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Artykuły w czasopismach na temat "China. Cai zheng bu"
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Sun, Shingwun, i Theodore Park. "Eunuchs: Angels or Devils in Disguise?" SHS Web of Conferences 174 (2023): 03028. http://dx.doi.org/10.1051/shsconf/202317403028.
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The contradictory role of eunuchs in ancient China has remained a long-term debate in history. Often viewed as either holy or profane, eunuchs occupied a unique space in society that defied easy categorization. This study challenges the prevailing negative perception of eunuchs in ancient dynastical China by re-examining historical records and highlighting their positive contributions to society. The historical portrayal of eunuchs as wicked and cunning individuals has been heavily influenced by Chinese literati who sought to defame their rivals in order to gain political influence. Eunuchs, who played crucial roles in the imperial court, have had a significant impact on China’s development both positively and negatively. By focusing on notable eunuchs such as Zheng He, Cai Lun, and Sima Qian, this paper demonstrates that their contributions to Chinese culture, technological advancement, and historical documentation significantly outweigh the harm caused by a few power-hungry individuals. Ultimately, the paper calls for a more nuanced understanding of eunuchs’ roles in ancient Chinese society and their impact on the country’s development.
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YAMADA, KAZUTAKA, TOMOHIDE YASUNAGA i TOSHIHIDE ICHIKAWA. "A new species of Lyctocoridae (Hemiptera: Heteroptera: Cimicoidea) feeding on the exuded sap of Sawtooth Oak, Quercus acutissima, in Japan". Zootaxa 3525, nr 1 (24.10.2012): 65. http://dx.doi.org/10.11646/zootaxa.3525.1.5.
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A new species of the family Lyctocoridae, Lyctocoris ichikawai Yamada & Yasunaga sp. nov., is described from Shikoku and Kyushu, southwestern Japan. The species was found to inhabit near the sap-exuding parts on the trunk of Sawtooth Oak, Quercus acutissima Carruth. (Fagaceae). Lyctocoris ichikawai is considered to be most closely related to L. zhangi Bu & Zheng, 2001 from continental China and L. variegatus Péricart, 1969 from the Caucasus. The unique biology of the new species, including its habitat use, feeding activities, and phenology, is documented and discussed. A key is provided to distinguish among the three Japanese species of Lyctocoris.
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Lim, An-King. "On Sino-Turkic verbal functional expressions". International Journal of Chinese Linguistics 8, nr 1 (1.06.2021): 102–38. http://dx.doi.org/10.1075/ijchl.00012.lim.
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Abstract Turkic conquest and rule of China since 386 CE for nearly two hundred years had exerted its profound and long-lasting influence on many levels of Chinese society. Turkic sinification policy had induced the Xianbei National Language (XNL), which was Turkic language with selected set of Chinese characters for phonetic spelling. XNL, being spelt in Chinese characters, managed to function in Turkic-Chinese code-mixing in the bilingual communities as evidenced in bianwen 變文. Because of the Turkic politico-socio-economical dominance, some the Turkic elements in the code-mixing eventually gained prominance and have become permanent part of the northern vernacular, predecessor of modern Mandarin. This paper discusses twelve such Turkic-rooted verbal functional expressions: (1). The causative-passive qu 取; (2). Transitive passive sha 殺, sha 煞, si 死; (3). Causative dou 鬥, dou 逗; (4). Continuative hai 還, que 卻; (5). Resultative que 卻; (6). Reflexive nə 呢 (7). Positive indirective mo shi 莫是; (8). Negative indirective bu dao 不道; (9). Future participle cai 才, cai 纔; (10). Conditional yao shi 要是, yao 要, yao bu shi 要不是; (11). INDUCE-base nong 弄; (12). The speech quote verb dao 道.
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Meidong, Chen. "A Study of Some Astronomical Data in Muslim Calendar". International Astronomical Union Colloquium 91 (1987): 169–74. http://dx.doi.org/10.1017/s0252921100106001.
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The Muslim Calendar spread into China in 1385 where it was immediately translated into Chinese by the astronomer Yuan Tong and came into use. In 1477, it was further translated by the astronomer Bei Lin and compiled into the “Qi Zheng Tui Bu”, a work more or less the same in substance with the Muslim Calendar recorded in the “Ming Shi Li Zhi”, both being works of the same source. They left for us the valuable data of the results of research of ancient Arabian astronomers.On different occasions in the Muslim Calendar, values different with one another are used for the same kind of data. In that case, which of them are used for them are accurate values surveyed and calculated by people who originally worked out the Muslim Calendar? And how are these values calculated from data now available?
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Cai, Xiaobo, Min Cao, Huihui Guo, Qingliang Yang, Yongxiang Chen, Xiangfei Kong, Yong Du i in. "Abstract 1883: A MUC1 antibody-conjugated with a tubulysin B analog, DXC005, demonstrates excellent synergistic effect in combination with gemcitabine for treatment of pancreatic tumors". Cancer Research 84, nr 6_Supplement (22.03.2024): 1883. http://dx.doi.org/10.1158/1538-7445.am2024-1883.
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Abstract Pancreatic cancer is a malignant tumor with high incidence and mortality. It is difficult to diagnose and detect in the early stage, with low surgical resection rate and high recurrence and metastasis rate after surgery. At present, the clinical therapeutic strategy is extremely limited. The first-line Standard of Care (SOC) for unresectable pancreatic cancer is chemotherapy. The preferred regimen includes gemcitabine combined with albumin paclitaxel or FOLFIRINOX (5-FU+Oxaliplatin+Irinotecan). Due to the limited long-term benefits and toxic side effects of chemotherapy, targeted therapy combined with chemotherapy has become a new therapeutic strategy. MUC1 is a highly glycosylated transmembrane mucin located on the lumen surface of epithelial cells. It can protect cells from extreme factors and plays an important role in tumor cell metabolism, apoptosis, epithelial-mesenchymal transition (EMT) and metastasis. Previous studies have confirmed that MUC1 is highly expressed in a variety of tumors, including pancreatic cancer, and is closely related to poor prognosis. DXC005 is a novel MUC1-targeting antibody-drug conjugate (ADC), generated by conjugating a Tubulysin B analogue to a recombinant humanized anti-Muc1 monoclonal antibody. DXC005 is the first MUC1-ADC IND in China, and is currently in phase I clinical trials. Preclinical studies have confirmed the efficacy of DXC005 monotherapy (2.5 mg/kg, 5 mg/kg, 10 mg/kg in one administration) in the HuPrime ® pancreatic cancer PDX model (PA1194). The tumor growth inhibition (TGI) was 42.53 %, 70.77 %, and 95.58 %, respectively. We want to further clarify whether DXC005 combined with chemotherapeutic drug Gemcitabine can ensure or even improve the efficacy while reducing the dosage of Gemcitabine. In PA1194 xenograft model, DXC005 (3 mg/kg or 6 mg/kg) in combination with Gemcitabine (10 mg/kg) showed significant anti-tumor efficacy with 58.77% and 93.17% TGIs, respectively. In contrast, the treatment with 10 mg/kg of Gemcitabine alone exhibited much less TGI. Furthermore, complete response (CR) was observed in some animals after treatment with DXC005 (6 mg/kg) plus Gemcitabine (10 mg/kg). All groups of treatment are tolerated well, no abnormal animal behavior and body weight loss were observed in the study. The above results concluded that DXC005 combined with Gemcitabine can achieve synergistic effect even with reduced dose of Gemcitabine, which will serve as a support for synergistic application in clinical studies. Citation Format: Xiaobo Cai, Min Cao, Huihui Guo, Qingliang Yang, Yongxiang Chen, Xiangfei Kong, Yong Du, Zhicang Ye, Zhixiang Guo, Lingli Zhang, Lu Bai, Junxiang Jia, Yunxia Zheng, Wei Zheng, Jun Zheng, Wenjun Li, Yuanyuan Huang, Zhongliang Fan, Binbin Chen, Yanlei Yang, Meng Dai, Robert Y. Zhao. A MUC1 antibody-conjugated with a tubulysin B analog, DXC005, demonstrates excellent synergistic effect in combination with gemcitabine for treatment of pancreatic tumors [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2024; Part 1 (Regular Abstracts); 2024 Apr 5-10; San Diego, CA. Philadelphia (PA): AACR; Cancer Res 2024;84(6_Suppl):Abstract nr 1883.
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Suk Choo Chang. "Abstracts and Reviews : Nihon To Chu-Koku Ni Okeru Seishin-Bunretsu- Eyo No Mo-So Shudai (a Comparison of Schizo Phrenic Delusions in Japan and China) by Fuji- Mori Hideyuki, Zhan Pei Zheng, Kizaki Yoshio, Zheng-Ji Cai. Sishin Igaku (Psychological Medicine): Tokyo, 30 (1988):517-527". Transcultural Psychiatric Research Review 26, nr 3 (wrzesień 1989): 229–33. http://dx.doi.org/10.1177/136346158902600311.
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Rahman, Mohammad L., Xiao-Ou Shu, Douglas Walker, Dean P. Jones, Wei Hu, Bu-tian Ji, Batel Blechter i in. "Abstract 6056: A nested case-control study of untargeted plasma metabolomics and lung cancer risk among never-smoking women in the prospective Shanghai Women’s Health Study". Cancer Research 83, nr 7_Supplement (4.04.2023): 6056. http://dx.doi.org/10.1158/1538-7445.am2023-6056.
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Abstract Background: The etiology of lung cancer among never-smokers is unclear despite 15% of cases in men and 53% in women worldwide are not smoking-related. Metabolomics provides a snapshot of dynamic biochemical activities, including those found to be driving tumor formation and progression. This study used untargeted metabolomics with network analysis to agnostically identify network modules and independent metabolites in pre-diagnostic blood samples among never-smokers to further understand the pathogenesis of lung cancer. Methods: Within the prospective Shanghai Women’s Health Study, we conducted a nested case-control study of 395 never-smoking incident lung cancer cases and 395 never-smoking controls matched on age. We performed liquid chromatography high-resolution mass spectrometry to quantify 20,348 unique metabolic features in plasma. Because metabolic features are expected to be highly correlated and more likely to be involved in biological processes as a network of intertwined features than individually, we agnostically constructed 28 network modules using a weighted correlation network analysis approach. The associations between metabolite network modules and individual metabolites with lung cancer were assessed using conditional logistic regression models, adjusting for age, body mass index, and exposure to environmental tobacco smoke. We accounted for multiple testing using a false discovery rate (FDR) < 0.20. Results: We identified a network module of 122 metabolic features enriched in lysophosphatidylethanolamines that was associated with all lung cancer combined (p = 0.001, FDR = 0.028) and lung adenocarcinoma (p = 0.002, FDR = 0.056) and another network module of 440 metabolic features that was associated with lung adenocarcinoma (p = 0.014, FDR = 0.196). Metabolic features were enriched in pathways associated with cell growth and proliferation, including oxidative stress, bile acid biosynthesis, and metabolism of nucleic acids, carbohydrates, and amino acids, including 1-carbon compounds. Conclusions: Our prospective study suggests that untargeted plasma metabolomics in pre-diagnostic samples could provide new insights into the etiology of lung cancer in never-smokers. Replication and further characterization of these associations are warranted. Citation Format: Mohammad L. Rahman, Xiao-Ou Shu, Douglas Walker, Dean P. Jones, Wei Hu, Bu-tian Ji, Batel Blechter, Jason YY Wong, Qiuyin Cai, Gong Yang, Tu-Tang Gao, Wei Zheng, Nathaniel Rothman, Qing Lan. A nested case-control study of untargeted plasma metabolomics and lung cancer risk among never-smoking women in the prospective Shanghai Women’s Health Study [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2023; Part 1 (Regular and Invited Abstracts); 2023 Apr 14-19; Orlando, FL. Philadelphia (PA): AACR; Cancer Res 2023;83(7_Suppl):Abstract nr 6056.
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Rahman, Mohammad L., Charles E. Breeze, Xiao-Ou Shu, Jason YY Wong, Andres Cardenas, Xuting Wang, Bu-Tian Ji i in. "Abstract 3483: Epigenome-wide association study of lung cancer among never-smokers in two prospective cohorts in Shanghai". Cancer Research 83, nr 7_Supplement (4.04.2023): 3483. http://dx.doi.org/10.1158/1538-7445.am2023-3483.
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Abstract Background: The etiology of lung cancer among never-smokers has not been adequately elucidated despite that globally15% of lung cancer cases in men and 53% in women are not smoking-related. Epigenetic modifications, including changes in DNA methylation (DNAm), have been suggested as possible underlying mechanisms. However, only a few prospective epigenome-wide association studies (EWAS) of lung cancer incidence have been conducted, all exclusively focused on DNAm in peripheral blood cells and included a minimal number of never-smokers. We aimed to investigate genome-wide DNAm associations and epigenetic age acceleration with future risk of lung cancer among never-smokers using pre-diagnostic oral rinse samples. Methods: We conducted a case-control study of 80 never-smoking incident lung cancer cases and 83 comparable never-smoking controls nested in two large prospective cohorts: the Shanghai Women’s Health Study and Shanghai Men’s Health Study. DNAm was measured using the Illumina EPIC array. The top 50 differentially methylated positions (DMPs) were identified from a discovery sample and tested for replication in a validation sample using robust linear regression models. We also conducted an EWAS in the pooled sample. We examined functional overlap enrichment across chromatin states and histone mark broadPeaks for the top 1000 DMPs using eFORGE and constructed enrichment biological pathways analyses. Results: Across discovery and pooled EWAS, we identified four DMPs associated with lung cancer at the epigenome-wide significance level of P<8.0x10-8 (cg01411366: SLC9A10, P=7.23x10-08; cg00811020: NAA30, P=3.95x10-08; cg05658193: EIF2A, SERP1, P=5.02x10-08; and cg09198866: unannotated, P=5.39x10-09). The top 1000 DMPs were significantly enriched in epithelial regulatory regions and were associated with small GTPase-mediated signal transduction pathways. Furthermore, epigenetic age acceleration, measured by the GrimAge clock was prospectively associated with an increased risk of lung cancer (OR=1.19 per year of acceleration; 95% CI: 1.06-1.34) in logistic regression models. Conclusions: To our knowledge, this is the first prospective EWAS of lung cancer among never-smokers using oral rinse samples. Our results show that DNAm in pre-diagnostic oral rinse samples can provide new insights into lung cancer etiology and risk factors. Citation Format: Mohammad L. Rahman, Charles E. Breeze, Xiao-Ou Shu, Jason YY Wong, Andres Cardenas, Xuting Wang, Bu-Tian Ji, Wei Hu, Batel Blechter, Qiuyin Cai, H Dean Hosgood, Gong Yang, Jianxin Shi, Jirong Long, Yu-Tang Gao, Douglas Bell, Wei Zheng, Qing Lan, Nathaniel Rothman. Epigenome-wide association study of lung cancer among never-smokers in two prospective cohorts in Shanghai [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2023; Part 1 (Regular and Invited Abstracts); 2023 Apr 14-19; Orlando, FL. Philadelphia (PA): AACR; Cancer Res 2023;83(7_Suppl):Abstract nr 3483.
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Blechter, Batel, Parichoy Pal Choudhury, Xiao-ou Shu, Wei Zheng, Qiuyin Cai, Gong Yang, Jason Y. Y. Wong i in. "Abstract 2254: Risk models for lung cancer in never-smoking women in Shanghai with implications for screening". Cancer Research 82, nr 12_Supplement (15.06.2022): 2254. http://dx.doi.org/10.1158/1538-7445.am2022-2254.
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Abstract Background: The majority of female lung cancer cases in Asia are never-smokers with distinct risk factor profiles. Given the high burden of disease in this population, there is an increasing need to improve the understanding of lung cancer. Current risk models for lung cancer focus on active smokers and individuals of European ancestry. Therefore, we developed statistical models by integrating genetic and environmental risk factors to estimate absolute and population attribute risk of lung cancer among never-smoking women in Asia. Methods: We built absolute risk models for lung cancer among never-smoking women using data from 71,300 women (760 incident cases) in the Shanghai Women’s Health Study (SWHS), a population-based prospective cohort study. Relative risks were estimated using a multivariable Cox regression model with questionnaire-based risk factors. To account for missing genetic data for some subjects, we simulated genotypes for 10 common single nucleotide polymorphisms (SNP) using information on minor allele frequencies (MAF) and odds ratio estimates from previous genome-wide association studies (GWAS), conditional on family history of lung cancer. We used the iCARE tool to build two models for predicting lifetime (40 years) and 6-year absolute risk of lung cancer using age-specific lung cancer incidence rates, age-specific competing mortality rates, and risk factor distribution with: 1) questionnaire-based risk factors only and 2) questionnaire and genetic data. We then used the full absolute risk model to estimate the population attributable risk (PAR) due to modifiable risk factors, namely coal use and exposure to environmental tobacco smoke (ETS). Results: The questionnaire-based only model included family history of lung cancer, coal use, exposure to ETS, and body mass index (BMI). The full model also included data on 10 lung cancer related SNPs from our previous GWAS and had a wider spread in distribution of absolute lifetime risk (median=2.41%; range=0.43-12.36) compared to the questionnaire-based only model (median=2.72%; range=1.93-4.87). We used the full model to estimate the PAR and found that 1.74% and 6.33% of lung cancer cases could be prevented if never-smoking women in Shanghai did not use coal and were not exposed to ETS, respectively. Furthermore, we found that the full model estimated that 2.5% of the study population had a 6-year absolute risk of lung cancer higher than 1.51%, which is the suggested risk threshold for screening by existing risk models. Conclusion: We built risk models for never-smoking Asian women and estimated the contribution of coal use and ETS to the burden of lung cancer in Shanghai. This initial work shows promise for expanding and validating risk models in this population with potential translational implications, such as providing insight to identifying high risk individuals that may be eligible for lung cancer screening and primary prevention efforts. Citation Format: Batel Blechter, Parichoy Pal Choudhury, Xiao-ou Shu, Wei Zheng, Qiuyin Cai, Gong Yang, Jason Y.Y. Wong, Bu-Tian Ji, Wei Hu, Anne Rositch, Nilanjan Chatterjee, Nathaniel Rothman, Qing Lan. Risk models for lung cancer in never-smoking women in Shanghai with implications for screening [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2022; 2022 Apr 8-13. Philadelphia (PA): AACR; Cancer Res 2022;82(12_Suppl):Abstract nr 2254.
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Perez-Sancristobal, I., L. León, M. P. Álvarez Hernandez, A. Madrid García, L. Lopez Pedraza, J. I. Colomer, S. Lerma Lara i in. "AB1171 PERSISTENT POST-DISCHARGE SYMPTOMS AFTER COVID-19 IN RHEUMATIC AND MUSCULOSKELETAL DISEASES". Annals of the Rheumatic Diseases 81, Suppl 1 (23.05.2022): 1701.1–1701. http://dx.doi.org/10.1136/annrheumdis-2022-eular.4699.
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BackgroundThe COVID-19 pandemic continues worldwide and has had a strong impact on public health. As the pandemic evolves, efforts have been intensified to identify persistent symptoms associated with the infection once resolved have intensified.ObjectivesWe aimed to describe persistent symptoms and sequelae in patients with rheumatic and musculoskeletal diseases (RMD) after admission due to Covid-19. We also compared the role of autoimmune rheumatic diseases (ARD) with that of non–autoimmune rheumatic and musculoskeletal diseases (NARD) in persistent symptoms and sequelae.MethodsWe performed an observational study of patients with RMD who attended a rheumatology outpatient clinic in Madrid and required admission to hospital due to Covid-19 (1st March-30th May 2020) and survived. The study began at discharge and ran until 1st October 2020. The main outcomes were persistence of symptoms and sequelae related to Covid19. The independent variable was the RMD group (ARD and NARD). The covariates were sociodemographic data, clinical findings, and treatment. We ran a multivariate logistic regression model to assess the risk of the main outcomes by RMD group.ResultsWe included 105 patients, of whom 51.5% had ARD and 68.57% reported at least 1 persistent symptom. The most frequent were dyspnea, fatigue, and chest pain. Sequelae were recorded in 31 patients. These included lung damage in 10.4% of patients, lymphopenia in 10%, central retinal vein occlusion (1 patient), and optic neuritis (1 patient). Two patients died. Eleven patients required readmission owing to Covid-19 problems (16.7% ARD vs 3.9% NARD; p=0.053). No statistically significant differences were found between RMD groups in the final models.ConclusionMany RMD patients have persistent symptoms, as in other populations. Lung damage is the most frequent sequela. Compared to NARD patients, ARD patients do not seem to differ in terms of persistent symptoms or sequelae, although ARD patients might generate more readmissions due to Covid-19.References[1]Zheng Z, Peng F, Xu B, Zhao J, Liu H, Peng J, et al. Risk factors of critical & mortal COVID-19 cases: A systematic literature review and meta-analysis. J Infect. 2020;81(2):e16–25[2]Wang D, Hu B, Hu C, Zhu F, Liu X, Zhang J, et al. Clinical Characteristics of 138 Hospitalized Patients with 2019 Novel Coronavirus-Infected Pneumonia in Wuhan, China. JAMA -J Am Med Assoc. 2020;323(11):1061–9[3]Jiang F, Deng L, Zhang L, Cai Y, Cheung CW, Xia Z. Review of the Clinical Characteristics of Coronavirus Disease 2019 (COVID-19). J Gen Intern Med. 2020;35(5):1545–9.[4]Sung HK, Kim JY, Heo J, Seo H, Jang Y, Kim H, et al. Clinical Course and Outcomes of 3, 060 Patients with Coronavirus Disease 2019 in Korea, January – May 2020. 2020;35(30):1–11.[5]Freites Nuñez DD, Leon L, Mucientes A, Rodriguez-Rodriguez L, Font Urgelles J, Madrid García A, et al. Risk factors for hospital admissions related to COVID-19 in patients with autoimmune inflammatory rheumatic diseases. Ann Rheum Dis. 2020;79(11):1393–9.Disclosure of InterestsNone declared
Rozprawy doktorskie na temat "China. Cai zheng bu"
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"上海癌症自助組織硏究: 組員參與、社會支持和社會學習的增權效果". 2001. http://library.cuhk.edu.hk/record=b6073818.
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張時飛.論文(哲學博士)--香港中文大學, 2001.
參考文獻 (p. 338-366)
中英文摘要.
Available also through the Internet via Dissertations & theses @ Chinese University of Hong Kong.
Electronic reproduction. Hong Kong : Chinese University of Hong Kong, [2012] System requirements: Adobe Acrobat Reader. Available via World Wide Web.
Electronic reproduction. Ann Arbor, MI : ProQuest Information and Learning Company, [200-] System requirements: Adobe Acrobat Reader. Available via World Wide Web.
Mode of access: World Wide Web.
Zhang Shifei.
Lun wen (Zhe xue bo shi)--Xianggang Zhong wen da xue, 2001.
Can kao wen xian (p. 338-366)
Zhong Ying wen zhai yao.
Książki na temat "China. Cai zheng bu"
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Mao, Lianquan. Xuan an bu xuan: Mao Pijiang zhu "Hong lou meng" qi shi san zheng. Beijing Shi: Huaxia chu ban she, 2016.
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Cândida da Silva Antunes Pires. Aomen zhong cai gong zheng: Nei bu zi yuan zhong cai : liu yue shi yi ri di 29/96/M hao fa ling zhu shi = Justiça arbitral em Macau : a arbitragem voluntária interna : anotações ao Decreto-Lei no. 29/96/M, de 11 de Junho. [Aomen]: Fa lü ji si fa pei xun zhong xin, 2012.
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Duxide. Tang dai cai zheng. Shanghai: Zhong xi shu ju, 2014.
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Jia, Dehuai. Minguo cai zheng jian shi. [Beijing: Beijing zhong xian tuo fang ke ji fa zhan you xian gong si, 2012.
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Bei'an Shi cai zheng ju zhi bian zuan wei yuan hui, red. Bei'an Shi cai zheng ju zhi. [Bei'an Shi: Bei'an Shi cai zheng ju zhi bian zuan wei yuan hui], 2009.
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Ye, Yuanlong. Zhongguo cai zheng wen ti. [Beijing: Beijing zhong xian tuo fang ke ji fa zhan you xian gong si, 2012.
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Mianchi Xian (China). Cai zheng ju. Mianchi Xian cai zheng zhi: Nei bu zi liao. Sanmenxia Shi: Mianchi Xian cai zheng ju cai zheng zhi bian xie zu, 1986.
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Quan guo tu shu guan wen xian suo wei fu zhi zhong xin (China). Min zheng bu zou zhe hui cun. Beijing: Quan guo tu shu guan wen xian suo wei fu zhi zhong xin, 2004.
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Tang, Xiaogang. Fei chang shi qi zhi cai zheng. [Beijing: Beijing zhong xian tuo fang ke ji fa zhan you xian gong si, 2012.
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Xun, Lu. Cai wei ben yue: "A Q zheng zhuan" san bu qu. Xinbei Shi: Pu tian chu ban she, 2015.
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