Rozprawy doktorskie na temat „Cardiovascular physiology”
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McMurdo, Lorraine. "Endothelin in cardiovascular physiology and pathophysiology". Thesis, Queen Mary, University of London, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.321680.
Pełny tekst źródłaSpratt, James Christopher Samuel. "Endothelin : cardiovascular pharmacology, physiology & pathophysiology". Thesis, University of Edinburgh, 2003. http://hdl.handle.net/1842/23202.
Pełny tekst źródłaDeGrande, Sean Thomas. "Phosphatase regulation in cardiovascular physiology and disease". University of Iowa, 2012. http://ir.uiowa.edu/etd/3443.
Pełny tekst źródłaO'Neill, Mark. "Cardiovascular regulation under physiological stress". Thesis, University of Oxford, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.294358.
Pełny tekst źródłaNyarko-Adomfeh, Charles. "Cardiovascular function in normal man". Thesis, University of Nottingham, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.317595.
Pełny tekst źródłaWebb, D. J. "The endothelin system in human cardiovascular physiology and pathophysiology". Thesis, University of Edinburgh, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.663587.
Pełny tekst źródłaMuntean, Brian. "Roles of Primary Cilia in Cardiovascular and Renal Physiology". University of Toledo Health Science Campus / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=mco1396014586.
Pełny tekst źródłaMcIntosh, Matthew. "Long-term cardiovascular adaptations to neonatal hypoxia". Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=110447.
Pełny tekst źródłaINTRODUCTION: Des travaux antérieurs entrepris au laboratoire Rohlicek ont montré que l'hypoxie néonatale est associée à une élévation de la pression artérielle systolique chez les rats mâles âgés de deux mois. Dans le cadre de la présente étude, on demande si cette élévation persiste plus tard dans la maturité et si elle est également présente chez les femelles. On essaye en outre de déterminer si les hormones sexuelles ou des altérations dans le contrôle autonome jouent un rôle dans cette élévation. MÉTHODE: Des études ont été menées sur des rats adultes Sprague-Dawley des deux sexes. Un groupe expérimental a été élevé en hypoxie (FiO2 = 0,12) durant les dix premiers jours de vie et subséquemment en normaxie. Un second groupe a été élevé entièrement en normaxie. Un sous-ensemble de mâles et de femelles ont été gonadectomisés un mois avant la prise de mesures. À deux, trois et six mois, des rats étaient branchés à un transducteur de pression artérielle intravasculaire avec télémétrie pour surveillance de la pression de l'aorte abdominale. Une semaine plus tard, la pression artérielle a été mesurée durant 24 heures chez des rats ambulatoires et non contenus. RÉSULTATS: La pression systolique a été considérablement plus élevée chez des rats mâles en hypoxie à tout âge durant leur période active (nocturne) et également à 3 et 6 mois durant la période de repos (diurne) par comparaison aux rats du groupe témoin. L'ampleur de l'effet de l'hypoxie néonatale s'est accrue avec l'âge, bien que cette augmentation n'ait pas été statistiquement significative. Les femelles en hypoxie néonatale n'ont montré aucune différence dans la pression artérielle générale par comparaison aux femelles du groupe témoin. Tout comme la castration n'a pu empêcher l'apparition d'une pression artérielle élevée chez les mâles en hypoxie néonatale âgés de deux mois, l'ovariectomie de même n'a pu montrer une quelconque différence entre les femelles en hypoxie néonatale par opposition aux femelles du groupe témoin à l'âge de trois mois. Chez les rats mâles âgés de deux mois en hypoxie néonatale, la sensibilité du baroréflexe a été considérablement atrophiée durant leur période active (nocturne). CONCLUSION: Nos résultats indiquent que l'élévation de la pression artérielle chez les rats mâles adultes en hypoxie néonatale persiste plus tard dans la maturité. Cet effet semble être spécifique selon le sexe chez les animaux mâles. La découverte de la sensibilité du baroréflexe atrophiée à la suite de l'hypoxie néonatale à deux mois indique que le tonus autonome altéré, conjugué à une augmentation relative de l'activité sympathique, jouent un rôle dans l'élévation de la pression artérielle.
Bearham, D. A. "The cardiovascular consequences of burn injury". Thesis, University of Southampton, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.374058.
Pełny tekst źródłaSidery, Michael B. "The cardiovascular effects of food ingestion in man". Thesis, University of Nottingham, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.335380.
Pełny tekst źródłaGreen, Lucy Rebecca. "Mechanisms of fetal cardiovascular responses to acute hypoxia". Thesis, University College London (University of London), 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.338743.
Pełny tekst źródłaBrindle, Ryan C. "Peripheral physiological mechanisms of cardiovascular stress reactivity". Thesis, University of Birmingham, 2016. http://etheses.bham.ac.uk//id/eprint/6428/.
Pełny tekst źródłaWhite, Christopher Iain. "Cardiovascular 11β-HSD1 : its role in myocardial physiology and pathophysiology". Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/23391.
Pełny tekst źródłaMcNulty, Clare. "Ageing, inflammation and cardiovascular function". Thesis, University of Birmingham, 2016. http://etheses.bham.ac.uk//id/eprint/6541/.
Pełny tekst źródłaOdblom, Maria Pernilla. "The innervation and physiology of the cardiac tissue in squid". Thesis, University of Plymouth, 1997. http://hdl.handle.net/10026.1/2545.
Pełny tekst źródłaHooper, Justin Shane. "Cardiovascular Effects Evoked by Airway Nociceptive Reflexes in Healthy and Cardiovascular Diseased Rats". Scholar Commons, 2016. http://scholarcommons.usf.edu/etd/6258.
Pełny tekst źródłaRobertson-Gray, Olivia Jane. "Studies on the role of GPR55 in cardiovascular physiology and pathophysiology". Thesis, Robert Gordon University, 2017. http://hdl.handle.net/10059/3127.
Pełny tekst źródłaGUALA, ANDREA. "Mathematical modelling of cardiovascular fluid mechanics: physiology, pathology and clinical practice". Doctoral thesis, Politecnico di Torino, 2015. http://hdl.handle.net/11583/2615064.
Pełny tekst źródłaGallacher, David John. "The role of the posterior cerebellar vermis in cardiovascular control". Thesis, University College London (University of London), 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.309284.
Pełny tekst źródłaFardipour, Parvin. "Computer modelling of the human cardiovascular system based on relational analysis". Thesis, City University London, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.328932.
Pełny tekst źródłaSundblad, Patrik. "Effects of physical activity and gravitational stress on cardiovascular control /". Stockholm, 1999. http://diss.kib.ki.se/1999/91-628-3791-5/.
Pełny tekst źródłaHawkley, Louise C. "Loneliness, psychosocial states, behavior, and cardiovascular physiology : a 12-hour field study /". The Ohio State University, 2001. http://rave.ohiolink.edu/etdc/view?acc_num=osu1486399160107369.
Pełny tekst źródłaBagatto, Brian. "The Developmental Physiology of the Zebrafish: Influence of Environment and Cardiovascular Attributes". Thesis, University of North Texas, 2001. https://digital.library.unt.edu/ark:/67531/metadc2854/.
Pełny tekst źródłaTuttle, Hillary Ann. "Inflammation in diabetic women with cardiovascular disease". Diss., The University of Arizona, 2003. http://hdl.handle.net/10150/289992.
Pełny tekst źródłaGorr, Matthew W. "Air Pollution and Cardiovascular Dysfunction". The Ohio State University, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=osu1428674045.
Pełny tekst źródłaJourneay, William Shane. "Thermoregulatory and nonthermoregulatory interaction in human cardiovascular control". Thesis, University of Ottawa (Canada), 2003. http://hdl.handle.net/10393/26496.
Pełny tekst źródłaJohnson, Darren A. "Cardiovascular responses to physical stressors in normotensive and exercise hypertensive individuals". Thesis, University of Ottawa (Canada), 1995. http://hdl.handle.net/10393/10100.
Pełny tekst źródłaHanna, Brian Dale. "Control of sympathetic neuron and cardiovascular effector activity by carbon dioxide". Thesis, McGill University, 1988. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=75884.
Pełny tekst źródłaSun, Bing, i 孫冰. "Vestibular influence on central cardiovascular regulation in the rat: functional and anatomical aspects". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B31244774.
Pełny tekst źródłaTur, Jared. "Cardiovascular regulation by Kvβ1.1 subunit". Scholar Commons, 2016. http://scholarcommons.usf.edu/etd/6596.
Pełny tekst źródłaWang, Lu Electrical Engineering & Telecommunications Faculty of Engineering UNSW. "Experimental investigation and mathematical modelling of human cardiovascular system during exercise". Awarded by:University of New South Wales. Electrical Engineering & Telecommunications, 2007. http://handle.unsw.edu.au/1959.4/40598.
Pełny tekst źródłaNunn, Nicolas. "The role of the signalling protein XLalphas in cardiovascular control in mice". Thesis, University of Liverpool, 2012. http://livrepository.liverpool.ac.uk/9893/.
Pełny tekst źródłaDunworth, William P. Caron Kathleen M. "Genetic mouse models elucidate the roles of adrenomedullin in cardiovascular development and physiology". Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2009. http://dc.lib.unc.edu/u?/etd,2832.
Pełny tekst źródłaTitle from electronic title page (viewed Jun. 4, 2010). "... in partial fulfillment of the requirements for the degree of Doctor of Philosophy in the Curriculum in Genetics and Molecular Biology." Discipline: Genetics and Molecular Biology; Department/School: Medicine.
Penny, Daniel James. "Changes in integrated cardiovascular physiology during inotropic stimulation in the early postnatal period". Monash University, Institute of Reproduction and Development, 2004. http://arrow.monash.edu.au/hdl/1959.1/9661.
Pełny tekst źródłaCoccarelli, Alberto. "Understanding aspects of cardiovascular physiology and disease via a multi-physics modelling methodology". Thesis, Cardiff University, 2018. http://orca.cf.ac.uk/111464/.
Pełny tekst źródłaHammer, Fabian. "Corticosteroid hormone action in the cardiovascular system". Thesis, University of Birmingham, 2011. http://etheses.bham.ac.uk//id/eprint/1436/.
Pełny tekst źródłaBrooke-Wavell, Katherine S. F. "Human body composition : measurement and relationship with exercise, dietary intakes and cardiovascular risk factors". Thesis, Loughborough University, 1992. https://dspace.lboro.ac.uk/2134/17193.
Pełny tekst źródłaStevens, Sarah CW. "Calcium and Cancer: Implications for Cardiovascular Function and Disease". The Ohio State University, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=osu1337211154.
Pełny tekst źródłaAde, Carl Jacob. "Cardiorespiratory and vascular function during stress". Diss., Kansas State University, 2013. http://hdl.handle.net/2097/15976.
Pełny tekst źródłaDepartment of Anatomy and Physiology
Thomas J. Barstow
The primary aim of this dissertation was to evaluate the factors that contribute to the cardiorespiratory and vascular responses following exercise conditioning and microgravity deconditioning. The first study of this dissertation (Chapter 2) revealed that exercise training in the head down tilt posture, which places increases central blood volume compared to upright, results in cardiorespiratory adaptations in both upright and head down tilt postures which are not completely expressed with exercise training in the upright posture. These findings suggest that augmentation of the ventricular volume load during exercise training may result in adaptations that transfer across multiple body positions. In the second and third studies measurements of blood velocity and flow were performed via Doppler ultrasound. In Chapter 3 we observed that in the brachial and femoral arteries blood moves with a slightly blunted parabolic velocity profile that is very stable across a range of mean arterial pressures and downstream limb resistances. We concluded that these findings support the current calculations of shear rate based on the assumptions of laminar flow. With these assumptions confirmed, the investigation in Chapter 4 could be performed. We observed that acute exposure to a sustained antegrade shear rate, via unilateral forearm heating, increased measurements of flow-mediated dilation and the overall rate of adjustment for forearm blood flow and vascular conductance during dynamic handgrip exercise. These findings suggest that one potential stimulus for improvements in vascular function and health following exercise conditioning may be exposure to elevations in antegrade shear. Lastly in Chapter 5 we changed focus to the cardiorespiratory deconditioning following long-duration microgravity exposure. We retrospectively reviewed and analyzed previous investigations of microgravity deconditioning and demonstrated that the decrease in maximal O2 consumption ( O2max) occurs as a function of duration of exposure and that both convective and diffusive O2 transport pathways substantially contribute to this decline. In addition we reviewed the current literature and highlighted potential mechanisms, across several organ systems, which may contribute to this decline in O2max. Collectively, these studies revealed the breath of plasticity for cardiorespiratory adaptations to a variety of stressors.
Rook, William. "The effects of chronic hypoxia in utero on cardiovascular regulation in the offspring". Thesis, University of Birmingham, 2011. http://etheses.bham.ac.uk//id/eprint/3040/.
Pełny tekst źródłaWilliams, Daniel Patrick. "Dehydroepiandrosterone sulfate, resistive exercise training and cardiovascular disease risk factors in premenopausal females". Diss., The University of Arizona, 1993. http://hdl.handle.net/10150/186281.
Pełny tekst źródłaMersereau, Eric, Cody Boyle, Shelby Poitra, Ana Espinoza, Joclyn Seiler, Robert Longie, Lisa Delvo i in. "Longitudinal Effects of Embryonic Exposure to Cocaine on Morphology, Cardiovascular Physiology, and Behavior in Zebrafish". MDPI AG, 2016. http://hdl.handle.net/10150/618721.
Pełny tekst źródłaWang, Tse-Yao. "Impacts of Moderate Cold Exposure on Energy Metabolism, Obesity, and Cardiovascular Function". The Ohio State University, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=osu1384449386.
Pełny tekst źródłaLamensdorf, Angela Mona-Lisa. "Cardiovascular risk and autonomic changes during high and low affect provocations". Thesis, University of British Columbia, 1988. http://hdl.handle.net/2429/28098.
Pełny tekst źródłaArts, Faculty of
Psychology, Department of
Graduate
Hemesath, Susan M. "Effects of acute aerobic exercise on cardiovascular reactivity to stress in healthy, untrained males". Thesis, This resource online, 1990. http://scholar.lib.vt.edu/theses/available/etd-06102009-063023/.
Pełny tekst źródłaAyers, Misty. "EFFECTS OF HYPOXIC REARING ENVIRONMENT ON THE DEVELOPMENT, PHYSIOLOGY, AND ECOLOGY OF ZEBRAFISH". University of Akron / OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=akron1153495631.
Pełny tekst źródłaKoon, Hon-wai Michael, i 管漢偉. "Role of endothelin-1 and nitric oxide on the cardiovascular function". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2002. http://hub.hku.hk/bib/B31245444.
Pełny tekst źródłaHartwich, Doreen. "Neural cardiovascular control during exercise : influence of sex and ovarian hormones". Thesis, University of Birmingham, 2012. http://etheses.bham.ac.uk//id/eprint/3218/.
Pełny tekst źródłaNephew, Benjamin Charles. "Simultaneous modulation of behavioral, cardiovascular, and corticosterone responses to acute stress, with an emphasis on arginine vasotocin /". Thesis, Connect to Dissertations & Theses @ Tufts University, 2003.
Znajdź pełny tekst źródłaAdviser: L. Michael Romero. Submitted to the Dept. of Biology. Includes bibliographical references (leaves 158-180). Access restricted to members of the Tufts University community. Also available via the World Wide Web;
Barrett-O'Keefe, Zachary. "Cardiovascular control during exercise and the role of the sympathetic nervous system in heart failure with reduced ejection fraction". Thesis, The University of Utah, 2016. http://pqdtopen.proquest.com/#viewpdf?dispub=10001028.
Pełny tekst źródłaThe objective of this dissertation was to systematically investigate the hemodynamic response to exercise in heart failure with reduced ejection fraction (HFrEF) and healthy individuals of a similar age, with an emphasis on how the sympathetic nervous system (SNS) may contribute to the dysregulation of the cardiovascular system in this cohort. The first study aimed to determine how varying levels of metaboreceptor activation alters the mean arterial pressure (MAP) response as well as the degree in which cardiac output (CO) and systemic vascular conductance (SVC) contribute to the metaboreflex-induced increase in MAP. We observed similar increases in MAP induced by metaboreceptor activation in both groups; however, this response was driven primarily by increases in CO in the control group and reductions in SVC in the HFrEF group. These data suggest a preserved role of the metaboreflex-induced increase in MAP in HFrEF, but suggest that this response is governed by the peripheral circulation in this cohort, a maladaptation that may exacerbate systolic dysfunction through an increase in afterload. The second study of this dissertation was focused on investigating the peripheral vasodilatory and hyperemic response to exercise in isolation of central hemodynamic limitations in both the upper and lower limbs. This study documented an impaired hyperemic response to both static-intermittent handgrip exercise as well as dynamic single-leg knee-extensor exercise in HFrEF patients - impairments primarily attributed to vasodilatory dysfunction, as the increase in MAP induced by these exercise modalities was preserved compared to healthy individuals. Together, these findings have identified a significant attenuation of the exercise-induced hyperemic response during both upper and lower limb exercise, implicating maladaptions in the peripheral hemodynamic response to exercise as a potential contributor limiting exercise capacity in this patient group. The third study sought to address the contribution of the alpha-adrenergic receptor pathway in the regulation of blood flow to exercising skeletal muscle in HFrEF patients. At rest, alpha-1-adrenergic receptor vasoconstriction induced by local intra-arterial infusion of phenylephrine (PE) was reduced in HFrEF compared to control subjects. During exercise, the vasoconstrictor responsiveness to PE was significantly attenuated in the control group and preserved in HFrEF patients compared to rest. Additionally, nonspecific alpha-adrenergic receptor antagonism induced by local intra-arterial infusion of phentolamine increased blood flow to a greater degree in HFrEF compared to the control subjects, both at rest and during exercise. Together, these findings demonstrate a marked contribution of alpha-adrenergic receptor restraint of leg blood flow in HFrEF patients during exercise. Collectively, these three studies have provided new insight into the role the SNS and peripheral hemodynamics play in the maladaptive cardiovascular response to exercise displayed in patients with HFrEF, further implicating the peripheral expression of SNS activity as a primary contributor to impaired exercise capacity in this patient group.