Gotowe bibliografie tematyczne / Cholesterol

Gotowa bibliografia na temat „Cholesterol”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 27 lipca 2024

Utwórz poprawne odniesienie w stylach APA, MLA, Chicago, Harvard i wielu innych

Wybierz rodzaj źródła:

Spis treści

Zobacz listy aktualnych artykułów, książek, rozpraw, streszczeń i innych źródeł naukowych na temat „Cholesterol”.

Przycisk „Dodaj do bibliografii” jest dostępny obok każdej pracy w bibliografii. Użyj go – a my automatycznie utworzymy odniesienie bibliograficzne do wybranej pracy w stylu cytowania, którego potrzebujesz: APA, MLA, Harvard, Chicago, Vancouver itp.

Możesz również pobrać pełny tekst publikacji naukowej w formacie „.pdf” i przeczytać adnotację do pracy online, jeśli odpowiednie parametry są dostępne w metadanych.

Artykuły w czasopismach na temat "Cholesterol"

1

Liang, Congxin, Liqun Yan, J�rg-R. Hill, Carl S. Ewig, Terry R. Stouch i Arnold T. Hagler. "Force field studies of cholesterol and cholesteryl acetate crystals and cholesterol-cholesterol intermolecular interactions". Journal of Computational Chemistry 16, nr 7 (lipiec 1995): 883–97. http://dx.doi.org/10.1002/jcc.540160706.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
2

Vill, V., J. Thiem i P. Rollin. "Flüssigkristalline aromatische Cholesterin-Derivate". Zeitschrift für Naturforschung A 47, nr 3 (1.03.1992): 515–20. http://dx.doi.org/10.1515/zna-1992-0313.

Pełny tekst źródła
Streszczenie:
Abstract Liquid Crystalline Aromatic Cholesterol Derivates A series of aromatic cholesteryl ethers, esters, phenylcarbonates and benzylcarbonates were prepared and their liquid crystalline properties studied. The occurence of ferroelectric phases as well as properties of cholesteric and blue phases alternate with the number of linking atomes between steroid and atomatic system
Style APA, Harvard, Vancouver, ISO itp.
3

Robins, S. J., J. M. Fasulo, C. R. Pritzker, J. M. Ordovas i G. M. Patton. "Diurnal changes and adaptation by the liver of hamsters to an atherogenic diet". American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 269, nr 6 (1.12.1995): R1327—R1332. http://dx.doi.org/10.1152/ajpregu.1995.269.6.r1327.

Pełny tekst źródła
Streszczenie:
Studies were performed in freely feeding, male (F1B) Syrian hamsters fed a high-fat diet to determine the extent and manner of adaptation of the liver to diurnal changes in eating patterns and an increase in serum lipids. Serum cholesterol and triglycerides strongly paralleled changes in food consumption and were 40-50% greater during the 12-h dark period than the 12-h light period of the diurnal cycle. Hepatic cholesterol changes closely approximated changes in serum cholesterol (r = 0.916) due principally to changes in hepatic cholesteryl esters that were on average about 10-fold greater with the high-fat diet than with a chow diet. With the high-fat diet, hepatic cholesteryl esters were, however, extremely variable and were 40% greater at the mid-dark than at the mid-light period. With high fat there was also a marked increase in the secretion of very low density lipoproteins (VLDL) from the liver that were cholesteryl ester rich and closely paralleled the diurnal changes in hepatic cholesteryl esters (r = 0.911). In contrast, although with a high-fat diet biliary cholesterol secretion was increased, the increase in cholesterol in bile exhibited no diurnal pattern and with the high-fat diet was far less in magnitude than the increase of cholesterol in VLDL. Biliary cholesterol secretion is dependent on bile acid secretion. However, with the high-fat diet, neither the bile acid pool size nor bile acid secretion was increased compared with chow-fed controls. Moreover, with high fat at mid-dark period, bile acid secretion was significantly less than controls at mid-dark period. Thus in these hamsters a high-fat diet produced a marked increase in serum cholesterol that was distinctly diurnal and was compensated for by a diurnal increase in hepatic cholesteryl ester stores and the secretion of cholesteryl esters in VLDL. In contrast, cholesterol secretion in bile did not correspond to the fluctuating changes of cholesterol in the liver and was far less in magnitude than would be necessary to reduce a greatly expanded pool of hepatic cholesterol.
Style APA, Harvard, Vancouver, ISO itp.
4

Rodriguez-Agudo, Daniel, Shunlin Ren, Eric Wong, Dalila Marques, Kaye Redford, Gregorio Gil, Phillip Hylemon i William M. Pandak. "Intracellular cholesterol transporter StarD4 binds free cholesterol and increases cholesteryl ester formation". Journal of Lipid Research 49, nr 7 (9.04.2008): 1409–19. http://dx.doi.org/10.1194/jlr.m700537-jlr200.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
5

Khan, B., H. G. Wilcox i M. Heimberg. "Cholesterol is required for secretion of very-low-density lipoprotein by rat liver". Biochemical Journal 258, nr 3 (15.03.1989): 807–16. http://dx.doi.org/10.1042/bj2580807.

Pełny tekst źródła
Streszczenie:
To study potential effects of hepatic cholesterol concentration on secretion of very-low-density lipoprotein (VLDL) by the liver, male rats were fed on unsupplemented chow, chow with lovastatin (0.1%), or chow with lovastatin (0.1%) and cholesterol (0.1%) for 1 week. Livers were isolated from these animals and perfused in vitro, with a medium containing [2-14C]acetate, bovine serum albumin and glucose in Krebs-Henseleit buffer, and with an oleate-albumin complex. With lovastatin feeding, the hepatic concentrations of cholesteryl esters and triacylglycerols before perfusion were decreased, although free cholesterol was unchanged. However, hepatic secretion of all the VLDL lipids was decreased dramatically by treatment with lovastatin. Although total secretion of VLDL triacylglycerol, phospholipid, cholesterol and cholesteryl esters was decreased, the decrease in triacylglycerol was greater than that in free cholesterol or cholesteryl esters, resulting in secretion of a VLDL particle enriched in sterols relative to triacylglycerol. In separate studies, the uptake of VLDL by livers from control animals or animals treated with lovastatin was measured. Uptake of VLDL was estimated by disappearance of VLDL labelled with [1-14C]oleate in the triacylglycerol moiety, and was observed to be similar in both groups. During perfusion, triacylglycerol accumulated to a greater extent in livers from lovastatin-fed rats than in control animals. The depressed output of VLDL triacylglycerols and the increase in triacylglycerol in the livers from lovastatin-treated animals was indicative of a limitation in the rate of VLDL secretion. Addition of cholesterol (either free cholesterol or human low-density lipoprotein) to the medium perfusing livers from lovastatin-fed rats, or addition of cholesterol to the diet of lovastatin-fed rats, increased the hepatic concentration of cholesteryl esters and the output of VLDL lipids. The concentration of cholesteryl esters in the liver was correlated with the secretion of VLDL by the liver. These data suggest that cholesterol is an obligate component of the VLDL required for its secretion. It is additionally suggested that cholesteryl esters are in rapid equilibrium with a small pool of free cholesterol which comprises a putative metabolic pool available and necessary for the formation and secretion of the VLDL. Furthermore, the specific radioactivity (d.p.m./mumol) of the secreted VLDL free cholesterol was much greater than that of hepatic free cholesterol, suggesting that the putative hepatic metabolic pool is only a minor fraction of total hepatic free cholesterol.
Style APA, Harvard, Vancouver, ISO itp.
6

Gylling, H., S. Pyrhönen, E. Mäntylä, H. Mäenpää, L. Kangas i T. A. Miettinen. "Tamoxifen and toremifene lower serum cholesterol by inhibition of delta 8-cholesterol conversion to lathosterol in women with breast cancer." Journal of Clinical Oncology 13, nr 12 (grudzień 1995): 2900–2905. http://dx.doi.org/10.1200/jco.1995.13.12.2900.

Pełny tekst źródła
Streszczenie:
PURPOSE Long-term effects of tamoxifen and toremifene, a new antiestrogen that closely resembles tamoxifen, were investigated on serum lipids and cholesterol metabolism. PATIENTS AND METHODS The study group consisted of 24 postmenopausal Finnish women with advanced breast cancer from an international multicenter study of 415 patients. Cholesterol metabolism was evaluated by measuring the cholesterol precursor (delta 8-cholestenol, desmosterol, and lathosterol, reflecting cholesterol synthesis) and plant sterol (markers of cholesterol absorption) and cholestanol levels by gas-liquid chromatography. RESULTS Tamoxifen and toremifene lowered significantly serum low-density lipoprotein (LDL) cholesterol levels after 12 months of treatment by 16% and 15%, with no change in high-density lipoprotein (HDL) cholesterol or serum triglyceride levels. Serum delta 8-cholestenol was increased 40- and 55-fold during toremifene and tamoxifen treatment, respectively, while the increase of desmosterol less than doubled and was lacking for lathosterol by toremifene. Plant sterols and cholestanol were only inconsistently increased in serum. CONCLUSION Tamoxifen and toremifene inhibit the conversion of delta 8-cholestenol to lathosterol so that serum total and LDL cholesterol levels are lowered by downregulation of cholesterol synthesis. Thus, inhibition of the delta 8-isomerase may be the major hypolipidemic effect of these agents. Reduced risk of coronary artery disease will probably occur also during long-term toremifene treatment, because the drug reduces cholesterol and its synthesis, similarly to tamoxifen.
Style APA, Harvard, Vancouver, ISO itp.
7

Lystvan, V., A. Zhmurchuk i V. Lystvan. "SYNTHESIS OF POTENTIAL LIQUID CRYSTALS WITH CHOLESTEROL FRAGMENT BY WITTIG REACTION". Ukrainian Journal of Natural Sciences, nr 2 (28.01.2023): 143–54. http://dx.doi.org/10.35433/naturaljournal.2.2023.144-154.

Pełny tekst źródła
Streszczenie:
Liquid crystals are substances that owing to the features of their structure and physical properties are of interest not only as objects for theoretical research, but also significantly important practically due to the possibilities of their effective application in various brunches of industry, medicine, in household etc. Among the known classes of liquid crystals, substances known as cholesterics are an important group. Cholesteric liquid crystals demonstrate very high optical activity, that significantly exceeds the optical activity of most other known classes of organic compounds. Their ability for appreciable change of color at change of the temperature and environment composition is also practically important. Among all compounds belonging to the class of cholesterics an important place possess cholesterol derivatives, especially cholesterol esters. Therefore, the elaboration of new methods of their synthesis and the introduction of new functional groups into their molecules are an urgent tasks indeed. This work is devoted to the investigation of the possibility of synthesis of new cholesterol derivatives, namely cholesteryl esters of unsaturated acids by the Wittig reaction - the interaction of various classes of aldehydes with phosphonium salts, with the intermediate formation of phosphorus ylides - alkylidene phosphoranes. We found that the Wittig reaction is a convenient method for the synthesis of cholesteryl esters of unsaturated carboxylic acids. We have developed methods for the synthesis of potential cholesteric liquid crystals by the Wittig reaction, which use a phosphonium salt containing a cholesterol fragment and corresponding aldehydes. The process proceed without the release of an intermediate compound – alkylidene phosphorane, which reduces the labor intensity of the method. The application of various aldehydes enables the easily obtaining of cholesteryl esters of unsaturated acids containing various aliphatic, aromatic and heterocyclic fragments in the acid radical. The reaction takes place in mild conditions, without using of high temperatures or aggressive environments. The resulting esters show signs of mesophase formation, which is a confirmation of their liquid crystalline properties.
Style APA, Harvard, Vancouver, ISO itp.
8

Scobey, M. W., F. L. Johnson i L. L. Rudel. "Delivery of high-density lipoprotein free and esterified cholesterol to bile by the perfused monkey liver". American Journal of Physiology-Gastrointestinal and Liver Physiology 257, nr 4 (1.10.1989): G644—G652. http://dx.doi.org/10.1152/ajpgi.1989.257.4.g644.

Pełny tekst źródła
Streszczenie:
The movement of cholesterol from high-density lipoproteins (HDL) into bile has been studied using perfused livers from cholesterol-fed African Green monkeys. Mass amounts of HDL were isolated from the plasma of African Green monkeys and were doubly labeled with either 125I-apolipoprotein and [3H]cholesteryl ester or with [3H]cholesteryl ester and [14C]cholesterol. For 3 h of perfusion HDL-free cholesterol was cleared from perfusate at a faster rate than HDL ester cholesterol which, in turn, was cleared at a faster rate than HDL protein. [14C]cholesterol from HDL appeared in biliary bile acids and cholesterol at a higher rate than [3H]esterified cholesterol from HDL. The specific activities of biliary [14C]cholesterol and HDL-free [14C]cholesterol had equilibrated by 60 min of perfusion, although the specific activity of whole liver free [14C]cholesterol was still only 46% of that in bile at 180 min of perfusion. In contrast, the specific activity of total liver free [3H]cholesterol was equal to that of biliary [3H]cholesterol by 180 min of perfusion. We conclude that, in this primate model, HDL-free cholesterol enters into a hepatic compartment that communicates with biliary cholesterol and bile acid precursor pools more efficiently than with other liver pools of cholesterol, whereas HDL-esterified cholesterol appears to mix with all liver pools with equal efficiency. Overall, these data support the concept of compartmentalization of cholesterol in the liver.
Style APA, Harvard, Vancouver, ISO itp.
9

Pincinato, Eder de Carvalho, Patricia Moriel i Dulcinéia Saes Parra Abdalla. "Cholesterol oxides inhibit cholesterol esterification by lecithin: cholesterol acyl transferase". Brazilian Journal of Pharmaceutical Sciences 45, nr 3 (wrzesień 2009): 429–35. http://dx.doi.org/10.1590/s1984-82502009000300007.

Pełny tekst źródła
Streszczenie:
Cholesterol oxides are atherogenic and can affect the activity of diverse important enzymes for the lipidic metabolism. The effect of 7β-hydroxycholesterol, 7-ketocholesterol, 25-hydroxycholesterol, cholestan-3β,5α,6β-triol,5,6β-epoxycholesterol, 5,6α-epoxycholesterol and 7α-hydroxycholesterol on esterification of cholesterol by lecithin:cholesterol acyl transferase (LCAT, EC 2.3.1.43) and the transfer of esters of cholesterol oxides from high density lipoprotein (HDL) to low density lipoproteins (LDL) and very low density lipoproteins (VLDL) by cholesteryl ester transfer protein (CETP) was investigated. HDL enriched with increasing concentrations of cholesterol oxides was incubated with fresh plasma as source of LCAT. Cholesterol and cholesterol oxides esterification was followed by measuring the consumption of respective free sterol and oxysterols. Measurements of cholesterol and cholesterol oxides were done by gas-chromatography. 14C-cholesterol oxides were incorporated into HDL2 and HDL3 subfractions and then incubated with fresh plasma containing LCAT and CETP. The transfer of cholesterol oxide esters was followed by measuring the 14C-cholesterol oxide-derived esters transferred to LDL and VLDL. All the cholesterol oxides studied were esterified by LCAT after incorporation into HDL particles, competing with cholesterol by LCAT. Cholesterol esterification by LCAT was inversely related to the cholesterol oxide concentration. The esterification of 14C-cholesterol oxides was higher in HDL3 and the transfer of the derived esters was greater from HDL2 to LDL and VLDL. The results suggest that cholesterol esterification by LCAT is inhibited in cholesterol oxide-enriched HDL particles. Moreover, the cholesterol oxides-derived esters are efficiently transferred to LDL and VLDL. Therefore, we suggest that cholesterol oxides may exert part of their atherogenic effect by inhibiting cholesterol esterification on the HDL surface and thereby disturbing reverse cholesterol transport.
Style APA, Harvard, Vancouver, ISO itp.
10

Hallikainen, Maarit, Henri Tuomilehto, Tarja Martikainen, Esko Vanninen, Juha Seppä, Jouko Kokkarinen, Jukka Randell i Helena Gylling. "Cholesterol Metabolism and Weight Reduction in Subjects with Mild Obstructive Sleep Apnoea: A Randomised, Controlled Study". Cholesterol 2013 (16.05.2013): 1–9. http://dx.doi.org/10.1155/2013/769457.

Pełny tekst źródła
Streszczenie:
To evaluate whether parameters of obstructive sleep apnoea (OSA) associate with cholesterol metabolism before and after weight reduction, 42 middle-aged overweight subjects with mild OSA were randomised to intensive lifestyle intervention (N=23) or to control group (N=18) with routine lifestyle counselling only. Cholesterol metabolism was evaluated with serum noncholesterol sterol ratios to cholesterol, surrogate markers of cholesterol absorption (cholestanol and plant sterols) and synthesis (cholestenol, desmosterol, and lathosterol) at baseline and after 1-year intervention. At baseline, arterial oxygen saturation (SaO2) was associated with serum campesterol (P<0.05) and inversely with desmosterol ratios (P<0.001) independently of gender, BMI, and homeostasis model assessment index of insulin resistance (HOMA-IR). Apnoea-hypopnoea index (AHI) was not associated with cholesterol metabolism. Weight reduction significantly increased SaO2and serum cholestanol and decreased AHI and serum cholestenol ratios. In the groups combined, the changes in AHI were inversely associated with changes of cholestanol and positively with cholestenol ratios independent of gender and the changes of BMI and HOMA-IR (P<0.05). In conclusion, mild OSA seemed to be associated with cholesterol metabolism independent of BMI and HOMA-IR. Weight reduction increased the markers of cholesterol absorption and decreased those of cholesterol synthesis in the overweight subjects with mild OSA.
Style APA, Harvard, Vancouver, ISO itp.
Więcej źródeł

Rozprawy doktorskie na temat "Cholesterol"

1

Edington, J. "The relationship of dietary cholesterol to plasma cholesteroll". Thesis, University of Oxford, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.236284.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
2

Malcolmson, Richard Joseph. "Physical studies of cholesterol and cholesteryl esters in model membranes". Thesis, Edinburgh Napier University, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.385910.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
3

Velarde, Laos Edmundo, i Ana Gonzalez. "Cholesterol and Cholesterol Oxides in Chicken Meat". Revista de Química, 2012. http://repositorio.pucp.edu.pe/index/handle/123456789/99119.

Pełny tekst źródła
Streszczenie:
Se determinó el contenido de lípidos totales, colesterol yóxidos de colesterol en carne de pollo adquiridos en 3avícolas de Lima. La carne de pollo presentó valores de lípidostotales y colesterol diferentes porpresentó los menores valores demientras que la piel presentó losavícola. La carne de pecholípidos totales y colesterol,mayores valores de estos. Por cromatografía de gases acoplado a espectrómetro demasas (CG-EM) se confirmó la estructura de los siguientes óxidosde colesterol: 7-cetocolesterol, en las 3 avícolas; 7 β -hidroxicolesterol en ala de la avícola 1 y piel de la avícola 2; 7 -hidroxicolesterol y α - epoxicolesterol en piel de la avícola 2.
The contents of tota lipids, cholesterol and cholesterol oxides we determined in chicken meat purchased fromen farms in Lima. Chicken meat presented differentvalues of total lipids and cholesterol. Breast chicken presentedthe lowest values of total lipids and cholesterol while the skinpresented the highest values.A gas chromatograph – mass spectrometer (GC – MS) was employed to confirm the structure of theoxides: 7-ketocholesterol was found infarms ; 7- hydroxycholesterol was foundfollowing cholesterolthe three chickenin wing of chickenfarm 1 and in the skin of chicken farm 2; 7-hydroxycholesteroland - epoxycholesterol in the skin of chicken farm 2.
Style APA, Harvard, Vancouver, ISO itp.
4

Kingdon, Lorraine B. "Speedy Cholesterol". College of Agriculture, University of Arizona (Tucson, AZ), 1990. http://hdl.handle.net/10150/295659.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
5

Famer, Daniel. "Implications of cholesterol and cholesterol-lowering therapy in Alzheimer's disease /". Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-260-6/.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
6

Burkett, Paul A. "Frequent cholesterol feedback as an aid in lowering cholesterol levels". Thesis, Virginia Tech, 1989. http://hdl.handle.net/10919/44704.

Pełny tekst źródła
Streszczenie:
Twenty six male and two female participants in the Cardiac Therapy Program at Virginia Tech were stratified, based upon level of total cholesterol (TC) and length of time in the Cardiac Program, and then randomly assigned to either experimental or control groups. Participants ranged in age from 43 to 68 years and all had baseline TC levels greater than 200 mg/dl. There were no significant differences between groups in terms of baseline TC (control M : 248 mg/dl; experimental M = 251 mg/dl), blood pressure (BP), weight, predicted percent body fat, dietary fat/cholesterol, age, education, or program attendance.
Master of Science
Style APA, Harvard, Vancouver, ISO itp.
7

Alasmi, Mahmood Mohamed. "EFFECTS OF CHOLESTEROL SUPPLEMENTATION ON CHOLESTEROL SYNTHESIS RATES IN INFANTS". University of Cincinnati / OhioLINK, 2000. http://rave.ohiolink.edu/etdc/view?acc_num=ucin974741712.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
8

Norlin, Maria. "Cytochrome P450 Enzymes in the Metabolism of Cholesterol and Cholesterol Derivatives". Doctoral thesis, Uppsala University, Department of Pharmaceutical Biosciences, 2000. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-1086.

Pełny tekst źródła
Streszczenie:

Cholesterol is metabolized to a variety of important biological products in the body including bile acids and vitamin D. The present investigation is focused on enzymes that catalyze 7α-hydroxylation or 27-hydroxylation in the metabolism of cholesterol, oxysterols (side chain-hydroxylated derivatives of cholesterol) and vitamin D3. The enzymes studied belong to the cytochrome P450 enzyme families CYP7 and CYP27.

The study describes purification of a cytochrome P450 enzyme fraction active in 7α-hydroxylation of 25-hydroxycholesterol, 27-hydroxycholesterol, dehydroepiandrosterone and pregnenolone from pig liver microsomes. Peptide sequence analysis indicated that this enzyme fraction contains an enzyme belonging to the CYP7B subfamily. The purified enzyme was not active towards cholesterol or testosterone. Purification and inhibition experiments suggested that hepatic microsomal 7α -hydroxylation of 27-hydroxycholesterol and dehydroepiandrosterone involves at least two enzymes, probably closely related.

The study shows that recombinantly expressed human and rat cholesterol 7α -hydroxylase (CYP7A) and partially purified pig liver cholesterol 7α -hydroxylase are active towards 20(S)-, 24-, 25- and 27-hydroxycholesterol. CYP7A was previously considered specific for cholesterol and cholestanol. The 7α -hydroxylation of 20(S)-, 25-, and 27-hydroxycholesterol in rat liver was significantly increased by treatment with cholestyramine, an inducer of CYP7A. Cytochrome P450 of renal origin showed 7α -hydroxylase activity towards 25- and 27-hydroxycholesterol, dehydroepiaundrosterone and pregnenolone but not towards 20(S)-, 24-hydroxycholesterol or cholesterol. The results indicate a physiological role for CYP7A as an oxysterol 7α -hydroxylase, in addition to the previously known human oxysterol 7α -hydroxylase CYP7B.

The role of renal sterol 27-hydroxylase (CYP27A) in the bioactivation of vitamin D3 was studied with cytochrome P450 fractions purified from pig kidney mitochondria. Purification and inhibition experiments and experiments with a monoclonal antibody against CYP27A indicated that CYP27A plays a role in renal 25-hydroxyvitamin D3 l α -hydroxylation.

The expression of CYP7A, CYP7B and CYP27A during development was studied. The levels of CYP27A in livers of newborn and six months old pigs were similar whereas the levels of CYP7A increased. The expression of CYP7B varied depending on the tissue. The expression of CYP7B increased with age in the liver whereas the CYP7B levels in kidney showed a marked age-dependent decrease.

Style APA, Harvard, Vancouver, ISO itp.
9

Trevenen, Alexandra H. "Investigations of phospholipid/cholesterol and cholesterol derivative interactions in model membranes". Thesis, Imperial College London, 2010. http://hdl.handle.net/10044/1/5582.

Pełny tekst źródła
Streszczenie:
Multinuclear solid state MAS NMR (1H and 31P), 31P CSA solid state NMR, 2H NMR and x-ray diffraction techniques have been used to compare the structure and properties of DPPC: Andro and DPPC: 4β-hydroxycholesterol with that of the properties known of DPPC: Chol mixtures in excess water. The formation of the Lo phase is known to occur with PC: Chol mixtures with sufficient concentrations of cholesterol. The formation and properties of the Lo phase was looked at with the cholesterol derivatives Andro (lacking in the hydrocarbon tail present in cholesterol) and 4β-hydroxycholesterol (possessing an extra hydroxyl group adjacent to the one present at the headgroup region of cholesterol). The Lo phase shows fluid-fluid immisibility when combined with the disordered Lα phase. It is this heterogeneity that is believed to be important in the formation of lipid rafts, which are thought to play a role in the function of living cells. Cholesterol desorption from DOPC model membranes was examined using methyl-β-cyclodextrin, which has a high affinity for cholesterol and facilitates the mechanism of cholesterol desorption from lipid membranes. By running a number of experiments, measuring cholesterol desorption from MLV’s and LUV’s a cholesterol flip-flop rate within DOPC model membranes was measured using 1H solution state NMR. A number of methods were also attempted to synthesise an asymmetric DOPC: DPPC membranes. These included a split glass slides method and a modified version of an emulsion method as described by Pautot et al. lipid asymmetry with respect to the split glass slides and emulsion methods was to be observed by NMR, where any cholesterol asymmetry formed via desorption by methyl-β- cyclodextrin was to be evaluated via the use of paramagnetic NMR experiments, however no asymmetry was observed in either experiment.
Style APA, Harvard, Vancouver, ISO itp.
10

Beehler, Kaitlyn. "MiR-1908 Is a Cholesterol Responsive MicroRNA Implicated In Cholesterol Regulation". Thesis, Université d'Ottawa / University of Ottawa, 2020. http://hdl.handle.net/10393/40422.

Pełny tekst źródła
Streszczenie:
Leveraging miRNA-Seq data and the 1000 Genomes imputed genotypes, we identified rs174561-C as a strong miRQTL for circulating miRNA-1908-5p (P=4.8x10-31) which has an inverse relationship with circulating LDL-C, fasting glucose and A1c. Here I investigated the molecular mechanism(s) linking miR1908-5p to cholesterol metabolism. First, by overexpression experiments in HuH-7 cells demonstrate that the presence of the C allele, associated with lower LDL-C levels, significantly increases miR-1908-5p by 2.15-fold relative to the T allele. Further experiments revealed that 72-hour cholesterol depletion increases miR-1908-5p expression (2.11-fold) whereas cholesterol loading decreases miR-1908-5p expression (0.69-fold). Differential miR-1908-5p expression was then used to profile genes involved in lipoprotein signaling and cholesterol metabolism using a PCR array to identify LDLR as a gene of interest. Although total RNA and protein expression of LDLR was unchanged in response to differential miR-1908-5p expression, the ratio of the mature form to the cleaved form of LDLR decreased following miR-1908-5p inhibition (0.85-fold) and conversely, increased with mimic treatment (1.63-fold). Cleavage of the mature LDLR is known to reduce cell surface affinity for LDL. These findings uncover a potential mechanism linking miR-1908-5p to lower LDL-cholesterol levels through reduced LDLR cleavage.
Style APA, Harvard, Vancouver, ISO itp.
Więcej źródeł

Książki na temat "Cholesterol"

1

Roth, Eli. Good cholesterol, bad cholesterol. Wyd. 2. Rocklin, CA: Prima Pub., 1995.

Znajdź pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
2

Roth, Eli. Good cholesterol, bad cholesterol. Rocklin, CA: Prima Pub. & Communications, 1988.

Znajdź pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
3

Roth, Eli. Good cholesterol, bad cholesterol. Rocklin, CA: Prima Pub. & Communications, 1988.

Znajdź pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
4

A, Kramer M., red. Cholesterol. Hauppauge, N.Y: Nova Science Publishers, 2005.

Znajdź pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
5

Møller, Jens. Cholesterol. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71600-3.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
6

Daninos, Jean-Michel. Cholesterol. Warszawa: W.A.B., 1995.

Znajdź pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
7

Carole, Michaud, red. Cholesterol. Montréal, Québec, Canada: Modus Vivendi, 2014.

Znajdź pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
8

Sabine, John R. Cholesterol. Ann Arbor, Mich: University Microfilms International, 1992.

Znajdź pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
9

G, Williams David. Cholesterol. Ingram, TX: Mountain Home Pub., 1988.

Znajdź pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
10

Lupovici, Zaharia. Good cholesterol, bad cholesterol, and the most discussed cholesterol-- HDL. New York: Vantage Press, 1992.

Znajdź pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
Więcej źródeł

Części książek na temat "Cholesterol"

1

Møller, Jens. "Cholesterol". W Cholesterol, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71600-3_1.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
2

Møller, Jens. "Infarction Due to Metabolic Processes Other than Vessel Occlusion". W Cholesterol, 18–21. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71600-3_10.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
3

Møller, Jens. "Testosterone as an Alternative to Surgery for CVD". W Cholesterol, 22–23. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71600-3_11.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
4

Møller, Jens. "Diabetic States and CVD". W Cholesterol, 24–28. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71600-3_12.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
5

Møller, Jens. "Use of Anabolic Steroids in Surgical Stress". W Cholesterol, 29–30. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71600-3_13.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
6

Møller, Jens. "Increasing Testosterone and Decreasing Cholesterol by Physical Training". W Cholesterol, 31–32. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71600-3_14.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
7

Møller, Jens. "The Negative Nitrogen Balance". W Cholesterol, 33–36. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71600-3_15.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
8

Møller, Jens. "Evaluation of the Parameters of CVD". W Cholesterol, 37–38. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71600-3_16.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
9

Møller, Jens. "Comments on Photographs". W Cholesterol, 39–45. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71600-3_17.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
10

Møller, Jens. "The Balance Between Hormone Sensitive Lipase and Postheparin Lipoprotein Lipase". W Cholesterol, 47. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71600-3_18.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.

Streszczenia konferencji na temat "Cholesterol"

1

Pescador, R., R. Porta, R. Niada, M. Mantovani i G. Prino. "DEFIBROTIDE DECREASES CHOLESTEROL CONTENT IN HYPERCHOLESTEROLEMIC RABBIT AORTA, WITH NO MODIFICATION OF PLASMA OR LIPOPROTEIN CHOLESTEROL". W XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643153.

Pełny tekst źródła
Streszczenie:
Defibrotide was shown to stimulate the production of endogenous PGI2 from rat and hamster aortic tissue, as well as the production of PGi2 frcm the coronary vascular bed in the platelet perfused heart model. This effect was only seen in presence of platelets. Durirg the infusion period with Defibrotide thromboxane release remained unaffected while platelet cAMP rised. Defibrotide, was also able to reduce the secretion of ATP from platelets as well as to deaggregate platelet cluTps. It has been postulated that long-term administration of stable PGI^ metabolites or analogues could be a more useful means of enhancing cholesterol and cholesteryl ester mobilization out of the arterial “ foam” cell during early stages of cardiovascular disease. These observations prompted us to administer Defibrotide i.v. to cholesterol fed rabbits to verify if it could cause a decrease in cholesterol content of aortas. Aorta cholesterol was evaluated by gas chromatography. Plasma or lipoprotein lipids were assayed by enzymatic kits from Boehringer Biochemia. Plasma lipoproteins were separated by density gradient ultracentrifugation. Platelet aggregation was carried out with ADP using an optical aggregometer to find out the effective aggregatory concentration fifty (EC). Histology of the rabbit hearts was performed by optical microscopy on heart sections coloured with hematoxylin-eosine. Defibrotide caused a decrease (-49%, P < 0.05, vs. cholesterol fed rabbits treated with placebo) in cholesterol content of aortas with no modification of total plasma cholesterol, triglyceride and phospholipid. The cholesterol, triglyceride, phospholipid and protein of plasma lipoproteins were not affected too. The EC of rabbits treated with Defibrotide was 50 normalized (-9%, N.S. vs. control animals fed the normal diet and treated with placebo; cholesterol fed animals treated with placebo: -32%, P< 0.05 vs. control animals fed the normal diet). Vascular lesions in the hearts of animals treated with Defibrotide had a lower rate (33%) in comparison to that (87%) of animals fed with cholesterol and treated with placebo. It is concluded that the ability of Defibrotide to stimulate vascular PGI2 formation and to reduce platelet sensitivity could be helpful in reducing the amount of cholesterol in the cardiovascular system in atherosclerotic prone situations.
Style APA, Harvard, Vancouver, ISO itp.
2

Perdani, Meka Saima, Muhamad Sahlan, Siti Farida, Dwini Normayulisa Putri, Sri Angky Soekanto i Heri Hermansyah. "Kinetic study of cholesterol oxidation by cholesterol oxidase enzyme as application for cholesterol biosensor". W SECOND INTERNATIONAL CONFERENCE OF MATHEMATICS (SICME2019). Author(s), 2019. http://dx.doi.org/10.1063/1.5096731.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
3

Heiremans, J., M. Claeys i A. G. Herman. "DETERMINATION OF CHOLESTERYL HYDROXYOCTADBCADIENOATES IN VASCULAR TISSUE BY HPLC AND ITS RELEVANCE TO ATHEROSCLEROSIS". W XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643084.

Pełny tekst źródła
Streszczenie:
Accumulation of lipids in the intimal arterial layer, and of cholesterol esters in particular, has been recognised as an early and prominent phenomenon in atherogenesis. Several attempts have been made to link putative peroxidation of these lipids in vivo to causal or deteriorating etiological determinants of plaque formation. The occurrence in advanced human atheromata of oxidized derivatives of cholesteryl linoleate -a major polyunsaturated cholesterol ester species in plasma and vessel wall - has been described by Brooks et al. (Atherosclerosis, 1970,13,223) and a positive correlation between the amount of cholesteryl hydroxyoctadecadienoates (CHODES) and the stage of the lesion has also been reported. In addition Funk and Powell (J. Biol. Chem., 1985,260,7481) have found hydroxyoctadecadienoic acids in normal aorta of different species, wich were strikingly increased after alkaline hydrolysis of total lipids, and this in contrast with the arachidonic acid analogs. The aim of this study was to develop a sensitive and practical method for specific assay of CHODES, without resorting to laborious saponification and derivatisation procedures required for gas chromatographic analysis, which could moreover augment the risk for artefacts.Dog thoracal aorta was homogenised and lipids were extracted using the Folch method with CHCl3/CH30H;2/l containing 0.05mM butylated hydroxytoluene. Fractionation of CHODES from neutral lipids was carried out by thin-layer chromatography. For detection and quantification a high-performance liquid chromatography (HPI/2) assay method was developed, with UV monitoring at 232nm , a wavelength characteristic for conjugated dienes with vicinal hydroxyl function. Reference compounds and the internal standard for HPLC analysis were synthesized from linoleic acid and 10,13,16-docosatrienoic acid, respectively, by preparation of hydroxy fatty acids with soybean lipoxygenase and subsequent esterification to cholesterol esters with pancreas cholesterol esterase. Confirmation of the structural identity was obtained by mass spectrometry. Artefactual formation of CHODES ex vivo was investigated by subjecting radiolabeled cholesteryl linoleate through the analysis procedure. This method allows the specific detection of CHODES in non-atherosclerotic arteries which was hitherto only reported for human advanced atherosclerotic lesions and is proposed as a sensitive and specific probe for prospective survey of lipid peroxidation in atherosclerotic blood vessels.
Style APA, Harvard, Vancouver, ISO itp.
4

Zhang, Peipei, Jun Huang, Mengshi Li, Pengfei Zhang i Liyun Ding. "A fiber optic cholesterol biosensor based on magnetic immobilized cholesterol oxidase". W Asia-Pacific Optical Sensors Conference. Washington, D.C.: OSA, 2016. http://dx.doi.org/10.1364/apos.2016.w4a.59.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
5

Ghelmez, Mihaela A., Maria Honciuc i Cristian Gheorghe. "Estimation of the cholesterol percentages in mixtures of arachidonic acid and cholesterol". W OE/LASE '94, redaktor Joseph R. Lakowicz. SPIE, 1994. http://dx.doi.org/10.1117/12.182774.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
6

Kokal, Sunil L., i Selim G. Sayegh. "Asphaltenes: The Cholesterol Of Petroleum". W Middle East Oil Show. Society of Petroleum Engineers, 1995. http://dx.doi.org/10.2118/29787-ms.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
7

Perdani, Meka Saima, Muhammad Faturrohman, Dwini Normayulisa Putri i Heri Hermansyah. "Oxidation of extracted cholesterol from fatty food by using crude cholesterol oxidase Streptomyces sp." W THE 4TH BIOMEDICAL ENGINEERING’S RECENT PROGRESS IN BIOMATERIALS, DRUGS DEVELOPMENT, HEALTH, AND MEDICAL DEVICES: Proceedings of the International Symposium of Biomedical Engineering (ISBE) 2019. AIP Publishing, 2019. http://dx.doi.org/10.1063/1.5139357.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
8

Rathinam, R., Devesh Pratap Singh, Arjun Dutta, S. Rudresha, Shaikh Rajesh Ali i Pratik Chatterjee. "TiO<sub>2</sub> Nanoparticles Based Peroxidase Mimics for Colorimetric Sensing of Cholesterol and Hydrogen Peroxide". W International Conference on Recent Advancements in Biomedical Engineering. Switzerland: Trans Tech Publications Ltd, 2022. http://dx.doi.org/10.4028/p-7in58j.

Pełny tekst źródła
Streszczenie:
It's still a challenge to detect cholesterol more rapidly and easily. As a result, using a colorimetric sensor to detect cholesterol may result in the development of a ready-to-use. This research shows how a TiO2 nanoparticles may be utilized as a peroxidase mimic to detect H2O2 and free cholesterol. The TiO2 Nanoparticles was carefully studied in terms of structural, morphological, and functional characteristics. In the existence of cholesterol oxidase, and the proposed approach comprises detecting H2O2 produced during cholesterol oxidation. The TiO2 nanoparticles sensor has shown good cholesterol detection sensitivity with a LOD as low as 0.061 M and a linear response in the 0.1 mM to 50 mM range.
Style APA, Harvard, Vancouver, ISO itp.
9

Pakapongpan, Saithip, Adisorn Tuantranont i Pornpimol Sritongkham. "Cholesterol biosensor based on direct electron transfer of cholesterol oxidase on multi-wall carbon nanotubes". W 2011 Biomedical Engineering International Conference (BMEiCON) - Conference postponed to 2012. IEEE, 2012. http://dx.doi.org/10.1109/bmeicon.2012.6172037.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
10

Li, Mengshi, Jun Huang, Peipei Zhang, Pengfei Zhang i Liyun Ding. "Immobilization of cholesterol oxidase on SiO2 nanoparticles and its application in Fiber optic cholesterol sensor". W Asia-Pacific Optical Sensors Conference. Washington, D.C.: OSA, 2016. http://dx.doi.org/10.1364/apos.2016.w4a.33.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.

Raporty organizacyjne na temat "Cholesterol"

1

Hausmann, Ricardo, i Eduardo Fernández-Arias. Foreign Direct Investment: Good Cholesterol? Inter-American Development Bank, marzec 2000. http://dx.doi.org/10.18235/0010777.

Pełny tekst źródła
Streszczenie:
This paper studies the proposition that capital inflows tend to take the form of FDI -i.e., the share of FDI in total liabilities tends to be higher- in countries that are safer, more promising and with better institutions and policies. It finds that this view is patently wrong since it stands the historical record on its head. It then uses alternative theories to make sense of the facts. It begins by studying the determinants of the size and composition of the flows of private capital across countries. It finds that while capital flows tend to go to countries that are safer and have better institutions and financial markets, the share of FDI in total flows is not an indication of good health. On the contrary, countries that are riskier, less financially developed and have weaker institutions tend to attract less capital but more of it in the form of FDI. Hence, interpreting the rising share of FDI as a sign of good health is unwarranted. This is even more so, given that FDI's recent rise has taken place while total private capital inflows have fallen.
Style APA, Harvard, Vancouver, ISO itp.
2

Hung, Hsuan-Yu, Hui-Hsiung Lai, Hui-Chuan Lin i Chung-Yu Chen. Impact of interferon-free antivirus therapy on lipid profiles in patients with chronic hepatitis C: A network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, lipiec 2022. http://dx.doi.org/10.37766/inplasy2022.7.0055.

Pełny tekst źródła
Streszczenie:
Review question / Objective: P: ("Hepatitis C"[Mesh] AND "Hepacivirus"[Mesh] AND "Hepatitis C, Chronic”[Mesh]) I: (direct acting antiviral OR asunaprevir OR boceprevir OR daclatasvir OR dasabuvir OR elbasvir OR glecaprevir OR grazoprevir OR ledipasvir OR ombitasvir OR paritaprevir OR pibrentasvir OR simeprevir OR sofosbuvir OR telaprevir OR velpatasvir OR voxilaprevir) C: placebo O: ( "Cholesterol, VLDL"[Mesh] OR "Cholesterol, LDL"[Mesh] OR "Cholesterol, HDL"[Mesh] OR "Dyslipidemias"[Mesh] OR "lipoprotein cholesterol ester, human" [Supplementary Concept] OR "lipoprotein cholesterol" [Supplementary Concept] ) OR ((lipoprotein cholesterol) OR ("lipidemia") OR (lipid metabolism) OR (lipid)). Information sources: We conducted a comprehensive literature search of PubMed, Cochrane Library, Embase, and Ovid MEDLINE electronic databases from their inception to May 20, 2022.
Style APA, Harvard, Vancouver, ISO itp.
3

Min, Byungrok, Il Suk Kim i Dong U. Ahn. Dietary Cholesterol Affects Lipid Metabolism in Rabbits. Ames (Iowa): Iowa State University, styczeń 2015. http://dx.doi.org/10.31274/ans_air-180814-1348.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
4

Freeman, Michael R. A Cholesterol-Sensitive Regulator of the Androgen Receptor. Fort Belvoir, VA: Defense Technical Information Center, lipiec 2010. http://dx.doi.org/10.21236/ada543533.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
5

Hur, Sun Jin, Kwon Il Seo i Dong U. Ahn. Effects of Dietary Cholesterol and its Oxidation Products on Pathological Lesions and Cholesterol and Lipid Oxidation in the Rabbit Liver. Ames (Iowa): Iowa State University, styczeń 2015. http://dx.doi.org/10.31274/ans_air-180814-1349.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
6

Wood, W. G. Mechanisms of Alcohol Induced Effects on Cellular Cholesterol Dynamics. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2001. http://dx.doi.org/10.21236/ada398121.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
7

Wood, W. G. Mechanisms of Alcohol Induced Effects on Cellular Cholesterol Dynamics. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2004. http://dx.doi.org/10.21236/ada431885.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
8

Surls, Jacqueline D. Regulation of CD4+ T-Cell Function by Membrane Cholesterol. Fort Belvoir, VA: Defense Technical Information Center, luty 2012. http://dx.doi.org/10.21236/ad1013280.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
9

Momchilova, Albena, Galya Staneva, Rumiana Tzoneva, Ralica Scrobanska, Georgi Georgiev, Mariana Hadzhilazova, Liliana Maslenkova i Roumen Pankov. Resveratrol Affects Sphingomyelin and Cholesterol in Three-dimensional Fibroblast Cultures. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, kwiecień 2019. http://dx.doi.org/10.7546/crabs.2019.04.07.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
10

Santhosh, Poornima, Julia Genova, Ales Iglič, Veronika Kralj-Iglič i Nataša Poklar Ulrih. Influence of Cholesterol on Bilayer Fluidity and Size Distribution of Liposomes. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, lipiec 2020. http://dx.doi.org/10.7546/crabs.2020.07.07.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
Możesz być także zainteresowany rozszerzonymi bibliografiami na temat „Cholesterol” dla poszczególnych typów źródeł:
Artykuły w czasopismach Rozprawy doktorskie Książki
Oferujemy zniżki na wszystkie plany premium dla autorów, których prace zostały uwzględnione w tematycznych zestawieniach literatury. Skontaktuj się z nami, aby uzyskać unikalny kod promocyjny!

Do bibliografii