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1

Hoepner, Lori A. Bisphenol A Exposure, Adipogenic Mechanism and Effect on Childhood Adiposity. [New York, N.Y.?]: [publisher not identified], 2015.

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Ashby-Thompson, Maxine. Maternal Diet Quality as a Predictor of Child Adiposity Among New York City Head Start Families. [New York, N.Y.?]: [publisher not identified], 2019.

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Wirth, Alfred. Adipositas. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-662-05601-1.

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Wirth, Alfred. Adipositas. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-662-05603-5.

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Wirth, Alfred, i Hans Hauner, red. Adipositas. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-22855-1.

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Wirth, Alfred. Adipositas-Fibel. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-18252-5.

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Wirth, Alfred. Adipositas-Fibel. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-97741-1.

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Toplak, Hermann, red. Praxishandbuch Adipositas. Vienna: Springer Vienna, 2002. http://dx.doi.org/10.1007/978-3-7091-6746-5.

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Humer, Elke Carina. Psychotherapie bei Adipositas. Wiesbaden: Springer Fachmedien Wiesbaden, 2019. http://dx.doi.org/10.1007/978-3-658-27539-6.

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Herpertz, Stephan, Martina de Zwaan i Stephan Zipfel, red. Handbuch Essstörungen und Adipositas. Berlin, Heidelberg: Springer Berlin Heidelberg, 2022. http://dx.doi.org/10.1007/978-3-662-63544-5.

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11

Ordemann, Jürgen, i Ulf Elbelt, red. Adipositas- und metabolische Chirurgie. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-48698-6.

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Herpertz, Stephan, Martina de Zwaan i Stephan Zipfel, red. Handbuch Essstörungen und Adipositas. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-76882-1.

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Herpertz, Stephan, Martina Zwaan i Stephan Zipfel, red. Handbuch Essstörungen und Adipositas. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-54573-3.

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14

Hannover, Akademie für Sozialmedizin, Wissenschaftliches Institut der Ortskrankenkassen (Bonn, Germany) i Seminar "Stoffwechselerkrankungen ", red. Stoffwechselerkrankungen: Diabetes, Gicht, Adipositas. Bonn: Das Institut, 1985.

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15

Horstmann, Rüdiger. Raus aus der Adipositas. Berlin, Heidelberg: Springer Berlin Heidelberg, 2022. http://dx.doi.org/10.1007/978-3-662-65808-6.

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16

Strauss, Alexander, i Carolin Strauss, red. Praxisbuch Adipositas in der Geburtshilfe. Berlin, Heidelberg: Springer Berlin Heidelberg, 2022. http://dx.doi.org/10.1007/978-3-662-61906-3.

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17

Wabitsch, Martin, Johannes Hebebrand, Wieland Kiess, Thomas Reinehr i Susanna Wiegand, red. Adipositas bei Kindern und Jugendlichen. Berlin, Heidelberg: Springer Berlin Heidelberg, 2022. http://dx.doi.org/10.1007/978-3-662-59216-8.

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18

de Zwaan, Martina, Stephan Herpertz i Stephan Zipfel, red. Psychosoziale Aspekte der Adipositas-Chirurgie. Berlin, Heidelberg: Springer Berlin Heidelberg, 2022. http://dx.doi.org/10.1007/978-3-662-65556-6.

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19

de Zwaan, Martina, Stephan Herpertz i Stephan Zipfel, red. Psychosoziale Aspekte der Adipositas-Chirurgie. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-57364-8.

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20

Wabitsch, Martin, Wieland Kiess, Johannes Hebebrand i Karl Zwiauer, red. Adipositas bei Kindern und Jugendlichen. Berlin, Heidelberg: Springer Berlin Heidelberg, 2005. http://dx.doi.org/10.1007/b138048.

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21

Danne, Thomas, i Olga Kordonouri, red. Adipositas, Diabetes und Fettstoffwechselstörungen im Kindesalter. Berlin, Boston: De Gruyter, 2016. http://dx.doi.org/10.1515/9783110460056.

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22

Schorb, Friedrich. Die Adipositas-Epidemie als politisches Problem. Wiesbaden: Springer Fachmedien Wiesbaden, 2015. http://dx.doi.org/10.1007/978-3-658-06614-7.

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23

Stier, Christine, i Sonja Chiappetta, red. Interdisziplinäre Langzeitbehandlung der Adipositas- und Metabolischen Chirurgie. Berlin, Heidelberg: Springer Berlin Heidelberg, 2022. http://dx.doi.org/10.1007/978-3-662-63705-0.

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24

Zwick, Michael M., Jürgen Deuschle i Ortwin Renn, red. Übergewicht und Adipositas bei Kindern und Jugendlichen. Wiesbaden: VS Verlag für Sozialwissenschaften, 2011. http://dx.doi.org/10.1007/978-3-531-93158-6.

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25

Herpertz, Stephan. Handbuch Essstörungen und Adipositas: Mit 21 Tabellen. Heidelberg: Springer, 2008.

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Zwick, Michael M. U bergewicht und Adipositas bei Kindern und Jugendlichen. Wiesbaden: VS Verlag fu r Sozialwissenschaften, 2011.

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Gordeladze, Jan Oxholm, red. Adiposity - Epidemiology and Treatment Modalities. InTech, 2017. http://dx.doi.org/10.5772/63121.

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Riccardi, Gabriele, i Maria Masulli. Overweight, obesity, and abdominal adiposity. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656653.003.0013.

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Streszczenie:
Obesity is a serious chronic disease of epidemic and global proportions. The incidence of cardiovascular diseases (CVD) is increased in obese people. Since overweight and obesity are associated with decreased lifespan, weight loss might be expected to improve long-term survival and to have beneficial effects on CVD risk. The therapeutic approaches for obesity are lifestyle changes, drugs, and bariatric surgery. Lifestyle modifications include modest weight loss and moderate-intensity physical activity. A low-fat (low saturated fat), low-sugar diet rich in fruit and vegetables, as well as legumes and whole grains, should be advised for its beneficial impact on weight and cardiovascular risk. Bariatric surgery represents an effective treatment in cases of severe obesity. Prevention of overweight and obesity at the population level will probably play a major role in combating the present obesity epidemic. Combining different intervention strategies is probably the best choice for maximizing the effects and minimizing the costs.
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29

Gordeladze, Jan Oxholm, red. Adiposity - Omics and Molecular Understanding. InTech, 2017. http://dx.doi.org/10.5772/67422.

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30

Shaw, Max. Evaluation and Management of Adiposity. HAYLE MEDICAL, 2020.

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31

Churchill, Jamie. Evaluation and Management of Adiposity. Murphy & Moore Publishing, 2022.

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32

Hurley, Daniel L., J. Michael Gonzalez-Campoy i W. Timothy Garvey. Bariatric Endocrinology: Evaluation and Management of Adiposity, Adiposopathy and Related Diseases. Springer, 2019.

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Bariatric Endocrinology: Evaluation and Management of Adiposity, Adiposopathy and Related Diseases. Springer, 2018.

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34

Bravenboer, Nathalie, Stephanie Lucas i Clifford James Rosen, red. Bone Marrow Adiposity: Establishing Harmonized, Mechanistic and Multidisciplinary Approaches to Reach Clinical Translation. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88966-283-8.

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35

Ward, Heather. The interrelationships of education, income, lifestyle factors, and adiposity in the Ontario Food Survey. 2005.

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36

Wolfgang, Langhans, i Geary Nori, red. Frontiers in eating and weight regulation. Basel: Karger, 2010.

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37

Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng i Anne Bardsley. Polyunsaturated fatty acids in pregnancy and breastfeeding. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0005.

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Evidence for the importance of the long-chain omega-3 polyunsaturated fatty acids in fetal and infant development is growing, as is interest in what constitutes an appropriate intake from sources such as oily fish or dietary supplements for pregnant women and/or infants. Polyunsaturated fatty acids have been implicated in maternal mental health and aspects of infant development, including cognitive and visual function, adiposity, and allergy. Western diets have become imbalanced with regard to the ratio of omega-6:omega-3 fatty acids, and recommendations to correct this imbalance include increasing the maternal intake of oily fish. However, this recommendation needs to be evaluated in light of the increased risk of exposure to contaminants such as mercury. Vegetable oils and cereals are important sources of polyunsaturated fatty acids for vegetarians.
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38

Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng i Anne Bardsley. Macronutrients and fibre requirements during pregnancy. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0004.

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In this chapter, the impact of varying intakes of protein, carbohydrate and lipids, which are the key nutrients that contribute to calorie intake, is examined. Fibre is also an important food component that needs to be considered. The maternal macronutrient profile can influence embryonic and fetal development. For instance, both low and excessively high protein intakes during pregnancy are associated with restricted growth, increased adiposity, and impaired glucose tolerance. High-fat maternal diets can significantly increase the susceptibility to diet-induced obesity and percentage total body fat in offspring, although types of fats need to be considered, as intake of polyunsaturated fatty acids is important for fetal development. The type and content of carbohydrate (high- vs low-glycaemic sources) in the maternal diet influences blood glucose concentration, which has a direct effect on fetal glucose levels and metabolism.
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39

Ruiz-Hermosa, Abel, Mairena Sánchez-López, Vicente Martínez-Vizcaíno i Andrés Redondo-Tébar. MOVI-da FIT!: An After-School Program of Physical Activity Based on the HIIT Methodology to Prevent Obesity and Improve Physical Fitness and Academic Performance. Ediciones de la Universidad de Castilla-La Mancha, 2021. http://dx.doi.org/10.18239/atenea_2021.29.00.

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Numerous studies have shown the positive effects of physical activity (PA) on schoolchildren’s physical, psychological and social health. However, children’s levels of physical inactivity and sedentary lifestyle have increased significantly in recent years, leading to a rise in childhood obesity and cardiovascular disease. The MOVI programmes to promote PA in school settings were launched with the aim of mitigating this public health problem. The latest edition implemented by the research group at Centro de Estudios Sociosanitarios analysed the e􀇱fectiveness of an extracurricular PA programme (MOVI-da FIT!) based on high intensity interval training (HIIT) in enhancing schoolchildren’s executive functioning, academic performance, physical fitness and in reducing adiposity and cardiometabolic risk in schoolchildren. This book is intended for professionals in education, health care and sports and PA sciences who are keen to develop and implement PA interventions aimed at preventin
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40

Westman, Eric C., Emily Maguire i William S. Yancy. Ketogenic Diets as Highly Effective Treatments for Diabetes Mellitus and Obesity. Redaktor Dominic P. D’Agostino. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0037.

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Obesity and type 2 diabetes mellitus (T2DM) have reached epidemic proportions worldwide. While characterized by chronic hyperglycemia, the underlying cause of T2DM is insulin resistance—most often related to an increase in abdominal adiposity caused by obesity. The goal of treatment of T2DM is to put the disease into remission by targeting the underlying insulin resistance. The observation that dietary carbohydrate is the major factor to cause glycosuria and hyperglycemia, has been known since the early days of modern medicine. As a result, low-carbohydrate, ketogenic diets were employed to treat obesity and diabetes in the nineteenth and early twentieth centuries. This chapter reviews the rationale and recent clinical research supporting the use of a low-carbohydrate, ketogenic diet in individuals with obesity and diabetes. For individuals affected by obesity-related T2DM, clinical studies have shown that carbohydrate restriction and weight loss can improve hyperglycemia, obesity, and T2DM.
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41

Chen, Mimi, i Rob Andrews. Obesity: epidemiology, prevention and management. Redaktorzy Patrick Davey i David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0336.

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Obesity has been around for more than 20 000 years, as evidenced by statuettes produced in the Stone Age. Body mass index (BMI), calculated as weight divided by the square of the height, is one of the simplest and most common ways of defining obesity. A BMI between 18.5 and 24.9 is classed as normal. BMI values of 25.0–29.9 suggest overweight, and any values over 30 are deemed obese. Across populations, BMI is closely associated with whole body adiposity, and the cut-off levels for overweight and obesity reflect the increasing risk of metabolic, cardiovascular, and other complications of obesity as BMI increases above the normal range. Obesity is widely agreed to be caused by a prolonged period of energy imbalance. In 95% of cases, it is due to the impact of an obesogenic lifestyle (overconsumption of energy and/or insufficient energy expenditure) on a variable background of genetic susceptibility. The remaining cases are caused by certain drugs, specific endocrine diseases, and monogenic syndromes.
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42

Adipositas. Hogrefe-Verlag, 2003.

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Adipositas. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-68078-9.

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44

Bischoff, Stephan C., red. Adipositas. De Gruyter, 2018. http://dx.doi.org/10.1515/9783110412802.

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45

LeBow, Michael D. Adipositas. Huber, Bern, 1991.

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46

Schwanstecher, Mathias. Diabetes - Perspectives in Drug Therapy. Springer, 2011.

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47

Diabetes Perspectives In Drug Therapy. Springer, 2011.

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48

Schwanstecher, Mathias. Diabetes - Perspectives in Drug Therapy. Springer, 2013.

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49

Lechleitner, M., Hermann Toplak, E. Böhnisch, F. Hoppichler i B. Ludvik. Praxishandbuch Adipositas. Springer London, Limited, 2013.

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(Contributor), E. Böhnisch, F. Hoppichler (Contributor), M. Lechleitner (Contributor), B. Ludvik (Contributor), P. Schmid (Contributor), R. Schoberberger (Contributor), H. Toplak (Contributor), H. Wallner (Contributor), T. Wascher (Contributor) i Hermann Toplak (Editor), red. Praxishandbuch Adipositas. Springer, 2002.

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