Gotowe bibliografie tematyczne / Bacterial resistance

Gotowa bibliografia na temat „Bacterial resistance”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 22 czerwca 2024

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Artykuły w czasopismach na temat "Bacterial resistance"

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Venkatesan, Nandakumar, Govindaraj Perumal i Mukesh Doble. "Bacterial resistance in biofilm-associated bacteria". Future Microbiology 10, nr 11 (listopad 2015): 1743–50. http://dx.doi.org/10.2217/fmb.15.69.

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Gentry, Layne O. "Bacterial Resistance". Orthopedic Clinics of North America 22, nr 3 (lipiec 1991): 379–88. http://dx.doi.org/10.1016/s0030-5898(20)31668-0.

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Bockstael, Katrijn, i Arthur Aerschot. "Antimicrobial resistance in bacteria". Open Medicine 4, nr 2 (1.06.2009): 141–55. http://dx.doi.org/10.2478/s11536-008-0088-9.

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AbstractThe development of antimicrobial resistance by bacteria is inevitable and is considered as a major problem in the treatment of bacterial infections in the hospital and in the community. Despite efforts to develop new therapeutics that interact with new targets, resistance has been reported even to these agents. In this review, an overview is given of the many therapeutic possibilities that exist for treatment of bacterial infections and how bacteria become resistant to these therapeutics.
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Shifa Begum, Tofa Begum, Naziza Rahman i Ruhul A. Khan. "A review on antibiotic resistance and way of combating antimicrobial resistance". GSC Biological and Pharmaceutical Sciences 14, nr 2 (28.02.2021): 087–97. http://dx.doi.org/10.30574/gscbps.2021.14.2.0037.

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Antibiotics are widely used most effective medication since the twentieth century against bacterial infections (Tetanus, Strep Throat, Urinary Tract Infections, etc.) and thus save one’s life. Before 20th-century infectious disease played the main role in the death. Thus, antibiotics opened a revolutionary era in the field of medication. These cannot fight against viral infections. Antibiotics are also known as an antibacterial that kill or slow down bacterial growth and prohibit the bacteria to harm. Resistance comes as a curse with antibiotics that occurs when bacteria change in some way that reduces or eliminates the effectiveness of drugs, chemicals or other agents designed to cure or prevent infections. It is now a significant threat to public health that is affecting humans worldwide outside the environment of the hospital. When a bacterium once become resistant to antibiotic then the bacterial infections cannot be cured with that antibiotic. Thus, the emergence of antibiotic-resistance among the most important bacterial pathogens causing more harm. In this context, the classification of antibiotics, mode of action of antibiotics, and mechanism of resistance and the process of overcoming antibiotic resistance are discussed broadly.
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Gangwar, Sonali, Keerti Kaushik i Maya Datt Joshi. "Current Mechanism of Bacterial Resistance to Antimicrobials". SAMRIDDHI : A Journal of Physical Sciences, Engineering and Technology 10, nr 01 (25.07.2018): 55–64. http://dx.doi.org/10.18090/samriddhi.v10i01.8.

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Serious infectious diseases are caused by bacterial pathogens that represents a serious public health concern. Antimicrobial agents are indicated for the treatment bacterial infections.Various bacteria carries several resistance genes also called multidrug resistant (MDR). Multidrug resistant organisms have emerged not only in the hospital environment but are now often identified in community settings, suggesting the reservoirs of antibiotic resistant bacteria are present outside the hospital. Drug resistant bacteria that are selected with a single drug are also frequently multi-drug resistant against multiple structurally different drugs, thus confounding the chemotherapeutic efficacy of infectious disease caused by such pathogenic variants. The molecular mechanisms by which bacteria have common resistance to antibiotics are diverse and complex. This review highlights the mechanism of bacterial resistance to antimicrobials.
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Johnson, Alan P., i Neil Woodford. "Bacterial antibiotic resistance". Current Opinion in Infectious Diseases 6, nr 4 (sierpień 1993): 515–19. http://dx.doi.org/10.1097/00001432-199308000-00005.

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Taylor, Diane E. "Bacterial tellurite resistance". Trends in Microbiology 7, nr 3 (marzec 1999): 111–15. http://dx.doi.org/10.1016/s0966-842x(99)01454-7.

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Tumah, H. N. "Bacterial Biocide Resistance". Journal of Chemotherapy 21, nr 1 (luty 2009): 5–15. http://dx.doi.org/10.1080/1120009x.2009.12030920.

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Iredell, Jon. "Bacterial resistance testing". Pathology 46 (2014): S46. http://dx.doi.org/10.1097/01.pat.0000443500.86097.3f.

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Wilson, Fred. "Battling Bacterial Resistance". Laboratory Medicine 32, nr 2 (1.02.2001): 73–76. http://dx.doi.org/10.1309/734f-d2x1-37fy-499k.

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Rozprawy doktorskie na temat "Bacterial resistance"

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Chung, Whasun Oh. "Macrolide resistance and its linkage to tetracycline resistance /". Thesis, Connect to this title online; UW restricted, 1999. http://hdl.handle.net/1773/9279.

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Powell, James Patrick. "Antibiotic Diversity and Bacterial Resistance". Available to users online at:, 2007. http://hdl.handle.net/10156/1303.

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Messer, Janet Mariam. "Bacterial resistance ot glycopeptide antibiotics". Thesis, University of Cambridge, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.239488.

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Brown, Keith L. "Heat resistance of bacterial spores". Thesis, University of Nottingham, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.317036.

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Sütterlin, Susanne. "Aspects of Bacterial Resistance to Silver". Doctoral thesis, Uppsala universitet, Klinisk mikrobiologi och infektionsmedicin, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-247472.

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Bacterial resistance to antibiotics has increased rapidly within recent years, and it has become a serious threat to public health. Infections caused by multi-drug resistant bacteria entail higher morbidity, mortality, and a burden to health care systems. The use of biocides, including silver compounds, may affect the resistance to both biocides and antibiotics and, thereby, can be a driving factor in this development. The aim of the following thesis was to investigate the frequency of silver resistance and the effects of silver exposure on bacterial populations being of clinical significance and from geographically different parts of the world. Furthermore, it explored the genetic background of silver resistance, and if silver could select directly or indirectly for antibiotic resistance. By a range of methods, from culture in broth to whole genome sequencing, bacterial populations from humans, birds and from the environment were characterized. The studies showed that sil genes, encoding silver resistance, occurred at a high frequency. Sil genes were found in 48 % of Enterobacter spp., in 41 % of Klebsiella spp. and in 21 % of all human Escherichia coli isolates with production of certain types of extended-spectrum beta-lactamases (CTX-M-14 and CTX-M-15). In contrast, silver resistance was not found in bird isolates or in bacterial species, such as Pseudomonas aeruginosa and Legionella spp., with wet environments as their natural habitat. One silver-resistant Enterobacter cloacae strain was isolated from a chronic leg ulcer after only three weeks of treatment with silver-based dressings. The in-vivo effects of these dressings were limited, and they failed to eradicate both Gram-positive and Gram-negative bacteria. The activity of silver nitrate in vitro was bacteriostatic on Gram-positive species such as S. aureus and bactericidal on Gram-negative species. In Enterobacteriaceae, sil genes were associated with silver resistance phenotypes in all but one case. Using whole genome sequencing, single nucleotide polymorphisms in the silS gene were discovered after silver exposure in isolates with expressed silver resistance. This resistance could co-select for resistance to beta-lactams, co-trimoxazole and gentamicin. The findings of this thesis indicate that silver exposure may cause phenotypic silver resistance, and it may reduce the susceptibility to mainly beta-lactams and select for bacteria with resistance to clinically important antibiotics.
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Crossland, Richard J. "Mathematical modelling of bacterial mercury resistance". Thesis, University of Nottingham, 2015. http://eprints.nottingham.ac.uk/30521/.

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A mathematical model of mercury resistance was designed which describes the following reactions: the cellular uptake and volatilisation of Hg2+, binding of the DNA by the regulator, mer protein synthesis, and dilution of quantities by cell growth. A total of 66 biological experiments were then selected from the scientific literature from studies of Tn21 and Tn501 in E. coli at 37 °C. These experiments were repeated in the computer simulation and the information from their 489 data points was incorporated into the 16 parameters of the model using the Metropolis-Hastings algorithm. This model is very useful biology for four reasons. Firstly, it shows whether the data from existing biological experiments are consistent with each other or not. Secondly, it predicts the previously unknown concentrations of mer proteins in cells of each mercury phenotype. In addition, it challenges the hypotheses that the rates of uptake and volatilisation are always equal in resistant cells and that the plasmid copy number effects replicated by the model are caused by the saturation of MerT in the membrane. Thirdly, the model can guide the design of future experiments. This guidance can minimise the use of laboratory resources and will ensure that sufficient data are created for every parameter in the model under standardised conditions. Finally, the modelling has identified many areas for future biological research: the absolute concentrations of mer proteins, the significance of MerC and MerD, plasmid copy number effects and substrate inhibition, the three uptake processes (non-mer import, MerA transport, and non-MerA transport), the order of DNA + MerR + Hg2+ binding, the nature of toxicity, and the concentrations of mercury in each of the five cellular binding sites.
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Leszcynski, Robert A. "Determination of the Relationship Between Bacterial Coculturing, Antibiotic Resistance and Bacterial Growth". University of Dayton / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=dayton1591787505690696.

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I, D'Agostino. "Polyalkylguanidines: new weapons to tackle bacterial resistance". Doctoral thesis, Università di Siena, 2019. http://hdl.handle.net/11365/1070884.

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In this historical period known as the antibiotic crisis era, the ever faster rise of bacterial strains resistant to the clinically used antibiotics along with the scientific research silent gap in the antibacterial field is treating seriously to the worldwide public health. Hence, we urgently need to develop new antibacterials agents with an innovative mode of action, able to trick the mechanisms of the pathogen resistance. In this alarming frame, aware of the antibacterial properties of guanidine moieties, Prof. M. Botta and his research group have evaluated the biological activity of a linear polyalkylguanidino series, synthesized for different medicinal purpose, toward a panel of bacterial microorganisms. Only one compound (1) emerged to have an interesting broad-spectrum antibacterial activity. Later, the serendipitous discovery that the test batch of compound 1 was actually a mixture of oligomers led us to identify the chemical structure of the main component, dimer 2, which was the responsible for the activity. From its scaffold, we designed and synthesized a small library of analogs to make some preliminary consideration on the pharmacophores with the aim of improving the selectivity index and studying the mode(s) of action.
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Cousin, Sydney Louis. "Macrolide resistance in Neisseria gonorrhoeae /". Thesis, Connect to this title online; UW restricted, 2003. http://hdl.handle.net/1773/5078.

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Nilsson, Annika. "Bacterial adaptation to novel selection pressures /". Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-192-X/.

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Książki na temat "Bacterial resistance"

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1934-, Wax Richard G., red. Bacterial resistance to antimicrobials. Wyd. 2. Boca Raton: CRC Press, 2008.

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Mandal, Santi M., i Debarati Paul, red. Bacterial Adaptation to Co-resistance. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-8503-2.

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Yi, Yŏng-sŏn. Chiyŏk sahoe nae hangsaengje naesŏng yŏnghyang pʻyŏngka =: Effective evaluations of antimicrobial resistant pathogens within the community. [Seoul]: Sikpʻum Ŭiyakpʻum Anjŏnchʻŏng, 2007.

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Amábile-Cuevas, Carlos F. Origin, evolution and spread of antibiotic resistance genes. Austin: R.G. Landes Co., 1993.

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Derek, Wakelin, i Blackwell J. M, red. Genetics of resistance to bacterial and parasitic infection. London: Taylor & Francis, 1988.

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Border, Peter. Diseases fighting back: The growing resistance of TB and other bacterial diseases to treatment. London: Parliamentary Office of Science and Technology, 1994.

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F, Amábile-Cuevas Carlos, red. Multiple drug resistant bacteria. Wymondham, Norfolk, England: Horizon Scientific Press, 2003.

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Bacterial biofilms. Berlin: Springer, 2008.

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author, Wilker Ian, i Ambrose Marylou author, red. Tuberculosis and superbugs: Examining TB and bacterial infections. Berkeley Heights, NJ: Jasmine Health, 2014.

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F, Baquero, red. Evolutionary biology of bacterial and fungal pathogens. Washington, D.C: ASM Press, 2007.

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Części książek na temat "Bacterial resistance"

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Livermore, David M. "Bacterial Resistance to Carbapenems". W Antimicrobial Resistance, 25–47. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4757-9203-4_3.

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Setlow, Peter. "Spore Resistance Properties". W The Bacterial Spore, 201–15. Washington, DC, USA: ASM Press, 2016. http://dx.doi.org/10.1128/9781555819323.ch10.

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Turner, Raymond J. "Bacterial Tellurite Resistance". W Encyclopedia of Metalloproteins, 219–23. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-1533-6_451.

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Grimsley, N., i J. F. Wang. "Chair’s Perspective: Host Resistance". W Bacterial Wilt Disease, 197–99. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-03592-4_29.

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Setlow, Peter. "Resistance of Bacterial Spores". W Bacterial Stress Responses, 319–32. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816841.ch18.

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Huletsky, A., i Michel G. Bergeron. "Bacterial Genotypic Drug Resistance Assays". W Antimicrobial Drug Resistance, 1465–99. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-47266-9_37.

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Datta, Naomi. "Bacterial Resistance to Antibiotics". W Ciba Foundation Symposium 102 - Origins and Development of Adaptation, 204–18. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470720837.ch13.

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Kumar, Anil, i Nikita Chordia. "Bacterial Resistance Against Antibiotics". W Drug Resistance in Bacteria, Fungi, Malaria, and Cancer, 171–92. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-48683-3_7.

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Woc-Colburn, Laila, i Denise Marie A. Francisco. "Multidrug Resistance Bacterial Infection". W Highly Infectious Diseases in Critical Care, 139–46. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-33803-9_8.

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Bradford, Patricia A. "Epidemiology of Bacterial Resistance". W Antimicrobial Resistance in the 21st Century, 299–339. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78538-7_10.

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Streszczenia konferencji na temat "Bacterial resistance"

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Rodrigues, Francisco, Raquel Teixeira i Patricia Coelho. "Antibiotic consumption in Portugal - An overview". W III SEVEN INTERNATIONAL MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/seveniiimulti2023-225.

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Infections caused by opportunistic bacteria resistant to multiple antibiotics continue to be a major challenge in the hospital environment. Growing bacterial resistance results in compromising the success of immunosuppressive therapies and surgical interventions (transplantation), which associated with a high risk of bacterial infections during the procedure and post-operatively often culminate in the death of the patient.
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Svanova, Pavla. "METAL-RESISTANCE IN RHODOCOCCUS BACTERIAL STRAIN". W 15th International Multidisciplinary Scientific GeoConference SGEM2015. Stef92 Technology, 2015. http://dx.doi.org/10.5593/sgem2015/b61/s25.066.

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Mao, Jianing, Mingfu Gong i Qinlan Guan. "Induced disease resistance of endophytic bacteria REB01 to bacterial blight of rice". W 2018 INTERNATIONAL CONFERENCE ON BIOTECHNOLOGY AND BIOENGINEERING (8TH ICBB). Author(s), 2019. http://dx.doi.org/10.1063/1.5092395.

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Iancu, Ionica, Viorel Herman, Ileana Nichita i Alexandru Gligor. "RESEARCH ON THE AEROBIC BACTERIAL FLORA ISOLATED FROM CATTLE WITH CLINICAL ENDOMETRITIS". W 23rd SGEM International Multidisciplinary Scientific GeoConference 2023. STEF92 Technology, 2023. http://dx.doi.org/10.5593/sgem2023/6.1/s25.23.

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In cattle, uterine infections are one of the major causes of reproductive disorders. These bacterial infections are frequently treated with antibiotics, but their effectiveness must be evaluated, due to the continuous emergence of bacterial strains with multiple drug resistances. The research was conducted on several 190 samples from cows with a presumptive diagnosis of endometritis. The exploitation technology practiced at this farm is the free stall type. Samples were collected using sterile swabs and transported under appropriate conditions to the laboratory. The definitive identification of suspected etiological agents was made with the help of diagnostic kits: API 20 E - for the identification of pathogens from the Enterobacteriaceae family, API Staph - for the identification of staphylococci (API - Analytical Profile Index). Out of the total 190 processed samples, more than half of them were gram-negative bacteria, 105 strains (55.26%) and 85 strains (44.74%) were gram-positive bacteria. From the 190 samples collected from cows with endometritis, 95 strains of Escherichia coli (50%), 20 strains of Staphylococcus spp. (10.52%), 37 strains of Klebsiella spp. (19.47%) were isolated, 19 strains of Streptococcus spp. (10%) 19 strains of Pseudomonas spp. (10%). The isolated strains had similar behavior, being sensitive to enrofloxacin, florfenicol, gentamicin, and lincospectin at 94.44%. Most of the gram-positive bacterial strains showed sensitivity to ceftiofur, cefoxitin, and cefotaxime. Among gram-negative bacterial strains, resistance to amoxicillin / clavulanic acid was found to be 100%, penicillin at 77%, and ampicillin at 72%. Among the gram-positive bacterial strains, 78.57% penicillin and 71.43% amoxicillin/clavulanic acid resistance were found.
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Kumarasinghe, N. M. A., Charitha Thambiliyagodage, Madara Jayanetti i Heshan Liyanaarachchi. "Antibacterial Activity of Zn Decorated TiO2 Nanocomposites". W SLIIT International Conference on Advancements in Sciences and Humanities 2023. Faculty of Humanities and Sciences, SLIIT, 2023. http://dx.doi.org/10.54389/usor2577.

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Bacterial infections have a significant public health impact. Infections are caused by bacteria in animals, plants as well as humans. Pathogenic bacteria can produce toxins, which are chemical poisons that interfere with cell function such as digestion of normal human enzymes, evasion of infection-fighting white blood cells, and immune clearance. Antibiotic prophylaxis is used to prevent bacterial infection. Antibiotic resistance is one of the most serious concerns in world health. Antibacterial nanoparticles are one possible answer to antimicrobial resistance. These nanomaterials not only kill antibiotic-resistant bacteria through various modes of action but, they can also be employed in conjunction with existing clinically relevant antibiotics to help overcome antimicrobial resistance mechanisms. In this study, anodized titanium dioxide (TiO2 ) nanorods were treated hydrothermally with zinc oxide (ZnO) nanoparticles to give titanium (Ti) antibacterial properties. The antibacterial activity of synthesized samples was investigated by Agar Well Diffusion method at 40 mg/ml concentration, against gram negative Klebsiella pneumoniae. To determine the antibacterial activity, the diameter of the zone of inhibition was measured, and the resulting data were statistically analyzed. Zn/TiO2 nano particles were characterized by using X-ray diffraction (XRD) Analysis.
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Rammadan ABDUL, Fatima, Maysoon Kh.ABBAS, Batool Abd Al Ameer BAQER i Ihsan Ali RAHEEM. "ASSESSMENT OF THE LEVEL OF INTERLEUKIN- 10 AND INTERFERON -GAMMA IN CHILDREN INFECTED WITH AEROMONAS HEMOPHILIA ISOLATED FROM DIARRHEA". W VIII.International ScientificCongressofPure,AppliedandTechnological Sciences. Rimar Academy, 2023. http://dx.doi.org/10.47832/minarcongress8-4.

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Background: Aeromonas hydrophila causes bacterial diseases that lead to intestinal and extra-intestinal infections. Objective: The study aimed to isolate Aeromonas hydrophila from patients suffering diarrhea and evaluate the level of some interleukins in patients and compare them with control and resistance of bacteria to antibiotics. Materials and methods collect 150 stool samples of patients with diarrhea. Six isolates of A. hemophilia were isolated from children suffering diarrhea from some Baghdad hospitals. Results, all bacterial isolates were identified by the biochemical, cultural and microbial characteristics and confirmed by VITK2 System. The study showed the resistance of A. hemophilia to many antibiotics, the highest resistance to Tetracycline and Oxacillin and the rate of resistance was 100%, while the bacteria had the lowest resistance 16.7%, to to Erythromycin. The present results exhibited Interleukin-10 level was elevated in the patients and it was controlled by a significant level. The present results besides exhibited that the concentration of Interferon- gamma (IFN-γ) of patients and control group with nonsignificant difference
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Mukkisa, Hareesh, Lauren Crisman, Sarah Davis, Stacie Wood i Deborah Heyl. "Fighting Bacterial Resistance: Modifying the Antimicrobial Peptide Tachyplesin". W The Twenty-Third American and the Sixth International Peptide Symposium. Prompt Scientific Publishing, 2013. http://dx.doi.org/10.17952/23aps.2013.050.

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Rozman, Urška, Mojca Cimerman, Darja Duh i Sonja Šostar Turk. "Zmanjšana občutljivost bakterij proti razkužilom, izoliranih iz površin na oddelku za fizioterapijo". W Interdisciplinarity Counts. University of Maribor, University Press, 2023. http://dx.doi.org/10.18690/um.fov.3.2023.72.

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Disinfectants are intensively used in medical institutions to control and prevent health care associated infections. With constant use and exposure to disinfectants, bacteria can develop reduced sensitivity or even resistance. In our research, we isolated bacterial strains from reusable devices in physiotherapy departments in four different institutions from Slovenia and Austria. The minimum inhibitory concentration (MIC) of various disinfectant wipes used in institutions was determined for the isolated strains. The wipes contained the active substance alcohol or quaternary ammonium compounds (QAC). Average MIC values for alcohol-based disinfectants were between 66.61 and 148.82 g/l, for QAC-based disinfectants between 2.4 and 3.5 mg/L. We identified five different strains in which the MIC values were four times higher than the average values, which may indicate a reduced sensitivity of the bacteria against the tested disinfectants. Monitoring the reduction of susceptibility and/or resistance of bacteria to disinfectants is important, as this can also affect the emergence of bacterial resistance to antibiotics. It is important to take care of the prudent use of available and effective antimicrobial agents, and it is also absolutely necessary to establish clear criteria and protocols for monitoring the resistance of microorganisms to disinfectants.
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Gesierich, FJ, F. Hucke, S. Bota i M. Peck-Radosavljevic. "Bacterial spectrum and antibiotic resistance in patients with spontaneous bacterial peritonitis in southern Austria". W 54. Jahrestagung & 31. Fortbildungskurs der Österreichischen Gesellschaft für Gastroenterologie & Hepatologie – ÖGGH (Hybrid Veranstaltung). Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1734315.

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Treccani, Laura, i Kurosch Rezwan. "Microstructuring and Biofunctionalization of Alumina Surfaces to Enhance Abrasion Resistance and Suppress Bacterial Biofilm Growth". W ASME 2008 International Manufacturing Science and Engineering Conference collocated with the 3rd JSME/ASME International Conference on Materials and Processing. ASMEDC, 2008. http://dx.doi.org/10.1115/msec_icmp2008-72189.

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The design and fabrication of alumina microstructured surfaces that simultaneously present high mechanical and chemical features and do not suffer biofouling are here reported. An aerosol based patterning technique was employed to fabricate alumina microstructures directly on alumina surfaces with the aim to enhance wear and chemical resistance. Microstructured alumina surfaces were subsequently biofunctionalised with antibacterial biomolecules to inhibit bacterial adhesion. Lysozyme, an antibacterial enzyme commonly found in body secretions, was used as antibacterial agent and directly deposited onto microstructured alumina surfaces. Lysozyme-biofunctionalised microstructured alumina surfaces were tested at flow condition using abrasive particles and viable bacteria. The very preliminary results showed that alumina microstructures presented high resistance against mechanical abrasion and that bacterial biofilm formation could be suppressed. In particular alumina microstructures protected lysozyme molecules from desorption and loss of enzymatic activity. Such biofunctionalised microstructures present a promising system for fundamental research in the field of biomolecule adsorption on surfaces and maybe a feasible alternative e. g. to protect surfaces of water transport systems where abrasive particles and microorganisms are present.
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Raporty organizacyjne na temat "Bacterial resistance"

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Maltz, Lauren. Bacterial Enzymes and Antibiotic Resistance- Oral Presentation. Office of Scientific and Technical Information (OSTI), sierpień 2015. http://dx.doi.org/10.2172/1213182.

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Sessa, Guido, i Gregory Martin. A functional genomics approach to dissect resistance of tomato to bacterial spot disease. United States Department of Agriculture, styczeń 2004. http://dx.doi.org/10.32747/2004.7695876.bard.

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The research problem. Bacterial spot disease in tomato is of great economic importance worldwide and it is particularly severe in warm and moist areas affecting yield and quality of tomato fruits. Causal agent of spot disease is the Gram-negative bacterium Xanthomonas campestris pv. vesicatoria (Xcv), which can be a contaminant on tomato seeds, or survive in plant debris and in association with certain weeds. Despite the economic significance of spot disease, plant protection against Xcvby cultural practices and chemical control have so far proven unsuccessful. In addition, breeding for resistance to bacterial spot in tomato has been undermined by the genetic complexity of the available sources of resistance and by the multiple races of the pathogen. Genetic resistance to specific Xcvraces have been identified in tomato lines that develop a hypersensitive response and additional defense responses upon bacterial challenge. Central goals of this research were: 1. To identify plant genes involved in signaling and defense responses that result in the onset of resistance. 2. To characterize molecular properties and mode of action of bacterial proteins, which function as avirulence or virulence factors during the interaction between Xcvand resistant or susceptible tomato plants, respectively. Our main achievements during this research program are in three major areas: 1. Identification of differentially expressed genes during the resistance response of tomato to Xcvrace T3. A combination of suppression subtractive hybridization and microarray analysis identified a large set of tomato genes that are induced or repressed during the response of resistant plants to avirulent XcvT3 bacteria. These genes were grouped in clusters based on coordinate expression kinetics, and classified into over 20 functional classes. Among them we identified genes that are directly modulated by expression of the type III effector protein AvrXv3 and genes that are induced also during the tomato resistance response to Pseudomonas syringae pv. tomato. 2. Characterization of molecular and biochemical properties of the tomato LeMPK3MAP kinase. A detailed molecular and biochemical analysis was performed for LeMPK3 MAP kinase, which was among the genes induced by XcvT3 in resistant tomato plants. LeMPK3 was induced at the mRNA level by different pathogens, elicitors, and wounding, but not by defense-related plant hormones. Moreover, an induction of LeMPK3 kinase activity was observed in resistant tomato plants upon Xcvinfection. LeMPK3 was biochemically defined as a dual-specificity MAP kinase, and extensively characterized in vitro in terms of kinase activity, sites and mechanism of autophosphorylation, divalent cation preference, Kₘand Vₘₐₓ values for ATP. 3. Characteriztion of molecular properties of the Xcveffector protein AvrRxv. The avirulence gene avrRxvis involved in the genetic interaction that determines tomato resistance to Xcvrace T1. We found that AvrRxv functions inside the plant cell, localizes to the cytoplasm, and is sufficient to confer avirulence to virulent Xcvstrains. In addition, we showed that the AvrRxv cysteine protease catalytic core is essential for host recognition. Finally, insights into cellular processes activated by AvrRxv expression in resistant plants were obtained by microarray analysis of 8,600 tomato genes. Scientific and agricultural significance: The findings of these activities depict a comprehensive and detailed picture of cellular processes taking place during the onset of tomato resistance to Xcv. In this research, a large pool of genes, which may be involved in the control and execution of plant defense responses, was identified and the stage is set for the dissection of signaling pathways specifically triggered by Xcv.
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Aleksova, Michaella, Anelia Kenarova, Silvena Boteva i Galina Radeva. Induced Bacterial Antibiotic Resistance under the Application of Fungicide Azoxystrobin. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, grudzień 2019. http://dx.doi.org/10.7546/crabs.2019.11.18.

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Aronson, Arthur I. Function of Bacterial Spore Coat Polypeptides in Structure, Resistance and Germination. Fort Belvoir, VA: Defense Technical Information Center, grudzień 1990. http://dx.doi.org/10.21236/ada232217.

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Michel Jr., Frederick C., Harry A. J. Hoitink, Yitzhak Hadar i Dror Minz. Microbial Communities Active in Soil-Induced Systemic Plant Disease Resistance. United States Department of Agriculture, styczeń 2005. http://dx.doi.org/10.32747/2005.7586476.bard.

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Induced Systemic Resistance (ISR) is a highly variable property that can be induced by compost amendment of potting media and soils. For example, previous studies showed that only 1 of 79 potting mixes prepared with different batches of mature composts produced from several different types of solid wastes were able to suppress the severity of bacterial leaf spot of radish caused by Xanthomonas campestris pv. armoraciae compared with disease on plants produced in a nonamended sphagnum peat mix. In this project, microbial consortia in the rhizosphere of plants grown in ISR-active compost-amended substrates were characterized. The plants used included primarily cucumber but also tomato and radish. Rhizosphere microbial consortia were characterized using multiple molecular tools including DGGE (Israel) and T -RFLP (Ohio) in both ISR-active field plots and potting media. Universal as well as population-specific bacterial and fungal PCR primers were utilized. T -RFLP analyses using universal bacterial primers showed few significant differences in overall bacterial community composition in ISR-active and inactive substrates (Ohio). In addition, the community members which were significantly different varied when different ISR-activecomposts were used (Ohio). To better characterize the shifts in microbial community structure during the development of ISR, population specific molecular tools were developed (Israel, Ohio).-PCR primers were designed to detect and quantify bacterial groups including Pyrenomycetes, Bacillus, Pan toea, Pseudomonas, Xanthomonas and Streptomyces as well as Trichoderma and Fusarium; two groups of fungi that harbor isolates which are ISR active (Isreal and Ohio). Bacterial consortia associated with cucumber plants grown in compost-amended potting mixtures were shown to be dominated by the phylogenetic taxon Bacteroidetes, including members of the genus Chryseobacterium, which in some cases have been shown to be involved in biocontrol (Israel). Nested-PCR-DGGE analyses coupled with long l6S rDNA sequencing, demonstrated that the Chryseobacteriumspp. detected on seed and the root in compost-amended treatments were derived from the compost itself. The most effective ISR inducing rhizobacterial strains were identified as Bacillus sp. based on partial sequencing of l6S rDNA. However, these strains were significantly less effective in reducing the severity of disease than Trichoderma hamatum382 (T382). A procedure was developed for inoculation of a compost-amended substrate with T -382 which consistently induced ISR in cucumber against Phytophthora blight caused by Phytophthora capsiciand in radish against bacterial spot (Ohio). Inoculation of compost-amended potting mixes with biocontrol agents such as T -382 and other microbes that induce systemic resistance in plants significantly increased the frequency of systemic disease control obtained with natural compost amendments.
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Aleksova, Michaella. Azoxystrobin Impact on a Selection of Soil Bacterial Resistance to Amynoglicoside Antibiotics. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, październik 2019. http://dx.doi.org/10.7546/crabs.2019.10.08.

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Abdullah, Nassreen, Lorraine Scanlon i Deirdre O’Brien. Bacterial resistance mechanisms in urological infections and how they can be overcome. BJUI Knowledge, kwiecień 2020. http://dx.doi.org/10.18591/bjuik.0538.

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Zhao, Bingyu, Saul Burdman, Ronald Walcott i Gregory E. Welbaum. Control of Bacterial Fruit Blotch of Cucurbits Using the Maize Non-Host Disease Resistance Gene Rxo1. United States Department of Agriculture, wrzesień 2013. http://dx.doi.org/10.32747/2013.7699843.bard.

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The specific objectives of this BARD proposal were: (1) To determine whether Rxol can recognize AacavrRxo1 to trigger BFB disease resistance in stable transgenic watermelon plants. (2) To determine the distribution of Aac-avrRxo1 in a global population of Aae and to characterize the biological function of Aac-avrRxo1. (3) To characterize other TIS effectors of Aae and to identify plant R gene(s) that can recognize conserved TIS effectors of this pathogen. Background to the topic: Bacterial fruit blotch (BFB) of cucurbits, caused by Acidovorax avenae subsp. citrulli (Aae), is a devastating disease that affects watermelon (Citrullus lanatus) and melon (Cucumis melo) production worldwide, including both Israel and USA. Two major groups of Aae strains have been classified based on their virulence on host plants, genetics and biochemical properties. Thus far, no effective resistance genes have been identified from cucurbit germplasm. In this project, we assessed the applicability of a non-host disease resistance gene, Rxol, to control BFB in watermelon. We also tried to identify Aae type III secreted (TIS) effectors that can be used as molecular probes to identify novel disease resistance genes in both cucurbits and Nieotianatabaeum. Major conclusions, solutions, achievements: We generated five independent transgenic watermelon (cv. Sugar Babay) plants expressing the Rxol gene. The transgenic plants were evaluated with Aae strains AAC001 and M6 under growth chamber conditions. All transgenic plants were found to be susceptible to both Aae strains. It is possible that watermelon is missing other signaling components that are required for Rxol-mediated disease resistance. In order to screen for novel BFB resistance genes, we inoculated two Aae strains on 60 Nieotiana species. Our disease assay revealed Nicotiana tabaeum is completely resistant to Aae, while its wild relative N. benthamiana is susceptible to Aae. We further demonstrated that Nieotiana benthamiana can be used as a surrogate host for studying the mechanisms of pathogenesis of Aae. We cloned 11 TIS effector genes including the avrRxolhomologues from the genomes of 22 Aae strains collected worldwide. Sequencing analysis revealed that functional avrRxol is conserved in group" but not group I Aae strains. Three effector genes- Aave_1548, Aave_2166 and Aave_2708- possessed the ability to trigger an HR response in N. tabacum when they were transiently expressed by Agrobaeterium. We conclude that N. tabacum carries at least three different non-host resistance genes that can specifically recognize AaeTIS effectors to trigger non-host resistance. Screening 522 cucurbits genotypes with two Aae strains led us to identify two germplasm (P1536473 and P1273650) that are partially resistant to Aae. Interestingly, transient expression of the TIS effector, Aave_1548, in the two germplasms also triggered HR-Iike cell death, which suggests the two lines may carry disease resistance genes that can recognize Aave_1548. Importantly, we also demonstrated that this effector contributes to the virulence of the bacterium in susceptible plants. Therefore, R genes that recognize effector Aave1548 have great potential for breeding for BFB resistance. To better understand the genome diversity of Aae strains, we generated a draft genome sequence of the Israeli Aae strain, M6 (Group I) using Iliumina technology. Comparative analysis of whole genomes of AAC001, and M6 allowed us to identify several effectors genes that differentiate groups I and II. Implications, both scientific and agricultural: The diversity of TIS effectors in group I and II strains of Aae suggests that a subset of effectors could contribute to the host range of group I and II Aae strains. Analysis of these key effectors in a larger Aae population may allow us to predict which cucurbit hosts may be at risk to BFB. Additionally, isolation of tobacco and cucurbit Rgenes that can recognize Aae type III effectors may offer new genetic resources for controlling BFB.
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Maltz, Lauren. Structural Studies of Bacterial Enzymes and their Relation to Antibiotic Resistance Mechanisms - Final Paper. Office of Scientific and Technical Information (OSTI), sierpień 2015. http://dx.doi.org/10.2172/1213135.

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Maltz, Lauren. Structural Studies of Bacterial Enzymes and their Relation to Antibiotic Resistance Mechanisms - General Abstract. Office of Scientific and Technical Information (OSTI), sierpień 2015. http://dx.doi.org/10.2172/1213137.

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