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Artykuły w czasopismach na temat "Cellules C26"
COSTA, CÉSAR S. B., JULIANO R. M. VICENTI, JOAQUÍN A. MORÓN-VILLARREYES, SERGIANE CALDAS, LIZIANE V. CARDOSO, RICARDO F. FREITAS i MARCELO G. M. D'OCA. "Extraction and characterization of lipids from Sarcocornia ambigua meal: a halophyte biomass produced with shrimp farm effluent irrigation". Anais da Academia Brasileira de Ciências 86, nr 2 (czerwiec 2014): 935–43. http://dx.doi.org/10.1590/0001-3765201420130022.
Pełny tekst źródłaMarkowski, Adam R., Agnieszka U. Błachnio-Zabielska, Karolina Pogodzińska, Anna J. Markowska i Piotr Zabielski. "Diverse Sphingolipid Profiles in Rectal and Colon Cancer". International Journal of Molecular Sciences 24, nr 13 (29.06.2023): 10867. http://dx.doi.org/10.3390/ijms241310867.
Pełny tekst źródłaAlbouery, Mayssa, Alexis Bretin, Bénédicte Buteau, Stéphane Grégoire, Lucy Martine, Ségolène Gambert, Alain M. Bron, Niyazi Acar, Benoit Chassaing i Marie-Agnès Bringer. "Soluble Fiber Inulin Consumption Limits Alterations of the Gut Microbiota and Hepatic Fatty Acid Metabolism Caused by High-Fat Diet". Nutrients 13, nr 3 (23.03.2021): 1037. http://dx.doi.org/10.3390/nu13031037.
Pełny tekst źródłaTobin, V., M. Le Gall, M. Romero, A. Leturque i E. Brot-Laroche. "C26 - L’insuline est un inhibiteur de la localisation apicale de GLUT2 dans les entérocytes : une étude in vivo chez la souris et in vitro dans les cellules CACO-2/TC7". Gastroentérologie Clinique et Biologique 30, nr 1 (styczeń 2006): 86. http://dx.doi.org/10.1016/s0399-8320(06)73108-6.
Pełny tekst źródłaDetzner, Johanna, Charlotte Püttmann, Gottfried Pohlentz, Hans-Ulrich Humpf, Alexander Mellmann, Helge Karch i Johannes Müthing. "Primary Human Colon Epithelial Cells (pHCoEpiCs) Do Express the Shiga Toxin (Stx) Receptor Glycosphingolipids Gb3Cer and Gb4Cer and Are Largely Refractory but Not Resistant towards Stx". International Journal of Molecular Sciences 22, nr 18 (16.09.2021): 10002. http://dx.doi.org/10.3390/ijms221810002.
Pełny tekst źródłaDetzner, Johanna, Elisabeth Krojnewski, Gottfried Pohlentz, Daniel Steil, Hans-Ulrich Humpf, Alexander Mellmann, Helge Karch i Johannes Müthing. "Shiga Toxin (Stx)-Binding Glycosphingolipids of Primary Human Renal Cortical Epithelial Cells (pHRCEpiCs) and Stx-Mediated Cytotoxicity". Toxins 13, nr 2 (12.02.2021): 139. http://dx.doi.org/10.3390/toxins13020139.
Pełny tekst źródłaGuo, Tianyao, Zhigui Duan, Jia Chen, Chunliang Xie, Ying Wang, Ping Chen i Xianchun Wang. "Pull-down combined with proteomic strategy reveals functional diversity of synaptotagmin I". PeerJ 5 (8.02.2017): e2973. http://dx.doi.org/10.7717/peerj.2973.
Pełny tekst źródłaDetzner, Johanna, Anna-Lena Klein, Gottfried Pohlentz, Elisabeth Krojnewski, Hans-Ulrich Humpf, Alexander Mellmann, Helge Karch i Johannes Müthing. "Primary Human Renal Proximal Tubular Epithelial Cells (pHRPTEpiCs): Shiga Toxin (Stx) Glycosphingolipid Receptors, Stx Susceptibility, and Interaction with Membrane Microdomains". Toxins 13, nr 8 (28.07.2021): 529. http://dx.doi.org/10.3390/toxins13080529.
Pełny tekst źródłaMarkowski, Adam R., Arkadiusz Żbikowski, Piotr Zabielski, Urszula Chlabicz, Patrycja Sadowska, Karolina Pogodzińska i Agnieszka U. Błachnio-Zabielska. "The Effect of Silencing the Genes Responsible for the Level of Sphingosine-1-phosphate on the Apoptosis of Colon Cancer Cells". International Journal of Molecular Sciences 24, nr 8 (13.04.2023): 7197. http://dx.doi.org/10.3390/ijms24087197.
Pełny tekst źródłaAl-Qarawi, AA, EF Abd Allah i Hashem Abeer. "Effect of Ephedra alata Decne. on lipids metabolism of Aspergillus flavus Link". Bangladesh Journal of Botany 42, nr 1 (24.07.2013): 45–50. http://dx.doi.org/10.3329/bjb.v42i1.15823.
Pełny tekst źródłaRozprawy doktorskie na temat "Cellules C26"
Chaouki, Ghita. "Etude du rôle de la voie de signalisation eIF2αATF4 au cours des états inflammatoires, dans le cadre du stress mitochondrial et de l’anorexie associée à la pathologie". Electronic Thesis or Diss., Université Clermont Auvergne (2021-...), 2022. http://www.theses.fr/2022UCFAC109.
Pełny tekst źródłaThe eIF2α-ATF4 signaling pathway is activated in cells in response to a wide range of cellular stresses. Its activation leads to the inhibition of the global protein synthesis and the regulation of the transcription factor ATF4 target genes expression. This pathway is activated in response to essential amino acid deficiency, mitochondrial stress, endoplasmic reticulum stress or viral infections. Its activation triggers adaptive mechanisms, both at the cellular level (such as inhibition of protein synthesis and increased autophagy) and at the whole organism level (such as regulation of metabolism, inflammation, immunity and food intake). Previous results generated by our laboratory as well as data from the scientific literature led us to investigate the role of eIF2α-ATF4 signaling in two different contexts. Firstly, we explored the role of eIF2α-ATF4 signaling in anorexia associated with catabolic inflammatory pathologies (sepsis and cancer). We hypothesized that this signaling pathway could contribute to the inhibition of food intake by its direct action at the central level and/or by stimulating the expression of anorectic cytokines, including GDF15, in the periphery (liver, intestine). We used two experimental models reproducing pathology-associated anorexia in mice: a sepsis model of acute and systemic inflammation (single administration of bacterial lipopolysaccharide) and a model of mice carrying a C26 colon carcinoma cell tumor. Both models were characterized in the early phase of anorexia by inflammation at the peripheral and central (hypothalamus) levels, increased circulating levels of IL-6 and GDF15, profound alterations in amino acid metabolism, and activation of the eIF2αATF4 signaling pathway in the hypothalamus and liver. Afterwards, the response of inducible models of ATF4 loss-of-function was tested in the sepsis model. ATF4 knock-out in the liver and intestine had no impact on either anorexia or the induction of GDF15 production. Constitutive invalidation of GDF15 also had no effect on the inhibition of food intake induced by LPS administration. The role of ATF4 function at the central level could not be tested and should be the subject of future experiments. The analysis of samples from mice knocked-out for ATF4 at the hepatic level, will allow us to evaluate ATF4 involvement in the reorientation of AA metabolism (transport, biosynthesis, autophagy). In the C26 cancer model, the transition from pre-anorexia to early anorexia was associated with an activation of the eIF2α-ATF4 signaling pathway at the hepatic and hypothalamic levels, and a pharmacological approach using ISRIB (ISR Inhibitor) will soon be implemented to study the involvement of the ISR in the regulation of appetite and AA metabolism (in this model, genes knock-out is not possible) Secondly, we focused on mitochondrial dysfunction, which represents a major threat to cellular homeostasis, promotes the development of many metabolic disorders and plays a crucial role in the pathogenesis of sepsis. Given the role played by the eIF2α-ATF4 signaling pathway in the adaptive response to mitochondrial stress, we investigated whether a pretreatment activating this pathway could be a way to increase the resilience of the mitochondrial pool during subsequent stressful events. We demonstrated in mice that a pretreatment activating the GCN2-eIF2α-ATF4 pathway upstream of inflammatory stress (LPS administration) counteracted some of the effects of this stress on mitochondrial homeostasis in the liver, an organ playing a major role in the metabolic and immune response to endotoxic stress. These results are presented as an article that will be submitted soon for publication
Allen, Frederick Jr. "CCL3 Augments Antitumor Responses in CT26 by Enhancing Cellular Trafficking and Interferon-Gamma Expression". Case Western Reserve University School of Graduate Studies / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=case1513124234665339.
Pełny tekst źródłaFilali-Ansary, Aziz. "Régulation de l'incorporation du cholestérol venant de liposomes ou de lipoprotéines, de la biosynthèse d'hormones stéroïdes en C21 et de leurs hydroxylations dans des cellules corticosurrénaliennes de rats transformées par l'oncogène Ha-rasEJ et dans les cellules de la lignée Y1". Dijon, 1990. http://www.theses.fr/1990DIJOS051.
Pełny tekst źródłaBezine, Maryem. "Implication du canal potassium Kv3.1 dans la lipotoxicité du 7-cétocholestérol, 24S-hydroxycholestérol et de l’acide tétracosanoïque sur des cellules nerveuses 158N et BV-2 : Etude des relations entre Kv3.1, homéostasie potassique et métabolisme peroxysomal dans la maladie d’Alzheimer". Thesis, Bourgogne Franche-Comté, 2017. http://www.theses.fr/2017UBFCI010/document.
Pełny tekst źródłaPotassium (K+) is involved in the regulation of cellular excitability, cell cycle regulation, cell viability, neuroprotection and maintenance of microglial and oligodendrocytic functions. Potassium dysfunction, described in several neurodegenerative diseases such as Alzheimer's Disease (AD), multiple sclerosis (MS), Parkinson's disease and Huntington's disease, may be a potential therapeutic target. The underlying toxic mechanisms of these neurodegenerative pathologies involve oxysterols, which are oxidized cholesterol derivatives, and fatty acids including those associated with peroxisomal metabolism. 7-ketocholesterol (7KC), 24S-hydroxycholesterol (24S-OHC) and tetracosanoic acid (C24:0), often found at increased levels in the brain and plasma of patients with neurodegenerative diseases (Nieman-Pick disease, MS, Parkinson's disease, Huntington's disease and X-ALD) lead to a breakdown of the redox equilibrium leading to neurodegeneration. In this context, it is interesting to determine the possible connection between the lipid environment and potassium homeostasis The in vitro study was carried out on 158N murine oligodendrocytes and microglial BV-2 cells. We have shown that the lipotoxicity of 7KC, 24S-OHC and C24:0 implies retention of K+ involving the voltage dependent potassium channels (Kv). These results have shown that inhibition of Kv channels lead to an increase in [K +] i contributing to the cytotoxicity of 7KC, 24S-OHC and C24:0. The retention of K+ induced by oxysterols (7KC and 24S-OHC) would be under the control of Kv3.1b. A clinical study, on plasma of patients with Alzheimer’s disease, revealed a negative correlation between docosahexaenoic acid (DHA) and K+ concentration. In the J20 mice, a transgenic model of Alzheimer’s disease, the expression of Kv3.1b and Abcd3 was decreased in the hippocampus and cortex. Overall, the results obtained established relationships between lipotoxicity, peroxisomal metabolism and potassium homeostasis in neurodegeneration and suggest a possible modulation of the expression and activity of kv3.1b in the pathophysiology of neurodegenerative diseases. So, modulation of Kv3.1 could constitute a new therapeuthic approach against some neurodegenerative diseases
MATIGNON, Christophe. "Etude de la détonation de deux mélanges stoechiométriques (CH4/H2/O2/N2 et CH4/C2H6/O2/N2) Influence de la proportion relative des deux combustibles et de la température initiale élevée". Phd thesis, Université de Poitiers, 2000. http://tel.archives-ouvertes.fr/tel-00010305.
Pełny tekst źródłaCzęści książek na temat "Cellules C26"
O’Neill, C. M., i R. J. Mathias. "Regeneration of Plants from Protoplasts of Arabidopsis Thaliana CV. Columbia L. (C24), Via Direct Embryogenesis". W Current Issues in Plant Molecular and Cellular Biology, 377–82. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0307-7_52.
Pełny tekst źródłaSharma, Sunny, i Karl-Dieter Entian. "Chemical Modifications of Ribosomal RNA". W Ribosome Biogenesis, 149–66. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2501-9_9.
Pełny tekst źródłaKelley, Brian. "Software-Defined Radio for Advanced Gigabit Cellular Systems". W Wireless, Networking, Radar, Sensor Array Processing, and Nonlinear Signal Processing, 1–13. CRC Press, 2009. http://dx.doi.org/10.1201/9781420046052-c22.
Pełny tekst źródła"11. Metabotropic Transmission: Receptors and G Proteins". W Molecular and Cellular Physiology of Neurons, 417–40. Harvard University Press, 2015. http://dx.doi.org/10.4159/harvard.9780674735644.c16.
Pełny tekst źródła"15. Mechanoreceptors". W Molecular and Cellular Physiology of Neurons, 545–82. Harvard University Press, 2015. http://dx.doi.org/10.4159/harvard.9780674735644.c21.
Pełny tekst źródła"16. Photoreceptors and Olfactory Receptor Neurons". W Molecular and Cellular Physiology of Neurons, 583–628. Harvard University Press, 2015. http://dx.doi.org/10.4159/harvard.9780674735644.c22.
Pełny tekst źródła"Appendix: Symbols Used". W Molecular and Cellular Physiology of Neurons, 629–32. Harvard University Press, 2015. http://dx.doi.org/10.4159/harvard.9780674735644.c23.
Pełny tekst źródła"References". W Molecular and Cellular Physiology of Neurons, 633–722. Harvard University Press, 2015. http://dx.doi.org/10.4159/harvard.9780674735644.c24.
Pełny tekst źródła"Index". W Molecular and Cellular Physiology of Neurons, 723–51. Harvard University Press, 2015. http://dx.doi.org/10.4159/harvard.9780674735644.c25.
Pełny tekst źródła"Vitamin B5: Pantothenate". W The Chemical Biology of Human Vitamins, 161–92. The Royal Society of Chemistry, 2018. http://dx.doi.org/10.1039/bk9781788014649-00161.
Pełny tekst źródłaStreszczenia konferencji na temat "Cellules C26"
Benedicto, Aitor, Joana Marquez, Elvira Olaso i Beatriz Arteta. "Abstract B10: LFA-1/ICAM-1 interaction switches on an orchestrated prometastatic microenvironmental shift during experimental liver metastasis of colon C26 cancer cells." W Abstracts: AACR Special Conference on Cellular Heterogeneity in the Tumor Microenvironment; February 26 — March 1, 2014; San Diego, CA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.chtme14-b10.
Pełny tekst źródłaGuagchinga Moreno, Genesis Yulisa, Andrea Carolina Serrano Larrea, Frank Alexis i Javier Santamaría. "Cellulose-based beads as a possible method for drug release in the treatment of Leishmaniasis disease". W VIII Congreso Internacional de Investigación REDU. Medwave, 2022. http://dx.doi.org/10.5867/medwave.2022.s1.ci26.
Pełny tekst źródłaAlande, C., i C. Landric. "Autotransplantation de germes dentaires au centre hospitalier de Pau : une série de cas". W 66ème Congrès de la SFCO. Les Ulis, France: EDP Sciences, 2020. http://dx.doi.org/10.1051/sfco/20206603008.
Pełny tekst źródłaKurnikova, Irina, Shirin Gulova, Guzal Akhmadullina, Natalia Danilina i Ikram Mokhammed. "Methods of computer simulation in the development of technology for the functional assessment of the state of the liver in patients". W 14th International Conference on Applied Human Factors and Ergonomics (AHFE 2023). AHFE International, 2023. http://dx.doi.org/10.54941/ahfe1003456.
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