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Książki na temat „Antiviral inhibitors”

Autor: Grafiati
Data publikacji: 11 września 2023

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Sprawdź 32 najlepszych książek naukowych na temat „Antiviral inhibitors”.

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1

March, Darren. Designing new antiviral drugs for AIDS: HIV-1 protease and its inhibitors. Austin: R.G. Landes, 1996.

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2

American Society for Microbiology. Eastern Pennsylvania Branch i Eastern Pennsylvania Branch of the American Society for Microbiology Symposium of Innovations in Antiviral Development and the Detection of Virus Infections (1990 : Philadelphia, Pa.), red. Innovations in antiviral development and the detection of virus infections. New York: Plenum Press, 1992.

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3

HIV-1 integrase: Mechanism and inhibitor design. Hoboken, N.J: Wiley, 2011.

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4

Esté, José Andrés. Mode of action and development of resistance to human immunodeficiency virus inhibitors that are targeted at early stages of infection. Leuven, Belgium: Leuven University Press, 1999.

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5

RNA interference and viruses: Current innovations and future trends. Norfolk, UK: Caister Academic Press, 2010.

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6

Martínez, Miguel Angel. RNA interference and viruses: Current innovations and future trends. Norfolk, UK: Caister Academic Press, 2010.

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7

Hans-Georg, Kräusslich, Oroszlan Stephen, Wimmer Eckard i Cold Spring Harbor Laboratory, red. Viral proteinases as targets for chemotherapy. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory Press, 1989.

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8

Bilimoria, Darius M. Studies involving measles virus receptor interaction and inhibitors of virus mediated membrane fusion (a prelude to a small animal model and antiviral agents directed). Ottawa: National Library of Canada, 1998.

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9

Lendeckel, Uwe, i Nigel M. Hooper, red. Viral Proteases and Antiviral Protease Inhibitor Therapy. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-2348-3.

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10

H, Wagman Gerald, i Cooper Raymond, red. Natural products isolation: Separation methods for antimicrobials, antivirals, and enzyme inhibitors. Amsterdam: Elsevier, 1989.

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11

Gupta, Satya Prakash. Viral Proteases and Their Inhibitors. Elsevier Science & Technology Books, 2017.

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12

Gupta, Satya P. Viral Proteases and Their Inhibitors. Elsevier Science & Technology Books, 2017.

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13

Gupta, Satya Prakash. Cancer-Causing Viruses and Their Inhibitors. Taylor & Francis Group, 2014.

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14

Cancer-Causing Viruses and Their Inhibitors. Taylor & Francis Group, 2018.

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15

Gupta, Satya Prakash. Cancer-Causing Viruses and Their Inhibitors. Taylor & Francis Group, 2014.

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16

Gupta, Satya Prakash. Cancer-Causing Viruses and Their Inhibitors. Taylor & Francis Group, 2014.

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17

Gupta, Satya Prakash. Cancer-Causing Viruses and Their Inhibitors. Taylor & Francis Group, 2014.

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18

Viral Proteases And Antiviral Protease Inhibitor Therapy. Springer, 2009.

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19

Hooper, Nigel M., i Uwe Lendeckel. Viral Proteases and Antiviral Protease Inhibitor Therapy. Springer, 2009.

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20

Oncoviruses and Their Inhibitors. Taylor & Francis Group, 2014.

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21

Advances in Studies on Enzyme Inhibitors As Drugs : Volume 1: Anticancer and Antiviral Drugs. Nova Science Publishers, Incorporated, 2017.

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22

Ogden, Richard, i Gail Skowron. Reverse Transcriptase Inhibitors in HIV/AIDS Therapy. Humana Press, 2010.

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23

Ogden, Richard, i Gail Skowron. Reverse Transcriptase Inhibitors in HIV/AIDS Therapy. Springer, 2008.

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24

Ogden, Richard, i Gail Skowron. Reverse Transcriptase Inhibitors in HIV/AIDS Therapy. Humana Press, 2007.

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25

Hooper, Nigel M., i Uwe Lendeckel. Viral Proteases and Antiviral Protease Inhibitor Therapy: Proteases in Biology and Disease. Springer, 2009.

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26

Hooper, Nigel M., i Uwe Lendeckel. Viral Proteases and Antiviral Protease Inhibitor Therapy: Proteases in Biology and Disease. Springer, 2011.

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27

Innovations in Antiviral Development and the Detection of Virus Infection: Proceedings of an Eastern Pennsylvania Branch of the American Society for Microbiology Symposium Held in Philadelphia, Pennsylvania, November 15-16, 1990. New York: Plenum, 1992.

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28

Separation Methods for Antimicrobials, Antivirals and Enzyme Inhibitors. Elsevier, 1989. http://dx.doi.org/10.1016/s0301-4770(08)x6019-1.

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29

H, Wagman Gerald, i Cooper Raymond, red. Natural productsisolation: Separation methods for antimicrobials, antivirals and enzyme inhibitors. Elsevier, 1989.

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30

Cooper, R., i G. H. Wagman. Natural Products Isolation: Separation Methods for Antimicrobials, Antivirals and Enzyme Inhibitors. Elsevier Science & Technology Books, 1988.

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31

Oldstone, Michael B. A. Viruses, Plagues, and History. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780190056780.001.0001.

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“Viruses, Plagues, & History” focuses on the effects of viral diseases on human history. Written by an eminent internationally respected virologist, it couples the fabric of history with major concepts developed in virology, immunology, vaccination, and accounts by people who first had, saw and acted at the times these events occurred. Much of the preventive and therapeutic progress (vaccines, antiviral drugs) has been made in the last 60 years. Many of those who played commanding roles in the fight to understand, control and eradicate viruses and viral diseases are (were) personally known to the author and several episodes described in this book reflect their input. The book records the amazing accomplishments that led to the control of lethal and disabling viral diseases caused by Smallpox, Yellow Fever, Measles, Polio, Hepatitis A, B and C, and HIV. These six success stories are contrasted with viral infections currently out of control—COVID-19, Ebola virus, Lassa Fever virus, Hantavirus, West Nile virus, and Zika. Influenza, under reasonable containment at present, but with the potential to revert to a world-wide pandemic similar to 1918–1919 where over 50 million people were killed. The new platforms to develop inhibitory and prophylactic vaccines to limit these and other viral diseases is contrasted to the anti-vaccine movement and the false prophets of autism.
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32

Alexander, D. J., N. Phin i M. Zuckerman. Influenza. Redaktor I. H. Brown. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0037.

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Influenza is a highly infectious, acute illness which has affected humans and animals since ancient times. Influenza viruses form the Orthomyxoviridae family and are grouped into types A, B, and C on the basis of the antigenic nature of the internal nucleocapsid or the matrix protein. Infl uenza A viruses infect a large variety of animal species, including humans, pigs, horses, sea mammals, and birds, occasionally producing devastating pandemics in humans, such as in 1918 when it has been estimated that between 50–100 million deaths occurred worldwide.There are two important viral surface glycoproteins, the haemagglutinin (HA) and neuraminidase (NA). The HA binds to sialic acid receptors on the membrane of host cells and is the primary antigen against which a host’s antibody response is targeted. The NA cleaves the sialic acid bond attaching new viral particles to the cell membrane of host cells allowing their release. The NA is also the target of the neuraminidase inhibitor class of antiviral agents that include oseltamivir and zanamivir and newer agents such as peramivir. Both these glycoproteins are important antigens for inducing protective immunity in the host and therefore show the greatest variation.Influenza A viruses are classified into 16 antigenically distinct HA (H1–16) and 9 NA subtypes (N1–9). Although viruses of relatively few subtype combinations have been isolated from mammalian species, all subtypes, in most combinations, have been isolated from birds. Each virus possesses one HA and one NA subtype.Last century, the sudden emergence of antigenically different strains in humans, termed antigenic shift, occurred on three occasions, 1918 (H1N1), 1957 (H2N2) and 1968 (H3N2), resulting in pandemics. The frequent epidemics that occur between the pandemics are as a result of gradual antigenic change in the prevalent virus, termed antigenic drift. Epidemics throughout the world occur in the human population due to infection with influenza A viruses, such as H1N1 and H3N2 subtypes, or with influenza B virus. Phylogenetic studies have led to the suggestion that aquatic birds that show no signs of disease could be the source of many influenza A viruses in other species. The 1918 H1N1 pandemic strain is thought to have arisen as a result of spontaneous mutations within an avian H1N1 virus. However, most pandemic strains, such as the 1957 H2N2, 1968 H3N2 and 2009 pandemic H1N1, are considered to have emerged by genetic re-assortment of the segmented RNA genome of the virus, with the avian and human influenza A viruses infecting the same host.Influenza viruses do not pass readily between humans and birds but transmission between humans and other animals has been demonstrated. This has led to the suggestion that the proposed reassortment of human and avian influenza viruses takes place in an intermediate animal with subsequent infection of the human population. Pigs have been considered the leading contender for the role of intermediary because they may serve as hosts for productive infections of both avian and human viruses, and there is good evidence that they have been involved in interspecies transmission of influenza viruses; particularly the spread of H1N1 viruses to humans. Apart from public health measures related to the rapid identification of cases and isolation. The main control measures for influenza virus infections in human populations involves immunization and antiviral prophylaxis or treatment.
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