Gotowa bibliografia na temat „Cell cycle progression”
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Artykuły w czasopismach na temat "Cell cycle progression"
Braun-Dullaeus, Ruediger C., Michael J. Mann i Victor J. Dzau. "Cell Cycle Progression". Circulation 98, nr 1 (7.07.1998): 82–89. http://dx.doi.org/10.1161/01.cir.98.1.82.
Pełny tekst źródłaTyrcha, Joanna. "Cell cycle progression". Comptes Rendus Biologies 327, nr 3 (marzec 2004): 193–200. http://dx.doi.org/10.1016/j.crvi.2003.05.002.
Pełny tekst źródłaAssoian, Richard K. "Anchorage-dependent Cell Cycle Progression". Journal of Cell Biology 136, nr 1 (13.01.1997): 1–4. http://dx.doi.org/10.1083/jcb.136.1.1.
Pełny tekst źródłaGiacinti, C., i A. Giordano. "RB and cell cycle progression". Oncogene 25, nr 38 (sierpień 2006): 5220–27. http://dx.doi.org/10.1038/sj.onc.1209615.
Pełny tekst źródłaJazwinski, S. M., B. H. Howard i R. K. Nayak. "Cell Cycle Progression, Aging, and Cell Death". Journals of Gerontology Series A: Biological Sciences and Medical Sciences 50A, nr 1 (1.01.1995): B1—B8. http://dx.doi.org/10.1093/gerona/50a.1.b1.
Pełny tekst źródłaTrotter, Eleanor Wendy, i Iain Michael Hagan. "Release from cell cycle arrest with Cdk4/6 inhibitors generates highly synchronized cell cycle progression in human cell culture". Open Biology 10, nr 10 (październik 2020): 200200. http://dx.doi.org/10.1098/rsob.200200.
Pełny tekst źródłaBaldassarre, Gustavo, Milena Nicoloso, Monica Schiappacassi, Emanuela Chimienti i Barbara Belletti. "Linking Inflammation to Cell Cycle Progression". Current Pharmaceutical Design 10, nr 14 (1.05.2004): 1653–66. http://dx.doi.org/10.2174/1381612043384691.
Pełny tekst źródłaBohnsack, Brenda L., i Karen K. Hirschi. "NUTRIENT REGULATION OF CELL CYCLE PROGRESSION". Annual Review of Nutrition 24, nr 1 (14.07.2004): 433–53. http://dx.doi.org/10.1146/annurev.nutr.23.011702.073203.
Pełny tekst źródłaDavid, Rachel. "Keeping cell cycle progression in check". Nature Reviews Molecular Cell Biology 13, nr 6 (23.05.2012): 341. http://dx.doi.org/10.1038/nrm3363.
Pełny tekst źródłaSmink, Luc J. "Acetylation can regulate cell-cycle progression". Trends in Molecular Medicine 7, nr 9 (wrzesień 2001): 384. http://dx.doi.org/10.1016/s1471-4914(01)02122-0.
Pełny tekst źródłaRozprawy doktorskie na temat "Cell cycle progression"
Santos, Carlo Steven. "Circadian Control of Cell Cycle Progression". Thesis, Virginia Tech, 2009. http://hdl.handle.net/10919/76987.
Pełny tekst źródłaMaster of Science
Joseph, Alton J. "Regulation of S6KL during cell cycle progression". Thesis, California State University, Long Beach, 2014. http://pqdtopen.proquest.com/#viewpdf?dispub=1527714.
Pełny tekst źródłamTOR (Mammalian Target ofRapamycin), PI3K (Phosphatidylinositol3-kinase) and MEK (Mitogen-activated protein kinase/ERK kinase) have been shown to be potent regulators ofS6Kl at G1 phase of the cell cycle. Research has been concentrated at the Gt phase to elucidate mTOR's role in cell growth and proliferation. Limited information is available on the activity ofmTOR, PI3K and ERKl/2 in cell cycle phases other than G1. Since we have observed that S6Kl is active in phases other than G1 our goal was to ascertain ifmTOR, PI3K or ERKl/2 have a role in regulating S6Kl during these cell cycle phases. Using cell cycle analysis and immunoblot analysis we have determined here that mTORand PI3K could play a role in regulating S6Kl at the G1/S transition iQ. the cell cycle but there is also indications that mTOR and PI3K are potentially involved in regulating S6Kl in the phases post-G1/S of the cell cycle, indicating a complex interaction between the kinases used to regulate S6Kl during the cell cycle. ERKl/2 is demonstrated to have limited involvement in the regulation of S6Kl during the cell cycle.
Fredlund, Jan O. "The role of polyanimes in cell cycle progression". Lund : Lund University Dept. of Animal Physiology, 1996. http://catalog.hathitrust.org/api/volumes/oclc/38100686.html.
Pełny tekst źródłaDelorme, Marilyne. "Downregulation of ATRX disrupts cell proliferation and cell cycle progression". Thesis, University of Ottawa (Canada), 2008. http://hdl.handle.net/10393/27627.
Pełny tekst źródłaStewart, Nancy G. "P53 control over cell cycle progression at G2". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ32022.pdf.
Pełny tekst źródłaRathbone, Christopher R. "Mechanisms regulating skeletal muscle satellite cell cycle progression". Diss., Columbia, Mo. : University of Missouri-Columbia, 2006. http://hdl.handle.net/10355/5866.
Pełny tekst źródłaThe entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Vita. "December 2006" Includes bibliographical references.
Weber, Tom. "Optimal timing of phase resolved cell cycle progression". Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2015. http://dx.doi.org/10.18452/17253.
Pełny tekst źródłaSelf-reproduction is one of the distinguishing marks of living organisms. The cell cycle is the underlying process by which self-reproduction is accomplished in single-celled organisms. In multi-cellular organisms, the cell cycle is in addition indispensable for other vital processes, including immune reactions. In this thesis a method is developed that allows to estimate the time it takes for a dividing cells to complete the CC phases. Knowledge of the CC phase durations allows to predict, for example, how fast a population of proliferating cells will grow in size, or how many new cells are born per hour in a given tissue. In Chapter 1 of this thesis, a cell cycle model with delays and variability in the completion times of each phase is developed. Analytical solutions are derived to describe a common experimental technique used for cell cycle analysis, namely pulse labeling with bromodeoxyuridine (BrdU). Comparison with data shows that the model reproduces closely measured cell cycle kinetics, however also reveals that some of the parameter values cannot be identified. This problem is addressed in Chapter 2. In a first approach, the framework of D-optimal experimental designs is employed, in order to choose optimal sampling schemes. In a second approach, the prevailing protocol with a single nucleoside is modified by adding a second nucleoside analog pulse. Both methods are tested and the results suggest that experimental design can significantly improve parameter estimation. In Chapter 3, the model is applied to the germinal center reaction. A substantial influx of cells into the dark zone of germinal centers is predicted. Moreover the wide-held view of rapid proliferation in germinal centers, appears, under this model, as an artifact of cell migration.
Poli, Alessandro <1985>. "New DAG-dependent mechanisms modulate cell cycle progression". Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2015. http://amsdottorato.unibo.it/6739/1/Tesi_Alessandro.Poli..pdf.
Pełny tekst źródłaPoli, Alessandro <1985>. "New DAG-dependent mechanisms modulate cell cycle progression". Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2015. http://amsdottorato.unibo.it/6739/.
Pełny tekst źródłaOuertani, A. "Determinants of cell cycle progression in human mammary epithelial MCF12 cells". Thesis, University College London (University of London), 2012. http://discovery.ucl.ac.uk/1362848/.
Pełny tekst źródłaKsiążki na temat "Cell cycle progression"
Johannes, Boonstra, red. G1 phase progression. Georgetown, Tex: Landes Bioscience/Eurekah.com, 2003.
Znajdź pełny tekst źródłaBoonstra, Johannes. G1 phase progression. Georgetown, Tex: Landes Bioscience/Eurekah.com, 2003.
Znajdź pełny tekst źródłaKlein, Alyssa Michelle. A p53-independent role for MDM2-MDMX in cell cycle progression. [New York, N.Y.?]: [publisher not identified], 2021.
Znajdź pełny tekst źródłaRadeva, Galina. Overexpression of the integrin-linked kinase (ILK) promotes anchorage-independent cell cycle progression. Ottawa: National Library of Canada, 1997.
Znajdź pełny tekst źródłaG1 phase progression. Georgetown, TX: Landes Bioscience/Eurekah.com ; Kluwer Academic/Plenum, 2004.
Znajdź pełny tekst źródłaBoonstra, Johannes. Regulation of G1 Phase Progression. Springer, 2003.
Znajdź pełny tekst źródłaPetrocelli, Teresa. UVB cell cycle checkpoint loss in melanoma progression. 2002.
Znajdź pełny tekst źródłaLiang, Jiyong. Regulation of p27(KIP1) by the PI3K/PKB pathway and its role in cell cycle progression in human cancer. 2004.
Znajdź pełny tekst źródłaTurner, Neil. Mechanisms of progression of chronic kidney disease. Redaktor David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0136.
Pełny tekst źródłaPozio, Edoardo. Trichinellosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0068.
Pełny tekst źródłaCzęści książek na temat "Cell cycle progression"
Wang, Zhixiang. "Cell Cycle Progression and Synchronization: An Overview". W Cell-Cycle Synchronization, 3–23. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2736-5_1.
Pełny tekst źródłaDeRan, Michael, Mary Pulvino i Jiyong Zhao. "Assessing G1-to-S-Phase Progression After Genotoxic Stress". W Cell Cycle Checkpoints, 221–30. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-273-1_16.
Pełny tekst źródłaSherr, Charles J., Hitoshi Matsushime, Jun-ya Kato, Dawn E. Quelle i Martine F. Roussel. "Control of G1 Progression by Mammalian D-Type Cyclins". W The Cell Cycle, 17–23. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2421-2_2.
Pełny tekst źródłaKianfard, Zohreh, Kyle Cheung, Daniel Rappaport, Sirasie P. Magalage i Sarah A. Sabatinos. "Detecting Cell Cycle Stage and Progression in Fission Yeast, Schizosaccharomyces pombe". W Cell-Cycle Synchronization, 235–46. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2736-5_18.
Pełny tekst źródłaAlberts, Arthur S., i Axel Schönthal. "Positive and Negative Regulation of Cell Cycle Progression by Serine/Threonine Protein Phosphatases". W The Cell Cycle, 33–40. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2421-2_4.
Pełny tekst źródłaGreenwood, Brianna L., i David T. Stuart. "Synchronization of Saccharomyces cerevisiae Cells for Analysis of Progression Through the Cell Cycle". W Cell-Cycle Synchronization, 145–68. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2736-5_12.
Pełny tekst źródłaLee, Jongkuen, i David Dominguez-Sola. "Mammalian Cell Fusion Assays for the Study of Cell Cycle Progression by Functional Complementation". W Cell Cycle Checkpoints, 145–57. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1217-0_9.
Pełny tekst źródłaPrall, Owen W. J., Eileen M. Rogan, Elizabeth A. Musgrove, Colin K. W. Watts i Robert L. Sutherland. "Estrogen Regulation of Cell Cycle Progression". W Hormonal Carcinogenesis III, 220–27. New York, NY: Springer New York, 2001. http://dx.doi.org/10.1007/978-1-4612-2092-3_21.
Pełny tekst źródłaSutherland, Robert L., Jenny A. Hamilton, Kimberley J. E. Sweeney, Colin K. W. Watts i Elizabeth A. Musgrove. "Steroidal Regulation of Cell Cycle Progression". W Novartis Foundation Symposia, 218–34. Chichester, UK: John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470514757.ch13.
Pełny tekst źródłaVitale, Ilio, Mohamed Jemaà, Lorenzo Galluzzi, Didier Metivier, Maria Castedo i Guido Kroemer. "Cytofluorometric Assessment of Cell Cycle Progression". W Methods in Molecular Biology, 93–120. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-239-1_6.
Pełny tekst źródłaStreszczenia konferencji na temat "Cell cycle progression"
Tamura, Rodrigo E., Juliano D. Paccez, Mirian G. Morale, Xuesong Gu, Towia Libermann, Luiz F. Zerbini i Kristal Duncan. "cdk11p58 regulation of cell cycle progression in cancer development". W AACR International Conference: Molecular Diagnostics in Cancer Therapeutic Development– Sep 27-30, 2010; Denver, CO. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/diag-10-a37.
Pełny tekst źródłaTaylor-Harding, Barbie, Hasmik Agadjanian, Sandra Orsulic, Christine Walsh, Beth Y. Karlan i Wolf-Ruprecht Wiedemeyer. "Abstract 1749: Cell cycle requirements shape ovarian cancer progression." W Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-1749.
Pełny tekst źródłade Souza Cruz, André Luiz, Patricia Torres Bozza i João Paulo de Biaso Viola. "Abstract 1744: Role of lipid bodies on cell cycle progression." W Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-1744.
Pełny tekst źródłaChoi, Byeong Hyeok, Xun Che, Changyan Chen, Luo Lu i Wei Dai. "Abstract 2726: WWP2 is required for normal cell cycle progression". W Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-2726.
Pełny tekst źródłaDovat, Elanora, Jonathon Payne, Carlos M. Casiano, Justin Sloane, Chandrika Gowda, Kimberly J. Payne, Sinisa Dovat i Chunhua Song. "Abstract 3504: Regulation of cell cycle progression by Ikaros in leukemia". W Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-3504.
Pełny tekst źródłaMuralidharan, Somsundar Veppil, Joydeep Bhadury, Lydia Green, Lisa M. Nilsson, Kevin G. Mclure i Jonas A. Nilsson. "Abstract 4565: Bet bromodomain inhibitors affects replication & cell cycle progression". W Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-4565.
Pełny tekst źródłaIbla, Juan C., Yan Su, Assefa Wondimu, Stephan Ladisch i Robert Freishtat. "Modulation Of Cell Cycle Progression By Continuous Hypoxia In Bronchial Epithelium". W American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5293.
Pełny tekst źródłaSHA, YOBAO, Lavannya M. Pandit, shenyan zeng, Li-Yuan Yu-Lee i Tony N. Eissa. "CHIP Is A Novel Centrosomal Protein Involved In Cell Cycle Progression". W American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4933.
Pełny tekst źródłaYang, Yingzi, Wantae Kim i Xiaohui Wang. "Abstract IA11: Hippo signaling Is Intrinsically regulated during cell cycle progression". W Abstracts: AACR Special Conference on the Hippo Pathway: Signaling, Cancer, and Beyond; May 8-11, 2019; San Diego, CA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1557-3125.hippo19-ia11.
Pełny tekst źródłaAlmiman, Abeer, Daotai Nie i Jamila Adom. "Abstract 307: TRIB3 regulates cell cycle progression and programmed cell death in non-small cell lung cancer". W Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-307.
Pełny tekst źródłaRaporty organizacyjne na temat "Cell cycle progression"
Kuhne, Wendy, Candace Langan, Lucas Angelette i Lesleyann Hawthorne. Deuterium Concentration Effects on Cell Cycle Progression. Office of Scientific and Technical Information (OSTI), sierpień 2020. http://dx.doi.org/10.2172/1651107.
Pełny tekst źródłaKUHNE, WENDY, i LUCAS ANGELETTE. DEUTERIUM CONCENTRATION EFFECTS ON CELL CYCLE PROGRESSION. Office of Scientific and Technical Information (OSTI), październik 2021. http://dx.doi.org/10.2172/1827682.
Pełny tekst źródłaKUHNE, WENDY. DEUTERIUM CONCENTRATION EFFECTS ON CELL CYCLE PROGRESSION. Office of Scientific and Technical Information (OSTI), październik 2021. http://dx.doi.org/10.2172/1827952.
Pełny tekst źródłaDirenzo, James. Differential Regulation of Cell Cycle Progression in Human Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, sierpień 1998. http://dx.doi.org/10.21236/adb240737.
Pełny tekst źródłaRajasekaran, Ayyappan K. Role of PSMA in Aberrant Cell Cycle Progression in Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, grudzień 2004. http://dx.doi.org/10.21236/ada433876.
Pełny tekst źródłaRajasekaran, Ayyappan. Role of PSMA in Aberrant Cell Cycle Progression in Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, listopad 2009. http://dx.doi.org/10.21236/ada560904.
Pełny tekst źródłaDirenzo, James, i Myles Brown. Differential Regulation of Cell Cycle Progression in Human Breast Cancer Cell Lines by the Estrogen Receptor. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2000. http://dx.doi.org/10.21236/ada394036.
Pełny tekst źródłaLew, Brian. The Loss of Pin1 Deregulates Cell Cycle Progression and Promotes the Development of Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, lipiec 2008. http://dx.doi.org/10.21236/ada488883.
Pełny tekst źródłaLew, Brian. The Loss of Pin1 Deregulates Cell Cycle Progression and Promotes the Development of Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, lipiec 2009. http://dx.doi.org/10.21236/ada511532.
Pełny tekst źródłaStiles, Bangyan, i Hong Wu. Functional Analysis of Oncogene Akt: Its Role in Tumorigenesis In Vivo and Cell Cycle Progression In Vitro. Fort Belvoir, VA: Defense Technical Information Center, lipiec 2002. http://dx.doi.org/10.21236/ada407666.
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