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Data publikacji: 18 maja 2024

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1

Prior, Philippe, Caitilyn Allen i John Elphinstone, red. Bacterial Wilt Disease. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-03592-4.

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2

1957-, Prior Ph, Allen C. 1957-, Elphinstone John G i International Bacterial Wilt Symposium (2nd : 1997 : Gosier, Guadeloupe), red. Bacterial wilt disease: Molecular and ecological aspects. Berlin: Springer, 1998.

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3

Thomas, George V. Coconut root (wilt) disease management. Kasaragod: Central Plantation Crops Research Institute, Indian Council of Agricultural Research, 2010.

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4

C, Hayward A., Hartman Glen Lee 1955- i Asian Vegetable Research and Development Center., red. Bacterial wilt: The disease and its causative agent, Pseudomonas solanacearum. Wallingford, UK: CAB International in association with the Asian Vegetable Research and Development Center, 1994.

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5

International, Verticillium Symposium (7th 1997 Athens Greece). Advances in verticillium: Research and disease management. St. Paul, Minn: APS Press, 2000.

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6

J, Persley G., Batugal Ponciano A i Philippine Council for Agriculture and Resources Research and Development., red. Bacterial wilt disease in Asia and the South Pacific: Proceedings of an international workshop held at PCARRD, Los Baños, Philippines, 8-10 October 1985. Canberra: Australian Centre for International Agricultural Research, 1986.

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7

A, Griesbach John, i Oregon. State Dept. of Agriculture., red. The Tomato spotted wilt virus: Information and disease management guidelines. [Salem, Or.?: Oregon State Dept. of Agriculture, 1991.

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8

Mbwika, J. M. Feasibility study on technologies for improving banana for resistance against bacterial wilt in sub-Saharan Africa. Nairobi: African Agricultural Technology Foundation, 2009.

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9

Gurung, T. B. Baseline study report of a community participatory bacterial wilt management programme in Ulleri and Jhilibarang villages of Western Nepal. Pokhara: Lumle Regional Agricultural Research Centre, 1997.

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10

Zwet, Tom Van Der. Fire blight--its nature, prevention, and control: A practice guide to integrated disease management. [Washington, D.C.?]: U.S. Dept. of Agriculture, 1992.

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Zwet, Tom Van Der. Fire blight--its nature, prevention, and control: A practice guide to integrated disease management. [Washington, D.C.?]: U.S. Dept. of Agriculture, 1992.

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12

V, Beer S., United States. Agricultural Research Service. i Cornell University, red. Fire blight--its nature, prevention, and control: A practical guide to integrated disease management. [Washington, DC]: U.S. Dept. of Agriculture, 1999.

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13

Zwet, Tom Van Der. Fire blight--its nature, prevention, and control: A practical guide to integrated disease management. [Washington, DC]: U.S. Dept. of Agriculture, 1999.

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14

Zwet, Tom Van Der. Fire blight--its nature, prevention, and control: A practical guide to integrated disease management. [Washington, DC]: U.S. Dept. of Agriculture, 1999.

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15

Zwet, Tom Van Der. Fire blight--its nature, prevention, and control: A practice guide to integrated disease management. [Washington, D.C.?]: U.S. Dept. of Agriculture, 1992.

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16

V, Beer S., United States. Agricultural Research Service i Cornell University, red. Fire blight--its nature, prevention, and control: A practical guide to integrated disease management. [Washington, DC]: U.S. Dept. of Agriculture, 1999.

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17

V, Beer S., Cornell University i United States. Agricultural Research Service, red. Fire blight--its nature, prevention, and control: A practice guide to integrated disease management. [Washington, D.C.?]: U.S. Dept. of Agriculture, 1992.

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18

V, Beer S., United States. Agricultural Research Service i Cornell University, red. Fire blight--its nature, prevention, and control: A practical guide to integrated disease management. [Washington, DC]: U.S. Dept. of Agriculture, 1999.

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19

V, Beer S., United States. Agricultural Research Service. i Cornell University, red. Fire blight--its nature, prevention, and control: A practical guide to integrated disease management. [Washington, DC]: U.S. Dept. of Agriculture, 1995.

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20

Zwet, Tom Van Der. Fire blight--its nature, prevention, and control: A practice guide to integrated disease management. [Washington, D.C.?]: U.S. Dept. of Agriculture, 1992.

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21

Zwet, Tom Van Der. Fire blight--its nature, prevention, and control: A practical guide to integrated disease management. [Washington, DC]: U.S. Dept. of Agriculture, 1995.

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22

Amyes, Sebastian G. B. Magic Bullets, Lost Horizons. London: Taylor & Francis Inc, 2004.

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23

Smith, Robert M. Other bacterial diseasesErysipeloid. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0025.

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Erysipeloid is an acute bacterial infection usually causing acute localised cellulitis as a secondary infection of traumatised skin. It is caused by Erysipelothrix rhusiopathiae (insidiosa), a non-sporulating Gram-positive rod-shaped bacterium, ubiquitous in the environment. It is the cause of swine erysipelas and also a pathogen or commensal in a variety of wild and domestic birds, animal and marine species. Human infection primarily associated with occupational exposure to infected or contaminated animals or handling animal products and therefore is commoner in farmers, butchers and abattoir workers and fisherman.Risk factors for the rare human invasive E. rhusiopathiae infection include conditions that affect the host immune response, such as alcoholism, cancer and diabetes. Treatment is with penicillin.Erysipelas can affect animals of all ages but is recognised more frequently in juveniles. Swine exhibit similar stages to the disease in man. Clinical manifestations in swine vary from the classical rhomboid urticaria (diamond skin), the condition of greatest prevalence and economic importance, to sepsis, polyarthritis, pneumonia and death.Prevention is largely a matter of good hygiene, herd management and by raising awareness in those at risk (especially butchers, farmers and fishermen); ensuring that clinicians are aware of E. rhusiopathiae as a possible cause of occupational skin lesions and bacterial endocarditis is important.
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24

(Editor), Philippe Prior, Caitilyn Allen (Editor) i John Elphinstone (Editor), red. Bacterial Wilt Disease: Molecular and Ecological Aspects. Springer, 1998.

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25

Prior, Philippe, Caitilyn Allen i John Elphinstone. Bacterial Wilt Disease: Molecular and Ecological Aspects. Springer, 2011.

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26

Prior, Philippe, Caitilyn Allen i John Elphinstone. Bacterial Wilt Disease: Molecular and Ecological Aspects. Springer London, Limited, 2013.

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27

(Editor), Caitilyn Allen, Philippe Prior (Editor) i A. C. Hayward (Editor), red. Bacterial Wilt Disease And The Ralstonia Solanacearum Species Comples. American Phytopathological Society, 2005.

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28

(Editor), A. C. Hayward, i G. L. Hartman (Editor), red. Bacterial Wilt: The Disease and Its Causative Agent, Pseudomonas solanacearum (Cabi Publishing). CABI, 1996.

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29

Persley, G. J. Bacterial Wilt Disease in Asia and the South Pacific (Aciar Proceedings, No 13). Australian Centre for International Agricultural Research, 1987.

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30

Greece) International Verticillium Symposium 1997 (Akra Sounion. Advances in Verticillium: Research and Disease Management. American Phytopathological Society, 2000.

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31

Griffith, David E. Nontuberculous Mycobacterial Disease: A Comprehensive Approach to Diagnosis and Management. Springer, 2018.

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32

Griffith, David E. Nontuberculous Mycobacterial Disease: A Comprehensive Approach to Diagnosis and Management. Humana, 2018.

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33

Espada, Margarida, Ryoji Shinya, Hong Mei Li, Anna Filipiak i Claudia S. L. Vicente, red. Global Occurrence of Pine Wilt Disease: Biological Interactions and Integrated Management. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88976-867-7.

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34

Brook, Itzhak. Anaerobic Infections: Diagnosis and Management (Infectious Disease and Therapy). Informa Healthcare, 2007.

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35

Merkuz Abera Admassu (Ph.D). EPIDEMIOLOGY AND MANAGEMENT OF CHICKPEA FUSARIUM WILT IN ETHIOPIA: Disease Distribution, Incidences and Management Options. LAP Lambert Academic Publishing, 2010.

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36

Fire blight--its nature, prevention, and control: A practical guide to integrated disease management. [Washington, DC]: U.S. Dept. of Agriculture, 1995.

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37

Gillespie, Stephen H. Management of Multiple Drug-Resistant Infections (Infectious Disease). Humana Press, 2004.

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38

Pediatric Anaerobic Infections: Diagnosis and Management (Infectious Disease and Therapy). Wyd. 3. Informa Healthcare, 2001.

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39

Infective Endocarditis: Management in the Era of Intravascular Devices (Infectious Disease and Therapy). Informa Healthcare, 2007.

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40

Shanmugam, Naresh P., i Dharam Basude. Complications of chronic liver disease. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759928.003.0067.

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The chapter on complications of chronic liver disease gives an overview of the definition, pathophysiology, diagnosis, and management of the various complications that accompany chronic liver disease. It includes among others, malnutrition and growth failure, hepatic encephalopathy, hepatopulmonary syndrome, hepatorenal syndrome, portal hypertension, and spontaneous bacterial peritonitis.
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41

Rudwaleit, Martin, i Atul Deodhar. Diagnosis, classification, and management of peripheral spondyloarthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198734444.003.0004.

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Spondyloarthritis (SpA) can affect the axial skeleton (axSpA) but also manifest as peripheral arthritis, enthesitis, and dactylitis (peripheral SpA). Peripheral SpA can occur after bacterial infections (reactive arthritis) or be associated with psoriasis or inflammatory bowel disease. The arthritis is usually asymmetric, affects predominantly the lower extremity, and can be self-limiting but can also run a chronic course. The frequency of HLA-B27 is around 50% in purely peripheral SpA, while it is 70–90% in axSpA. For classification, the Amor, ESSG, or more recent ASAS criteria for peripheral SpA can be used. The ASAS criteria are likely to capture early peripheral SpA better than the other two. Therapy includes NSAIDs, local steroid injections, and synthetic disease-modifying antirheumatic drugs, of which sulfasalazine is best studied and the preferred drug for peripheral arthritis. A recent, placebo-controlled clinical trial with adalimumab may lead to the first approval of a biologic in peripheral SpA.
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42

Chiumello, Davide, i Silvia Coppola. Management of pleural effusion and haemothorax. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0125.

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The main goal of management of pleural effusion is to provide symptomatic relief removing fluid from the pleural space. The options depend on type, stage, and underlying disease. The first diagnostic instrument is the chest radiography, while ultrasound can be very useful to guide thoracentesis. Pleural effusion can be a transudate or an exudate. Generally, a transudate is uncomplicated effusion treated by medical therapy, while an exudative effusion is considered complicated effusion and should be managed by drainage. Refractory non-malignant effusions can be transudative (congestive heart failure, cirrhosis, nephrosis) or exudative (pancreatitis, connective tissue disease, endocrine dysfunction), and the management options include repeated therapeutic thoracentesis, in-dwelling pleural catheter for intermittent external drainage, pleuroperitoneal shunts for internal drainage, or surgical pleurectomy. Parapneumonic pleural effusions can be classified as complicated when there is persistent bacterial invasion of the pleural space, uncomplicated and empyema with specific indications for pleural fluid drainage. Malignancy is the most common cause of exudative pleural effusions in patients aged >60 years and the decision to treat depends upon the presence of symptoms and the underlying tumour type. Options include in-dwelling pleural catheter drainage, pleurodesis, pleurectomy, and pleuroperitoneal shunt. Haemothorax needs to be differentiated from a haemorrhagic pleural effusion and, when suspected, the essential management is intercostal drainage. It achieves two objectives to drain the pleural space allowing expansion of the lung and to allow assessment of rates of blood loss to evaluate the need for emergency or urgent thoracotomy.
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43

Amyes, Sebastian G. B. Magic Bullets, Lost Horizons: The Rise and Fall of Antibiotics. Taylor & Francis Group, 2001.

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Amyes, Sebastian G. B. Magic Bullets, Lost Horizons: The Rise and Fall of Antibiotics. Taylor & Francis Group, 2001.

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45

Amyes, Sebastian G. B. Magic Bullets, Lost Horizons: The Rise and Fall of Antibiotics. CRC, 2001.

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Amyes, Sebastian G. B. Magic Bullets, Lost Horizons: The Rise and Fall of Antibiotics. Taylor & Francis Group, 2001.

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Amyes, Sebastian G. B. Magic Bullets, Lost Horizons: The Rise and Fall of Antibiotics. Taylor & Francis Group, 2001.

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Amyes, Sebastian G. B. Magic Bullets, Lost Horizons: The Rise and Fall of Antibiotics. Taylor & Francis Group, 2001.

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Magic Bullets, Lost Horizons: The Rise and Fall of Antibiotics. CRC, 2001.

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Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson i David Ledingham. Neurological infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0015.

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This chapter reviews the clinical features and management of meningitis (community-acquired bacterial meningitis and chronic meningitis), Mycobacterium tuberculosis, encephalitis and myelitis, Lyme disease, brain abscess and parameningeal infection, neurological infections in the immunocompromised, fungal infection, parasitic infection, and bacterial neurotoxins.
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