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Artykuły w czasopismach na temat "Bones – Molecular aspects"
Safarova, S. S. "Pathogenetic aspects of bone metabolism in diabetes mellitus." Clinical Medicine (Russian Journal) 96, nr 8 (20.12.2018): 707–12. http://dx.doi.org/10.18821/0023-2149-2018-96-8-707-712.
Pełny tekst źródłaMaltsev, S. V., A. I. Safina i T. V. Mihajlova. "Гипофосфатемический рахит у детей — клинические и генетические аспекты, подходы к терапии". Practical medicine 19, nr 1 (2021): 38–49. http://dx.doi.org/10.32000/2072-1757-2021-1-38-49.
Pełny tekst źródłaKushchayev, Sergiy V., Yevgeniya S. Kushchayeva, Sri Harsha Tella, Tetiana Glushko, Karel Pacak i Oleg M. Teytelboym. "Medullary Thyroid Carcinoma: An Update on Imaging". Journal of Thyroid Research 2019 (7.07.2019): 1–17. http://dx.doi.org/10.1155/2019/1893047.
Pełny tekst źródłaRice, Ritva, Aki Kallonen, Judith Cebra-Thomas i Scott F. Gilbert. "Development of the turtle plastron, the order-defining skeletal structure". Proceedings of the National Academy of Sciences 113, nr 19 (25.04.2016): 5317–22. http://dx.doi.org/10.1073/pnas.1600958113.
Pełny tekst źródłaAbe, Shinichi, i Masahito Yamamoto. "Factors Involved in Morphogenesis in the Muscle–Tendon–Bone Complex". International Journal of Molecular Sciences 22, nr 12 (14.06.2021): 6365. http://dx.doi.org/10.3390/ijms22126365.
Pełny tekst źródłaJentgen-Ceschino, Benjamin, Koen Stein i Valentin Fischer. "Case study of radial fibrolamellar bone tissues in the outer cortex of basal sauropods". Philosophical Transactions of the Royal Society B: Biological Sciences 375, nr 1793 (13.01.2020): 20190143. http://dx.doi.org/10.1098/rstb.2019.0143.
Pełny tekst źródłaRichardson, Jo, Takanori Shono, Masataka Okabe i Anthony Graham. "The presence of an embryonic opercular flap in amniotes". Proceedings of the Royal Society B: Biological Sciences 279, nr 1727 (czerwiec 2011): 224–29. http://dx.doi.org/10.1098/rspb.2011.0740.
Pełny tekst źródłaGentile, Cristina, i Francesco Chiarelli. "Rickets in Children: An Update". Biomedicines 9, nr 7 (27.06.2021): 738. http://dx.doi.org/10.3390/biomedicines9070738.
Pełny tekst źródłaIwaszczuk, Urszula, Justyna Niderla-Bielińska i Aneta Ścieżyńska. "Kings and peasants from El-Zuma/El-Detti microregion in the Early Makurian period. Economic aspects of animal bones from funerary contexts". PLOS ONE 14, nr 2 (15.02.2019): e0212423. http://dx.doi.org/10.1371/journal.pone.0212423.
Pełny tekst źródłaYANG, XIUPING, FENGJU SUN, LONGTAO WANG, CHUNQIU ZHANG i XIZHENG ZHANG. "SOLUTE TRANSPORT IN ARTICULAR CARTILAGE UNDER ROLLING-COMPRESSION LOAD". Journal of Mechanics in Medicine and Biology 19, nr 06 (wrzesień 2019): 1950054. http://dx.doi.org/10.1142/s0219519419500544.
Pełny tekst źródłaRozprawy doktorskie na temat "Bones – Molecular aspects"
Cheng, Tak Sum. "Molecular identification and characterization of novel osteoclast V-ATPase subunits". University of Western Australia. School of Surgery and Pathology, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0068.
Pełny tekst źródłaDai, Zhijie, i 戴志洁. "The role of sodium/myo-inositol cotransporter 1 and myo-inositol in osteogenesis and bone formation". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43783533.
Pełny tekst źródłaChan, Cheuk-wing Wilson, i 陳卓榮. "ER stress in the pathogenesis of osteochondrodysplasia". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43085192.
Pełny tekst źródłaWang, Cathy Ting-Peng. "Molecular dissection of RANKL signaling pathways in osteoclasts". University of Western Australia. School of Surgery and Pathology, 2007. http://theses.library.uwa.edu.au/adt-WU2008.0037.
Pełny tekst źródłaCheng, Yin-wo, i 鄭燕和. "Molecular basis for the increased osteoblast activity in a mouse modelwith hyperostosis". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B34612981.
Pełny tekst źródłaLee, B. C. Bob, i 李卜駿. "Probing the molecular mechanisms of how polymorphisms in Cerberus-likeresult in low bone mineral density". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B39793771.
Pełny tekst źródłaCestari, Tania Mary. "Aspectos celulares, teciduais e moleculares da osteogênese ectópica e ortotópica induzida pela matriz alogênica óssea e dentinária". Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/25/25142/tde-02062009-111408/.
Pełny tekst źródłaThe aim of the present work was to correlate the cellular and tissue events with the expression of VEGF, BMP-7, RANKL and OPG during ectopic and orthotopic osteogenesis, induced by bone and dentin allogeneic matrix. Allogenic matrices obtained from femur and incisor of rats and demineralized in 0.6 N HCl were implanted into a intramuscular pocket and a 8mm-diameter bone defect in the skull. The radiographic and histomorphometric analysis of new bone formation, and immunohistochemistry and western blotting for VEGF, BMP, RANKL and OPG proteins, showed that: a) the total volume of the graft region in orthotopic site decreased 19% at 42 days b) in both graft types and implantation sites occurred formation of cartilaginous and bone tissues, c) in intramuscular sites, the resorption of allogenic matrix and remodeling of the new formed cartilage and bone were faster, in relation to orthotopic implantation sites; d) the increase in the volume density of blood vessels and in the number of osteoblasts/osteocytes and osteoclasts occurred simultaneously and was associated with greater reabsorption of the allogenic matrix and hematopoietic bone marrow formation; e) VEGF, BMP-7, RANKL, OPG proteins were expressed in chondrocytes, active osteoblasts, newly osteocytes confined and stromal cells located near the osteoblasts or in the surface of the reabsorbed matrix; and f) the VEGF, BMP-7, RANKL and OPG expression was higher in MO grafts than in the MD. The peak of expression of these proteins each occurred at 14 and 21 days in MO and 21 and 28 days in MD. We concluded that, the osteoinductive capacity of allogeneic demineralized matrix is related to matrix origin and implantation site and that the VEGF, BMP-7, RANKL and OPG proteins are associated with greater reabsorption of the implanted matrix, promoting rapid and continuous matrix-release morphogens that induces spatially and temporally the bone and bone marrow formation.
Albuquerque, Dulcinéia Martins de. "Aspectos moleculares do citomegalovirus humano durante infecção ativa em pacientes submetidos ao transplante de medula ossea". [s.n.], 2006. http://repositorio.unicamp.br/jspui/handle/REPOSIP/311935.
Pełny tekst źródłaTese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas
Made available in DSpace on 2018-08-08T16:11:05Z (GMT). No. of bitstreams: 1 Albuquerque_DulcineiaMartinsde_D.pdf: 3473302 bytes, checksum: daa8d0ff76ca0850d748389f7bb2ea56 (MD5) Previous issue date: 2006
Resumo: O Citomegalovírus Humano (HCMV) continua sendo uma causa significante de morbidade em pacientes imunocomprometidos, especialmente em transplantados de medula óssea, e pode manifestar diversas complicações que incluem hepatite, doença gastrointestinal e pneumonia intersticial ou a denominada "Síndrome Viral por HCMV"caracterizada por febre, leucopenia e trombocitopenia. O HCMV pode também ter um efeito imuno-modulador, fazendo da infecção por esse vírus um fator de risco importante para o desenvolvimento de rejeição ao enxerto aguda e crônica e para co-infecção com outras herpesviroses. A detecção do genoma do HCMV pela PCR (Reação em Cadeia da Polimerase) é específica e sensível, e pode ser usada como uma poderosa ferramenta para o diagnóstico precoce da infecção causada por este vírus. Variações em regiões funcionalmente relevantes do genoma do HCMV têm sido utilizadas como marcadores genéticos em diversos estudos clínicos para diferenciar as linhagens do vírus e associá-las com a patogênese viral e com as manifestações clínicas no paciente. A glicoproteína B (gB) é a maior glicoproteína do envelope do HCMV e tem sido relacionada à entrada na célula hospedeira, transmissão célula-a-célula, e conseqüentemente à fusão das células infectadas. A amplificação do gene gB pela PCR combinada com análise de restrição por RFLP em regiões polimórficas deste gene são eficientes para a identificação dos genótipos do HCMV, tornando possível a distinção de pelo menos 4 padrões eletroforéticos. Por outro lado, a determinação da carga viral em pacientes imunologicamente afetados tem sido associada como marcador ou preditor do desenvolvimento de doença por HCMV órgão-específica. Sendo assim, a determinação da carga viral, especificamente nestes pacientes, é fundamental para a supervisão da terapia antiviral. Além disso, os valores da carga viral estão relacionados aos níveis de imunossupressão, à patogênese do HCMV e ao grupo de pacientes e/ou ao tipo de transplante e podem indicar o início da administração da terapia antiviral.O método de real-time PCR (RT-PCR) foi aplicado para a quantificação do genoma do HCMV em amostras clínicas e a detecção e posterior quantificação do DNA do HCMV em amostras de soro por esta técnica é capaz de distinguir entre pacientes com infecções sintomáticas daqueles com infecções inativas ou latente.Avanços têm sido feitos na prevenção da doença por HCMV após o transplante de medula óssea, inclusive a administração profilática, por períodos prolongados, de antivirais como o Acyclovir e o Ganciclovir e como conseqüência, pode originar linhagens resistentes relacionadas principalmente a dois genes virais: a fosfotransferase viral (UL97) e a DNA polimerase viral (UL54). Sabendo-se da importância da identificação das linhagens do HCMV em pacientes transplantados de medula óssea e da possível relação com a infecção e apresentação clínica; da relevância em determinar a carga viral como preditor de doença; e finalmente, da detecção de linhagens resistentes aos agentes antivirais disponíveis, este estudo avaliou, prospectivamente, pacientes transplantados de medula óssea em seguimento no Hemocentro/UNICAMP. Além disso, teve como principais objetivos: determinar a prevalência dos genótipos do HCMV e avaliar uma possível associação com a apresentação clínica nesses pacientes; determinar a carga viral para o monitoramento da terapia antiviral; e identificar e correlacionar mutações que conferem resistência ao Ganciclovir com carga viral e apresentação clínica. Foram incluídas na casuística, 169 amostras de DNA de sangue periférico e 187 amostras de DNA de soro de 22 pacientes transplantados de medula óssea. Dentre as 47 amostras de DNA de sangue periférico HCMV positivas, 42 foram genotipadas e observamos a prevalência do genótipo gB1 (47%) como descrito em literatura, e embora sem comprovação estatística, notamos a tendência deste genótipo com melhor prognóstico. Aplicamos a RT-PCR em 96 amostras de DNA de soro de 12 pacientes transplantados de medula óssea seguidos no Ambulatório de Hematologia, e observamos que o método é adequado para a avaliação da carga viral neste grupo de pacientes. No entanto, é necessário estabelecer um valor de corte a fim de se utilizar esta metodologia para obtenção de um valor que seja preditivo de doença e para o monitoramento do tratamento dos pacientes. Este método mostrou-se mais preciso que a ?nested?-PCR no mesmo tipo de amostra. Além disso, identificamos 8 novas mutações no gene UL97, uma delas pode estar relacionada à resistência viral ao Ganciclovir. Dentre os polimorfismos identificados, 3 parecem estar relacionados ao genótipo gB1 e possivelmente podem ser utilizadas como marcadores genéticos para a genotipagem do HCMV. Para o gene UL54 foram identificadas 5 novas mutações na região IV do gene e que geralmente é relacionada à resistência ao Ganciclovir. Nós concluímos que a determinação da carga viral é importante, mas não é o único modo de avaliar a eficiência do tratamento antiviral. Dessa forma, a avaliação de outros parâmetros moleculares, como a genotipagem e mutações relacionadas à resistência aos antivirais, são informações complementares e devem ser consideradas para o monitoramento da evolução clínica em pacientes transplantados de medula óssea
Abstract: Human Cytomegalovirus (HCMV) remains a significant cause of morbidity in immunocompromised patients, especially in bone marrow transplant recipients. It may manifest severe complications including hepatitis, gastrointestinal disease, and interstitial pneumonitis or as so-called ?HCMV viral syndrome? with fever, leukopenia, and thrombocytopenia. The HCMV may also has an immunomodulatory effect, potentially making HCMV infection an important risk factor for the development of an acute and chronic allograft rejection and for coinfection with other herpesviruses. The detection of the HCMV genome by PCR (Polymerase Chain Reaction) is specific and sensitive. Besides this, it can be used as a powerful tool for the early diagnoses of the infection caused by this virus. Variations in functionally relevant areas of the HCMV genome have been used as genetic markers in numerous clinical studies to differentiate the HCMV strains and to associate them with the viral pathogenesis further with the patients? clinical manifestations. The glycoprotein B (gB) is the major glycoprotein of HCMV?s envelope and it has been implicated in host cell entry, cell-to-cell virus transmission, consequently in the fusion of infected cells. The gB amplification by PCR combined with the restriction analysis by RFLP in polymorphic areas are effective for the identification of the HCMV genotypes, becoming possible the distinction of at least 4 electrophoretic patterns. On the other hand, the determination of the viral load in the immunologically affected patients has been associated as marker or predictor for the development of the organ specific disease by the HCMV. Hence, the determination of the viral load in these specific patients is fundamental for the management of the antiviral therapy. In addition, the viral load values are related to levels of the immune-suppression, the pathogenesis of the HCMV and the group of patients and/or the type of transplant. Furthermore, the viral load values can indicate the beginning of the antiviral therapy administration. A real-time PCR (RT-PCR) assay was applied for quantifying the HCMV genome load in clinical samples and the detection and quantification of HCMV DNA in blood serum through RT-PCR are able to distinguish patients with symptomatic infections among those with latent or inactive infections. Advances have been made in the prevention of HCMV disease after bone marrow transplantation, including prophylactic administration of antivirals such as Acyclovir and Ganciclovir. The HCMV prophylaxis with antiviral in this patients? group is administered for prolonged periods of therapy, consequently it can originate resistant viruses related mainly to two genes: the viral phosphotransferase (UL97) and the viral DNA polymerase (UL54). Ahead the importance of the identification of HCMV strains in bone marrow transplant patients, the HCMV strains performance in the patients? infection and clinical presentation, the relevance of determinating the viral load as a disease predictor, and finally, the detection of the resistant strains to the available antivirals, this study prospectively evaluated bone marrow transplant recipients followed at Hemocentro/UNICAMP. Moreover, it had as main goals: to determine the prevalence of the HCMV gB genotypes, to evaluate a possible gB genotype association with the patients? clinical presentation; to determinate the viral load for monitoring the antiviral therapy, and to correlate Ganciclovir resistant mutations in UL97 and UL54 gene with the viral load and patients? clinical presentation. From 22 bone marrow transplant recipients, DNA samples of peripheral blood (169) and DNA samples of blood serum (187) were included in this casuistic. Among 47 HCMV positive samples, 42 were genotyped. We observed the prevalence of gB1 genotype (47%), as described in the specific literature, however without statistical analysis, the raw data exhibited that gB1 genotype can be related to patients? better prognostics. From 12 followed bone marrow transplant recipients, we applied the RT-PCR in 96 DNA blood serum samples and we observed that the method was accurate for the viral load evaluation in this patients? group. However, it is necessary to establish a crucial cutoff to consider whether a specific value of viral load is a predictive value to cause HCMV disease and to monitor the patients? treatment. This method was more precise than the nested-PCR for blood serum samples. Additionally, we identified 8 new mutations in UL97 gene, one of them can be related to Ganciclovir HCMV resistance. Among all of identified polymorphisms, 3 of them can be related to gB1 genotype and may be used as genetic marker to HCMV genotyping. In the region IV of the UL54 gene, 5 new mutations were identified, and can possibly be related to Ganciclovir HCMV resistance. We concluded that the determination of the patients? viral load is crucial, even so it is not the only way to evaluate the antiviral treatment efficacy. Then, the evaluation of other molecular parameters as genotyping and mutations related to the HCMV antiviral resistance, are complementary information and must be considered to monitor the clinical evolution of bone marrow transplant recipients
Doutorado
Ciencias Basicas
Doutor em Clínica Médica
PEREIRA, Andrea de Castro. "Sítios implantáveis da maxila e mandíbula: correlação entre aspectos clínico-radiográficos e histomorfométrico-moleculares". Universidade Federal de Goiás, 2011. http://repositorio.bc.ufg.br/tede/handle/tde/1367.
Pełny tekst źródłaThe study of bone microarchitecture and its molecular aspects may provide new information for better understanding of "bone quality". Histomorphometry is a recommended reference method for bone-dimensional analysis. At the molecular level, possible changes in the process of resorption and bone formation has not been studied in different patterns of bone "normal." Objective: To analyze the correlation between clinical, radiographic and histomorphometric bone-molecular sites for dental implants in humans. Methods: The sample consisted of 44 sites of 32 volunteers implantable. These sites were classified according to three different methods: a classification based only on periapical and panoramic images (PP), the classification of Lekholm and Zarb, based on methods of diagnostic imaging in conjunction with the tactile perception of the surgeon during surgery and classification of Lindh. The bone specimens were removed with the use of trephine during the first drilling site for placement of dental implants. The samples were subjected to staining with hematoxylin-eosin and histomorphometric analysis to obtain the following histomorphometric parameters: trabecular thickness (Tb.Th), trabecular number, bone volume fraction (BV / TV), trabecular bone surface area by bone volume (BS / BV), bone surface fraction and trabecular separation (Tb.Sp). We also made the technique for analysis of proteins immunoistoquímica RANK, RANKL, OPG and osteocalcin (OC) in samples of bone tissue. Results: Rankings PP & L and Z correlated with BV / TV, BS / BV, Tb.Th and Tb.Sp. The classification of Lindh did not correlate with any histomorphometric parameter. L & Z show differences between types when compared to bone BV / TV, BS / BV, Tb.Th and Tb.Sp. We found a weak correlation between ratings PP / L & Z and the expression of regulators of bone metabolism (RANK, RANKL, OPG and OC). Conclusions: It may be that the subjective ratings conculir types of bone are influenced by aspects of histomorphometry and the regulatory molecules of bone remodeling seems not to exert influence in the morphology of the maxilla and mandible.
O estudo da microarquitetura óssea e dos seus aspectos moleculares podem trazer novas informações para a melhor compreensão da qualidade óssea . A histomorfometria é um método de referência recomendado para análise óssea bidimensional. A nível molecular, possíveis alterações das vias de reabsorção e formação óssea ainda não foram estudadas nos diferentes padrões de osso normal . Objetivo: Analisar a correlação entre aspectos clínico-radiográficos e histomorfométrico-moleculares de sítios ósseos para implantes dentários em humanos. Material e métodos: A amostra foi composta por 44 sítios implantáveis de 32 voluntários. Estes sítios foram classificados de acordo com 3 diferentes métodos: uma classificação baseada somente em imagens periapical e panorâmica (PP); a classificação de Lekholm e Zarb, baseada em métodos de diagnóstico por imagens em conjunto com a percepção tátil do cirurgião durante a cirurgia e a classificação de Lindh. Os espécimes ósseos foram removidos com o uso da trefina durante a primeira perfuração do sítio para colocação dos implantes dentários. As amostras foram submetidas à técnica de coloração com hematoxilina-eosina e análise histomorfométrica para obtenção dos seguintes parâmetros histomorfométricos: espessura trabecular (Tb.Th), número de trabéculas, fração de volume ósseo (BV/TV), área de superfície óssea trabecular pelo volume ósseo (BS/BV), fração de superfície óssea e separação trabecular (Tb.Sp). Também foi realizada a técnica da immunoistoquímica para análise das proteínas RANK, RANKL, OPG e Osteocalcina (OC) nas amostras de tecido ósseo. Resultados: As classificações PP e L&Z apresentaram correlação com BV/TV, BS/BV, Tb.Th e Tb.Sp. A classificação de Lindh não apresentou correlação com nenhum parâmetro histomorfométrico. L&Z demonstrou diferença entre os tipos ósseos quando comparado a BV/TV, BS/BV, Tb.Th e Tb.Sp. Foi encontrada uma fraca correlação entre as classificações PP/L&Z e a expressão dos reguladores do metabolismo ósseo (RANK, RANKL, OPG e OC). Conclusões: Pode-se conculir que as classificações subjetivas dos tipos ósseos são influenciadas pelos aspectos histomorfométricos e que as moléculas reguladoras da remodelação óssea parecem não exercer influência nos aspectos morfológicos da maxila e mandíbula.
Irani, Dilshad Minocher. "Role of the surface associated material of Eikenella corrodens in bone resorption associated with periodontal disease : a research thesis submitted in fulfilment of the requirements for the degree of Master of Science in Dentistry". Title page, contents and summary only, 1998. http://web4.library.adelaide.edu.au/theses/09DSM/09dsmi65.pdf.
Pełny tekst źródłaKsiążki na temat "Bones – Molecular aspects"
Raisz, Lawrence G. (Lawrence Gideon), 1925-, Martin T. John i ScienceDirect (Online service), red. Principles of bone biology. Wyd. 3. Amsterdam: Elsevier, 2008.
Znajdź pełny tekst źródłaDietz, Georg. Calcium hydroxide and bone regeneration: Odontological aspects of induced osteogenesis in experiment and clinical practice. München: G. Dietz, 1998.
Znajdź pełny tekst źródłaRosen, Vicki. The cellular and molecular basis of bone formation and repair. New York: Springer, 1995.
Znajdź pełny tekst źródła1957-, Thies Robert Scott, red. The cellular and molecular basis of bone formation and repair. Austin: R.G. Landes, 1995.
Znajdź pełny tekst źródłaCardew, Gail. The molecular basis of skeletogenesis. Chichester: Wiley, 2001.
Znajdź pełny tekst źródłaWackym, Phillip A. Molecular temporal bone pathology, parts III and IV. Philadelphia, PA: Lippincott-Raven, 1998.
Znajdź pełny tekst źródłaMasaki, Noda, red. Cellular and molecular biology of bone. San Diego: Academic Press, 1993.
Znajdź pełny tekst źródłaHong-wen, Deng, i Liu Yao-zhong, red. Current topics in bone biology. Singapore: World Scientific, 2005.
Znajdź pełny tekst źródła(Editor), Hong-Wen Deng, Yao-zhong Liu (Editor), Chun-Yuan Guo (Editor) i Di Chen (Editor), red. Current Topics in Bone Biology. World Scientific Publishing Company, 2005.
Znajdź pełny tekst źródła(Foreword), G. A. Rodan, Felix Bronner (Editor) i Mary C. Farach-Carson (Editor), red. Bone Formation (Topics in Bone Biology). Springer, 2003.
Znajdź pełny tekst źródłaCzęści książek na temat "Bones – Molecular aspects"
Bab, Itai A., i Jona J. Sela. "Cellular and Molecular Aspects of Bone Repair". W Principles of Bone Regeneration, 11–41. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-2059-0_2.
Pełny tekst źródłaWinston, Drew J. "Cytomegalovirus Infection in Bone Marrow Transplantation". W Molecular Aspects of Human Cytomegalovirus Diseases, 183–204. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-84850-6_11.
Pełny tekst źródłaDawidowska, Małgorzata. "Isolation of Mononuclear Cells from Human Blood and Bone Marrow by Density Gradient Centrifugation". W Molecular Aspects of Hematologic Malignancies, 305–8. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-29467-9_19.
Pełny tekst źródłaWiren, Kristine M. "Androgen Action in Bone: Basic Cellular and Molecular Aspects". W Osteoporosis, 359–83. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-459-9_16.
Pełny tekst źródłaCrocker, Paul R., i Siamon Gordon. "Studies on the Interaction between Murine Resident Bone Marrow Macrophages and Haematopoietic Cells". W Molecular and Cellular Aspects of Erythropoietin and Erythropoiesis, 259–72. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-72652-1_20.
Pełny tekst źródłaSpiro, Ira J., i Herman D. Suit. "Radiation-induced bone and soft tissue sarcomas: Clinical aspects and molecular biology". W Cancer Treatment and Research, 143–55. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-6121-7_10.
Pełny tekst źródłaWoo, Je-Tae, Yasuo Ohba, Kahori Tagami, Koji Sumitani, Kohji Yamaguchi, Tomoko Tsuji, Takao Kataoka i Kazuo Nagai. "Low Molecular Weight Microbial Metabolites that Suppress Bone Resorption by Inhibiting Vacuolar Type Proton Pump Activity of Osteoclasts". W Animal Cell Technology: Basic & Applied Aspects, 627–32. Dordrecht: Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5746-9_102.
Pełny tekst źródłaLuque, J., M. D. Delgado, E. Ferrer, M. Moreno, M. Pinilla i P. Sancho. "The Use of Two-Phase Systems for the Fractionation of Heterogeneous Populations of Bone Marrow Cells and Erythrocytes: Bisphosphoglycerate Mutase as an Enzyme Marker for Erythroid Cells". W Molecular and Cellular Aspects of Erythropoietin and Erythropoiesis, 353–71. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-72652-1_26.
Pełny tekst źródłaTirode, Franck, i Thomas G. P. Grünewald. "Molecular aspects of Ewing's sarcomas". W Bone Cancer, 617–30. Elsevier, 2022. http://dx.doi.org/10.1016/b978-0-12-821666-8.00022-0.
Pełny tekst źródła"Cellular and Molecular Aspects". W Stem Cells and Bone Tissue, 123. CRC Press, 2013. http://dx.doi.org/10.1201/b14590-9.
Pełny tekst źródłaStreszczenia konferencji na temat "Bones – Molecular aspects"
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Pełny tekst źródłaSancaktar, E., i J. Kuznicki. "Stress-Dependent Water Uptake Behavior of Clay Reinforced Nanocomposite Epoxy". W ASME 2005 International Mechanical Engineering Congress and Exposition. ASMEDC, 2005. http://dx.doi.org/10.1115/imece2005-80549.
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