Artykuły w czasopismach na temat „Cardiac hypertrophy”
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Li, Wei-ming, Yi-fan Zhao, Guo-fu Zhu, Wen-hui Peng, Meng-yun Zhu, Xue-jing Yu, Wei Chen, Da-chun Xu i Ya-wei Xu. "Dual specific phosphatase 12 ameliorates cardiac hypertrophy in response to pressure overload". Clinical Science 131, nr 2 (23.12.2016): 141–54. http://dx.doi.org/10.1042/cs20160664.
Pełny tekst źródłaWehbe, Nadine, Suzanne Nasser, Gianfranco Pintus, Adnan Badran, Ali Eid i Elias Baydoun. "MicroRNAs in Cardiac Hypertrophy". International Journal of Molecular Sciences 20, nr 19 (23.09.2019): 4714. http://dx.doi.org/10.3390/ijms20194714.
Pełny tekst źródłaStrøm, Claes C., Mogens Kruhøffer, Steen Knudsen, Frank Stensgaard-Hansen, Thomas E. N. Jonassen, Torben F. Ørntoft, Stig Haunsø i Søren P. Sheikh. "Identification of a Core Set of Genes That Signifies Pathways Underlying Cardiac Hypertrophy". Comparative and Functional Genomics 5, nr 6-7 (2004): 459–70. http://dx.doi.org/10.1002/cfg.428.
Pełny tekst źródłaBazgir, Farhad, Julia Nau, Saeideh Nakhaei-Rad, Ehsan Amin, Matthew J. Wolf, Jeffry J. Saucerman, Kristina Lorenz i Mohammad Reza Ahmadian. "The Microenvironment of the Pathogenesis of Cardiac Hypertrophy". Cells 12, nr 13 (4.07.2023): 1780. http://dx.doi.org/10.3390/cells12131780.
Pełny tekst źródłaZhang, Yan, Qiang Da, Siyi Cao, Ke Yan, Zhiguang Shi, Qing Miao, Chen Li i in. "HINT1 (Histidine Triad Nucleotide-Binding Protein 1) Attenuates Cardiac Hypertrophy Via Suppressing HOXA5 (Homeobox A5) Expression". Circulation 144, nr 8 (24.08.2021): 638–54. http://dx.doi.org/10.1161/circulationaha.120.051094.
Pełny tekst źródłaLu, Peilei, Danyu Zhang, Fan Ding, Jialu Ma, Yang K. Xiang i Meimi Zhao. "Silencing of circCacna1c Inhibits ISO-Induced Cardiac Hypertrophy through miR-29b-2-5p/NFATc1 Axis". Cells 12, nr 12 (19.06.2023): 1667. http://dx.doi.org/10.3390/cells12121667.
Pełny tekst źródłaLi, Yuhao, Yoshihiko Saito, Koichiro Kuwahara, Xianglu Rong, Ichiro Kishimoto, Masaki Harada, Yuichiro Adachi i in. "Guanylyl Cyclase-A Inhibits Angiotensin II Type 2 Receptor-Mediated Pro-Hypertrophic Signaling in the Heart". Endocrinology 150, nr 8 (16.04.2009): 3759–65. http://dx.doi.org/10.1210/en.2008-1353.
Pełny tekst źródłaLi, Yu, Bo He, Chao Zhang, Yanji He, Tianyang Xia i Chunyu Zeng. "Naringenin Attenuates Isoprenaline-Induced Cardiac Hypertrophy by Suppressing Oxidative Stress through the AMPK/NOX2/MAPK Signaling Pathway". Nutrients 15, nr 6 (9.03.2023): 1340. http://dx.doi.org/10.3390/nu15061340.
Pełny tekst źródłaJohansson, Markus, Benyapa Tangruksa, Sepideh Heydarkhan-Hagvall, Anders Jeppsson, Peter Sartipy i Jane Synnergren. "Data Mining Identifies CCN2 and THBS1 as Biomarker Candidates for Cardiac Hypertrophy". Life 12, nr 5 (12.05.2022): 726. http://dx.doi.org/10.3390/life12050726.
Pełny tekst źródłaHu, Chengyun, Feibiao Dai, Jiawu Wang, Lai Jiang, Di Wang, Jie Gao, Jun Huang i in. "Peroxiredoxin-5 Knockdown Accelerates Pressure Overload-Induced Cardiac Hypertrophy in Mice". Oxidative Medicine and Cellular Longevity 2022 (29.01.2022): 1–12. http://dx.doi.org/10.1155/2022/5067544.
Pełny tekst źródłaRaghunathan, Suchi, Ramesh K. Goyal i Bhoomika M. Patel. "Selective inhibition of HDAC2 by magnesium valproate attenuates cardiac hypertrophy". Canadian Journal of Physiology and Pharmacology 95, nr 3 (marzec 2017): 260–67. http://dx.doi.org/10.1139/cjpp-2016-0542.
Pełny tekst źródłaGallo, Simona, Annapia Vitacolonna, Alessandro Bonzano, Paolo Comoglio i Tiziana Crepaldi. "ERK: A Key Player in the Pathophysiology of Cardiac Hypertrophy". International Journal of Molecular Sciences 20, nr 9 (1.05.2019): 2164. http://dx.doi.org/10.3390/ijms20092164.
Pełny tekst źródłaJohansson, Markus, Benjamin Ulfenborg, Christian X. Andersson, Sepideh Heydarkhan-Hagvall, Anders Jeppsson, Peter Sartipy i Jane Synnergren. "Multi-Omics Characterization of a Human Stem Cell-Based Model of Cardiac Hypertrophy". Life 12, nr 2 (16.02.2022): 293. http://dx.doi.org/10.3390/life12020293.
Pełny tekst źródłaBi, Hai-Lian, Xiao-Li Zhang, Yun-Long Zhang, Xin Xie, Yun-Long Xia, Jie Du i Hui-Hua Li. "The deubiquitinase UCHL1 regulates cardiac hypertrophy by stabilizing epidermal growth factor receptor". Science Advances 6, nr 16 (kwiecień 2020): eaax4826. http://dx.doi.org/10.1126/sciadv.aax4826.
Pełny tekst źródłaDeng, Yawen, Zhitong Li, Xiangbo An, Rui Fan, Yao Wang, Jiatian Li, Xiaolei Yang, Jiawei Liao i Yunlong Xia. "Hyperhomocysteinemia Promotes Cardiac Hypertrophy in Hypertension". Oxidative Medicine and Cellular Longevity 2022 (22.08.2022): 1–16. http://dx.doi.org/10.1155/2022/1486157.
Pełny tekst źródłaGu, Wei, Yutong Cheng, Su Wang, Tao Sun i Zhizhong Li. "PHD Finger Protein 19 Promotes Cardiac Hypertrophy via Epigenetically Regulating SIRT2". Cardiovascular Toxicology 21, nr 6 (21.02.2021): 451–61. http://dx.doi.org/10.1007/s12012-021-09639-0.
Pełny tekst źródłaGolomb, E., Z. A. Abassi, G. Cuda, M. Stylianou, V. R. Panchal, D. Trachewsky i H. R. Keiser. "Angiotensin II maintains, but does not mediate, isoproterenol-induced cardiac hypertrophy in rats". American Journal of Physiology-Heart and Circulatory Physiology 267, nr 4 (1.10.1994): H1496—H1506. http://dx.doi.org/10.1152/ajpheart.1994.267.4.h1496.
Pełny tekst źródłaHarmsen, Eef, i Frans H. H. Leenen. "Dietary sodium induced cardiac hypertrophy". Canadian Journal of Physiology and Pharmacology 70, nr 4 (1.04.1992): 580–86. http://dx.doi.org/10.1139/y92-073.
Pełny tekst źródłaCooper, George. "Cytoskeletal networks and the regulation of cardiac contractility: microtubules, hypertrophy, and cardiac dysfunction". American Journal of Physiology-Heart and Circulatory Physiology 291, nr 3 (wrzesień 2006): H1003—H1014. http://dx.doi.org/10.1152/ajpheart.00132.2006.
Pełny tekst źródłaGao, Si, Xue-ping Liu, Li-hua Wei, Jing Lu i Peiqing Liu. "Upregulation of α-enolase protects cardiomyocytes from phenylephrine-induced hypertrophy". Canadian Journal of Physiology and Pharmacology 96, nr 4 (kwiecień 2018): 352–58. http://dx.doi.org/10.1139/cjpp-2017-0282.
Pełny tekst źródłaLi, Peng-Long, Hui Liu, Guo-Peng Chen, Ling Li, Hong-Jie Shi, Hong-Yu Nie, Zhen Liu i in. "STEAP3 (Six-Transmembrane Epithelial Antigen of Prostate 3) Inhibits Pathological Cardiac Hypertrophy". Hypertension 76, nr 4 (październik 2020): 1219–30. http://dx.doi.org/10.1161/hypertensionaha.120.14752.
Pełny tekst źródłaTappia, Paramjit, Vijayan Elimban i Naranjan Dhalla. "Involvement of phospholipase C in the norepinephrine-induced hypertrophic response in Cardiomyocytes". Scripta Medica 53, nr 2 (2022): 149–57. http://dx.doi.org/10.5937/scriptamed53-36527.
Pełny tekst źródłaXie, Xin, Hai-Lian Bi, Song Lai, Yun-Long Zhang, Nan Li, Hua-Jun Cao, Ling Han, Hong-Xia Wang i Hui-Hua Li. "The immunoproteasome catalytic β5i subunit regulates cardiac hypertrophy by targeting the autophagy protein ATG5 for degradation". Science Advances 5, nr 5 (maj 2019): eaau0495. http://dx.doi.org/10.1126/sciadv.aau0495.
Pełny tekst źródłaRuzicka, M., i F. H. Leenen. "Renin-angiotensin system and minoxidil-induced cardiac hypertrophy in rats". American Journal of Physiology-Heart and Circulatory Physiology 265, nr 5 (1.11.1993): H1551—H1556. http://dx.doi.org/10.1152/ajpheart.1993.265.5.h1551.
Pełny tekst źródłaSari, Nurmila, Yasufumi Katanasaka, Hiroki Honda, Yusuke Miyazaki, Yoichi Sunagawa, Masafumi Funamoto, Kana Shimizu i in. "Cacao Bean Polyphenols Inhibit Cardiac Hypertrophy and Systolic Dysfunction in Pressure Overload-induced Heart Failure Model Mice". Planta Medica 86, nr 17 (9.07.2020): 1304–12. http://dx.doi.org/10.1055/a-1191-7970.
Pełny tekst źródłaChung, Eunhee, Fan Yeung i Leslie A. Leinwand. "Akt and MAPK signaling mediate pregnancy-induced cardiac adaptation". Journal of Applied Physiology 112, nr 9 (1.05.2012): 1564–75. http://dx.doi.org/10.1152/japplphysiol.00027.2012.
Pełny tekst źródłaLiu, Yao-Lung, Chiu-Ching Huang, Chiz-Chung Chang, Che-Yi Chou, Shih-Yi Lin, I.-Kuan Wang, Dennis Jine-Yuan Hsieh, Gwo-Ping Jong, Chih-Yang Huang i Chao-Min Wang. "Hyperphosphate-Induced Myocardial Hypertrophy through the GATA-4/NFAT-3 Signaling Pathway Is Attenuated by ERK Inhibitor Treatment". Cardiorenal Medicine 5, nr 2 (2015): 79–88. http://dx.doi.org/10.1159/000371454.
Pełny tekst źródłaKee, Hae Jin, i Hyun Kook. "Roles and Targets of Class I and IIa Histone Deacetylases in Cardiac Hypertrophy". Journal of Biomedicine and Biotechnology 2011 (2011): 1–10. http://dx.doi.org/10.1155/2011/928326.
Pełny tekst źródłaZeng, Si-yu, Qiu-jiang Yan, Li Yang, Qing-hua Mei i Hui-qin Lu. "Inhibition of the ROS-EGFR Pathway Mediates the Protective Action of Nox1/4 Inhibitor GKT137831 against Hypertensive Cardiac Hypertrophy via Suppressing Cardiac Inflammation and Activation of Akt and ERK1/2". Mediators of Inflammation 2020 (4.08.2020): 1–14. http://dx.doi.org/10.1155/2020/1078365.
Pełny tekst źródłaLi, Shujun, Bo Huang, Changfei Zhou, Jingshan Shi, Qin Wu i Qingsong Jiang. "Rutaecarpine Ameliorates Pressure Overload Cardiac Hypertrophy by Suppression of Calcineurin and Angiotensin II". Evidence-Based Complementary and Alternative Medicine 2021 (15.01.2021): 1–8. http://dx.doi.org/10.1155/2021/8857329.
Pełny tekst źródłaGopinath, Bamini, Ronald J. Trent i Bing Yu. "Molecular characterisation of neonatal cardiac hypertrophy and its regression". Cardiology in the Young 14, nr 5 (październik 2004): 498–505. http://dx.doi.org/10.1017/s1047951104005062.
Pełny tekst źródłaYan, Xiaoying, Ran Zhao, Xiaorong Feng, Jingzhou Mu, Ying Li, Yue Chen, Chunmei Li i in. "Sialyltransferase7A promotes angiotensin II-induced cardiomyocyte hypertrophy via HIF-1α-TAK1 signalling pathway". Cardiovascular Research 116, nr 1 (11.03.2019): 114–26. http://dx.doi.org/10.1093/cvr/cvz064.
Pełny tekst źródłaDorn, Lisa E., William Lawrence, Jennifer M. Petrosino, Xianyao Xu, Thomas J. Hund, Bryan A. Whitson, Matthew S. Stratton i in. "Microfibrillar-Associated Protein 4 Regulates Stress-Induced Cardiac Remodeling". Circulation Research 128, nr 6 (19.03.2021): 723–37. http://dx.doi.org/10.1161/circresaha.120.317146.
Pełny tekst źródłaXie, Yifan, Yang Gao, Rifeng Gao, Wenlong Yang, Zheng Dong, Robb E. Moses, Aijun Sun, Xiaotao Li i Junbo Ge. "The proteasome activator REGγ accelerates cardiac hypertrophy by declining PP2Acα–SOD2 pathway". Cell Death & Differentiation 27, nr 10 (18.05.2020): 2952–72. http://dx.doi.org/10.1038/s41418-020-0554-8.
Pełny tekst źródłaYu, Qing, Wenxin Kou, Xu Xu, Shunping Zhou, Peipei Luan, Xiaopeng Xu, Hailing Li i in. "FNDC5/Irisin inhibits pathological cardiac hypertrophy". Clinical Science 133, nr 5 (1.03.2019): 611–27. http://dx.doi.org/10.1042/cs20190016.
Pełny tekst źródłaXu, Si-Chi, Zhen-Guo Ma, Wen-Ying Wei, Yu-Pei Yuan i Qi-Zhu Tang. "Bezafibrate Attenuates Pressure Overload-Induced Cardiac Hypertrophy and Fibrosis". PPAR Research 2017 (2017): 1–12. http://dx.doi.org/10.1155/2017/5789714.
Pełny tekst źródłaGeraets, Ilvy M. E., Will A. Coumans, Agnieszka Strzelecka, Patrick Schönleitner, Gudrun Antoons, Francesco Schianchi, Myrthe M. A. Willemars i in. "Metabolic Interventions to Prevent Hypertrophy-Induced Alterations in Contractile Properties In Vitro". International Journal of Molecular Sciences 22, nr 7 (31.03.2021): 3620. http://dx.doi.org/10.3390/ijms22073620.
Pełny tekst źródłaZhang, Ning, Wen-Ying Wei, Zheng Yang, Yan Che, Ya-Ge Jin, Hai-Han Liao, Sha-sha Wang, Wei Deng i Qi-Zhu Tang. "Nobiletin, a Polymethoxy Flavonoid, Protects Against Cardiac Hypertrophy Induced by Pressure-Overload via Inhibition of NAPDH Oxidases and Endoplasmic Reticulum Stress". Cellular Physiology and Biochemistry 42, nr 4 (2017): 1313–25. http://dx.doi.org/10.1159/000478960.
Pełny tekst źródłaYao, Rui, Lingyao Kong, Chunlei Yang, Jiaqi Du, Guojun Zhao i Yapeng Li. "Tumor Necrosis Factor-α-Induced Protein 8-Like 2 Ameliorates Cardiac Hypertrophy by Targeting TLR4 in Macrophages". Oxidative Medicine and Cellular Longevity 2022 (26.04.2022): 1–13. http://dx.doi.org/10.1155/2022/9469143.
Pełny tekst źródłaVilleneuve, C., A. Caudrillier, C. Ordener, N. Pizzinat, A. Parini i J. Mialet-Perez. "Dose-dependent activation of distinct hypertrophic pathways by serotonin in cardiac cells". American Journal of Physiology-Heart and Circulatory Physiology 297, nr 2 (sierpień 2009): H821—H828. http://dx.doi.org/10.1152/ajpheart.00345.2009.
Pełny tekst źródłaLucas, Aline Maria Brito, Joana Varlla de Lacerda Alexandre, Maria Thalyne Silva Araújo, Cicera Edna Barbosa David, Yuana Ivia Ponte Viana, Beatriz Neves Coelho, Francisco Rodrigo Lemos Caldas, Anna Lídia Nunes Varela, Alicia Juliana Kowaltowski i Heberty Tarso Facundo. "Diazoxide Modulates Cardiac Hypertrophy by Targeting H2O2 Generation and Mitochondrial Superoxide Dismutase Activity". Current Molecular Pharmacology 13, nr 1 (15.01.2020): 76–83. http://dx.doi.org/10.2174/1874467212666190723144006.
Pełny tekst źródłaLiu, Yaoqiu, Yahui Shen, Jingai Zhu, Ming Liu, Xing Li, Yumei Chen, Xiangqing Kong, Guixian Song i Lingmei Qian. "Cardiac-Specific PID1 Overexpression Enhances Pressure Overload-Induced Cardiac Hypertrophy in Mice". Cellular Physiology and Biochemistry 35, nr 5 (2015): 1975–85. http://dx.doi.org/10.1159/000374005.
Pełny tekst źródłaMontiel, Virginie, Ramona Bella, Lauriane Y. M. Michel, Hrag Esfahani, Delphine De Mulder, Emma L. Robinson, Jean-Philippe Deglasse i in. "Inhibition of aquaporin-1 prevents myocardial remodeling by blocking the transmembrane transport of hydrogen peroxide". Science Translational Medicine 12, nr 564 (7.10.2020): eaay2176. http://dx.doi.org/10.1126/scitranslmed.aay2176.
Pełny tekst źródłaWei, Xiangxiang, Jiayu Jin, Jian Wu, Yunquan He, Jieyu Guo, Zhaohua Yang, Liang Chen i in. "Cardiac-specific BACH1 ablation attenuates pathological cardiac hypertrophy by inhibiting the Ang II type 1 receptor expression and the Ca2+/CaMKII pathway". Cardiovascular Research, 3.06.2023. http://dx.doi.org/10.1093/cvr/cvad086.
Pełny tekst źródłaKwon, Duk-hwa, Gwang Hyeon Eom, Hae Jin Kee, Yoonseok Nam, Young Kuk Cho, Don-Kyu Kim, Jayoung Koo i in. "Abstract 071: Estrogen-related Receptor Gamma Induces Cardiac Hypertrophy By Activating Gata4". Circulation Research 113, suppl_1 (sierpień 2013). http://dx.doi.org/10.1161/res.113.suppl_1.a071.
Pełny tekst źródłaWang, Wei, Nian Liu, Li Xin, Yanfei Ruan, Xin Du, Rong Bai, Jianzeng Dong i ChangSheng Ma. "RETRACTED ARTICLE: Inhibition of miR-296-5p protects the heart from cardiac hypertrophy by targeting CACNG6". Gene Therapy, 16.12.2019. http://dx.doi.org/10.1038/s41434-019-0109-0.
Pełny tekst źródłaSu, Haibi, Jie Xu, Zhenghua Su, Chenxi Xiao, Jinghuan Wang, Wen Zhong, Chen Meng, Di Yang i Yizhun Zhu. "Poly (ADP-ribose) polymerases 16 triggers pathological cardiac hypertrophy via activating IRE1α–sXBP1–GATA4 pathway". Cellular and Molecular Life Sciences 80, nr 6 (23.05.2023). http://dx.doi.org/10.1007/s00018-023-04805-9.
Pełny tekst źródłaRind, Jubran, Nagib Chalfoun i Richard McNamara. "Cardiac amyloidosis: The great masquerader". Global Cardiology Science and Practice 2018, nr 2 (23.07.2018). http://dx.doi.org/10.21542/gcsp.2018.18.
Pełny tekst źródłaXiao, Zheng, Bin Kong, Hongjie Yang, Chang Dai, Jin Fang, Tianyou Qin i He Huang. "Key Player in Cardiac Hypertrophy, Emphasizing the Role of Toll-Like Receptor 4". Frontiers in Cardiovascular Medicine 7 (26.11.2020). http://dx.doi.org/10.3389/fcvm.2020.579036.
Pełny tekst źródłaChen, Wen-Jing, Yan Cheng, Wen Li, Xiao-Kang Dong, Jian-liang Wei, Chuan-Hua Yang i Yue-Hua Jiang. "Quercetin Attenuates Cardiac Hypertrophy by Inhibiting Mitochondrial Dysfunction Through SIRT3/PARP-1 Pathway". Frontiers in Pharmacology 12 (28.10.2021). http://dx.doi.org/10.3389/fphar.2021.739615.
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