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Artykuły w czasopismach na temat "Cellular Proliferation"
Yao, Guang. "Modelling mammalian cellular quiescence". Interface Focus 4, nr 3 (6.06.2014): 20130074. http://dx.doi.org/10.1098/rsfs.2013.0074.
Pełny tekst źródłaHatchell, D. L., T. McAdoo, S. Sheta, R. T. King i J. V. Bartolome. "Quantification of Cellular Proliferation in Experimental Proliferative Vitreoretinopathy". Archives of Ophthalmology 106, nr 5 (1.05.1988): 669–72. http://dx.doi.org/10.1001/archopht.1988.01060130731033.
Pełny tekst źródłaZhang, Jian Chun, Howard E. Savage, Peter G. Sacks, Thomas Delohery, R. R. Alfano, A. Katz i Stimson P. Schantz. "Innate cellular fluorescence reflects alterations in cellular proliferation". Lasers in Surgery and Medicine 20, nr 3 (1997): 319–31. http://dx.doi.org/10.1002/(sici)1096-9101(1997)20:3<319::aid-lsm11>3.0.co;2-8.
Pełny tekst źródłaCLARKE, CHRISTINE L., i ROBERT L. SUTHERLAND. "Progestin Regulation of Cellular Proliferation*". Endocrine Reviews 11, nr 2 (maj 1990): 266–301. http://dx.doi.org/10.1210/edrv-11-2-266.
Pełny tekst źródłaLenkala, Divya, Eric R. Gamazon, Bonnie LaCroix, Hae Kyung Im i R. Stephanie Huang. "MicroRNA biogenesis and cellular proliferation". Translational Research 166, nr 2 (sierpień 2015): 145–51. http://dx.doi.org/10.1016/j.trsl.2015.01.012.
Pełny tekst źródłaMankoff, David A., Anthony F. Shields i Kenneth A. Krohn. "PET imaging of cellular proliferation". Radiologic Clinics of North America 43, nr 1 (styczeń 2005): 153–67. http://dx.doi.org/10.1016/j.rcl.2004.09.005.
Pełny tekst źródłaVINCENT, P. C. "Leukemic Cellular Proliferation: A Perspective". Annals of the New York Academy of Sciences 459, nr 1 Hematopoietic (grudzień 1985): 308–27. http://dx.doi.org/10.1111/j.1749-6632.1985.tb20839.x.
Pełny tekst źródłaZlotorynski, Eitan, i Reuven Agami. "A PASport to Cellular Proliferation". Cell 134, nr 2 (lipiec 2008): 208–10. http://dx.doi.org/10.1016/j.cell.2008.07.003.
Pełny tekst źródłaVerdoorn, Cornelis. "Cellular Migration, Proliferation, and Contraction". Archives of Ophthalmology 104, nr 8 (1.08.1986): 1216. http://dx.doi.org/10.1001/archopht.1986.01050200122064.
Pełny tekst źródłaAbrisqueta, Pau, Neus Villamor, Ana Muntañola, Carles Codony, Mireia Camós, Eva Calpe, Maria Joao Baptista i in. "Biological Analysis and Prognostic Significance of Proliferative Cellular Compartment in Chronic Lymphocytic Leukemia (CLL)." Blood 114, nr 22 (20.11.2009): 667. http://dx.doi.org/10.1182/blood.v114.22.667.667.
Pełny tekst źródłaRozprawy doktorskie na temat "Cellular Proliferation"
Gan, Lisha. "Corneal cellular proliferation and wound healing /". Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4505-5/.
Pełny tekst źródłaKranc, Kamil. "The role of Cited2 in cellular proliferation". Thesis, University of Oxford, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.398233.
Pełny tekst źródłaSangfelt, Olle. "Effects of interferon on cellular proliferation and apoptosis /". Stockholm, 1998. http://diss.kib.ki.se/search/diss.se.cfm?19981014sang.
Pełny tekst źródłaStacy, Andrew Jared. "Regulation of ΔNp63α by TIP60 promotes cellular proliferation". Wright State University / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1596151919161674.
Pełny tekst źródłaChakravarthy, Usha. "The effect of gamma radiation on intraocular cellular proliferation". Thesis, Queen's University Belfast, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.317046.
Pełny tekst źródłaMaiti, Baidehi. "E2F and survivin - key players in cellular proliferation and transformation". Columbus, Ohio : Ohio State University, 2007. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1173801044.
Pełny tekst źródłaKhav, Eddie. "Visualizing an RB-E2F Cellular Switch that Controls Cell Proliferation". Thesis, The University of Arizona, 2013. http://hdl.handle.net/10150/297627.
Pełny tekst źródłaSimmons, Ambrosia. "The Role of Polarity Complex Proteins in Neural Progenitor Proliferation". Diss., Temple University Libraries, 2019. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/552083.
Pełny tekst źródłaPh.D.
Cortical malformations arise from defects in any stage of brain development and often result in life-long disability ranging from epilepsy to developmental delay and even perinatal lethality. The neuroepithelium of the emergent cortex lays the foundation on which the future cortex will develop, and as such, neuroepithelial tissue and the neural progenitor cells (NPCs) which comprise it are critical to the proper growth and development of the cortex. Here I demonstrate the significance of neuroepithelial cell polarity determinants in cortical development and how they affect both junctional integrity and the regulation of NPC proliferation leading to a variety of cortical malformations. Until now, the role of basal polarity complex protein Lgl1 in cortical development remained elusive due to perinatal lethality in animal models. To bypass this, we developed a novel conditional knockout mouse model of Lgl1 in the neuroepithelium and show that Lgl1 is essential to the maintenance of neuroepithelial integrity and regulation of NPC proliferation. Loss of Lgl1 results in a displaced ventricular zone with widespread ectopic proliferation resulting in severe periventricular nodular heterotopia (PNH). Furthermore, Lgl1 loss reduces the cell cycle length resulting in hyperproliferation leading to neuronal overproduction. Together, this work identifies a novel genetic cause of PNH. Next, I aimed to characterize the interaction of Lgl1 with other polarity proteins and downstream signaling pathways in cortical development. Apical and basal polarity proteins have demonstrated mutual antagonism in the establishment/maintenance of epithelial polarity; however, little is known about the role of this antagonism on cortical size and structure or the signaling pathways through which it acts. To address these questions we generated multiple genetic mouse models to investigate the opposing roles of basal protein, Lgl1, and either apical proteins Pals1 or Crb2. Concurrent loss of Pals1 and Lgl1 was able to prevent heterotopic nodules and increase proliferation compared to loss of Pals1 alone. However, cortical size was severely diminished due to overriding effects of Pals1 on cell survival that was unmitigated by Lgl1 loss. Remarkably, loss of both Crb2 and Lgl1 restored the cortex and hippocampus to near normal morphology with a profound rescue of cortical size, suggesting their essential antagonism in both cortical and hippocampal development. Importantly, genetic manipulation through reduction of YAP/TAZ expression in the Lgl1 CKO eliminates periventricular nodules and restores cortical thickness to that of WT cortices. This important finding implicates Lgl1 in the regulation of YAP/TAZ in cortical development. Finally, we investigated a possible downstream target of Pals1 in cell survival, BubR1. My work demonstrates that loss of Pals1 reduces BubR1 expression, which is an essential regulator of the mitotic checkpoint and causative gene of the human disorder Mosaic Variegated Aneuploidy. I show that loss of BubR1 results in significant apoptosis across all cell types in the cortex leading to microcephaly. These data provide the first link between cell polarity determinants and mitotic regulation in the cortex and suggests that BubR1 reduction likely contributes to the decreased cell survival following Pals1 loss. Overall these findings implicate impaired polarity complex function in a wide variety of NPC defects resulting in multiple cortical malformations. My work shows that polarity proteins regulate every stage of the NPCs life cycle from cell division and proliferation to cell survival through regulation of mitosis and YAP/TAZ signaling.
Temple University--Theses
Reed, Jennifer. "Interferon-gamma increases CD4+ T cell survival and proliferation". Click here for download, 2006. http://wwwlib.umi.com/cr/villanova/fullcit?p1432655.
Pełny tekst źródłaAnderson, Elizabeth. "Co-ordinate regulation of cellular proliferation and apoptosis in rodent liver". Thesis, University of Surrey, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.441719.
Pełny tekst źródłaKsiążki na temat "Cellular Proliferation"
Guest, Simon Sean. Strathmin is an intracellular regulator of cellular proliferation. Birmingham: University of Birmingham, 1996.
Znajdź pełny tekst źródłaRenato, Baserga, red. Biological regulation of cell proliferation. New York: Raven Press, 1986.
Znajdź pełny tekst źródłaJones, Neil Austin. The role of a major cytosolic protein in cellular proliferation. Birmingham: University of Birmingham, 1992.
Znajdź pełny tekst źródłaL, Boynton Alton, i Leffert H. L, red. Control of animal cell proliferation. Orlando: Academic Press, 1985.
Znajdź pełny tekst źródłaM, Veneziale Carlo, red. Control of cell growth and proliferation. New York, N.Y: Van Nostrand Reinhold, 1985.
Znajdź pełny tekst źródłariazi, Sheila. Pathophysiological links between impaired elastogenesis and increased cellular proliferation in development of cardiovascular disorders. Ottawa: National Library of Canada, 2002.
Znajdź pełny tekst źródłaBurton, Jean. A study of cellular proliferation rates in squamous cell carcinomas of the lung, with relation to p53 status. [S.l: The Author], 1994.
Znajdź pełny tekst źródłaMeridith, Alan T. Handbook of prostate cancer cell research: Growth, signalling, and survival. New York: Nova Biomedical Books, 2009.
Znajdź pełny tekst źródłaInternational Conference on Gene Regulation, Oncogenesis, and AIDS (1st 1989 Loutráki, Greece). Oncogenesis: Oncogenes in signal transduction and cell proliferation : papers delivered at the First International Conference on Gene Regulation, Oncogenesis, and AIDS, Loutraki, Greece, September 15-21, 1989. Redaktor Papas Takis S. Woodlands, Tex: Portfolio Pub. Co., 1990.
Znajdź pełny tekst źródłaWei, Dai, red. Checkpoint responses in cancer therapy. Totowa, NJ: Humana Press, 2008.
Znajdź pełny tekst źródłaCzęści książek na temat "Cellular Proliferation"
Brockhoff, Gero. "DNA and Proliferation Analysis by Flow Cytometry". W Cellular Diagnostics, 390–425. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000209173.
Pełny tekst źródłaMatatall, Katie A., Claudine S. Kadmon i Katherine Y. King. "Detecting Hematopoietic Stem Cell Proliferation Using BrdU Incorporation". W Cellular Quiescence, 91–103. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7371-2_7.
Pełny tekst źródłaJalbert, Emilie, i Eric M. Pietras. "Analysis of Murine Hematopoietic Stem Cell Proliferation During Inflammation". W Cellular Quiescence, 183–200. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7371-2_14.
Pełny tekst źródłaMierke, Claudia Tanja. "Cell Proliferation, Survival, Necrosis and Apoptosis". W Cellular Mechanics and Biophysics, 743–824. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-58532-7_16.
Pełny tekst źródłaDover, R. "Basic Methods for Assessing Cellular Proliferation". W Assessment of Cell Proliferation in Clinical Practice, 63–81. Tokyo: Springer Japan, 1992. http://dx.doi.org/10.1007/978-4-431-68287-5_4.
Pełny tekst źródłaDover, R. "Basic Methods for Assessing Cellular Proliferation". W Assessment of Cell Proliferation in Clinical Practice, 63–81. London: Springer London, 1992. http://dx.doi.org/10.1007/978-1-4471-3190-8_4.
Pełny tekst źródłaHoran, Paul Karl, Sue E. Slezak i Bruce D. Jensen. "Cellular Proliferation History by Fluorescent Analysis". W Flow Cytometry, 133–39. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-84616-8_8.
Pełny tekst źródłaGuerrieri, Ferruccio. "The F0F1-ATP Synthase in Cell Proliferation and Aging". W Frontiers of Cellular Bioenergetics, 677–92. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4843-0_27.
Pełny tekst źródłaHerbig, A. Katherine, Sameh Girgis i Patrick J. Stover. "Effects of Cellular Glycine on Cell Proliferation". W Chemistry and Biology of Pteridines and Folates, 491–94. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0945-5_83.
Pełny tekst źródłaMacieira-Coelho, Alvaro. "Slowing Down of the Cell Cycle During Fibroblast Proliferation". W Cellular Ageing and Replicative Senescence, 29–47. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26239-0_3.
Pełny tekst źródłaStreszczenia konferencji na temat "Cellular Proliferation"
Qian, Xu, He Hujun, Yang Guangtao i Yang Xu. "Effect of Formaldehyde on Cellular Proliferation of HEK293 Cells". W 2007 1st International Conference on Bioinformatics and Biomedical Engineering. IEEE, 2007. http://dx.doi.org/10.1109/icbbe.2007.122.
Pełny tekst źródłaDho, So Hee, Ji Young Kim, Chang-Jin Kim, William M. Nauseef, So-Young Choi, Kwang-Pyo Lee i Ki-Sun Kwon. "Abstract 2916: NOXX: Friend or foe for cellular proliferation." W Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-2916.
Pełny tekst źródłaBlahna, Matthew T., Matthew R. Jones, Lee J. Quinton i Joseph P. Mizgerd. "Zcchc11 Enhances Cellular Proliferation Independent Of Its Uridyltransferase Activity". W American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2124.
Pełny tekst źródła"The Effect of Hydroalcoholic Extract of Junipers communis on Proliferation BHK Cells". W International Conference on Cellular & Molecular Biology and Medical Sciences. Universal Researchers (UAE), 2016. http://dx.doi.org/10.17758/uruae.ae0916411.
Pełny tekst źródłaSavage, Howard E., Venkateswara Kolli, Jian C. Zhang, Robert R. Alfano, Peter G. Sacks i Stimson P. Schantz. "Tissue autofluorescence spectroscopy: in-vivo alterations may reflect cellular proliferation". W OE/LASE '94, redaktor Robert R. Alfano. SPIE, 1994. http://dx.doi.org/10.1117/12.175991.
Pełny tekst źródłaChung, Eunna, i M. N. Rylander. "Thermal Preconditioning Protocols for Cartilage Tissue Engineering". W ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193107.
Pełny tekst źródłaSolarte, Efrain, Hernan Urrea, William Criollo i Oscar Gutierrez. "LED illumination effects on proliferation and survival of meningioma cellular cultures". W BiOS, redaktorzy Valery V. Tuchin, Donald D. Duncan i Kirill V. Larin. SPIE, 2010. http://dx.doi.org/10.1117/12.843060.
Pełny tekst źródłaBlahna, Matthew T., Matthew R. Jones, Lee J. Quinton i Joseph P. Mizgerd. "The Uridyl-Transferase Enzyme Zcchc11 Prevents Senescence And Promotes Cellular Proliferation". W American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4926.
Pełny tekst źródłaShi, Caleb, Robert Chang i Donna Leonardi. "The Effects of Mechanical Vibration on Cellular Health in Differentiated Neuroblastoma Cells". W ASME 2018 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/detc2018-86280.
Pełny tekst źródłaNeish, Andrew Scott. "Abstract PL01-02: Influence of the microbiota on cellular proliferation and survival". W Abstracts: Thirteenth Annual AACR International Conference on Frontiers in Cancer Prevention Research; September 27 - October 1, 2014; New Orleans, LA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1940-6215.prev-14-pl01-02.
Pełny tekst źródłaRaporty organizacyjne na temat "Cellular Proliferation"
Sun, Lina, Yanan Han, Hua Wang, Huanyu Liu, Shan Liu, Hongbin Yang, Xiaoxia Ren i Ying Fang. MicroRNAs as Potential Biomarkers for the Diagnosis of Inflammatory Bowel Disease: A Systematic Review and Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, luty 2022. http://dx.doi.org/10.37766/inplasy2022.2.0027.
Pełny tekst źródłaEldar, Avigdor, i Donald L. Evans. Streptococcus iniae Infections in Trout and Tilapia: Host-Pathogen Interactions, the Immune Response Toward the Pathogen and Vaccine Formulation. United States Department of Agriculture, grudzień 2000. http://dx.doi.org/10.32747/2000.7575286.bard.
Pełny tekst źródłaZhou, Ting, Roni Shapira, Peter Pauls, Nachman Paster i Mark Pines. Biological Detoxification of the Mycotoxin Deoxynivalenol (DON) to Improve Safety of Animal Feed and Food. United States Department of Agriculture, lipiec 2010. http://dx.doi.org/10.32747/2010.7613885.bard.
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