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Gotowe bibliografie tematyczne / Cold Sensitive Phenotypes

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Artykuły w czasopismach

Gotowa bibliografia na temat „Cold Sensitive Phenotypes”

Autor: Grafiati

Data publikacji: 6 września 2023

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Artykuły w czasopismach na temat "Cold Sensitive Phenotypes"

1

Baliga, Chetana, Sandipan Majhi, Kajari Mondal, Antara Bhattacharjee, K. VijayRaghavan, and Raghavan Varadarajan. "Rational elicitation of cold-sensitive phenotypes." Proceedings of the National Academy of Sciences 113, no. 18 (2016): E2506—E2515. http://dx.doi.org/10.1073/pnas.1604190113.

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Cold-sensitive phenotypes have helped us understand macromolecular assembly and biological phenomena, yet few attempts have been made to understand the basis of cold sensitivity or to elicit it by design. We report a method for rational design of cold-sensitive phenotypes. The method involves generation of partial loss-of-function mutants, at either buried or functional sites, coupled with selective overexpression strategies. The only essential input is amino acid sequence, although available structural information can be used as well. The method has been used to elicit cold-sensitive mutants

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Novick, P., B. C. Osmond, and D. Botstein. "Suppressors of yeast actin mutations." Genetics 121, no. 4 (1989): 659–74. http://dx.doi.org/10.1093/genetics/121.4.659.

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Abstract Suppressors of a temperature-sensitive mutation (act1-1) in the single actin gene of Saccharomyces cerevisiae were selected that had simultaneously acquired a cold-sensitive growth phenotype. Five genes, called SAC (suppressor of actin) were defined by complementation tests; both suppression and cold-sensitive phenotypes were recessive. Three of the genes (SAC1, SAC2 and SAC3) were subjected to extensive genetic and phenotypic analysis, including molecular cloning. Suppression was found to be allele-specific with respect to actin alleles. The sac mutants, even in ACT1+ genetic backgro

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3

Flower, Ann M. "SecG Function and Phospholipid Metabolism inEscherichia coli." Journal of Bacteriology 183, no. 6 (2001): 2006–12. http://dx.doi.org/10.1128/jb.183.6.2006-2012.2001.

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ABSTRACT SecG is an auxiliary protein in the Sec-dependent protein export pathway of Escherichia coli. Although the precise function of SecG is unknown, it stimulates translocation activity and has been postulated to enhance the membrane insertion-deinsertion cycle of SecA. Deletion of secG was initially reported to result in a severe export defect and cold sensitivity. Later results demonstrated that both of these phenotypes were strain dependent, and it was proposed that an additional mutation was required for manifestation of the cold-sensitive phenotype. The results presented here demonstr

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4

Nonet, M. L., and R. A. Young. "Intragenic and extragenic suppressors of mutations in the heptapeptide repeat domain of Saccharomyces cerevisiae RNA polymerase II." Genetics 123, no. 4 (1989): 715–24. http://dx.doi.org/10.1093/genetics/123.4.715.

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Abstract The largest subunit of RNA polymerase II contains a repeated heptapeptide sequence at its carboxy terminus. Yeast mutants with certain partial deletions of the carboxy-terminal repeat (CTR) domain are temperature-sensitive, cold-sensitive and are inositol auxotrophs. Intragenic and extragenic suppressors of the cold-sensitive phenotype of CTR domain deletion mutants were isolated and studied to investigate the function of this domain. Two types of intragenic suppressing mutations suppress the temperature-sensitivity, cold-sensitivity and inositol auxotrophy of CTR domain deletion muta

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5

Skiadopoulos, Mario H., Sonja Surman, Joanne M. Tatem, et al. "Identification of Mutations Contributing to the Temperature-Sensitive, Cold-Adapted, and Attenuation Phenotypes of the Live-Attenuated Cold-Passage 45 (cp45) Human Parainfluenza Virus 3 Candidate Vaccine." Journal of Virology 73, no. 2 (1999): 1374–81. http://dx.doi.org/10.1128/jvi.73.2.1374-1381.1999.

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ABSTRACT The live-attenuated human parainfluenza virus 3 (PIV3) cold-passage 45 (cp45) candidate vaccine was shown previously to be safe, immunogenic, and phenotypically stable in seronegative human infants. Previous findings indicated that each of the three amino acid substitutions in the L polymerase protein of cp45 independently confers the temperature-sensitive (ts) and attenuation (att) phenotypes but not the cold-adaptation (ca) phenotype (29).cp45 contains 12 additional potentially important point mutations in other proteins (N, C, M, F, and hemagglutinin-neuraminidase [HN]) or in cis-a

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6

Kuchka, Michael R., and Jonathan W. Jarvik. "Short-Flagella Mutants of Chlamydomonas reinhardtii." Genetics 115, no. 4 (1987): 685–91. http://dx.doi.org/10.1093/genetics/115.4.685.

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ABSTRACT Six short-flagella mutants were isolated by screening clones of mutagenized Chlamydomonas for slow swimmers. The six mutants identify three unlinked Mendelian genes, with three mutations in gene shf-1, two in shf-2 and one in shf-3. shf-1 and shf-2 have been mapped to chromosomes VI and I, respectively. Two of the shf-1 mutations have temperature-sensitive flagellar-assembly phenotypes, and one shf-2 mutant has a cold-sensitive phenotype. shf shf double mutants were constructed; depending on the alleles present they showed either flagellaless or short-flagella phenotypes. Phenotypic r

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7

Puziss, J. W., T. A. Hardy, R. B. Johnson, P. J. Roach, and P. Hieter. "MDS1, a dosage suppressor of an mck1 mutant, encodes a putative yeast homolog of glycogen synthase kinase 3." Molecular and Cellular Biology 14, no. 1 (1994): 831–39. http://dx.doi.org/10.1128/mcb.14.1.831-839.1994.

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The yeast gene MCK1 encodes a serine/threonine protein kinase that is thought to function in regulating kinetochore activity and entry into meiosis. Disruption of MCK1 confers a cold-sensitive phenotype, a temperature-sensitive phenotype, and sensitivity to the microtubule-destabilizing drug benomyl and leads to loss of chromosomes during growth on benomyl. A dosage suppression selection was used to identify genes that, when present at high copy number, could suppress the cold-sensitive phenotype of mck1::HIS3 mutant cells. Several unique classes of clones were identified, and one of these, de

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8

Puziss, J. W., T. A. Hardy, R. B. Johnson, P. J. Roach, and P. Hieter. "MDS1, a dosage suppressor of an mck1 mutant, encodes a putative yeast homolog of glycogen synthase kinase 3." Molecular and Cellular Biology 14, no. 1 (1994): 831–39. http://dx.doi.org/10.1128/mcb.14.1.831.

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The yeast gene MCK1 encodes a serine/threonine protein kinase that is thought to function in regulating kinetochore activity and entry into meiosis. Disruption of MCK1 confers a cold-sensitive phenotype, a temperature-sensitive phenotype, and sensitivity to the microtubule-destabilizing drug benomyl and leads to loss of chromosomes during growth on benomyl. A dosage suppression selection was used to identify genes that, when present at high copy number, could suppress the cold-sensitive phenotype of mck1::HIS3 mutant cells. Several unique classes of clones were identified, and one of these, de

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9

Fane, B. A., and M. Hayashi. "Second-site suppressors of a cold-sensitive prohead accessory protein of bacteriophage phi X174." Genetics 128, no. 4 (1991): 663–71. http://dx.doi.org/10.1093/genetics/128.4.663.

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Abstract This study describes the isolation of second-site suppressors which correct for the defects associated with cold-sensitive (cs) prohead accessory proteins of bacteriophage phi X174. Five phenotypically different suppressors were isolated. Three of these suppressors confer novel temperature-sensitive (ts) phenotypes. They were unable to complement a ts mutation in gene F which encodes the major coat protein of the phage. All five suppressor mutations confer nucleotide changes in the gene F DNA sequence. These changes define four amino acid sites in the gene F protein. Three suppressor

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10

Hecht, Ralph M., Mary A. Norman, Tammy Vu, and William Jones. "A novel set of uncoordinated mutants inCaenorhabditis elegansuncovered by cold-sensitive mutations." Genome 39, no. 2 (1996): 459–64. http://dx.doi.org/10.1139/g96-058.

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A set of uncoordinated (Unc) cold-sensitive (cs) mutants was isolated at a stringent condition of 11 °C. About half of the 13 independently isolated cs-Unc mutants were alleles of three X-linked Unc mutants that exhibited the "kinker" phenotype. The remaining four isolates identified new mutants that exhibited "kinker," "coiler," or severe paralytic phenotypes. The temperature-sensitive period (TSP) for each gene was determined. As a homozygous or heterozygous dominant, unc-125 exhibited a TSP throughout all stages of development. Its severe paralysis was immediately observed upon a shift down

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