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Prado, Fabio Ornellas. "Avaliação clinicopatologica de condrossarcomas de cabeça e pescoço". [s.n.], 2006. http://repositorio.unicamp.br/jspui/handle/REPOSIP/287860.
Pełny tekst źródłaTese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba
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Resumo: Os condrossarcomas são tumores malignos de etiologia desconhecida, em que as células tumorais formam tecido cartilaginoso. Embora a ocorrência seja rara, principalmente na região de cabeça e pescoço, é o segundo tumor ósseo primário maligno mais freqüente. O objetivo deste trabalho foi correlacionar os dados clinicopatológicos ao prognóstico dos pacientes portadores de condrossarcomas de cabeça e pescoço tratados no Departamento de Cabeça e Pescoço e Otorrinolaringologia do Hospital do Câncer A.C. Camargo. Foram selecionados 16 casos tratados no Hospital do Câncer A.C. Camargo entre 1953 e 2002. A idade média de acometimento no momento do diagnóstico foi de 36,5 anos, variando de 11 a 70 anos. Observou-se ligeira predileção pelo gênero masculino (56,2%). De acordo com a localização, 7 casos (43,8%) acometeram a maxila; 5 casos ocorreram em outros sítios (região etmoidal, fossa nasal [2 casos], fossa infra-temporal, região parietooccipital) e 4 pacientes (25,0%) desenvolveram condrossarcomas em mandíbula. A maioria dos casos foi tratada somente com cirurgia (6 casos, 40% do total). A sobrevida global, observada foi de 66,7% em 3 anos e 56,4% em 5 anos
Abstract: Chondrosarcomas are malignant tumors of unknown etiology, in which tumoral cells form cartilagenous tissue. Although rare in head and neck region, chondrosarcomas are the second primary osseous tumor in frequency. The aim of the present study was correlate clinicopathological data to prognostic of patients with head and neck chondrosarcomas treated in the Head and Neck and Otolaringology Department of the A.C. Camargo Cancer Hospital. There were 16 cases treated in the institution from 1953 to 2002. The mean age at diagnosis was 36.5 years, ranging from 11 to 70 years. A slight male preference was observed (56,2%). According to the location, 7 cases (43,8%) accomited maxilla; 5 cases occured at other sites (ethmoidal region, nasal fossa [2 cases], infratemporal fossa, parieto-occipital region) and 4 patients (25,0%) had mandibular lesions. Most of cases were treated with surgery alone (6 cases, 40%). The observed overall survival was 66,7% for 3-year and 56,4% for 5-year
Doutorado
Patologia
Doutor em Estomatopatologia
Dickinson, Sally Clare. "Cartilage oligomeric matrix protein and cartilage degradation". Thesis, University of Sheffield, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.323419.
Pełny tekst źródłaGirdler, N. M. "The role of mandibular condylar cartilage in articular cartilage repair". Thesis, King's College London (University of London), 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.309110.
Pełny tekst źródłaCook, James L. "Three-dimensional chondrocyte culture : in vitro and in vivo applications /". free to MU campus, to others for purchase, 1998. http://wwwlib.umi.com/cr/mo/fullcit?p9924877.
Pełny tekst źródłaHoch, Johanna M. "SERUM CARTILAGE OLIGOMERIC MATRIX PROTEIN: A BIOMARKER FOR ACUTE ARTICULAR CARTILAGE DAMAGE". UKnowledge, 2012. http://uknowledge.uky.edu/rehabsci_etds/3.
Pełny tekst źródłaHamm, Christopher Allan Soares Marcelo B. "Functional genomic analyses of the impact of global hypomethylation and of tumor microenvironment in a rat model of human chondrosarcoma". [Iowa City, Iowa] : University of Iowa, 2009. http://ir.uiowa.edu/etd/372.
Pełny tekst źródłaWong, Brian Jet-Fei. "Laser mediated cartilage reshaping". [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2001. http://dare.uva.nl/document/60182.
Pełny tekst źródłaGetgood, Alan Martin John. "Articular cartilage tissue engineering". Thesis, University of Cambridge, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608764.
Pełny tekst źródłaMakower, Anne-Marie. "Regulation of chondrocyte growth i̲n̲ v̲i̲t̲r̲o̲". Stockholm : Kongl. Carolinska Medico Chirurgiska Institutet, 1989. http://books.google.com/books?id=j0pqAAAAMAAJ.
Pełny tekst źródłaTsang, Kwok-yeung. "Molecular pathogenesis of abnormal chondrocyte differentiation in a transgenic mouse model /". View the Table of Contents & Abstract, 2006. http://sunzi.lib.hku.hk/hkuto/record/B35132796.
Pełny tekst źródłaArora, Arvind. "Potential of qMRI in the study of articular cartilage and cartilage repair tissue". Thesis, University of Cambridge, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.613644.
Pełny tekst źródłaYang, Ziquan. "Repair of cartilage injury using gene modified stem cells and acellular cartilage matrix". Thesis, Queen's University Belfast, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.501585.
Pełny tekst źródłaSeol, Dong Rim. "Chondrogenic progenitor cell response to cartilage injury and its application for cartilage repair". Diss., University of Iowa, 2011. https://ir.uiowa.edu/etd/1262.
Pełny tekst źródłaFenwick, S. A. "Models of cartilage vascularisation : the vasculature and its effects on developing and osteoarthritic cartilage". Thesis, University of Leicester, 1997. http://hdl.handle.net/2381/29481.
Pełny tekst źródłaTogo, Takeshi. "Identification of cartilage progenitor cells in the adult ear perichondrium : utilization for cartilage reconstruction". Kyoto University, 2008. http://hdl.handle.net/2433/135826.
Pełny tekst źródłaAndrade, Andre Luis Lugnani de. "Expressão do fator de transcrição HIF - 1'alfa' em condrocitos humanos cultivados em condições normais de oxigenio". [s.n.], 2006. http://repositorio.unicamp.br/jspui/handle/REPOSIP/309649.
Pełny tekst źródłaDissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas
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Resumo: Introdução: Os condrócitos da cartilagem articular vivem em um ambiente com baixa concentração de oxigênio. Nestas condições, a proteína do fator induzido por hipóxia (HIF-1a) mantém-se estável e ativa genes que são fundamentais na homeostase do oxigênio. A expressão do HIF-1a aumenta, em joelhos com osteoartrite (OA), principalmente nas áreas mais afetadas pela degeneração. Os condrócitos são capazes de produzir mediadores inflamatórios, como a interleucina-1 (IL-1) e o fator de necrose tumoral a (TNF-a), que estimulam a produção de prostaglandinas, metaloproteinases e óxido nítrico e relacionam-se com o início e com a progressão da osteoartrite. Os antiinflamatórios são drogas freqüentemente utilizadas no tratamento sintomático da OA. Material e Método: condrócitos humanos de joelhos osteoartríticos cultivados em suspensão e em condições normais de oxigênio foram divididos em quatro grupos: 1) controle, 2) estimulados com IL-1 ou TNF-a, 3) estimulados com meloxicam ou parecoxibe e 4) estimulados com meloxicam ou parecoxibe associados a IL-1 ou TNF-a. Os grupos foram submetidos à extração de RNA (ácido ribonucléico) e de proteína nuclear. O RNA foi convertido em cDNA, sendo então realizada a reação de PCR em tempo real para verificar a expressão do HIF-1a. As proteínas nucleares foram extraídas, quantificadas e analisadas pela técnica de Western Blotting. Resultados: Foi detectada a expressão de HIF-1a e cDNA de HIF-1a em todos os grupos de condrócitos cultivados em suspensão em tensões normais de oxigênio, não havendo diferenças significativas entre os grupos. Discussão: a meia-vida do HIF-1a é extremamente curta em normóxia e marcadamente prolongada em hipóxia, por isso muitos pesquisadores acreditam não ser possível a detecção da proteína do HIF-1a em condrócitos cultivados em condições normais de oxigênio. Neste estudo foi possível constatar a expressão do HIF-1a em normóxia, possivelmente devido ao modelo de cultura utilizado. O estímulo com IL-1, TNF-a e inibidores da COX-2 não alterou a expressão de HIF-1a. Condrócitos oriundos de articulações osteoartríticas avançadas poderiam apresentar resistência à ação das citocinas
Abstract: Introduction: The chondrocytes of joint surface live in low concentration of oxygen environment. In this condition, the hypoxia inducible factor 1 a (HIF-1a) becomes stable and regulates the expression of genes that are important for oxygen homeostasis. The expression of HIF-1a mRNA is augmented in chondrocytes from osteoarthritic knees, especially in more degenerated areas. Chondrocytes are capable of producing inflammatory mediators, such as interleukin 1 (IL-1) and tumoral necrosis factor a (TNF-a), that stimulate the production of prostaglandin, metalloproteinases and nitric oxide, correlated with the onset and progression of osteoarthritis. Antiinflammatory drugs are frequently used in the treatment of symptoms of osteoarthritis. Material e Methods: human chondrocytes from osteoarthritic knees were cultivated in suspension and in normal tension of oxygen. The cells were divided in 4 groups: control, stimulated with IL-1 or TNF-a, stimulated with meloxicam or parecoxib and the last one stimulated with meloxicam or parecoxib and IL-1 or TNF-a. Nuclear protein and RNA were extracted from these cells. cDNA was synthesized from RNA and real time PCR was performed with this product in order to determine HIF-1a expression. Nuclear protein was analyzed using the Western-Blotting method. Results: HIF-1a and HIF-1a mRNA was detectable in all cell groups, and there was not a statistic significant difference between them. Discussion: As half live of HIF-1a is extremely short when in normoxic and greater in hypoxic conditions, many researchers believe it is not possible to detect this protein in chondrocytes cultivated in normoxic environment. Our results presented expression of HIF-1a in normal oxygen tensions, probably due to the fact that chondrocytes were cultivated in suspension. As chondrocytes were obtained from advanced osteoarthritic knees and in such conditions the cells can be more resistant to the action of cytokines, this could explain why IL-1, TNF-a and antiinflamatory did not result in modification of HIF-1a
Mestrado
Clinica Medica
Mestre em Clinica Medica
Sitterle, Valerie B. "Photoactivated Fixation of Cartilage Tissue". Diss., Georgia Institute of Technology, 2004. http://hdl.handle.net/1853/7609.
Pełny tekst źródłaRains, Jeffrey K. "Mechanical properties of tracheal cartilage". Thesis, University of British Columbia, 1989. http://hdl.handle.net/2429/27994.
Pełny tekst źródłaApplied Science, Faculty of
Chemical and Biological Engineering, Department of
Graduate
Gratz, Kenneth R. "Biomechanics of articular cartilage defects". Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2007. http://wwwlib.umi.com/cr/ucsd/fullcit?p3284116.
Pełny tekst źródłaTitle from first page of PDF file (viewed January 9, 2008). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references.
Mansfield, Jessica. "Multi-photon microscopy of cartilage". Thesis, University of Exeter, 2008. http://hdl.handle.net/10036/42345.
Pełny tekst źródłaArkill, Kenton Paul. "Mass transport in articular cartilage". Thesis, University of Exeter, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.421565.
Pełny tekst źródłaBurgin, Leanne Victoria. "Impact loading of articular cartilage". Thesis, University of Aberdeen, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.288339.
Pełny tekst źródłaTreppo, Steven. "Physical diagnostics of cartilage degeneration". Thesis, Massachusetts Institute of Technology, 1999. http://hdl.handle.net/1721.1/85263.
Pełny tekst źródła"January 1999."
Includes bibliographical references (leaves 219-239).
by Steven Treppo.
Ph.D.
Merly, Liza. "Immunomodulation by Shark Cartilage Extracts". FIU Digital Commons, 2011. http://digitalcommons.fiu.edu/etd/420.
Pełny tekst źródłaLoeuille, Damien. "Micro-imagerie RMN du cartilage". Nancy 1, 2002. http://www.theses.fr/2002NAN11307.
Pełny tekst źródłaZhang, Le. "Neutral solute transport in cartilage". Access to citation, abstract and download form provided by ProQuest Information and Learning Company; downloadable PDF file, 165 p, 2008. http://proquest.umi.com/pqdweb?did=1601524361&sid=8&Fmt=2&clientId=8331&RQT=309&VName=PQD.
Pełny tekst źródłaParker, Eleanor. "Mechanical loading and cartilage physiology". Thesis, University of Westminster, 2011. https://westminsterresearch.westminster.ac.uk/item/8zzqy/mechanical-loading-and-cartilage-physiology.
Pełny tekst źródłaRowles, Christopher. "Visualisation of Articular Cartilage Microstructure". Thesis, Curtin University, 2016. http://hdl.handle.net/20.500.11937/52984.
Pełny tekst źródłaSCARPA, TOMMASO. "BIOPOLYMERS FOR CARTILAGE TISSUE-ENGINEERING". Doctoral thesis, Università degli studi di Trieste, 2007. http://thesis2.sba.units.it/store/handle/item/12302.
Pełny tekst źródłaBONNET, ISABELLE. "Les traumatismes du cartilage triradie". Besançon, 1991. http://www.theses.fr/1991BESA3041.
Pełny tekst źródłaBrodkin, Kathryn Rhea. "Chondrocyte behavior in monolayer culture : the effects of protein substrates and culture media". Thesis, Georgia Institute of Technology, 2002. http://hdl.handle.net/1853/20216.
Pełny tekst źródłaJin, Moonsoo 1971. "Regulation of cartilage metabolism by dynamic tissue shear strain and the mechanical characterization of cartilage". Thesis, Massachusetts Institute of Technology, 1999. http://hdl.handle.net/1721.1/9787.
Pełny tekst źródłaIncludes bibliographical references (leaves 76-82).
I investigated the physical regulation of cartilage metabolism induced by dynamic tissue shear strain, and also the mechanical behavior under shear strain, especially focused on shear modulus, under different shear strain, frequency, compressive offset, and physiochemical environment. For this purpose, a new instrument was developed to apply axial deformations as small as lµm and sinusoidal rotations as small as 0.5% up to 4% based on 1 mm thickness of tissue under feedback control. This apparatus is small enough (30 cm high x 25 cm x 20 cm) to be placed in a standard incubator for long-term tissue culture loading studies. Consistent with previous studies, articular cartilage showed a typical viscoelastic material behavior under shear strain, and the shear modulus increased when the frequency and compressive offset was increased, or the applied shear strain was decreased. This shear softening effect was found to be related to the transient response of cartilage. The equilibrium stress was linear with shear strain. Under different ionic strengths, articular cartilage showed a decrease in the shear modulus up to 1.0 M NaCl bath concentration, but interestingly above this point the shear modulus began to increase while axial stiffness monotonically decreased. Biosynthetic response of chondrocytes under 0.1 Hz and 1 % sinusoidal shear strain, which was measured by the incorporation rate of 35 S-sulfate and 3 H-proline, was significantly increased compared to the incorporation level of statically compressed or unloaded free-swelling controls. To check the local stimulation by relative fluid flow which can be induced in the outer peripheral region, the incorporation rate of 2 mm center region and outer ring region was compared to those of static and free swelling controls. Unlike axial compression, where the incorporation rate in the outer ring region was greater than the 2 mm center region due to fluid flow and cell deformation, the effect of shear strain was uniformly distributed over the entire area, so the increased biosynthetic effect under shear strain is more related with direct mechanical deformation of chondrocytes rather than fluid flow, changes in hydrostatic pressure, or electrical or chemical environment.
by Moonsoo Jin.
S.M.
Stoker, Aaron. "Evaluation of the metabolic responses of normal and osteoarthritic cartilage in vitro and in vivo /". Free to MU Campus, others may purchase, 2004. http://wwwlib.umi.com/cr/mo/fullcit?p3144460.
Pełny tekst źródłaHwang, Jennifer. "Integration of cartilage and bone through a calcified cartilage interface to form a functional osteochondral graft". Diss., [La Jolla] : University of California, San Diego, 2010. http://wwwlib.umi.com/cr/ucsd/fullcit?p3407906.
Pełny tekst źródłaTitle from first page of PDF file (viewed June 22, 2010). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references.
Ghanavi, Parisa. "Effects of cartilage dust on cartilage formation in in vitro and in ectopic in vivo models". Thesis, Queensland University of Technology, 2016. https://eprints.qut.edu.au/101499/1/Parisa_Ghanavi_Thesis.pdf.
Pełny tekst źródłaSrinivasan, Jayendran. "Investigation of internal fluid pressure in cells". Morgantown, W. Va. : [West Virginia University Libraries], 2005. https://eidr.wvu.edu/etd/documentdata.eTD?documentid=4177.
Pełny tekst źródłaTitle from document title page. Document formatted into pages; contains x, 114 p. : ill. (some col.). Includes abstract. Includes bibliographical references (p. 69-77).
Meirelles, Vanessa Morales [UNESP]. "Avaliação do laser de baixa intensidade na regeneração de cartilagem articular do joelho de coelhos submetidos à trocleoplastia". Universidade Estadual Paulista (UNESP), 2011. http://hdl.handle.net/11449/101168.
Pełny tekst źródłaA osteoartrose (OA) do joelho é uma doença de caráter inflamatório e degenerativo que provoca a destruição da cartilagem articular e leva à deformidade da articulação. A luxação patelar é um problema ortopédico comum na clínica de pequenos animais. A cirurgia não impede a progressão da OA. O laser de baixa intensidade (LBI) tem sido utilizado para acelerar processos de reparação tecidual, por aumentar o fluxo sangüíneo, possuir ação antiinflamatória, antiedematosa, analgésica e estimular o metabolismo celular. Este trabalho estudou a influência da laserterapia em dois espectros eletromagnéticos, vermelho e infravermelho, na reparação cartilagínea, diferenciando histologicamente o tipo de cartilagem formada durante a reparação articular. Foram utilizados 36 coelhos machos raça Norfolk divididos em 3 grupos com 12 animais cada: G1 (controle, trocleoplastia sem tratamento pós-operatório), G2 (trocleoplastia + irradiação laser 670 nm) e G3 (trocleoplastia + irradiação laser 904 nm). Os animais dos grupos G2 e G3 foram irradiados diariamente, com intervalo de 24 horas, durante 10 dias utilizando um laser In-Ga-Al com comprimento de onda (λ) de 670 e um laser de As-Ga com λ de 904 nm respectivamente, na dose de 2 J por ponto totalizando 4 pontos. Os grupos G2 e G3 apresentaram recuperação funcional mais rápida que G1. Histologicamente, G3 apresentou um aspecto osteocondral mais regular, com formação de cartilagem hialina enquanto em G1 e G2 formou-se fibrocartilagem
Stifle osteoarthrosis is an inflammatory and degenerative disease that causes destruction of the articular cartilage and leads to deformity in the articulation. Patellar luxation is a common orthopedic problem in the clinic of small animals. The surgery does not avoid the progression of the osteoarthrosis. Low intensity laser has been used to improve degenerated processes for it increases blood flow, it has antiinflammatory, antiedematous, analgesic effects and stimulates cellular metabolism. This work studied the influence of laser therapy of two electromagnetic spectra, red and infrared, for repairing cartilage, histologically telling apart the kind of cartilage formed during articular regeneration. We use 36 male rabbits bred Norfolk divided into 3 groups with 12 animals each: G1 (control, trochleoplasty without treatment post-operatory), G2 (trochleoplasty + laser therapy 670 nm) and G3 (trochleoplasty + laser therapy 904 nm). The animals from G2 and G3 were irradiated daily with an interval of 24 hours over 10 days using a laser Ga-Al-In with wavelength (λ) of 670 and a Ga-As laser with λ 904 nm respectively. At a dose of 2 J per point total of 4 points. The groups G2 and G3 showed faster functional recovery that G1. Histologically, G3 had a most regular osteochondral aspect, with hyaline cartilage formation while in G1 and G2 formed fibrocartilage
Coelho, Lívia de Paula. "Células-tronco mesenquimais autólogas no tratamento da osteoartrite induzida da articulação coxofemoral em coelhos (Oryctolagus cuniculus) /". Jaboticabal, 2017. http://hdl.handle.net/11449/150483.
Pełny tekst źródłaBanca: Luis Gustavo Gosuen Gonçalves Dias
Banca: Paulo César Jark
Resumo: A cartilagem articular possui capacidade de reparação limitada, aumentado a predisposição ao desenvolvimento de alterações degenerativas, muitas vezes irreversíveis. Diversas formas de tratamento, cirúrgicas ou conservativas, são descritas, entretanto a terapêutica da osteoartrite continua sendo grande desafio ao médico veterinário. Neste contexto, a pesquisa envolvendo células-tronco mesenquimais destaca-se na busca de melhorias e avanços na reparação da cartilagem articular. Objetivou-se, no presente projeto, comparar a regeneração cartilaginosa da articulação coxofemoral de coelhos, com e sem o transplante de células-tronco mesenquimais autólogas, por meio de exames radiográficos e histopatológicos. Dois grupos, com 15 animais da espécie leporina cada, foram submetidos à indução química de osteoartrite com solução de colagenase 2% na articulação coxofemoral direita. No Grupo 1 (Células-tronco) realizou-se a aplicação intra-articular de células-tronco mesenquimais autólogas, enquanto que, o Grupo 2 (Controle) foi constituído por animais submetidos à aplicação intra-articular de solução salina estéril. Foram realizadas avaliações radiográficas e histopatológicas aos 30, 60 e 90 dias após a aplicação. Os resultados histológicos deste ensaio indicam que células-tronco mesenquimais (Grupo 1) melhoraram discretamente a qualidade do tecido de reparo, de acordo com os critérios da escala semi-quantitativa ICRS 1 ("International Cartilage Repair Society"). O Grupo 1 (Células-Tronco... (Resumo completo, clicar acesso eletrônico abaixo)
Abstract: The articular cartilage has limited repair capacity, leading to an increased risk for degenerative changes, potentially irreversible. Several treatments, surgical or not, are described, however osteoarthritis remains a major challenge for the veterinarian. In this context, research involving mesenchymal stem cells stands out. The aim of this study was to compare cartilage regeneration of the hip in rabbits, with and without the transplantation of autologous mesenchymal stem cells. Radiographic and histopathological evaluation were used. Thirty rabbits were submitted to chemical induction of osteoarthritis with a 2% colagenase in the right hip. They were divided into 2 groups of 15 animals each: Group 1 (intra-articular application of autologous mesenchymal stem cells) and Group 2 (control - intra-articular application of sterile saline solution). Radiographic and histopathological evaluations were performed at 30, 60 and 90 days after application. The mesenchymal stem cells group (Group 1) showed slight improvement of the quality of the repair tissue, according to the semi-quantitative scale criteria ICRS 1 (International Cartilage Repair Society). The Group 1 (Stem Cells) showed superiority in relation to Group 2, specially in the parameters joint surface, extracellular matrix and cellular distribution.
Mestre
Ishiguro, Naoki, Toshihisa Kojima i A. Robin Poole. "Mechanism of cartilage destruction in osteoarthritis". Nagoya University School of Medicine, 2002. http://hdl.handle.net/2237/5380.
Pełny tekst źródłaRafi, Ali. "Estrogen action in growth plate cartilage". Thesis, Högskolan i Skövde, Institutionen för vård och natur, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-5463.
Pełny tekst źródłaBarco, Andres. "Self-assembling peptides for cartilage regeneration". Thesis, University of Leeds, 2017. http://etheses.whiterose.ac.uk/19600/.
Pełny tekst źródłaChan, Alex Dart Ming. "Neurogenic modulation of articular cartilage degeneration". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ41123.pdf.
Pełny tekst źródłaCovert, Rebeccah Jean. "Durability evaluation of articular cartilage prostheses". Diss., Georgia Institute of Technology, 2003. http://hdl.handle.net/1853/17596.
Pełny tekst źródłaGribbon, Philip. "Biophysical properties of glycoaminoglycans and cartilage". Thesis, Imperial College London, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.294795.
Pełny tekst źródłaFinlay, Scott. "Towards acellular constructs for cartilage repair". Thesis, University of Leeds, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.612556.
Pełny tekst źródłaKatopodi, Theoni. "Cell based strategies for cartilage repair". Thesis, University of Manchester, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.491864.
Pełny tekst źródłaGoldsmith, Andrew Alan John. "Biphasic modelling of synthetic articular cartilage". Thesis, University of Bath, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.321846.
Pełny tekst źródłaArdill, Jennifer Maureen. "Optical measurement of articular cartilage roughness". Thesis, Queen's University Belfast, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.241325.
Pełny tekst źródłaBarton, Nicholas J. "Accurate assessment of articular cartilage roughness". Thesis, Queen's University Belfast, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.334495.
Pełny tekst źródłaGadher, S. J. "An enzymatic study of cartilage degradation". Thesis, University of Manchester, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.234208.
Pełny tekst źródła