Gotowa bibliografia na temat „Cell cycle”
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Artykuły w czasopismach na temat "Cell cycle"
MACKEY, M. C. "The Cell Division Cycle: Cell Cycle Clocks." Science 227, nr 4691 (8.03.1985): 1221. http://dx.doi.org/10.1126/science.227.4691.1221.
Pełny tekst źródłaLavi, O., i Y. Louzoun. "What cycles the cell? -Robust autonomous cell cycle models". Mathematical Medicine and Biology 26, nr 4 (6.07.2009): 337–59. http://dx.doi.org/10.1093/imammb/dqp016.
Pełny tekst źródłaKornitskaya, Y. V., S. D. Bykova i N. L. Gusakova. "Cell cycle". Тенденции развития науки и образования 96, nr 7 (2023): 106–8. http://dx.doi.org/10.18411/trnio-04-2023-366.
Pełny tekst źródłaEdgar, Bruce A. "Cell Cycle: Cell-cycle control in a developmental context". Current Biology 4, nr 6 (czerwiec 1994): 522–24. http://dx.doi.org/10.1016/s0960-9822(00)00113-5.
Pełny tekst źródłaForsburg, Susan L. "Cell Cycle: In and out of the cell cycle". Current Biology 4, nr 9 (wrzesień 1994): 828–30. http://dx.doi.org/10.1016/s0960-9822(00)00184-6.
Pełny tekst źródłaJacks, T. "CELL CYCLE: The Expanding Role of Cell Cycle Regulators". Science 280, nr 5366 (15.05.1998): 1035–36. http://dx.doi.org/10.1126/science.280.5366.1035.
Pełny tekst źródłaMaddox, Amy Shaub, i Jan M. Skotheim. "Cell cycle, cell division, cell death". Molecular Biology of the Cell 30, nr 6 (15.03.2019): 732. http://dx.doi.org/10.1091/mbc.e18-12-0819.
Pełny tekst źródłaWells, D. N. "Keith's MAGIC: Cloning and the Cell Cycle". Cellular Reprogramming 15, nr 5 (październik 2013): 348–55. http://dx.doi.org/10.1089/cell.2013.0038.
Pełny tekst źródłaChia, Gloryn, i Dieter Egli. "Connecting the Cell Cycle with Cellular Identity". Cellular Reprogramming 15, nr 5 (październik 2013): 356–66. http://dx.doi.org/10.1089/cell.2013.0041.
Pełny tekst źródłaWiney, Mark. "Cell cycle: Driving the centrosome cycle". Current Biology 9, nr 12 (czerwiec 1999): R449—R452. http://dx.doi.org/10.1016/s0960-9822(99)80279-6.
Pełny tekst źródłaRozprawy doktorskie na temat "Cell cycle"
Chauhan, Anuradha. "Cell cycle". Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2011. http://dx.doi.org/10.18452/16301.
Pełny tekst źródłaCell replication is a controlled process with sequential and timely activation and degradation of cyclins leading to swift transitions between the phases of the cell cycle. The essential achievement in identifying the key components and in dissecting the mechanisms of the cell cycle circuitry has been attributed to the simultaneous use of model systems like yeast, frogs, and flies. Present understanding of the cell cycle needs to be extended to investigate whether those findings also apply to mammalian in-vivo models like mice. We chose liver regeneration in mammals as the model system because it is the most synchronised cell proliferation phenomenon, where 95\% of the cells simultaneously enter cell cycle. The G1-S phase transition was modelled, focusing on how injury induced pro-inflammatory signals \textit{prime} the cells in G1 phase and consequently both cytokine and growth factor induced pathways lead to further cell cycle progression. The model was further extended to mitotic events leading to the all-or-none G2-M transition and mitotic exit. I focussed on the emerging role of Cdh1 in the mammalian cell cycle. Cdh1 known for its role in G1 phase was further investigated for its role G2 delay. Cdh1 was suggested to be at the core of the cell cycle machinery controlling cyclin dynamics. This model is an attempt in understanding core machinery of the mammalian cell cycle. Better understanding of the cell cycle control system in mammalian cells would enable understanding perturbations of the human cell cycle machinery which lead to diseases like cancers.
Radmaneshfar, Elahe. "Mathematical modelling of the cell cycle stress response". Thesis, University of Aberdeen, 2012. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=192232.
Pełny tekst źródłaThanky, Niren Rasik. "The mycobacterial cell cycle". Thesis, Imperial College London, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.405727.
Pełny tekst źródłaChaffey, Gary S. "Modelling the cell cycle". Thesis, University of Surrey, 2015. http://epubs.surrey.ac.uk/807189/.
Pełny tekst źródłaLi, Victor Chun. "The Cell Cycle and Differentiation in Stem Cells". Thesis, Harvard University, 2012. http://dissertations.umi.com/gsas.harvard:10536.
Pełny tekst źródłaGauger, Michele Ann Sancar Aziz. "Cryptochrome, circadian cycle, cell cycle checkpoints, and cancer". Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2007. http://dc.lib.unc.edu/u?/etd,971.
Pełny tekst źródłaTitle from electronic title page (viewed Dec. 18, 2007). "... in partial fulfillment of the requirements for the degree of Doctor of Philosophy in the Department of Biochemistry and Biophysics." Discipline: Biochemistry and Biophysics; Department/School: Medicine.
Gad, Annica. "Cell cycle control by components of cell anchorage /". Stockholm : Division of Pathology, Karolinska institutet, 2005. http://diss.kib.ki.se/2005/91-7140-359-0/.
Pełny tekst źródłaCadart, Clotilde. "Cell size homeostasis in animal cells". Thesis, Université Paris-Saclay (ComUE), 2017. http://www.theses.fr/2017SACLS103/document.
Pełny tekst źródłaThe way proliferating mammalian cells maintain a constant size through generations is still unknown. This question is however central because size homeostasis is thought to occur through the coordination of growth and cell cycle progression. In the yeast S. pombe for example, the trigger for cell division is the reach of a target size (Fantes, 1977). This mechanism is referred to as ‘sizer’. The homeostatic behavior of bacteria and daughter cells of the yeast S. cerevisiae on the contrary was recently characterized as an ‘adder’ where all cells grow by the same absolute amount of volume at each cell cycle. This leads to a passive regression towards the mean generation after generation (Campos et al., 2014; Soifer et al., 2016; Taheri-Araghi et al., 2015). These findings were made possible by the development of new technologies enabling direct and dynamic measurement of volume over full cell cycle trajectories. Such measurement is extremely challenging in mammalian cells whose shape constantly fluctuate over time and cycle over 20 hours long periods. Studies therefore privileged indirect approaches (Kafri et al., 2013; Sung et al., 2013; Tzur et al., 2009) or indirect measurement of cell mass rather than cell volume (Mir et al. 2014; Son et al., 2012). These studies showed that cells overall grew exponentially and challenged the classical view that cell cycle duration was adapted to size and instead proposed a role for growth rate regulation. To date however, no clear model was reached. In fact, the nature and even the existence of the size homeostasis behavior of mammalian cells is still debated (Lloyd, 2013).In order to characterize the homeostatic process of mammalian cells, we developed a technique that enable measuring, for the first time, single cell volume over full cell cycle trajectories (Cadart et al., 2017; Zlotek-Zlotkiewicz et al. 2015). We found that several cell types, HT29, HeLa and MDCK cells behaved in an adder-like manner. To further test the existence of homeostasis, we artificially induced asymmetrical divisions through confinement in micro-channels. We observed that asymmetries of sizes were reduced within the following cell cycle through an ‘adder’-like behavior. To then understand how growth and cell cycle progression were coordinated in way that generates the ‘adder’, we combined our volume measurement method with cell cycle tracking. We showed that G1 phase duration is negatively correlated with initial size. This adaptation is however limited by a minimum duration of G1, unraveled by the study of artificially-induced very large cells. Nevertheless, the adder behavior is maintained even in the absence of time modulation, thus suggesting a complementary growth regulatory mechanism. Finally, we propose a method to estimate theoretically the relative contribution of growth and timing modulation in the overall size control and use this framework to compare our results with that of bacteria. Overall, our work provides the first evidence that proliferating mammalian cells behave in an adder-like manner and suggests that both growth and cell cycle duration are involved in size control
Poplawski, Andrzej. "Cell cycle analysis of archaea". Doctoral thesis, Uppsala University, Department of Cell and Molecular Biology, 2000. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-1078.
Pełny tekst źródłaIn my thesis, the cell cycle analysis of archaea and hyperthermophilic organisms is presented for the first time. Crenarchaea from the genus Sulfolobus were used as a model system. Plow cytometry and light microscopy were applied to investigate the timing and coordination of different cell cycle events. Furthermore, DNA content, nucleoid structure, and nucleoid distribution at different stages during the cell cycle were studied. The Sulfolobus cell cycle was characterized as having a short pre-replication and a long post-replication period. The presence of a low proportion of cells with segregated genomes in the exponentially growing population suggested 3 considerable time delay between termination of chromosome replication and completion of nucleoid partition, reminiscent of the G2 period in eukaryotic cells.
The first available collection of conditional-lethal mutants of any archaeon or hyperthemophile was used to elucidate the coordination of cell cycle events. The studies showed that chromosome replication, nucleoid partition and cell division in Sulfolobus acidocaldarius, which are normally tightly coordinated during cellular growth, could be separately inhibited or uncoupled by mutation.
The ftsZ gene, which is involved in cell division in bacteria and euryarchaea, was isolated from the halophilic archaeon Haloferax mediterranei. Transcriptional start sites were mapped, and putative translation initiation elements were identified. In both the upstream and downstream regions of the ftsZ gene, open reading frames were found to be conserved within the genus Haloferax. Furthermore, at the 3' end of the ftsZ gene, the homologs of the bacterial secE and nusG genes are conserved in almost all euryarchaea analyzed so far. The studies also demonstrated the functional conservation of the FtsZ protein in different archaeal species, as well as between euryarchaea and bacteria.
Shirazi, Fard Shahrzad. "The Heterogenic Final Cell Cycle of Retinal Horizontal Cells". Doctoral thesis, Uppsala universitet, Medicinsk utvecklingsbiologi, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-222559.
Pełny tekst źródłaKsiążki na temat "Cell cycle"
Pippa, Cristina. Cell cycle. Burlington, MA]: JAC Pub. & Promotions, 2007.
Znajdź pełny tekst źródłaA, Bryant J., i Francis D, red. The eukaryotic cell cycle. New York: Taylor & Francis, 2008.
Znajdź pełny tekst źródła1943-, Hunt Tim, red. The cell cycle: An introduction. New York: W.H. Freeman, 1993.
Znajdź pełny tekst źródłaMurray, Andrew Wood. The cell cycle: An introduction. New York: Oxford University Press, 1993.
Znajdź pełny tekst źródłaEnders, Greg H. Cell cycle deregulation in cancer. New York: Springer, 2010.
Znajdź pełny tekst źródłaHumphrey, Tim, i Gavin Brooks. Cell Cycle Control. New Jersey: Humana Press, 2004. http://dx.doi.org/10.1385/1592598579.
Pełny tekst źródłaManfredi, James J., red. Cell Cycle Checkpoints. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1217-0.
Pełny tekst źródłaCoutts, Amanda S., i Louise Weston, red. Cell Cycle Oscillators. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1538-6.
Pełny tekst źródłaWang, Zhixiang, red. Cell-Cycle Synchronization. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2736-5.
Pełny tekst źródłaBanfalvi, Gaspar, red. Cell Cycle Synchronization. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6603-5.
Pełny tekst źródłaCzęści książek na temat "Cell cycle"
Takahashi, Naoki, i Masaaki Umeda. "Cell Cycle". W Cell Biology, 1–19. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-7881-2_11-1.
Pełny tekst źródłaNahler, Gerhard. "cell cycle". W Dictionary of Pharmaceutical Medicine, 23. Vienna: Springer Vienna, 2009. http://dx.doi.org/10.1007/978-3-211-89836-9_173.
Pełny tekst źródłaCsikász-Nagy, Attila. "Cell Cycle". W Encyclopedia of Systems Biology, 220–31. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-9863-7_115.
Pełny tekst źródłaAdlung, Lorenz. "Cell Cycle". W Cell and Molecular Biology for Non-Biologists, 75–88. Berlin, Heidelberg: Springer Berlin Heidelberg, 2022. http://dx.doi.org/10.1007/978-3-662-65357-9_7.
Pełny tekst źródłaMira, Grdisa, i Ana-Matea Mikecin. "Cell Cycle". W New Frontiers in Nanochemistry, 85–89. Includes bibliographical references and indexes. | Contents: Volume 1. Structural nanochemistry – Volume 2. Topological nanochemistry – Volume 3. Sustainable nanochemistry.: Apple Academic Press, 2020. http://dx.doi.org/10.1201/9780429022951-5.
Pełny tekst źródłaSiudeja, Katarzyna, Jannie de Jong i Ody C. M. Sibon. "Studying Cell Cycle Checkpoints Using Drosophila Cultured Cells". W Cell Cycle Checkpoints, 59–73. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-273-1_6.
Pełny tekst źródłaNikaido, Toshio, Koji Ono, Masuji Yamamoto, Toshiyuki Sakai i Yasushi Magami. "Cell Cycle Regulation in Normal Versus Leukemic T Cells". W The Cell Cycle, 347–57. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2421-2_41.
Pełny tekst źródłaKohn, Kurt W., Patrick M. O’Connor i Joany Jackman. "Cell Cycle Regulation and the Chemosensitivity of Cancer Cells". W The Cell Cycle, 379–88. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2421-2_44.
Pełny tekst źródłaBehl, Christian, i Christine Ziegler. "Cell Cycle: The Life Cycle of a Cell". W Cell Aging: Molecular Mechanisms and Implications for Disease, 9–19. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-45179-9_2.
Pełny tekst źródłaQuaranta, Vito, Darren Tyson i Peter Frick. "Cell Cycle, Cancer Cell Cycle and Oncogene Addiction". W Encyclopedia of Systems Biology, 341–43. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-9863-7_49.
Pełny tekst źródłaStreszczenia konferencji na temat "Cell cycle"
Gurkan-Cavusoglu, Evren, Jane E. Schupp, Timothy J. Kinsella i Kenneth A. Loparo. "Analysis of cell cycle dynamics using probabilistic cell cycle models". W 2011 33rd Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2011. http://dx.doi.org/10.1109/iembs.2011.6089914.
Pełny tekst źródłaSpitzley, David V., Tiffany A. Brunetti i Bruce W. Vigon. "Assessing Fuel Cell Power Sustainability". W Total Life Cycle Conference and Exposition. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 2000. http://dx.doi.org/10.4271/2000-01-1490.
Pełny tekst źródłaStephenson, Dawn, i Ian Ritchey. "Parametric Study of Fuel Cell and Gas Turbine Combined Cycle Performance". W ASME 1997 International Gas Turbine and Aeroengine Congress and Exhibition. American Society of Mechanical Engineers, 1997. http://dx.doi.org/10.1115/97-gt-340.
Pełny tekst źródłaCoulson, Guy, i Richard Stobart. "Life–Cycle Analysis and the Fuel Cell Car". W Total Life Cycle Conference and Exposition. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 2000. http://dx.doi.org/10.4271/2000-01-1485.
Pełny tekst źródłaSchneider, Eugenia, i Michael Mangold. "Problems During Cell Cycle in Artificial Cell Modeling". W Artificial Life 14: International Conference on the Synthesis and Simulation of Living Systems. The MIT Press, 2014. http://dx.doi.org/10.7551/978-0-262-32621-6-ch110.
Pełny tekst źródłaSchneider, Eugenia, i Michael Mangold. "Problems During Cell Cycle in Artificial Cell Modeling". W Artificial Life 14: International Conference on the Synthesis and Simulation of Living Systems. The MIT Press, 2014. http://dx.doi.org/10.1162/978-0-262-32621-6-ch110.
Pełny tekst źródłaPascalie, Jonathan, Valérie Lobjois, Hervé Luga, Bernard Ducommun i Yves Duthen. "Checkpoint oriented cell-cycle simulation". W the fourteenth international conference. New York, New York, USA: ACM Press, 2012. http://dx.doi.org/10.1145/2330784.2330966.
Pełny tekst źródłaAzizi, Aydin, i Navid Seifipour. "Modeling and Control of Cell Cycle". W 2009 3rd International Conference on Bioinformatics and Biomedical Engineering (iCBBE). IEEE, 2009. http://dx.doi.org/10.1109/icbbe.2009.5163070.
Pełny tekst źródłaFaltenbacher, M., M. Betz i P. Eyerer. "Alternative Fuels for Fuel Cell Powered Buses in Comparison to Diesel powered Buses". W Total Life Cycle Conference and Exposition. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 2000. http://dx.doi.org/10.4271/2000-01-1484.
Pełny tekst źródłaSaito, Norihiko, Nozomi Hirai, Kazuya Aoki, Satoshi Fujita, Haruo Nakayama, Morito Hayashi, Takatoshi Sakurai i Satoshi Iwabuchi. "Abstract 2621: OLIG2 regulates stem cell maintenance and cell cycle in glioma stem cells". W Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-2621.
Pełny tekst źródłaRaporty organizacyjne na temat "Cell cycle"
Williams, Thomas. Cell Biology Board Game: Cell Life Cycle Top Trumps. University of Dundee, styczeń 2023. http://dx.doi.org/10.20933/100001277.
Pełny tekst źródłaReed, Steven I. Cell Cycle in Normal and Malignant Breast Epithelial Cells. Fort Belvoir, VA: Defense Technical Information Center, lipiec 1996. http://dx.doi.org/10.21236/ada315811.
Pełny tekst źródłaReed, Steven I. Cell Cycle in Normal and Malignant Breast Epithelial Cells. Fort Belvoir, VA: Defense Technical Information Center, lipiec 1995. http://dx.doi.org/10.21236/ada300387.
Pełny tekst źródłaReed, Steven I. Cell Cycle in Normal and Malignant Breast Epithelial Cells. Fort Belvoir, VA: Defense Technical Information Center, lipiec 1998. http://dx.doi.org/10.21236/ada354074.
Pełny tekst źródłaDooner, Mark, Jason M. Aliotta, Jeffrey Pimental, Gerri J. Dooner, Mehrdad Abedi, Gerald Colvin, Qin Liu, Heinz-Ulli Weier, Mark S. Dooner i Peter J. Quesenberry. Cell Cycle Related Differentiation of Bone Marrow Cells into Lung Cells. Office of Scientific and Technical Information (OSTI), grudzień 2007. http://dx.doi.org/10.2172/936517.
Pełny tekst źródłaMcConkey, David J. Cell Cycle Dependence of TRAIL Sensitivity in Prostate Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, listopad 2006. http://dx.doi.org/10.21236/ada466697.
Pełny tekst źródłaMcConkey, David J. Cell Cycle Dependence of TRIAL Sensitivity in Prostate Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, listopad 2007. http://dx.doi.org/10.21236/ada481365.
Pełny tekst źródłaRich, Alexander. Transcriptional Regulation in the Cell Cycle. Fort Belvoir, VA: Defense Technical Information Center, październik 1988. http://dx.doi.org/10.21236/ada200715.
Pełny tekst źródłaChaney, Larry J., Mike R. Tharp, Tom W. Wolf, Tim A. Fuller i Joe J. Hartvigson. FUEL CELL/MICRO-TURBINE COMBINED CYCLE. Office of Scientific and Technical Information (OSTI), grudzień 1999. http://dx.doi.org/10.2172/802823.
Pełny tekst źródłaKuhne, Wendy, Candace Langan, Lucas Angelette i Lesleyann Hawthorne. Deuterium Concentration Effects on Cell Cycle Progression. Office of Scientific and Technical Information (OSTI), sierpień 2020. http://dx.doi.org/10.2172/1651107.
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