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Gotowe bibliografie tematyczne / Calcium Metabolism / Artykuły w czasopismach

Artykuły w czasopismach na temat „Calcium Metabolism”

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Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 4 marca 2023

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1

Pak, Charles Y. C. "Calcium Metabolism". Journal of the American College of Nutrition 8, sup1 (grudzień 1989): 46S—53S. http://dx.doi.org/10.1080/07315724.1989.10737969.

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2

Licata, Angelo A. "Calcium metabolism". Trends in Endocrinology & Metabolism 2, nr 6 (listopad 1991): 240. http://dx.doi.org/10.1016/1043-2760(91)90031-h.

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3

Stiffler, D. F. "Amphibian calcium metabolism". Journal of Experimental Biology 184, nr 1 (1.11.1993): 47–61. http://dx.doi.org/10.1242/jeb.184.1.47.

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Calcium is present in amphibian blood at a concentration similar to that in other vertebrates, about 1–2 mmol l-1. The fraction of free calcium in amphibians is lower than that in other tetrapod vertebrates because about 50% of the plasma Ca2+ is bound to plasma proteins and perhaps other molecules. Plasma [Ca2+] varies seasonally, increasing in spring and summer and decreasing in winter. Changes in plasma [Ca2+] also occur during larval development, as the concentration of this ion increases in larval forms as they approach metamorphosis. Calcium is exchanged at a variety of sites in animals. There is evidence for Ca2+ uptake across the skin and gills of larval anurans. It is also transported into the blood from the small intestine (especially the duodenum) and reabsorbed in renal tubules from the glomerular filtrate. The possibility of Ca2+ absorption from urine stored in the urinary bladder has not been confirmed, however. Calcium is stored in bone and in specialized endolymphatic sacs. This Ca2+ can be mobilized when the need arises. There are a number of endocrine and other humoral factors that appear to be involved in amphibian calcium metabolism. These include parathyroid hormone, calcitonin, vitamin D and prolactin.

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4

WALPERT, NAOMI. "CALCIUM METABOLISM DISORDERS". Nursing 20, nr 7 (lipiec 1990): 60–64. http://dx.doi.org/10.1097/00152193-199007000-00022.

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5

Emkey, Ronald D., i Gregory R. Emkey. "Calcium Metabolism and Correcting Calcium Deficiencies". Endocrinology and Metabolism Clinics of North America 41, nr 3 (wrzesień 2012): 527–56. http://dx.doi.org/10.1016/j.ecl.2012.04.019.

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6

Walters, Barry NJ. "Calcium metabolism in pregnancy". Fetal and Maternal Medicine Review 1, nr 2 (lipiec 1989): 213–22. http://dx.doi.org/10.1017/s0965539500000188.

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During pregnancy and lactation there are many changes in maternal calcium physiology which maintain homeostasis in the face of greatly altered calcium balance. In the course of fetal growth and development, 30g of calcium is incorporated into the fetus by term, an amount derived wholly from the maternal system. Most of this accumulates in the latter half of pregnancy, representing a net transfer of 200mg calcium/day (5mmoles). The fact that this is not achieved at the expense of the maternal skeleton is testimony to the conservative and protective adjustments that are seen in calcium metabolism in pregnancy. Furthermore, the changes must persist both in the puerperium and later when lactation presents a source of continuing maternal calcium loss to the suckling infant. The calcium content of human breast milk s i 6–9mmols calcium/l, two to three times the maternal serum level. In the course of one week a normal breast-fed at term infant takes two to three litres of milk, containing 10–30mmols of calcium. The maternal daily calcium intake recommended by the World Health Organization s i 1.25g (30mmol) of which only 25% is absorbed. Thus calcium loss from mother to baby is significant and may not be replaced by diet in many parts of the world.

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7

Massey, Linda K., i Susan J. Whiting. "Caffeine, Urinary Calcium, Calcium Metabolism and Bone". Journal of Nutrition 123, nr 9 (1.09.1993): 1611–14. http://dx.doi.org/10.1093/jn/123.9.1611.

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8

TERASHITA, Kenzo, Tetsuro NAKAMURA, Joji OSHIMA, Hajime ORIMO i Masayoshi YAMAGUCHI. "Calcium Metabolism and Arteriosclerosis". Journal of Japan Atherosclerosis Society 14, nr 4 (1986): 951–56. http://dx.doi.org/10.5551/jat1973.14.4_951.

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9

TERASHITA, Kenzo, Tetsuro NAKAMURA, Joji OOSHIMA, Hajime ORIMO i Masayoshi YAMAGUCHI. "Calcium Metabolism and Arteriosclerosis". Journal of Japan Atherosclerosis Society 15, nr 1 (1987): 315–18. http://dx.doi.org/10.5551/jat1973.15.1_315.

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10

TERASHITA, Kenzo, Tetsuro NAKAMURA, Joji OOSHIMA, Hajime ORIMO i Yoshiyuki SEYAMA. "Calcium Metabolism and Arteriosclerosis". Journal of Japan Atherosclerosis Society 16, nr 6 (1988): 895–901. http://dx.doi.org/10.5551/jat1973.16.6_895.

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11

McCarron, David A. "Calcium Metabolism in Hypertension." Keio Journal of Medicine 44, nr 4 (1995): 105–14. http://dx.doi.org/10.2302/kjm.44.105.

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12

O’Toole, John F. "Disorders of Calcium Metabolism". Nephron Physiology 118, nr 1 (2011): p22—p27. http://dx.doi.org/10.1159/000320884.

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13

McCarron, David A. "Calcium metabolism and hypertension". Kidney International 35, nr 2 (luty 1989): 717–36. http://dx.doi.org/10.1038/ki.1989.44.

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14

Rosol, Thomas J., Dennis J. Chew, Larry A. Nagode i Charles C. Capen. "Pathophysiology of Calcium Metabolism". Veterinary Clinical Pathology 24, nr 2 (czerwiec 1995): 49–63. http://dx.doi.org/10.1111/j.1939-165x.1995.tb00937.x.

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15

Stewart, Andrew F. "Calcium metabolism without anguish". Postgraduate Medicine 77, nr 1 (styczeń 1985): 283–94. http://dx.doi.org/10.1080/00325481.1985.11698858.

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16

Sutton, Roger A. L. "Diuretics and Calcium Metabolism". American Journal of Kidney Diseases 5, nr 1 (styczeń 1985): 4–9. http://dx.doi.org/10.1016/s0272-6386(85)80128-1.

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17

Redrobe, Sharon. "Calcium metabolism in rabbits". Seminars in Avian and Exotic Pet Medicine 11, nr 2 (kwiecień 2002): 94–101. http://dx.doi.org/10.1053/saep.2002.125100.

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18

Bouillon, Roger, Geert Carmeliet i Steven Boonen. "Ageing and calcium metabolism". Baillière's Clinical Endocrinology and Metabolism 11, nr 2 (lipiec 1997): 341–65. http://dx.doi.org/10.1016/s0950-351x(97)80332-1.

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19

Bronner, Felix. "Calcium nutrition and metabolism". Dental Clinics of North America 47, nr 2 (kwiecień 2003): 209–24. http://dx.doi.org/10.1016/s0011-8532(02)00097-6.

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20

TOMITA, Akio, i Kensuke TAKATSUKI. "Aging and calcium metabolism." Nihon Naika Gakkai Zasshi 74, nr 10 (1985): 1366–72. http://dx.doi.org/10.2169/naika.74.1366.

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21

Bowden, A. N. "ANTICONVULSANTS AND CALCIUM METABOLISM". Developmental Medicine & Child Neurology 16, nr 2 (12.11.2008): 214–16. http://dx.doi.org/10.1111/j.1469-8749.1974.tb02745.x.

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22

Bergenfelz, Anders, i Bo Ahrén. "Calcium Metabolism after Hemithyroidectomy". Hormone Research 39, nr 1-2 (1993): 56–60. http://dx.doi.org/10.1159/000182696.

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23

de Matos, Ricardo. "Calcium Metabolism in Birds". Veterinary Clinics of North America: Exotic Animal Practice 11, nr 1 (styczeń 2008): 59–82. http://dx.doi.org/10.1016/j.cvex.2007.09.005.

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24

Allgrove, Jeremy. "Disorders of calcium metabolism". Current Paediatrics 13, nr 7 (grudzień 2003): 529–35. http://dx.doi.org/10.1016/j.cupe.2003.08.007.

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25

Walkon, Lauren L., Jasiel O. Strubbe-Rivera i Jason N. Bazil. "Calcium Overload and Mitochondrial Metabolism". Biomolecules 12, nr 12 (17.12.2022): 1891. http://dx.doi.org/10.3390/biom12121891.

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Mitochondria calcium is a double-edged sword. While low levels of calcium are essential to maintain optimal rates of ATP production, extreme levels of calcium overcoming the mitochondrial calcium retention capacity leads to loss of mitochondrial function. In moderate amounts, however, ATP synthesis rates are inhibited in a calcium-titratable manner. While the consequences of extreme calcium overload are well-known, the effects on mitochondrial function in the moderately loaded range remain enigmatic. These observations are associated with changes in the mitochondria ultrastructure and cristae network. The present mini review/perspective follows up on previous studies using well-established cryo–electron microscopy and poses an explanation for the observable depressed ATP synthesis rates in mitochondria during calcium-overloaded states. The results presented herein suggest that the inhibition of oxidative phosphorylation is not caused by a direct decoupling of energy metabolism via the opening of a calcium-sensitive, proteinaceous pore but rather a separate but related calcium-dependent phenomenon. Such inhibition during calcium-overloaded states points towards mitochondrial ultrastructural modifications, enzyme activity changes, or an interplay between both events.

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26

Recker, R. R., i R. P. Heaney. "The effect of milk supplements on calcium metabolism, bone metabolism and calcium balance". American Journal of Clinical Nutrition 41, nr 2 (1.02.1985): 254–63. http://dx.doi.org/10.1093/ajcn/41.2.254.

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27

Thomas, Annie, Robert McVie i Steven Levine. "DISORDERS OF MATERNAL CALCIUM METABOLISM IMPLICATED BY ABNORMAL CALCIUM METABOLISM IN THE NEONATE". American Journal of Perinatology 16, nr 10 (1999): 0515–20. http://dx.doi.org/10.1055/s-1999-7280.

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28

YILDIZ, Saliha, i Mustafa Öztürk. "Relationship Between Calcium Metabolism Parameters". Eastern Journal Of Medicine 24, nr 4 (2019): 490–96. http://dx.doi.org/10.5505/ejm.2019.46873.

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29

LICATA, A. A. "Clinical Endocrinology of Calcium Metabolism". Cleveland Clinic Journal of Medicine 55, nr 6 (1.11.1988): 570. http://dx.doi.org/10.3949/ccjm.55.6.570.

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30

KAWAGISHI, Takahiko, Kiichirou SEKIYA, Yasuhisa OKUNO, Takami MIKI, Yoshiki NISHIZAWA i Hirotoshi MORII. "Calcium Metabolism in Diabetes Mellitus". Journal of Nutritional Science and Vitaminology 37, Supplement (1991): S51—S56. http://dx.doi.org/10.3177/jnsv.37.supplement_s51.

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31

IKEDA, KATSUHISA. "Calcium metabolism and hearing disorder." AUDIOLOGY JAPAN 33, nr 5 (1990): 615–16. http://dx.doi.org/10.4295/audiology.33.615.

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32

Ikeda, Katsuhisa. "Calcium metabolism and hearing disturbance." AUDIOLOGY JAPAN 34, nr 2 (1991): 75–83. http://dx.doi.org/10.4295/audiology.34.75.

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33

Young, E. W., R. D. Bukoski i D. A. McCarron. "Calcium Metabolism in Experimental Hypertension". Experimental Biology and Medicine 187, nr 2 (1.02.1988): 123–41. http://dx.doi.org/10.3181/00379727-187-42646.

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34

ALLEN, W. M., i B. F. SANSOM. "Milk fever and calcium metabolism". Journal of Veterinary Pharmacology and Therapeutics 8, nr 1 (marzec 1985): 19–29. http://dx.doi.org/10.1111/j.1365-2885.1985.tb00920.x.

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35

Stier, C. T., i H. D. Itskovitz. "Renal Calcium Metabolism and Diuretics". Annual Review of Pharmacology and Toxicology 26, nr 1 (kwiecień 1986): 101–16. http://dx.doi.org/10.1146/annurev.pa.26.040186.000533.

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36

TOMITA, Akio, i Kensuke TAKATSUKI. "6. Aging and Calcium Metabolism". Japanese Journal of Medicine 25, nr 1 (1986): 95b—96. http://dx.doi.org/10.2169/internalmedicine1962.25.95b.

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37

Kazantzis, George. "Cadmium, osteoporosis and calcium metabolism". BioMetals 17, nr 5 (październik 2004): 493–98. http://dx.doi.org/10.1023/b:biom.0000045727.76054.f3.

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38

Fuss, M., A. Bergans, C. Gillet, R. Karmali, T. Pepcrsack, J. A. Bagon, G. Mandart i J. Corvilain. "Calcium Metabolism Disturbances in Sarcoidosis". Acta Clinica Belgica 42, nr 6 (styczeń 1987): 421–30. http://dx.doi.org/10.1080/22953337.1987.11719260.

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39

Horner, Janet H. "Bone structure and calcium metabolism". Surgery (Oxford) 22, nr 1 (styczeń 2004): 24a—24d. http://dx.doi.org/10.1383/surg.22.1.24.27039.

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40

Martyn, C. N., S. Singh i P. J. Wood. "Calcium Metabolism in Alzheimer’s Disease". Gerontology 35, nr 2-3 (1989): 153–57. http://dx.doi.org/10.1159/000213014.

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41

Barzel, Uriel S. "Anion Effects on Calcium Metabolism". Journal of Bone and Mineral Research 12, nr 2 (1.02.1997): 298. http://dx.doi.org/10.1359/jbmr.1997.12.2.298.1.

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42

Straffen, Anne M., DJS Carmichael, Angela Fairney, B. Hulme i M. Snell. "Calcium Metabolism following Renal Transplantation". Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 31, nr 2 (marzec 1994): 125–28. http://dx.doi.org/10.1177/000456329403100202.

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Abnormalities of calcium homeostasis are a recognized feature of end-stage renal disease. The treatment of choice is renal transplantation, but this does not always result in normalization of the biochemical profile. Persistent hypercalcaemia is well documented and our study was undertaken to investigate the status of the calcium regulating hormones in renal patients post-transplantation. Serum calcium, parathyroid hormone, 1,25-dihydroxyvitamin D (1,25(OH)2D) and osteocalcin concentrations were measured in post-transplant patients. Twenty per cent of the patients had subnormal 1,25(OH)2D concentrations while 55% had biochemical evidence of hyperparathyroidism but only 5% were hypercalcaemic. Time elapsed since transplantation was not correlated with any of the analytes investigated and there was no relationship between persistent impairment of renal function and abnormalities of calcium homeostasis.

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43

Al-Jubouri, M. A. "Calcium Metabolism following Renal Transplantation". Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 31, nr 6 (listopad 1994): 587. http://dx.doi.org/10.1177/000456329403100615.

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44

Katz, I. "Calcium Metabolism following Renal Transplantation". Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 32, nr 2 (marzec 1995): 234. http://dx.doi.org/10.1177/000456329503200225.

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45

Thakker, RV. "Genetic disorders of calcium metabolism". Bone 27, nr 4 (październik 2000): 2. http://dx.doi.org/10.1016/s8756-3282(00)89001-8.

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46

Ray, J., K. Vasishta, S. Kaur, S. Majumdar i H. Sawhney. "Calcium metabolism in pre-eclampsia". International Journal of Gynecology & Obstetrics 66, nr 3 (wrzesień 1999): 245–50. http://dx.doi.org/10.1016/s0020-7292(99)00096-x.

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47

KRALL, A. R., HASSAN QAZZAZ, E. J. DUKE i ANNE R. BEHAN. "Mitochondrial calcium metabolism in Drosophila". Biochemical Society Transactions 17, nr 1 (1.02.1989): 235. http://dx.doi.org/10.1042/bst0170235.

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48

Weaver, Connie M. "Assessing Calcium Status and Metabolism". Journal of Nutrition 120, suppl_11 (1.11.1990): 1470–73. http://dx.doi.org/10.1093/jn/120.suppl_11.1470.

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49

Mihai, R., i J. R. Farndon. "Parathyroid disease and calcium metabolism". British Journal of Anaesthesia 85, nr 1 (lipiec 2000): 29–43. http://dx.doi.org/10.1093/bja/85.1.29.

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50

Farnell, B. J., D. R. Crapper McLachlan, K. Baimbridge, U. De Boni, L. Wong i P. L. Wood. "Calcium metabolism in aluminum encephalopathy". Experimental Neurology 88, nr 1 (kwiecień 1985): 68–83. http://dx.doi.org/10.1016/0014-4886(85)90114-1.

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