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Artykuły w czasopismach na temat "C26 cells"
Kutkowska-Kaźmierczak, Anna, Małgorzata Rydzanicz, Aleksander Chlebowski, Kamila Kłosowska-Kosicka, Adriana Mika, Jakub Gruchota, Elżbieta Jurkiewicz i in. "Dominant ELOVL1 mutation causes neurological disorder with ichthyotic keratoderma, spasticity, hypomyelination and dysmorphic features". Journal of Medical Genetics 55, nr 6 (1.03.2018): 408–14. http://dx.doi.org/10.1136/jmedgenet-2017-105172.
Pełny tekst źródłaSchutgens, R. B., I. W. Bouman, A. A. Nijenhuis, R. J. Wanders i M. E. Frumau. "Profiles of very-long-chain fatty acids in plasma, fibroblasts, and blood cells in Zellweger syndrome, X-linked adrenoleukodystrophy, and rhizomelic chondrodysplasia punctata". Clinical Chemistry 39, nr 8 (1.08.1993): 1632–37. http://dx.doi.org/10.1093/clinchem/39.8.1632.
Pełny tekst źródłaYamaguchi, A., T. Katagiri, T. Ikeda, J. M. Wozney, V. Rosen, E. A. Wang, A. J. Kahn, T. Suda i S. Yoshiki. "Recombinant human bone morphogenetic protein-2 stimulates osteoblastic maturation and inhibits myogenic differentiation in vitro." Journal of Cell Biology 113, nr 3 (1.05.1991): 681–87. http://dx.doi.org/10.1083/jcb.113.3.681.
Pełny tekst źródłaZarrouk, Amira, Anne Vejux, Thomas Nury, Hammam I. El Hajj, Madouda Haddad, Mustapha Cherkaoui-Malki, Jean-Marc Riedinger, Mohamed Hammami i Gérard Lizard. "Induction of Mitochondrial Changes Associated with Oxidative Stress on Very Long Chain Fatty Acids (C22:0, C24:0, or C26:0)-Treated Human Neuronal Cells (SK-NB-E)". Oxidative Medicine and Cellular Longevity 2012 (2012): 1–15. http://dx.doi.org/10.1155/2012/623257.
Pełny tekst źródłaSantalova, Elena A., Alexandra S. Kuzmich, Ekaterina A. Chingizova, Ekaterina S. Menchinskaya, Evgeny A. Pislyagin i Pavel S. Dmitrenok. "Phytoceramides from the Marine Sponge Monanchora clathrata: Structural Analysis and Cytoprotective Effects". Biomolecules 13, nr 4 (14.04.2023): 677. http://dx.doi.org/10.3390/biom13040677.
Pełny tekst źródłaYamada, T., N. Kubushiro, K. Shigemasa, T. Ikeda i M. Takagi. "Effects of Transforming Growth Factor-β1 on Decorin Expression by Undifferentiated Osteoblastic Cells". Microscopy and Microanalysis 3, S2 (sierpień 1997): 187–88. http://dx.doi.org/10.1017/s1431927600007820.
Pełny tekst źródłaEisenkolb, Marlis, Christoph Zenzmaier, Erich Leitner i Roger Schneiter. "A Specific Structural Requirement for Ergosterol in Long-chain Fatty Acid Synthesis Mutants Important for Maintaining Raft Domains in Yeast". Molecular Biology of the Cell 13, nr 12 (grudzień 2002): 4414–28. http://dx.doi.org/10.1091/mbc.e02-02-0116.
Pełny tekst źródłaSCHNEITER, Roger, Britta BRÜGGER, Clare M. AMANN, Glenn D. PRESTWICH, Raquel F. EPAND, Günther ZELLNIG, Felix T. WIELAND i Richard M. EPAND. "Identification and biophysical characterization of a very-long-chain-fatty-acid-substituted phosphatidylinositol in yeast subcellular membranes". Biochemical Journal 381, nr 3 (27.07.2004): 941–49. http://dx.doi.org/10.1042/bj20040320.
Pełny tekst źródłaRodolfo, M., C. Zilocchi, C. Melani, B. Cappetti, I. Arioli, G. Parmiani i M. P. Colombo. "Immunotherapy of experimental metastases by vaccination with interleukin gene-transduced adenocarcinoma cells sharing tumor-associated antigens. Comparison between IL-12 and IL-2 gene-transduced tumor cell vaccines." Journal of Immunology 157, nr 12 (15.12.1996): 5536–42. http://dx.doi.org/10.4049/jimmunol.157.12.5536.
Pełny tekst źródłaSmakman, Niels, Diana J. M. van den Wollenberg, Inne H. M. Borel Rinkes, Rob C. Hoeben i Onno Kranenburg. "Sensitization to Apoptosis Underlies KrasD12-Dependent Oncolysis of Murine C26 Colorectal Carcinoma Cells by Reovirus T3D". Journal of Virology 79, nr 23 (15.12.2005): 14981–85. http://dx.doi.org/10.1128/jvi.79.23.14981-14985.2005.
Pełny tekst źródłaRozprawy doktorskie na temat "C26 cells"
Chaouki, Ghita. "Etude du rôle de la voie de signalisation eIF2αATF4 au cours des états inflammatoires, dans le cadre du stress mitochondrial et de l’anorexie associée à la pathologie". Electronic Thesis or Diss., Université Clermont Auvergne (2021-...), 2022. http://www.theses.fr/2022UCFAC109.
Pełny tekst źródłaThe eIF2α-ATF4 signaling pathway is activated in cells in response to a wide range of cellular stresses. Its activation leads to the inhibition of the global protein synthesis and the regulation of the transcription factor ATF4 target genes expression. This pathway is activated in response to essential amino acid deficiency, mitochondrial stress, endoplasmic reticulum stress or viral infections. Its activation triggers adaptive mechanisms, both at the cellular level (such as inhibition of protein synthesis and increased autophagy) and at the whole organism level (such as regulation of metabolism, inflammation, immunity and food intake). Previous results generated by our laboratory as well as data from the scientific literature led us to investigate the role of eIF2α-ATF4 signaling in two different contexts. Firstly, we explored the role of eIF2α-ATF4 signaling in anorexia associated with catabolic inflammatory pathologies (sepsis and cancer). We hypothesized that this signaling pathway could contribute to the inhibition of food intake by its direct action at the central level and/or by stimulating the expression of anorectic cytokines, including GDF15, in the periphery (liver, intestine). We used two experimental models reproducing pathology-associated anorexia in mice: a sepsis model of acute and systemic inflammation (single administration of bacterial lipopolysaccharide) and a model of mice carrying a C26 colon carcinoma cell tumor. Both models were characterized in the early phase of anorexia by inflammation at the peripheral and central (hypothalamus) levels, increased circulating levels of IL-6 and GDF15, profound alterations in amino acid metabolism, and activation of the eIF2αATF4 signaling pathway in the hypothalamus and liver. Afterwards, the response of inducible models of ATF4 loss-of-function was tested in the sepsis model. ATF4 knock-out in the liver and intestine had no impact on either anorexia or the induction of GDF15 production. Constitutive invalidation of GDF15 also had no effect on the inhibition of food intake induced by LPS administration. The role of ATF4 function at the central level could not be tested and should be the subject of future experiments. The analysis of samples from mice knocked-out for ATF4 at the hepatic level, will allow us to evaluate ATF4 involvement in the reorientation of AA metabolism (transport, biosynthesis, autophagy). In the C26 cancer model, the transition from pre-anorexia to early anorexia was associated with an activation of the eIF2α-ATF4 signaling pathway at the hepatic and hypothalamic levels, and a pharmacological approach using ISRIB (ISR Inhibitor) will soon be implemented to study the involvement of the ISR in the regulation of appetite and AA metabolism (in this model, genes knock-out is not possible) Secondly, we focused on mitochondrial dysfunction, which represents a major threat to cellular homeostasis, promotes the development of many metabolic disorders and plays a crucial role in the pathogenesis of sepsis. Given the role played by the eIF2α-ATF4 signaling pathway in the adaptive response to mitochondrial stress, we investigated whether a pretreatment activating this pathway could be a way to increase the resilience of the mitochondrial pool during subsequent stressful events. We demonstrated in mice that a pretreatment activating the GCN2-eIF2α-ATF4 pathway upstream of inflammatory stress (LPS administration) counteracted some of the effects of this stress on mitochondrial homeostasis in the liver, an organ playing a major role in the metabolic and immune response to endotoxic stress. These results are presented as an article that will be submitted soon for publication
Enyindah-Asonye, Gospel. "PATHOPHYSIOLOGICAL ROLE OF CD6 AND ITS NEW LIGAND IN DISEASES". Case Western Reserve University School of Graduate Studies / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=case1491389112552353.
Pełny tekst źródłaFalkenberg, Christiane. "Optimizing Organic Solar Cells". Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2012. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-89214.
Pełny tekst źródłaChang, Shang-wen. "Cu₂S/ZnCdS thin film heterojunction solar cell studies". Diss., Virginia Polytechnic Institute and State University, 1985. http://hdl.handle.net/10919/54740.
Pełny tekst źródłaPh. D.
Allen, Frederick Jr. "CCL3 Augments Antitumor Responses in CT26 by Enhancing Cellular Trafficking and Interferon-Gamma Expression". Case Western Reserve University School of Graduate Studies / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=case1513124234665339.
Pełny tekst źródłaHassan, Namir. "Interactions of the leukocyte cell-surface proteins CD5 and CD6". Thesis, University of Oxford, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.398158.
Pełny tekst źródłaYeh, Ming-Hsin. "Identification of CD8+ T cell-stimulating shared antigens that are uncovered in CT26 vaccinated mice in the absence of CD25+ regulatory T cells". Thesis, University of Southampton, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.431953.
Pełny tekst źródłaLiu, Gin-Yun. "Analysis of the effects of Leptomycin B on Cells Exiting Mitosis". The Ohio State University, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=osu1153488860.
Pełny tekst źródłaAlam, Israt Shamima. "Imaging tumour cell death using the C2A domain of Synaptotagmin-I". Thesis, University of Cambridge, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.609494.
Pełny tekst źródłaLau, Mike Rudi. "Characterisation of the class II phosphoinositide 3-kinase, PI 3K-C2β". Thesis, Institute of Cancer Research (University Of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.342221.
Pełny tekst źródłaCzęści książek na temat "C26 cells"
van de Beek, Malu-Clair, Inge M. E. Dijkstra i Stephan Kemp. "Method for Measurement of Peroxisomal Very Long-Chain Fatty Acid Beta-Oxidation and De Novo C26:0 Synthesis Activity in Living Cells Using Stable-Isotope Labeled Docosanoic Acid". W Methods in Molecular Biology, 45–54. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6937-1_5.
Pełny tekst źródłaSen, Dipankar. "Whole-Cell Protein Profiles of Soil Bacteria by Gel Electrophoresis". W SSSA Book Series, 619–34. Madison, WI, USA: Soil Science Society of America, 2018. http://dx.doi.org/10.2136/sssabookser5.2.c29.
Pełny tekst źródłaDübendorfer, A. "Feedback-Regulation of Ecdysone C20-Hydroxylation in Primary Cell Cultures from Drosophila Embryos". W Invertebrate and Fish Tissue Culture, 39–42. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73626-1_10.
Pełny tekst źródłaMorschhauser, Franck, i Pier Luigi Zinzani. "Indolent Lymphomas". W The EBMT/EHA CAR-T Cell Handbook, 83–86. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-94353-0_15.
Pełny tekst źródłaAktories, K. "C2 toxin (Clostridium botulinum type C and D)". W Guidebook to Protein Toxins and Their Use in Cell Biology, 66–68. Oxford University PressOxford, 1997. http://dx.doi.org/10.1093/oso/9780198599555.003.0023.
Pełny tekst źródłaTurk, Seyhan. "The Impact of Biochemical Alterations in the Tumor Microenvironment on Cancer Progression and Treatment". W Current Researches in Health Sciences-II. Özgür Yayınları, 2023. http://dx.doi.org/10.58830/ozgur.pub128.c626.
Pełny tekst źródłaZinola, C. "The Electrochemical Fuel Cell Reactor". W Surfactant Science, 385–402. CRC Press, 2010. http://dx.doi.org/10.1201/9781420045451-c16.
Pełny tekst źródłaWeete, J., i S. Gandhi. "Enhancement of C20 Polyunsaturated Fatty Acid Production in Pythium ultimum". W Industrial Applications of Single Cell Oils. AOCS Publishing, 1992. http://dx.doi.org/10.1201/9781439821855.ch6.
Pełny tekst źródłaBowen, Phyllis. "Lycopene Oxidation, Uptake, and Activity in Human Prostate Cell Cultures". W Carotenoids, 437–64. CRC Press, 2009. http://dx.doi.org/10.1201/9781420052312-c21.
Pełny tekst źródła"CHAPTER 22. The Vision in the Penitentiary Cell". W Prison Blossoms, 199–203. Harvard University Press, 2011. http://dx.doi.org/10.4159/harvard.9780674066618.c23.
Pełny tekst źródłaStreszczenia konferencji na temat "C26 cells"
Angelotti, Austin, Rachel Cole, Amy Webb, Maciej Pietrzak i Martha Belury. "Diet-induced Gene Expression Changes of Cachectic Muscle, Adipose, and Liver". W 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/gvbe2596.
Pełny tekst źródłaBenedicto, Aitor, Joana Marquez, Elvira Olaso i Beatriz Arteta. "Abstract B10: LFA-1/ICAM-1 interaction switches on an orchestrated prometastatic microenvironmental shift during experimental liver metastasis of colon C26 cancer cells." W Abstracts: AACR Special Conference on Cellular Heterogeneity in the Tumor Microenvironment; February 26 — March 1, 2014; San Diego, CA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.chtme14-b10.
Pełny tekst źródłaLeyva Gutierrez, Francisco, i Tong Wang. "Crystallography and Functionality of Natural Waxes: Insights for the Development of Tailored Lipid Materials". W 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/nyok4571.
Pełny tekst źródłaFranich, Andjela, Ivana Vasić, Snežana Rajković, Aleksandar Arsenijević, Marija Milovanović, Nebojša Arsenijević, Jelena Milovanović i Marija Živković. "CYTOTOXICITY OF CATIONIC DINUCLEAR PLATINUM(II) COMPLEXES IN AN EXPERIMENTAL MODEL OF MOUSE COLON CANCER". W 1st INTERNATIONAL Conference on Chemo and BioInformatics. Institute for Information Technologies, University of Kragujevac, 2021. http://dx.doi.org/10.46793/iccbi21.293f.
Pełny tekst źródłaHolliday, Michael W., Steven B. Cox, Min H. Kang i Barry J. Maurer. "Abstract 4661: Levels of C22:0- and C24:0-dihydroceramide correlate with cytotoxicity in T-cell acute lymphoblastic leukemia cell lines". W Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-4661.
Pełny tekst źródłaLarrouilh, B., K. Mogensen i A. Dehane. "Very Rich Laboratory Data Set Paves the Way for Miscible Gas Injection Evaluation Onshore Abu Dhabi". W ADIPEC. SPE, 2023. http://dx.doi.org/10.2118/216680-ms.
Pełny tekst źródłaKolev, Vihren, Yan Wang, Kam Sprott, Irina Shapiro, Jennifer Ring, Jonathan Pachter i David Weaver. "Abstract C29: FAK inhibition targets cancer stem cells". W Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics; November 5-9, 2015; Boston, MA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1535-7163.targ-15-c29.
Pełny tekst źródłaIbrahim, Omar, Stephen G. Grant, Nicole T. Myers, Amie B. Courtney, Nancy A. lalanne i Jean J. Latimer. "Abstract C26: Analysis of stem cell number & potency in African-American breast tissue". W Abstracts: Ninth AACR Conference on the Science of Cancer Health Disparities in Racial/Ethnic Minorities and the Medically Underserved; September 25-28, 2016; Fort Lauderdale, FL. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7755.disp16-c26.
Pełny tekst źródłaKim, Jeong‐Ah, Wonshik Han, Eun‐Mi Jung, Ki‐Tae Hwang i Dong‐Young Noh. "Abstract C26: Nuclear factor I/B regulates cell proliferation of ER negative breast cancer". W Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Nov 15-19, 2009; Boston, MA. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/1535-7163.targ-09-c26.
Pełny tekst źródłaAn, Ho Jung, Eun Kyoung Choi, Jin-Sun Kim, Seung-Woo Hong, Jai-Hee Moon, Jae-Sik Shin, Seung-Hee Ha i in. "Abstract C276: Ruxolitinib induces apoptotic cell death through the suppression of pJAK1 in human colon cancer cells." W Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Oct 19-23, 2013; Boston, MA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1535-7163.targ-13-c276.
Pełny tekst źródłaRaporty organizacyjne na temat "C26 cells"
Gafni, Yedidya, Moshe Lapidot i Vitaly Citovsky. Dual role of the TYLCV protein V2 in suppressing the host plant defense. United States Department of Agriculture, styczeń 2013. http://dx.doi.org/10.32747/2013.7597935.bard.
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