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Artykuły w czasopismach na temat "Alcohol-related brain damage"
Erdozain, Amaia M., Benito Morentin, Lynn Bedford, Emma King, David Tooth, Charlotte Brewer, Declan Wayne i in. "Alcohol-Related Brain Damage in Humans". PLoS ONE 9, nr 4 (3.04.2014): e93586. http://dx.doi.org/10.1371/journal.pone.0093586.
Pełny tekst źródłaLovinger, David M. "Excitotoxicity and Alcohol-Related Brain Damage". Alcoholism: Clinical and Experimental Research 17, nr 1 (luty 1993): 19–27. http://dx.doi.org/10.1111/j.1530-0277.1993.tb00720.x.
Pełny tekst źródłaHalliday, Glenda, Kerry Baker i Clive Harper. "Serotonin and alcohol-related brain damage". Metabolic Brain Disease 10, nr 1 (marzec 1995): 25–30. http://dx.doi.org/10.1007/bf01991780.
Pełny tekst źródłaSmith, Caine C., Donna L. Sheedy, Holly P. McEwen, Anthony S. Don, Jillian J. Kril i Greg T. Sutherland. "Lipidome changes in alcohol‐related brain damage". Journal of Neurochemistry 160, nr 2 (11.11.2021): 271–82. http://dx.doi.org/10.1111/jnc.15530.
Pełny tekst źródłaRao, Rahul, i Brian Draper. "Alcohol-related brain damage in older people". Lancet Psychiatry 2, nr 8 (sierpień 2015): 674–75. http://dx.doi.org/10.1016/s2215-0366(15)00215-1.
Pełny tekst źródłaGuerrini, I., A. D. Thomson i H. M. Gurling. "Molecular Genetics of Alcohol-Related Brain Damage". Alcohol and Alcoholism 44, nr 2 (16.01.2009): 166–70. http://dx.doi.org/10.1093/alcalc/agn101.
Pełny tekst źródłaHarper, C. "The Neuropathology of Alcohol-Related Brain Damage". Alcohol and Alcoholism 44, nr 2 (16.01.2009): 136–40. http://dx.doi.org/10.1093/alcalc/agn102.
Pełny tekst źródłaAziz, Victor M. "Alcohol-related brain damage (ARBD): a service need". International Psychogeriatrics 26, nr 10 (3.07.2014): 1747–49. http://dx.doi.org/10.1017/s1041610214001252.
Pełny tekst źródłaPlace, Charlie. "Alcohol related brain damage – a case of neglect?" Advances in Dual Diagnosis 7, nr 3 (12.08.2014): 129–36. http://dx.doi.org/10.1108/add-02-2014-0008.
Pełny tekst źródłaJauhar, Sameer, i Iain D. Smith. "Alcohol-related brain damage: not a silent epidemic". British Journal of Psychiatry 194, nr 3 (marzec 2009): 287–88. http://dx.doi.org/10.1192/bjp.194.3.287b.
Pełny tekst źródłaRozprawy doktorskie na temat "Alcohol-related brain damage"
Alexander-Kaufman, Kimberley Louise. "Proteomics of the human alcoholic brain: Implications for the pathophysiology of alcohol-related brain damage". The University of Sydney, 2008. http://hdl.handle.net/2123/2692.
Pełny tekst źródłaProteomics is rapidly achieving recognition as a complimentary and perhaps superior approach to examine global changes in protein abundance in complex biological systems and the value of these techniques in neuropsychiatry is beginning to be acknowledged. Characterizing the brain’s regional proteomes provides a foundation for the detection of proteins that may be involved in disease-related processes. Firstly, optimal conditions were achieved for the application of two dimensional-gel electrophoresis (2D-GE)-based proteomics with postmortem human brain tissue. These optimized techniques were then applied to soluble fractions of adjacent grey and white matter of a single cytoarchitecturally defined area (Brodmann area 9; BA9) and of two adjacent regions of frontal white matter (BA9 and CC body) from healthy individuals. These normative proteomic comparisons highlighted the importance of correct tissue sampling, i.e. proper separation of regional white matter, as heterogeneity in the respective proteomes was demonstrated. Furthermore, they stressed the necessity for future molecular brain mapping studies. The main focus of this thesis however, was to examine the proteomes of brain regions specifically vulnerable to alcohol-induced damage underlying cognitive dysfunction. Alcoholic patients commonly experience mild to severe cognitive decline. It is postulated that cognitive dysfunction is caused by an alcohol-induced region selective brain damage, particularly to the prefrontal cortex. The cerebellum is increasingly recognized for its role in various aspects of cognition and alcohol–induced damage to the cerebellar vermis could indirectly affect neurocognitive functions attributed to the frontal lobe. We used a 2D-GE-based proteomics approach to compare protein abundance profiles of BA9 grey and white matter and the cerebellar vermis from human alcoholics (neurologically uncomplicated and alcoholics complicated with liver cirrhosis) and healthy control brains. Among the protein level changes observed are disturbances in the levels of a number of thiamine-dependent enzymes. A derangement in energy metabolism perhaps related to thiamine deficiency seems to be important in all regions analysed, even where there are no clinical or pathological findings of Wernicke-Korsakoff Syndrome. Evidence of oxidative changes was also seen in all regions and effects of liver dysfunction in the vermis found. However, overall, these results highlight the complexity of this disease process in that a number of different proteins from different cellular pathways appear to be affected. By identifying changes in protein abundance levels in the prefrontal grey and white matter and the cerebellar vermis, hypotheses may draw upon more mechanistic explanations as to how chronic ethanol consumption causes the structural and functional alterations associated with alcohol-related brain damage. Furthermore, by comparing these results, we may be able to isolate disturbances in molecular pathways specific to the brain damage caused by alcohol, severe liver dysfunction and thiamine deficiency.
Simpson, Heather Jayne. "Transformation through adaptation : a grounded theory of the patient experience of Alcohol-Related Brain Damage". Thesis, University of Edinburgh, 2015. http://hdl.handle.net/1842/15671.
Pełny tekst źródłaHård, Julia. "Långvarigt bruk av alkohol ger kramper och epilepsi : Ett arbete om alkohols effekter på hjärnan". Thesis, Linnéuniversitetet, Institutionen för kemi och biomedicin (KOB), 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-65066.
Pełny tekst źródłaAlcohol has been used for drinking for many years and is a substance that is well known to most teenagers and adults. Most people also know that alcohol, when misused, can cause damage to both the liver and the kidneys but not as many people know about the damage alcohol can cause the brain. The damage that alcohol causes in the brain can lead to conditions where the patient can experience seizures, whitch can further devlop into epilepsy. Alcohol has different effects on the body. An immidiate response to alcohol is that the inhibitory signaling in the brain increases and the excitatory signaling decreases. When it comes to a prolonged misuse of alcohol the effects on the brain are the opposite and it can also increase the tolerance for alcohol. Inhibitory and excitatory signaling in the brain are essential and disturbance of those signals can be very damaging to the brain. The damages can develop and become permanent and it can also trigger different kinds of seizures. The seizures can in turn become very serious and fatal. Studies on the connection between alcohol and epilepsy has been conducted by Samokhvalov et al. (2010), Devetag et al. (1983), Bråthen et al. (1999), Tartara et al. (1983), Bartolomei et al. (1997), Victor och Brausch (1967) och Hillbom (1980) and have shown different results. The results however have shown a clear correlation between alcohol and epilepsi. In the study performed by Devetag et al. (1983) 58 % of 153 patients experienced seizures not related to alcohol withdrawl, alcohol induction or injury/disease. Of 60 patients who presented seizures in the study conducted by Bartolomei et al. (1997), 30 (50 %) had seizures not related to alcohol withdrawl, alcohol induction or injury/disease. A study performed by Bråthen et al. (1999) showed 16 patients (36 %) of 142 with seizures not related to alcohol withdrawl, alcohol indiction or injury/disease. Furthermore, a study conducted by Tartara et al. (1983) showed 30 patients with seizures, where 3 (10 %) of them were not related to alcohol withdrawl, alcohol induction or injury/disease. Seizures not related to alcohol withdrawl, alcohol abuse or injury/disease are difficult to investigate. Many scientists have tried to get insight in as to how alcohol can influence the ethiopathogenesis of epilepsy. What is alcohol-related seizures, what is the cause behind the seizures and how does one decide if the seizures can be defines as epilepsy. This literature review investigates the link between alcoholism and epilepsy to better understand this connection. The question of issue was ”if prolonged misuse of alcohol can lead to epilepsy” and to unravel the question, 7 studies were used. The studies main focus was alcoholism and seizures. The results from the studies indicated in total that alcohol prabably can cause epilepsy since none of the studies showed the opposite. A prolonged misuse of alcohol can lead to seizures and even epilepsy, but how this comes to be is not clear and needs to be properly investigated. Not to forget, some people who misuse alcohol do not get epilepsy and some never experience even a single seizure.
Cocks, Trevor J. "Screening for alcohol related brain damage among Australian aboriginals with drinking problems". Master's thesis, 1992. http://hdl.handle.net/1885/141424.
Pełny tekst źródłaKsiążki na temat "Alcohol-related brain damage"
D, Richter. Addiction & Brain Damage. Lippincott Williams & Wilkins Publishers, 1998.
Znajdź pełny tekst źródłaCzęści książek na temat "Alcohol-related brain damage"
Marshall, Jane. "Alcohol-related dementia (alcohol-induced dementia; alcohol-related brain damage)". W New Oxford Textbook of Psychiatry, 399–402. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199696758.003.0051.
Pełny tekst źródłaWilson, Ken. "Neuropsychiatry of alcohol-related brain damage". W Oxford Textbook of Neuropsychiatry, redaktorzy Niruj Agrawal, Rafey Faruqui i Mayur Bodani, 323–28. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198757139.003.0027.
Pełny tekst źródłaBrooks, PJ, JA Theruvathu i RG Nath. "DNA Damage Resulting from Alcohol Abuse with Special Reference to the Brain". W Comprehensive Handbook of Alcohol Related Pathology, 1049–67. Elsevier, 2005. http://dx.doi.org/10.1016/b978-012564370-2/50084-2.
Pełny tekst źródłaNunes, Polliana Toledo, Brian T. Kipp, Nicole L. Reitz i Lisa M. Savage. "Aging with alcohol-related brain damage: Critical brain circuits associated with cognitive dysfunction". W International Review of Neurobiology, 101–68. Elsevier, 2019. http://dx.doi.org/10.1016/bs.irn.2019.09.002.
Pełny tekst źródłavan den Brink, Wim, i Falk Kiefer. "Alcohol use disorder". W New Oxford Textbook of Psychiatry, redaktorzy John R. Geddes, Nancy C. Andreasen i Guy M. Goodwin, 498–506. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198713005.003.0050.
Pełny tekst źródłaLi, Jie Jack. "Drugs of the Mind". W Laughing Gas, Viagra, and Lipitor. Oxford University Press, 2006. http://dx.doi.org/10.1093/oso/9780195300994.003.0010.
Pełny tekst źródłaTaber, Douglass F. "New Methods for C-C Bond Construction". W Organic Synthesis. Oxford University Press, 2013. http://dx.doi.org/10.1093/oso/9780199965724.003.0023.
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