Rozprawy doktorskie na temat „Cancer – Microbiology”
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Arat, Seda. "A Systems Biology Approach to Microbiology and Cancer". Diss., Virginia Tech, 2015. http://hdl.handle.net/10919/75149.
Pełny tekst źródłaPh. D.
Al, Kamal Nasrah Ali. "Immunotherapy for human breast cancer". Wright State University / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=wright1452814566.
Pełny tekst źródłaCiesielski, Francisco Isaak Nícolas [UNESP]. "Microrganismos oportunistas e exógenos na microbiota bucal de pacientes oncológicos submetidos à radioterapia de cabeça e pescoço". Universidade Estadual Paulista (UNESP), 2010. http://hdl.handle.net/11449/91426.
Pełny tekst źródłaFundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)
O objetivo deste estudo foi avaliar a ocorrência de microrganismos exógenos e oportunistas (bactérias entéricas, pseudomonados, leveduras e Helicobacter pylori) na cavidade bucal de pacientes submetidos à radioterapia para tratamento de câncer de cabeça e pescoço. Cincoenta pacientes que iria receber radioterapia foram examinados antes, durante e 30 dias após radioterapia. Amostras de saliva, mucosa e biofilme foram coletadas e os microrganismos foram detectados por cultura e Reação em Cadeia da Polimerase (PCR). Candida albicans, C. tropicalis, C. krusei, C. glabrata e C. parapsilosis foram as leveduras mais prevalentes nos pacientes submetidos a radioterapia. Gêneros Citrobacter, Enterobacter, Enterococcus, Klebsiella, Proteus, e Pseudomonas forma as bactérias mais frequentemente cultivadas. As bactérias alvo foram cultivadas de 77.8% dos pacientes edêntulos e 46.9% dos pacientes dentados 30 dias após a radioterapia. Por PCR, estes microrganismos foram detectados em todos os pacientes edêntulos e 78.1% dos pacientes dentados. Bactérias não orais e espécies de Cândida foram mais prevalentes nestes pacientes. Modificações no meio ambiente oral devido a radioterapia parecem facilitar a colonização por estes microrganismos.
The aim of this study was to evaluate the occurrence of opportunistic and exogenous microrganisms (enteric bacteria, pseudomonads, yeasts and Helicobacter pylori) in the oral cavity of patients undergoing radiotherapy (RT) for treatment of head and neck cancer. Fifty patients receiving RT were examined before, during and 30 days after RT. Saliva, mucosa, and biofilm samples were collected and microorganisms were detected by culture and Polymerase Chain Reaction (PCR). Candida albicans, C. tropicalis, C. krusei, C. glabrata and C. parapsilosis were the most prevalent yeasts in patients submitted to RT. Genera Citrobacter, Enterobacter, Enterococcus, Klebsiella, Proteus, and Pseudomonas were the most frequently cultivated bacteria. Targeted bacteria were cultivated from 77.8% edentulous and 46.9% dentate patients 30 days after RT. By PCR, these microorganisms were detected from all edentulous patients and from 78.1% of dentate patients. Non-oral bacteria and Candida species were prevalent in these patients. Modifications of the oral environment due to RT seem to facilitate the colonization of these microorganisms.
Mas, de Xaxars Rivero Teresa. "Descripció i quantificació de la microbiota intestinal associada al càncer colorectal". Doctoral thesis, Universitat de Girona, 2012. http://hdl.handle.net/10803/94513.
Pełny tekst źródłaEl càncer colorectal és el tipus de càncer més abundant a Espanya. Fins el 90% dels casos són d'origen espontani i la seva etiologia és desconeguda malgrat existeixen diversos factors que poden afectar en el desenvolupament tumoral, com la microbiota. L'objectiu d'aquesta tesi ha estat analitzar la composició de la comunitat microbiana en mostres de mucosa intestinal quantitativa i qualitativament. Els resultats mostren una disbiosi en els malalts de càncer colorectal i una associació amb l'augment o disminució d'espècies bacterianes, així com l'augment de determinats filotips/gèneres. També s'ha analitzat les poblacions d'estreptococs i l'exposició d'un cas clínic d'un pacient amb càncer colorectal amb una infecció causada per E. faecalis. Estudis focalitzats en aspectes més específics de la relació hoste-microbiota, així com explorar nous mecanismes induïts per bacteris són necessaris per comprendre alguns aspectes de la carcinogènesis colorectal.
Ciesielski, Francisco Isaak Nícolas. "Microrganismos oportunistas e exógenos na microbiota bucal de pacientes oncológicos submetidos à radioterapia de cabeça e pescoço /". Araçatuba : [s.n.], 2010. http://hdl.handle.net/11449/91426.
Pełny tekst źródłaBanca: Alvimar Lima de Castro
Banca: Luis Fernando Landucci
Resumo: O objetivo deste estudo foi avaliar a ocorrência de microrganismos exógenos e oportunistas (bactérias entéricas, pseudomonados, leveduras e Helicobacter pylori) na cavidade bucal de pacientes submetidos à radioterapia para tratamento de câncer de cabeça e pescoço. Cincoenta pacientes que iria receber radioterapia foram examinados antes, durante e 30 dias após radioterapia. Amostras de saliva, mucosa e biofilme foram coletadas e os microrganismos foram detectados por cultura e Reação em Cadeia da Polimerase (PCR). Candida albicans, C. tropicalis, C. krusei, C. glabrata e C. parapsilosis foram as leveduras mais prevalentes nos pacientes submetidos a radioterapia. Gêneros Citrobacter, Enterobacter, Enterococcus, Klebsiella, Proteus, e Pseudomonas forma as bactérias mais frequentemente cultivadas. As bactérias alvo foram cultivadas de 77.8% dos pacientes edêntulos e 46.9% dos pacientes dentados 30 dias após a radioterapia. Por PCR, estes microrganismos foram detectados em todos os pacientes edêntulos e 78.1% dos pacientes dentados. Bactérias não orais e espécies de Cândida foram mais prevalentes nestes pacientes. Modificações no meio ambiente oral devido a radioterapia parecem facilitar a colonização por estes microrganismos.
Abstract: The aim of this study was to evaluate the occurrence of opportunistic and exogenous microrganisms (enteric bacteria, pseudomonads, yeasts and Helicobacter pylori) in the oral cavity of patients undergoing radiotherapy (RT) for treatment of head and neck cancer. Fifty patients receiving RT were examined before, during and 30 days after RT. Saliva, mucosa, and biofilm samples were collected and microorganisms were detected by culture and Polymerase Chain Reaction (PCR). Candida albicans, C. tropicalis, C. krusei, C. glabrata and C. parapsilosis were the most prevalent yeasts in patients submitted to RT. Genera Citrobacter, Enterobacter, Enterococcus, Klebsiella, Proteus, and Pseudomonas were the most frequently cultivated bacteria. Targeted bacteria were cultivated from 77.8% edentulous and 46.9% dentate patients 30 days after RT. By PCR, these microorganisms were detected from all edentulous patients and from 78.1% of dentate patients. Non-oral bacteria and Candida species were prevalent in these patients. Modifications of the oral environment due to RT seem to facilitate the colonization of these microorganisms.
Mestre
Leveille, Simon. "Combination strategies in the development of oncolytic cancer therapy". Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=106483.
Pełny tekst źródłaCertains virus possèdent la capacité d'exploiter les défauts métaboliques des cellules cancéreuses pour leur propre réplication. Ces virus, nommés virus oncolytiques, représentent une remarquable opportunité pour le développement de thérapies contre le cancer. Malgré cette prédisposition, certaines caractéristiques des virus oncolytiques ne sont pas optimales pour cette fonction et pourraient être améliorées. Dans cette optique, des stratégies visant à augmenter l'oncolyse induite par le virus Vesicular Stomatitis (VSV) ont été développées et testées chez la souris au cours de ce doctorat. Dans un premier temps, VSV a été modifié génétiquement afin qu'il exprime l'enzyme suicide CD ::UPRT lui permettant de réaliser la conversion d'un composé non-toxique administré de façon systémique en composé cytotoxique uniquement au site de la tumeur. La stratégie a permis de démontrer une augmentation synergique de la lyse des cellules cancéreuses ainsi que l'induction de la mort de cellules cancéreuses non infectées et partiellement résistantes au VSV. De plus, la combinaison in vivo a été optimisée afin de tenir compte de la cinétique de réplication du virus à la tumeur ainsi que de la biodisponibilité de la drogue. Les résultats ont permis non seulement d'obtenir une amélioration de l'effet thérapeutique mais également de souligner d'importantes caractéristiques de la réplication virale in vivo. Dans une seconde stratégie, VSV a été combiné avec une approche d'immunomodulation ayant pour but d'engendrer une réponse immunitaire acquise spécifique à la tumeur. En employant le facteur de croissance Flt3L qui favorise la prolifération et la différentiation des cellules dendritiques, la capacité de présentation d'antigènes a été grandement renforcée simultanément à l'oncolyse induite par VSV. En dépit du fait que la combinaison n'a que partiellement amélioré l'effet thérapeutique, elle a révélé un aspect inattendu de la réponse immunitaire engendrée par VSV. Les résultats ont démontré que VSV affecte grandement la viabilité des cellules immunitaires et des cellules dendritiques à la tumeur, qu'il bloque leur migration aux organes lymphatiques et que, par conséquent, la présentation d'antigènes tumoraux est abolie. La démonstration de l'absence de présentation d'antigènes tumoraux suivant le traitement oncolytique de VSV représente un important concept expliquant la piètre capacité de VSV en ce qui a trait à l'induction d'une réponse immunitaire aquise spécifique à la tumeur. En conclusion, les stratégies développées aux cours de ces travaux ont permis d'améliorer les propriétés oncolytiques de VSV ainsi que de grandement contribuer à la compréhension de la thérapie anti-cancer de VSV.
Lampraki, Eirini-Maria. "Role of the histone demethylase KDM6A in pancreatic cancer". Thesis, University of Glasgow, 2018. http://theses.gla.ac.uk/38933/.
Pełny tekst źródłaPlummer, Kathleen Hope. "Cancer and Infection". Scholar Commons, 2014. https://scholarcommons.usf.edu/etd/5293.
Pełny tekst źródłaHand, Nicholas. "Development of a Recombinant Attenuated Salmonella Cancer Vaccine". Thesis, The George Washington University, 2017. http://pqdtopen.proquest.com/#viewpdf?dispub=10635177.
Pełny tekst źródłaNew treatments for neuroblastoma are desperately needed; high-risk neuroblastoma patients have a less than 50% five-year survival rate despite intensive treatment. The greatest impact on improving survival rates for cancer patients in recent years is the result of a number of immunotherapeutic approaches. A proportion of high-risk neuroblastoma patients undergo spontaneous regression, possibly mediated by the immune system, indicating the potential of immunotherapies targeting neuroblastoma-associated antigens. Recombinant attenuated Salmonella vaccines (RASV) are cost-effective and have shown efficacy against a number of pathogen-associated antigens and are easily adapted for use as cancer immunotherapies. Here we cloned survivin, a neuroblastoma tumor-associated antigen into RASV expression plasmids to develop 24 RASV candidate vaccines with an array of select phenotypes. While conventional recombinant attenuated Salmonella vaccines are permanently attenuated, the RASV used here are engineered with inducible in vivo attenuation and other delayed phenotypes shown to improve immune responses. Survivin expression did not impact the growth or stability of any of the RASV constructs. Six of the constructs were tested in vivo, the RASV survived in the gut lumen, and all RASV-immunized mice produced anti-Salmonella responses. Protein/adjuvant immunized mice produced humoral and cellular survivin specific immune responses; however two independent in vivo experiments showed that no survivin specific immune responses were induced in survivin-expressing RASV immunized mice. Based on the results, a number of improvements to the future development of the vaccine are suggested.
Riggio, Alessandra I. "The role of Runx1 in genetic models of breast cancer". Thesis, University of Glasgow, 2017. http://theses.gla.ac.uk/9103/.
Pełny tekst źródłaMacDonald, Patricia. "Defining functional domains within GPNMB important for breast cancer cell invasion". Thesis, McGill University, 2011. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=96829.
Pełny tekst źródłaGlycoprotein non-metastatic melanoma protein B (GPNMB), aussi connu sous le nom de Ostéoactivine (OA), est une protéine transmembranaire fréquemment exprimée dans les tumeurs mammaires appartement au sous-type triple négatif. Notre groupe a mis en évidence que l'expression de GPNMB/OA est suffisante pour accroître, in vitro, les capacités migratoires et invasives associées aux cellules murines de cancer du sein faiblement métastatiques. In vivo, l'expression de GPNMB/OA est caractérisée par une augmentation de la formation des métastases osseuses et pulmonaires. Dans cette étude, nous avons cherché à caractériser le rôle pro-invasif associé à l'expression de GPNMB/OA dans les cellules humaines de cancer du sein en identifiant les domaines ou motifs de GPNMB/OA impliqués dans le phénotype invasif des cellules de cancer du sein. D'une part, nous avons entrepris l'identification de protéines interagissant avec GPNMB/OA et qui pourraient participer aux phénotypes associés à l'expression de GPNMB/OA. Pour ce faire, nous avons généré une série de mutants pour la protéine GPNMB et nous les avons exprimées dans les lignées cellulaires BT-549 et MDA-MB-453, qui n'exprime normalement pas GPNMB/OA, que nous avons par la suite soumis à des essais d'invasion in vitro. D'autre part, une étude de la littérature scientifique, associée à une analyse par spectrométrie de masse, ont été utilisées pour identifier les protéines partenaires potentielles de GPNMB/OA. Finalement, nous avons généré une lignée de souris transgénique qui exprime la forme humaine de GPNMB/OA sous le contrôle du promoteur Mouse Mammary Tumor Virus (MMTV). Ce modèle murin a été utilisé pour étudier le rôle de GPNMB/OA sur le développement de la glande mammaire et sur la tumorigenèse in vivo. Ainsi, ces travaux ont démontré que la forme humaine de GPNMB/OA est suffisante pour induire une augmentation des propriétés invasives des cellules BT549 et que ce phénotype requiert à la fois la région cytoplasmique et le motif RGD de la protéine GPNMB/OA. La caractérisation, des souris transgéniques MMTV-GPNMB/OA a révélé, pour sa part, que GPNMB/OA n'interfère pas avec le développement normal des glandes mammaires chez les femelles vierges et aucune tumeur n'a été détectée à ce jour. L'ensemble de nos données démontre que GPNMB/OA est capable d'induire les propriétés invasives associées aux cellules de cancer du sein, et que ce phénotype requiert l'interaction du motif RGD de la protéine GPNMB avec les intégrines et/ou des résidus ou motifs présents dans la partie cytoplasmique de GPNMB/OA et qui pourraient induire le recrutement de molécule de signalisation dans cette région de GPNMB/OA.
Port, Jennifer Lynne Forbes. "Investigating the therapeutic potential of NUAK1 for the treatment of colorectal cancer". Thesis, University of Glasgow, 2018. http://theses.gla.ac.uk/9125/.
Pełny tekst źródłaRodgers, Lisa. "The phosphagen system in prostate cancer". Thesis, University of Glasgow, 2017. http://theses.gla.ac.uk/8520/.
Pełny tekst źródłaGiampazolias, Evangelos. "Investigating non-apoptotic cell death in cancer". Thesis, University of Glasgow, 2017. http://theses.gla.ac.uk/8056/.
Pełny tekst źródłaDewar, Emma Louise. "Alpha-1-acid glycoprotein as a potential biomarker of breast cancer in 'at risk' individuals". Thesis, Edinburgh Napier University, 2015. http://researchrepository.napier.ac.uk/Output/9835.
Pełny tekst źródłaGupta, Elera Gaytri Devi. "Effect of Antioxidants and Oxidative Stress on Different Cancer Cell Types". BYU ScholarsArchive, 2012. https://scholarsarchive.byu.edu/etd/3227.
Pełny tekst źródłaBraidwood, Lynne. "Oncolytic HSV1716 in combination with targeted anti cancer agents : identification of synergistic interactions and their mechanisms of synergy". Thesis, University of Glasgow, 2016. http://theses.gla.ac.uk/7999/.
Pełny tekst źródłaMurray, Abner A. "Plant Virus Nanoparticle In Situ Cancer Immunotherapies". Case Western Reserve University School of Graduate Studies / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=case1532370850718292.
Pełny tekst źródłaLungu, Gina Florentina. "Role of hypoxia and hypoxia induced factors in the development of breast cancer brain metastasis". [College Station, Tex. : Texas A&M University, 2008. http://hdl.handle.net/1969.1/ETD-TAMU-3082.
Pełny tekst źródłaDerkits, Sahra. "Investigating the role of Pten and Lkb1 in traditional and serrated mouse models of colorectal cancer". Thesis, University of Glasgow, 2014. http://theses.gla.ac.uk/4909/.
Pełny tekst źródłaSowerby, Zoe. "Bacterial n-nitrosation and nitrite reduction in the model organism Neisseria subflava". Thesis, Nottingham Trent University, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.241830.
Pełny tekst źródłaSteer, Toni. "Bacteriology of dietary anti-carcinogens in relation to colon cancer and the potential use of dietary intervention : effects of prebiotics and probiotics". Thesis, University of Reading, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.272262.
Pełny tekst źródłaFOY, KEVIN CHU. "COMBINATION IMMUNOTHERAPY WITH HER-2/NEU AND VEGF PEPTIDE MIMICS IN BOTH TRANSGENIC AND TRANSPLANTABLE MOUSE MODELS OF HUMAN BREAST CANCER". The Ohio State University, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=osu1299532419.
Pełny tekst źródłaGuffey, Catherine R. "Linking obesity to colorectal cancer| Recent insights into plausible biological mechanisms". Thesis, University of South Carolina, 2013. http://pqdtopen.proquest.com/#viewpdf?dispub=1537648.
Pełny tekst źródłaObesity has emerged as a leading environmental risk factor for the development of CRC. However, the mechanisms underlying this relationship have not yet been fully explained. Recent literature has focused on 1) inflammatory processes, 2) adipokines, and 3) estrogen. Obesity-enhanced inflammation is largely orchestrated by increases in adipose tissue macrophages leading to the secretion of TNF-alpha, MCP-1, and IL-6, all of which are linked to CRC. Adiponectin is decreased with obesity and has been reported to be negatively associated with CRC, while leptin, which is increased, is positively associated with the disease. Estrogen has been shown to influence CRC, although its role remains controversial; some studies have implicated estrogen as being protective, while others have suggested it to be a risk factor. We highlight the most important recent advances that have been made on the aforementioned mechanisms that are thought to link obesity to CRC.
Miller, Megan Jo. "Novel HER3 and IGF-1R Peptide Mimics and Synthetic Cancer Vaccines". The Ohio State University, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=osu1408981670.
Pełny tekst źródłaPhillips, Elisabeth. "Tamoxifen resistance in breast cancer : a proteomic approach". Thesis, University of Birmingham, 2012. http://etheses.bham.ac.uk//id/eprint/3270/.
Pełny tekst źródłaWeagel, Evita Giraldez. "Biomarker Analysis and Clinical Relevance of Thymidine Kinase 1 in Solid and Hematological Malignancies". BYU ScholarsArchive, 2018. https://scholarsarchive.byu.edu/etd/6881.
Pełny tekst źródłaWebster, Rebecca. "Complementary investigations of the molecular biology of cancer : assessment of the role of Grb7 in the proliferation and migration of breast cancer cells; and prediction and validation of microRNA targets involved in cancer". University of Western Australia. School of Medicine and Pharmacology, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0179.
Pełny tekst źródłaMonteverde, Tiziana. "Investigating the function and regulation of NUAK1 and its role in non-small cell lung cancer". Thesis, University of Glasgow, 2017. http://theses.gla.ac.uk/8701/.
Pełny tekst źródłaSmith, Richard LeRoy. "Cis-regulatory Sequence and Co-regulatory Transcription Factor Functions in ERα-Mediated Transcriptional Repression". BYU ScholarsArchive, 2009. https://scholarsarchive.byu.edu/etd/2261.
Pełny tekst źródłaFoster, Jessica. "Investigating the relationship between UbcH10 and the anaphase promoting complex/cyclosome". Thesis, University of Birmingham, 2018. http://etheses.bham.ac.uk//id/eprint/8024/.
Pełny tekst źródłaTsim, Selina. "Diagnostic and prognostic biomarkers of malignant pleural mesothelioma". Thesis, University of Glasgow, 2018. http://theses.gla.ac.uk/30687/.
Pełny tekst źródłaMuthalagu, Nathiya. "Understanding the mechanism of MYC induced vulnerabilities". Thesis, University of Glasgow, 2015. http://theses.gla.ac.uk/7464/.
Pełny tekst źródłaWoodham, Emma. "Investigating the role of the Rho GTPase Cdc42 in the migration and invasion of the melanocyte lineage". Thesis, University of Glasgow, 2017. http://theses.gla.ac.uk/8195/.
Pełny tekst źródłaLi, Xiaona. "Mass spectrometry-based metabolomics study on KRAS-mutant colorectal cancer and rheumatoid arthritis". HKBU Institutional Repository, 2018. https://repository.hkbu.edu.hk/etd_oa/540.
Pełny tekst źródłaNatarajan, Gayathri. "THE USE OF A TEC KINASE INHIBITOR, IBRUTINIB, FOR THE DEVELOPMENT OF IMMUNOTHERAPIES AGAINST CANCER AND LEISHMANIASIS". The Ohio State University, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=osu1461200133.
Pełny tekst źródłaMiranda, Alexandra de Sousa Montenegro. "Characterisation of LVI-1 (WDR76) as a candidate tumour suppressor gene". Thesis, University of Glasgow, 2006. http://theses.gla.ac.uk/4967/.
Pełny tekst źródłaAlegre, Melissa Marie. "Thymidine Kinase 1: Diagnostic and Prognostic Significance in Malignancy". BYU ScholarsArchive, 2013. https://scholarsarchive.byu.edu/etd/4049.
Pełny tekst źródłaKershaw, Rachael Maria. "Delayed response of the CYS326 variant of the DNA repair protein OGG1 to cellular oxidative stress". Thesis, University of Birmingham, 2011. http://etheses.bham.ac.uk//id/eprint/3035/.
Pełny tekst źródłaMachado, Lee Richard. "Molecular mechanisms of T cell homing in Hodgkin's lymphoma : implications for T-cell-based therapies". Thesis, University of Birmingham, 2004. http://etheses.bham.ac.uk//id/eprint/579/.
Pełny tekst źródłaZhao, Hongyan. "Development and application of high-field asymmetric waveform ion mobility spectrometry and mass spectrometry for the investigation of fibroblast growth factor signalling". Thesis, University of Birmingham, 2016. http://etheses.bham.ac.uk//id/eprint/7022/.
Pełny tekst źródłaIrvine, David Arthur. "Defining novel therapeutic targets in chronic myeloid leukaemia stem cells : targeting self-renewal through hedgehog pathway inhibition". Thesis, University of Glasgow, 2013. http://theses.gla.ac.uk/4584/.
Pełny tekst źródłaAQBI, HUSSEIN F. "Preconditioning of the tumor microenvironment by means of low dose chemotherapies for an effective immunotherapy of breast cancer". VCU Scholars Compass, 2019. https://scholarscompass.vcu.edu/etd/6025.
Pełny tekst źródłaGarrett, Andrew Robert. "Antioxidants in Cancer Research and Prevention: Assay Comparison, Structure-Function Analysis, and Food Product Analysis". BYU ScholarsArchive, 2011. https://scholarsarchive.byu.edu/etd/2735.
Pełny tekst źródłaLu, Daniel Kee. "The Rad51 family of proteins: Interactions, vitamin D, and implications in head and neck cancer". Scholarly Commons, 2013. https://scholarlycommons.pacific.edu/uop_etds/191.
Pełny tekst źródłaMacpherson, Iain Roderick James. "A study of p120-catenin and its tyrosine phosphorylation in cancer cell adhesion and invasion". Thesis, Connect to e-thesis, 2007. http://theses.gla.ac.uk/1008/.
Pełny tekst źródłaKodigepalli, Madhav Karthik. "Role and Regulation of SnoN/SkiL and PLSCR1 Located at 3q26.2 and 3q23, Respectively, in Ovarian Cancer Pathophysiology". Scholar Commons, 2014. https://scholarcommons.usf.edu/etd/5426.
Pełny tekst źródłaKennedy, Helen F. "Viridans streptococcal bacteraemia in paediatric immunocompromised patients with malignant disease". Thesis, University of Glasgow, 2001. http://theses.gla.ac.uk/2214/.
Pełny tekst źródłaNichols, Brandt Alan. "The Role of Nuclear BMP2 in the Cell Cycle and Tumorigenesis". BYU ScholarsArchive, 2013. https://scholarsarchive.byu.edu/etd/4167.
Pełny tekst źródłaFullwood, Rebecca A. "The Role of Viral Interleukin-6 in Tumor Development of Kaposi's Sarcoma-Associated Herpesvirus Lymphomas". BYU ScholarsArchive, 2016. https://scholarsarchive.byu.edu/etd/6194.
Pełny tekst źródła