Gotowa bibliografia na temat „Cardio-Metabolism risk”
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Artykuły w czasopismach na temat "Cardio-Metabolism risk"
Chandiok, Ketaki, i Rakash Ranjan Monda. "One Carbon Metabolism and Cardio-Metabolic risk". Voice of Intellectual Man- An International Journal 7, nr 2 (2017): 135. http://dx.doi.org/10.5958/2319-4308.2017.00029.9.
Pełny tekst źródłaLi, Ling-Jun, Izzuddin M. Aris, Lin Lin Su, Yap Seng Chong, Tien Yin Wong, Kok Hian Tan i Jie Jin Wang. "Effect of gestational diabetes and hypertensive disorders of pregnancy on postpartum cardiometabolic risk". Endocrine Connections 7, nr 3 (marzec 2018): 433–42. http://dx.doi.org/10.1530/ec-17-0359.
Pełny tekst źródłaKashukh, Ye A., i V. T. Ivashkin. "Probiotics, metabolism and the functional condition of cardio-vascular system". Russian Journal of Gastroenterology, Hepatology, Coloproctology 26, nr 1 (12.08.2016): 8–14. http://dx.doi.org/10.22416/1382-4376-2016-26-1-8-14.
Pełny tekst źródłaFatunde, Olubadewa A., i Sherry-Ann Brown. "The Role of CYP450 Drug Metabolism in Precision Cardio-Oncology". International Journal of Molecular Sciences 21, nr 2 (17.01.2020): 604. http://dx.doi.org/10.3390/ijms21020604.
Pełny tekst źródłaRonchi, Cristina L., Elisa Verrua, Emanuele Ferrante, Gwendolyn Bender, Elisa Sala, Andrea G. Lania, Martin Fassnacht i in. "Long-term effects of radiotherapy on cardiovascular risk factors in acromegaly". European Journal of Endocrinology 164, nr 5 (maj 2011): 675–84. http://dx.doi.org/10.1530/eje-10-1105.
Pełny tekst źródłaRajak, Devraj, Deepali Sahu, Anushree Jain, Rubeena Khan, Basant Khare, Prateek Kumar Jain i Bhupendra Singh Thakur. "Review on Toxicity of Antihypertensive Drugs". Asian Journal of Dental and Health Sciences 2, nr 4 (15.12.2022): 64–68. http://dx.doi.org/10.22270/ajdhs.v2i4.28.
Pełny tekst źródłaKiluk, Małgorzata, Janina Lewkowicz, Dariusz Pawlak i Anna Tankiewicz-Kwedlo. "Crosstalk between Tryptophan Metabolism via Kynurenine Pathway and Carbohydrate Metabolism in the Context of Cardio-Metabolic Risk—Review". Journal of Clinical Medicine 10, nr 11 (4.06.2021): 2484. http://dx.doi.org/10.3390/jcm10112484.
Pełny tekst źródłaCuciureanu, Tudor, Stefan Chiriac, Madalina Chiorescu, Irina Girleanu i Anca Trifan. "Chronic hepatitis C virus infection: a new modifiable cardio-metabolic risk factor ?" Medicine and Pharmacy Reports 90, nr 3 (25.07.2017): 251–55. http://dx.doi.org/10.15386/cjmed-793.
Pełny tekst źródłaKovtun, O. P., i P. B. Tsyvian. "MATERNAL NUTRITION AND THE PROBLEM OF INTRAUTERINE DISEASE PROGRAMMING IN CHILDREN". Annals of the Russian academy of medical sciences 73, nr 3 (9.07.2018): 190–94. http://dx.doi.org/10.15690/vramn988.
Pełny tekst źródłaPodzolkov, V. I., E. A. Gladysheva i A. E. Bragina. "Moxonidin in combined therapy of metabolic syndrome". "Arterial’naya Gipertenziya" ("Arterial Hypertension") 13, nr 4 (28.08.2007): 246–49. http://dx.doi.org/10.18705/1607-419x-2007-13-4-246-249.
Pełny tekst źródłaRozprawy doktorskie na temat "Cardio-Metabolism risk"
Rigal, Eve. "Impact à long terme de la programmation nutritionnelle postnatale sur le risque cardio-métabolisme et sur la sensibilité aux lésions d'ischémie-reperfusion in vivo chez la souris". Electronic Thesis or Diss., Bourgogne Franche-Comté, 2023. http://www.theses.fr/2023UBFCI017.
Pełny tekst źródłaNutritional disorders occurring during the foetal and postnatal periods may be responsible for a predisposition to cardio-metabolic diseases in adulthood. The PostNatal OverFeeding (PNOF) model, induced by litter size reduction, pertinently recapitulates the pathophysiological mechanisms of a programmed increase of cardiometabolic risk in Humans. However, most of preclinical data has been obtained in young male animals.In male mice, PNOF induced an increase in body weight associated with glucose metabolism impairment at young and older ages (4 to 18 months). Cardiac contractile function was reduced by PNOF and the sensitivity of hearts to in vivo ischemia-reperfusion (I/R) injury was increased and associated with an augmentation in pericardial adipose tissue mass and inflammatory status. In females, PNOF induced similar cardio-metabolic alterations, however, the sensitivity of hearts to I/R lesions induced in vivo was not impacted.These results show that PNOF predisposes the organism of male and female mice to cardio-metabolic diseases from the young (4 months) to the older ages (up to 18 months); however, PNOF only affects males for an increased sensitivity to ischemic cardiac lesions.This original model of PNOF could constitute a model of “natural” metabolic syndrome on which physiopathological and therapeutic studies could be carried out
Części książek na temat "Cardio-Metabolism risk"
Jacob, Stephan, Matthias Leschke i Andrew J. Krentz. "Type 2 diabetes management: from a glucose-centric approach to cardio-renal risk reduction". W Cardiovascular Endocrinology and Metabolism, 221–32. Elsevier, 2023. http://dx.doi.org/10.1016/b978-0-323-99991-5.00013-9.
Pełny tekst źródłaStreszczenia konferencji na temat "Cardio-Metabolism risk"
Kolyaskina, M. M., A. A. Kislyakova i P. Sh Osmanova. "EMPLOYEES OPERATING AND MAINTAINING WIRED AND WIRELESS COMMUNICATION DEVICES CARDIOVASCULAR RISK DEVELOPMENT MONITORING". W The 4th «OCCUPATION and HEALTH» International Youth Forum (OHIYF-2022). FSBSI «IRIOH», 2022. http://dx.doi.org/10.31089/978-5-6042929-6-9-2022-1-124-127.
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