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Artykuły w czasopismach na temat „Central nervous system”

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Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 29 lipca 2024

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1

Adamson, D. Cory, B. Ahmed K. Rasheed, Roger E. McLendon i Darell D. Bigner. "Central nervous system". Cancer Biomarkers 9, nr 1-6 (26.10.2011): 193–210. http://dx.doi.org/10.3233/cbm-2011-0177.

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2

Desole, M. S., P. Enrico, M. Miele, L. Fresu, G. Esposito, G. De Natale i E. Miele. "Central nervous system". Pharmacological Research 25 (maj 1992): 19–20. http://dx.doi.org/10.1016/1043-6618(92)90265-d.

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3

Goldberg, Herbert I., i Robert A. Zimmerman. "Central nervous system". Seminars in Roentgenology 22, nr 3 (lipiec 1987): 205–12. http://dx.doi.org/10.1016/0037-198x(87)90034-4.

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4

Vimal, Shruti. "Histopathological Spectrum of Central Nervous System Tumours in a Tertiary Care Centre". Indian Journal of Pathology: Research and Practice 9, nr 2 (Part- I) (1.05.2020): 103–10. http://dx.doi.org/10.21088/ijprp.2278.148x.9220.18.

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5

Colman Ozuna, Víctor Manuel, Natalia María Antonella Rojas Almirón, Edgar Eugenio Ortega Portillo, Sandra María Soto Valiente, Vivian María Liz Pérez i Graciela Medina Insfran. "Vasculitis del sistema nervioso central". Revista del Instituto de Medicina Tropical 18, nr 1 (9.08.2023): 90–93. http://dx.doi.org/10.18004/imt/2023.18.1.12.

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La vasculitis primaria del Sistema Nervioso Central (VPSNC) se refiere a un grupo de enfermedades que resultan de la inflamación y destrucción de los vasos sanguíneos de la médula espinal, encéfalo y meninges, tanto en el sector venoso como arterial, esto puede conducir a la oclusión o formación de aneurismas, con las consiguientes alteraciones isquémico-hemorrágicas.1 La presentación es heterogénea y poco sistematizable. El diagnóstico se establece con un cuadro clínico compatible, una angiografía o biopsia del parénquima encefálico y/o meninges que evidencien vasculitis. Presentamos el caso de un paciente portador de retrovirus con probable VPSNC con clínica compatible, hallazgos imagenológicos sugestivos, con escasa alteración de LCR y EEG.2
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6

Canillas, M., B. Moreno-Burriel i E. Chinarro. "Materials directed to implants for repairing Central Nervous System". Boletín de la Sociedad Española de Cerámica y Vidrio 53, nr 6 (30.12.2014): 249–59. http://dx.doi.org/10.3989/cyv.302014.

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7

K, Sailaja. "A STUDY ON CONGENITAL ANOMALIES OF CENTRAL NERVOUS SYSTEM". International Journal of Anatomy and Research 5, nr 2.2 (31.05.2017): 3819–23. http://dx.doi.org/10.16965/ijar.2017.189.

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8

Lipton, Jordan D., i Robert W. Schafermeyer. "Central Nervous System Infections". Emergency Medicine Clinics of North America 13, nr 2 (maj 1995): 417–43. http://dx.doi.org/10.1016/s0733-8627(20)30358-8.

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9

Frost, Elizabeth A. M. "Central Nervous System Trauma". Anesthesiology Clinics of North America 5, nr 3 (wrzesień 1987): 565–85. http://dx.doi.org/10.1016/s0889-8537(21)00334-5.

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10

Brem, Steven S., Philip J. Bierman, Henry Brem, Nicholas Butowski, Marc C. Chamberlain, Ennio A. Chiocca, Lisa M. DeAngelis i in. "Central Nervous System Cancers". Journal of the National Comprehensive Cancer Network 9, nr 4 (kwiecień 2011): 352–400. http://dx.doi.org/10.6004/jnccn.2011.0036.

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11

Nabors, Louis Burt, Mario Ammirati, Philip J. Bierman, Henry Brem, Nicholas Butowski, Marc C. Chamberlain, Lisa M. DeAngelis i in. "Central Nervous System Cancers". Journal of the National Comprehensive Cancer Network 11, nr 9 (wrzesień 2013): 1114–51. http://dx.doi.org/10.6004/jnccn.2013.0132.

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12

D'Addario, Vincenzo, i Capuano Pasquale. "Central Nervous System Malformations". Donald School Journal of Ultrasound in Obstetrics and Gynecology 10, nr 3 (2016): 235–55. http://dx.doi.org/10.5005/jp-journals-10009-1472.

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ABSTRACT Ultrasound (US) is a useful tool to evaluate the normal morphology, the developmental changes, and the malformations of the fetal central nervous system (CNS). The development of the fetal CNS is a complex and continuous process progressing till the end of pregnancy and even after delivery. Although, a limited number of CNS anomalies may be suspected in the 1st trimester, the 2nd trimester is the best period of pregnancy to screen for CNS anomalies, but some malformations may be recognized only in the 3rd trimester or become evident only in the postnatal period. Screening for CNS anomalies relies on the use of the basic examination, which requires two simple axial planes on the fetal head (transventricular and transcerebellar). For a more detailed evaluation of brain malformations, an expanded fetal neurosonogram is needed, based on the use of multiple sagittal and coronal planes. The correct diagnosis of a CNS anomaly must be followed by an accurate counseling since the prognosis is varying widely. How to cite this article Vincenzo D, Pasquale C. Central Nervous System Malformations. Donald School J Ultrasound Obstet Gynecol 2016;10(3):235-255.
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13

Laughlin, Suzanne, i Walter Montanera. "Central nervous system imaging". Postgraduate Medicine 104, nr 5 (listopad 1998): 73–88. http://dx.doi.org/10.3810/pgm.1998.11.402.

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14

Gallek, Matthew J., i Leslie Ritter. "Central Nervous System Genomics". Annual Review of Nursing Research 29, nr 1 (grudzień 2011): 205–26. http://dx.doi.org/10.1891/0739-6686.29.205.

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In the past 25 years, remarkable progress has been made in our understanding of genomics and its influence on central nervous system diseases. In this chapter, common diseases of the central nervous system will be reviewed along with the genomics associated with these diseases. The diseases/injuries that will be investigated include neurovascular disorders such as ischemic stroke, hemorrhagic stroke, subarachnoid hemorrhage, and traumatic brain injury. This chapter will also explore Apolipoprotein E (APOE), a 299-aminoacid protein encoded by the APOE gene, and its associations with many of the previously named diseases. APOE was first tied to the risk of Alzheimer's disease and has since then been investigated in traumatic brain injury and hemorrhagic strokes. In addition, we will discuss the future of genomic research in central nervous system diseases.
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15

Yoshida, Mari. "1. Central Nervous System". Nihon Naika Gakkai Zasshi 99, nr 8 (2010): 1845–52. http://dx.doi.org/10.2169/naika.99.1845.

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16

Cataño, Juan, i Jessica Porras. "Central Nervous System Tuberculoma". American Journal of Tropical Medicine and Hygiene 105, nr 1 (7.07.2021): 3. http://dx.doi.org/10.4269/ajtmh.20-1495.

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17

KIYMAZ, NEJMI, i BAYRAM CIRAK. "Central Nervous System Lipomas." Tohoku Journal of Experimental Medicine 198, nr 3 (2002): 203–6. http://dx.doi.org/10.1620/tjem.198.203.

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18

Zimmerman, Robert A. "Central Nervous System Lymphoma". Radiologic Clinics of North America 28, nr 4 (lipiec 1990): 697–721. http://dx.doi.org/10.1016/s0033-8389(22)01256-8.

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19

King, Deidre S. "CENTRAL NERVOUS SYSTEM INFECTIONS". Nursing Clinics of North America 34, nr 3 (wrzesień 1999): 761–71. http://dx.doi.org/10.1016/s0029-6465(22)02416-1.

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20

Miller, Douglas C., Frederick F. Lang i Fred J. Epstein. "Central nervous system gangliogliomas". Journal of Neurosurgery 79, nr 6 (grudzień 1993): 859–66. http://dx.doi.org/10.3171/jns.1993.79.6.0859.

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Histopathological features that suggest the diagnosis of ganglioglioma require, in most cases, confirmation by special stains to distinguish these tumors from other gliomas. For this purpose, immunostaining for synaptophysin, which has previously been shown to selectively label the cell surface of neoplastic ganglion cells, was used to retrospectively examine glioma tumor specimens. Sixty-three cases of ganglioglioma were identified. The files of the Division of Neuropathology of New York University Medical Center contained 45 tumors that had been diagnosed as ganglioglioma, of which 42 were verified by synaptophysin; three cases were reclassified, two as astrocytomas and one as a gangliocytic paraganglioma. Thus, a tumor identified as ganglioglioma based on other criteria was likely to be a ganglioglioma. The other 21 cases of gangliogliomas were originally diagnosed as astrocytoma or mixed glioma, but were shown by synaptophysin staining to be gangliogliomas. In some cases the ultimate diagnosis was obtained after radical surgery provided relatively abundant amounts of tissue, thereby limiting sampling errors, in contrast to the biopsies from which the original diagnoses were made. Histopathological review of these cases demonstrated that four features represent important clues to the correct diagnosis: 1) clusters of large cells potentially representing neurons (without such cells the tumor cannot be classified as a ganglioglioma); 2) no perineuronal clustering of the glial cells around the alleged neoplastic neurons; 3) fibrosis (desmoplasia); and 4) calcification. Binucleate neurons, previously suggested to be common in gangliogliomas, were not frequently found in this series, and lymphocytic infiltrates, while common, are so often found in other tumors that they gave no specific hint that any single neoplasm was a ganglioglioma. The glial elements were astrocytic in all cases, except that one tumor also had oligodendroglial and ependymal patterns. Four tumors also had small mature neurons, as seen in neurocytomas. Cells from one tumor were successfully grown in short-term tissue culture; the culture contained large dividing neurons with synaptophysin immunoreactivity as well as smaller dividing cells, demonstrating that the neuronal cells are a proliferating element in gangliogliomas.
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21

Lang, Frederick F., Fred J. Epstein, Joseph Ransohoff, Jeffrey C. Allen, Jeffrey Wisoff, I. Richmond Abbott i Douglas C. Miller. "Central nervous system gangliogliomas". Journal of Neurosurgery 79, nr 6 (grudzień 1993): 867–73. http://dx.doi.org/10.3171/jns.1993.79.6.0867.

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The records of 58 patients with gangliogliomas surgically treated between January 1, 1980, and June 30, 1990, were retrospectively reviewed in order to determine long-term survival, event-free survival, and functional outcome resulting after radical resection and to assess the impact of histological grading on outcome. Tumors were located in the cerebral hemisphere in 19 cases, the spinal cord in 30, and the brain stem in nine. Forty-four patients had gross total resection and 14 had radical subtotal resection. Only six patients underwent postoperative irradiation or chemotherapy and, therefore, the outcome was generally related to surgery alone. Of the 58 gangliogliomas, 40 were classified as histological grade I, 16 were grade II, and two were grade III. The median follow-up period was 56 months. There were no operative deaths, and the operative morbidity rate was 5%, 37%, and 33% for cerebral hemisphere, spinal cord, and brain-stem gangliogliomas, respectively. The 5-year actuarial survival rates for cerebral hemisphere, spinal cord, and brain-stem gangliogliomas were 93%, 84%, and 73%, respectively (p = 0.7). The event-free survival rate at 5 years was 95% for cerebral hemisphere gangliogliomas and 36% for spinal cord gangliogliomas (p < 0.05); for brain-stem gangliogliomas the event-free survival rate at 3 years was 53% (p < 0.05). Neurological function at recent follow-up evaluation was stable or improved in 81% of patients. Multivariate analysis (Cox linear regression) revealed tumor location to be the only variable predictive of outcome, with spinal cord and brain-stem gangliogliomas having a 3.5- and 5-fold increased relative risk of recurrence, respectively, compared to cerebral hemisphere gangliogliomas. Histological grade was not predictive of outcome, although in each location there was a trend for higher-grade tumors to have a shorter time to recurrence. It is concluded that radical surgery leads to long-term survival of patients with gangliogliomas, regardless of location, and adjuvant therapy can probably be reserved for special cases.
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22

Reardon, David A. "Central Nervous System Malignancies". Hematology/Oncology Clinics of North America 36, nr 1 (luty 2022): i. http://dx.doi.org/10.1016/s0889-8588(21)00149-0.

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23

Nieblas-Bedolla, Edwin, Jeffrey Zuccato, Harriet Kluger, Gelareh Zadeh i Priscilla K. Brastianos. "Central Nervous System Metastases". Hematology/Oncology Clinics of North America 36, nr 1 (luty 2022): 161–88. http://dx.doi.org/10.1016/j.hoc.2021.08.004.

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24

Rothwell, John, Andrea Antal, David Burke, Antony Carlsen, Dejan Georgiev, Marjan Jahanshahi, Dagmar Sternad, Josep Valls-Solé i Ulf Ziemann. "Central nervous system physiology". Clinical Neurophysiology 132, nr 12 (grudzień 2021): 3043–83. http://dx.doi.org/10.1016/j.clinph.2021.09.013.

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25

Dian, Sofiati, Ahmad Rizal Ganiem i Arjan van Laarhoven. "Central nervous system tuberculosis". Current Opinion in Neurology 34, nr 3 (3.03.2021): 396–402. http://dx.doi.org/10.1097/wco.0000000000000920.

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26

Varadarajan, Supraja G., John L. Hunyara, Natalie R. Hamilton, Alex L. Kolodkin i Andrew D. Huberman. "Central nervous system regeneration". Cell 185, nr 1 (styczeń 2022): 77–94. http://dx.doi.org/10.1016/j.cell.2021.10.029.

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27

Kawahata, Kimito. "1. Central Nervous System". Nihon Naika Gakkai Zasshi 102, nr 10 (2013): 2532–42. http://dx.doi.org/10.2169/naika.102.2532.

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28

Buckner, Jan C., Paul D. Brown, Brian P. O'Neill, Fredric B. Meyer, Cynthia J. Wetmore i Joon H. Uhm. "Central Nervous System Tumors". Mayo Clinic Proceedings 82, nr 10 (październik 2007): 1271–86. http://dx.doi.org/10.4065/82.10.1271.

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29

Blaney, Susan M., i David G. Poplack. "Central nervous system leukemia". Current Opinion in ONCOLOGY 8, nr 1 (styczeń 1996): 13–19. http://dx.doi.org/10.1097/00001622-199601000-00003.

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30

Klein, Joshua P., i Robin C. Ryther. "Central Nervous System Hemorrhage". New England Journal of Medicine 361, nr 18 (29.10.2009): 1786. http://dx.doi.org/10.1056/nejmicm0900232.

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31

Walshe, J. M. "Central nervous system regeneration". QJM 107, nr 8 (6.06.2014): 687. http://dx.doi.org/10.1093/qjmed/hcu120.

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32

Elster, Allen D. "MRI: Central Nervous System". Radiology 178, nr 2 (luty 1991): 392. http://dx.doi.org/10.1148/radiology.178.2.392.

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33

Rumboldt, Zoran, Majda M. Thurnher i Rakesh K. Gupta. "Central Nervous System Infections". Seminars in Roentgenology 42, nr 2 (kwiecień 2007): 62–91. http://dx.doi.org/10.1053/j.ro.2006.08.012.

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34

Mahley, Robert W. "Central Nervous System Lipoproteins". Arteriosclerosis, Thrombosis, and Vascular Biology 36, nr 7 (lipiec 2016): 1305–15. http://dx.doi.org/10.1161/atvbaha.116.307023.

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35

Mallia, Charles C., i Martin J. Wood. "Central nervous system parasitoses". Current Opinion in Infectious Diseases 7, nr 6 (grudzień 1994): 692–95. http://dx.doi.org/10.1097/00001432-199412000-00012.

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36

Frost, E. A. M. "Central nervous system trauma". Current Opinion in Anaesthesiology 1, nr 3 (wrzesień 1988): 298–304. http://dx.doi.org/10.1097/00001503-198801030-00006.

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Frost, E. A. M. "Central nervous system trauma". Current Opinion in Anaesthesiology 1, nr 3 (wrzesień 1988): 298–304. http://dx.doi.org/10.1097/00001503-198809000-00006.

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38

Grommes, Christian. "Central Nervous System Lymphomas". CONTINUUM: Lifelong Learning in Neurology 26, nr 6 (grudzień 2020): 1476–94. http://dx.doi.org/10.1212/con.0000000000000936.

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39

Kourkoumpetis, Themistoklis K., Athanasios Desalermos, Maged Muhammed i Eleftherios Mylonakis. "Central Nervous System Aspergillosis". Medicine 91, nr 6 (listopad 2012): 328–36. http://dx.doi.org/10.1097/md.0b013e318274cd77.

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40

Kuo, Vincent C., Louis M. Sloan i Michael Emmett. "Central Nervous System Tuberculosis". Baylor University Medical Center Proceedings 23, nr 4 (październik 2010): 359–60. http://dx.doi.org/10.1080/08998280.2010.11928652.

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41

Moore, Patricia M. "Central nervous system vasculitis". Current Opinion in Neurology 11, nr 3 (czerwiec 1988): 241–46. http://dx.doi.org/10.1097/00019052-199806000-00009.

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42

Lister, Andrew, Lauren E. Abrey i John T. Sandlund. "Central Nervous System Lymphoma". Hematology 2002, nr 1 (1.01.2002): 283–96. http://dx.doi.org/10.1182/asheducation-2002.1.283.

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Abstract Central nervous system involvement with malignant lymphoma whether primary or secondary is an uncommon but not rare complication observed in the management of patients with hematological malignancy. Its importance lies in the considerable morbidity and mortality with which it is associated and the inadequacy of therapy. In Section I, Dr. Lauren Abrey addresses the totality of the problem of primary central nervous system lymphoma, with emphasis on strategies increasingly dependent on systemic chemotherapy. In Section II, Dr. John Sandlund reviews the success of sequential clinical trials of overall therapy for acute lymphoblastic leukemia in childhood, identifying those patients at high risk of central nervous system leukemia and the development of a rational therapeutic strategy for prevention. In Section III, Dr. Andrew Lister discusses the issue of secondary central nervous system involvement with lymphoma and the indications for prophylaxis.
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43

Liebel, Francois‐Xavier, i Peter M. Smith. "Central nervous system neoplasia". In Practice 36, S1 (wrzesień 2014): 24–29. http://dx.doi.org/10.1136/inp.g5097.

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44

Schwartz, Ilsa R. "Central Nervous System Studies". Annals of Otology, Rhinology & Laryngology 98, nr 12_suppl (grudzień 1989): 40–42. http://dx.doi.org/10.1177/0003489489098s1220.

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45

Desmonts, G. "CENTRAL NERVOUS SYSTEM TOXOPLASMOSIS". Pediatric Infectious Disease Journal 6, nr 9 (wrzesień 1987): 872. http://dx.doi.org/10.1097/00006454-198709000-00022.

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46

Johnston, Michael V. "Central nervous system drugs". Current Opinion in Pediatrics 2, nr 2 (kwiecień 1990): 230–33. http://dx.doi.org/10.1097/00008480-199004000-00004.

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47

Scolding, N. J. "CENTRAL NERVOUS SYSTEM ANGIITIS." Brain 123, nr 11 (listopad 2000): 2364–65. http://dx.doi.org/10.1093/brain/123.11.2364.

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48

Bleasel, Andrew. "The Central Nervous System". Journal of Clinical Neurophysiology 11, nr 4 (lipiec 1994): 466–67. http://dx.doi.org/10.1097/00004691-199407000-00010.

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49

Bleyer, W. Archie. "Central Nervous System Leukemia". Pediatric Clinics of North America 35, nr 4 (sierpień 1988): 789–814. http://dx.doi.org/10.1016/s0031-3955(16)36510-5.

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50

Bozzola, Elena. "Central Nervous System Infections". Journal of Pediatric Infectious Diseases 14, nr 01 (23.01.2018): 001. http://dx.doi.org/10.1055/s-0037-1618595.

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