Gotowe bibliografie tematyczne / Autonomic changes

Gotowa bibliografia na temat „Autonomic changes”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 19 lutego 2022

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Artykuły w czasopismach na temat "Autonomic changes"

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Delamont, Robert S., i Matthew C. Walker. "Pre-ictal autonomic changes". Epilepsy Research 97, nr 3 (grudzień 2011): 267–72. http://dx.doi.org/10.1016/j.eplepsyres.2011.10.016.

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Jin, Heung Yong, Hong Sun Baek i Tae Sun Park. "Morphologic Changes in Autonomic Nerves in Diabetic Autonomic Neuropathy". Diabetes & Metabolism Journal 39, nr 6 (2015): 461. http://dx.doi.org/10.4093/dmj.2015.39.6.461.

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Heydarpour, F. "SODIUM CHANGES AUTONOMIC NERVOUS RESPONSE". Journal of Hypertension 22, Suppl. 2 (czerwiec 2004): S210. http://dx.doi.org/10.1097/00004872-200406002-00733.

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Cook, Glen A. "Toenail changes in autonomic neuropathy". Clinical Autonomic Research 28, nr 4 (19.06.2018): 437–38. http://dx.doi.org/10.1007/s10286-018-0538-7.

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Igamova, Saodat, i Aziza Djurabekova. "CHANGES OF PSYCHOMOTOR DEVELOPMENT IN CHILDREN WITH PERINATAL BRAIN HYPOXIA". JOURNAL OF NEUROLOGY AND NEUROSURGICAL RESEARCH 3, nr 1 (30.03.2020): 35–37. http://dx.doi.org/10.26739/2181-0982-2020-3-6.

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The central nervous system is the main mechanism that determines the nature of the reactivity and adaptation of the body to a set of environmental factors. At the same time, autonomic imbalance acts as the root cause of the pathological process or asa predisposing factor, and it is advisable to use the cardiovascular system as an indicator of neurohumoral regulationits reactions are associated with the activity of the central nervous system, autonomic nervous system and subcortical centers
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De Caprio, Lorenzo, Marco Papa, Domenico Acanfora, Carlo Vigorito, Vincenzo Canonico, Pasquale Abete, Sergio Cuomo, Fernando Gallucci i Franco Rengo. "Autonomic Tone Changes during Isometric Exercise". American Journal of Noninvasive Cardiology 3, nr 1 (1989): 58–63. http://dx.doi.org/10.1159/000470584.

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Reinsberger, Claus, Rani Sarkis, Christos Papadelis, Chiran Doshi, David L. Perez, Gaston Baslet, Tobias Loddenkemper i Barbara A. Dworetzky. "Autonomic Changes in Psychogenic Nonepileptic Seizures". Clinical EEG and Neuroscience 46, nr 1 (styczeń 2015): 16–25. http://dx.doi.org/10.1177/1550059414567739.

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Moseley, Brian D. "Seizure-Related Autonomic Changes in Children". Journal of Clinical Neurophysiology 32, nr 1 (luty 2015): 5–9. http://dx.doi.org/10.1097/wnp.0000000000000138.

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Yun, WH, SW Min, J. Huh, YJ Ro i CS Kim. "Autonomic Changes in Preoperative Uncomplicated Diabetic Patients with Postural Changes". Journal of International Medical Research 38, nr 5 (październik 2010): 1764–71. http://dx.doi.org/10.1177/147323001003800522.

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Schultz, Jordan L., i Peg C. Nopoulos. "Autonomic Changes in Juvenile-Onset Huntington’s Disease". Brain Sciences 10, nr 9 (26.08.2020): 589. http://dx.doi.org/10.3390/brainsci10090589.

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Patients with adult-onset Huntington’s Disease (AOHD) have been found to have dysfunction of the autonomic nervous system that is thought to be secondary to neurodegeneration causing dysfunction of the brain–heart axis. However, this relationship has not been investigated in patients with juvenile-onset HD (JOHD). The aim of this study was to compare simple physiologic measures between patients with JOHD (n = 27 participants with 64 visits) and participants without the gene expansion that causes HD (GNE group; n = 259 participants with 395 visits). Using data from the Kids-JOHD study, we compared mean resting heart rate (rHR), systolic blood pressure (SBP), and diastolic blood pressure (DBP) between the JOHD and GNE groups. We also divided the JOHD group into those with childhood-onset JOHD (motor diagnosis received before the age of 13, [n = 16]) and those with adolescent-onset JOHD (motor diagnosis received at or after the age of 13 [n = 11]). We used linear mixed-effects models to compare the group means while controlling for age, sex, and parental socioeconomic status and including a random effect per participant and family. For the primary analysis, we found that the JOHD group had significant increases in their rHR compared to the GNE group. Conversely, the JOHD group had significantly lower SBP compared to the GNE group. The JOHD group also had lower DBP compared to the GNE group, but the results did not reach significance. SBP and DBP decreased as disease duration of JOHD increased, but rHR did not continue to increase. Resting heart rate is more sensitive to changes in autonomic function as compared to SBP. Therefore, these results seem to indicate that early neurodegenerative changes of the central autonomic network likely lead to an increase in rHR while later progression of JOHD leads to changes in blood pressure. We hypothesize that these later changes in blood pressure are secondary to neurodegeneration in brainstem regions such as the medulla.
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Rozprawy doktorskie na temat "Autonomic changes"

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Nosakhare, Ehimwenma. "QT-interval adaptation to changes in autonomic balance". Thesis, Massachusetts Institute of Technology, 2013. http://hdl.handle.net/1721.1/84865.

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Thesis (S.M.)--Massachusetts Institute of Technology, Dept. of Electrical Engineering and Computer Science, 2013.
Cataloged from PDF version of thesis.
Includes bibliographical references (pages 93-98).
ECG variability, as it relates to the influence of the autonomic nervous system on the heart, is primarily studied via frequency-domain and time-domain analysis of heart rate variability (HRV). HRV studies the variability of the RR intervals in the ECG; these intervals are modulated by the autonomic influence on the periodicity of the the heart's pacemaker, the sino-atrial node. The autonomic influence at this level is dominated by the parasympathetic nervous system. In order to have a robust assessment of autonomic balance, there is a need for an ECG-based approach to assess the influence of the sympathetic nervous system. In this thesis, using spectral analysis, we quantify the variability of the QT interval, which is primarily modulated by the sympathetic nervous system. We also estimate the time constant of the sympathetic nervous system by least-squares fitting of the QT time series resulting from step perturbations in autonomic balance. This study is carried out on graded head-up tilt test data. Our results demonstrate the potential of QT interval variability as a non-invasive assessment of the sympathetic nervous system activity on the heart.
by Ehimwenma Nosakhare.
S.M.
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Lamensdorf, Angela Mona-Lisa. "Cardiovascular risk and autonomic changes during high and low affect provocations". Thesis, University of British Columbia, 1988. http://hdl.handle.net/2429/28098.

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Does having a positive family history of essential hypertension predispose one to greater cardiovascular reactivity? Could reactivity be assessed with stress tasks that have greater external validity than traditional laboratory stressors? To answer these questions? 2b subjects with parental history of essential hypertension and 3b subjects without) were induced to converse with an experimenter on (a) a neutral topic (the weather)? and (b) an affective topic (a frustrating person or event). The topics were selected from a Iist of 2b because they had been rated by undergraduates as being the least and most arousing topics to talk about with a stranger in an experimental situation. The ratings yielded no interactions of sex of experimenter with sex of the subject. Subjects also performed a mental arithmetic task which is a standard laboratory stressor. The order of task presentation was randomly assigned within groups but matched across groups and sex to control sequence effects. For each subject? a 15-minute base I ine period was al lowed before each task. Readings of blood pressure? heart rate and rate of respiration were made at minute one? three? and five of each task phase. Each conversation task consisted of five minutes of talking followed by Iistening for five minutes to the experimenter. The tasks were separated by five-minute intervals to allow return to baseline levels. Results indicated that compared to individuals without parental history of hyper tension? individuals with parental history displayed higher levels of blood pressure (but not heart rate and rate of respiration) whether talking or listening. When peak values were considered; positive parental history subjects showed greater reactivity to the affective topic on diastolic blood pressure. The results also indicated that the three kinds of stressors yielded different levels of physiological responses with the math task and talk phase of the affect task yielding higher levels of blood pressure and heart rate than talk about the weather. The difference between the math and affective tasks was not significant on systolic blood pressure? but math yielded higher responses on heart rate and lower responses on diastolic blood pressure than talking about a frustrating event or person. These results suggest that a more generalizabIe stress stimulus such as an affect-laden conversation? can be reasonably standardized across subjects and elicits an aIpha-adrenergic vaso-constrictive response? a response more readily given by individuals with positive parental history than individuals without. The results also suggest that individuals with positive parental history of hypertension have higher blood pressure levels than individuals without. With respect to the similarity of the findings of this study? with those of other studies which have used older populations? it is proposed that these results are generalizable to older populations and provide evidence that a positive family history of essential hypertension may be considered a risk factor for later cardiovascular disease.
Arts, Faculty of
Psychology, Department of
Graduate
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Roshan, Moniri Nazanin. "Vascular changes in spinal cord injured animals with repetitive episodes of autonomic dysreflexia". Thesis, University of British Columbia, 2012. http://hdl.handle.net/2429/43531.

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Spinal cord injury (SCI) is a devastating condition that not only leads to paralysis, but also causes dramatic changes in cardiovascular function. Individuals with cervical or high thoracic SCI commonly suffer from a life threatening condition known as autonomic dysreflexia (AD). AD is characterized by episodic hypertension─ an exaggerated sympathetic response triggered by irritating stimulus below the level of injury e.g. distended bladder. As a lifespan of SCI patients increases, cardiovascular-related illnesses become more prevalent. Recent studies suggest marked vascular dysfunction within the critical splanchnic vascular bed. Mesenteric arteries from rats with chronic high-thoracic SCI are hypersensitive to the α₁-adrenoceptor agonist PE. The hypersensitivity of splanchnic vascular bed in response to PE develops over time after SCI and may contribute to the development of AD. In this dissertation, I examined the morphological changes in peripheral vasculature following repetitive episodes of AD in animals with high SCI. I hypothesized that recurrent episodes of AD will trigger an inward eutrophic remodeling in peripheral resistance arteries of SCI rats. In this study, male Wistar rats with complete spinal cord transection at third (T3) thoracic segment were utilized. At 2 weeks after the injury, AD was induced in rats with T3 SCI using CRD. 4 weeks following injury superior mesenteric (SMA) arteries and primary branches (PMA) were collected from T3 SCI-only, T3+CRD and control uninjured rats. Morphological characteristics such as media thickness, lumen diameter, wall-to-lumen ratio and wall cross sectional area (CSA) of the arteries were evaluated. Results suggest that AD induced through CRD lead to structural remodeling of PMAs, but no changes were observed in SMAs of CRD group. Media thickness, wall-to-lumen ratio significantly increased in PMAs of CRD group; lumen diameter and CSA of PMAs in CRD did not change when compared to T3 SCI-only and uninjured groups. The data support eutrophic (no change in CSA) remodeling of PMAs in CRD group, but failed to show a reduction in lumen diameter (inward changes) of these arteries. The findings of the study highlight the underlying effect of AD on structural remodeling of vasculature following an injury.
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Zheng, Mei Mu Zi (Annie). "Above and below : changes in conduit artery after spinal cord injury, autonomic dysreflexia, and passive exercise". Thesis, University of British Columbia, 2016. http://hdl.handle.net/2429/59093.

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Spinal cord injury (SCI) is a devastating condition that not only results in motor and sensory loss, but also autonomic dysfunctions. Individuals with SCI experience a 3-4 fold increased risk of cardiovascular disease (CVD), the leading cause of mortality in this population. Endothelial dysfunction is among the earliest markers of CVD progression. This thesis aims to: 1) clarify previous reports showing a counterintuitive improvement in endothelial function after SCI, 2) examine the effect of autonomic dysreflexia (AD) on conduit vasculature, and 3) assess the efficacy of passive exercise (PE) to reverse vascular dysfunction. In uninjured controls (CON), T3-complete spinal cord transected Wistar rats (SCI), T3-transected with induced AD by colorectal distension (SCI+CRD), and T3-transected with PE (SCI+PE), we assessed endothelium-dependent vasodilation and specific mechanisms for relaxation in brachial (BA) and femoral artery (FA) using wire-myography. Sympathetic innervation, mechanotransducer expression [transient receptor potential channel V4 (TRPV4)], arterial morphology, and profibrotic markers were assessed using immunohistochemistry. Impaired reactivity to acetylcholine was seen in FA after SCI via decreased contribution of endothelium dependent hyperpolarizing factor (EDHF) mediated pathways, while BA showed preserved endothelial function. Moreover, FA in SCI exhibited inward remodelling, 37.7% less sympathetic nerve fiber density, and increased collagen I expression (53.0%). Chronic repetitive AD resulted in a shift in vasodilatory mechanisms away from nitric oxide and towards EDHF, hypersensitivity to phenylephrine, and reduced elastin expression (13.9%). Passive hind-limb exercise after SCI led to improved sensitivity of FA to acetylcholine, through an increase in TRPV4 and prostacyclin-mediated pathways for vasodilation. Outward remodelling, as well as decreased expression of transforming growth factor beta (47.7%) and collagen I (39.0%) was seen in FA after PE. We have shown, for the first time, the expected endothelial dysfunction in the inactive/supraspinally disrupted FA after SCI and that chronic repetitive AD resulted in exacerbation of vascular dysfunction caudal to injury. Furthermore, PE was effective in reversing endothelial dysfunction and provided atheroprotective benefit, indicating PE may be a viable therapeutic intervention for preventing CVD after SCI. The observed changes provide insight into the mechanisms of endothelial dysfunction and possible directions on improvement of vascular health after SCI.
Medicine, Faculty of
Graduate
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Reeder, Matthew, i res cand@acu edu au. "The Emotional Congruence of Experience and Bodily Change". Australian Catholic University. School of Psychology, 2001. http://dlibrary.acu.edu.au/digitaltheses/public/adt-acuvp10.09042006.

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This study examined the association of the experience of emotion and somatic changes. The study compared reported somatic changes generally experienced when anxious with the actual association of the experience of emotion and somatic changes as measured during a specific event. Emotions were measured as both general negative emotion as well as specific emotions: anger, disgust, fear, sadness and shame. Participants were volunteers from a Victorian university who agreed to watch a video depicting the dramatisation of child abuse. Throughout the video, participants indicated their experience of emotion. Measures were also taken throughout the procedure of facial expression and Galvanic Skin Response (GSR). In order to examine emotional-congruence, subjects were divided into three groups. These groups were divided according to the congruence of subjects’ experienced emotion with autonomic changes and facial expressivity. Groups were divided separately for each of the emotion types. Where there was little difference between the reported experience of emotion and that, which would have been expected from the observed somatic changes, the subject was deemed to be in the Congruent Group. Subjects whose reported experience of emotion was greater or less than would be expected from observed somatic changes were allocated to the Over-reporter and Under-Reporter Groups respectively. This data was then compared to participants’ reports of the number of somatic symptoms usually experienced when anxious. It was found that participants who under-report the experience of general negative-emotion compared with their observed somatic changes (both GSR and facial expressivity) had lower trait-somatic-anxiety (reported fewer somatic symptoms usually experienced when anxious). There was no significant difference between the Congruent Group and Over-Reporter Group. The Under-Reporter Groups had significantly lower trait-somatic-anxiety than the Congruent Group when emotional-congruence was defined by fear and GSR, anger and GSR and sadness and facial expressivity. The actual association of shame and disgust with either somatic change, sadness with autonomic change and anger and fear with facial expressivity was unrelated to the number of somatic symptoms reported to be usually experienced when anxious. The results supported the idea that subjective reports of the number of somatic symptoms reported to be usually experienced when anxious reflect the actual association of somatic change and experience, but with limitations. The actual association of experience of fear with autonomic change seems to reflect the number of somatic symptoms reported to be usually experienced when anxious more than other emotions. Further for those for whom the experience of anger and negative-emotion has a greater association with somatic change, there was a greater number of somatic symptoms reported to be usually experienced when anxious. This would suggest that some people have a greater association of some experiences of emotion and somatic change. Furthermore, while there is an association between reported somatic changes generally experienced when anxious with the actual association of the experience of emotion and somatic changes as measured during a specific event, this was dependant on the association of the emotion types rather than being generalised for all emotions.
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Shiraiwa, Keigo. "Changes in electroencephalography and cardiac autonomic function during craft activities : experimental evidence for the effectiveness of occupational therapy". Doctoral thesis, Kyoto University, 2021. http://hdl.handle.net/2433/263589.

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Jokinen, V. (Vesa). "Longitudinal changes and prognostic significance of cardiovascular autonomic regulation assessed by heart rate variability and analysis of non-linear heart rate dynamics". Doctoral thesis, University of Oulu, 2003. http://urn.fi/urn:isbn:9514272005.

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Abstract Several studies have shown that altered cardiovascular autonomic regulation is associated with hypertension, diabetes, aging, angiographic severity of coronary artery disease (CAD), and increased mortality after acute myocardial infarction (AMI). The purpose of this study was to assess the temporal changes and prognostic significance of various measures of heart rate (HR) behaviour and their possible associations to coronary risk variables, and the progression of CAD in different populations. This study comprised five patient populations. The first consisted of 305 patients with recent coronary artery bypass graft surgery (CABG) and lipid abnormalities, the second of 109 male patients with recent CABG, the third of 53 type II diabetic patients with CAD, the fourth of 600 patients with recent AMI, and the fifth of 41 elderly subjects. HR variability and non-linear measures of HR dynamics were analysed. Among the patients with prior CABG, a significant correlation existed between the baseline HR variability (standard deviation of N-N intervals, SDNN) and the progression of CAD (r = 0.26, p < 0.001)). In the longitudinal study of patients with prior CABG, only the fractal indexes of HR dynamics, such as the power law slope (β) and the short-term fractal exponent (α1), decreased significantly. In diabetic patients, SDNN decreased significantly (p < 0.001) during the three-year period. The reduction of SDNN was related to cholesterol, triglyceride, and glucose levels, and also to progression of CAD (r = 0.36, p < 0.01). In the longitudinal follow-up study of patients with recent AMI, reduced fractal indices (α1 and β), and reduced HR turbulence predicted cardiac death when measured at the convalescent phase after AMI. Reduced β and turbulence slope predicted cardiac death when measured at 12 months after AMI. In the elderly population, β (p < 0.001) and α1 (p < 0.01) reduced significantly. Low-frequency power spectra were the only traditional measure of HR variability that decreased significantly during the 16-year period. HR variability is associated with many risk factors of atherosclerosis and with progression of CAD among patients with ischemic heart disease. Fractal HR dynamics are more sensitively able to detect age-related changes in cardiovascular autonomic regulation. Altered fractal HR dynamics and HR turbulence are associated with increased mortality after AMI.
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Cassam, Aly K. "Characterization of changes in dopamine ß-hydroxylase, neuropeptide Y, substance P and growth associated protein-43 expression in spinal autonomic nuclei after spinal cord injury". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp04/mq21083.pdf.

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Zhang, Hongling. "Sigma Receptors Modulation of Voltage-gated Ion Channels in Rat Autonomic Neurons". [Tampa, Fla.] : University of South Florida, 2005. http://purl.fcla.edu/fcla/etd/SFE0001183.

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Krishnan, Binu. "Hypothalamic and autonomic responses to change in glucose homeostasis". Thesis, Exeter and Plymouth Peninsula Medical School, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.544012.

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Książki na temat "Autonomic changes"

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Autonomía y transformación universitaria. [Caracas?]: Fondo Editorial Tropykos, 2011.

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Pechriggl, Alice. Utopiefähigkeit und Veränderung: Der Zeitbegriff und die Möglichkeit kollektiver Autonomie. Pfaffenweiler: Centaurus-Verlagsgesellschaft, 1993.

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Miller, E. J. From dependency to autonomy: Studies in organization and change. London: Free Association Books, 1993.

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Bottani, Norberto. Insegnanti al timone?: Fatti e parole dell'autonomia scolastica. Bologna: Il Mulino, 2002.

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editor, Fehmel Thilo, Lessenich Stephan editor i Preunkert Jenny editor, red. Systemzwang und Akteurswissen: Theorie und Empirie von Autonomiegewinnen. Frankfurt: Campus Verlag, 2014.

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Zlatkin-Troitschanskaia, Olga. Steuerbarkeit von Bildungssystemen mittels politischer Reformstrategien: Interdisziplinäre theoretische Analyse und empirische Studie zur Erweiterung der Autonomie im öffentlichen Schulwesen. Frankfurt am Main: Lang, 2006.

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Northern Ireland: What is it? : Professor Mansergh changes his mind. Belfast: Belfast Magazine, 2011.

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Inghilleri, P. From subjective experience to cultural change. New York: Cambridge University Press, 1999.

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Millán, Patricio. Reformas educacionais e autonomia das escolas: Os casos da cidade de Nova Iorque, do Chile e do Estado de Minas Gerais. [Brasília, Brazil]: Banco Mundial, Departamento de Desenvolvimento Humano, Diretoria do Brasil, 1999.

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Hroch, Miroslav. Comparative studies in modern European history: Nation, nationalism, social change. Aldershot: Ashgate Variorum, 2007.

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Części książek na temat "Autonomic changes"

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Romigi, Andrea, i Nicola Toschi. "Cardiac Autonomic Changes in Epilepsy". W Complexity and Nonlinearity in Cardiovascular Signals, 375–86. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-58709-7_14.

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Pezzella, Francesca Romana, Sabrina Anticoli i Claudio Pozzessere. "Muscle, Peripheral Nerve and Autonomic Changes". W Frontiers of Neurology and Neuroscience, 79–82. Basel: KARGER, 2012. http://dx.doi.org/10.1159/000333421.

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Borel, C., i G. Hamann. "Autonomic Changes in Acute Cerebrovascular Disorders". W Update in Intensive Care and Emergency Medicine, 24–32. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-60264-1_4.

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Amici, Roberto, i Giovanna Zoccoli. "Physiological Changes in the Autonomic Nervous System During Sleep". W Autonomic Nervous System and Sleep, 43–50. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-62263-3_5.

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Steele, Andrew M. "Developmental Changes in Neural Control of Respiration". W Developmental Neurobiology of the Autonomic Nervous System, 327–401. Totowa, NJ: Humana Press, 1986. http://dx.doi.org/10.1007/978-1-59259-459-7_12.

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Ford, G. A. "Clinical and Therapeutic Implications of Aging Changes in Autonomic Function". W Autonomic Nervous System in Old Age, 24–31. Basel: KARGER, 2004. http://dx.doi.org/10.1159/000077922.

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Attavar, P., i D. I. Silverman. "Normal and Pathological Changes in Cardiovascular Autonomic Function with Age". W Autonomic Nervous System in Old Age, 32–44. Basel: KARGER, 2004. http://dx.doi.org/10.1159/000077923.

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McDonald, D. M., i A. Haskell. "Ultrastructural Changes Induced in Rat Carotid Bodies and Superior Laryngeal Nerve Paraganglia by Chronic Hypoxia". W Histochemistry and Cell Biology of Autonomic Neurons and Paraganglia, 260–65. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-72749-8_45.

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Apura, João, Jorge Tiago, A. Sequeira i L. B. Rosário. "Numerical modeling of cardiovascular physiology Study of dynamic changes during autonomic reflexes". W EMBEC & NBC 2017, 278–81. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-5122-7_70.

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Victor, Ronald G. "Autonomic Changes during the Acute Phase of Acute Myocardial Infarction and Ischemia". W Acute Coronary Care 1986, 3–18. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2633-5_1.

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Streszczenia konferencji na temat "Autonomic changes"

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Zhang, Xiangliang, Cecile Germain i Michele Sebag. "Adaptively detecting changes in Autonomic Grid Computing". W 2010 11th IEEE/ACM International Conference on Grid Computing (GRID). IEEE, 2010. http://dx.doi.org/10.1109/grid.2010.5698017.

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Johnson, Kristina T., Sara Taylor, Szymon Fedor, Natasha Jaques, Weixuan Chen i Rosalind W. Picard. "Vomit Comet Physiology: Autonomic Changes in Novice Flyers". W 2018 40th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2018. http://dx.doi.org/10.1109/embc.2018.8512414.

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Bahati, Raphael M., i Michael A. Bauer. "Adapting to Run-Time Changes in Policies Driving Autonomic Management". W 2008 Fourth International Conference on Autonomic and Autonomous Systems (ICAS). IEEE, 2008. http://dx.doi.org/10.1109/icas.2008.47.

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Toriumi, Kyouhei, i Yutaka Fukuoka. "Changes in Autonomic Nervous System and Axillary Temperature Caused by Electric Potential Therapy". W 2020 42nd Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC) in conjunction with the 43rd Annual Conference of the Canadian Medical and Biological Engineering Society. IEEE, 2020. http://dx.doi.org/10.1109/embc44109.2020.9176319.

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Sollers III, John J., Yoshiharu Yonezawa, Rebecca A. Silver, Marcellus M. Merritt i Julian F. Thayer. "An ambulatory recording system for the assessment of autonomic changes across multiple days". W Defense and Security, redaktorzy John A. Caldwell i Nancy Jo Wesensten. SPIE, 2005. http://dx.doi.org/10.1117/12.604413.

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Shinar, Z., A. Baharav i S. Akselrod. "Changes in autonomic nervous system activity and in electro-cortical activity during sleep onset". W Computers in Cardiology, 2003. IEEE, 2003. http://dx.doi.org/10.1109/cic.2003.1291151.

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Llamedo Soria, M., P. D. Arini, M. R. Risk i P. Laguna. "Effect of QT interval correction during autonomic blockade in combination with changes in posture". W Computers in Cardiology, 2005. IEEE, 2005. http://dx.doi.org/10.1109/cic.2005.1588152.

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Seppanen, Tiina M., Olli-Pekka Alho i Tapio Seppanen. "Concomitant dynamic changes in autonomic nervous system function and nasal airflow resistance during allergen provocation". W 2015 37th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2015. http://dx.doi.org/10.1109/embc.2015.7319107.

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Themelis, Kristy, Robyn Stocks, Patrick Tano, Zdenka Cipinova, Lorraine Shah-Goodwin, Andrew Barritt, Hugo Critchley, Kevin Davies i Jessica Eccles. "AB0907 AUTONOMIC AND INFLAMMATORY CHANGES IN FM AND ME/CFS AND THE CONTRIBUTION TO SIGNS AND SYMPTOMS". W Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.1917.

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Makowiec, Danuta, i Zbigniew R. Struzik. "Visualization of age-dependent circadian changes in autonomic drive on heart rhythm by network representation of RR-increments". W 2015 Computing in Cardiology Conference (CinC). IEEE, 2015. http://dx.doi.org/10.1109/cic.2015.7411087.

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Raporty organizacyjne na temat "Autonomic changes"

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Bankole, Akinrinola, Lisa Remez, Onikepe Owolabi, Jesse Philbin i Patrice Williams. From Unsafe to Safe Abortion in Sub-Saharan Africa: Slow but Steady Progress. Guttmacher Institute, grudzień 2020. http://dx.doi.org/10.1363/2020.32446.

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This report represents the first comprehensive compilation of information about abortion in Sub-Saharan Africa and its four subregions. It offers a panorama of this hard-to-measure practice by assembling data on the incidence and safety of abortion, the extent to which the region’s laws restrict abortion, and how these laws have changed between 2000 and 2019. Many countries in this region have incrementally broadened the legal grounds for abortion, improved the safety of abortions, and increased the quality and reach of postabortion care. There is still much progress to be made, however, including enabling the region’s women to avoid unintended pregnancies and unsafe abortions. The report concludes with recommendations for a broad range of actors to improve the sexual and reproductive health and autonomy of the region’s 255 million women of reproductive age.
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Aiginger, Karl, Andreas Reinstaller, Michael Böheim, Rahel Falk, Michael Peneder, Susanne Sieber, Jürgen Janger i in. Evaluation of Government Funding in RTDI from a Systems Perspective in Austria. Synthesis Report. WIFO, Austria, sierpień 2009. http://dx.doi.org/10.22163/fteval.2009.504.

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In the spring of 2008, WIFO, KMU Forschung Austria, Prognos AG in Germany and convelop were jointly commissioned by the Austrian Federal Ministry for Transport, Innovation and Technology and the Austrian Federal Ministry of Economy, Family and Youth to perform a systems evaluation of the country's research promotion and funding activities. Based on their findings, six recommendations were developed for a change in Austrian RTDI policy as outlined below: 1. to move from a narrow to a broader approach in RTDI policy (links to education policy, consideration of the framework for innovation such as competition, international perspectives and mobility); 2. to move from an imitation to a frontrunner strategy (striving for excellence and market leadership in niche and high-quality segments, increasing market shares in advanced sectors and technology fields, and operating in segments of relevance for society); 3. to move from a fragmented approach to public intervention to a more coordinated and consistent approach(explicit economic goals, internal and external challenges and reasoning for public intervention); 4. to move from a multiplicity of narrowly defined funding programmes to a flexible, dynamic policy that uses a broader definition of its tasks and priorities (key technology and research segments as priority-action fields, adequate financing of clusters and centres of excellence); 5. to move from an unclear to a precisely defined allocation of responsibilities between ministries and other players in the field (high-ranking steering group at government level, monitoring by a Science, Research and Innovation Council); 6. to move from red-tape-bound to a modern management of public intervention (institutional separation between ministries formulating policies and agencies executing them, e.g., by "progressive autonomy").
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