny tekst źródłaPoll-The, Bwee Tien, Ronald J. A. Wanders i Hans R. Waterham. Peroxisomal Disorders. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0062.
Pełny tekst źródłaStanworth, Simon, i Stuart McKechnie. Pathophysiology of disordered coagulation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0269.
Pełny tekst źródłaCzęści książek na temat "Deficiency of assets"
Nauseef, William M. "Diagnostic Assays for Myeloperoxidase Deficiency". W Neutrophil Methods and Protocols, 525–30. Totowa, NJ: Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-467-4_32.
Pełny tekst źródłaNauseef, William M. "Diagnostic Assays for Myeloperoxidase and Myeloperoxidase Deficiency". W Neutrophil Methods and Protocols, 537–46. Totowa, NJ: Humana Press, 2014. http://dx.doi.org/10.1007/978-1-62703-845-4_32.
Pełny tekst źródłaZeng, Lin, Alain Sarasin i Mauro Mezzina. "Novel Complementation Assays for DNA Repair-Deficient Cells". W DNA Repair Protocols, 87–100. Totowa, NJ: Humana Press, 1999. http://dx.doi.org/10.1007/978-1-4612-1608-7_8.
Pełny tekst źródłaMurrell, T. Scott, i Dharma Pitchay. "Evaluating Plant Potassium Status". W Improving Potassium Recommendations for Agricultural Crops, 219–61. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-59197-7_9.
Pełny tekst źródłaDick, John E. "Establishment of Assays for Human Hematopoietic Cells in Immune Deficient Mice". W Current Topics in Microbiology and Immunology, 219–24. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74974-2_26.
Pełny tekst źródłaSpangler-Bickell, Caitlin. "Integrating Front-of-House with Behind-the-Scenes Practice in Contemporary Art Conservation". W Conservation of Contemporary Art, 341–63. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-42357-4_17.
Pełny tekst źródłaOuellette, Nadine, France Meslé, Jacques Vallin i Jean-Marie Robine. "Supercentenarians and Semi-supercentenarians in France". W Demographic Research Monographs, 105–23. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-49970-9_9.
Pełny tekst źródłaRusinamhodzi, Leonard, James Njeru, John E. Sariah, Rama Ngatoluwa i Phlorentin P. Lagwen. "Tillage effect on agronomic efficiency of nitrogen under rainfed conditions of Tanzania." W Conservation agriculture in Africa: climate smart agricultural development, 246–55. Wallingford: CABI, 2022. http://dx.doi.org/10.1079/9781789245745.0014.
Pełny tekst źródłaBergmann, Jonas. "Costa: Flood Displacement During the 2017 Coastal El Niño in Peru". W Studien zur Migrations- und Integrationspolitik, 249–318. Wiesbaden: Springer Fachmedien Wiesbaden, 2023. http://dx.doi.org/10.1007/978-3-658-42298-1_7.
Pełny tekst źródłaAli, Paul A. U. "Introduction". W Marshalling Of Securities, 2–8. Oxford University PressOxford, 1999. http://dx.doi.org/10.1093/oso/9780198268659.003.0001.
Pełny tekst źródłaStreszczenia konferencji na temat "Deficiency of assets"
Geddes, Connor, David R. Flatla i Ciabhan L. Connelly. "30 Years of Solving the Wrong Problem: How Recolouring Tool Design Fails those with Colour Vision Deficiency". W ASSETS '23: The 25th International ACM SIGACCESS Conference on Computers and Accessibility. New York, NY, USA: ACM, 2023. http://dx.doi.org/10.1145/3597638.3608407.
Pełny tekst źródłaUehara, Akira. "Colorable Band: A Wearable Device to Encourage Daily Decision Making Based on Behavior of Users with Color Vision Deficiency". W ASSETS '21: The 23rd International ACM SIGACCESS Conference on Computers and Accessibility. New York, NY, USA: ACM, 2021. http://dx.doi.org/10.1145/3441852.3476570.
Pełny tekst źródłaAlzaki, A., Nawaf S. Al-Shammari, Rodny Masoud Zuleta i Alejandro De la Cruz Sasso. "Longest 9 5/8" Casing Cementing in ERD Well, A Worldwide-Record". W International Petroleum Technology Conference. IPTC, 2022. http://dx.doi.org/10.2523/iptc-22082-ms.
Pełny tekst źródłaMarko, Kenneth. "Machine Learning and Model Based Reasoning for Prognostics of Complex Systems". W ASME 2005 International Mechanical Engineering Congress and Exposition. ASMEDC, 2005. http://dx.doi.org/10.1115/imece2005-81625.
Pełny tekst źródłaEllis, Eric. "Deficiency Management as Part of Asset Integrity - a Case Study". W Abu Dhabi International Petroleum Exhibition and Conference. Society of Petroleum Engineers, 2014. http://dx.doi.org/10.2118/172012-ms.
Pełny tekst źródłaGeddes, Connor, David R. Flatla, Garreth W. Tigwell i Roshan L. Peiris. "Improving Colour Patterns to Assist People with Colour Vision Deficiency". W CHI '22: CHI Conference on Human Factors in Computing Systems. New York, NY, USA: ACM, 2022. http://dx.doi.org/10.1145/3491102.3502024.
Pełny tekst źródłaMorrien-Salomons, M. M., A. Sturk, M. V. Huisman, J. Borm, H. R. Büller i J. W. ten Cate. "EVALUATION OF COMMERCIAL PROTEIN C ASSAYS". W XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644314.
Pełny tekst źródłaAlmeida Filho, Benedito de Sousa, Michelle Sako Omodei, Eduardo Carvalho Pessoa, Heloisa de Luca Vespoli i Eliana Aguiar Petri Nahas. "NEGATIVE IMPACT OF SERUM VITAMIN D DEFICIENCY ON BREAST CANCER SURVIVAL". W XXIV Congresso Brasileiro de Mastologia. Mastology, 2022. http://dx.doi.org/10.29289/259453942022v32s1058.
Pełny tekst źródłaYarkova, V. G., V. A. Zhmurov i E. B. Klester. "THE ROLE OF MAGNESIUM DEFICIENCY IN THE PATHOGENESIS OF THE FORMATION OF ARTERIAL HYPERTENSION IN WORKERS OF LOCOMOTIVE CREWS. CORRECTION METHODS". W The 16th «OCCUPATION and HEALTH» Russian National Congress with International Participation (OHRNC-2021). FSBSI “IRIOH”, 2021. http://dx.doi.org/10.31089/978-5-6042929-2-1-2021-1-607-611.
Pełny tekst źródłaKovalenko, E. "LACTASE DEFICIENCY IN RUSSIA: MULTIETHNIC GENETIC STUDY". W I International Congress “The Latest Achievements of Medicine, Healthcare, and Health-Saving Technologies”. Kemerovo State University, 2023. http://dx.doi.org/10.21603/-i-ic-53.
Pełny tekst źródłaRaporty organizacyjne na temat "Deficiency of assets"
Eshel, Amram, Jonathan P. Lynch i Kathleen M. Brown. Physiological Regulation of Root System Architecture: The Role of Ethylene and Phosphorus. United States Department of Agriculture, grudzień 2001. http://dx.doi.org/10.32747/2001.7585195.bard.
Pełny tekst źródłaAyers, R., G. P. Course i G. R. Pasco. Scottish Inshore Fisheries Integrated Data System (SIFIDS): work package (2) final report WP2A: development and pilot deployment of a prototypic autonomous fisheries data harvesting system, and WP2B: investigation into the availability and adaptability of novel technological approaches to data collection. Redaktorzy Mark James i Hannah Ladd-Jones. Marine Alliance for Science and Technology for Scotland (MASTS), 2019. http://dx.doi.org/10.15664/10023.23443.
Pełny tekst źródłaKonstantinou, Theodora, Donghui Chen, Konstantinos Flaris, Kyubyung Kang, Dan Daehyun Koo, Jonathon Sinton, Konstantina Gkritza i Samuel Labi. A Strategic Assessment of Needs and Opportunities for the Wider Adoption of Electric Vehicles in Indiana. Purdue University, 2022. http://dx.doi.org/10.5703/1288284317376.
Pełny tekst źródłaGómez Vidal, Analía, Fabiana Machado i Darcia Datshkovsky. Water and Sanitation Services in Latin America: Access and Quality Outlook. Inter-American Development Bank, kwiecień 2021. http://dx.doi.org/10.18235/0003285.
Pełny tekst źródłaGranot, David, Scott Holaday i Randy D. Allen. Enhancing Cotton Fiber Elongation and Cellulose Synthesis by Manipulating Fructokinase Activity. United States Department of Agriculture, 2008. http://dx.doi.org/10.32747/2008.7613878.bard.
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