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Książki na temat „Autoimmune diseases – Animal models”

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Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 31 stycznia 2023

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1

The use of SCID mice in the investigation of human autoimmune disease. Austin: R.G. Landes, 1994.

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2

Animal models for retinal diseases. [Totowa, N.J.]: Humana Press, 2010.

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3

Chan, Chi-Chao, red. Animal Models of Ophthalmic Diseases. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-19434-9.

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4

Pang, Iok-Hou, i Abbot F. Clark, red. Animal Models for Retinal Diseases. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-541-5.

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5

J, Pfeiffer Carl, red. Animal models for intestinal disease. Boca Raton, Fla: CRC Press, 1985.

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6

Animal models of human disease. Amsterdam: Elsevier/AP, 2011.

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7

Gross, David R. Animal models in cardiovascular research. Wyd. 3. Dordrecht: Springer, 2009.

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8

S, Chan Lawrence, red. Animal models of human inflammatory skin diseases. Boca Raton: CRC Press, 2004.

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9

Animal models in cardiovascular research. Boston: Nijhoff, 1985.

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10

Animal models in cardiovascular research. Wyd. 2. Dordrecht: Kluwer Academic, 1994.

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11

F, Sima Anders A., i Shafrir Eleazar, red. Animal models of diabetes: A primer. Amsterdam: Harwood Academic, 2001.

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12

O, Cantor Jerome, red. CRC handbook of animal models of pulmonary disease. Boca Raton, Fla: CRC Press, 1989.

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Casadesus, Gemma. Handbook of animal models in Alzheimer's disease. Amsterdam: IOS Press, 2011.

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14

V, Kalueff Allan, i LaPorte Justin L, red. Experimental animal models in neurobehavioral research. New York: Nova Science Publishers, 2008.

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15

1943-, Greenwald Robert A., i Diamond Herbert S. 1938-, red. CRC handbook of animal models for the rheumatic diseases. Boca Raton, Fla: CRC Press, 1988.

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16

Models for biomedical research: A new perspective. Washington, D.C: National Academy Press, 1985.

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17

International Association of Biomedical Gerontology. International Congress. Molecular mechanisms and models of aging. Boston: Blackwell Pub. on behalf of the New York Academy of Sciences, 2007.

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18

Animal models for stem cell therapy. [New York]: Humana Press, 2014.

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19

Biodefense: Research methodology and animal models. Wyd. 2. Boca Raton: Taylor & Francis, 2012.

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20

R, Swearengen James, red. Biodefense: Research methodology and animal models. Boca Raton, Fla: Taylor & Francis, 2006.

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21

Smith, Cynthia Petrie. Animal models of disease: January 1981 - July 1992. Beltsville, Md: National Agricultural Library, 1992.

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22

Cohen, Irun R., i Ariel Miller. Autoimmune Disease Models. Elsevier Science & Technology Books, 2014.

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23

R, Cohen Irun, i Miller Ariel, red. Autoimmune disease models: A guidebook. San Diego: Academic Press, 1994.

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24

Masahisa, Kyōgoku, i Wigzell Hans 1938-, red. New horizons in animal models for autoimmune disease. Tokyo: Academic Press, 1987.

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25

1926-, Volpé Robert, red. Autoimmunity and endocrine disease. New York: Dekker, 1985.

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26

K, Abbas Abul, Serono Symposia USA i International Symposium on Genetic Models of Immune and Inflammatory Diseases (1994 : Savannah, Ga.), red. Genetic models of immune and inflammatory diseases. New York: Springer, 1996.

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27

Genetic Models of Immune and Inflammatory Diseases. Springer, 2011.

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28

1937-, Cruse Julius M., i Lewis R. E. 1947-, red. Cellular aspects of autoimmunity. Basel: Karger, 1988.

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29

Yang, Hai-Tao. Genetic Analysis of Autoimmune Diseases Using Animal Models: Mapping Susceptibility Genes for Multiple Sclerosis and Rheumatoid Arthritis (Comprehensive Summaries of Uppsala Dissertations, 927). Uppsala Universitet, 2001.

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30

L, Adorini, red. Immunointervention in autoimmunity by Th1/Th2 regulation. Austin, Tex: Landes Bioscience, 1997.

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31

(Editor), Howard L. Weiner, Lloyd F. Mayer (Editor) i Warren Strober (Editor), red. Oral Tolerance: New Insights and Prospects for Clinical Application (Annals of the New York Academy of Sciences). New York Academy of Sciences, 2004.

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32

Adorini. Immunointervention in Autoimmunity. Springer, 1998.

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33

L, Adorini, red. Immunointervention in autoimmunity by Th1/Th2 regulation. New York: Springer, 1997.

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34

Marshall, Tarnya, i Rita Abdulkader. Anti-rheumatic drugs in pregnancy and lactation. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0097.

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Streszczenie:
Anti-rheumatic drugs in pregnancy and lactation are increasingly a common clinical dilemma. With the shift towards early, aggressive control of autoimmune diseases and with the advent of newer therapeutic agents, there is a need to understand the effects of these medicines in pregnancy and lactation, on fertility in both men and women, and on the process of spermatogenesis, in order to understand the risk of teratogenesis. Although there are some limited data available for the use of anti-rheumatic drugs in pregnancy and lactation, much of our knowledge is derived from animal models and from limited clinical experience in human pregnancy. The balance of therapeutic benefits and risks of harm to mother and fetus should always be carefully considered: it may vary between individuals and should be assessed on a case by case basis. Because of these issues, pregnancy should always be discussed and planned in advance, in part to reduce disease activity prior to conception but also to minimize risk to the fetus. In this chapter we use the available evidence to discuss medicines which are commonly used in the treatment of rheumatological autoimmune diseases, and cover disease-modifying anti-rheumatic drugs (DMARDS) and biological agents.
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35

Marshall, Tarnya, Rita Abdulkader i Poonam Sharma. Antirheumatic drugs in pregnancy and lactation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0097_update_003.

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Antirheumatic drugs in pregnancy and lactation are increasingly a common clinical dilemma. With the shift towards early, aggressive control of autoimmune diseases and with the advent of newer therapeutic agents, there is a need to understand the effects of these medicines in pregnancy and lactation, on fertility in both men and women, and on the process of spermatogenesis, in order to understand the risk of teratogenesis. Although there are some limited data available for the use of antirheumatic drugs in pregnancy and lactation, much of our knowledge is derived from animal models and from limited clinical experience in human pregnancy. The balance of therapeutic benefits and risks of harm to mother and fetus should always be carefully considered: it may vary between individuals and should be assessed on a case by case basis. Because of these issues, pregnancy should always be discussed and planned in advance, in part to reduce disease activity prior to conception but also to minimize risk to the fetus. In this chapter we use the available evidence to discuss medicines which are commonly used in the treatment of rheumatological autoimmune diseases, and cover disease-modifying antirheumatic drugs (DMARDS) and biological agents.
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36

Chan, Chi-Chao. Animal Models of Ophthalmic Diseases. Springer London, Limited, 2015.

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37

Chan, Chi-Chao. Animal Models of Ophthalmic Diseases. Springer, 2019.

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38

Koch, Michael. Animal Models of Neuropsychiatric Diseases. Imperial College Press, 2006.

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39

Human Polygenic Diseases: Animal Models. Taylor & Francis, 1998.

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40

Baglietto-Vargas, David, Rahasson R. Ager, Rodrigo Medeiros i Frank M. LaFerla. Animal Models of Neurodegenerative Diseases. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190233563.003.0014.

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The incidence and prevalence of neurodegenerative disorders (e.g., Alzheimer’s disease (AD), Parkinson’s disease (PD), and Huntington’s disease (HD), etc.) are growing rapidly due to increasing life expectancy. Researchers over the past two decades have focused their efforts on the development of animal models to dissect the molecular mechanisms underlying neurodegenerative disorders. Existing models, however, do not fully replicate the symptomatic and pathological features of human diseases. This chapter focuses on animal models of AD, as this disorder is the most prevalent of the brain degenerative conditions afflicting society. In particular, it briefly discusses the current leading animal models, the translational relevance of the preclinical studies using such models, and the limitations and shortcomings of using animals to model human disease. It concludes with a discussion of potential means to improve future models to better recapitulate human conditions.
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41

Koch, Michael. Animal Models of Neuropsychiatric Diseases. PUBLISHED BY IMPERIAL COLLEGE PRESS AND DISTRIBUTED BY WORLD SCIENTIFIC PUBLISHING CO., 2006. http://dx.doi.org/10.1142/p421.

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Chan, Chi-Chao. Animal Models of Ophthalmic Diseases. Springer, 2015.

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43

Pang, Iok-Hou, i Abbot F. Clark. Animal Models for Retinal Diseases. Humana Press, 2016.

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44

Abbas, Abul K., i Richard A. Flavell. Genetic Models of Immune and Inflammatory Diseases. Springer, 2011.

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Abbas, Abul K., i Richard A. Flavell. Genetic Models of Immune and Inflammatory Diseases. Springer, 2012.

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46

Animal models of neurological disease. Totowa, N.J: Humana Press, 1992.

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47

E, Cornelius Charles, red. Animal models in liver research. San Diego: Academic Press, 1993.

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48

Ward, Billy C., Norman H. Altman i Edwin J. Andrews. Spontaneous Animal Models of Human Disease. Elsevier Science & Technology Books, 2012.

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49

Gross, David. Animal Models in Cardiovascular Research. Springer, 2010.

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Gross, David. Animal Models in Cardiovascular Research. Springer, 2009.

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