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Artykuły w czasopismach na temat "Alveolar recruitment"
Baumgartner, William A., Eric M. Jaryszak, Amanda J. Peterson, Robert G. Presson i Wiltz W. Wagner. "Heterogeneous capillary recruitment among adjoining alveoli". Journal of Applied Physiology 95, nr 2 (sierpień 2003): 469–76. http://dx.doi.org/10.1152/japplphysiol.01115.2002.
Pełny tekst źródłaSlutsky, A. S. "Barotrauma and alveolar recruitment". Intensive Care Medicine 19, nr 7 (lipiec 1993): 369–71. http://dx.doi.org/10.1007/bf01724874.
Pełny tekst źródłaHajari, A. J., D. A. Yablonskiy, A. L. Sukstanskii, J. D. Quirk, M. S. Conradi i J. C. Woods. "Morphometric changes in the human pulmonary acinus during inflation". Journal of Applied Physiology 112, nr 6 (15.03.2012): 937–43. http://dx.doi.org/10.1152/japplphysiol.00768.2011.
Pełny tekst źródłaAlbert, Scott P., Joseph DiRocco, Gilman B. Allen, Jason H. T. Bates, Ryan Lafollette, Brian D. Kubiak, John Fischer, Sean Maroney i Gary F. Nieman. "The role of time and pressure on alveolar recruitment". Journal of Applied Physiology 106, nr 3 (marzec 2009): 757–65. http://dx.doi.org/10.1152/japplphysiol.90735.2008.
Pełny tekst źródłaGhadiali, Samir N. "Making “time” for alveolar recruitment". Journal of Applied Physiology 106, nr 3 (marzec 2009): 751–52. http://dx.doi.org/10.1152/japplphysiol.91652.2008.
Pełny tekst źródłaCereda, Maurizio, i Yi Xin. "Alveolar Recruitment and Lung Injury". Critical Care Medicine 41, nr 12 (grudzień 2013): 2837–38. http://dx.doi.org/10.1097/ccm.0b013e31829cb083.
Pełny tekst źródłaKacmarek, Robert M. "Strategies to optimize alveolar recruitment". Current Opinion in Critical Care 7, nr 1 (luty 2001): 15–20. http://dx.doi.org/10.1097/00075198-200102000-00003.
Pełny tekst źródłaMancebo, J. "PEEP, ARDS, and alveolar recruitment". Intensive Care Medicine 18, nr 7 (lipiec 1992): 383–85. http://dx.doi.org/10.1007/bf01694337.
Pełny tekst źródłaLista, G., F. Castoldi, F. Cavigioli, S. Bianchi i P. Fontana. "Alveolar recruitment in the delivery room". Journal of Maternal-Fetal & Neonatal Medicine 25, sup1 (5.03.2012): 39–40. http://dx.doi.org/10.3109/14767058.2012.663164.
Pełny tekst źródłaEsquinas, Antonio M., i Luca S. De Santo. "Alveolar recruitment manoeuvres after cardiac surgery". European Journal of Anaesthesiology 35, nr 1 (styczeń 2018): 61–62. http://dx.doi.org/10.1097/eja.0000000000000652.
Pełny tekst źródłaRozprawy doktorskie na temat "Alveolar recruitment"
Andrade, Felipe Silveira Rêgo Monteiro de. "Estudo clínico da mecânica respiratória em equinos sob ventilação com volume controlado durante cirurgia artroscópica". Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/10/10137/tde-04092015-180239/.
Pełny tekst źródłaIt is known that general anaesthesia by it’s self is capable of causing substantial cardiovascular and respiratory depression in horses and this characteristic can be enhanced even more by patient positioned in dorsal recumbence and the administration of high intrathoracic pressures during recruitment manoeuvres used to reverse hypoxemia. Therefore, the aim of this study was to evaluate the respiratory mechanics and arterial blood gas analysis after recruitment manoeuvre and PEEP for maintenance, in horses ASA I and II undergoing general isoflurane-anaesthesia for arthroscopic surgery and what is the best value PEEP to maintain alveolar recruitment. Therefore, we used 30 horses, weighing on average 454 kg, which underwent arthroscopic surgery in the dorsal recumbence, randomly allocated into one of the 4 groups, as follows: Control; PEEP 7; PEEP 12; and PEEP 17. Animals received xylazine (0,6 mg/kg) as pre anaesthetic medication followed by anaesthesia induction (ketamine 2,2 mg/kg associated to diazepam 0,05 mg/kg and EGG 10% 50 mg/kg) and maintenance with isoflurane-anaesthesia. The animals were positioned in dorsal recumbence and submitted the volume-controlled ventilation (14ml/kg), RR: 7 mpm, I:E ratio 1:3, 7 cmH2O of PEEP and FiO2 0,7. After instrumentation period was performed RM by PEEP titration every 5 minutes until reach 22 cmH2O of PEEP, and the animals of control group did not receive RM, only maintenance with PEEP 7 cmH2O. The animals of other groups went through the RM followed by maintenance with their treatment PEEP (7, 12 or 17 cmH2O). The respiratory parameters and blood gas samples were assessed immediately before the RM; and 5, 10, 15, 20, 40, 60 and 80 minutes after the manoeuvre. We also assessed the HR, SAP, MAP and DAP, percentage of inhaled anaesthetic: inhaled and exhaled, ETCO2 and vasoactive drug consumption. Animals receiving RM showed an increase in static compliance and oxygenation parameters after the manoeuvre, maintenance of the increase coming from the RM were observed in animals from PEEP 12 and 17 group, for at least 80 minutes. The animals in PEEP 7 group decreased the increase after 20 minutes of manoeuvre and the control group decreased the parameters of oxygenation and ventilation over time, both indicating a probable pulmonary closure due to insufficient PEEP to maintain the alveoli opened. Cardiovascular changes were observed in the study animals, only mild transient tachycardia in PEEP 17group soon after RM. Therefore, the PEEP 12 and 17 cmH2O used after RM were able to keep the lung opened, thereby performing better gas exchange and the increase in the oxygenation and ventilation of patients. The animals receiving RM and maintenance PEEP 7 cmH2O were able to maintain the alveoli open for only 20 minutes
Mott, Lara Lopes Facó. "Avaliação de diferentes protocolos de recrutamento alveolar durante a ventilação mecânica em equinos submetidos a laparotomia". Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/10/10137/tde-13042018-142412/.
Pełny tekst źródłaThe equine patient suffering from acute abdomen syndrome anesthesia remains one of the biggest challenges for the veterinary anesthesiologist. Despite many advances on mechanical ventilation, volemic status monitoring and critical care patient management it is still extremely difficult to achieve the balance between the best ventilation strategy to minimize the occurrence of ventilation mismatch and the optimization of cardiovascular parameters to keep blood perfusion to peripheral tissues within normal ranges. Therefore, the aim of this study was to assess the effects of a stepwise increase in airway pressure recruitment and two different values of positive end expiratory pressure (PEEP) on ventilatory, oxygenation and hemodynamics parameters as well as vasoactive drugs consumption on horses undergoing exploratory laparotomy. Thus, twenty client-owned horses, weighing 431± 95 kg, refered to the Large Animal Surgery Service of the Faculty of Veterinary Medicine and Animal Science, at the University of São Paulo were included on the study. The animals were premedicated with xilazine (0,6mg/kg-1) and after 10 minutes the anesthesia was induced with ketamine (2,2 mg/kg-1) and diazepam (0,05 mg/kg-1). The subjects were positioned in dorsal recumbence, maintained in volume controlled mechanical ventilation, with tidal volume of 14ml/kg. After 30 min of instrumentation the recruitment maneuver (RM) was performed by PEEP titration with increments of 5 cmH2O every 5 min up to a PEEP of 20 cmH2O followed by decremental PEEP titration until 12 cmH20 (Group PEEP12, n=10) or 17 cmH2O (Group PEEP 17, n=10) until the end of surgical procedure. The Group PEEP12 had significantly higher values of mean arterial blood pressure (MAP) and pH for 100 minutes after the RM was performed. The animals in this group also needed less vasoactive drugs (dobutamine and noradrenaline) to keep the MAP>60mmHg during the whole anesthesia time. There was no difference between group PEEP12 a group PEEP 17 regarding oxygenation parameters 30 minutes after the anesthesia was ended.
Ambrósio, Aline Magalhães. "Estudo da influência das manobras de recrutamento alveolar sobre a mecânica, a ventilação e o parênquima pulmonar durante lesão aguda promovida pela instilação de ácido clorídrico: estudo experimental em porcos". Universidade de São Paulo, 2005. http://www.teses.usp.br/teses/disponiveis/10/10137/tde-29092006-201943/.
Pełny tekst źródłaDifferent mechanical ventilation strategies which define limits of intrathoracic pressures and volumes are being proposed for patients with acute respiratory distress syndrome (ARDS). These recommendations are based on observations that mechanical ventilation with excessive tidal volumes or insufficient values of positive end expiratory pressure (PEEP) can cause severe lung injury due to overinflation. The aim of the present study was to apply recruitment maneuvers (RM) and PEEP in lungs submitted to acute lung injury (ALI) due to the administration of hydrochloride acid. Twenty four female Landrace Largewhite pigs, weighing 25 to 35 Kg were used. After anesthesia, animals were submitted to volume controlled mechanical ventilation (6 to 8ml/kg) and were randomly allocated in four groups of 6 animals each: GI animals without ALI and treated with progressive values of PEEP (5, 10, 15 and 20 cmH2O) or regressive (20 to 5 cm H2O); GII animals without ALI and treated with progressive values of PEEP (5, 10, 15 and 20 cmH2O) or regressive (20 to 5 cm H2O) plus 3 consecutive recruitment maneuvers with 30 cmH2O; GIII animals submitted to 1 hour of ALI and treated as GI; GIV animals submitted to 1 hour of ALI and treated as GII. Parameters of respiratory mechanics, ventilation and oxygenation were measured each 20 minutes according to the change of the PEEP values. ALI could be observed by the severe changes of oxygenation and respiratory mechanics noted. The use of RM and PEEP were able to restore control values. Nevertheless, application of high values of PEEP and CPAP were accompanied by significant hemodynamic changes which could be evidenced in animals without ALI. Derecruitment probably occurred when PEEP value reached 5 cmH2O. The lung lesions were uniform in the HCL-injured animals and consisted of necrosis, hemorrhage, congestion, and inflammatory cells infiltration that involved both the interstitium and the alveoli. The experimental model of lung injury was adequate to the study of RM followed by PEEP since significant changes of the oxygenation and compliance values could be observed 1 hour after acid instillation. PEEP values of 5cmH2O were incapable to maintain recruitment at the end of the observation period, while 10 cmH2O were sufficient to promote the reestablishment of oxygenation index with minimal hemodynamic changes. Compliance did not improve during the maneuvers. Further studies are necessary to confirm the results obtained, especially to show that the maintenance of a PEEP value of 10 cmH2O are sufficient to maintain recruitment after the RM
Amaral, Ronaldo Alves do. "Origem f?sica das curvas sigmoidais respirat?rias press?o-volume: recrutamento alveolar e elasticidade n?o-linear". Universidade Federal do Rio Grande do Norte, 2011. http://repositorio.ufrn.br:8080/jspui/handle/123456789/13248.
Pełny tekst źródłaAn important unsolved problem in medical science concerns the physical origin of the sigmoidal shape of pressure volume curves of healthy (and some unhealthy) lungs. Such difficulties are expected because the lung, which is the most important structure in the respiratory system, is extremely complex. Its rheological properties are unknown and seem to depend on phenomena occurring from the alveolar scale up to the thoracic scale. Conventional wisdom holds that linear response, i.e., Hooke s law, together with alveolar overdistention, play a dominant role in respiration, but such assumptions cannot explainthe crucial empirical sigmoidal shape of the curves. In this doctorate thesis, we propose an alternative theory to solve this problem, based on the alveolar recruitment together with the nonlinear elasticity of the alveoli. This theory suggests that recruitment may be the predominant factor shaping these curves in the entire range of pressures normally employed in experiments. The proposed model correctly predicts the observed sigmoidal pressure volume curves, allowing us to discuss adequately the importance of this result, as well as its implications for medical practice
Um importante problema n?o resolvido na ci?ncia m?dica diz respeito ? origem f?sica da forma sigmoidal das curvas press?o-volume de pulm?es saud?veis (e de alguns n?o saud?veis). Tais dificuldades s?o esperadas tendo em vista que o pulm?o, a mais importante estrutura do sistema respirat?rio, ? extremamente complexo. Suas propriedades reol?gicas s?o desconhecidas, e parecem depender dos fen?menos que ocorrem a partir da escala alveolar at? a escala tor?cica. O modelo cl?ssico diz que a resposta linear, ou seja, a lei de Hooke, juntamente com a superdistens?o alveolar desempenha um papel predominante na formata??o dessas curvas, mas tais pressupostos n?o podem explicar a crucial forma emp?rica sigmoidal das curvas P-V respirat?rias est?ticas. Nesta tese de doutorado propomos uma teoria alternativa para resolver este problema, baseada no recrutamento alveolar, juntamente com a hip?tese do comportamento el?stico n?o-linear dos alv?olos. Esta teoria sugere o recrutamento alveolar como o fator predominante que modela as curvas press?o-volume em um completo intervalo de press?o normalmente utilizado nas experi?ncias. O modelo proposto prediz corretamente a forma sigmoidal observada nas curvas press?o-volume, nos permitindo discutir adequadamente a import?ncia deste resultado, assim como as suas implica??es para a pr?tica m?dica
Rodrigues, Jéssica Corrêa [UNESP]. "Efeitos do pneumoperitônio e de uma manobra de recrutamento alveolar seguida por pressão positiva no final da expiração na função cardiopulmonar em ovinos anestesiados com isoflurano e fentanil". Universidade Estadual Paulista (UNESP), 2016. http://hdl.handle.net/11449/136391.
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Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)
A realização da laparoscopia cirúrgica requer a insuflação de gás carbônico (CO2) na cavidade abdominal. O pneumoperitônio formado eleva a pressão intra-abdominal (PIA), deslocando o diafragma em sentido cranial, o que resulta em diminuição da complacência pulmonar e consequentemente formação de áreas atelectásicas. Este estudo objetivou investigar os efeitos do pneumoperitônio e de uma manobra de recrutamento alveolar (MRA) seguida por aplicação de pressão positiva ao final da expiração (PEEP) na função cardiorrespiratória em ovinos. Em um delineamento prospectivo aleatório cruzado, nove ovinos (36–52 kg) foram anestesiados com isoflurano e fentanil e submetidos à ventilação com volume controlado (volume corrente: 12 mL/kg) com o emprego do bloqueador neuromuscular atracúrio. Cada animal recebeu três tratamentos com intervalo de dez dias entre cada experimento: Controle (sem intervenção); Pneumo (pneumoperitônio mantido por 120 minutos sob PIA de 15 mmHg); Pneumo+MRA/PEEP (pneumoperitônio mantido por 120 minutos sob PIA de 15 mmHg e realização de uma MRA aos 60 minutos após insuflação abdominal seguida por 10 cmH2O de PEEP). A MRA consistiu em aumentos progressivos na pressão expiratória a cada minuto até alcançar o valor de 20 cmH2O de PEEP. As variáveis estudadas foram coletadas até 30 minutos após a interrupção do pneumoperitônio. A insuflação abdominal com CO2 diminuiu significativamente (P < 0.05) os valores de PaO2 de 435–462 mmHg (intervalo dos valores médios observados) no tratamento Controle para 377–397 mmHg e 393–413 mmHg nos tratamentos Pneumo e Pneumo+MRA/PEEP, respectivamente. A complacência estática (Cstat, mL/cmH2O/kg) diminuiu significativamente de 0,83–0,86 (Controle) para 0,49–0,52 (Pneumo) e 0,51–0,54 (Pneumo+MRA/PEEP) após a indução do pneumoperitônio. A MRA/PEEP elevou significativamente a PaO2 (429–444 mmHg) e a Cstat (0,68–0,72) quando comparada com o os animais sob pneumoperitônio que não receberam a MRA/PEEP (PaO2: 383–385 mmHg e Cstat: 0,48–0,49). A realização do pneumoperitônio aumentou significativamente a formação de “shunt” intrapulmonar; porém após a aplicação da MRA/PEEP houve uma diminuição significativa nos valores de “shunt”. Trinta minutos após a desinsuflação abdominal, a PaO2 e a Cstat encontravam-se significativamente menores e o “shunt” intrapulmonar significativamente maior no tratamento Pneumo quando comparado ao tratamento Controle. Durante os últimos 60 minutos de pneumoperitônio (Pneumo e Pneumo+MRA/PEEP), os valores médios de índice cardíaco (IC) foram 20–28 % menores (P < 0.05) que os valores observados no tratamento Controle. Após a MRA/PEEP, a pressão média da artéria pulmonar (PMAP) apresentou-se significativamente maior (47-56%) e a pressão arterial média (PAM) apresentou-se significativamente menor (16%) em relação ao tratamento Controle. Concluiu-se que a desinsuflação abdominal não foi suficiente para reverter os impactos negativos na função pulmonar associados à realização do pneumoperitônio e que a realização de uma MRA seguida por PEEP foi capaz de melhorar a complacência do sistema pulmonar e reverter o prejuízo na oxigenação ocasionados pela insuflação abdominal, sem, no entanto, induzir alterações hemodinâmicas inaceitáveis.
Laparoscopic surgical procedures usually require carbon dioxide (CO2) insufflation into the peritoneal cavity. The pneumoperitoneum increases intra-abdominal pressure (IAP) displaces the diaphragm cranially, and decreases respiratory system compliance, leading to the development of atelectasis. This study aimed to investigate the effects of pneumoperitoneum and of an alveolar recruitment maneuver (ARM) followed by positive end-expiratory pressure PEEP on cardiopulmonary function in sheep. In a prospective randomized crossover study, nine sheep (36–52 kg) received 3 treatments with 10-day intervals during isoflurane-fentanyl anesthesia and volume-controlled ventilation (tidal volume: 12 mL/kg): Control (no intervention); Pneumo (120 minutes of CO2 pneumoperitoneum until achieving an intra-abdominal pressure of 15 mmHg); Pneumo+ARM/PEEP (same pneumoperitoneum protocol with an ARM after 60 minutes of abdominal inflation). The ARM consisted of stepwise increases in end-expiratory pressures every minute until 20 cmH2O of PEEP, followed by 10 cmH2O of PEEP. Data were recorded until 30 minutes after abdominal deflation. Abdominal inflation significantly (P < 0.05) decreased PaO2 from 435–462 mmHg (range of recorded mean values) in controls to 377–397 mmHg and 393–413 mmHg in the Pneumo and Pneumo+ARM/PEEP treatments, respectively. Static compliance (Cstat, mL/cmH2O/kg) was significantly decreased from 0.83–0.86 (Control) to 0.49–0.52 (Pneumo), and 0.51–0.54 (Pneumo+ARM/PEEP) after induction of pneumoperitoneum. The ARM/PEEP significantly increased PaO2 [429–444 mmHg and Cstat (0.68–0.72)] from values recorded during pneumoperitoneum alone [PaO2: 383–385 mmHg and Cstat: 0.48–0.49]. Pneumoperitoneum significantly increased intrapulmonary Shunt; while the ARM/PEEP significantly decreased the Shunt. Thirty minutes after abdominal deflation (Pneumo), PaO2 and Cstat were significantly lower and the Shunt was higher than in controls. During the last 60 minutes of pneumoperitoneum (Pneumo and Pneumo+ARM/PEEP), cardiac index values were 20–28 % lower than in controls. After the ARM/PEEP, mean pulmonary artery pressure was significantly higher (47-56%) and mean systemic arterial pressure was significantly lower (16%) than controls. It was concluded that abdominal deflation is not enough to reverse the impairment in pulmonary function associated with pneumoperitoneum and the ARM/PEEP may improve respiratory system compliance and reverse the oxygenation impairment induced by pneumoperitoneum with clinically acceptable hemodynamic changes.
Kozian, Alf. "Pathophysiological and Histomorphological Effects of One-Lung Ventilation in the Porcine Lung". Doctoral thesis, Uppsala universitet, Klinisk fysiologi, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-108850.
Pełny tekst źródłaNamati, Eman, i eman@namati com. "Pre-Clinical Multi-Modal Imaging for Assessment of Pulmonary Structure, Function and Pathology". Flinders University. Computer Science, Engineering and Mathematics, 2008. http://catalogue.flinders.edu.au./local/adt/public/adt-SFU20081013.044657.
Pełny tekst źródłaMori, Audie Rollin Roldan. "Impacto de duas estratégias de titulação da PEEP em modelo suíno de síndrome do desconforto respiratório agudo: guiada por pressão esofágica versus guiada por tomografia de impedância elétrica". Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/5/5150/tde-27092017-114609/.
Pełny tekst źródłaINTRODUCTION: The use of higher levels of positive end-expiratory pressure (PEEP) in the acute respiratory distress syndrome (ARDS), aimed at reducing the amount of lung collapse, making the ventilation more homogeneous, has been pointed out by randomized clinical trials and meta-analysis as an effective strategy to improve some clinical outcomes. Currently, there is no ideal method for adjustment PEEP in ARDS. Two methods are distinguished by their physiological rationality and the possibility of being used in the clinical practice: PEEP titration guided by Esophageal Pressure (Pes) and PEEP titration guided by Electrical Impedance Tomography (EIT). The objectives of the study were: 1) To evaluate through computed tomography of thorax (X-ray), which strategy induces better pulmonary aeration: greater lung recruitment and less hyperdistension; (2) To evaluate changes in the regional distribution of ventilation, pulmonary volume and regional compliance, measured by electrical impedance tomography; (3) To assess changes in lung mechanics and gas exchange produced by both PEEP titration strategies. METHODS: Ten pigs were submitted to a two-hit model of severe ARDS: Surfactant depletion plus ventilator-induced lung injury. After a recruitment maneuver (RM), two strategies of PEEP titration were tested in a randomized sequence: 1) Using electric impedance tomography to calculate the lowest PEEP keeping recruitable-lungcollapse < 1%; 2) Using esophageal pressure to calculate the PEEP needed to achieve an end-expiratory transpulmonary pressure between 5-6 cmH2O. Then, animals were ventilated for 1 hour with the optimum-PEEP estimated by each method. Physiological and computed tomography (CT) parameters were recorded before RM (baseline) and after ventilation at optimum-PEEP (15 min and 60 min). RESULTS: At 60 min, both strategies reduced lung collapse but with significantly (P < 0.05) greater effects in EIT-group: nonaerated tissue (20.3 ± 11.8% vs 38.6 ± 13.1%, EIT vs. Pes, respectively), tidal recruitment (4.8 ± 3.7% vs 8.7 ± 2.7%), PaO2/FIO2 (289 ± 78 vs 209 ± 92 mmHg), driving-pressure (14.5 ± 2.3 vs 16.1 ± 2.3 cmH2O) and transpulmonary driving-pressure (11.9 ± 1.7 vs 13.6 ± 1.8 cmH2O). Despite the choice for a higher optimum-PEEP in the EIT-group; plateau pressure (33.2 ± 3.7 vs 31.5 ± 3.1 cmH2O), end-inspiratory transpulmonary pressure (20.0 ± 2.8 vs 19.2 ± 1.7 cmH2O) and compliance of non-dependent areas measured by EIT (0.07 ± 0.04 vs 0.06 ± 0.05 arbitrary units/cmH2O) or CT (1.52 ± 0.90 vs 1.41 ± 0.98 mL/cmH2O) varied similarly in both groups (P > 0.05). Hyperaerated tissue and tidal hyperinflation were very low in both groups. CONCLUSION: In this model, the choice of PEEP guided by EIT leads to higher lung recruitment and physiological signals of a better lung protection, when compared to the strategy guided by Pes
Ya-i, Hsieh, i 謝雅宜. "Clinical Efficacy of Manual Hyperinflation on Alveolar Recruitment in Difficult Weaning Patients". Thesis, 2002. http://ndltd.ncl.edu.tw/handle/01694989282061929660.
Pełny tekst źródła長庚大學
護理學研究所
90
Use of mechanical ventilator support through tracheostomy or intubation has been one of extremely important medical intervention to help support life for ICU patients. However, as the result of using current medical intervention for patients with lung collapse during mechanical ventilation is limited, manual hyperinflation helps alveolar recruitment that results in secretion clearance, ventilation improvement, lung collapse prevention, lung collapse re-inflation, dynamic compliance improvement, artery oxygenation improvement and work of breathing reduction according to certain clinic reports from foreign sources. This study seeks to verify success of sputum clearance and ventilation in giving manual hyperinflation to patients suffering from lung collapse who has been using mechanical ventilation for more than one week. The study takes the assumption that through manual hyperinflation to recruit alveolar, it will help patients sputum clearance and ventilation. A controlled, randomized, single blind, experimental design was used. Each patient was assigned a random number upon entering the research project, which allowed randomly allocation to two groups: experimental group and control group. The samples were collected in the ICU of a Medical Center in northern Taiwan and the Chronic Respiratory Treatment Center of the Regional Hospital under the cooperation program. A total of 28 patients joined the study in two groups, i.e., experimental group and control group, 14 patients in each group. Cases from the experimental group were treated with ventilation support and additional manual hyperinflation while cases from the control group were only treated with ventilation support. Patients in experimental group received the manual hyperinflation once of 20 minutes in 3 times per day for 5 days. Outcome measurement was evaluated by noting changes in the patient’s score in dry/wet ratio of sputum, sputum viscosity, tidal volume, maximal respiratory pressure, PaO2/FiO2, upper chest X-ray film, dynamic lung compliance and the index of rapid shallow breathing. Outcome measurements were assessed 3 times for all patients: at the day admitted to this study, the 3rd day, and 6th day. Upon giving the conclusions are as follows: (1)For the sputum clearance, patients from the experiment group indicate significant improvement than those from control group does depending on the time-point in terms of sputum viscosity (p=0.002); while patients from both group indicate no significant variance in dry/wet ratio of sputum; (2)For the ventilation, patients from the experimental group indicate significant improvement than those from control group does depending on the time-point in terms of improved tidal volume (p=0.033), maximal respiratory pressure (p=0.05), PaO2/FiO2 (p=0.016), upper chest X-ray film (p=0.069), the index of rapid shallow breathing (p=0.008); and while patients from both groups indicate no significant variance in terms of dynamic compliance.
Herold, Susanne [Verfasser]. "Macrophage-epithelial interactions during influenza virus pneumonia : alveolar recruitment pathways and impact on epithelial barrier integrity / by Susanne Valerie Herold". 2008. http://d-nb.info/992276659/34.
Pełny tekst źródłaKsiążki na temat "Alveolar recruitment"
Muders, Thomas, i Christian Putensen. Pressure-controlled mechanical ventilation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0096.
Pełny tekst źródłaDeCampos, Kleber N. Lung preservation for transplantation: The role of reperfusion flow rate and alveolar recruitment on post-ischemic pulmonary function in rats. 1996.
Znajdź pełny tekst źródłaWare, Lorraine B. Pathophysiology of acute respiratory distress syndrome. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0108.
Pełny tekst źródłaLucangelo, Umberto, i Massimo Ferluga. Pulmonary mechanical dysfunction in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0084.
Pełny tekst źródłaCuartero, Mireia, i Niall D. Ferguson. High-frequency ventilation and oscillation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0098.
Pełny tekst źródłaHedenstierna, Göran, i Hans Ulrich Rothen. Physiology of positive-pressure ventilation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0088.
Pełny tekst źródłaCzęści książek na temat "Alveolar recruitment"
Bigatello, L. M., i P. Caironi. "Alveolar Recruitment". W Anaesthesia, Pain, Intensive Care and Emergency Medicine — A.P.I.C.E., 215–24. Milano: Springer Milan, 2002. http://dx.doi.org/10.1007/978-88-470-2099-3_18.
Pełny tekst źródłaMergoni, M., A. Volpi i A. Rossi. "Inflection Point and Alveolar Recruitment in ARDS". W Yearbook of Intensive Care and Emergency Medicine, 556–67. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-662-13450-4_46.
Pełny tekst źródłaKuraszkiewicz, Bozenna. "Modelling of Alveolar Recruitment Phenomena in Human Lungs". W Advances in Soft Computing, 231–39. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-04462-5_23.
Pełny tekst źródłaSydow, M., i H. Burchardi. "Influence of Time on Alveolar Recruitment in Acute Lung Injury". W Yearbook of Intensive Care and Emergency Medicine, 127–40. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79154-3_11.
Pełny tekst źródłaLovas, A., i Z. Molnár. "Alveolar Recruitment in Patients with Assisted Ventilation: Open Up the Lung in Spontaneous Breathing". W Annual Update in Intensive Care and Emergency Medicine 2018, 205–15. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-73670-9_17.
Pełny tekst źródłaScott, J. Aaron, i Vivek Moitra. "Positive End-Expiratory Pressure Setting in Adults with ALI and ARDS". W 50 Studies Every Intensivist Should Know, redaktorzy Edward A. Bittner i Michael E. Hochman, 147–51. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190467654.003.0024.
Pełny tekst źródłaShah, Chirag V. "What Is the Role of Alveolar Recruitment Maneuvers in the Management of ARDS?" W Evidence-Based Practice of Critical Care, 118–24. Elsevier, 2010. http://dx.doi.org/10.1016/b978-1-4160-5476-4.00019-5.
Pełny tekst źródłaCheung, Cindy, i Christopher W. Tam. "Robotic Mitral Valve Surgery and Unilateral Pulmonary Edema". W Cardiothoracic Critical Care, 171–78. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780190082482.003.0017.
Pełny tekst źródłaStreszczenia konferencji na temat "Alveolar recruitment"
Dianti, J. M., M. S. Venuti, E. Gogniat, M. Ducrey, M. Madorno, M. Las Heras, S. Giannasi, E. San Roman i G. Tusman. "Stress Index Predicts Alveolar Recruitment After a Lung Recruitment Maneuver". W American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1657.
Pełny tekst źródłaCereda, Maurizio, Kiarash Emami, Stephen Kadlecek, Yi Xin, Puttisarn Mongkolwisetwara, Harilla Profka, Amy Barulic i in. "Quantitative Imaging Of Alveolar Recruitment With Hyperpolarized Gas MRI". W American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2632.
Pełny tekst źródłaDegryse, Amber L., Xiaochuan C. Xu, Harikrishna Tanjore, Vasiliy V. Polosukhin, Brittany Jones, Frank B. Mc Mahon, Camilla Ortiz, Timothy S. Blackwell i William E. Lawson. "TGFBeta Signalling In Epithelium Regulates Bleomycin Induced Alveolar Injury And Fibroblast Recruitment". W American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a6144.
Pełny tekst źródłaSilva, Pedro L., Lilian Moraes, Raquel Santos, Cynthia S. Samary, Felipe Saddy, Humberto C. Junior, Tatiana Maron-Gutierrez i in. "Effects Of Different Recruitment Maneuvers On Lung Morpho-function And Alveolar Stress". W American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1688.
Pełny tekst źródłaStewart, Glenn, Caitlin Fermoyle, Courtney Wheatley, Briana Ziegler i Bruce Johnson. "Influence of ultra-endurance exercise on alveolar-capillary recruitment and lung diffusion". W ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2441.
Pełny tekst źródłaJose, Ann Mary. "Alveolar recruitment improving oxygenation in Acute Respiratory Distress Syndrome: A prospective observational study". W ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2338.
Pełny tekst źródłaRisquez, Cristobal F., Avignat Patel, Juan C. Osorio, Isis E. Fernandez, Andrew Goodwin, Ying Shi, Xiaomeng Tang, Danielle Morse, Ivan O. Rosas i Yuanyuan Shi. "Syndecan-2 And CCL2 Interactions Promote Alveolar Macrophage Recruitment During Acute Lung Injury". W American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3700.
Pełny tekst źródłaAmin, Samir, Henrique T. Moriya, Béla Suki i Adriano M. Alencar. "Airway Tree Model Of Lung Recruitment: Effect Of Alveolar Compliance On Pressure Volume Fluctuations". W American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3656.
Pełny tekst źródłaWall, Wolfgang A., Andrew Comerford, Lena Wiechert i Sophie Rausch. "Coupled and Multi-Scale Building Blocks for a Comprehensive Computational Lung Model". W ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206407.
Pełny tekst źródłaVadasz, I., Y. Buchaeckert, R. Ruehl, W. Seeger i RE Morty. "Recruitment Maneuvers Impair Alveolar Epithelial Function by Inhibiting Na,K-ATPase Function in Rabbit Lungs." W American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a4945.
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