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Artykuły w czasopismach na temat "Chikungunya"
Gómez Alba, Virgen, Maylen Chalas i Eduardo Michelen. "Epidermolisis por chikungunya por transmisión autóctona en República Dominicana. Caso atípico y severo en un recién nacido (RN)". Ciencia y Salud 4, nr 1 (3.03.2020): 71–78. http://dx.doi.org/10.22206/cysa.2020.v4i1.pp71-78.
Pełny tekst źródłaNAYAK, KAUSTUV, Vineet Jain, Manpreet Kaur, Naushad Khan, Ramesh Chandra Rai, Kritika Dixit, Rohit Sagar i in. "Human immunity to chikungunya infection". Journal of Immunology 204, nr 1_Supplement (1.05.2020): 249.3. http://dx.doi.org/10.4049/jimmunol.204.supp.249.3.
Pełny tekst źródłaBurnett, Mark W. "Chikungunya". Journal of Special Operations Medicine 14, nr 4 (2014): 129. http://dx.doi.org/10.55460/8h36-wo5p.
Pełny tekst źródłaRodriguez-Cintron, William. "Chikungunya". Annals of Internal Medicine 162, nr 7 (7.04.2015): 531. http://dx.doi.org/10.7326/l15-5076.
Pełny tekst źródłaAl-Araimi, Tariq, i Shikha Mittoo. "Chikungunya". Annals of Internal Medicine 162, nr 7 (7.04.2015): 531. http://dx.doi.org/10.7326/l15-5076-2.
Pełny tekst źródłaHamer, Davidson H., i Lin H. Chen. "Chikungunya". Annals of Internal Medicine 162, nr 7 (7.04.2015): 532. http://dx.doi.org/10.7326/l15-5076-3.
Pełny tekst źródłaHORWOOD, P. F., i P. BUCHY. "Chikungunya". Revue Scientifique et Technique de l'OIE 34, nr 2 (1.08.2015): 479–89. http://dx.doi.org/10.20506/rst.34.2.2373.
Pełny tekst źródłaAnadarajah, Cholan. "Chikungunya". London Student Journal of Medicine 1, nr 1 (15.06.2009): 66–67. http://dx.doi.org/10.4201/lsjm.gch.001.
Pełny tekst źródłaAmin, Md Robed, Md Mujibur Rahman i Quazi Tarikul Islam. "Chikungunya". Journal of Medicine 18, nr 2 (24.08.2017): 92–108. http://dx.doi.org/10.3329/jom.v18i2.33687.
Pełny tekst źródłaPowers, Ann M. "Chikungunya". Clinics in Laboratory Medicine 30, nr 1 (marzec 2010): 209–19. http://dx.doi.org/10.1016/j.cll.2009.10.003.
Pełny tekst źródłaRozprawy doktorskie na temat "Chikungunya"
Delogu, Ilenia. "Virus chikungunya et traitement antiviral". Thesis, Aix-Marseille 2, 2011. http://www.theses.fr/2011AIX20668/document.
Pełny tekst źródłaThe Alphavirus RNA viruses are enveloped with a diameter of 70 nm, icosahedral structure with symmetry of type T = 4 (Choi et al. 1991; Cheng et al. 1995; Garoff et al. 2004). These viruses, whose distribution is worldwide, can infect a wide variety of vertebrates (mammals, birds, fish). These viruses are arboviruses, is à_dire viruses transmitted by arthropods. In the case of Alphavirus, the vectorization is done by mosquitoes from several species.To date, 29 species of Alphavirus have been identified, including at least six are pathogenic for humans. In humans, some are responsible for Alphavirus encephalitis, arthritis, fever, rash and can be fatal (Thiruvengadam et al. 1965; Pialoux et al. 2006).The first was isolated Alphavirus Equine Encephalitis West (Weeve) in 1930 (Meyer et al. 1931). The encephalitis virus Eastern (VEEV) and virus Venezuelan equine encephalitis (VEEV) were isolated respectively in 1933 and 1938 (Gibbs EP. 1976; Beck et al. 1938; Kubes et al. 1939 ). Sindbis virus isolated in Egypt in 1952 (Taylor et al. 1955), was the first Alphavirus responsible for arthritis to be isolated. The demonstration of the existence of CHIKV in Tanzania will be 1952 (Robinson 1955) (Lumsden 1955). Then follow the discoveries of all other Alphavirus. The South Elephent Seal virus (SESV), identified in 2000 on the Australian island of Macquarie is now the last Alphavirus discovered. The phylogenetic strains of Chikungunya can identify different clades for strains of East African, Western or Asian, and phylogenetic analysis is very close O'Nyong-Nyong (Powers and al. 2000), The sequencing of different isolates of the epidemic of 2005, helped to highlight some of them a mutation in the envelope glycoprotein more specifically in E1 (Schuffenecker et al. 2006). This mutation causes the substitution of an arginine at position 226 instead of valine (A226V), is a key element in determining the choice of a new vector for the transmission or Aedes albopictus (which transmits on the island of La meeting) with respect to the vector Aedes aegypti (Tsetsarkin et al. 2007). This mutation was later also found in India in 2007. (Arankalle et al. 2007; Kumar et al. 2008; Santhosh et al. 2008).The classic presentation often begins with sudden onset of high fever (40°C) for 3 / 10 days intermittent chills (Deller and al.1967). Fever is, in some cases, bi-phasic, that is to say, it decreases during a day or two before rising sharply. It is usually followed by erythema, pain or stiffness of muscle pain and muscle aches (Ozden et al. 2007) especially those involving pain in the extremities (wrists, ankles and knuckles) (Robinson 1955; Jadhav et al. 1965; Thiruvengadam et al. 1965). Also headache, rash maculopapular itchy sometimes. The rash affecting the chest and face hands and feet, children were seen eruptions like bullous skin accompanied by a detachment (Talarmin et al. 2007). The evolution of the disease regresses gradually. There is no antiviral therapy effective against CHIKV. Treatment is essentially symptomatic and consists of non-analgesic salicylates, paracetamol and anti-inflammatory drugs. This work consists of two parts: one part on the phylogenetic study of CHIKV and one part of the study of antiviral molecules. [...]
Koga, Rosemary de Carvalho Rocha. "ASPECTOS CLÍNICOS E SOROLÓGICOS DE INDIVÍDUOS COM SINAIS E SINTOMAS DE FEBRE CHIKUNGUNYA". Pontifícia Universidade Católica de Goiás, 2017. http://tede2.pucgoias.edu.br:8080/handle/tede/3664.
Pełny tekst źródłaMade available in DSpace on 2017-04-27T14:37:47Z (GMT). No. of bitstreams: 1 ROSEMARY DE CARVALHO ROCHA KOGA.pdf: 1840064 bytes, checksum: 5be7272271d61789b6dc93e32af0b7a3 (MD5) Previous issue date: 2017-03-15
Introduction: Chikungunya fever (FCHIK) is a disease of abrupt onset, transmitted by arthropod mosquitoes intermediate hosts of the Chikungunya virus (CHIKV). The illness has a significant impact on the quality of life of the affected person. Since a disease causes intense and prolonged symptoms of polyarthralgia and myalgia, it requires health care, during a recovery, more than other arboviruses. The objective of this study was to study clinicians and clinicians suggestive of FCHIK, residing in the States of Amapá and Goiás, aiming to correlate the results of laboratory tests with the presented symptomatology. Materials and methods: The study was carried out at the Center for Immunological Studies and Research of the Pontifical Catholic University of Goiás, Goiânia, and in Emergency Care Units in the cities of Macapá, Oiapoque and Santana-AP. The study population consisted of 80 individuals with suspected FCHIK and for investigators of inflammatory markers, the control group consisted of 20 blood samples from healthy donors from Goiana Central de Serologia e Imunohematologia. Viral RNA extraction was performed, followed by RNA detection by Real-Time Polymerase Chain Reaction. In addition to ELISA for detection of IgM and IgG against Chikungunya virus. Participants symptoms were correlated with serology and Creactive protein (CRP), which was evaluated in healthy subjects and in people with FCHIK. Results: No data presented for detection of viral RNA by RT-qPCR for CHIKV, but three samples were positive in this technique for zika virus and one for dengue subtype 1 (DENV1). In an enzyme-linked immunosorbent assay, 26 samples were positive for IgG and 3 for IgM. Regarding the stage of the disease, 10 were in the acute phase, 04 in the subacute phase and 12 in the chronic phase. Correlated the results of the serology with a symptomatology it was observed that the acute phase, all have fever, 90% headache, 70% arthralgia and 60% edema. (100%), myalgia and edema (75%). (100%), arthralgia (92%) and myalgia (75%). When comparing participants with negative serology, n = 54, the most prevalent symptoms were rash, headache, fever, and arthralgia. The CRP levels in individuals infected with more than four symptoms were higher when compared with healthy individuals. Conclusion: The study focused on people with a clinical picture characteristic of FCHIK. The most common symptom in the three phases presented for arthralgia, followed by edema and myalgia, a fever was frequent only in the acute phase. All participants were negative in the evaluation of viral RNA by RT-qPCR for CHIKV, for the virus has a short duration in the body, and this methodology is limited to the time of symptom onset and sample collection, DENV and ZIKV. IG G. Those with negative serology for CHIKV, despite taking into account the joints, symptoms common to other arboviruses. CRP levels have been shown to be high relative to healthy subjects.
Introdução: A Febre Chikungunya (FCHIK) é uma doença de início abrupto, transmitida por mosquitos artrópodes hospedeiros intermediários do vírus Chikungunya (CHIKV). A enfermidade representa um significativo impacto na qualidade de vida da pessoa afetada. Uma vez que a doença causa sintomas intensos e prolongados de poliartralgia e mialgia, requerendo atenção de saúde, durante a recuperação, mais do que outras arboviroses. Objetivou-se estudar aspectos clínicos e sorológicos de indivíduos apresentando quadro clínico sugestivo de FCHIK, residentes nos Estados de Amapá e Goiás, visando correlacionar os resultados de testes laboratoriais com a sintomatologia apresentada. Materiais e métodos: O estudo foi realizado no Núcleo de Estudos e Pesquisa Imunológica da Pontifícia Universidade Católica de Goiás, em Goiânia, e em Unidades de Pronto Atendimento de Saúde das cidades de Macapá, Oiapoque e Santana-AP. A população de estudo foi constituída de 80 indivíduos com suspeita de FCHIK e para comparar os marcadores inflamatórios, o grupo controle foi constituído de 20 amostras de sangue de doadores saudáveis da Central Goiana de Sorologia e Imunohematologia. Foi realizada a extração do RNA viral, seguido de detecção do RNA por meio de Reação em Cadeia de Polimerase em Tempo Real. Além de ELISA para detecção de IgM e IgG específicos para o CHIKV. Os sintomas dos participantes foram correlacionados com o resultado da sorologia e da proteína C reativa (PCR), que foi avaliada em indivíduos saudáveis e em pessoas com FCHIK. Resultados: Nenhuma amostra apresentou limiar de detecção do RNA viral por RT-qPCR para CHIKV, porém três amostras foram positivas nessa técnica para vírus zika (ZIKV) e uma para dengue subtipo 1 (DENV1). Em ensaio imunoenzimático, 26 amostras foram positivas para IgG e 3 dessas para IgM. Em relação ao estágio da doença, 10 encontravam-se em fase aguda, 04 em fase subaguda e 12 em fase crônica. Correlacionados os resultados da sorologia com a sintomatologia observou-se que os de fase aguda, todos tiveram febre, 90% cefaleia, 70% artralgia e 60% edema. Enquanto que, os de fase subaguda tiveram: artralgia e cefaleia (100%), mialgia e edema (75%). Os de fase crônica tiveram edema (100%), artralgia (92%) e mialgia (75%). Quando comparados os participantes com sorologia negativa, n=54, os sintomas mais apresentados foram exantema, cefaleia, febre e artralgia. Os níveis de PCR nos indivíduos infectados e que apresentavam mais de quatro sintomas foram maiores quando comparados com indivíduos saudáveis. Conclusão: O estudo focou em pessoas com quadro clínico característico para FCHIK. O sintoma mais comum nas três fases apresentadas foi a artralgia, seguido de edema e mialgia, a febre foi frequente somente na fase aguda. Todos os participantes foram negativos na avaliação do RNA viral por RT-qPCR para CHIKV, pois o vírus tem uma curta duração no organismo, e esta metodologia é limitada ao tempo de início dos sintomas e coleta de amostra, ainda assim foi encontrado RNA viral do DENV e ZIKV. Alguns participantes foram positivos para sorologia IgG. Aqueles com sorologia negativa para CHIKV, apesar de terem dor nas articulações, tinham sintomas comuns a outras arboviroses. Os níveis de PCR demonstraram-se elevados em relação aos indivíduos saudáveis.
Hiroki, Carlos Hiroji. "Papel das Neutrophil Extracellular Traps no controle da infecção por Chikungunya". Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/17/17147/tde-01022019-105719/.
Pełny tekst źródłaChikungunya is a reemerging virus which causes a disease characterized by an incapacitanting arthralgia and affects thousands of people. Innate response against this virus is well described by participation of macrophages, dendritic cells and NK cells, however few works demonstrate the roe of neutrophils in this infection. Neutrophils Extracellular Traps are a web of DNA complexed with antimicrobial enzymes which were described for fighting against many pathogens. However, there are no works which demonstrate its relevance in Chikungunya infection. Our objective was to evaluate if there is release of NETs in Chikungunya infection, describe its mechanisms and demonstrate its relevance in vitro and in vivo. We observed that mouse and human neutrophils incubated with Chikungunya are able to produce NETs via Toll-Like 7 and reactive oxygen species production. These NETs were able to capture the virus and inhibit its infection in vitro. Moreover, animals infected with Chikungunya virus and treated with rhDNAse demonstrated higher viral load and are more susceptible to the infection, showing its importance in vivo. Lastly, patients diagnosed during acute infection of Chikungunya infection have high levels of NETs correlated with a high viral load.
MARQUES, Nuno Miguel da Silva. "Dengue e chikungunya: arboviroses emergentes em Angola". Doctoral thesis, Instituto de Higiene e Medicina Tropical, 2017. http://hdl.handle.net/10362/57140.
Pełny tekst źródłaIn recent decades, there has been a reduction in the number of malaria cases in many sub- Saharan African countries. However, the overdiagnosis of malaria in endemic areas is frequent. The clinical relevance of arboviruses in the differential diagnosis of malaria in Angola is unknown. Historically the main arbovirosis described in Angola has been Yellow Fever. However, there was evidence of circulation of other arboviruses such as the Chikungunya virus during the colonial period. After the country's independence, which occurred in 1975, sporadic dengue cases imported from Angola were registered in several countries. Thus, until 2012, the lack of knowledge about the prevalence of arboviruses, such as Dengue and Chikungunya, was a reality. An observational and cross - sectional study was carried out to identify the presence of Dengue virus and Chikungunya virus. Patients with febrile syndrome (body temperature at admission ≥37.5 ° C and / or history of fever) and clinically compatible with malaria were included. Rapid diagnostic tests (TDR) [SD BIOLINE®], which are immunochromatographic assays for the detection of: Ag NS1 and IgG / IgM antibodies against Dengue virus have been applied; Ig M against the Chikungunya virus; Ag HRPII- P.f and pLDH-P.v from Plasmodium spp. Biological samples were also collected for molecular biology techniques (PCR or RT-PCR) for Plasmodium spp., Dengue and Chikungunya. The study was conducted in two phases, the first from February to April 2012 in Huambo province and the second from May to June 2015 in Benguela province, with a total of 542 patients. In the first phase 242 patients were included, mostly female (59.9%). The average age was 16 years. The most common symptoms were respiratory such as cough (60.7%) and nasal discharge (48.3%) followed by painful complaints, headache (40.1%), abdominal pain (36.4%), arthralgia (33 , 9%) and myalgias (30.6%). The rates of positive TDR were as follows: malaria (2.1%), Chikungunya (1.7%) and Dengue (0.8%). There was a case of concomitant positivity for Dengue (Ag NS1 +) and Chikungunya. In the second phase 300 patients were included, also mostly female (61%). The average age was 19 years. The most frequent clinic complaints were headaches (53%), myalgias (45%), arthralgia (43.3%), abdominal pains (38.3%) followed by cough (26.3%) and runny nose (16.3%). The rates of positive TDR were as follows: malaria (36.7%), Chikungunya (18.3%) and Dengue fever (3.3%). There were 28 cases of concomitant positivity for malaria and Chikungunya and 4 cases for malaria and Dengue (1 with Ag NS1 + and 3 with IgM +). In this second phase a Chikungunya genomic sequence was also identified by RT-PCR which revealed a high identity with a circulating strain in Cameroon in 2006. This study was relevant for the recent investigation of arbovirus in Angola, documenting its circulation, as well as for allowing for the first time the large-scale application of TDR to Dengue and Chikungunya in this country.
Thiberville, Simon-Djamel. "Investigations épidémiologiques, cliniques et thérapeutiques du chikungunya". Thesis, Aix-Marseille, 2016. http://www.theses.fr/2016AIXM5016.
Pełny tekst źródłaChikungunya virus (CHIKV) is an arthropod-borne virus transmitted by Aedes mosquitoes that cause debilitating arthralgia and possible chronic rheumatism. In the first part we describe the clinical, biological and virological presentation of outpatients with chikungunya disease (CHIK) from the acute stage to the chronic stage at day 300, during the outbreak in the Reunion Island in 2006. We elaborated scores for CHIK diagnosis and we also analysed the intra-host genetic diversity.To complete our first results, we investigated a CHIKV outbreak, which occurred in the Republic of Congo in 2011. The clinical presentation was similar to the first description of the Reunion island outbreak. We assessed the clinical score which appeared to be unusable at the individual level but was still relevant to follow the epidemic curve. This work was completed by seroprevalence and phylogenetic analyses.The last study presented in this thesis focused on the use of chloroquine during the acute stage of CHIK in a non-human primate (NHP) model (prophylactic use) and during a clinical trial (therapeutic use). The main effect of chloroquine treatment at the acute stage of CHIK appeared to be related to its immuno-modulatory action; in prophylactic taking, chloroquine exacerbated acute symptoms while treatment during the early stages of the disease increased the risk of acquiring chronic arthralgia.In conclusion, we provide a detailed description of CHIK outpatients and identify risk factors for the chronic stage of the disease. We propose tentative diagnostic scores and we firmly establish that the use of chloroquine at the acute phase of CHIK is contraindicated
Enguehard, Margot. "Interaction between chikungunya and dengue viruses during co-infection in Aedes mosquito cells and in Aedes aegypti mosquito". Thesis, Lyon, 2017. http://www.theses.fr/2017LYSE1161/document.
Pełny tekst źródłaEmergence and geographical extension of dengue (DENV), Zika (ZIKV) and chikungunya (CHIKV) viruses increase simultaneous outbreak in an increasing number of countries. To date, no vaccine or cure have yet been developed against these diseases those cause a tremendous impact on human health and in the economy worldwide. During recent simultaneous outbreaks, up to 12% of patients have been diagnosed to be co-infected by CHIKV and DENV. In addition, it was shown that the mosquitoes Aedes albopictus could carry and transmit simultaneously CHIKV and DENV. However, the pathology, as well as the epidemiology of a pathogen, relies on the interactions between several infectious agents present within an organism or a community in the environment. It is crucial to consider to which extent a host infected by a first microorganism is modified and whether its reaction to the infection by a second microorganism is consequently altered. However, there is no extensive report of Alphavirus-Flavivirus or Flavivirus- Flavivirus interactions. Our global objective is to characterize these co-infections in both mosquitoes and humans, at the cell and molecular level. To this aim, we started this project by performing sequential co- infection in different cell lines from Aedes albopictus and Aedes aegypti. We found that the permissiveness and production of DENV is enhanced in presence of CHIKV. On the contrary, there is no effect of DENV pre-infection on subsequent CHIKV co-infection. We generalized the synergistic phenomena and we showed that CHIKV pre-infection also increased the infection by DENV-1, DENV-3 and DENV-4, but also by two others re-emerging Flaviviruses, the Yellow Fever Virus (YFV), and the Zika Virus (ZIKV). Remarkably, we succeeded to establish a mosquito model of co-infection of Aedes aegypti mosquito after by different two feedings at 4 days interval. Using this sequential co-infection, we were able to show that a pre-infection of Aedes aegypti by CHIKV increase the level of DENV-2 RNA in salivary glands compare to mono-infected mosquitos. This phenotype is reminiscent of the phenotype we observed in vitro during successive infections. Altogether, our study paves the way to the characterization of molecular interaction between Flaviviruses and Alphaviruses in mosquito in vitro and in vivo. This study can be crucial for a better understanding of disease and epidemiology during simultaneous outbreaks
VENTORIM, D. P. "DIVERSIDADE Genética de Chikungunya no Estado do Espírito Santo". Universidade Federal do Espírito Santo, 2018. http://repositorio.ufes.br/handle/10/7120.
Pełny tekst źródłaA febre chikungunya é uma arbovirose altamente debilitante, causada pelo vírus chikungunya, o qual é transmitido pela picada de mosquitos do gênero Aedes. Em 2014 foram registrados os primeiros casos da doença no Brasil, sendo constatada a presença dos genótipos asiático e Leste/Centro/Sul africano do vírus. No final de 2015, pela primeira vez, foram reportados casos no Espírito Santo (ES) e entre 2016-2017 o estado enfrentou um surto da doença. Diante disso, nós, juntamente à Secretaria Estadual de Saúde/ES e ao Laboratório Central/ES objetivamos identificar qual linhagem do vírus circula no ES; analisar características genéticas virais nas amostras estudadas e levantar dados epidemiológicos sobre a doença no estado. As amostras do estudo foram provenientes do Laboratório Central/ES e referentes ao período de março/2016 - dezembro/2017. O diagnóstico viral foi realizado por sorologia ou por técnicas moleculares. Vinte e sete amostras (diagnosticadas molecularmente) foram utilizadas na amplificação parcial e sequenciamento de dois genes codificantes de proteínas do envelope viral, E1 e E2. Seis dessas amostras foram utilizadas nas análises filogenéticas. Os resultados epidemiológicos demonstraram que no período do estudo foram reportados 2.021 casos suspeitos da febre chikungunya, sendo 412 (20,38%) confirmados. Além disso, a distribuição geográfica desses casos constatou que Vitória e Vila Velha representaram mais de 50% de todos os casos do estado. Os achados mostraram que a frequência da infecção pelo vírus chikungunya, em relação ao número de amostras referenciadas ao Laboratório Central/ES, pode ser considerada baixa. No entanto, constatou-se que a doença apresenta relevância epidemiológica e grande distribuição no estado. Os resultados filogenéticos evidenciaram que o vírus circulante pertence à linhagem Leste/Centro/Sul africana, a qual também foi constatada em diversos surtos na Europa, África e Ásia. Além disso, a caracterização molecular dos fragmentos das proteínas E1 e E2 não mostraram a presença das mutações adaptativas E1-K211E; E1-A226V; E2-L210Q e E2-I211T. Esse resultado permite sugerir que o vírus circulante no ES apresenta um potencial de disseminação menor em comparação aos vírus circulantes em grandes epidemias mundiais recentes. Devido à falta de uma vacina e à dificuldade no controle populacional do mosquito vetor, estudos sobre a diversidade genética como este tornam-se alternativas viáveis em busca de melhor entendimento e controle da febre chikungunya no Brasil e, especificamente, no ES.
Dagley, Ashley L. "Amelioration of Chikungunya through Inhibition of the Inflammatory Response". DigitalCommons@USU, 2016. https://digitalcommons.usu.edu/etd/4996.
Pełny tekst źródłaYapa, Badal Madiththegedara Chamini Randika Wimalasiri. "Chikungunya virus transmission dynamics and immune responses in mosquitoes". Thesis, Queensland University of Technology, 2020. https://eprints.qut.edu.au/206132/1/Badal%20Madiththegedara%20Chamini%20Randika%20Yapa%20Thesis.pdf.
Pełny tekst źródłaVega, Rua Anubis. "Émergence du virus chikungunya en Amérique et en Europe". Thesis, Paris 6, 2015. http://www.theses.fr/2015PA066197/document.
Pełny tekst źródłaChikungunya virus (CHIKV), transmitted mainly by the mosquitoes Aedes aegypti and Aedes albopictus, is a major public health problem. Since 2004, CHIKV epidemics have been reported in Africa, Asia, the Indian Ocean Islands, and Europe. Only the Americas seemed spared despite high densities of mosquitoes and multiple introductions of the virus to the continent by travelers returning from countries where CHIKV was circulating. We have assessed the risk of CHIKV emergence in the Americas by evaluating the vector competence of 35 local populations of Ae. aegypti and Ae. albopictus infected with different strains of CHIKV. These populations were shown to be susceptible to CHIKV infection, highlighting the predominant role of salivary glands as a "filter" of transmission. Genotyping of Ae. albopictus from the Americas using microsatellites allowed the identification of a genetic cluster of populations characterized by a low transmission of CHIKV strains of the East-Central-South-African genotype. In October 2013, Asian strains of CHIKV began circulating in the Caribbean. Thus, we evaluated the susceptibility of 11 populations of Ae. aegypti and Ae. albopictus to the Asian CHIKV genotype and showed that the two species were sufficiently competent to ensure dissemination of the virus throughout the continent. Furthermore, we showed that Ae. albopictus was likely to facilitate the spread of CHIKV to Europe. However, the vector competence of French Ae. albopictus to the Asian CHIKV genotype was negatively affected by temperatures lower than those usually found in tropical countries
Książki na temat "Chikungunya"
Heise, Mark, red. Chikungunya Virus. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-90610-8.
Pełny tekst źródłaChu, Justin Jang Hann, i Swee Kim Ang, red. Chikungunya Virus. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3618-2.
Pełny tekst źródłaOkeoma, Chioma M., red. Chikungunya Virus. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-42958-8.
Pełny tekst źródłaDileep, Mavalankar, i Indian Institute of Management, Ahmedabad., red. A preliminary estimate of immediate cost of chikungunya and dengue to Gujarat, India. Ahmedabad: Indian Institute of Management, 2009.
Znajdź pełny tekst źródłaDileep, Mavalankar, i Indian Institute of Management, Ahmedabad., red. Quantifying the impact of chikungunya and dengue on tourism revenues. Ahmedabad: Indian Institute of Management, 2009.
Znajdź pełny tekst źródłaChikungunya: La médiatisation d'une crise : presse, humour, communication publique. Cortil-Wodon: E.M.E., 2011.
Znajdź pełny tekst źródłaCentral Council for Research in Ayurveda and Siddha (India), red. Management of chikungunya through ayurveda and siddha: A technical report. New Delhi: Central Council for Research in Ayurveda and Siddha, Dept. of AYUSH, Ministry of Health & Family Welfare, Govt. of India, 2009.
Znajdź pełny tekst źródłaCentral Council for Research in Ayurveda and Siddha (India), red. Management of chikungunya through ayurveda and siddha: A technical report. New Delhi: Central Council for Research in Ayurveda and Siddha, Dept. of AYUSH, Ministry of Health & Family Welfare, Govt. of India, 2009.
Znajdź pełny tekst źródłaL' épidémie moderne et la culture du malheur: Petit traité du chikungunya. Paris: L'Harmattan, 2006.
Znajdź pełny tekst źródłamédecin, Aubry Pierre, red. Le chik, le choc, le chèque: L'épidémie de Chikungunya à La Réunion 2005-2006 en questions. Sainte-Marie [Réunion]: Azalées éditions, 2006.
Znajdź pełny tekst źródłaCzęści książek na temat "Chikungunya"
Arriaga, Pedro, i Jorge Hidalgo. "Chikungunya". W Evidence-Based Critical Care, 513–16. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-43341-7_59.
Pełny tekst źródłaMahendradas, Padmamalini. "Chikungunya". W Emerging Infectious Uveitis, 119–24. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-23416-8_12.
Pełny tekst źródłaGabrys, Beata, John L. Capinera, Jesusa C. Legaspi, Benjamin C. Legaspi, Lewis S. Long, John L. Capinera, Jamie Ellis i in. "Chikungunya". W Encyclopedia of Entomology, 841–44. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6359-6_625.
Pełny tekst źródłaFischer, Marcellus. "Chikungunya". W Reisedermatosen, 101–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-44705-5_22.
Pełny tekst źródłade Góes Cavalcanti, Luciano Pamplona, André Machado Siqueira, José Alfredo de Sousa Moreira i André Ricardo Ribas Freitas. "Chikungunya". W Infectious Tropical Diseases and One Health in Latin America, 19–36. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-99712-0_2.
Pełny tekst źródłaMahendradas, Padmamalini. "Chikungunya". W Intraocular Inflammation, 1247–51. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-540-75387-2_119.
Pełny tekst źródłaAbel, Sylvie, i André Cabié. "Clinical Syndrome and Therapy". W Chikungunya Virus, 1–17. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-42958-8_1.
Pełny tekst źródłaHawman, David W., i Thomas E. Morrison. "Chikungunya Virus Pathogenesis". W Chikungunya Virus, 161–83. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-42958-8_10.
Pełny tekst źródłaMahauad-Fernandez, Wadie D., i Chioma M. Okeoma. "Restriction Factors and Chikungunya Virus". W Chikungunya Virus, 185–97. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-42958-8_11.
Pełny tekst źródłaNg, Lisa F. P., i Pierre Roques. "Host Response and Mechanisms of Subversion of Chikungunya". W Chikungunya Virus, 19–32. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-42958-8_2.
Pełny tekst źródłaStreszczenia konferencji na temat "Chikungunya"
Higgs, Stephen. "Chikungunya and mosquito vectors". W 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.95071.
Pełny tekst źródłaBarud, Ana Rebecca Rodrigues, Fernando Maia Peixoto Filho i Paulo Roberto Nassar de Carvalho. "Infecção fetal pela chikungunya". W 44° Congresso da SGORJ - XXIII Trocando Ideias. Zeppelini Editorial e Comunicação, 2020. http://dx.doi.org/10.5327/jbg-0368-1416-2020130254.
Pełny tekst źródłaMOTA CARVALHO, NAYARA, CAROLINA PELLISON CARVALHO, WILLIAM BARROS HYPPOLITO FERREIRA, VANESSA FÉLIX NASCIMENTO COELHO, MARIA VERONICA RUSSO MACCHI, ANDRÉ MARUN LYRIO, RUBENS BONFIGLIOLI i in. "CHRONIC POLYARTHRITIS AFTER CHIKUNGUNYA FEVER". W SBR 2021 Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2021.2228.
Pełny tekst źródłaPereira, Maria Eduarda da Silva, José Carlos da Rocha Neto, Isa Cordeiro da Silva, Brenda Marques de Cerqueira, Maríllya Morais da Silva, Cibele Carine Silva i Solange Laurentino dos Santos. "COMPORTAMENTO EPIDEMIOLÓGICO DA FEBRE CHIKUNGUNYA EM TEMPOS DA PANDEMIA". W XXVII Semana de Biomedicina Inovação e Ciência. Editora IME, 2021. http://dx.doi.org/10.51161/9786588884119/48.
Pełny tekst źródłaAquino, Ana Alice de. "PERFIL EPIDEMIOLÓGICO DAS ARBOVIROSES URBANAS NO BRASIL NO ANO DE 2021". W II Congresso Nacional de Microbiologia Clínica On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/ii-conamic/13.
Pełny tekst źródłaHincapié-Palacio, Doracelly, i Juan Ospina. "Mathematical modeling of Chikungunya fever control". W SPIE Sensing Technology + Applications, redaktorzy Brian M. Cullum i Eric S. McLamore. SPIE, 2015. http://dx.doi.org/10.1117/12.2177066.
Pełny tekst źródłaCansian Tuão, Raiza, Paula dos Santos Athayde, Ketty Lysie Libardi Lira Machado, Estephania Pignaton Naseri, Bárbara Ferraço Dalmaso, Gustavo Pinto de Oliveira Gomes, Débora Marques Veghini i in. "AUTOIMMUNE INFLAMMATORY MYOPATHY AFTER CHIKUNGUNYA INFECTION". W SBR 2021 Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2021.2101.
Pełny tekst źródłaFreire de Carvalho, Jozélio, Darja Kanduc, Felipe Freire da Silva, Amir Tanay, Alberta Lucchese i Yehuda Shoenfeld. "SJÖGREN SYNDROME ASSOCIATED WITH CHIKUNGUNYA INFECTION". W Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.17526.
Pełny tekst źródła"Reemergence and Global Spread of Chikungunya". W 3rd International Conference on Biological, Chemical and Environmental Sciences. International Institute of Chemical, Biological & Environmental Engineering, 2015. http://dx.doi.org/10.15242/iicbe.c0915052.
Pełny tekst źródłaSilva, Victor Luiz Luciano da, URSULA RAIANNY LACERDA DA SILVA, AMANDA MARIA SANTANA DA COSTA, ITALO MATHEUS DA SILVA PEQUENO i FRANCISCO ISRAEL MAGALHÃES FEIJAO. "CHIKUNGUNYA NO BRASIL: UM ESTUDO OBSERVACIONAL TRANSVERSAL COMPARANDO AS 16 PRIMEIRAS SEMANAS EPIDEMIOLÓGICAS DE 2021 E 2022". W I Congresso Brasileiro de Estudos Epidemiológicos On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/epidemion/7622.
Pełny tekst źródłaRaporty organizacyjne na temat "Chikungunya"
Ramírez-Tamayo, Amparo Aurora, i Juan Luciano Olivella-Jacquín. Chicungnya, CHIKV, Chikungunya o Chicunguña. Universidad Cooperativa de Colombia, 2016. http://dx.doi.org/10.16925/greylit.1612.
Pełny tekst źródłaHayes, Curtis, i Thomas O'Rourke. Chikungunya Fever Among U.S. Peace Corps Volunteers - Republic of the Philippines. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 1986. http://dx.doi.org/10.21236/ada173506.
Pełny tekst źródłaChikungunya. Instituto Nacional de Salud, styczeń 2022. http://dx.doi.org/10.33610/infoeventos.2.
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