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Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 27 lipca 2024

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1

Brown, Rhonda, i Einar Thorsteinsson, red. Comorbidity. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-32545-9.

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Schoenen, Jean, David W. Dodick i Peter S. Sándor, red. Comorbidity in Migraine. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444394047.

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Schoenen, Jean. Comorbidity in migraine. Chichester, West Sussex, UK: Wiley-Blackwell, 2011.

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El Miedany, Yasser, red. Comorbidity in Rheumatic Diseases. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-59963-2.

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-U, Wittchen H., red. Comorbidity of mood disorders. London: Royal College of Psychiatrists, 1996.

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Petry, Nancy M. Pathological gambling: Etiology, comorbidity, and treatment. Washington: American Psychological Association, 2005. http://dx.doi.org/10.1037/10894-000.

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Lee, Andrew G., Yi-Hsien Renee Yeh, Ashwini Kini i Bayan Al Othman. Referred Comorbidity Diplopia in Geriatric Patients. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-25945-7.

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8

B, Lipton Richard, i Silberstein Stephen D, red. Neurologic and psychiatric comorbidity with migraine. New York: Advanstar Communications, 1994.

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9

D, Maser Jack, i Cloninger C. Robert, red. Comorbidity of mood and anxiety disorders. Washington, DC: American Psychiatric Press, 1990.

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10

Comorbidity: Addiction and other mental illnesses. Wyd. 2. Rockville, MD]: U.S. Dept. of Health and Human Services, National Institutes of Health, National Institute on Drug Abuse, 2010.

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Pathological gambling: Etiology, comorbidity, and treatments. Washington, DC: American Psychological Association, 2005.

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12

S, Miller Norman, i Stimmel Barry 1939-, red. Comorbidity of addictive and psychiatric disorders. New York: Haworth Press, 1993.

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13

Dalle Grave, Riccardo, Massimiliano Sartirana i Simona Calugi. Complex Cases and Comorbidity in Eating Disorders. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-69341-1.

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14

Henriksson, Markus. Mental disorders in suicide: A comorbidity approach. Helsinki: National Public Health Institute, Dept. of Mental Health, 1996.

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15

1955-, Wetzler Scott, i Sanderson William C, red. Treatment strategies for patients with psychiatric comorbidity. New York: Wiley, 1997.

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16

Gunderson, John G., i Lois W. Choi-Kain. Borderline personality and mood disorders: Comorbidity and controversy. New York, NY: Springer, 2015.

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17

H, McConnell, i Snyder Peter J. 1964-, red. Psychiatric comorbidity in epilepsy: Basic mechanisms, diagnosis, and treatment. Washington, DC: American Psychiatric Press, 1998.

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18

1951-, Ḳaminer Yifraḥ, i Bukstein Oscar Gary 1955-, red. Adolescent substance abuse: Psychiatric comorbidity and high-risk behaviors. New York: Haworth Press, 2007.

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19

W, Eaton William, red. Medical and psychiatric comorbidity over the course of life. Washington, DC: American Psychiatric Pub., 2006.

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20

Y, Hwang Michael, i Bermanzohn Paul C, red. Schizophrenia and comorbid conditions: Diagnosis and treatment. Washington, DC: American Psychiatric Press, 2001.

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21

Australian Institute of Health and Welfare. National comorbidity initiative: A review of data collections relating to people with coexisting substance use and mental health disorders. Canberra: Australian Institute of Health and Welfare, 2005.

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22

Moggi, Franz. Doppeldiagnosen: Komorbität von psychischen Störungen und Sucht. Bern, Switzerland: Huber, 2002.

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23

Oxley, Cristal, i Argyris Stringaris. Comorbidity. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198739258.003.0022.

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Anxiety and depression often co-occur in children with ADHD and this comorbidity can also occur across the lifespan. Such comorbidity is associated with adverse outcomes across several domains. The origin of the overlap between these disorders is discussed, including the role of shared risk factors such as common genes, environmental factors, potential association with a third disorder, or as a separate nosological entity. Abnormalities in neurochemistry and findings from imaging studies are discussed. Key components of clinical assessment are discussed together with differential diagnoses, including challenges that clinicians may encounter. Treatment approaches for comorbid ADHD with emotional disorders are outlined.
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24

Brugha, Traolach S. Comorbidity assessment. Redaktor Traolach S. Brugha. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198796343.003.0010.

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This final chapter in Part II captures the distinctions between autism and other mental disorders (and intellectual disability). Issues and challenges in comorbidity assessment are discussed including development and course of social interaction. Under recognition of autism by psychiatrists and the conditions they tend to diagnose in such cases (depression, BPD, anxiety) are considered. Possible harmful effects of misdiagnosis in clinical contexts and in advice (employers, benefits system, courts, etc.) are discussed. How to differentiate symptoms that might seem to be part of two conditions (e.g. OCD versus RRBs) is discussed as is the possible confusion between autism and other similar clinical presentations. Specific comorbidities covered include ADHD, Intellectual Disability, suicidality, anxiety, depression, and masking issues and any major mental disorder in adulthood. Issues of law are also covered.
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25

Schoenen, Jean, David W. Dodick i Peter Sándor. Comorbidity in Migraine. Wiley & Sons, Incorporated, John, 2011.

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Klein, Daniel N., Sara J. Bufferd, Eunyoe Ro i Lee Anna Clark. Depression and Comorbidity. Redaktorzy C. Steven Richards i Michael W. O'Hara. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.025.

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This chapter examines the relation between personality disorder (PD) and depression, disorders that are commonly comorbid in clinical and community populations. This comorbidity presents both clinical and conceptual challenges. In anticipation of the upcoming introduction of theDiagnostic and Statistical Manual of Mental Disorders(fifth edition;DSM-5), we review research on the associations of depression with both PD and traits in order to help bridge the current and future literatures. Issues distinguishing PD and depression are reviewed, including conceptual concerns, the nature of the associations between depression and PD and traits, and current evidence on associations between depression and PD and chief personality trait dimensions. Data are presented from an ongoing study examining associations between depressive symptoms, maladaptive-range personality, and psychosocial functioning using proposedDSM-5criteria for depression and PD trait domains and facets. Depressive disorders exhibit large associations with negative affect and more moderate links with positive affect and conscientiousness/disinhibition, though there appear to be even more differentiated patterns of associations at the facet level. However, our understanding of the processes responsible for the associations of PD and depression is still limited. Despite this lack of clarity, the links between depression and PD and traits have important clinical implications for assessment and treatment of both disorders. Assessment approaches and challenges are discussed, as well as the implications of co-occurring PD and traits for the treatment of depressive disorders. Finally, future research directions are summarized.
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27

Rabe, Klaus F., Jadwiga A. Wedzicha i Emiel F. M. Wouters, red. COPD and Comorbidity. European Respiratory Society, 2013. http://dx.doi.org/10.1183/1025448x.erm5913.

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Schoenen, Jean, David W. Dodick i Peter S�ndor. Comorbidity in Migraine. Wiley & Sons, Incorporated, John, 2011.

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29

Schoenen, Jean, David W. Dodick i Peter S�ndor. Comorbidity in Migraine. Wiley & Sons, Limited, John, 2011.

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30

Schoenen, Jean, David W. Dodick i Peter S�ndor. Comorbidity in Migraine. Wiley & Sons, Incorporated, John, 2011.

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31

Markon, Kristian E. From Comorbidity to Constructs. Redaktorzy C. Steven Richards i Michael W. O'Hara. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.014.

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Comorbidity models have become central to psychopathology theory and research, not only because they have clarified our understanding of how and why disorders co-occur but also because they have clarified our understanding of what the disorders are. This chapter reviews basic types of comorbidity models, recurring issues in comorbidity modeling, and discuss emerging issues in the area. Using recent epidemiological, repeated-measures data on depression and anxiety as an example, two different models of comorbidity are compared, one in which comorbidity arises due a shared liability dimension (i.e., a reflective or latent variable model) and another in which comorbidity arises as an epiphenomenon of correlated symptoms (i.e., a formative or network model). This comparison, relatively novel in the literature, illustrates a number of issues that are encountered in comorbidity modeling, and clearly demonstrates how questions pertaining to comorbidity can shape our understanding of psychopathology constructs.
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32

Carrión, Victor G., John A. Turner i Carl F. Weems. Comorbidity in Pediatric PTSD. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190201968.003.0007.

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The current chapter focuses on the idea that the constructs reviewed in the previous chapters such as executive function, emotion regulation, and memory processing abnormalities are not unique to PTSD. Many of the neurobiological correlates of PTSD are also found in depression, anxiety, and substance use disorders. The research reviewed in the current chapter suggests that the constructs of executive function, emotion regulation, and memory processing abnormalities may underlie these conditions and define core mechanisms across mental health disorders. Future directions, such as a shift in the field to incorporate neuroscience into personalized treatment for PTSD, as well as current imperatives, such as the need to assess for trauma histories in differential diagnosis, are discussed.
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33

Bloch, Michael H. Comorbidity in Pediatric OCD. Redaktor Christopher Pittenger. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228163.003.0053.

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Tic disorders, including Tourette syndrome (TS), are not formally part of the category of “OCD-related disorders” in the DSM; but the association with OCD is sufficiently strong, and clinically important, that the OCD diagnosis now carries an optional “tic-related” specifier. Comorbidity is the norm in TS; in addition to OCD, attention deficit symptoms are particularly common. The presence of these comorbidities can affect both behavioral and pharmacological treatments, which are reviewed in this chapter. Tics commonly begin in childhood (part of the definition of TS), often improving in late adolescence. Approximately 30% of children with TS will develop OCD; the onset of OCD symptoms is usually later than that of tics, and they are more likely to persist into adulthood. Tic-associated OCD has a male preponderance and is more likely to be characterized by symmetry-related obsessions and compulsions. Like OCD, tic disorders are characterized by abnormalities in the cortico-striatal circuitry.
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34

Wheaton, Michael G., i Anna Van Meter. Comorbidity in Hoarding Disorder. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199937783.013.003.

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Thomas, Stuart D. M. Diagnostic prevalence and comorbidity. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199360574.003.0032.

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Prisons and jails remain a growth industry, with many countries increasing correctional services to cope with the ever-burgeoning inmate population. One longstanding issue is the perceived increase in prevalence of mental disorders that are found in correctional settings compared to the community. Definitions of mental illness and methods of assessment vary substantially. That said, emerging data reflect some consistency in the range of estimated prevalence. Personality disorder (predominantly antisocial personality disorder) is the most common mental disorder among prisoners, accounting for 65% of male and 42% of female prisoners. Estimated rates of psychosis in some settings are as high as 3.7% for males and 4.0% for females, while major depressive disorders are found in up to 10% of male and 12% of female prisoners. Estimated point prevalence rates for alcohol abuse and dependence varied between 18 and 30% for male prisoners and between 10 and 24% for female prisoners; these estimates were between 10 and 48% for males and 30 to 60% for female prisoners with respect to drug dependence and abuse. The rates of almost all disorders are several times higher than those found in the general community, and the rates of comorbidity are exceptionally high. This chapter outlines the best available correctional prevalence of common mental disorders and considers the key assumptions and methodological challenges around ascertaining rates of these different diagnoses.
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36

STAHL, NICOLE D. CONDUCT DISORDER AND COMORBIDITY. 1999.

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Miedany, Yasser El. Comorbidity in Rheumatic Diseases. Springer, 2017.

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Comorbidity perspectives across Europe. London, UK: European Collaborating Centres in Addiction Studies, 2006.

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Miedany, Yasser El. Comorbidity in Rheumatic Diseases. Springer, 2018.

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Brown, J. J. Comorbidity: Expressions of Love. Independently Published, 2018.

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Comorbidity in affective disorders. New York: M. Dekker, 1999.

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Gershkovich, Marina, Olivia Pascucci i Joanna Steinglass. Eating Disorder Comorbidity with OCD. Redaktor Christopher Pittenger. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228163.003.0056.

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This chapter discusses the comorbidity of eating disorders with OCD, including epidemiology, neurobiology, clinical features, and treatment. These disorders frequently cooccur. There are hints that eating disorders and OCD may share underlying neural mechanisms of illness, but these neurobiological models are preliminary. Cooccurrence of eating disorders and OCD is more common in clinical populations than community samples, highlighting the importance of assessing eating and weight history among patients with OCD. Treatment for bulimia nervosa includes psychotherapy and medication options, all with good empirical support. Treatment for anorexia nervosa emphasizes behaviorally based approaches to weight restoration treatment, with no clear evidence for medication treatments. When these illnesses occur together, treatment focused on the eating disorder yields improvement in OCD symptoms. Integrating exposure and response prevention techniques into eating disorder treatment may yield synergistic improvement in both illnesses. Future research may shed light on shared and distinct neurobiological mechanisms of these illnesses.
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43

Huppert, Jonathan D. Anxiety Disorders and Depression Comorbidity. Oxford University Press, 2008. http://dx.doi.org/10.1093/oxfordhb/9780195307030.013.0044.

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Belik, Shay-Lee, Jitender Sareen i Murray B. Stein. Anxiety Disorders and Physical Comorbidity. Oxford University Press, 2008. http://dx.doi.org/10.1093/oxfordhb/9780195307030.013.0046.

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Casanova, Manuel Fernando, Emily L. Casanova, Richard Eugene Frye i Christopher Gillberg, red. Comorbidity and Autism Spectrum Disorder. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88966-358-3.

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(Editor), Ruth Gross-isseroff, i Abraham Weizman (Editor), red. Obsessive-Compulsive Disorder And Comorbidity. Nova Science Publishers, 2006.

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Kumperscak, Hojka Gregoric. ADHD: From Etiology to Comorbidity. Intechopen, 2021.

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48

Youngstrom, Eric, i Anna Van Meter. Comorbidity of Bipolar Disorder and Depression. Redaktorzy C. Steven Richards i Michael W. O'Hara. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.003.

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There has been speculation about the relationship between depression and mania for centuries. Modern psychiatry and psychology have mostly viewed these as different subtypes within a “family” of mood disorders. Conceptual models of comorbidity provide an opportunity to re-examine the association between depression and other pathological mood states. We examine the evidence pertaining to rates of “comorbidity,” which, in this case, refer to the lifetime occurrence of depression and hypomanic, mixed, or manic episodes in the same individual. We explore factors that could contribute to artifactual comorbidity. We also examine data pertaining to similarities or differences in phenomenology, longitudinal course, associated features, family history, and treatment response. Multiple factors are likely involved in the comorbidity of depression and hypomania or mania, and the problems of poor reliability and inconsistent diagnostic definitions and methodology attenuate the significance of most research findings. However, evidence appears sufficient to conclude that not all depression is on the bipolar spectrum, that bipolar features moderate the course and outcome of depressive illness, and that depression and bipolar disorder most likely involve a blend of some shared and some specific mechanisms. Research and clinical work both will advance substantially by more systematically assessing for potential bipolar features “comorbid” with depression and following how these factors change the trajectory of depression over time.
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Capaldi, Deborah M., i Hyoun K. Kim. Comorbidity of Depression and Conduct Disorder. Redaktorzy C. Steven Richards i Michael W. O'Hara. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.015.

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Both depression and conduct disorders are relatively prevalent and are related to poor long-term outcomes. Despite being characterized by very different symptoms, it is well established that these two disorders co-occur at higher rates than expected by chance, resulting in poorer adjustment for the individual than would result from either problem alone. The termcomorbidityis usually reserved to refer to the association of diagnosed disorders, whereasco-occurrencerefers more broadly to the association of levels of symptoms of conduct problems and depression, which are usually calculated with means or possibly symptoms counts. In the past two decades, researchers have focused particularly on the following issues regarding the comorbidity of depression and conduct disorder: (1) possible causal associations of the two problem behaviors (i.e., do depressive disorders tend to onset after conduct disorders or vice versa); (2) theory regarding causes of the association (i.e., common versus unique risk factors for these two problem behaviors); (3) changes across development (i.e., with age); (4) risks from diagnosed disorders versus symptoms that do not reach diagnostic criteria; (5) outcomes or prognosis (e.g., are outcomes more severe for co-occurring problems than for either problem alone, are there distinct patterns of outcomes associated with co-occurring problems). Within each of these areas there is considerable interest in moderation of effects by gender or gender similarities and differences. This chapter reviews findings pertaining to these issues and presents suggestions for future research. In addition, assessment approaches and clinical implications are discussed.
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Castriotta, Natalie, i Michelle G. Craske. Depression and Comorbidity with Panic Disorder. Redaktorzy C. Steven Richards i Michael W. O'Hara. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.027.

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Comorbidity between panic disorder and major depression is found in the majority of individuals with panic disorder and a substantial minority of individuals with major depression. Comorbidity between panic disorder and depression is associated with substantially more severe symptoms of each of the disorders, greater persistence of each disorder, more frequent hospitalization and help-seeking behavior, more severe occupational impacts, and a significantly higher rate of suicide attempts. These two disorders share many risk factors, such as neuroticism, exposure to childhood abuse, informational processing biases, and elevated amygdala activation in response to negative facial expressions. Research on the temporal priority of panic disorder and major depression has most frequently found that panic attacks and other symptoms of anxiety predate the onset of the first major depressive episode, but the first depressive episode predates the onset of full panic disorder. Treatment studies indicate that cognitive behavioral therapy (CBT) is the most effective treatment for panic disorder. Other forms of treatment include medication, particularly selective serotonin reuptake inhibitors. Comorbid depression does not appear to affect the outcome of CBT for a principal diagnosis of panic disorder, and CBT for panic disorder has positive, yet limited, effects on symptoms of depression.
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