Gotowe bibliografie tematyczne / Age-Related

Gotowa bibliografia na temat „Age-Related”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 10 marca 2023

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Artykuły w czasopismach na temat "Age-Related"

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Ishaq, Aliya. "Age Related Disparities in Colorectal Cancer Patients". Journal of Clinical and Laboratory Research 3, nr 3 (11.09.2021): 01–04. http://dx.doi.org/10.31579/2768-0487/064.

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Background: There is an evident change in the colorectal cancer demographic over the period. This change is more marked in the age distribution and location of the tumor. It has practical implications, in regards to develop cancer awareness programs and screening protocols. Keeping in view that Pakistan is one of the countries with a high number of the young population this study is carried out to make a comparative analysis of this trend in our population. Material and methods: Colorectal cancer patients presented in Sindh Institute of urology and transplantation from January 2011 till December 2020 was reviewed retrospectively. All patients were divided into two groups, Group A young age population and Group B old age population. Subgroup analysis of study period was performed to check the progressive change in the trend of stage and clinical characteristics of colorectal cancer patients. Data reviewed from the patient’s files and collected as per Proforma requirement. Result: Total of 612 patients with colorectal cancer presented between 2011 till 2020.Among these patients 243 (39.7%) presented between January 2011 till December 2015. Patients age 50 years and younger were 410 (66.8%). Results showed a statistically significant association between and patient’s age and location of tumor such that left-sided colonic cancer and rectal cancer were more common in the young population. Subgroup analysis according to the study period showed that there is a change in the trend of disease presentation. Right-sided colonic cancer presentation decreased in the younger population over the period while simultaneously left-sided colonic cancer and rectal cancer presentation increased. Conclusion: The incidence of left-sided colonic and rectal cancer has been increased in the younger population over the specified period while there was no association between right-sided colon cancer and age noticed.
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Oh, Se-Joon, i Il-Woo Lee. "Age-Related Tinnitus". Journal of Clinical Otolaryngology Head and Neck Surgery 25, nr 1 (czerwiec 2014): 7–13. http://dx.doi.org/10.35420/jcohns.2014.25.1.7.

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Lubinski, Rosemary. "Age Related Glossary". Perspectives on Gerontology 7, nr 3 (grudzień 2002): 16. http://dx.doi.org/10.1044/gero7.3.16.

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SPROTT, RICHARD L. "Age-related Variability". Annals of the New York Academy of Sciences 515, nr 1 Central Deter (styczeń 1988): 121–23. http://dx.doi.org/10.1111/j.1749-6632.1988.tb32974.x.

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Forbus, Sara B. "Age-Related Infertility". AWHONN Lifelines 9, nr 2 (kwiecień 2005): 126–32. http://dx.doi.org/10.1177/1091592305277051.

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Gillespie, Lorna M., i Jonathan Wyllie. "Age-related physiology". Anaesthesia & Intensive Care Medicine 6, nr 3 (marzec 2005): 84–88. http://dx.doi.org/10.1383/anes.6.3.84.62218.

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Bell, Sarah, i Diane Monkhouse. "Age-related pharmacology". Anaesthesia & Intensive Care Medicine 6, nr 3 (marzec 2005): 89–92. http://dx.doi.org/10.1383/anes.6.3.89.62227.

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Sivak, J. G. "AGE-RELATED CATARACT". Optometry and Vision Science 68, nr 6 (czerwiec 1991): 480–81. http://dx.doi.org/10.1097/00006324-199106000-00017.

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Matthews, Michelle L. "Age-Related Infertility". Postgraduate Obstetrics & Gynecology 29, nr 5 (marzec 2009): 1–7. http://dx.doi.org/10.1097/01.pgo.0000345587.71775.9d.

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&NA;. "Age-Related Infertility". Postgraduate Obstetrics & Gynecology 29, nr 5 (marzec 2009): 8. http://dx.doi.org/10.1097/01.pgo.0000345588.79398.fe.

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Rozprawy doktorskie na temat "Age-Related"

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Dark-Freudeman, Alissa. "Memory-related possible selves exploring age-related differences /". [Gainesville, Fla.] : University of Florida, 2004. http://purl.fcla.edu/fcla/etd/UFE0005642.

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van, Otterdijk Sanne Dorien. "The role of age-related DNA methylation in the development of age-related disease". Thesis, University of Newcastle upon Tyne, 2013. http://hdl.handle.net/10443/2337.

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Alterations in DNA methylation can have dramatic effects on gene transcription, and in particular, hypermethylation of promoter associated CpG islands is known to lead to gene inactivation. Altered patterns of DNA methylation play a key role in the development of cancer and may also play important roles in many other diseases. However, the mechanisms which lead to these changes in DNA methylation are unknown. DNA methylation patterns have also been found to change during normal ageing and these changes have similarities to those that occur during the development of cancer. This suggests that for some age-related diseases, most notably cancer, altered patterns of methylation may be an early initiating event and that disease may develop in cells which already possess changes in their DNA methylation landscape. Therefore, this study was designed to examine how methylation levels at a group of genes alters over the life-course and how these relate to methylation changes observed in major age-related diseases (cancer, specifically acute lymphoblastic leukaemia (ALL) and Hereditary Nonpolyposis Colorectal Cancer (HNPCC) patients, and atherosclerosis). DNA was collected from healthy volunteers from different ages and from ALL, HNPCC and atherosclerosis patients. Methylation was quantified using pyrosequencing. The study produced a number of findings: 1) Genes exhibiting variable methylation in PBL samples from healthy volunteers are also highly methylated in leukaemia, suggesting a common underlying mechanism. 2) Increased methylation levels were observed in lymphoid compared to myeloid cells, in healthy individuals, mirroring the patterns seen in leukaemia. 3) A subset of genes exhibiting variable methylation in PBL samples from healthy volunteers and that are highly methylated in leukaemia are aberrantly methylated in HNPCC patients and atherosclerosis patients, suggesting shared risk factors. 4) While methylation levels increase during ageing, a substantial proportion of methylation is already present at birth and may thus alter disease susceptibility throughout life. 5) Blood samples from ALL patients in remission exhibit increased methylation levels (versus controls), not directly related to their leukaemic clone, and the extent of methylation correlates with overall survival. The studies to date are compatible with a hypothesis in which altered methylation of disease-related genes pre-exists in a subset of haematopoietic cells and that these cells may be at a significantly increased risk of progression to age-related diseases. Furthermore, monitoring DNA methylation may be a valuable tool for early diagnosis of these diseases, as well as for monitoring disease progression in patients.
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Patil, Parag Ramchandra. "Age related effects of SSRIs". Thesis, University of Nottingham, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.442260.

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Koszyca, Barbara. "Age-related changes within the knee /". Title page, contents and abstract only, 1992. http://web4.library.adelaide.edu.au/theses/09PH/09phk86.pdf.

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Thesis (Ph. D.)--University of Adelaide, Dept. of Pathology, 1993.
Attempts to understand the effects of ageing on the condition of a synovial joint. Knee joints of individuals with no known history of joint disease were examined and the pattern of cartilage damage was mapped macroscopically in a manner that allowed quantitation of the affected areas. Includes bibliographical references (leaves 267-277).
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Despriet, Dominiek Denise Gasparine. "Genetics of age-related macular degenreation". [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2008. http://hdl.handle.net/1765/11512.

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Aydin, Senay. "Age-related deficits in perceptual stability". Thesis, Glasgow Caledonian University, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555684.

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The world's population is ageing, which represents challenges and opportunities concerning different aspects of life. Some older people exhibit various perceptual and cognitive declines, which are related to their inability to construct a stable representation of the external world, it being usually ambiguous and degraded. These declines require thorough research in order to understand their cortical mechanisms. Changes in perceptual stability as a function of ageing were explored in this PhD work using methods of psychophysics, visual evoked potentials, and eye- movement recordings. Specifically studied were attentional control in perceptual rivalry, the ability to construct a stable and efficient internal representation of degraded words and finally, saccadic suppression as an important contributory factor to the underlying perceptual stability of the visual world during saccadic eye movements. Perceptual rivalry, produced by an ambiguous Rubin vase-faces figure, was slower in older people during passive viewing than in young participants. Additionally, older adults, in contrast to young adults, were unable to hold the dominant percept longer than in passive viewing. Using current models of perceptual rivalry and studies of age-related changes in cortical activity, the increased response gain in older adults might be a possible factor leading to prolonged dominance durations in older people during passive viewing as well as impaired ability to hold the dominant percept longer. Visual evoked potentials, elicited by frequency-tagged stimulation, showed that perceptual rivalry under passive viewing caused in both age group suppression of visual evoked potentials to a contrast-modulated pattern. This finding is only true for the vase percept, when the vase percept was not dominant. Thus, the mechanism, underlying perceptual rivalry under passive viewing in early visual cortical areas, could be inhibition of neuronal populations associated with the non-dominant percept, rather than excitation of neuronal populations, corresponding to the dominant percept. Attentional control of holding the dominant percept caused an enhancement of the visual evoked potentials, representing the dominant percept, in young subjects but not in older people. Holding and switching the dominant percept also produced enhancement of gamma activity, recorded with electrodes over the occipital and parietal cortices, only in young adults. These results could be related to age-related deficits in top-down modulated attentional and figure-ground segregation processes. A novel paradigm for recognising words embedded in positional letter noise and a model for estimating internal noise components were developed. Older people showed noise-exclusion deficits when reading normal words and reversed words, but not when reading nonwords. These deficits suggest that older people may experience perceptual instability of words due to inefficient noisy perceptual representations of words. This method has the potential for use as a tool in the clinical testing of noise- exclusion deficits in normal and pathological ageing and in children with reading dysfunctions. Investigation of age-related changes in saccadic suppression revealed for the first time that older adults experienced reduced and delayed saccadic suppression compared to young adults. This could result in perceiving instability of the external world and thus affect their mobility and performance in everyday environments. The results of this thesis provide evidence of age-related deficits in the ability of older people to voluntarily perceive a stable dominant percept during perceptual rivalry, to efficiently exclude positional noise during reading degraded words and to suppress visual information during saccadic eye movements. Although these deficits involve different cortical mechanisms, the underlying cause for them could be related to impairments in inhibitory mechanisms at various cortical processing stages.
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Scott, Jenny Louise. "Age-related changes in osteoarthritic chondrocytes". Thesis, University of Newcastle Upon Tyne, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.501074.

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Age is the most important risk factor in the development of osteoarthritis (OA). Numerous studies have now demonstrated that age-related changes occur in both the extracellular matrix and chondrocytes of OA cartilage and such changes are proposed to alter the tissue's biomechanical properties, thus predisposing to OA onset. The study presented here describes further investigation of two such age-related changes, cellular senescence and oxidative stress.
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Badham, S. P. "Age-related changes in associative memory". Thesis, University of Warwick, 2011. http://wrap.warwick.ac.uk/50837/.

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Older adults suffer from many cognitive impairments relative to young adults and one of the most established types of age-related cognitive decline is a reduction in memory performance. Memory for single units of information (item memory) have been shown to be less susceptible to cognitive ageing than memory for associations among units of information (associative memory). An associative deficit hypothesis has been used to describe these observations as an age-related impairment in forming links between single units of information. The thesis elucidated specific differences between item and associative memory and evaluated how such differences correspond to their differential susceptibility to the effects of cognitive ageing. This indicated links between the associative deficit hypothesis and other theories of age-related memory decline, in particular, to the notion of age deficits in memory resulting from age deficits in self-initiated processing (in the absence of environmental support). Experiments 1-3 considered associative memory where the processing of associations was encouraged by distinctiveness of memory stimuli. Environmental support provided by distinctiveness was shown to improve associative memory in older adults. Experiments 4-7 considered how item and associative memory differ in their support from preexisting knowledge. Experimentally equating preexisting knowledge for item and associative memory tests eliminated the age-related associative deficit. Furthermore, it was found that preexisting knowledge could be used to enhance associative memory performance in older adults by providing support to encoding and/or retrieval processes. Experiment 8 established that item and associative memory processes were equally disrupted by a concurrent task, which indicated that both memory types are similarly affected by levels of available cognitive resources. In general, age-related associative deficits were considered to result from differing levels of environmental support for item and associative memory as opposed to a differential decline of item and associative memory processes.
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Gu, Jiayin. "Biomarkers for Age-Related Macular Degeneration". Case Western Reserve University School of Graduate Studies / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=case1225388164.

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Feigl, Beatrix Karoline. "Age-related Maculopathy: A Multifocal Approach". Thesis, Queensland University of Technology, 2005. https://eprints.qut.edu.au/16026/1/Beatrix_Feigl_Thesis.pdf.

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Age-related maculopathy (ARM) is a central retinal disease with unclear pathogenesis. It is the major cause of permanent vision loss in adults over 50 years and is increasing in prevalence and incidence, faster than the aging population would suggest. Early in the disease process (early ARM) there is little or no vision loss and there are only slight retinal changes with abnormal deposits within Bruch's membrane. As the disease progresses (late ARM or age-related macular degeneration, AMD) vision loss may be quite severe due to atrophy (dry AMD) or the development of chorioretinal neovascularisation (CNV, wet AMD). It is hard to predict from conventional eye examinations and clinical vision tests which cases will progress to the severe, dry or wet forms of the disease. Moreover, most of the conventional clinical tests are based upon subjective vision measures. Objective tests which detect ARM earlier would be a useful aid to diagnosis and to monitoring progression. The multifocal electroretinogram (mfERG) is a relatively new clinical tool which enables the recording of electrical potentials from multiple, small areas of the central retina and thus assesses function from specific retinal locations. It is therefore useful in detecting focal retinal diseases such as hereditary or acquired maculopathies or in monitoring retinal laser or surgical treatment effects. There is cone and rod impairment in ARM and histopathological and psychophysical evidence for a preferential vulnerability of rods compared to cones. This research project investigated if an objective tool such as the mfERG could detect early ARM,its progression and the treatment effects of multiple photodynamic therapies (PDT) on retinal function in late ARM, prior to a battery of subjective vision measures. For comparison purposes a subjective assessment of central retinal function was performed using high and low contrast distance visual acuities (VA), near VA, low luminance VA (SKILL cards), contrast sensitivity (Pelli-Robson, P-R), saturated and desaturated Panel D-15 (sat Panel D-15, desat Panel D-15) and central visual fields (Humphrey 10-2, mean sensitivity, MS and mean defects, MD). As an objective assessment of central retinal function the cone- and rod-mediated multifocal electroretinograms were recorded. Subjective and objective tests of retinal function were compared in early ARM and an age-matched control group (chapter 3). Seventeen eyes of seventeen subjects with early ARM and twenty control subjects with normal vision were measured. For the cone-mediated mfERG responses conventional averaging methods were used and results were correlated with subjective vision tests. The conventional cone-mediated mfERG failed to distinguish between the early ARM and control subjects whereas subjective vision measures such as HC- and LC-VA, desat Panel D-15, MS, P-R were significantly reduced in the ARM group. However, there were significant correlations between the cone-mediated mfERG and the desat Panel D-15 results in the ARM group. This suggests that the mfERG measures similar retinal processes that detect colour vision deficiency under desaturated conditions. There was no significant correlation between cone-mediated mfERG measures and funduscopic changes. The conclusion from this study was that the subjective vision tests detected early ARM better than the objective cone-mediated mfERG. Thus the aim of detecting early ARM objectively was not met by the cone-mediated mfERG suggesting the need to develop other objective tests such as a rod-mediated mfERG. Whether the preferential rod vulnerability others have reported in early ARM could be detected by the rod-mediated mfERG was determined in the next study (chapter 4). A protocol for recording rod-mediated mfERG responses was developed by determining the optimal testing luminance to reduce the effect of stray light and elicit maximal rod-mediated responses. Sixteen of the seventeen ARM subjects and seventeen control subjects from the previous study were tested. For analysis, a customized computer template fitting method was developed in MATLAB (Mathworks, Natick, MA, USA). This method has been shown to be useful for low signal-to-noise ratio responses that characterize the rod-mediated mfERG. Significantly delayed rod-mediated mfERG responses were found whereas cone-mediated mfERG responses were within the normal range. This suggested that the effect of ARM on the rod system could be detected objectively with the rod-mediated mfERG before changes in the cone-mediated mfERG. Which of the tests best detected progression of vision loss was investigated in chapter 5. Visual function of 26 (13 ARM and 13 control subjects) of the original 37 subjects (17 ARM and 20 control subjects) had cone- and rod-mediated mfERG and the subjective vision measures repeated after one year. The main purpose was to determine which of the tests best detected progression of vision loss. The mfERG results were analysed by using both averaged and local responses and by using the computer template fitting procedure. On average no significant worsening of either objective or subjective function measures was evident after one year. These results reinforce the slow progression of the disease. With a longer follow-up period progression of ARM may translate into measurable changes in the mfERG and the other visual function tests. The effect of multiple photodynamic therapies (PDT) on cone- and rod-mediated function was assessed with the mfERG in the last study (chapter 6). The cumulative treatment effects of PDT in five subjects with late ARM were determined. Having demonstrated that the rod-mediated mfERG was applicable in early ARM, this study also aimed to investigate how useful it was in late ARM where there is substantially greater rod loss. Cone- and rod-mediated mfERGs, visual acuities, contrast sensitivities and central visual fields were investigated a week before treatment began and then one month after each PDT treatment. The subjects received three treatments each over an average period of five and a half months. In some subjects there were significant transient reductions in cone- and rod-mediated amplitudes possibly reflecting alterations in choroidal hypoperfusion dynamics one month after treatment. Further, b-wave component of the mfERG became increasingly misshapen after each PDT treatment suggesting an ischemic insult mainly targeting post-receptoral sites. However, objective and subjective function was stabilized after multiple PDT treatments in most of the subjects. This pilot study of five cases showed that there was no additional damage to cone- and rod-mediated outer retinal function after three PDT treatments. One of the novel findings of this research was that the rod-mediated function measured with the mfERG was impaired in early ARM. This finding supports histopathological and psychophysical evidence of rod vulnerability in early ARM. The results of these studies also suggest that early ARM affects different aspects of visual function which is reflected by different outcomes from objective and subjective vision tests. A model (chapter 7) based upon the results was developed proposing a hypoxic insult with a preferential alteration of post-receptoral sites in early ARM. The cone-mediated mfERG documented the retinal damage and possible treatment effects on outer retinal function of the multiple PDTs which did not further deteriorate. Thus, this technique might assist in the development of optimal treatment modalities for ARM, especially in retreatment regimes. Greater variability was found for the rod-mediated mfERG and its clinical use in PDT treatment regimes still needs to be investigated. In conclusion, this research has provided a better understanding of the disease process and treatment effects in ARM and might contribute to improvements in diagnosis and treatment of ARM.
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Książki na temat "Age-Related"

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Age-related cataract. New York: Oxford University Press, 1991.

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Chew, Emily Y., i Anand Swaroop, red. Age-related Macular Degeneration. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-66014-7.

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Holz, Frank G., Daniel Pauleikhoff, Richard F. Spaide i Alan C. Bird, red. Age-related Macular Degeneration. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-22107-1.

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Wykoff, Charles, Mariam Hussain, T. Ashwini Kini i Bayan Al Othman. Age-Related Macular Degeneration. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-36973-6.

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Alberti, Winfried E., Gisbert Richard i Robert H. Sagerman, red. Age-Related Macular Degeneration. Berlin, Heidelberg: Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-56439-0.

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Holz, Frank G., Daniel Pauleikhoff, Richard F. Spaide i Alan C. Bird. Age-related macular degeneration. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-662-05199-3.

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Windom, Robert Emerson. Age related macular degeneration. Bethesda, Md: U.S. Dept. of Health and Human Services, U.S. Public Health Service, 1988.

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National Institutes of Health (U.S.), red. Age-related macular degeneration. Bethesda, Md: National Institutes of Health, 1988.

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G, Holz Frank, red. Age-related macular degeneration. Berlin: Springer, 2004.

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National Institutes of Health (U.S.), red. Age-related macular degeneration. Bethesda, Md: National Institutes of Health, 1988.

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Części książek na temat "Age-Related"

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Buckles, Virginia Diggles. "Age-Related Slowing". W Sensorimotor Impairment in the Elderly, 73–87. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-1976-4_6.

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Ahmed, Touqeer, Abida Zulfiqar i Sara Ishaq. "Age-Related Diseases". W Nutrients and Nutraceuticals for Active & Healthy Ageing, 27–51. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-3552-9_3.

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Mehlhorn, Heinz. "Age-Related Prevalence". W Encyclopedia of Parasitology, 69. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_88.

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Esposito, Irene, i Lena Häberle. "Age-Related Changes". W Pathology of the Pancreas, 17–19. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-62416-3_5523.

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Esposito, Irene, i Lena Haeberle. "Age-Related Changes". W Encyclopedia of Pathology, 1–3. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-28845-1_5523-1.

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Szalma, Ivett. "Age-Related Dilemmas". W essentials, 27–30. Wiesbaden: Springer Fachmedien Wiesbaden, 2021. http://dx.doi.org/10.1007/978-3-658-35628-6_6.

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Scharnagl, Hubert, Winfried März, Markus Böhm, Thomas A. Luger, Federico Fracassi, Alessia Diana, Thomas Frieling i in. "Age-related Maculopathy". W Encyclopedia of Molecular Mechanisms of Disease, 49. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_6149.

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Mehlhorn, Heinz. "Age Related Prevalence". W Encyclopedia of Parasitology, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_88-2.

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Dysli, Chantal, i Lydia Sauer. "Age-Related Macular Degeneration". W Fluorescence Lifetime Imaging Ophthalmoscopy, 57–64. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-22878-1_10.

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Rabina, Gilad, i Anat Loewenstein. "Age-Related Macular Degeneration". W Encyclopedia of Ophthalmology, 1–6. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-35951-4_984-1.

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Streszczenia konferencji na temat "Age-Related"

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Mayer, Melanie J., Brian Ward, Ronald Klein, Joel B. Talcott i Robert F. Dougherty. "Predicting Exudative Age-Related Maculopathy". W Noninvasive Assessment of the Visual System. Washington, D.C.: Optica Publishing Group, 1993. http://dx.doi.org/10.1364/navs.1993.nmb.6.

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Exudative age-related maculopathy (ARM) is the leading cause of vision loss in the United States among the elderly.1,2 The goal of our research is to identify those most at risk for exudative ARM before the development of exudative and neovascular complications and before severe loss of acuity. Those identified as "Most At Risk" could monitor their vision carefully for visual distortion or loss or for further flicker sensitivity loss, allowing early clinical evaluation and possible treatment should exudative symptoms appear. Predictors of exudative ARM could also provide information about the progress and rate of development of pre-exudative stages of ARM and could help to evaluate proposed treatments for the earlier stages of ARM.
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Cherenkova, Ludmila, i Lyudmila Sokolova. "AGE-RELATED CHANGES IN THE VISUAL PRIMING IN PRESCHOOL AGE". W XVI International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2020. http://dx.doi.org/10.29003/m1327.sudak.ns2020-16/507.

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Harutyunyan, Eline, A. Aghajanyan, L. Shahumyan, A. Asatryan, H. Shahumyan, S. Khachatryan, I. Atoyan i in. "333 Age-related disorders in children". W 10th Europaediatrics Congress, Zagreb, Croatia, 7–9 October 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-europaediatrics.333.

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Wuertz-Kozak, Karin. "Age-Related Changes of the Spine". W eccElearning Postgraduate Diploma in Spine Surgery. eccElearning, 2017. http://dx.doi.org/10.28962/01.3.004.

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Chayalo, P., i B. Grubnik. "Quantum Medicine and Age-Related Pathology". W 2006 16th International Crimean Microwave and Telecommunication Technology. IEEE, 2006. http://dx.doi.org/10.1109/crmico.2006.256249.

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Brown, Brian, i Jan Lovie-Kitchin. "Temporal function in age related maculopathy". W Noninvasive Assessment of the Visual System. Washington, D.C.: Optica Publishing Group, 1988. http://dx.doi.org/10.1364/navs.1988.tub3.

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Sensitivity to temporal change in red light in elderly patients with age related maculopathy (ARM) is depressed across a wide range of temporal frequencies compared to elderly control subjects. Patients with normal acuity and pigmentary changes and/or drusen at the macula (pre-ARM) have normal temporal sensitivity.
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Shan, G., i X. Zhang. "Development of a novel platform for quantifying age-related sensori-motor degradation to control age-related falls". W BIOMED 2013. Southampton, UK: WIT Press, 2013. http://dx.doi.org/10.2495/bio130181.

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Niksirat, Kavous Salehzadeh, Chaklam Silpasuwanchai, Zhenxin Wang, Jing Fan i Xiangshi Ren. "Age-Related Differences in Gross Motor Skills". W the International Symposium. New York, New York, USA: ACM Press, 2016. http://dx.doi.org/10.1145/2996267.2996278.

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Nikoloeva, Elena. "Age-Related Effects In Retrieval-Induced Forgetting". W ICPE 2018 - International Conference on Psychology and Education. Cognitive-Crcs, 2018. http://dx.doi.org/10.15405/epsbs.2018.11.02.64.

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Díaz, Mark, Isaac Johnson, Amanda Lazar, Anne Marie Piper i Darren Gergle. "Addressing Age-Related Bias in Sentiment Analysis". W Twenty-Eighth International Joint Conference on Artificial Intelligence {IJCAI-19}. California: International Joint Conferences on Artificial Intelligence Organization, 2019. http://dx.doi.org/10.24963/ijcai.2019/852.

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Recent studies have identified various forms of bias in language-based models, raising concerns about the risk of propagating social biases against certain groups based on sociodemographic factors (e.g., gender, race, geography). In this study, we analyze the treatment of age-related terms across 15 sentiment analysis models and 10 widely-used GloVe word embeddings and attempt to alleviate bias through a method of processing model training data. Our results show significant age bias is encoded in the outputs of many sentiment analysis algorithms and word embeddings, and we can alleviate this bias by manipulating training data.
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Raporty organizacyjne na temat "Age-Related"

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Subudhi, M., W. Shier i E. MacDougall. Age-related degradation of Westinghouse 480-volt circuit breakers. Office of Scientific and Technical Information (OSTI), lipiec 1990. http://dx.doi.org/10.2172/7028755.

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Jaganathan Geethalakshmi, Kruthica, Iyshwarya Bhaskar Kalarani i Ramakrishnan Veerabathiran. Stem cell technology to cure age-related macular degeneration (AMD). Peeref, listopad 2022. http://dx.doi.org/10.54985/peeref.2211p4104893.

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Davis, W. J., R. R. Parrish, J. C. Roddick i L. M. Heaman. Isotopic age determinations of kimberlites and related rocks: methods and applications. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1996. http://dx.doi.org/10.4095/210936.

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Nie, J., J. Braverman, C. Hofmayer, Y.-S. Choun, MK Kim i I.-K. Choi. Seismic Fragility Analysis of a Condensate Storage Tank with Age-Related Degradations. Office of Scientific and Technical Information (OSTI), kwiecień 2011. http://dx.doi.org/10.2172/1169547.

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Liu, Yunjie, Mengmei Ni, Rui Wu, Zhirui Yang, Xuejiao Zhu i Jinyao Chen. Lutein and age-related macular degeneration: a comprehensive systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, listopad 2021. http://dx.doi.org/10.37766/inplasy2021.11.0091.

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Donohue, Henry J., Christopher Niyibizi i Alayna Loiselle. Induced Pluripotent Stem Cell Derived Mesenchymal Stem Cells for Attenuating Age-Related Bone Loss. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2013. http://dx.doi.org/10.21236/ada606237.

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Donahue, Henry J. Induced Pluripotent Stem Cell Derived Mesenchymal Stem Cells for Attenuating Age-Related Bone Loss. Fort Belvoir, VA: Defense Technical Information Center, lipiec 2012. http://dx.doi.org/10.21236/ada581680.

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Canizio, Nancy. A study of age and sex-related differences in the perception of emotional stimuli. Portland State University Library, styczeń 2000. http://dx.doi.org/10.15760/etd.3142.

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Hu, P. S., J. R. Young i An Lu. Highway crash rates and age-related driver limitations: Literature review and evaluation of data bases. Office of Scientific and Technical Information (OSTI), sierpień 1993. http://dx.doi.org/10.2172/10149328.

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Kane, M.D., Robert L., Mary Butler, Ph.D., M.B.A. i Howard A. Fink, M.D., M.P.H. Interventions To Prevent Age-Related Cognitive Decline, Mild Cognitive Impairment, and Clinical Alzheimer’s-Type Dementia. Agency for Healthcare Research and Quality (AHRQ), 2017. http://dx.doi.org/10.23970/ahrqepccer188.

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