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Artykuły w czasopismach na temat "Cancer de poumon"
Chaleat, S., i L. Greillier. "Cancer du poumon". Revue des Maladies Respiratoires Actualités 11 (październik 2019): S29—S35. http://dx.doi.org/10.1016/s1877-1203(19)30161-2.
Pełny tekst źródłaBoucly, A., i B. Milleron. "Dépistage du cancer du poumon". Revue des Maladies Respiratoires Actualités 6, nr 2 (maj 2014): 115–17. http://dx.doi.org/10.1016/s1877-1203(14)70525-7.
Pełny tekst źródłaAntoni, D., i F. Mornex. "Cancer du poumon : « Les » radiothérapies". Revue des Maladies Respiratoires Actualités 8, nr 5 (wrzesień 2016): 332–40. http://dx.doi.org/10.1016/s1877-1203(16)30122-7.
Pełny tekst źródłaToffart, A. C., M. Giaj Levra, L. Ferrer, J. Pinsolle, N. Terzi i D. Moro-Sibilot. "Réanimation et cancer du poumon". Revue des Maladies Respiratoires Actualités 10, nr 3 (październik 2018): 262–68. http://dx.doi.org/10.1016/s1877-1203(18)30019-3.
Pełny tekst źródłaRochon, Madeleine. "Tabagisme et cancer du poumon". Gérontologie et société 26 / n° 105, nr 2 (2003): 69. http://dx.doi.org/10.3917/gs.105.0069.
Pełny tekst źródłaLüthi, François, Christian Monnerat, Roger Stupp, Hans-Beat Ris, Abderrrahim Zouhair i Thomas McKee. "Cancer du poumon : portrait 2004". Revue Médicale Suisse 62, nr 2477 (2004): 776–82. http://dx.doi.org/10.53738/revmed.2004.62.2477.0776.
Pełny tekst źródłaMilleron, B., S. Couraud, E. Grolleau i V. Gounant. "Le dépistage du cancer du poumon". Revue des Maladies Respiratoires Actualités 14, nr 2 (październik 2022): 2S37–2S41. http://dx.doi.org/10.1016/s1877-1203(22)00098-2.
Pełny tekst źródłaGrolleau, E., M. Darrason, J. de Bermont, B. Milleron i S. Couraud. "Le dépistage du cancer du poumon". Revue des Maladies Respiratoires Actualités 13, nr 2 (wrzesień 2021): 2S21–2S25. http://dx.doi.org/10.1016/s1877-1203(21)00092-6.
Pełny tekst źródłaMilleron, B., V. Gounant, A. Khalil i S. Couraud. "Le dépistage du cancer du poumon". Revue de Pneumologie Clinique 73, nr 1 (luty 2017): 27–33. http://dx.doi.org/10.1016/j.pneumo.2016.10.002.
Pełny tekst źródłaPerez, Noemí Busquets, Berta Bernad, Javier Narváez i José Valverde. "Érythème noueux et cancer du poumon". Revue du Rhumatisme 73, nr 5 (maj 2006): 525–26. http://dx.doi.org/10.1016/j.rhum.2005.06.011.
Pełny tekst źródłaRozprawy doktorskie na temat "Cancer de poumon"
Meary, Alexandre. "Schizophrenie et cancer du poumon". Paris 6, 2001. http://www.theses.fr/2001PA062055.
Pełny tekst źródłaBenizri, Emmanuel. "HIF-1alpha : le poumon du cancer". Nice, 2007. http://www.theses.fr/2007NICE4044.
Pełny tekst źródłaAny alive organism can survive without energy, which involves a satisfactory oxygen and nutriments supply. At the initial phase of tumor progression, cells proliferate without their own vascularization ; indeed, oxygen and nutrients are delivered by simple diffusion. Nevertheless, beyond a certain size, these mechanisms are no more effective and oxygen deficiencies appear. The transcription factor HIF (Hypoxia Inducible Factor) is a key regulator required to maintain oxygen homeostasis. HIF is composed of a constitutive HIF-â sub-unit and a HIF- 1á sub-unit, specific of the hypoxic cascade. Three hifá genes have been identified : hif1a, epas 1 and hif3a that code for HIF-1á, HIF-2á and HIF-3á, respectively. Because of its ubiquitous tissue distribution and its central impact on the regulation of the target genes, HIF- 1á is the mosty studied isoform. During my laboratory training, I have contributed to 3 main projects : • First, I have been focused on the regulatory mechanisms that control HIF-1á stability and more precisely, on the study of the HIF prolyl-hydroxylase (PHDs). Using a RNA interference (RNAi) approach, we have highlighted PHD2 as the key isoform that controls HIF-1á stability in normoxia. Related to clinical studies, we have evaluated the impact of HIF-1á immunostaining in a serie of breast cancer. We have shown that HIF-1á is an independent prognostic factor and a bad preditive factor for classical adjuvant treatments. • Finally, in a pre-clinical study, we have analyzed the impact of HIF-1á invalidation by RNAi in a xenograft melanoma model. We have reported a reduction of tumor growth in mice treated with siRNAs targeting HIF-1á
Michel, Noémie. "Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire". Thesis, Tours, 2013. http://www.theses.fr/2013TOUR3303/document.
Pełny tekst źródłaRecently, human tissue kallikreins (KLK) emerged as a new family of serine proteases which might play a major role in the tumorigenesis. The project aims at determining the contribution of KLK6 and 12 in lung pathophysiology. We showed that ectopic KLK6 promoted A549 cell proliferation in a protease activity-dependant manner, inhibited cell apoptosis and induced β-catenin nuclear translocation. Furthermore, this study uncovered a signaling pathway mediated by KLK6 in promoting A549 cell proliferation trough activation of the PAR2-EGFR-ERK pathway.We have also identified novel substrates of KLK12, the CCN family. We reported that KLK12-mediated proteolysis of CCN1 and CCN5 can reduce or abolish the binding of VEGF, BMP2, and TGF-β1
Pardo, Olivier. "Thérapie génique du cancer du poumon par la HSP65". Paris 5, 1997. http://www.theses.fr/1997PA05P120.
Pełny tekst źródłaDerniame, Sophie. "Cancer du poumon / Réponse immunitaire locale - modulation tumeur dépendante". Nancy 1, 2006. http://docnum.univ-lorraine.fr/public/SCD_T_2006_0097_DERNIAME.pdf.
Pełny tekst źródłaLung cancer is the most frequent type of cancer in the world. Smoking is clearly the major cause of this pathology. The proliferation of tumor cells induces an inflammatory stromal reaction comprising numerous tumor-infiltrating lymphocytes. In this study, four complementary approaches have been used to study the tumor-dependent modulation of the immune system : TCR Vβ repertoire usage in flow cytometry, TCRγ gene clonal rearrangements in denaturing gradient gel electrophoresis (DGGE/TTGE), tumor and healthy lung tissue infiltration as well as lymph nodes characteristics in immunohistology and cytokine production by RNA RT-PCR. The results obtained have demonstrated the oligoclonality of T-cells in the three types of tissues tested. A Vβ13. 1 clone and a gamma clone appeared to be specifics of epidermoid carcinoma. Similarly, two TCRγ clones appeared to be restricted to adenocarcinoma. Moreover, the CD3/TCR complex was clearly down regulated in tumors compared to healthy tissue or lymph nodes. Similarly, HLA-DR, HLA-DQ and β2 microglobuline, strongly expressed on healthy pneumocytes were nearly absent from tumor cells. Several cytokines with antagonistic effects were detected within tumoral tissue, especially TGFβ and IL-10, which favour tumor growth and TNFalpha and IFNγ which potentialize the anti-tumoral immune response. In conclusion, the clones identified in healthy lung tissue could be specific of early pre-tumoral lesions induced by tobacco smoke, and some of the clones appear to be tumor-specific. However, the immune system has been defeated by several mechanisms, including a decrease of the expression of partners of the immunological synapse and the production of antagonistic cytokines
CAILLET, BRUNO. "Genetique et cancer du poumon : interet des etudes familiales". Lyon 1, 1991. http://www.theses.fr/1991LYO1M273.
Pełny tekst źródłaSève, Pascal. "Pharmacorésistance et cancer du poumon Non à petites cellules". Lyon 1, 2006. http://www.theses.fr/2006LYO10008.
Pełny tekst źródłaCauchi, Stéphane. "Etude des polymorphismes génétiques du récepteur aux hydrocarbures polycycliques aromatiques (AHR) et de son répresseur (AHRR) : relation avec l'inductibilité du CYP1A1 et le cancer pulmonaire". Paris 5, 2003. http://www.theses.fr/2003PA05N030.
Pełny tekst źródłaPolycyclic aromatic hydrocarbons (PAH) found in cigarette smoke play a key role in the appearance of the lung cancer at the smokers. Cytochrome P-4501A1, metabolizes these HPA like benzo(a)pyrene in reactive and mutagen compounds. The CYP1A1 is inductible by the HPA and others compounds like dioxine. This inductibility is itself variable and largely dependent on the receptor Ah (AhR). The AhR is partially responsible for a polymorphic expression of the CYP1A1 and thus could be an important factor in carcinogenicity. We investigated the genetic polymorphisms of the AhR gene in a case-control study of lung cancer. Two new polymorphisms were found, none of them were found to play a key role. . .
RUGGIERI, SOPHIE. "Traitement des cancers du poumon non anaplasiques, non resecables, non metastatiques : a propos de 505 cas". Angers, 1989. http://www.theses.fr/1989ANGE1084.
Pełny tekst źródłaTLEMSANI, LARBI. "Prise en charge du cancer du poumon au centre hospitalier general de cahors de 1989 a 1992". Toulouse 3, 1993. http://www.theses.fr/1993TOU31084.
Pełny tekst źródłaKsiążki na temat "Cancer de poumon"
Trédaniel, Jean. Le cancer du poumon. Paris: Masson, 2004.
Znajdź pełny tekst źródłaLaboratoire de lutte contre la maladie (Canada). Bureau du cancer. Le cancer du poumon au Canada. Ottawa, Ont: Santé Canada, 1998.
Znajdź pełny tekst źródłaLitt, Claudia. J'ai le cancer, et alors? Lyon: Chronique sociale, 2012.
Znajdź pełny tekst źródłaGiovanni, Motta, i Agrigento International Conference on Imaging and Therapy of Lung Cancer. (1st : 1988), red. Lung cancer: Advanced concepts and present status. Genoa: Grafica, L.P., 1989.
Znajdź pełny tekst źródłaMackay, Bruce. Tumors of the lung. Philadelphia: W.B. Saunders, 1991.
Znajdź pełny tekst źródłaLemonde, Manon. Qualité de vie de personnes atteintes de cancer du poumon avancé. Montréal, Qué: Université de Montréal, 1999.
Znajdź pełny tekst źródłaStap, Sophie van der. La fille aux neuf perruques: Témoignage. [Paris]: Presses de la Cité, 2009.
Znajdź pełny tekst źródłaMayer, Carole R. Les directives avancées à l'usage des patients atteints du cancer du poumon. Sudbury, Ont: École de Service Social, Université Laurentienne, 1996.
Znajdź pełny tekst źródłaStatistique Canada. Direction des études analytiques. Utilisation de POHEM pour l'estimation des coûts médicaux directs associés à la pratique actuelle du traitement du cancer du poumon ainsi que pour l'évaluation économique de nouveaux traitements. Ottawa, Ont: Statistique Canada, 1997.
Znajdź pełny tekst źródłaNATO Advanced Research Workshop on Mechanisms in Fibre Carcinogenesis (1990 Albuquerque, N.M.). Mechanisms in fibre carcinogenesis. New York: Plenum Press, 1991.
Znajdź pełny tekst źródłaCzęści książek na temat "Cancer de poumon"
Molinié, F., A. Danzon, G. Hédelin i E. A. Sauleau. "Poumon". W Survie des patients atteints de cancer en France, 185–92. Paris: Springer Paris, 2007. http://dx.doi.org/10.1007/978-2-287-39310-5_25.
Pełny tekst źródłaLozano, S., i J. Mazières. "Le cancer du poumon du sujet âgé". W Oncogériatrie, 79–84. Paris: Springer Paris, 2011. http://dx.doi.org/10.1007/978-2-8178-0142-1_12.
Pełny tekst źródłaDauchy, S., i C. Lopez. "Prise en charge psychologique du patient atteint de cancer du poumon". W Oncologie thoracique, 367–78. Paris: Springer Paris, 2011. http://dx.doi.org/10.1007/978-2-287-99041-0_24.
Pełny tekst źródłaSocinski, Mark A. "Cancer du poumon". W Médecine interne de Netter, 609–16. Elsevier, 2011. http://dx.doi.org/10.1016/b978-2-294-70951-7.00078-5.
Pełny tekst źródła"Cancers du poumon". W Méga Guide STAGES IFSI, 89–97. Elsevier, 2015. http://dx.doi.org/10.1016/b978-2-294-74529-4.00022-7.
Pełny tekst źródłaAlexandre, J., A. Balian, L. Bensoussan, A. Chaïb, G. Gridel, K. Kinugawa, F. Lamazou i in. "Cancers du poumon". W Le tout en un révisions IFSI, 82–89. Elsevier, 2009. http://dx.doi.org/10.1016/b978-2-294-70633-2.50022-6.
Pełny tekst źródłaALLIO, I., A. M. DEMARTINI, J. J. MENEC, V. CARDE i V. TREGUER-HERROU. "Le dépistage par le Laboratoire d’analyses, de surveillance et d’expertise de la Marine de Brest du radon, un gaz radioactif naturel en Bretagne". W Médecine et Armées Vol. 44 No.4, 309–16. Editions des archives contemporaines, 2016. http://dx.doi.org/10.17184/eac.6820.
Pełny tekst źródłaTaber, Douglass. "Alkaloid Synthesis: Crispine A (Zhou), Cermizine C (Zhang), Tangutorine (Poupon), FR901483 (Kerr), Serratezomine A (Johnston)". W Organic Synthesis. Oxford University Press, 2011. http://dx.doi.org/10.1093/oso/9780199764549.003.0061.
Pełny tekst źródłaStreszczenia konferencji na temat "Cancer de poumon"
Dias, Andrey Luis de Oliveira Gonçalves, Thaíz da Silva Gome i Leila Cristina Soares Brollo. "Câncer de endométrio associado a radiação pélvica prévia paratratamento de câncer de colo uterino: relato de caso". W 45º Congresso da SGORJ XXIV Trocando Ideias. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/jbg-0368-1416-20211311018.
Pełny tekst źródłaMacedo, Aruan Kawally Coutinho De, Felipe De Andrade Moreira, Fernanda Clara Marinheiro Oliveira i Neidivania Medeiros Da Nobrega. "O DIAGNÓSTICO PRECOCE DA LEUCEMIA MIELOIDE AGUDA". W II Congresso Brasileiro de Hematologia Clínico-laboratorial On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/hematoclil/10.
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