Rozprawy doktorskie na temat „Childhood smoking”
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Woodward, Alistair. "Passive smoking and acute respiratory illness in childhood". Title page, contents and summary only, 1988. http://web4.library.adelaide.edu.au/theses/09PH/09phw899.pdf.
Pełny tekst źródłaLee, So-lun. "Foetal exposure to passive maternal smoking and childhood asthma". View the Table of Contents & Abstract, 2006. http://sunzi.lib.hku.hk/hkuto/record/B36887468.
Pełny tekst źródłaLee, So-lun, i 李素輪. "Foetal exposure to passive maternal smoking and childhood asthma". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B39724840.
Pełny tekst źródłaWANG, Calvin, i c. wang@ecu edu au. "EXPLORING YOUNG PEOPLE'S CONCEPTS OF SMOKING ADDICTION: PERCEIVED OPPORTUNITIES TO TRY SMOKING WITHOUT BECOMING ADDICTED". Edith Cowan University. Business And Law: School Of, 2006. http://adt.ecu.edu.au/adt-public/adt-ECU2006.0033.html.
Pełny tekst źródłaWang, Calvin. "Exploring young people's concepts of smoking addiction: Perceived opportunities to try smoking without becoming addicted". Thesis, Edith Cowan University, Research Online, Perth, Western Australia, 2006. https://ro.ecu.edu.au/theses/102.
Pełny tekst źródłaKauffman, Ross M. "Smoking and Tobacco in Ohio Prisons". The Ohio State University, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=osu1243363292.
Pełny tekst źródłaJaakkola, Niina. "Passive smoking during pregnancy and early childhood : occurrence, determinants, health effects and prevention". Helsinki : University of Helsinki, 2002. http://ethesis.helsinki.fi/julkaisut/laa/kansa/vk/jaakkola/.
Pełny tekst źródłaStrine, Tara Wynn. "The mediating role of psychological distress in the relationship between adverse childhood experiences and adult smoking". ScholarWorks, 2010. https://scholarworks.waldenu.edu/dissertations/791.
Pełny tekst źródłaPugmire, Juliana. "Health Effects of Childhood Exposure to Environmental Tobacco Smoke in Children followed to Adulthood". Diss., The University of Arizona, 2011. http://hdl.handle.net/10150/202985.
Pełny tekst źródłaYahaya, Ismail. "Childhood Sexual Abuse Against Girls in Sub-Saharan Africa : Individual and Contextual Risk Factors". Doctoral thesis, Mittuniversitetet, Avdelningen för hälsovetenskap, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:miun:diva-21919.
Pełny tekst źródłaHernandez, Denise Marie. "Immunotoxicological Evaluation Of Critical Windows Of Development Following Exposure to 1,2:5,6 Dibenzanthracene in B6C3F1 Mice". VCU Scholars Compass, 2006. http://hdl.handle.net/10156/1605.
Pełny tekst źródłaBaxter, Richard Turner. "Using Digital Microscopy to Evaluate Enamel Defects in Young Children: A Novel Method". The Ohio State University, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=osu1403188806.
Pełny tekst źródłaBonaventure, Audrey. "Facteurs de risque des leucémies aiguës de l’enfant et interactions gènes-environnement". Thesis, Paris 11, 2014. http://www.theses.fr/2014PA11T008.
Pełny tekst źródłaThe aim of this thesis was to analyze the associations between several environmental (maternal consumption of tobacco, alcohol or caffeinated drinks during pregnancy) and medical (history of asthma or eczema) factors and childhood acute leukemia, and to study genetic polymorphisms suspected to modify those associations.The analyses were performed using data from the national population-based case-control ESCALE study conducted in 2003 and 2004. Information about medical history and maternal consumptions during pregnancy was obtained through a standardized telephone interview with the mothers. The genetic polymorphisms were selected using a candidate approach based on their functionality, in genes involved in the metabolism of tobacco (CYP1A1*2, CYP2E1*5, NQO1*2, EPHX1 and NAT2*5), alcohol (CYP2E1*5, ADH1C*2) or caffeine (NAT2*5), and in allergy (IL4, IL4R, IL10 and IL13). Biological samples consisting of blood for cases and saliva for controls allowed for the genotyping of 370,000 SNPs in the cases and 4,500 SNPs in the controls. Where the candidate polymorphisms were not available from the genotyping, genotypic imputation was used to infer those. In total, data was available for 493 acute leukemia cases and 442 controls of European origin. Maternal coffee drinking during pregnancy and, to a lesser extent, cola soda drinking, was positively associated with childhood leukemia in the ESCALE study. No significant association was observed with maternal smoking or alcohol consumption during pregnancy. Carrying two NAT2*5 alleles was associated with acute lymphoblastic leukemia (Odds Ratio OR=1.9 [1.3-2.7]), although the analyses showed no association with the other candidate alleles involved in metabolism. There was no significant interaction between the candidate genetic polymorphisms and maternal consumptions of tobacco, alcohol or caffeinated drinks during pregnancy. However, the candidate alleles of CYP2E1, NQO1 and EPHX1, three enzymes involved in benzene metabolism, seemed to interact together.The variant alleles in IL13, IL4, IL10 and IL4R genes were not associated with childhood leukemia. A history of asthma or eczema was more frequently reported in controls than in cases (OR=0.7 [0.6-0.9]). This inverse association was mostly observed in children carrying a variant haplotype regulating the expression of IL10 (p for interaction=0.08), and carrying two reference alleles for IL13-rs20541 (p for interaction=0.06).As a conclusion, these results suggest a role of maternal coffee drinking during pregnancy in childhood leukemia that had already been reported in a previous French study of the same research team, and needing in-depth study and replication. However, no association was observed with maternal smoking or alcohol drinking, even after taking into account the candidate genetic polymorphisms. The gene-gene interaction of the three enzymes involved in benzene metabolism is interesting and needs to be investigated in other studies. Finally, the inverse association between childhood acute leukemia risk and medical history of asthma or eczema seems to be limited to the children with specific polymorphisms of interleukins IL10 and IL13, which could reflect underlying biological mechanisms. Those hypotheses should be further tested in other studies, such as the ESTELLE study, that has been recently conducted by the team
Woodward, Alistair J. "Passive smoking and acute respiratory illness in childhood / Alistair Woodward". Thesis, 1988. http://hdl.handle.net/2440/18671.
Pełny tekst źródłaJin, Cui. "The effects of passive smoking on respiratory illness in early childhood in Shanghai, P.R.China". Thesis, 1993. http://hdl.handle.net/1957/35873.
Pełny tekst źródłaGraduation date: 1993
Pryor, Laura E. "Developmental trajectories of body mass index in early childhood : an 8-year longitudinal study". Thèse, 2010. http://hdl.handle.net/1866/3917.
Pełny tekst źródłaDevelopmental Trajectories of Body Mass Index in Early Childhood: An 8-Year Longitudinal Study. Introduction: Childhood obesity has become one of the greatest Public Health challenges this century, affecting not only developed nations, but increasingly low- and middle-income countries as well. Estimating developmental trajectories of Body Mass Index (BMI) during early childhood represents an innovative approach towards a better understanding of the development of this health problem. Objective: To identify groups of children with distinct developmental trajectories of Body Mass Index (BMI) between the ages of five months and eight years, and to identify early-life risk factors that distinguish children in an atypically elevated BMI trajectory group. Methods: Group-based developmental trajectories of BMI were estimated from annual maternal assessments (5 months to 8 years) in a large population sample (n=1957). Measures of height and weight, as well as family and child characteristics were obtained yearly from mothers. Multivariate logistic regression was used to distinguish children with elevated BMI from other children, using pre and early post-natal risk factors. Results: Three trajectories of BMI were identified: low-stable BMI (54.5%), moderate BMI (41.0%) and high-rising BMI (4.5%). The high-rising group included children whose BMI, at eight years of age, exceeded the cut-off value for obesity. Multinomial logit regression analyses revealed that two maternal risk factors were significantly associated with the high-rising BMI trajectory group as compared to both the low and moderate groups: smoking during pregnancy and maternal overweight. Conclusions: Antecedents of childhood obesity can be identified during pregnancy. Intervention studies are needed in order to test the possibility that targeting maternal smoking and maternal obesity during pregnancy would reduce the risk of childhood obesity in the offspring. Keywords: Body Mass Index (BMI), child obesity, Group-based developmental trajectories, early life predictors, population-based study, maternal smoking, maternal obesity.