Gotowe bibliografie tematyczne / Basal ganglia

Gotowa bibliografia na temat „Basal ganglia”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 1 sierpnia 2024

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Artykuły w czasopismach na temat "Basal ganglia"

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Redgrave, Peter. "Basal ganglia". Scholarpedia 2, nr 6 (2007): 1825. http://dx.doi.org/10.4249/scholarpedia.1825.

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Brüggemann, Norbert, Susanne A. Schneider, Thurid Sander, Christine Klein i Johann Hagenah. "Distinct basal ganglia hyperechogenicity in idiopathic basal ganglia calcification". Movement Disorders 25, nr 15 (27.08.2010): 2661–64. http://dx.doi.org/10.1002/mds.23264.

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Tso, Winnie Wan-Yee, Ada Wing-Yan Yung, Hin-Yue Lau i Godfrey Chi-Fung Chan. "Basal Ganglia Germinoma". Journal of Pediatric Hematology/Oncology 36, nr 7 (październik 2014): e443-e447. http://dx.doi.org/10.1097/mph.0000000000000014.

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Patell, R. D., R. V. Dosi, H. K. Joshi i P. C. Shah. "Basal ganglia calcification". Case Reports 2013, aug13 1 (13.08.2013): bcr2013200399. http://dx.doi.org/10.1136/bcr-2013-200399.

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Albin, Roger L. "BASAL GANGLIA NEUROTOXINS". Neurologic Clinics 18, nr 3 (sierpień 2000): 665–80. http://dx.doi.org/10.1016/s0733-8619(05)70217-6.

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Graybiel, Ann M. "The basal ganglia". Current Biology 10, nr 14 (lipiec 2000): R509—R511. http://dx.doi.org/10.1016/s0960-9822(00)00593-5.

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Graybiel, Ann M. "The basal ganglia". Trends in Neurosciences 18, nr 2 (luty 1995): 60–62. http://dx.doi.org/10.1016/0166-2236(95)80019-x.

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KONSTANTINOU, GERASIMOS, ANASTASIA STAVRINOU, SAVVINA MYLONA, GEORGE PASCHALAKIS i POLYXENI VASILOPOULOU. "Basal Ganglia Calcification". Journal of Psychiatric Practice 25, nr 5 (wrzesień 2019): 391–94. http://dx.doi.org/10.1097/pra.0000000000000410.

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Braunlich, Kurt, i Carol Seger. "The basal ganglia". Wiley Interdisciplinary Reviews: Cognitive Science 4, nr 2 (21.12.2012): 135–48. http://dx.doi.org/10.1002/wcs.1217.

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Vaštík Kateřina Menšíková Sandra Kurčová, Miroslav, i Michaela Kaiserová Zuzana Matejčíková Petr Kaňovský. "Functional basal ganglia interconnection". Neurologie pro praxi 18, nr 4 (1.10.2017): 266–70. http://dx.doi.org/10.36290/neu.2017.097.

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Rozprawy doktorskie na temat "Basal ganglia"

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Moss, Jonathan. "Microcircuitry of the Basal Ganglia". Thesis, University of Oxford, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.514971.

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Drinnan, Suzane Loraine. "G proteins in the basal ganglia". Thesis, University of British Columbia, 1990. http://hdl.handle.net/2429/28981.

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G proteins are alpha-beta-gamma heterotrimers in the resting state, bound to GDP and complexed with the unbound receptor. Once the receptor becomes occupied, the alpha subunit exchanges GDP for GTP, becomes activated, and dissociates from the receptor and can stimulate or inhibit many intracellular activities such as phosphorylation and channel conductance. For example, Gs and Golf alpha subunits stimulate and Gi alpha subunits inhibit adenylyl cyclase. Go alpha subunits are abundant in brain, but are of unknown function. cDNAs for the alpha subunit have been cloned. In order to examine the relative distributions of G proteins in the brain, we used in situ hybridization with radiolabelled synthetic oligonucleotide probes. By using a tyrosine hydroxylase antibody, we found that the dopaminergic neurons of the substantia nigra and the noradrenergic neurons of the locus ceruleus express mRNA for the alpha subunits for each of Gi, Go, and Gs. We noted a paucity of Gs mRNA in the striatum. This was surprising because the basal ganglia contain a dopamine-stimulated adenylyl cyclase activity which has been assumed to be transduced by Gs. Also, immunohistochemistry, immunoblotting, and cholera ADP-ribosylation indicated a very high level of Gs alpha-like protein in the striatum. In order to ascertain which specific G protein we were detecting, we made probes to a new G protein previously identified in the olfactory system. Golf is a stimulatory G protein with size and sequence characteristics similar to those of Gs. The cholera toxin ADP-ribosylation site and C-terminal region to which the antibody was made are identical. We made oligonucloetide probes to the translated and untranslated portions of Golf alpha. High levels Golf mRNA and protein were detected in the striatum and nucleus accumbens, in addition to the expected high levels in the olfactory tubercle. Northern blot studies indicated that Golf transcripts are approximately ten-fold more abundant than Gs alpha transcripts in the striatum. These data indicate that Golf in not an olfactory-specific G protein. It is also the major stimulatory G protein in the basal ganglia. The selective expression of high levels of Golf in dopamine-rich forebrain areas suggest that it may couple DI dopamine receptors to adenylyl cyclase. The role of Golf in dopaminergic neurotransmission and neuropsychiatric disease should be considered.
Medicine, Faculty of
Graduate
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Mogoseanu, Diana. "Basal ganglia connections with orofacial muscles". Thesis, University of Oxford, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.260710.

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Smith, Denise P. A. "The Basal Ganglia and Sequential Learning". Kent State University / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=kent1353430597.

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Church, Andrew John. "Anti-basal ganglia antibodies in movement disorders". Thesis, University College London (University of London), 2006. http://discovery.ucl.ac.uk/1444607/.

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Sydenham's chorea (SC) is a neurological manifestation following group A Streptococcus infection (GABHS) and has been proposed as an antibody-mediated autoimmune disease. Other movement and psychiatric manifestations following GABHS have been recognised and termed Paediatric Autoimmune Neuropsychiatric Disorders Associated with Streptococcal infections (PANDAS). It is proposed that PANDAS may be caused by the same antibody as SC. As the symptoms of PANDAS are identical to Tourette's syndrome (TS), the possibility that TS might turn out to be an autoimmune disorder has implications for the treatment and understanding of these disorders. Evidence of GABHS was found in all patients with SC and PANDAS and 60% of patients with TS. Autoantibodies against basal ganglia (ABGA) were found in all acute SC and PANDAS patients. Only 25% of TS patients were ABGA positive. There was little evidence for ABGA in controls. There was a higher prevalence of ABGA in systemic diseases associated with GABHS but this did not reach significance. ABGA bound to proteins with molecular weights (40, 45, 60 and 98 kDa) and these responses were variably found in SC, PANDAS and TS. The identification of these antigens proved to be problematic due to contamination with other proteins with the same molecular weights. Neurone specific enolase (NSE) was identified as one of the antigens. As this protein was not specific to basal ganglia it cast doubt as to the specificity of ABGA. Interestingly, however, enolase is also found on the surface of GABHS and has extensive homology with human enolase, thus lending support to the possibility of molecular mimicry derived autoimmunity.
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Brown, Jennifer. "Feedback motor control and the basal ganglia". Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648678.

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Noy, G. "Abnormal motor behaviour and the basal ganglia". Thesis, University of Manchester, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.370974.

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Hutton, Elizabeth Anne May. "Somatosensory cortical input to the basal ganglia". Thesis, University of Edinburgh, 1998. http://webex.lib.ed.ac.uk/abstracts/hutton01.pdf.

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van, Albada Sacha. "Mean-field analysis of basal ganglia and thalamocortical dynamics". Connect to full text, 2008. http://ses.library.usyd.edu.au/handle/2123/5124.

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Thesis (Ph. D.)--University of Sydney, 2009.
Includes graphs and tables. Includes list of publications. Title from title screen (viewed June 17, 2009) Submitted in fulfilment of the requirements for the degree of Doctor of Philosophy to the School of Physics, Faculty of Science. Degree awarded 2009; thesis submitted 2008. Includes bibliographical references. Also available in print form.
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Deus, Yela Juan. "Sistema fronto-basal y aprendizaje incidental". Doctoral thesis, Universitat de Barcelona, 1996. http://hdl.handle.net/10803/670405.

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La memoria no es un proceso unitario. Se han definido diferentes subsistemas mnesicos que operan mediante circuitos neuroanatómicos distintos. Tradicionalmente, los estudios de la memoria se han centrado en el aprendizaje intencionado, especificando que el lóbulo temporal y las estructuras diencefálicas son esenciales para este tipo de memoria. Contrariamente, hay pocos estudios sobre el aprendizaje incidental y no han concretado las estructuras anatómicas implicadas en este aprendizaje. Ya que se considera que este último tipo de aprendizaje es un proceso mnésico automático de adquisición de la información, cabe presuponer la posible implicacion del estriado, de forma similar a lo que ocurre con el sistema motor. Se han seleccionado una muestra de 114 sujetos, 70 como pacientes y 44 pertenecen al grupo control de sujetos sanos. La muestra de pacientes está constituida por sujetos afectos de la enfermedad de Alzheimer (N=20), de la enfermedad de Huntington (N=21), pacientes con calcificaciones bilaterales en los ganglios basales (N=18) y pacientes con una lesión vascular unilateral en el estriado (N=11). Se ha diseñado una exploración neuropsicológica que ha evaluado funciones cognitivas generales, visoperceptivas y visoespaciales, aprendizaje intencionado, aprendizaje procedimental y funciones frontales en base a pruebas clínicamente estandarizadas. De forma expresa, se han diseñado tareas para evaluar el aprendizaje incidental del espacio, frecuencia y orden temporal. Nuestros resultados sugieren que el aprendizaje incidental puede estar controlado por circuitos frontoneoestriados bilaterales y puede utilizarse como un indicador del funcionamiento mnésico que permita ayudar a discriminar entre la demencia subcortical y cortical, considerando conjuntamente el funcionamiento de la memoria declarativa y procedimental.
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Książki na temat "Basal ganglia"

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Soghomonian, Jean-Jacques, red. The Basal Ganglia. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-42743-0.

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Chihiro, Ohye, Kimura M. 1947-, McKenzie John S i International Basal Ganglia Society. Symposium, red. The basal ganglia V. New York: Plenum Press, 1996.

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Bolam, J. Paul, Cali A. Ingham i Peter J. Magill, red. The Basal Ganglia VIII. Boston, MA: Springer US, 2005. http://dx.doi.org/10.1007/0-387-28066-9.

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Percheron, Gérard, John S. McKenzie i Jean Féger, red. The Basal Ganglia IV. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4613-0485-2.

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Carpenter, Malcolm B., i A. Jayaraman, red. The Basal Ganglia II. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5347-8.

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Graybiel, Ann M., Mahlon R. Delong i Stephen T. Kitai, red. The Basal Ganglia VI. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0179-4.

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Bernardi, Giorgio, Malcolm B. Carpenter, Gaetano Di Chiara, Micaela Morelli i Paolo Stanzione, red. The Basal Ganglia III. Boston, MA: Springer New York, 1991. http://dx.doi.org/10.1007/978-1-4684-5871-8.

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Groenewegen, Hendrik Jan, Pieter Voorn, Henk W. Berendse, Antonius B. Mulder i Alexander R. Cools, red. The Basal Ganglia IX. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-1-4419-0340-2.

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Nicholson, Louise F. B., i Richard L. M. Faull, red. The Basal Ganglia VII. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0715-4.

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Ohye, Chihiro, Minoru Kimura i John S. McKenzie, red. The Basal Ganglia V. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4899-0194-1.

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Części książek na temat "Basal ganglia"

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Marmarou, Christina R., Matthew R. Parry i Ekaterina Dobryakova. "Basal Ganglia". W Encyclopedia of Clinical Neuropsychology, 481–87. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_298.

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Marmarou, Christina R., Matthew R. Parry i Ekaterina Dobryakova. "Basal Ganglia". W Encyclopedia of Clinical Neuropsychology, 1–7. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_298-2.

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Cho, Youngsun T. "Basal Ganglia". W Encyclopedia of Autism Spectrum Disorders, 1–2. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4614-6435-8_102059-1.

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McKinlay, Audrey. "Basal Ganglia". W Encyclopedia of Child Behavior and Development, 206–9. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-0-387-79061-9_287.

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ten Donkelaar, Hans J., Bart van de Warrenburg, Michèl Willemsen, Benno Küsters, Yoshio Hashizume i Akira Hori. "Basal Ganglia". W Clinical Neuroanatomy, 591–667. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-41878-6_11.

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Cho, Youngsun T. "Basal Ganglia". W Encyclopedia of Autism Spectrum Disorders, 600–602. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-91280-6_102059.

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Ewert, Jörg-Peter. "Basal Ganglia". W Encyclopedia of Sciences and Religions, 187. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-1-4020-8265-8_200144.

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ten Donkelaar, Hans J., Bart van de Warrenburg, Michèl Willemsen, Benno Küsters, Yoshio Hashizume i Akira Hori. "Basal Ganglia". W Clinical Neuroanatomy, 495–564. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-19134-3_11.

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Marmarou, Christina R., i Matthew R. Parry. "Basal Ganglia". W Encyclopedia of Clinical Neuropsychology, 346–51. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_298.

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Feng, Shao. "Basal Ganglia". W The ECPH Encyclopedia of Psychology, 1–2. Singapore: Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-99-6000-2_705-1.

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Streszczenia konferencji na temat "Basal ganglia"

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Miglierini, Marcel, Adriana Lančok, Martin Kopáni i Roman Boča. "Mössbauer spectroscopy of Basal Ganglia". W MOSSBAUER SPECTROSCOPY IN MATERIALS SCIENCE - 2014. AIP Publishing LLC, 2014. http://dx.doi.org/10.1063/1.4898624.

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Mohagheghi-Nejad, Mohammad Reza, Fariba Bahrami i Mahyar Janahmadi. "Conductance-based computational model of basal ganglia". W 2014 22nd Iranian Conference on Electrical Engineering (ICEE). IEEE, 2014. http://dx.doi.org/10.1109/iraniancee.2014.6999867.

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Medini, Chaitanya, Anjitha Thekkekuriyadi, Surya Thayyilekandi, Manjusha Nair, Bipin Nair i Shyam Diwakar. "Modeling basal ganglia microcircuits using spiking neurons". W 2016 International Conference on Advances in Computing, Communications and Informatics (ICACCI). IEEE, 2016. http://dx.doi.org/10.1109/icacci.2016.7732383.

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Ferreira, Marcos Venâncio Araújo, Rafael Henrique Neves Gomes, Fabiana Carla dos Santos Correia, Mariana Beber Chamon, Sérgio Roberto Pereira da Silva Júnior, Isadora Chain Lima, Marcus Vinicius de Sousa, Murilo Justino de Almeida, Daniel Sabino de Oliveira i Thiago Cardoso Vale. "Idiopathic basal ganglia calcification and Hoarding disorder". W XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.499.

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Introduction: Basal ganglia calcifications are associated with many neurological and metabolic disorders, being present also on asymptomatic patients. It may present in its primary form, including familial and sporadic cases. Its secondary form is associated especially to hypoparathyroidism but also associated to infections, toxic exposure, rheumatologic diseases, mitochondrial disorders. It has an heterogenous clinical presentation with movement disorders and neuropsychiatric symptoms. Case presentation: A 66-year-old patient presented with a progressive hoarding disorder for the last six years. In the last 2 years started an aggressive behavior, loss of acquired skills, urinary incontinence, sleep-wake cicle disorder and one episode of focal seizure. Physical examination revealed bilateral asymmetrical tremor, bradykinesia and cogwheel rigidity. MoCA test was 23/30 for 12 years of schooling. Brain Computed Tomography showed calcifications in basal ganglia affecting predominantly pallidum e thalamus and cerebellar hemispheres. Brain Magnetic Resonance Imaging revealed hypointensites in the same regions and in nucleus caudate suggestive of calcification. Laboratory testing for endocrine and calcium metabolism was normal. No clinical signs of other disorders. Discussion: We presented a case of probable Idiopathic Basal Ganglia Calcification initially treated as a hoarding disorder. The normal laboratory results, lack of other clinical signs and familial history suggests a primary sporadic form that might be due to de novo mutations or transmitted by asymptomatic parent. The most commonly mutations in SLC20A2, PDGFB and PDGFRB but genetic testing is commonly unavailable. Parkinsonism is the most common movement disorder and the neuropsychiatric features include cognitive impairment, psychotic and obsessive compulsive disorders. Conclusion: This case demonstrates that attention is needed to the progression of psychiatric disorders suggesting some rare neurological disorders.
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Wang, Yuhao, Armin Lak, Sanjay Manohar i Rafal Bogacz. "Uncertainty-driven exploration in the basal ganglia". W 2023 Conference on Cognitive Computational Neuroscience. Oxford, United Kingdom: Cognitive Computational Neuroscience, 2023. http://dx.doi.org/10.32470/ccn.2023.1378-0.

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Gilmour, Timothy P., Constantino Lagoa, W. Kenneth Jenkins, Anand N. Rao, Matthew A. Berk, Kala Venkiteswaran i Thyagarajan Subramanian. "Transfer entropy between cortical and basal ganglia electrophysiology". W 2012 IEEE Signal Processing in Medicine and Biology Symposium (SPMB). IEEE, 2012. http://dx.doi.org/10.1109/spmb.2012.6469453.

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Song, Enmin, Valerie A. Cardenas, Frank Ezekiel i Michael W. Weiner. "Automatic boundary modification of warped basal ganglia template". W Medical Imaging 2001, redaktorzy Milan Sonka i Kenneth M. Hanson. SPIE, 2001. http://dx.doi.org/10.1117/12.431023.

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Elibol, Rahmi, i Neslihan Serap Sengor. "Modeling basal ganglia circuits with mass model equations". W 2016 Medical Technologies National Congress (TIPTEKNO). IEEE, 2016. http://dx.doi.org/10.1109/tiptekno.2016.7863131.

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Cavalcante Franca Junior, Marcondes, i Lara GusmÃo Vicente Dos Anjos. "Basal Ganglia abnormalities in ALS: Texture and volumetric analysis". W XXIII Congresso de Iniciação Científica da Unicamp. Campinas - SP, Brazil: Galoá, 2015. http://dx.doi.org/10.19146/pibic-2015-37478.

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Guiyeom Kang i M. M. Lowery. "Conductance-based model of the basal ganglia in Parkinson's Disease". W IET Irish Signals and Systems Conference (ISSC 2009). IET, 2009. http://dx.doi.org/10.1049/cp.2009.1692.

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Raporty organizacyjne na temat "Basal ganglia"

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Arbib, Michael A. Mechanisms of the Basal Ganglia for Arm-Hand Coordination. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 1996. http://dx.doi.org/10.21236/ada315470.

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Andrades, Oscar, David Ulloa, Dario Martinez, Francisco Guede, Gustava Muñoz, Luis Javier Chirosa i Amador García. Effect of the manipulation of the variables that configure the stimulus of strength training on motor symptoms in people with Parkinson's disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, listopad 2022. http://dx.doi.org/10.37766/inplasy2022.11.0079.

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Review question / Objective: To analyze the evidence on studies that have manipulated the variables that make up the strength training stimulus and its effects on motor symptoms in people with Parkinson's disease. Condition being studied: Parkinson's is a multisystemic neurodegenerative disease that affects the central nervous system and is caused by a loss of dopaminergic neurons in the compact part of the substantia nigra of the basal ganglia of the midbrain. People with Parkinson's disease (PEP) have non-motor and motor clinical symptoms. Classic motor symptoms are rest tremor, joint stiffness, bradykinesia, decreased balance, gait disturbances (speed, temporality, spatiality, support, and freezing) and decreased functional performance.
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Wang, Xiao, Hong Shen, Yujie Liang, Yixin Wang, Meiqi Zhang i Hongtao Ma. Effectiveness of Tango Intervention on Motor Symptoms in Patients with Parkinson's Disease: A Protocol for Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, maj 2022. http://dx.doi.org/10.37766/inplasy2022.5.0009.

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Review question / Objective: Parkinson's disease (PD) is a degenerative neurological disease caused by the loss of dopaminergic neurons in the pars compacta of the substantia nigra of the brain, resulting in lesions in the basal ganglia. The main motor symptoms of PD include resting tremor, rigidity, akinesia or bradykinesia and postural instability. As an exercise intervention based on musical accompaniment, tango dance has shown positive effects on the rehabilitation of motor symptoms in PD patients in recently. In this study, we systematically reviewed the efficacy of tango intervention in alleviating the motor symptoms of patients with PD. Condition being studied: Parkinson. Information sources: The following electronic databases will be searched: PubMed, Cochrane Central Register of Controlled Trials (CENTRAL), Web of Science Core collection, and China National Knowledge Infrastructure Database (CNKI) and WanFang Database.
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Wang, Jiepu, Zihao An, Lianxuan Liu i Xiaobo Yu. Stereotactic Drainage Treatment is Superior to Conservative Medical Treatment in Patients with Small Volume Basal Ganglion Hemorrhage: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, marzec 2024. http://dx.doi.org/10.37766/inplasy2024.3.0111.

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Utility of the ADC Map in Identification of Diffusion Restriction on a Background of Basal Ganglia Iron Deposition. Science Repository OU, kwiecień 2019. http://dx.doi.org/10.31487/j.rdi.2019.02.01.

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