Gotowe bibliografie tematyczne / Cell ultrastructure

Gotowa bibliografia na temat „Cell ultrastructure”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 4 lutego 2022

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Artykuły w czasopismach na temat "Cell ultrastructure"

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Hegewald, Eberhard, Seon Sook An, Eberhard Schnepf i Peter Tsarenko. "Taxonomy and cell wall ultrastructure of Scenedesmus lunatus (Chlorophyta, Chlorococcales)". Algological Studies/Archiv für Hydrobiologie, Supplement Volumes 91 (30.11.1998): 11–25. http://dx.doi.org/10.1127/algol_stud/91/1998/11.

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Kimbrough, James W., i Jack L. Gibson. "Ultrastructural and cytological observations of apothecial tissues of Geopyxis carbonaria (Pezizales, Ascomycetes)". Canadian Journal of Botany 68, nr 2 (1.02.1990): 243–57. http://dx.doi.org/10.1139/b90-034.

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Cytological observations are made on apothecial tissues of Geopyxis carbonaria, using transmission electron microscopy. Characteristic features of both the medullary and ectal excipula are examined. Changes in ascus apex and wall structures are examined during ascus ontogeny, especially in relation to operculum position and structure. Ultrastructure of septum configuration is observed and compared in the excipulum, ascogenous hyphae, paraphyses, and at the base of young asci. Ascosporogenesis is observed from the ascus mother cell stage and initial spore delimitation until secondary wall formation. The cytological and ultrastructural observations on this species are discussed in relation to their possible taxonomic or phylogenetic value. Key words: ascosporogenesis, Discomycetes, ascospore ultrastructure, septal ultrastructure, cytochemistry.
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Popov, V. L., A. A. Shatkin, V. N. Pankratova, N. S. Smirnova, C. H. Bonsdorff, M. R. Ekman, A. Mörttinen i P. Saikku. "Ultrastructure ofChlamydia pneumoniaein cell culture". FEMS Microbiology Letters 84, nr 2 (listopad 1991): 129–34. http://dx.doi.org/10.1111/j.1574-6968.1991.tb04584.x.

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Henrik, Per, i Becher Carstens. "ULTRASTRUCTURE OF GRANULAR CELL MYOBLASTOMA". Acta Pathologica Microbiologica Scandinavica Section A Pathology 78A, nr 6 (15.08.2009): 685–94. http://dx.doi.org/10.1111/j.1699-0463.1970.tb03521.x.

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Vollenweider, Iren, i P. Groscurth. "Ultrastructure of cell mediated cytotoxicity". Electron Microscopy Reviews 4, nr 2 (styczeń 1991): 249–67. http://dx.doi.org/10.1016/0892-0354(91)90005-w.

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Bonarski, Jan T., Girma Kifetew i Wiesław Olek. "Effects of cell wall ultrastructure on the transverse shrinkage anisotropy of Scots pine wood". Holzforschung 69, nr 4 (1.05.2015): 501–7. http://dx.doi.org/10.1515/hf-2014-0075.

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Abstract A hypothesis for explaining the differential anisotropic shrinkage behavior of wood has been proposed, and it was based on the differences in the cell wall ultrastructure. The starting point of the consideration is that wood shrinkage is governed by its chemical composition, ultrastructure, and gross anatomy. It is also well known that the transverse shrinkage anisotropy of earlywood (EW) is more pronounced than that of the latewood (LW). In the paper, the cell wall ultrastructure and shrinkage anisotropy has been related to each other, and to this purpose, a set of crystallographic texture descriptors was applied. The descriptors are based on X-ray diffraction (XRD) experiments conducted on matched EW samples from different growth rings of Scots pine. The range of the microfibril angle (MFA) was identified. The ratio of the maxima of inverse pole figures (IPFs) of both the tangential (T) and radial (R) directions was determined. The ratios quantify the inhomogeneity of the spatial arrangement of the ordered areas. The results of the study clearly indicate that the transverse shrinkage of wood is governed mostly by a specific ultrastructural organization of moderately organized cell wall compounds.
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Saucedo, José Edmundo Nava, Jean-Noël Barbotin, Martine Velut i Daniel Thomas. "Ultrastructural examination of Gibberella fujikuroi mycelia: effect of immobilization in calcium alginate beads". Canadian Journal of Microbiology 35, nr 12 (1.12.1989): 1118–31. http://dx.doi.org/10.1139/m89-187.

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Ultrastructural examination of free and calcium alginate immobilized Gibberella fujikuroi mycelia showed that in addition to the changes occurring during the transition phase from primary to secondary metabolism, there are several alterations in the ultrastructure of hyphae as a response to microenvironmental changes owing to immobilization constraints. Internal changes included (i) the presence of large glyoxisomelike bodies and of active vesicle-generating systems, which appeared as cloudy structures in electron micrographs; (ii) the formation of endocells, resulting in hyphae with up to three cell walls and the concomitant accumulation of secondary metabolites, mainly pigments, in peripheral cell compartments; and (iii) the progressive development of autophagic vacuoles involved in the turnover of cell constituents.Key words: Gibberella fujikuroi, immobilized fungi, alginate entrapment, ultrastructure modification, immobilization.
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Khursigara, Cezar M., Susan F. Koval, Dianne M. Moyles i Robert J. Harris. "Inroads through the bacterial cell envelope: seeing is believing". Canadian Journal of Microbiology 64, nr 9 (wrzesień 2018): 601–17. http://dx.doi.org/10.1139/cjm-2018-0091.

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A singular feature of all prokaryotic cells is the presence of a cell envelope composed of a cytoplasmic membrane and a cell wall. The introduction of bacterial cell fractionation techniques in the 1950s and 1960s along with developments in procedures for electron microscopy opened the window towards an understanding of the chemical composition and architecture of the cell envelope. This review traces the contribution of Terry Beveridge in these endeavours, beginning with his doctoral studies in the 1970s on the structure of paracrystalline surface arrays (S-layers), followed by an exploration of cryogenic methods for preserving bacteria for ultrastructural analyses. His insights are reflected in a current example of the contribution of cryo-electron microscopy to S-layer studies — the structure and assembly of the surface array of Caulobacter crescentus. The review then focuses on Terry’s contributions to imaging the ultrastructure of bacterial cell envelopes and to the development of cryo-electron microscopy techniques, including the use of CEMOVIS (Cryo-electron Microscopy of Vitreous Sections) to “see” the ultrastructure of the Gram-positive cell envelope — his last scientific endeavour.
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Šmarda, Jan. "Cell ultrastructure changes accompanying the annual life cycle of the cyanobacterium Microcystis aeruginosa". Algological Studies 130 (1.10.2009): 27–38. http://dx.doi.org/10.1127/1864-1318/2009/0130-0027.

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SITTART, J. "Ultrastructure of skin large cell acanthoma". Journal of the European Academy of Dermatology and Venereology 11 (wrzesień 1998): S218. http://dx.doi.org/10.1016/s0926-9959(98)95381-8.

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Rozprawy doktorskie na temat "Cell ultrastructure"

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Chao, Ying L. (Ying Liang). "Ultrastructure of Azotobacter vinelandii". Thesis, North Texas State University, 1993. https://digital.library.unt.edu/ark:/67531/metadc798461/.

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The purpose of this research was to reveal the morphological and cytological characteristics of Azotobacter vinelandii cells cultured in dialyzed soil medium. Culture samples taken at two, four, eight, sixteen and thirty-two days were prepared and examined with the electron microscope. Comparisons of the morphology of Azotobacter vinelandii grown in dialyzed soil medium with those grown in Burk's nitrogen-free, chemically-defined medium were done.
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Goss, Steven Philip Allan. "Ultrastructure and Mitosis of Glaucosphaera vacuolata". W&M ScholarWorks, 1993. https://scholarworks.wm.edu/etd/1539625804.

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Hudson, Liam. "Ultrastructure of the A-band unit cell in relaxed muscle". Thesis, Imperial College London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.310340.

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Ridden, John. "Studies on the cell biology of the human sebaceous gland". Thesis, University of Oxford, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.258034.

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Fahlén, Jesper. "The cell wall ultrastructure of wood fibres : effects of the chemical pulp fibre line". Doctoral thesis, KTH, Fiber- och polymerteknik, 2005. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-129.

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Knowledge of the ultrastructural arrangement within wood fibres is important for understanding the mechanical properties of the fibres themselves, as well as for understanding and controlling the ultrastructural changes that occur during pulp processing. The object of this work was to explore the use of atomic force microscopy (AFM) in studies of the cell wall ultrastructure and to see how this structure is affected in the kraft pulp fibre line. This is done in order to eventually improve fibre properties for use in paper and other applications, such as composites. On the ultrastructural level of native spruce fibres (tracheids), it was found that cellulose fibril aggregates exist as agglomerates of individual cellulose microfibrils (with a width of 4 nm). Using AFM in combination with image processing, the average side length (assuming a square cross-section) for a cellulose fibril aggregate was found to be 15–16 nm although with a broad distribution. A concentric lamella structure (following the fibre curvature) within the secondary cell wall layer of native spruce fibres was confirmed. These concentric lamellae were formed of aligned cellulose fibril aggregates with a width of about 15 nm, i.e. of the order of a single cellulose fibril aggregate. It was further found that the cellulose fibril aggregates had a uniform size distribution across the fibre wall in the transverse direction. During the chemical processing of wood chips into kraft pulp fibres, a 25 % increase in cellulose fibril aggregate dimension was found, but no such cellulose fibril aggregate enlargement occurred during the low temperature delignification of wood into holocellulose fibres. The high temperature in the pulping process, over 100 ºC, was the most important factor for the cellulose fibril aggregate enlargement. Neither refining nor drying of kraft or holocellulose pulp changed the cellulose fibril aggregate dimensions. During kraft pulping, when lignin is removed, pores are formed in the fibre cell wall. These pores were uniformly distributed throughout the transverse direction of the wood cell wall. The lamellae consisting of both pores and matrix material (“pore and matrix lamella”) became wider and their numeral decreased after chemical pulping. In holocellulose pulp, no such changes were seen. Refining of kraft pulp increased the width of the pore and matrix lamellae in the outer parts of the fibre wall, but this was not seen in holocellulose. Upon drying of holocellulose, a small decrease in the width of the pore and matrix lamellae was seen, reflecting a probable hornification of the pulp. Refining of holocellulose pulp led to pore closure probably due to the enhanced mobility within the fibre wall. Enzymatic treatment using hemicellulases on xylan and glucomannan revealed that, during the hydrolysis of one type of hemicellulose, some of the other type was also dissolved, indicating that the two hemicelluloses were to some extent linked to each other in the structure. The enzymatic treatment also decreased the pore volume throughout the fibre wall in the transverse direction, indicating enzymatic accessibility to the entire fibre wall. The results presented in this thesis show that several changes in the fibre cell wall ultrastructure occur in the kraft pulp fibre line, although the effects of these ultrastructural changes on the fibre properties are not completely understood.
QC 20101012
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Fahlén, Jesper. "The cell wall ultrastructure of wood fibres : effects of the chemical pulp fibre line /". Stockholm : Fibre and Polymer technology, KTH, 2005. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-129.

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Stander, Cornelia Steynberg. "An ultrastructural and light microscopic study of melanocyte differentiation in chick embryos". Master's thesis, University of Cape Town, 1991. http://hdl.handle.net/11427/27149.

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The embryonic source and chemical nature of those factor/s directing the in vivo differentiation of melanocytes from crest cells are as yet unknown. To begin to address this issue, it is important to establish exactly when and where these signa/s first exert their effects. Therefore, in the present study, overtly differentiated melanocytes containing melanin were quantitated in developing Black Australorp X New Hampshire Red chick embryos. In contrast to previous studies, it was found that embryos synthesize melanin from as early as Day 5 of development, and that at this stage, the melanocytes are predominantly dermally located. Between 5 and 8 days, the numbers of both dermal and epidermal melanocytes increase, after which the dermal melanocyte population declines rapidly while the number of epidermal melanocytes continues to increase. These findings suggest that premelanocytes do not have to be epidermally located to initiate terminal differentiation and implicate the dermis as a possible source of melanocyte inducing factor/s. The next step was to examine stages of development prior to the onset of pigment production. For this reason, tyrosinase was purified for use as antigen in the production of a polyclonal antibody. The antibody was tested for specificity by western blotting, - immunocytochemistry and immunoinhibition procedures. Lack of specificity was demonstrated, rendering it unsuitable as an antibody marker for early melanocytes. Fowl melanocytes are thought to differentiate into either eumelanosome- or pheomelanosome synthesizing cells. To test the validity of this concept, embryonic skin of the red/black cross breed were screened for possible mixed type melanocytes by electron microscopy. The melanocytes contained melanosomes with a matrix of irregularly arranged filaments amongst typical eumelanogenic melanosomes. This suggests that these chick melanocytes may synthesize both eumelanosomes and pheomelanosomes in single cells. In a further study on pure breeding New Hampshire Reds, it was found that the melanocytes contained a mixture of typical and less typical pheomelanosomes. Outer membrane indentations in the latter melanosome type suggest that tyrosinase may enter these pheomelanosomes by a mechanism related to that proposed for the melanosomes of goldfish.
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au, jeremy shaw@uwa edu, i Jeremy Shaw. "Biomineralisation processes in the radula teeth of the chiton Acanthopleura hirtosa (Mollusca: Polyplacophora)". Murdoch University, 2007. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20080703.163505.

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A detailed row by row investigation of major lateral tooth cusp mineralisation, together with the concomitant development of the superior epithelial tissue surrounding the teeth of the chiton Acanthopleura hirtosa has been undertaken using a combination of light microscopy, and scanning and transmission electron microscopy. A holistic approach has been adopted that encompasses observations over a range of spatial scales, from whole radula mineralisation processes to those occurring within individual tooth cusps at various stages of development. In addition, mineralisation in radulae from freshly collected animals has been compared to that of animals maintained for extensive periods within a newly developed iron limited system, which restricts radula mineralisation without impeding the formation of the organic matrix. An evaluation of the iron limitation technique has revealed that maintaining specimens of A. hirtosa within an iron poor environment results in a significant departure from the normal pattern of mineralisation in these animals. As a consequence of iron limitation, there is an obvious increase in the number of unmineralised tooth rows in addition to associated alterations in structure and composition at all stages of tooth development. In normal specimens of A. hirtosa, the onset of mineralisation in the tooth cusps occurs following the prior accumulation of iron at the junction zone and the sudden accumulation of iron containing granules in the cusp epithelium at tooth row 13. The superior epithelium surrounding the tooth cusps undergoes a series of developmental changes leading up to, and following, the onset of mineralisation. In particular, the abundance of mitochondria within the apical cusp epithelium increases, presumably in order to provide the ideal conditions of pH, and thus solubility, needed for the supersaturation of iron and its nucleation at row 13. Once mineralisation has commenced, the microvilli attached to the cusps develop rapidly, and are suggested to do so in order to facilitate the transport of iron, and thereby ensure that a high concentration gradient of this element into the cusps is maintained. The delivery of iron into the cusps occurs from two fronts, the first from the superior epithelium via the posterior surface, and the second from the junction zone via an internal pathway situated along the lepidocrocite boundary between the magnetite and core regions of the tooth. The existence of a plume of elements between this internal mineralisation pathway and the junction zone, provides the first direct evidence that the junction zone is involved in the storage and release of elements for cusp mineralisation. Data from iron limited radulae also indicate that iron continues to be deposited at the junction zone in preference to the superior epithelium or cusps, despite the disruption of mineralisation, highlighting the importance of this region in the mineralisation process. Iron reinstatement experiments have also shown that the internal pathways of iron delivery within the organic matrix remain viable, despite prolonged periods of iron limitation. In addition, the reinstatement of iron has revealed that the plumes, situated between the junction zone and internal mineralising pathway of the cusp, stem from the centre of the plate like junction zone, directly above the stylus canal, a tube like cavity situated within the styli of each major lateral tooth. An in depth study of the stylus canal has revealed that cells within the canal are remarkably similar to those of the epithelium surrounding the cusps, suggesting that this structure may also be involved in the delivery of ions to the junction zone. The stylus canal is shown to be present in the major lateral tooth cusps of 38 chiton species distributed worldwide, and is therefore likely to be a feature common to all chitons. The presence of the canal, and indeed its absence from the bases of all remaining non iron mineralised teeth, irrespective of chiton species, also points strongly to a functional relationship between the stylus canal and tooth cusp mineralisation.
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Merced-Alejandro, Amelia. "Evolution of stomata in mosses (Bryophyta): From molecules to form and function". OpenSIUC, 2015. https://opensiuc.lib.siu.edu/dissertations/1038.

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As one of the first land plant groups to diversify, mosses are central in understanding the origin, diversification, and early function of stomata. Unlike tracheophytes that have stomata on anatomically complex leaves and stems, mosses bear stomata exclusively on spore-bearing organs (capsules). However, stomata do not occur in all mosses and, indeed, are absence in the earliest-divergent mosses (Takakia, Andreaea, Andreaeobryum and Sphagnum), suggesting that stomata originated in mosses independently of other plants. The occurrence of structurally unique pseudostomata in Sphagnum further confounds the resolution of homology of moss stomata with those of other plants. The five studies included in this dissertation are aimed at clarifying the structure, development and evolution of moss stomata. The first study focuses on the sporophyte anatomy and stomatal ultrastructure in two structurally and phylogenetically divergent mosses, Oedipodium and Ephemerum. Oedipodium is the sister to peristomate mosses and the first extant moss with true stomata. This monospecific genus has an elaborated capsule with an extended apophysis bearing numerous long-pored stomata. In contrast, Ephemerum nests within the peristomate mosses and has a reduced capsule that lacks an apophysis and has a few round-pored stomata. Ultrastructure of stomata is similar in these two mosses and comparable to that of tracheophytes, except that the stomata of mosses are not as structurally distinct from epidermal cells as are tracheophyte stomata. Anatomical features such as the presence of a cuticle, water-conducting cells, and spongy tissues with large areas for gas exchange are more pronounced in Oedipodium sporophytes and support the role of stomata in gas exchange and water transport during development and maturation. The second study examines changes in pectin composition during development in the model moss Funaria. Stomatal movement in tracheophytes requires guard cell walls to be strong, yet flexible, because they have to undergo reversible deformation to open and close the pore. Pectins are necessary for wall flexibility and proper stomatal functioning in seed plants. In this study of Funaria, immunogold-labeling using five antibodies to pectin epitopes was conducted on guard cell walls during development to relate these features to the limited movement of stomata in moss. Movement of Funaria stomata coincides with capsule expansion when guard cell walls are thin and pectinaceous. Walls dramatically increase in thickness after pore formation and the pectin content significantly decreases in mature guard cell walls, suggesting that a decrease in flexibility is responsible for the inability to open a close previously reported in older moss guard cells. Because this was the first study to demonstrate changes in pectin composition during stomatal development in any plant, a similar study was done on Arabidopsis to identify the main types of pectins in guard cell walls. Localization of pectins in guard cell walls of Arabidopsis is similar to mosses in the stage they can move, with homogeneous walls rich in arabinan pectins that are required for wall flexibility. This study extends knowledge of pectin composition from stomata of the moss Funaria with limited stomatal movement to an angiosperm in which stomatal activity is crucial to the physiological health of the plant. The fourth study describes stomata development and internal changes in sporophyte anatomy that lead to formation of air spaces in the moss Funaria. Developing sporophytes at different stages were examined using light, fluorescence and electron microscopy; immunogold-labeling was used to investigate the presence of pectin in the newly formed cavities. Stomata in mosses do not develop from a self-generating meristemoid like in Arabidopsis, but instead they originate from a protodermal cell that differentiates directly into a guard mother cell. Epidermal cells develop from protodermal or other epidermal cells, i.e., there are no stomatal lineage ground cells. This developmental pattern is congruent with the presence of a gene ortholog of FAMA, but not SPCH and MUTE, in Physcomitrella. The final study in this dissertation focuses on the enigmatic Sphagnum. Although true stomata are absent in early-divergent mosses, Sphagnum has specialized epidermal cells, pseudostomata, that partially separate but do not open to the inside. To further understand the structure, function and evolution of pseudostomata, capsule anatomy and ultrastructure of pseudostomata were detailed. As in moss stomata, pseudostomata wall architecture and behavior facilitate capsule dehydration, shape change, and dehiscence, supporting this common function. Unlike other moss stomata, pseudostomata collapse along their ventral walls and they lack a substomatal cavity. Similarities to true stomata include two modified epidermal cells with specialized cell walls that separate by cuticle deposition and respond to drying. Pseudostomata may be interpreted as modified stomata that suppressed substomatal cavity formation, which in turn eliminated pore development. However, clarification of the homology of pseudostomata and moss stomata will require genomic studies integrated with physiological and structural data. The studies described in this dissertation significantly advance our understanding of moss stomatal development and structure, and provide a comparison point to better evaluate the evolution of stomata. Moss capsule anatomy coupled with the exclusive existence of stomata on capsules supports the concept that stomata in moss are involve in gas exchange but also facilitate drying and dispersal of spores. Changes in wall architecture coupled with a decrease in total pectin explain the inability of mature stomata to move. Development and distribution of stomata in Funaria provides evidence of a direct and less elaborated mechanism for stomatal development than described in Arabidopsis. Resolving relationships among early land plants, especially hornworts and mosses, the only bryophyte groups with stomata, is critical to understanding stomata evolution. Evaluated together, the results of this dissertation are consistent with a single origin of stomata in land plants.
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Kamakura, Tsukasa. "Ultrastructural Maturation of Human-Induced Pluripotent Stem Cell-Derived Cardiomyocytes in a Long-Term Culture". Kyoto University, 2015. http://hdl.handle.net/2433/199202.

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Książki na temat "Cell ultrastructure"

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Cell ultrastructure. Belmont, Calif: Wadsworth Pub. Co., 1985.

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Bubel, Andreas. Microstructure and function of cells: Electron micrographs of cell ultrastructure. Chichester, West Sussex, England: E. Horwood, 1989.

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Lynne, Mercer K., red. Cell and tissue ultrastructure: A functional perspective. New York: W.H. Freeman, 1993.

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service), ScienceDirect (Online, red. Methods in nano cell biology. Amsterdam: Academic Press, 2008.

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Gunning, Brian Edgar Scourse. Plant cell biology: An ultrastructural approach. Dublin: M.W.Steer, 1986.

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Gunning, Brian E. S. Plant cell biology: Structure and function. Boston, Mass: Jones and Bartlett Publishers, 1996.

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W, Steer Martin, red. Plant cell biology: Structure and function. Sudbury, Mass: Jones and Bartlett Publishers, 1996.

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1943-, Graham J. M., red. Membrane structure and function. Oxford, England: IRL Press at Oxford University Press, 1989.

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Kouwets, Frans. The cell cycle in multinucleate coccoid green algae: Ultrastructure & systematics. Leiden: Rijksherbarium/Hortus Botanicus, 1994.

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Ultrastructural pathology of the cell and matrix. Wyd. 4. Boston: Butterworth-Heinemann, 1997.

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Części książek na temat "Cell ultrastructure"

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Pavelka, Margit, i Jürgen Roth. "Cell-Cell and Cell-Matrix Contacts and Disorders". W Functional Ultrastructure, 196–221. Vienna: Springer Vienna, 2015. http://dx.doi.org/10.1007/978-3-7091-1830-6_13.

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Pavelka, Margit, i Jürgen Roth. "Selectin — Ligand-Mediated Cell-Cell Interaction". W Functional Ultrastructure, 174–75. Vienna: Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99390-3_91.

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Pavelka, Margit, i Jürgen Roth. "Brush Cell". W Functional Ultrastructure, 158–59. Vienna: Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99390-3_83.

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Pavelka, Margit, i Jürgen Roth. "Liver: Hepatocytes, Kupffer cell, Cell of Ito". W Functional Ultrastructure, 214–15. Vienna: Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99390-3_111.

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Pavelka, Margit, i Jürgen Roth. "Eosinophilic Granulocyte, Plasma Cell, Macrophage, Mast Cell". W Functional Ultrastructure, 280–81. Vienna: Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99390-3_144.

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Pavelka, Margit, i Jürgen Roth. "I-Cell Disease". W Functional Ultrastructure, 110–11. Vienna: Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99390-3_57.

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Pavelka, Margit, i Jürgen Roth. "Glycocalyx (Cell Coat)". W Functional Ultrastructure, 160–61. Vienna: Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99390-3_84.

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Pavelka, Margit, i Jürgen Roth. "Mitosis and Cell Division". W Functional Ultrastructure, 20–21. Vienna: Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99390-3_11.

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Pavelka, Margit, i Jürgen Roth. "Umbrella Cell — Surface Specialisations". W Functional Ultrastructure, 250–51. Vienna: Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99390-3_129.

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Pavelka, Margit, i Jürgen Roth. "Umbrella Cell — Fusiform Vesicles". W Functional Ultrastructure, 252–53. Vienna: Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99390-3_130.

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Streszczenia konferencji na temat "Cell ultrastructure"

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Rodemer, Claus, Jürgen Jenne, Marc Fatar, Michael G. Hennerici i Stephen Meairs. "Effects of ultrasound upon endothelial cell ultrastructure". W 12TH INTERNATIONAL SYMPOSIUM ON THERAPEUTIC ULTRASOUND. AIP, 2012. http://dx.doi.org/10.1063/1.4769908.

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Backman, V., H. Subramanian, P. Pradhan, Y. Liu, I. Capoglu, J. D. Rogers, H. K. Roy i A. Taflove. "Detecting alterations in cell ultrastructure with optical imaging". W 2009 Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2009. http://dx.doi.org/10.1109/iembs.2009.5333173.

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Hu, Shaoshan, Ruyou Zhang i Yongri Zheng. "Photodynamic therapy on the ultrastructure of glioma cell". W 2004 Shanghai international Conference on Laser Medicine and Surgery, redaktor Jing Zhu. SPIE, 2005. http://dx.doi.org/10.1117/12.639223.

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Francis, John Paul, Hongzhi Wang, Kate White, Tanveer Syeda-Mahmood i Raymond Stevens. "Neural Network Segmentation of Cell Ultrastructure Using Incomplete Annotation". W 2020 IEEE 17th International Symposium on Biomedical Imaging (ISBI). IEEE, 2020. http://dx.doi.org/10.1109/isbi45749.2020.9098739.

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Ford, Thomas W., Anton M. Page, Guy F. Foster i Anthony D. Stead. "Effects of soft x-ray irradiation on cell ultrastructure". W San Diego '92, redaktorzy Chris J. Jacobsen i James E. Trebes. SPIE, 1993. http://dx.doi.org/10.1117/12.138748.

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Dalby Kristensen, S., K. M. Roberts, J. Lawry i J. F. Martin. "SHORT TERM HIGH CHOLESTEROL DIET CAUSES CHANGES IN MEGAKARYOCYTE SIZE AND IN VASCULAR ULTRASTRUCTURE". W XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643411.

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Platelets produced by megakaryocytes (MK) have a role in atherogenesis. Six pairs of male litter mate rabbits were randomised to feeding with either 2g of cholesterol daily in addition to their normal diet or normal diet alone. After seven days the animals were killed and serum cholesterol, platelet count, MK total, cytoplasmic and nuclear area (microscopic planimetry) and MK DNA content cell distribution (fluorescent activated cell sorting) were measured and compared between the two groups. The results are given in the table as medians with range values in brackets.After perfusion-fixation the aortas were examined by transmission electron microscopy. In the aortas from the animals on high cholesterol diet cells with ultrastructural features resembling smooth muscle cells were found in the intima. Changes in megakaryocyte size are associated with the occurrence of smooth muscle cell proliferation and migration. The platelet-megakaryocyte axis may be activated in early atherogenesis.
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Shannon, Garrett, Tyler Novak, Sherry L. Voytik-Harbin i Corey P. Neu. "Temperature Control During Fibrillogenesis Allows for Improved Magnetic Alignment of Collagen". W ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14314.

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Osteoarthritis (OA), commonly known as ‘wear and tear’ in human joints, affects over 27 million people in the United States [1]. There is currently no encompassing solution for the regeneration of damaged articular cartilage. One potential solution involves the close emulation of the native structure of articular cartilage, with special consideration given to maintaining the distinct organized zonal ultrastructure, characterized by both random and highly aligned zones of collagen fibrils, in order to preserve mechanical and cell signaling properties of the extracellular matrix [2]. Techniques such as electrospinning achieve high degrees of alignment, but do so at the cost of denaturing the collagen molecule [3] that may lead to inferior cell recognition and mechanical strength.
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Wilson, Christopher G., i Marc E. Levenston. "Chondrocytes and Fibrochondrocytes Differentially Process Aggrecan During De Novo Extracellular Matrix Assembly". W ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176669.

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The material properties of articular cartilage and meniscal fibrocartilage depend on the composition and ultrastructure of the extracellular matrix (ECM). Aggrecan is the predominant large proteoglycan in these tissues, and confers compressive stiffness through immobilization of negatively charged sulfated glycosaminoglycans (sGAG). The abundance of sGAG is in part regulated by cell-mediated proteolysis of the aggrecan core protein, and transforming growth factor-β (TGF-β) family cytokines upregulate aggrecan synthesis in chondrocytes and fibrochondrocytes. The function(s) of aggrecan and mechanisms of aggrecan processing in the meniscus, however, are not well understood. The objective of this study was to examine tissue-specific kinetics and mechanisms of TGF-β-induced aggrecan turnover using the cell-agarose culture system. In addition, the tissue-specific functional implications of increased proteoglycan production were evaluated in terms of construct material properties.
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Novak, Tyler, Adam Griebel i Corey P. Neu. "Strains in Magnetically Aligned Collagen Scaffolds as Determined by Displacement-Encoded MRI". W ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80897.

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Collagen is the most abundant structural protein in a broad range of tissues, including ligament, cartilage, and bone. Collagen-based tissue constructs, including those with defined fibril alignment, have shown considerable potential for repair and regeneration of diseased collagen-rich tissues [1,2], where fibril ultrastructure of constructs emulate the native tissue counterpart. Magnetic alignment of collagen is an established method to control fiber orientation without undesirable changes in molecular stability and structure [5]. To date, studies concerning the magnetic alignment of collagen have shown the ability of fibril alignment to drive preferential cell growth characteristics [6]. While the bulk mechanical characteristics due to alignment has been investigated in our laboratory (data not shown), the mechanical strain throughout the interior of the scaffolds has not been investigated. Knowledge of internal strains is required to determine the degree that fiber alignment affects spatiotemporal mechanics of microenvironments within the collagen structures. Spatially-dependent mechanics, which could be tailored based on unique fabrication methods that control fibril alignment in three dimensions, would be expected to directly influence local cell-matrix interactions.
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Zhou, Xiaozhou, Bin Wang, Christopher Price, Wen Li, Jun Pan i Liyun Wang. "Investigating the Sieving and Structural Property of the Osteocyte Pericellular Matrix: Experiments and Modeling". W ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80307.

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Growing evidence shows that osteocytes, the most abundant bone cells, serve as the primary sensory cells that detect external mechanical forces [1], enabling the bone to adapt its mass and structure to meet its environmental requirements and to fulfill its weight bearing functions [2]. Although the cellular and molecular mechanisms of such adaptation phenomena are not fully understood, recent experiments and theoretical models suggest that the pericellular matrix (PCM) filling the tiny gap between the cell membrane and the canalicular matrix wall plays a critical role in the osteocytes’ outside-in signaling process [1]. Weinbaum first hypothesized that a proteoglycan-like fiber matrix, similar to the endothelial glycocalyx, must exist within the PCM to account for the surprisingly long relaxation times of the strain-generated potentials measured in bone [3]. Such a filling matrix was predicted to impose hydraulic resistance, impede fluid pressure relaxation and reduce fluid flow in the tiny lacunar-canalicular pore system in bone, thus protecting the cell membranes from being ruptured under shear. Later electronic microscopic studies confirmed the existence and the proteoglycan nature of the PCM [4]. Previous models using idealized PCM ultrastructure suggested that the hydrodynamic interactions between the PCM and fluid could determine the magnitude of drag forces that deform cytoskeleton via tethered transmembrane components or the focal contacts containing integrins [5,6]. In both scenarios, the PCM is the key to force transmission and strain signal amplification, and responsible for downstream mechanotransduction. In addition, once the mechanically excited osteocytes affect the release of molecular signals such as ATP, NO, PGE2, OPG, RANKL, and sclerostin [2], the PCM, as a molecular sieve and temporary storage, may influence the transport and availability of these bioactive molecules [7]. Therefore, the structural and sieving properties of PCM are important in regulating bone’s mechanotransduction and adaptation. However, due to the small dimensions of the PCM (∼100nm thick) and the difficulty in preserving the PCM in situ, its detailed structure and properties have remained elusive [4]. The objective of this study was to elucidate the sieving property of the PCM in mechanically loaded bone with an innovative imaging approach and to further deduce plausible PCM structures using mathematical modeling.
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