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Artykuły w czasopismach na temat "Cortical neuronal cultures"
Boespflug, Odile, i Kenneth F. Swaiman. "Neurotransmitter Changes during Development of Cortical Neuronal Cultures". Developmental Neuroscience 8, nr 2 (1986): 102–10. http://dx.doi.org/10.1159/000112245.
Pełny tekst źródłaJoo, Chul Hyun, Yoo Kyum Kim, Heuiran Lee, HeaNam Hong, Seung-Yong Yoon i DongHou Kim. "Coxsackievirus B4-induced neuronal apoptosis in rat cortical cultures". Neuroscience Letters 326, nr 3 (lipiec 2002): 175–78. http://dx.doi.org/10.1016/s0304-3940(02)00340-3.
Pełny tekst źródłaLustig, Heather S., Kristine von B. Ahern i David A. Greenberg. "ω-Agatoxin IVA and excitotoxicity in cortical neuronal cultures". Neuroscience Letters 213, nr 2 (sierpień 1996): 142–44. http://dx.doi.org/10.1016/0304-3940(96)12849-4.
Pełny tekst źródłaJIA, L. C., C. R. HAN, PIK-YIN LAI i C. K. CHAN. "CONNECTIVITY INDUCED SYNCHRONIZATION IN CORTICAL NEURONAL NETWORKS". International Journal of Modern Physics B 21, nr 23n24 (30.09.2007): 4117–23. http://dx.doi.org/10.1142/s0217979207045293.
Pełny tekst źródłaNguyen, Lan, Sarah Wright, Mike Lee, Zhao Ren, John-Michael Sauer, Wherly Hoffman, Wagner Zago, Gene G. Kinney i Michael P. Bova. "Quantifying Amyloid Beta (Aβ)–Mediated Changes in Neuronal Morphology in Primary Cultures". Journal of Biomolecular Screening 17, nr 6 (2.04.2012): 835–42. http://dx.doi.org/10.1177/1087057112441972.
Pełny tekst źródłaGuaraldi, Mary, Sangmook Lee i Thomas B. Shea. "Synaptic Signals from Glutamate-Treated Neurons Induce Aberrant Post-Synaptic Signals in Untreated Neuronal Networks". Open Neurology Journal 14, nr 1 (24.08.2020): 59–62. http://dx.doi.org/10.2174/1874205x02014010059.
Pełny tekst źródłaKim, Sun H., Seok J. Won, Seonghyang Sohn, Hyuk J. Kwon, Jee Y. Lee, Jong H. Park i Byoung J. Gwag. "Brain-derived neurotrophic factor can act as a pronecrotic factor through transcriptional and translational activation of NADPH oxidase". Journal of Cell Biology 159, nr 5 (2.12.2002): 821–31. http://dx.doi.org/10.1083/jcb.200112131.
Pełny tekst źródłaZhang, Junhui, Geoffrey Thomas Gibney, Peng Zhao i Ying Xia. "Neuroprotective role of δ-opioid receptors in cortical neurons". American Journal of Physiology-Cell Physiology 282, nr 6 (1.06.2002): C1225—C1234. http://dx.doi.org/10.1152/ajpcell.00226.2001.
Pełny tekst źródłaC., L., C. R., C. H., M. T., Y. S., Pik-Yin Pik-Yin i C. K. "Synchronized Bursting Induced by Network Connectivity in Cortical Neuronal Cultures". Journal of the Korean Physical Society 50, nr 91 (15.01.2007): 207. http://dx.doi.org/10.3938/jkps.50.207.
Pełny tekst źródłaWise-Faberowski, Lisa, Robert D. Pearlstein i David S. Warner. "NMDA-induced Apoptosis in Mixed Neuronal/Glial Cortical Cell Cultures". Journal of Neurosurgical Anesthesiology 18, nr 4 (październik 2006): 240–46. http://dx.doi.org/10.1097/00008506-200610000-00004.
Pełny tekst źródłaRozprawy doktorskie na temat "Cortical neuronal cultures"
Boulos, Sherif. "Identification and characterisation of potential neuroprotective proteins induced by erythropoietin (EPO) preconditioning of cortical neuronal cultures". University of Western Australia. School of Biomedical and Chemical Sciences, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0128.
Pełny tekst źródłaSattler, Rita. "Effects of moderate and profound hypothermia on excitatory amino acid-induced neuronal injury in cortical cell cultures". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1996. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/MQ51597.pdf.
Pełny tekst źródłaMohandas, Bhavna. "The Effect of Exogenous Nitric Oxide on Neuronal Zn 2+ Homeostasis". Ohio : Ohio University, 2005. http://www.ohiolink.edu/etd/view.cgi?ohiou1126041116.
Pełny tekst źródłaWu, Calvin. "In Vitro Cortical Networks for Disease Modeling and Drug Evaluation". Thesis, University of North Texas, 2013. https://digital.library.unt.edu/ark:/67531/metadc407860/.
Pełny tekst źródłaQin, Yan. "Studies of Zinc Transport and Its Contribution to Zinc Homeostasis in Cultured Cortical Neurons". View abstract, 2008. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&res_dat=xri:pqdiss&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&rft_dat=xri:pqdiss:3339515.
Pełny tekst źródłaHonda, Kazuhiro. "Nongenomic antiapoptotic signal transduction by estrogen in cultured cortical neurons". Kyoto University, 2002. http://hdl.handle.net/2433/149665.
Pełny tekst źródłaTurina, Dean. "Propofol changes the cytoskeletal function in neurons : An experimental study in cortical cultures". Doctoral thesis, Linköpings universitet, Anestesiologi med intensivvård, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-77219.
Pełny tekst źródłaDurant, Stormy R. "In Vitro Exploration of Functional Acrolein Toxicity with Cortical Neuronal Networks". Thesis, University of North Texas, 2018. https://digital.library.unt.edu/ark:/67531/metadc1157516/.
Pełny tekst źródłaLavergne, Pauline, i Pauline Lavergne. "Caractérisation des réponses de neurones corticaux de rat en culture suite à des stimulations glutamatergiques grâce à la microscopie holographique numérique : vers une mesure de la balance excitation/inhibition". Master's thesis, Université Laval, 2020. http://hdl.handle.net/20.500.11794/38153.
Pełny tekst źródłaDe nouvelles preuves suggèrent que les dysfonctionnements des circuits sous-jacents aux symptômes et aux déficits cognitifs des maladies psychiatriques pourraient être causés par une altération des paramètres d'équilibre d’excitation/inhibition (E/I). Cependant, les preuves physiologiques directes de cette hypothèse à partir de données électrophysiologiques et de neuro-imagerie non invasives sont jusqu'à présent rares. Pour apporter un soutien supplémentaire à l’hypothèse de l’équilibre E/I, la présente étude a appliqué une approche avancée de microscopie holographique numérique (MHN) pour examiner la dynamique des systèmes excitateurs/inhibiteurs suite à une stimulation glutamatergique dans des réseaux de neurones à différents stades de maturation neuronale. Cette approche fournissant une mesure approximative très précise des variations de mouvement de l’eau dans les cellules permet d’étudier certains processus physiologiques, tels que ceux reliés à l’activité neuronale. Cette étude a ainsi permis d’améliorer les connaissances sur la dynamique de la réponse neuronale induite par le glutamate, notamment en la caractérisant dans des cultures de neurones corticaux primaires de rats postnataux. L’activation des neurones engendrée par le glutamate, le principal neurotransmetteur excitateur, a révélé des changements plus ou moins persistants de la morphologie et des propriétés intracellulaires des neurones. De plus, les différentes réponses obtenues indiquent que le glutamate engendre des mécanismes d’activation et des processus de régulation du volume neuronal distincts d’un neurone à l’autre, probablement dépendant de l’état d’excitabilité de ce dernier qui résulte de l’interaction complexe des neurones inhibiteurs et excitateurs. Ainsi, la régulation de l’équilibre E/I de réseaux neuronaux pourrait potentiellement être reflétée par la proportion des différentes réponses de phase induites lors de stimulation de réseaux neuronaux au glutamate.
New evidences suggest that circuit dysfunctions underlying symptoms and cognitive deficits of psychiatric disorders may be caused by impaired excitation/inhibition equilibrium parameters (E/I). However, direct physiological evidences supporting this hypothesis from non-invasive electrophysiological and neuroimaging remain scarce. To provide additional support concerning the E/I balance hypothesis, this study uses an advanced digital holographic microscopy (DHM) approach to explore the dynamics of excitatory/inhibitory systems following glutamatergic stimulation in neural networks at different stages of neuronal maturation. This approach provides a very accurate approximate measurement of the water movement variations in cells allowing to study certain specific physiological processes, such as those related to neuronal activity. This study improves the knowledge regarding the dynamics of the glutamate-induced neuronal response, especially by characterizing it in cultures of primary cortical neurons of postnatal rats. The activation of neurons induced by glutamate, which is the main excitatory neurotransmitter, revealed more or less permanent changes in the morphology and intracellular properties of neurons. Moreover, the various responses obtained indicate that glutamate generates different neuronal activation mechanisms and neuronal volume regulation processes from a neuron to another, probably depending to the excitability state of the neuron that results from the complex interaction of inhibitory and excitatory neurons. Thus, the E/I balance regulation of neural networks could potentially be reflected by the proportion of different phase responses induced during glutamate neural network stimulation.
New evidences suggest that circuit dysfunctions underlying symptoms and cognitive deficits of psychiatric disorders may be caused by impaired excitation/inhibition equilibrium parameters (E/I). However, direct physiological evidences supporting this hypothesis from non-invasive electrophysiological and neuroimaging remain scarce. To provide additional support concerning the E/I balance hypothesis, this study uses an advanced digital holographic microscopy (DHM) approach to explore the dynamics of excitatory/inhibitory systems following glutamatergic stimulation in neural networks at different stages of neuronal maturation. This approach provides a very accurate approximate measurement of the water movement variations in cells allowing to study certain specific physiological processes, such as those related to neuronal activity. This study improves the knowledge regarding the dynamics of the glutamate-induced neuronal response, especially by characterizing it in cultures of primary cortical neurons of postnatal rats. The activation of neurons induced by glutamate, which is the main excitatory neurotransmitter, revealed more or less permanent changes in the morphology and intracellular properties of neurons. Moreover, the various responses obtained indicate that glutamate generates different neuronal activation mechanisms and neuronal volume regulation processes from a neuron to another, probably depending to the excitability state of the neuron that results from the complex interaction of inhibitory and excitatory neurons. Thus, the E/I balance regulation of neural networks could potentially be reflected by the proportion of different phase responses induced during glutamate neural network stimulation.
Hetsch, Florian Jan Alexander [Verfasser]. "Induction of Synapses by Agrin in Cultured Cortical Neurons / Florian Jan Alexander Hetsch". Berlin : Freie Universität Berlin, 2015. http://d-nb.info/1072622262/34.
Pełny tekst źródłaKsiążki na temat "Cortical neuronal cultures"
Sattler, Rita. Effects of moderate and profound hypothermia on excitatory amino-acid-induced neuronal injury in cortical cell cultures. Ottawa: National Library of Canada, 1996.
Znajdź pełny tekst źródłaMassimini, Marcello, i Giulio Tononi. Sizing up Consciousness. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198728443.001.0001.
Pełny tekst źródłaCzęści książek na temat "Cortical neuronal cultures"
Choi, Dennis W., Hannelore Monyer, Rona G. Giffard, Mark P. Goldberg i Chadwick W. Christine. "Acute Brain Injury, NMDA Receptors, and Hydrogen Ions: Observations in Cortical Cell Cultures". W Excitatory Amino Acids and Neuronal Plasticity, 501–4. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-5769-8_55.
Pełny tekst źródłaKvist, Giedre. "Derivation of Adult Human Cortical Organotypic Slice Cultures for Coculture with Reprogrammed Neuronal Cells". W Methods in Molecular Biology, 253–59. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1601-7_17.
Pełny tekst źródłaLau, Anthony C., Hong Cui i Michael Tymianski. "The Use of Propidium Iodide to Assess Excitotoxic Neuronal Death in Primary Mixed Cortical Cultures". W Methods in Molecular Biology, 15–29. Totowa, NJ: Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-504-6_2.
Pełny tekst źródłaDarbinyan, Armine, Rafal Kaminski, Martyn K. White, Nune Darbinian i Kamel Khalili. "Isolation and Propagation of Primary Human and Rodent Embryonic Neural Progenitor Cells and Cortical Neurons". W Neuronal Cell Culture, 45–54. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-640-5_5.
Pełny tekst źródłaChoi, D. W. "Excitotoxicity on Cultured Cortical Neurons". W Research and Perspectives in Neurosciences, 125–36. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-84526-0_11.
Pełny tekst źródłaFacci, Laura, i Stephen D. Skaper. "Culture of Rodent Cortical and Hippocampal Neurons". W Neurotrophic Factors, 49–56. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-536-7_5.
Pełny tekst źródłaFacci, Laura, i Stephen D. Skaper. "Culture of Rodent Cortical, Hippocampal, and Striatal Neurons". W Neurotrophic Factors, 39–47. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7571-6_3.
Pełny tekst źródłaGonthier, Bertrand, C�cile Nasarre, Tina R�diger i Dominique Bagnard. "Protocol for the Primary Culture of Cortical Neurons". W New Methods for Culturing Cells from Nervous Tissues, 12–22. Basel: KARGER, 2005. http://dx.doi.org/10.1159/000083430.
Pełny tekst źródłaFacci, Laura, i Stephen D. Skaper. "Amyloid β-Peptide Neurotoxicity Assay Using Cultured Rat Cortical Neurons". W Neurotrophic Factors, 57–65. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-536-7_6.
Pełny tekst źródłaYamamoto, Seiji, Y. Matsumoto, Y. Suzuki, T. Tsuboi, S. Terakawa, N. Ohashi i K. Umemura. "An Na+/H+ exchanger inhibitor suppresses cellular swelling and neuronal death induced by glutamate in cultured cortical neurons". W Brain Edema XII, 223–26. Vienna: Springer Vienna, 2003. http://dx.doi.org/10.1007/978-3-7091-0651-8_48.
Pełny tekst źródłaStreszczenia konferencji na temat "Cortical neuronal cultures"
Gritsun, T., J. Stegenga, J. le Feber i W. L. C. Rutten. "Network bursts in cortical neuronal cultures ‘Noise- versus pacemaker’- driven neural network simulations". W 2009 4th International IEEE/EMBS Conference on Neural Engineering (NER). IEEE, 2009. http://dx.doi.org/10.1109/ner.2009.5109374.
Pełny tekst źródłale Feber, Joost, Jaap van Pelt i Wim Rutten. "Latency dependent development of related firing patterns of cultured cortical neurons". W 2007 29th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2007. http://dx.doi.org/10.1109/iembs.2007.4352960.
Pełny tekst źródłaHe, Jun, Hui Gong, Shaoqun Zeng, Yanling Li i Qingming Luo. "Enhancement of synaptic transmission induced by BDNF in cultured cortical neurons". W Biomedical Optics 2005, redaktor Valery V. Tuchin. SPIE, 2005. http://dx.doi.org/10.1117/12.592072.
Pełny tekst źródłaZorina, Inna, Irina Zakharova, Lubov Bayunova, Ekaterina Fokina, Alexander Shpakov i Natalia Avrova. "MECHANISMS OF NEUROPROTECTIVE EFFECT OF INSULIN AND α-TOCOPHEROL AT THEIR JOINT ACTION ON BRAIN CORTICAL NEURONS IN CULTURE UNDER CONDITIONS OF H2O2-INDUCED OXIDATIVE STRESS". W XVII INTERNATIONAL INTERDISCIPLINARY CONGRESS NEUROSCIENCE FOR MEDICINE AND PSYCHOLOGY. LCC MAKS Press, 2021. http://dx.doi.org/10.29003/m2133.sudak.ns2021-17/157-158.
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