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Artykuły w czasopismach na temat "Cellules de Purkinje – toxicité"
MOILLERON, R., C. MORIN, L. PAULIC, A. MARCONI, V. ROCHER, R. MAILLER, A. BRESSY i L. GARRIGUE-ANTAR. "Caractérisation du potentiel toxique des eaux urbaines par bioessais – Cas de l’agglomération parisienne". Techniques Sciences Méthodes, nr 12 (20.01.2020): 175–94. http://dx.doi.org/10.36904/tsm/201912175.
Pełny tekst źródłaDjeneb, Camara, Yapi Adon Basile, Fofié N’guessan Bra Yvette, Ouattara Katinan Etienne i Zirihi Guédé Noël. "Etude Comparative des Toxicités Cellulaires et Aigües de Ageratum conyzoides L. et de Acanthospermum hispidum DC". European Scientific Journal ESJ 17, nr 40 (30.11.2021): 74–87. http://dx.doi.org/10.19044/esj.2021.v17n40p74.
Pełny tekst źródłaViala, Marie, i Diego Tosi. "Déterminer la dose à injecter lors des premières études cliniques menées avec un anticorps monoclonal". médecine/sciences 35, nr 12 (grudzień 2019): 1121–29. http://dx.doi.org/10.1051/medsci/2019209.
Pełny tekst źródłaKostine, Marie, Aurélien Marabelle, Thierry Schaeverbeke i Maria Kfoury. "Les limites des inhibiteurs de points de contrôle immunitaire et la gestion de leur toxicité". médecine/sciences 35, nr 12 (grudzień 2019): 949–56. http://dx.doi.org/10.1051/medsci/2019191.
Pełny tekst źródłaLuce, Eléanor, Antonietta Messina, Amandine Caillaud, Karim Si-Tayeb, Bertrand Cariou, Etienne Bur, Anne Dubart-Kupperschmitt i Jean-Charles Duclos-Vallée. "Les organoïdes hépatiques". médecine/sciences 37, nr 10 (październik 2021): 902–9. http://dx.doi.org/10.1051/medsci/2021119.
Pełny tekst źródłaPeirone, S. M., i G. Filogamo. "Caractérisation immunophénotypique des cellules de Purkinje du cœur". Morphologie 88, nr 281 (lipiec 2004): 90. http://dx.doi.org/10.1016/s1286-0115(04)98072-1.
Pełny tekst źródłaFeuillard, J. C. "Les toxines des cyanobactéries : revue de synthèse". Revue des sciences de l'eau 5, nr 4 (12.04.2005): 489–508. http://dx.doi.org/10.7202/705143ar.
Pełny tekst źródłaAgier, Catherine, Michèle Bury, Robert Farinotti i Claude Viel. "Toxicité de la chloroquine vis-à-vis de cellules deSaponaria officinalisL. (Caryophyllacées)". Bulletin de la Société Botanique de France. Lettres Botaniques 138, nr 1 (styczeń 1991): 39–45. http://dx.doi.org/10.1080/01811797.1991.10824904.
Pełny tekst źródłaCittanova, M. L., F. Wahbé, D. Prié, P. M. Ronco i P. Viars. "Toxicité Directe des Ions Fluorure sur une Lignée de Cellules Rénales Humaines". Annales Françaises d'Anesthésie et de Réanimation 12, nr 12 (1993): R70. http://dx.doi.org/10.1016/s0750-7658(16)30070-3.
Pełny tekst źródłaHosni, H., A. Salama, A. Abudunia, Y. Cherrah, A. Ibrahimi i K. Alaoui. "Toxicité aiguë, cytotoxicité et effet antiradicalaire de l’extrait méthanolique des feuilles de l’asphodèle, Asphodelus microcarpus". Phytothérapie 18, nr 5 (2.07.2019): 284–90. http://dx.doi.org/10.3166/phyto-2019-0136.
Pełny tekst źródłaRozprawy doktorskie na temat "Cellules de Purkinje – toxicité"
Heitz, Stéphane. "Neuronal death mechanisms in cerebellar Purkinje cells". Strasbourg 1, 2008. https://publication-theses.unistra.fr/public/theses_doctorat/2008/HEITZ_Stephane_2008.pdf.
Pełny tekst źródłaNeuropathologies involve specific apoptotic and autophagic pathways which identification is important to gain insight into preventing loss of central neurons. My studies were focused on cerebellar Purkinje cells (PC) the death of which was investigated in GluRd2 mutant mice and in NP0/0 mice knock-out for prion protein and overexpressing its neurotoxic paralogue Doppel. The study of GluRd2Lc indicates a link between autophagy and excitotoxicity in PCs which are rescued from excitotoxic cell death and autophagy blockade of excitotoxicity. In the NP0/0 mouse, Doppel induces the BAX-dependent apoptotic cell death of PCs. Nevertheless, an alternative process could be either autophagy detected early in PCs or an unidentified apoptotic mechanism. These data suggest that different pathogenic stimuli trigger different cell death modalities involving apoptosis and autophagy in the same neuron
Rakotomamonjy, Jennifer. "Mort développementale des cellules de Purkinje". Thesis, Paris 11, 2011. http://www.theses.fr/2011PA11T031.
Pełny tekst źródłaProgrammed cell death is an essential feature of the central nervous system during development. Purkinje cells, in cerebellar organotypic slice cultures, die when tissue is taken from one-week-old animals. Beyond this critical period, they survive. This massive death is supposed to reflect a naturel process occurring in the developing cerebellum. The synthetic steroid mifepristone allows neuron to survive by a mechanism involving depolarization. We show that the spontaneous release of the neurotransmitter GABA induces the activation of GABAA receptors which leads to Purkinje cell firing and death. This GABA toxicity is also accompanied by an intracellular calcium release. Mifepristone depolarizes Purkinje cell membrane potential to a value above chloride reversal potential, thus shunting spiking activity and GABAergic conductance. Moreover, the steroid neuroprotective effect is mediated by the neurotrophic factor BDNF and involves the inhibition of p38 MAP-kinase pathway. Our data provide new insights in the search for treatments preventing GABA toxicity in the developing brain
Heitz, Stéphane Bailly Yannick Kapfhammer Josef P. Poulain Bernard. "Neuronal death mechanisms in cerebellar Purkinje cells". Strasbourg : Université Louis Pasteur, 2008. http://eprints-scd-ulp.u-strasbg.fr:8080/1012/01/HEITZ_Stephane_2008.pdf.
Pełny tekst źródłaThèse soutenue en co-tutelle. Titre provenant de l'écran-titre. Bibliogr. 37 p.
MORAIN, PHILIPPE. "Proprietes electrophysiologiques des cellules de purkinje de rat : modification apres desafferentation". Paris 6, 1989. http://www.theses.fr/1989PA066357.
Pełny tekst źródłaOrduz, Pérez David Andrés. "Propriétés électrophysiologiques des synapses Purkinje-Purkinje du cervelet de souris et caractérisation des dynamiques calciques des boutons présynaptiques de leurs collatérales récurrentes". Paris 6, 2007. http://www.theses.fr/2007PA066247.
Pełny tekst źródłaLay, Russo Nadège. "Différenciation des cellules souches embryonnaires murines et des cellules souches pluripotentes induites humaines en cellules dendritiques : cellules d'intérêt pour les tests de toxicologie". Nice, 2012. http://www.theses.fr/2012NICE4077.
Pełny tekst źródłaThe seventh amendment in the European cosmetic directive imposes an abandonment of the tests on animals to measure the allergenic or irritant effects of some compounds used in cosmetic. The allergenic response in animals ‘models includes five aspects but in the in vitro test each aspect is studied separately. Among the in vitro tests of toxicity which are envisaged, dendritic cells, which are antigen presenting cells, were revealed as cells of choice for study one of these aspects. However today it is difficult to obtain a reliable and strong source of dendritic cells allowing working out a reglementary test. The aim of my thesis project was to propose an alternative model in these tests on animals. For it we set up the conditions allowing generating dendritic cells derived of stern cells. For it we have two sources of stem cells (hiPS) which having all the characteristics of the embryonic stem cells without raise ethical problems. These two types of cells allowed having an inexhaustible and plentiful source of dendritic cells. We set up one protocol allowing generating and purifying a population of dendritic cells from mouse embryonic stem cells. Cells were characterized by gene expression like CD45, CD86. Furthermore we realized as functional test that consists to measure the dextran-FICT endocytosis and the answer of this cellular population to allergenic reference molecules such as MCI/MI or TNBS. We also tried to generate “dendritic-like” cells from human iPS based to expression of specifics markers as CD45, CD34, CD1a, CD14, CD209, CD207, CD86 and HLA-DR. Several protocols were envisaged. However dendritic-like cells obtained represent a low percentage of differentiated cells and the protocol is in the course of optimization. Increasingly tests use keratinocytes cells for evaluate another aspect of allergenic response. So we were interesting also to these cells and we will present first steps differentiation of hiPS that will allow generating keratinocytes
Vincent, Pierre. "Courants synaptiques gabergiques enregistrés dans les cellules de Purkinje du cervelet de rat". Paris 6, 1994. http://www.theses.fr/1994PA066709.
Pełny tekst źródłaAzouri, Tannous Hayat. "Étude de la toxicité de deux complexes du platine : description d'un nouveau modèle d'appréciation de la toxicité testiculaire". Paris 11, 1989. http://www.theses.fr/1989PA114822.
Pełny tekst źródłaChaumont, Joseph. "Organisation fonctionnelle de la boucle olivo-cortico-nucléaire : influence de l'activité des cellules de Purkinje". Thesis, Strasbourg, 2014. http://www.theses.fr/2014STRAJ128/document.
Pełny tekst źródłaThe cerebellum plays a fundamental role in coordination, adjustment, planning and automation of movements, in the modulation of reflexes and in some cognitive functions. To do this, it will collect motor and sensory information from both the cerebral cortex and the rest of the body. These information are relayed to the cortex and cerebellar nuclei via climbing fibers and mossy fibers. Climbing fibers, the projections from the inferior olive to the cerebellar cortex, carry sensorimotor error and clock signals that trigger motor learning by controlling cerebellar Purkinje cell synaptic plasticity and discharge. Purkinje cells target the deep cerebellar nuclei, which are the output of the cerebellum and include an inhibitory GABAergic projection to the inferior olive. This pathway identifies a potential closed loop in the olivo-cortico-nuclear network. Therefore, sets of Purkinje cells may phasically control their own climbing fiber afferents. Here, using in vitro and in vivo recordings, we describe a genetically modified mouse model that allows the specific optogenetic control of Purkinje cell discharge. Tetrode recordings in the cerebellar nuclei demonstrate that focal stimulations of Purkinje cells strongly inhibit spatially restricted sets of cerebellar nuclear neurons. Strikingly, such stimulations trigger delayed climbing-fiber input signals in the stimulated Purkinje cells. Therefore, our results demonstrate that Purkinje cells phasically control the discharge of their own olivary afferents and thus might participate in the regulation of cerebellar motor learning
Cavelier, Pauline. "Rôle des canaux ioniques sensibles au potentiel dans l'excitabilité des cellules de Purkinje du cervelet". Université Louis Pasteur (Strasbourg) (1971-2008), 2002. http://www.theses.fr/2002STR13170.
Pełny tekst źródłaElectrical properties of excitable cells are didacted by the ion channels present in the membrane. Purkinje cells (PCs) of the cerebellar cortex represent a unique model to study the role of ion channels in determining the firing pattern generated by various synaptic inputs as well as, owing to their large dendritic tree, the electrophysiology of the dendro-somatic interactions. My thesis focused on the role of different ion channels in determining the electrical properties of PCs dendrites and soma. Dendritically and somatically evoked discharges of action potentials (APs) were recorded under current clamp conditions. The whole cell configuration of the patch-clamp technique was used in PCs from rat cerebellar slice cultures. The relation between the firing pattern and a given channel type was determined by using specific pharmacological tools. First, I examined the role of calcium entries in the establishment of PCs firing pattern. My results demonstrate that Ca2+ entry through low-voltage gated calcium channels underlies a dendritic AP rarely eliciting a somatic burst of APs. Second, I investigated the ionic mechanisms controlling the dendrosomatic propagation of this low-threshold Ca2+ spike in PCs. My results suggest that the propagation of dendritic LTS is controlled directly by 4-AP sensitive K+ channels, and indirectly modulated by P/Q channel activity probably by activating the calcium dependant K+ channels of the BK family. Similar conclusions can be drawn studying the electrical properties of PCs in mice lacking functional P/Q channels. Additionnal results suggest that TEA and 4-AP sensitive channel activity affects the electrical properties of dendrites such that the LTS is attenuated and slowed-down when propagating to the soma. Finally, after determining the pharmacological characteristics of the LTS I demonstrated that it is involved in the genesis of the complex spike elicited by climbing fiber stimulation. Lice cultures. The relation between the firing pattern and a given channel type was determined by using specific pharmacological tools. First, I examined the role of calcium entries in the establishment of PCs firing pattern. My results demonstrate that Ca2+ entry through low-voltage gated calcium channels underlies a dendritic AP rarely eliciting a somatic burst of APs. Second, I investigated the ionic mechanisms controlling the dendrosomatic propagation of this low-threshold Ca2+ spike in PCs. My results suggest that the propagation of dendritic LTS is controlled directly by 4-AP sensitive K+ channels, and indirectly modulated by P/Q channel activity probably by activating the calcium dependant K+ channels of the BK family. Similar conclusions can be drawn studying the electrical properties of PCs in mice lacking functional P/Q channels. Additionnal results suggest that TEA and 4-AP sensitive channel activity affects the electrical properties of dendrites such that the LTS is attenuated and slowed-down when propagating to the soma. Finally, after determining the pharmacological characteristics of the LTS I demonstrated that it is involved in the genesis of the complex spike elicited by climbing fiber stimulation