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Artykuły w czasopismach na temat "Activation/tolérance des lymphocytes T"
Bonnefoy, Nathalie, Daniel Olive i Bernard Vanhove. "Les futures générations d’anticorps modulateurs des points de contrôle de la réponse immunitaire". médecine/sciences 35, nr 12 (grudzień 2019): 966–74. http://dx.doi.org/10.1051/medsci/2019193.
Pełny tekst źródłaBockenstedt, Linda K., Mark A. Goldsmith, Gary A. Koretzky i Arthur Weiss. "The Activation of T Lymphocytes". Rheumatic Disease Clinics of North America 13, nr 3 (grudzień 1987): 411–30. http://dx.doi.org/10.1016/s0889-857x(21)00926-1.
Pełny tekst źródłaBertotto, Alberto, Roberto Gerli, Luisa Lanfrancone, Silvana Crupi, Carla Arcangeli, Cristina Cernetti, Fabrizio Spinozzi i Pietro Rambotti. "Activation of cord T lymphocytes". Cellular Immunology 127, nr 2 (maj 1990): 247–59. http://dx.doi.org/10.1016/0008-8749(90)90130-j.
Pełny tekst źródłaGerli, Roberto, Elisabetta Agea, Chiristopher Muscat, Rita Tognellini, Giuliana Fiorucci, Fabrizio Spinozzi, Cristina Cernetti i Alberto Bertotto. "Activation of Cord T Lymphocytes". Cellular Immunology 148, nr 1 (kwiecień 1993): 32–47. http://dx.doi.org/10.1006/cimm.1993.1089.
Pełny tekst źródłaGerli, Roberto, Elisabetta Agea, Christopher Muscat, Raffaella Ercolani, Onelia Bistoni, Rita Tognellini, Maria A. Mariggió, Fabrizio Spinozzi i Alberto Bertotto. "Activation of Cord T Lymphocytes". Cellular Immunology 155, nr 1 (kwiecień 1994): 205–18. http://dx.doi.org/10.1006/cimm.1994.1113.
Pełny tekst źródłaQuintana, Ariel, D�sir�e Griesemer, Eva C. Schwarz i Markus Hoth. "Calcium-dependent activation of T-lymphocytes". Pfl�gers Archiv - European Journal of Physiology 450, nr 1 (26.11.2004): 1–12. http://dx.doi.org/10.1007/s00424-004-1364-4.
Pełny tekst źródłaSchulze, Juliane, Juliane Gellrich, Michael Kirsch, Alexander Dressel i Antje Vogelgesang. "Central Nervous System-Infiltrating T Lymphocytes in Stroke Are Activated via Their TCR (T-Cell Receptor) but Lack CD25 Expression". Stroke 52, nr 9 (wrzesień 2021): 2939–47. http://dx.doi.org/10.1161/strokeaha.120.032763.
Pełny tekst źródłaSharom, Frances J., Anita L. H. Chiu i T. Elaine Ross. "Gangliosides and glycophorin inhibit T-lymphocyte activation". Biochemistry and Cell Biology 68, nr 4 (1.04.1990): 735–44. http://dx.doi.org/10.1139/o90-106.
Pełny tekst źródłaGrivel, Jean-Charles, Oxana Ivanova, Natalia Pinegina, Paul S. Blank, Alexander Shpektor, Leonid B. Margolis i Elena Vasilieva. "Activation of T Lymphocytes in Atherosclerotic Plaques". Arteriosclerosis, Thrombosis, and Vascular Biology 31, nr 12 (grudzień 2011): 2929–37. http://dx.doi.org/10.1161/atvbaha.111.237081.
Pełny tekst źródłaPippia, Proto, Luigi Sciola, Marianne Cogoli-greuter, Maria Antonia Meloni, alessandra Spano i Augusto Cogoli. "Activation signals of T lymphocytes in microgravity". Journal of Biotechnology 47, nr 2-3 (czerwiec 1996): 215–22. http://dx.doi.org/10.1016/0168-1656(96)01387-9.
Pełny tekst źródłaRozprawy doktorskie na temat "Activation/tolérance des lymphocytes T"
Bercovici, Nadège. "Activation et induction de tolérance des lymphocytes T dans des modèles de souris transgéniques". Paris 11, 1999. http://www.theses.fr/1999PA11T030.
Pełny tekst źródłaAntigen recognition by T cell can lead to immunity but also to antigen-specific T-cell tolerance. Immunological tolerance can be induced experimentally and may be useful for the treatment of organ-specific autoimmune diseases such as autoimmune diabetes. In this work, I have investigated the mechanisms of activation and tolerance induction in mature CD4+ and CDS+ T cells from TCR-transgenic mice. Systemic administration of soluble peptide is remarkably efficient to induce peripheral T-cell tolerance in vivo. Although one single injection induced transient T-cell tolerance, chronic intravenous (i. V. ) injections of soluble peptide is able to maintain CD4+ T-cell tolerance for more than 12 weeks. I have also shown that i. V. Injection of soluble peptide can tolerize naive CDS+ T cells but can also target effector CDS+ T cells thereby blocking the progression of an ongoing CDS-mediated autoimmune diabetes. Importantly, CDS+ T cell infiltrates are eliminated without bystander tissue damage. Furthermore, I have demonstrated that i. V. Injection of soluble MHC class I : peptide complexes represent an alternative strategy to induce CDS+ T cell tolerance in vivo. Tolerance was achieved by deletion and anergy of antigen-specific CDS+ T cells and allow to down-regulate an ongoing CDS mediated autoimmune diabetes. In experiments conducted in vitro with naïve T cells from TCR-transgenic mice, we have shown that antigen recognition by CD4+ T cells rapidly induced cytoskeletal alterations that are crucial for calcium responses and proliferation. Under conditions in which equal numbers of specific MHC class Il :peptide complexes are presented by dendritic cells (DC) and B cells, we could demonstrate that DC are always more efficient antigen presenting cells underlying the importance of adhesion/costimulatory molecules abundantly expressed by DC. Moreover, we provide evidence for the induction of small calcium signals in CD4+ T cells interacting with DC in the absence of specific antigen that involve MHC/TCR interactions. Finally, we have shown that naive CDS+ T cells can be fully activated and differentiated after antigenic stimulation in the absence of co-stimulatory signals. Altogether, these data contribute to our understanding of the mechanisms of activation and tolerance induction of CD4+ and CDS+ T cells
Adriouch, Sahil. "Immunorégulation par le NAD extracellulaire : activation via les ADP-ribosyl transférases du récepteur cytolytique P2X7". Paris 7, 2003. https://tel.archives-ouvertes.fr/tel-00003698.
Pełny tekst źródłaMono ADP-ribosyl transferases (ART) catalyse a posttranslational modification of proteins by transferring the ADP-ribose moiety of NAD to targets proteins. We investigated the functions of this newly identified proteins family and the effects of they substrate, extracellular NAD, on T lymphocytes. Exposure of T cells to micromolar NAD concentrations triggers rapid induction of apoptosis. This phenomenon requires the presence of ART2. 2 and of a functional P2X7 ATP receptor. ART2. 2-catalysed cell surface protein ADP-ribosylation activates the cytolitic P2X7 purinoreceptor causing pore formation and cell death. Interestingly, much lower NAD than ATP concentration are required to activate P2X7. In vivo, NAD concentration increases in inflamed tissue as a consequence of tissue injury. Based on the results that only peripheral naive T cells are sensitive to NAD, we proposed that NAD would participate to the control of autoimmunty by killing naive T cells surrounding inflamed tissue thereby limiting the activation of bystander T lymphocytes
Rivera, Cifuentes Claudia Andrea. "Intraepithelial dendritic cells : origin and function". Electronic Thesis or Diss., Université Paris Cité, 2021. http://www.theses.fr/2021UNIP5167.
Pełny tekst źródłaDendritic cells (DCs) patrol tissues and transport antigens to lymph nodes to initiate adaptive immune responses. Within tissues, DCs constitute a complex cell population made of distinct subsets that can exhibit different activation states and functions. How tissue-specific cues orchestrate DC diversification remains elusive. Particularly, the small intestine (SI) Lamina Propria (LP) is enriched in a peculiar population of cDC2s expressing the integrins CD103 and CD11b. Interestingly, a fraction of these cells can transmigrate into the epithelial layer both at steady state and in higher proportion upon infection. However, the consequences of such event on the identity and fate of these cells is unknown. By using single cell RNAseq analysis, we found that their epithelial colonization deeply modifies their transcriptomic profile, downregulating inflammatory genes expression and stimulating the transcription of antimicrobial genes. We then further described that the small intestine includes two pools of cDC2s originating from common preDC precursors: (1) lamina propria CD103+CD11b+ cDC2s that are mature-like pro-inflammatory cells and (2) intraepithelial cDC2s that exhibit an immature-like phenotype and induce tolerogenic T lymphocyte properties. Intraepithelial cDC2 phenotype results from the action of food-derived retinoic acid (ATRA), which enhances actomyosin contractility and promotes LP cDC2 transmigration into the epithelium. There, cDC2s are imprinted by environmental cues including ATRA itself and the mucus component Muc2. Hence, by reaching distinct sub-tissular niches, DCs can exist as immature and mature cells within the same tissue, revealing a novel mechanism of DC functional diversification
ADRIOUCH, Sahil. "Immunorégulation par le NAD extracellulaire : activation via les ADP-ribosyl transférases du récepteur cytolytique P2X7". Phd thesis, Université Paris-Diderot - Paris VII, 2003. http://tel.archives-ouvertes.fr/tel-00003698.
Pełny tekst źródłaKakwata-Nkor, Deluce Nora. "Induction de sous-populations de cellules dendritiques humaines pro-tolérogènes par des fragments d’anticorps bispécifiques". Thesis, Tours, 2019. http://www.theses.fr/2019TOUR3805.
Pełny tekst źródłaDendritic cells (DCs) have a central role in immunity and induce both specific immunity and immune tolerance thanks to their surface pathogen receptors (PPRs). The immune tolerance induced by tolerant DCs (Tol-DCs) appears as an interesting way to explore in order to improve the long-term transplantation outcome. Four DC subsets, at least, have been identified including conventional DCs (BDCA-1; BDCA-3), plasmacytoid DCs (pDC), Inflammatory DCs(MoDC) and Langerhans cells (LC). For each DC subset, an array of pathogen recognition receptors (PRRs) have been identified on their surface. The PRRs profile differs between DC subsets providing an individual responsiveness to target specific pathogens as well as to trigger and modulate immunological responses. The aim is to target DC subset PRRs by bispecific antibodies (BsAb) in order to induce physiological tolerance. Monocyte derived DC (moDC) and monocyte derived Langerhans DC (moLC) were obtained from CD14+ cells. The plasmacytoïd DC (pDC) were purified from an enriched DC cells fraction obtained by Percoll® gradient of PBMCs. The moDC, pDC and moLC subsets were analyzed by phenotype labelling and FACS. A Bispecific Ab (tandem scFv) were built to target PRR on DC subsets. The tandem is made of 2 scFv of 55KDa. The BsAb were produced using insect S2 (BIC05) or CHO cell (BIC15 or BIC25) and purified by protein L column. Each scFV recognize a PRR on DC. Each BsAb have been evaluated on its DC target and on PBMC at the phenotypic and functional levels by evaluating the maturation markers (CD83, CD86, CD25 and HLA-DR), cytokine secretions (IL-10, IL-12p70 and IFN- ) and the capacity to activate naïve T-cell as well as to induce regulatory T-cell (Treg)
De, Wit Dominique. "Tolérance immunologique induite: propriétés des lymphocytes T et des lymphocytes B". Doctoral thesis, Universite Libre de Bruxelles, 1991. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/213001.
Pełny tekst źródłaBilliard, Fabienne. "Etude de l'intéraction entre lymphocytes T régulateurs CD4+CD25+ et lymphocytes T effecteurs in vivo, chez la souris". Paris 6, 2007. http://www.theses.fr/2007PA066091.
Pełny tekst źródłaVan, Mechelen Marcelle. "Activation et inactivation des lymphocytes T :implications immunothérapeutiques". Doctoral thesis, Universite Libre de Bruxelles, 1991. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/213053.
Pełny tekst źródłaBoshell, Martina. "Activation and apoptosis requirements in human T lymphocytes". Thesis, University of Bath, 1996. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.307137.
Pełny tekst źródłaPoitrasson-Rivière, Maud. "Cellules T DC4+ FOXP3+ régulatrices et tolérance des lymphocytes T CD8+ à la périphérie". Paris 5, 2009. http://www.theses.fr/2009PA05T008.
Pełny tekst źródłaThe first part of this work showed, in an original mice model, that regulatory CD4+ T cells play an important role in preventing peripheral CD8+ T cell-mediated autoimmunity. We suggest that regulatory CD4+ Foxp3+ T cells induce the generation at the periphery of regulatory CD8+ T cells that can then regulate conventional CD8+ T cells activity. The second part of this work suggests strongly that self-recognition events are required to control autoreactive, potentially pathogenic, conventional T cells. Autoreactivity would thus be necessary for its own control
Książki na temat "Activation/tolérance des lymphocytes T"
Charles, Snow E., red. T-cell dependent and independent B-cell activation. Boca Raton: CRC Press, 1991.
Znajdź pełny tekst źródła1950-, Grinstein Sergio, i Rotstein Ori D, red. Mechanisms of leukocyte activation. San Diego: Academic Press, 1990.
Znajdź pełny tekst źródłaCarlos, Rodríguez-Gallego, i Arnaiz-Villena Antonio, red. Human T-lymphocyte activation deficiencies. Austin, TX: R.G. Landes, 1994.
Znajdź pełny tekst źródłaBranch, Moody D., red. T cell activation by CD1 and lipid antigens. Berlin: Springer, 2007.
Znajdź pełny tekst źródłaT cell protocols. Wyd. 2. New York: Humana Press, 2009.
Znajdź pełny tekst źródłaCooper, David. Suppressed PHA activation of T lymphocytes in simulated microgravity is restored by direct activation of protein kinase C with phorbol ester. [Washington, DC: National Aeronautics and Space Administration, 1997.
Znajdź pełny tekst źródłaCooper, David. Suppressed PHA activation of T lymphocytes in simulated microgravity is restored by direct activation of protein kinase C with phorbol ester. [Washington, DC: National Aeronautics and Space Administration, 1997.
Znajdź pełny tekst źródłaMarc, Feldmann, Maini R. N, Woody James N i United States. Naval Medical Research and Development Command., red. T-cell activation in health and disease: Disorders of immune regulation infection and autoimmunity : papers from an international meeting in Oxford, UK, in September 1988. London ; San Diego: Academic Press, 1989.
Znajdź pełny tekst źródła1955-, Suttles Jill, red. T-cell signaling of macrophage activation: Cell contact-dependent and cytokine signals. Austin: R.G. Landes, 1995.
Znajdź pełny tekst źródłaJean-Marie, Andrieu, Lu Wei i International Symposium on Cellular Approaches to the Control of HIV Disease (1st : 1994 : Paris, France), red. Cell activation and apoptosis in HIV infection: Implications for pathogenesis and therapy. New York: Plenum Press, 1995.
Znajdź pełny tekst źródłaCzęści książek na temat "Activation/tolérance des lymphocytes T"
Cahalan, Michael D., K. George Chandy, Thomas E. DeCoursey, Sudhir Gupta, Richard S. Lewis i Jeffrey B. Sutro. "Ion Channels in T Lymphocytes". W Mechanisms of Lymphocyte Activation and Immune Regulation, 85–101. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5323-2_9.
Pełny tekst źródłaMacDonald, H. R., i F. Erard. "Activation Requirements for Resting T Lymphocytes". W Current Topics in Microbiology and Immunology, 187–94. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71152-7_22.
Pełny tekst źródłaHadden, John W. "Transmembrane Signals in the Activation of T Lymphocytes". W Mechanisms of Lymphocyte Activation and Immune Regulation, 69–83. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5323-2_8.
Pełny tekst źródłaPillai, Shiv. "The Activation and Differentiation of Mature T Lymphocytes". W Lymphocyte Development, 434–64. Boston, MA: Birkhäuser Boston, 2000. http://dx.doi.org/10.1007/978-1-4612-2444-0_10.
Pełny tekst źródłaLouis-Dit-Sully, Christine, i Wolfgang W. A. Schamel. "Activation of the TCR Complex by Small Chemical Compounds". W T Lymphocytes as Tools in Diagnostics and Immunotoxicology, 25–39. Basel: Springer Basel, 2013. http://dx.doi.org/10.1007/978-3-0348-0726-5_3.
Pełny tekst źródłaLouis-Dit-Sully, Christine, Britta Blumenthal, Marlena Duchniewicz, Katharina Beck-Garcia, Gina J. Fiala, Esmeralda Beck-García, Markus Mukenhirn, Susana Minguet i Wolfgang W. A. Schamel. "Activation of the TCR Complex by Peptide-MHC and Superantigens". W T Lymphocytes as Tools in Diagnostics and Immunotoxicology, 9–23. Basel: Springer Basel, 2013. http://dx.doi.org/10.1007/978-3-0348-0726-5_2.
Pełny tekst źródłaWagner, H., i K. Heeg. "Signal Requirements for the Primary Activation of Murine CD8 T Lymphocytes". W Progress in Immunology, 581–86. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83755-5_78.
Pełny tekst źródłaIlinskaya, Anna, Ankit Shah, Andrew Van Dusen i Marina A. Dobrovolskaia. "Detection of Intracellular Complement Activation by Nanoparticles in Human T Lymphocytes". W Methods in Molecular Biology, 109–20. New York, NY: Springer US, 2024. http://dx.doi.org/10.1007/978-1-0716-3786-9_11.
Pełny tekst źródłaSiliciano, Robert F., i Ellis L. Reinherz. "Activation of Precursor and Mature T Lymphocytes via the 50KD T11 Molecule". W Regulation of Immune Gene Expression, 95–110. Totowa, NJ: Humana Press, 1986. http://dx.doi.org/10.1007/978-1-4612-5014-2_9.
Pełny tekst źródłaMadden, John J., David Ketelsen, William L. Whaley, Robert M. Donahoe i Dorothy Oleson. "Mitogenic Activation of Human T Lymphocytes Induces a High Affinity Morphine Binding Site". W The Brain Immune Axis and Substance Abuse, 37–40. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1951-5_6.
Pełny tekst źródłaStreszczenia konferencji na temat "Activation/tolérance des lymphocytes T"
Manteifel, Valentina, i Tiina I. Karu. "Activation of chromatin in T-lymphocytes under He-Ne laser radiation". W BiOS Europe '98, redaktorzy Giovanni F. Bottiroli, Tiina I. Karu i Rachel Lubart. SPIE, 1998. http://dx.doi.org/10.1117/12.334394.
Pełny tekst źródłaRemans, PH, KA Reedquist, SI Gringhuis, FC Breedveld, JM Van laar i CL Verweij. "THU0021 Defective rap1 activation in synovial fluid t lymphocytes from patients with rheumatoid arthritis". W Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.530.
Pełny tekst źródłaQasim, Ali, Touboul Eric, Gruy Frederic i Lambert Claude. "Mathematical Modeling for the Activation of T-Lymphocytes: Population Balance Modeling with Non Conventional Growth Law". W 5th Asian Particle Technology Symposium. Singapore: Research Publishing Services, 2012. http://dx.doi.org/10.3850/978-981-07-2518-1_195.
Pełny tekst źródłaParmantier, Eric, Georges Kalouche, Clara Soulard, Christophe Lanneau, Sophie Boisrobert-Blais, Céline Nicolazzi i Cécile Orsini. "Abstract 3269: Ablation of Cbl-b gene function leads to increased activation and resistance to immunosuppression in human primary T lymphocytes". W Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-3269.
Pełny tekst źródłaCarvalho. "P1.40 Presence of langerhans cells and activation of t lymphocytes in differents stages of uterine colo lesions associated with human papiloma virus infection". W STI and HIV World Congress Abstracts, July 9–12 2017, Rio de Janeiro, Brazil. BMJ Publishing Group Ltd, 2017. http://dx.doi.org/10.1136/sextrans-2017-053264.148.
Pełny tekst źródłaLuo, Lusong, Xiaoran Wu, Tong Zhang, Chunyan Fu, Yanjuan Zhang, Amy Guo, Dongping Zhou i in. "Abstract 5626: Investigation of T cell activation by anti-human PD-1 antibodies Nivolumab, Pembrolizumab and BGB-A317 using tumor-infiltrating lymphocytes (TILs) from colorectal cancer and colorectal liver metastasis patients". W Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-5626.
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