Rozprawy doktorskie na temat „Cognition disorders”
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Airaksinen, Eija. "Cognitive functions in depression and anxiety disorders : findings from a population-based study /". Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-954-8/.
Pełny tekst źródłaLynham, Amy. "Measuring cognition across mood and psychotic disorders". Thesis, Cardiff University, 2018. http://orca.cf.ac.uk/116386/.
Pełny tekst źródłaNowotny, Ewa. "Social cognition and HIV-associated neurocognitive disorders". Thesis, University of East London, 2016. http://roar.uel.ac.uk/5399/.
Pełny tekst źródłaCrino, Natalie. "Metacognition and eating disorders". Thesis, The University of Sydney, 2014. http://hdl.handle.net/2123/12643.
Pełny tekst źródłaEddy, Clare Margaret. "Social cognition in disorders of the basal ganglia". Thesis, University of Birmingham, 2009. http://etheses.bham.ac.uk//id/eprint/366/.
Pełny tekst źródłaFish, Scott Christopher. "Pupillary response measures of processing resource allocation during theory of mind task performance in schizophrenia". Diss., [La Jolla] : University of California, San Diego, 2009. http://wwwlib.umi.com/cr/ucsd/fullcit?p3360156.
Pełny tekst źródłaTitle from first page of PDF file (viewed August 11, 2009). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references (p. 36-39).
Whitcomb-Smith, Stacy. "The Role of Cognitive Factors in the Development of Seasonal Affective Disorder Episodes". Fogler Library, University of Maine, 2003. http://www.library.umaine.edu/theses/pdf/Whitcomb-SmithS2003.pdf.
Pełny tekst źródłaLee, W. "Subjective cognitive impairments in Schizophrenia and related disorders". Click to view the E-thesis via HKUTO, 2004. http://sunzi.lib.hku.hk/hkuto/record/B31384948.
Pełny tekst źródła王得寶 i Tak-po Mike Wong. "Memory profile of people with mild cognitive impairment". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B41547834.
Pełny tekst źródłaWong, Tak-po Mike. "Memory profile of people with mild cognitive impairment". Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B41547834.
Pełny tekst źródłaHallock, Hariharan Michael. "Cognitive Training Interventions for Neurocognitive Disorders". Thesis, The University of Sydney, 2017. http://hdl.handle.net/2123/17840.
Pełny tekst źródłaPossin, Katherine L. "Visuospatial and visual object cognition in early Parkinson's disease". Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2007. http://wwwlib.umi.com/cr/ucsd/fullcit?p3250074.
Pełny tekst źródłaTitle from first page of PDF file (viewed April 4, 2007). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references (p. 128-166).
李穎 i W. Lee. "Subjective cognitive impairments in Schizophrenia and related disorders". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B31384948.
Pełny tekst źródłaKrane, Erica A. "Functional impairments associated with DSM-IV diagnosed adult attention-deficithyperactivity disorder". Thesis, McGill University, 2002. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=82905.
Pełny tekst źródłaHumphreys, Clare Thomson. "Sleep disorders and cognition in older adults with cardiovascular disease". Diss., University of Iowa, 2010. https://ir.uiowa.edu/etd/685.
Pełny tekst źródłaRamirez, Joanna. "Cognitive distortions in adolescents with substance-related disorders /". Full text (PDF) from UMI/Dissertation Abstracts International, 2000. http://wwwlib.umi.com/cr/utexas/fullcit?p3004362.
Pełny tekst źródłaBarnes, Kelly Anne. "Implicit learning in typical development and children with developmental disorders". Connect to Electronic Thesis (ProQuest), 2008. http://0-gateway.proquest.com.library.lausys.georgetown.edu/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:3320707.
Pełny tekst źródłaLindquist, Barbro. "Hydrocephalus in children : cognition and behaviour /". Göteborg : Department of Pediatrics, Institute of Clinical Sciences, Sahlgrenska Academy at Göteborg University, 2007. http://hdl.handle.net/2077/2557.
Pełny tekst źródłaWhite, Elliott P. "Social cognition skills in borderline personality disorder". Thesis, Canterbury Christ Church University, 2014. http://create.canterbury.ac.uk/12836/.
Pełny tekst źródłaButler, Lucy. "Social cognition and HIV : exploring the profile of cognitive impairments in HIV-Associated Neurocognitive Disorders (HAND)". Thesis, University of East London, 2016. http://roar.uel.ac.uk/5407/.
Pełny tekst źródłaHo, Wendy Karen, i 何慧盈. "The examination of endothelin-1 effects on vascular-neurodegenerative pathways contributing to cognitive impairment". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/196432.
Pełny tekst źródłapublished_or_final_version
Psychiatry
Master
Master of Philosophy
Chua, Eldrich Norwin Siy, i 蔡季延. "The effects of noninvasive brain stimulation on cognitive function in patients with stroke : a systematic review". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/206919.
Pełny tekst źródłapublished_or_final_version
Public Health
Master
Master of Public Health
Kauhanen, M. L. (Marja-Liisa). "Quality of life after stroke:clinical, functional, psychosocial and cognitive correlates". Doctoral thesis, University of Oulu, 1999. http://urn.fi/urn:isbn:9514254279.
Pełny tekst źródłaEhlhardt, Laurie Anne. "E-steps : evaluation of an instructional sequence for persons with impaired memory and executive functions /". view abstract or download file of text, 2003. http://wwwlib.umi.com/cr/uoregon/fullcit?p3095242.
Pełny tekst źródłaTypescript. Includes vita and abstract. Includes bibliographical references (leaves 124-128). Also available for download via the World Wide Web; free to University of Oregon users.
DeVito, Elise Eva. "Cognition in disorders of frontostriatal dysfunction : neuropsychological, neuroimaging and psychopharmacological studies". Thesis, University of Cambridge, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.611615.
Pełny tekst źródłaLindinger, Nadine Michelle. "Social cognition in South African children with Fetal Alcohol Spectrum Disorders". Doctoral thesis, University of Cape Town, 2016. http://hdl.handle.net/11427/20333.
Pełny tekst źródłaDuff, Barbara Jane. "Cognition in t(1;11) translocation carriers and patients with psychotic disorders". Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/28826.
Pełny tekst źródłaPettersson, Anna. "Motor function and cognition : aspects on gait and balance /". Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-431-7/.
Pełny tekst źródłaCrouse, Jacob Jeffrey. "Mapping the Early Functional Course of Emerging Mental Disorders". Thesis, University of Sydney, 2020. https://hdl.handle.net/2123/23974.
Pełny tekst źródłaBenas, Jessica Sara. "Cognitive biases in depression and eating disorders". Diss., Online access via UMI:, 2009.
Znajdź pełny tekst źródłaWithall, Adrienne Lee. "Cognitive function and recovery in major depressive disorder". Thesis, The University of Sydney, 2006. https://hdl.handle.net/2123/28184.
Pełny tekst źródłaOberman, Lindsay Meredith. "A disembodied mind the role of dysfunctional simulation systems in the social and cognitive deficits of autism spectrum disorders /". Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2007. http://wwwlib.umi.com/cr/ucsd/fullcit?p3258394.
Pełny tekst źródłaTitle from first page of PDF file (viewed June 1, 2007). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references.
TÃvora, Daniel Gurgel Fernandes. "AlteraÃÃes do sono, alteraÃÃo cognitiva e avaliaÃÃo de estruturas cerebrais atravÃs de ressonÃncia magnÃtica e morfometria baseada em voxel na doenÃa de Parkinson". Universidade Federal do CearÃ, 2013. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=14114.
Pełny tekst źródłaO trabalho avalia em duas fases as alteraÃÃes clÃnicas, alteraÃÃes do sono, a alteraÃÃo cognitiva e as alteraÃÃes de estruturas cerebrais atravÃs de RessonÃncia MagnÃtica (RM) e morfologia baseada em voxel (VBM) em pacientes com DoenÃa de Parkinson. Foram estudados 100 pacientes (71% masculino), com idade entre 40 e 80 anos (66,1+9,5), recrutados do AmbulatÃrio de DistÃrbios do Movimento do Hospital UniversitÃrio Walter CantÃdio. A amostra faz parte de uma coorte longitudinal (Sleep-For-PD study). Foram estudadas as alteraÃÃes do sono e seus fatores associados e preditivos. A escala de sono da DoenÃa de Parkinson (PDSS) que avalia alteraÃÃes de sono na DP, a escala Pittsburgh Sleep Quality Index (PSQI) que avalia a qualidade do sono, a escala Epworth Sleepiness Scale (ESS) que avalia o grau subjetivo de sonolÃncia e a escala Unified Parkinsonâs Disease Scale (UPDRS partes I, II, III e IV) que avalia a gravidade da doenÃa foram estudados. Os sintomas depressivos foram avaliados atravÃs das escalas Beck Depression Inventory (BDI-II) e Hospital Anxiety Depression (HAD). Os pacientes foram submetidos ao Mini Exame do Estado Mental (MEEM) que avaliou o grau de alteraÃÃo cognitiva e a escala de distÃrbio comportamental do sono REM (RBD). A dose de levodopa (DEL) foi avaliada. Pacientes com alteraÃÃes do sono (PDSS) apresentaram mais alucinaÃÃes diurnas, mais alteraÃÃo cognitiva, mais ansiedade, depressÃo e maior gravidade dos sintomas parkinsonianos (p<0,05). Pacientes com mà qualidade de sono (PSQI) tiveram mais sintomas depressivos. Os escores PDSS correlacionaram-se ainda com a funÃÃo cognitiva (MEEM), os sintomas depressivos (BDI e HAD), a qualidade do sono (PSQI), a gravidade da doenÃa e com a escala de RBD. Os escores PSQI correlacionaram-se com o MEEM, atividades de vida diÃria (UPDRS II) e sintomas de depressÃo/ansiedade (BDI e HAD). Gravidade de sintomas relacionados a atividades da vida diÃria (p=0,002), sintomas depressivos (p=0,01) e ansiedade (p=0,01) foram fatores independentes preditivos das alteraÃÃes do sono (PDSS). A DEL e o MEEM foram preditores da mà qualidade do sono (p=0,02). A escala RBD (p=0,002) e a UPDRS I (p=0,02) foram preditores do grau de sonolÃncia. ConcluÃmos que alteraÃÃes de sono, mà qualidade de sono e sonolÃncia diurna excessiva sÃo comuns na DP. As escalas PDSS, PSQI e ESS tÃm fatores associados e preditivos distintos. A escala PDSS apresenta maior abrangÃncia na avaliaÃÃo do sono na DP. Na segunda fase, foram avaliadas as estruturas cerebrais por RM, a presenÃa de alteraÃÃes cognitivas e fatores associados em 39 pacientes com DP e em 10 indivÃduos controles pareados por idade. As imagens de RM foram processadas de acordo com o protocolo para processamento de VBM. A variÃvel de desfecho usada foi o volume de substÃncia cinzenta. Nossos dados evidenciaram que pacientes com DP apresentaram maior comprometimento cognitivo e maior ansiedade. Pacientes com DP e alteraÃÃes cognitivas apresentaram maior gravidade da doenÃa. NÃo houve diferenÃa no volume de substÃncia cinzenta entre os pacientes com DP com e sem alteraÃÃes cognitivas. Estes achados provavelmente deveram-se a atrofia cerebral precoce nos pacientes cognitivamente intactos. Pacientes com DP, quando comparados aos controles, revelaram reduÃÃes de volume de substÃncia cinzenta na Ãnsula esquerda e cÃrtex prÃ-frontal esquerdo, demonstrando envolvimento assimÃtrico do cÃrebro na DP.
The present study evaluates clinical abnormalities, sleep disturbances, cognitive alterations and structural brain changes using Magnetic Resonance Imaging (MRI) with Voxel Based Morphometry (VBM) in patients with Parkinson`s Disease (PD). In the first phase of the study one hundred patients (71% male), aged between 40 and 80 years (66,1+9,5) were studied. Patients were recruited from a movement disorders clinics at Walter CantÃdio University Hospital. The study is part of a larger longitudinal cohort study (Sleep-For-PD study). Sleep abnormalities and their associated and predictive factors were scrutinized. Many clinical questionnaires were used, including the Parkinson`s Disease Sleep Scale (PDSS), Pittsburgh Sleep Quality Index (PSQI), Epworth Sleepiness Scale (ESS) and the Unified Parkinsonâs Disease Rating Scale (UPDRS part I, II, III e IV). Depressive symptoms were evaluated with Beck Depression Inventory (IDB-II) and Hospital Anxiety Depression scale (HAD). The Minimental state examination (Folstein) (MMSE) evaluated the extent of cognitive dysfunction. REM sleep symptoms were evaluated by the REM Sleep Behavior Disorder (RBD) Scale. The levodopa equivalent dose was evaluated (DEL). Patients with sleep abnormalities (PDSS) had more diurnal visual hallucinations, cognitive dysfunction, anxiety, depression and worse parkinsonian symptoms (p<0.05). Patients with worse sleep quality (PSQI) had more depressive symptoms. PDSS scores were correlated with cognitive function (MEEM), depressive symptoms (BDI and HAD), sleep quality (PSQI), severity of PD and the RBD scale. PSQI scores were correlated with MMSE scores, activity of daily living symptoms (UPDRS II) and depression / anxiety (BDI e HAD). Activities of daily living (p=0.002), depressive symptoms (p=0.01) and anxiety (p=0.01) were independent predictors of sleep abnormalities (PDSS). The levodopa equivalent dose and MMSE scores were independent predictors of worse sleep quality (p=0.02). The RBD scale (p=0.002) and UPDRS I (p=0.02) were independent predictors of somnolence. We conclude that sleep disorders, disturbed sleep quality and excessive diurnal somnolence are common in PD. The PDSS, PSQI and ESS scales have distinct associated and predictive factors. PDSS scale is associated with a greater number of factors in PD patients. In the second phase of the study thirty-nine PD patients and ten control subjects were evaluated with regard to the presence of cognitive alterations. Structural brain abnormalities were also evaluated with MRI and VBM technique. The Gray matter volume was used as the ending variable. PD patients had more cognitive impairment and more anxiety. Patients with PD and cognitive alterations had worse disease severity. We found no difference in the volume of gray matter between the subgroups of PD patients with and without cognitive alterations, probably due to early brain atrophy in the patients without cognitive abnormalities. A significant reduction in gray matter volume in the left insula and left prefrontal cortex was observed when comparing PD patients in relation to controls. These findings indicate an asymmetrical brain involvement in PD, the left hemisphere being more affected.
McMillan, Elaine S. "Processing Social Information: An Investigation of the Modification of Attentional Biases in Social Anxiety". Fogler Library, University of Maine, 2008. http://www.library.umaine.edu/theses/pdf/McMillanES2008.pdf.
Pełny tekst źródłaHaig, Albert R. "Missing links the role of phase synchronous gamma oscillations in normal cognition and their dysfunction in schizophrenia /". Connect to full text, 2002. http://hdl.handle.net/2123/848.
Pełny tekst źródłaTitle from title screen (viewed Apr. 28, 2008). Submitted in fulfilment of the requirements for the degree of Doctor of Philosophy to the Dept. of Psychological Medicine, Faculty of Medicine. Includes bibliography. Also available in print form.
Soukup, Vicki Marlene. "The Assessment of Cognitive Functioning among Patients with Unilateral Visual Neglect: Effects of Field of Presentation and Cueing". Thesis, University of North Texas, 1992. https://digital.library.unt.edu/ark:/67531/metadc278752/.
Pełny tekst źródłaBurns, Amy Minh Nhat. "Theory of mind, social cognition, and neural functioning in schizophrenia spectrum disorders". Thesis, University of British Columbia, 2016. http://hdl.handle.net/2429/59475.
Pełny tekst źródłaArts, Faculty of
Psychology, Department of
Graduate
Chen, Zen-Yong. "Language, cognition and the corpus callosum in adults with developmental language disorders". Thesis, University of Sheffield, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.412721.
Pełny tekst źródłaIreland, Elizabeth. "Exploring social cognition and executive function in HIV-Associated Neurocognitive Disorders (HAND)". Thesis, University of East London, 2011. http://roar.uel.ac.uk/3711/.
Pełny tekst źródłaSit, Bik-yan Sonia. "Cognitive function in Chinese stroke patients /". View the Table of Contents & Abstract, 2005. http://sunzi.lib.hku.hk/hkuto/record/B31595923.
Pełny tekst źródłaCheung, Vinci, i 張穎思. "Cognitive dysfunction implicated in the expression of attentional blink in schizophrenia". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2002. http://hub.hku.hk/bib/B29741890.
Pełny tekst źródłaCrouch, Barry. "Cognitive dysfunction in schizophrenia : novel models and behavioural methods for preclinical research". Thesis, University of Aberdeen, 2015. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=229384.
Pełny tekst źródłaTsang, Yee-ha Lucia. "Neurocognitive sequelae of children born prematurely". Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B41712596.
Pełny tekst źródłaMcLaughlin, Karen A. "Initial investigation of a collaborative intervention model for individuals with brain injury and their families /". view abstract or download file of text, 2001. http://wwwlib.umi.com/cr/uoregon/fullcit?p3003997.
Pełny tekst źródłaTypescript. Includes vita and abstract. Includes bibliographical references (leaves 111-115). Also available for download via the World Wide Web; free to University of Oregon users.
Cheung, Vinci. "Cognitive dysfunction implicated in the expression of attentional blink in schizophrenia /". Hong Kong : University of Hong Kong, 2002. http://sunzi.lib.hku.hk/hkuto/record.jsp?B25248534.
Pełny tekst źródłaMcClain, Maryellen Chute Douglas L. "Trends in symptom validity, memory and psychological test performance as functions of time and malingering rating /". Philadelphia, Pa. : Drexel University, 2004. http://dspace.library.drexel.edu/handle/1860/380.
Pełny tekst źródłaMitsis, Effie M. "Construct validity of executive functions in normal adults and in adults with mild cognitive impairment". Full text available, 2003. http://images.lib.monash.edu.au/ts/theses/mitsis.pdf.
Pełny tekst źródłaSaxena, Sarah. "Preventive and therapeutic physio-pathological aspects of peri-operative neurocognitive disorders". Electronic Thesis or Diss., Université de Lille (2022-....), 2022. http://www.theses.fr/2022ULILS034.
Pełny tekst źródłaNeurocognitive disorders present a major postoperative complication. After many years of speculation about the etiology of this complication, recent studies indicate that an inflammatory cascade, leading to the activation of microglia, may be the cause. In this work, we investigated the mechanisms responsible for postoperative cognitive disorders along two axes, one within the framework of clinical studies, the other by a pre-clinical approach on murine models.Thus, in the clinical study, the influence of lifestyle habits on the prevalence of perioperative neurocognitive disorders as well as known peripheral inflammatory markers (IL-6; HMGB1) known to be associated with perioperative neurocognitive disorders was studied. This study suggests that preoperative sdentarybehaviour is not a risk factor but that the absence of depressive tendencies/ presence of multilingualism would be protective factors. In addition, it reveals that the level of serum IL-6, unlike that of HMGB1, varies according to the type of surgery and the age of the patient. From this study, a formula was defined to predict postoperative cognitive decline based on perioperative blood IL-6 and HMGB1 levels.Several K+ channels (Kca3.1; Kv1.3; Kvir) have been identified on the cell surface of microglia, essential for its activation in pro-inflammatory phenotype. Attenuating microglial activation by blocking these channels may be one approach to preventing the development of neuroinflammation and cognitive decline.In our preclinical study, we showed that pharmacological and genetic inhibition of the Kv1.3 channel reduces postoperative neuroinflammation and cognitive decline. Similarly, we investigated the study of the role of the KCa3.1 channel. Our results suggest that TRAM34 administration caused cognitive decline and neuroinflammation compared to baseline.The KCa3.1 -/- model causes cognitive decline and is associated with more peripheral inflammation. Tibia fracture causes cognitive decline, microglial proliferation and peripheral inflammation. Isoflurane-based anesthesia causes cognitive decline and microglial proliferation
Palser, Eleanor R. "The role of interoception in cognition, and its application to autism spectrum disorders". Thesis, University College London (University of London), 2018. http://discovery.ucl.ac.uk/10060469/.
Pełny tekst źródłaFalter, Christine Michaela. "The influence of testosterone on cognition in typical development and Autism Spectrum Disorders". Thesis, University of Cambridge, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.612007.
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