Bibliografie tematiche / Vascular risk factors

Letteratura scientifica selezionata sul tema "Vascular risk factors"

Autore: Grafiati
Pubblicato: 4 giugno 2021
Ultima modifica: 11 marzo 2023

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Articoli di riviste sul tema "Vascular risk factors"

1

de la Torre, Jack C. "Vascular Risk Factors". American Journal of Alzheimer's Disease & Other Dementiasr 28, n. 6 (28 giugno 2013): 551–59. http://dx.doi.org/10.1177/1533317513494457.

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Hanyu, Haruo. "Treatment of vascular risk factors". Nippon Ronen Igakkai Zasshi. Japanese Journal of Geriatrics 49, n. 3 (2012): 284–87. http://dx.doi.org/10.3143/geriatrics.49.284.

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3

Ouldred, Emma, e Catherine Bryant. "Vascular risk factors and dementia". British Journal of Cardiac Nursing 5, n. 5 (maggio 2010): 240–46. http://dx.doi.org/10.12968/bjca.2010.5.5.47882.

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4

B??chetoille, Alain. "Vascular risk factors in glaucoma". Current Opinion in Ophthalmology 7, n. 2 (aprile 1996): 39–43. http://dx.doi.org/10.1097/00055735-199604000-00007.

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Tanaka, Yutaka, Hitoshi Hara e Takenori Yamaguchi. "Risk factors of vascular dementia." Nosotchu 18, n. 4 (1996): 282–87. http://dx.doi.org/10.3995/jstroke.18.282.

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Schmidt, R., H. Schmidt e F. Fazekas. "Vascular risk factors in dementia". Journal of Neurology 247, n. 2 (21 febbraio 2000): 81–87. http://dx.doi.org/10.1007/s004150050021.

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Israelsson, Hanna, Bo Carlberg, Carsten Wikkelsö, Katarina Laurell, Babar Kahlon, Göran Leijon, Anders Eklund e Jan Malm. "Vascular risk factors in INPH". Neurology 88, n. 6 (6 gennaio 2017): 577–85. http://dx.doi.org/10.1212/wnl.0000000000003583.

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Abstract (sommario):
Objective:To assess the complete vascular risk factor (VRF) profile of idiopathic normal pressure hydrocephalus (INPH) using a large sample of representative patients with INPH and population-based controls to determine the extent to which vascular disease influences INPH pathophysiology.Methods:All patients with INPH who underwent shunting in Sweden in 2008–2010 were compared to age- and sex-matched population-based controls. Inclusion criteria were age 60–85 years and no dementia. The 10 most important VRFs and cerebrovascular and peripheral vascular disease were prospectively assessed using blood samples, clinical examinations, and standardized questionnaires. Assessed VRFs were hypertension, hyperlipidemia, diabetes, obesity, psychosocial factors, smoking habits, diet, alcohol intake, cardiac disease, and physical activity.Results:In total, 176 patients with INPH and 368 controls participated. Multivariable logistic regression analysis indicated that hyperlipidemia (odds ratio [OR] 2.380; 95% confidence interval [CI] 1.434–3.950), diabetes (OR 2.169; 95% CI 1.195–3.938), obesity (OR 5.428; 95% CI 2.502–11.772), and psychosocial factors (OR 5.343; 95% CI 3.219–8.868) were independently associated with INPH. Hypertension, physical inactivity, and cerebrovascular and peripheral vascular disease were also overrepresented in INPH. Moderate alcohol intake and physical activity were overrepresented among the controls. The population-attributable risk percentage was 24%.Conclusions:Our findings confirm that patients with INPH have more VRFs and lack the protective factors present in the general population. Almost 25% of cases of INPH may be explained by VRFs. This suggests that INPH may be a subtype of vascular dementia. Targeted interventions against modifiable VRFs are likely to have beneficial effects on INPH.
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Albu, Carmen, Vlad Padureanu, Mihail Boldeanu, Ana Bumbea, Anca Enescu, Dana Albulescu, Cristian Silosi e Aurelia Enescu. "Vascular neurocognitive disorders and the vascular risk factors". Journal of Mind and Medical Sciences 5, n. 1 (1 aprile 2018): 7–15. http://dx.doi.org/10.22543/7674.51.p715.

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Rao, Rahul. "Documentation of vascular risk factors in vascular dementia". Psychiatric Bulletin 22, n. 2 (febbraio 1998): 97–99. http://dx.doi.org/10.1192/pb.22.2.97.

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The diagnosis of vascular dementia may often be made on the basis of structural neuroimaging, this may not always be reliable. In view of this, a retrospective study of 30 case notes with diagnosis of vascular dementia was undertaken to examine the documentation of vascular risk factors and presence of cerebrovascular disease. Specific recommendations were then made for the future case note documentation. Nine months later, 10 case notes were examined to assess the impact of the recommendations. A noticeable improvement in the documentation of most risk factors was observed. The wider implications of these findings are discussed.
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BARBARASH, N. A., e D. Yu KUVSHINOV. "Smoking and cardio -vascular risk factors". Complex Issues of Cardiovascular Diseases, n. 1 (8 maggio 2016): 51–54. http://dx.doi.org/10.17802/2306-1278-2016-1-51-54.

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Tesi sul tema "Vascular risk factors"

1

Cornett, Patricia F. "Risk Factors for Vascular Dementia". Thesis, University of North Texas, 2005. https://digital.library.unt.edu/ark:/67531/metadc4781/.

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Dementia is a devastating disorder that commonly affects people over the age of 65. Alzheimer's disease and vascular dementia are the most common forms of dementias. A number of studies have implicated cardiovascular risks as important factors in the development of dementia. These risks include high-risk behaviors such as smoking and risks related at least partially to health behaviors such as diet and exercise. This study examines a group of cardiovascular risk factors, as defined by the Framingham study, to ascertain if they are predictors of dementia. A retrospective chart review of 481consecutive patients seen in a geriatric medicine clinic produced a sample of 177 individuals diagnosed with dementia and 304 individuals without a dementia diagnosis. Relative risk ratio (RRR) results indicate that a history of hypertension (RRR= 1.80, p = .009) and a history of hypercholesterolemia (RRR = 1.85, p = .016) are significant predictors of Alzheimer's disease. A history of tobacco use (RRR = 2.18, p = .01) is a significant predictor of vascular dementia. Stepwise regression analyses indicate that hypercholesterolemia is an independent predictor of dementia (b = -.113, p = .009) and hypercholesterolemia (b = -.104, p = .018) and hypertension (b = -.094, p = .031) clustered together have an additive risk factor effect. These results are discussed in terms of the importance of specific health behaviors in the development and possible prevention of dementia.
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Leeson, Christopher Paul Maxted. "Early risk factors for later vascular disease". Thesis, University of Cambridge, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.624836.

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Seshadri, Swathi. "Retinal vascular function and cardiovascular risk factors". Thesis, Aston University, 2015. http://publications.aston.ac.uk/27347/.

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The current platform of conventional cardiovascular risk assessments tends to forsake the importance of endothelial function - a key biological mechanism by which cardiovascular risk factors exert their propensity for adverse vascular events. Moreover, the presence and severity of endothelial dysfunction in ‘low-risk’ individuals suggests considerable variability in pre-clinical risk that could potentially be detected well before the onset of disease. The aim of the present thesis was to investigate the presence and impact of retinal vascular dysfunction, as a barometer of endothelial function, in otherwise healthy individuals with one or more cardiovascular risk factors, but low to moderate cardiovascular risk. Systemic circulatory influences on retinal vascular function were also evaluated. The principle sections and findings of this work are: 1. Ageing effect on retinal vascular function • In low-risk individuals, there are age differences in retinal vascular function throughout the entire functional response curve for arteries and veins. Gender differences mainly affect the dilatory phase and are only present in young individuals. 2. Retinal vascular function in healthy individuals with a family history of cardiovascular disease • In low-risk individuals with a family history of cardiovascular disease, impairments in microvascular function at the retinal level correlate with established plasma markers for cardiovascular risk. 3. Ethnic differences in retinal vascular function • When compared to age-matched White Europeans, in low-risk middle-aged South Asians, there are impairments in retinal vascular function that correlate with established cardiovascular risk indicators. 4. Systemic circulatory influences on retinalµvascular function • Systemic antioxidant capacity (redox index) and plasma markers for cardiovascular risk (lipids) influence retinal microvascular function at both arterial and venous levels. 5. Retinal vascular function in individuals with obstructive sleep apnoea: a preliminarystudy • Patients with moderate to severe sleep apnoea exhibit attenuated retinal vascular function.
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VERTICCHIO, VERCELLIN ALICE CHANDRA. "VASCULAR RISK FACTORS AND GLAUCOMA OPTIC NEUROPATHY". Doctoral thesis, Università degli studi di Pavia, 2021. http://hdl.handle.net/11571/1434314.

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Kerr, Gillian. "Cerebrovascular diseases, vascular risk factors and socioeconomic status". Thesis, University of Glasgow, 2010. http://theses.gla.ac.uk/1892/.

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Abstract (sommario):
Cerebrovascular disease, has an enormous, and increasing, impact on global health. As well as causing clinical stroke, cerebrovascular disease is thought to be a major contributor to cognitive decline and dementia. Socioeconomic status (SES) is associated with risk of stroke. Those in the lowest SES group are estimated to be at twice the risk of stroke compared to those in the highest SES group. Those with low SES may also have a more severe stroke and a poorer outcome. It is imperative that the extent and mechanism of this association is clarified. This thesis aims to determine if the association between SES and stroke is explained by a greater prevalence of traditional vascular risk factors amongst those of low SES. It also explains the link with a novel risk factor, poor oral health. Lastly it addresses the long-term cognitive outcome in older people at risk of vascular disease. A systematic review and meta-analysis was undertaken to establish if vascular risk factors explain the association between SES and stroke incidence / post-stroke mortality. This demonstrated that lower SES was associated with an increased risk of stroke and that a greater burden of vascular risk factors in those with low SES explained about 50% of the additional risk of stroke. However this meta-analysis could not clarify what vascular risk factors are most critical. Low SES was also associated with increased mortality risk in those who have a stroke although study results were heterogeneous and this link was not readily explained by known vascular risk factors. A prospective study of 467 consecutive stroke and transient ischameic attack (TIA) patients from three Scottish hospitals was undertaken with the aim of establishing whether those with low SES carry higher levels of vascular risk factors, have a more severe stroke and have equal access to stroke care services and investigations. Stroke / TIA patients with low SES were younger and more likely to be current smokers but there was no association with other vascular risk factors /co-morbidity. Those who had lower SES had a more severe stroke. The lowest SES group were less likely to have neuroimaging or an electrocardiogram although differences were not significant on multivariate analysis. There was however equal access to stroke unit care. A secondary analysis of a prospective cohort study of 412 stroke patients was conducted. The aim was to explore oral health after acute stroke and assess if poor oral health explains the association between SES and stroke. Dry mouth amongst acute stroke patients was very common, however there was no association between oral health and low SES. There was an association of dry mouth with pre-stroke disability and Urinary Tract Infection. There was also a link with oral Candida glabrata colonisation, although the clinical relevance of this is uncertain. In the acute phase after stroke there was no convincing association of dry mouth with dysphagia or pneumonia. Therefore there was no association between SES and poor oral health as measured in this study but oral health may still be part of the explanation of the association between SES and acute stroke and this needs further investigation. Vascular disease is an important contributor to cognitive decline and dementia. Low SES may be associated with an increased risk of cognitive decline in later life and vascular disease may be a mediating factor. More effective prevention of vascular disease may slow cognitive decline and prevent dementia in later life, particularly in low SES groups. Lipid lowering with statins might be effective in preventing dementia but so far evidence from randomised control trials does not show benefit from statins in preventing cognitive decline and dementia. However the duration of follow-up in these trials was short and there may be benefit in the long-term. My aim was therefore to establish if long-term follow-up of the Prospective Study of Pravastatin in the Elderly at Risk (PROSPER) study was feasible. I found that it was feasible to follow-up 300 elderly survivors from the Scottish arm of the PROSPER study and the methods could be extended to the whole group. As expected nearly half of the PROSPER participants were dead. Additionally a large proportion of traceable participants had significant cognitive impairment. Smoking cessation, control of blood pressure and management of other vascular risk factors should be made a priority in areas of low SES. Additionally further research is needed to fully clarify the association between SES and stroke incidence. Avenues for exploration might include the possibilities of poorer access to effective stroke care, reduced uptake of care and poorer oral health in lower SES groups. In addition public health campaigns regarding smoking cessation should be directed at lower SES groups. I have shown that a large scale follow-up of the PROSPER participants is feasible and may determine new and novel risk factors for dementia and assess the long-term effect of a period of treatment with pravastatin.
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Warsch, Jessica. "Subclinical Vascular Brain Damage, Vascular Risk Factors, and Depression in Successful Cognitive Aging". Scholarly Repository, 2010. http://scholarlyrepository.miami.edu/oa_dissertations/644.

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Currently, about one in every eight Americans is age 65 or older; by the year 2050, it will be one in five people. Given this “graying” of the population, research into successful aging is of increasing relevance. The question of how to precisely define successful aging, however, has not been completely answered. Likewise, the role of vascular risk factors, subclinical vascular brain damage, and other biopsychosocial characteristics in normal cognitive aging are not well understood. This Dissertation focused on the identification of some of the physiological, behavioral, and social risk factors that distinguish people able to maintain extraordinary health at an advanced age. Specifically, we aimed to create an ecologically valid definition of successful aging that incorporates both physical well-being and cognitive abilities, and to report the prevalence of successful cognitive aging in a population-based multi-ethnic cohort of older adults. We sought to describe how the prevalence varies by several sociodemographic and psychosocial determinants, and to investigate global vascular risk, depressive symptomatology, and MRI markers of subclinical vascular brain damage as correlates of successful cognitive aging. We observed the prevalence of successful cognitive aging to be 37% in the study sample (N=1,162) of a diverse racial/ethnic population in Northern Manhattan (NYC, NY). The prevalence decreased with increasing age; we did not observe any differences by racial/ethnic group, but did note a lower prevalence with lower socioeconomic status. Several social resources and self-reported quality of life were related to successful cognitive aging, and appeared more important than demographic variables alone. We found that the likelihood of successful cognitive aging decreases with increasing global vascular risk score, more severe depressive symptomatology, and greater white matter damage. The field of successful aging requires further study. Consideration of such biopsychosocial factors as socioeconomic status, social support, quality of life, and depressive symptoms alongside novel indicators of disease and disability including global vascular risk and white matter hyperintensity burden is essential. It may lead to a more robust definition of successful cognitive aging replete with opportunities to modify the aging process, as many of the factors investigated in this study are modifiable.
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Stanyer, Lee. "Beta-amyloid/plasma lipoprotein interactions : implications for vascular damage". Thesis, University College London (University of London), 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.270774.

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Rönnemaa, Elina. "Predictors of Dementia : Insulin, Fatty Acids and Vascular Risk Factors". Doctoral thesis, Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-164528.

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Identification of modifiable risk factors for Alzheimer’s disease (AD) is crucial in order to diminish suffering from this devastating disease. The aim of this thesis was to investigate if different aspects of glucose metabolism, insulin, fatty-acid composition or other vascular risk factors predict the future development of AD and dementia. This thesis is based on the Uppsala Longitudinal Study of Adult Men (ULSAM) cohort, which started in 1970. A total of 2322 men at age 50 were examined with focus on vascular risk factors. The cohort was re-examined at ages 60, 71, 77, 82 and 88. Incident diagnoses of AD, vascular dementia, other dementias and cognitive impairment were assessed in 2005–2010. The risk of AD was increased in subjects with lower early insulin response measured with both an intravenous glucose tolerance test at 50 years and an oral glucose tolerance test at 71 years of age. The presence of vascular risk factors such as hypertension, obesity, hypercholesterolemia and smoking increased the risk of future vascular dementia but not of AD. Furthermore, saturated fatty acids at midlife were inversely associated with risk of AD. No evidence of a protective effect of omega-3 fatty acids against dementia was found. The susceptibility allele, APOE ε4, was the strongest individual risk factor. APOE ε4 carriers with vascular risk factors had the greatest risk of developing dementia. Low insulin response was a risk factor for AD mainly in APOE ε4 non-carriers. Disturbances in insulin and glucose metabolism, vascular risk factors and fatty acids are linked differentially to the pathogenesis of AD and vascular dementia. These observations should be considered when future clinical approaches are planned to prevent and postpone the onset of dementia.
ULSAM
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Howard, Kellee. "The relationship between vascular risk factors and vascular cognitive impairment, a secondary analysis of a dataset". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0033/MQ66626.pdf.

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Al-Shoumer, Kamal Abdul Aziz Sulaiman. "Studies on intermediary metabolism and vascular risk factors in hypopituitary patients". Thesis, Imperial College London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.289334.

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Libri sul tema "Vascular risk factors"

1

Tousoulis, Dimitris. Risk factors and vascular endothelium. Hauppauge, N.Y: Nova Science Publishers, 2011.

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2

Jacobsen, Sarah R. Vascular dementia: Risk factors, diagnosis, and treatment. Hauppauge, N.Y: Nova Science, 2011.

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Andris, Kazmers, a cura di. Cardiac risk assessment before vascular surgery. Armonk, NY: Futura Pub. Co., 1994.

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Román, Gustavo C. Managing vascular dementia: Concepts, issues, and management. London: Science Press, 2003.

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M, Drance Stephen, a cura di. Vascular risk factors and neuroprotection in glaucoma: Update 1996. Amsterdam: Kugler Publications, 1997.

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Boers, G. H. J. Homocysttinuria: A risk factor of premature vascular disease. Dortrecht: Foris Publications, 1986.

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McKinley, Michelle. B-Vitamin status and plasma homocysteine: A risk factor for vascular disease. [S.l: The Author], 1999.

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Drance, Stephen M. Vascular Risk Factors and Neuroprotection in Glaucoma: Update 1996. Kugler Pubns B V (Medical), 1997.

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M, Gotto Antonio, e International Symposium on Multiple Risk Factors in Cardiovascular Disease (3rd : 1994 : Florence, Italy), a cura di. Multiple risk factors in cardiovascular disease: Vascular and organ protection. Dordrecht: Kluwer Academic Publishers, 1995.

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Huang, Yuli, Zhen Yang e Ji Bihl, a cura di. Cardiovascular Risk Factors: Related Vascular Injury and New Molecular Biomarkers. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-83250-313-3.

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Capitoli di libri sul tema "Vascular risk factors"

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Amin, Hardik P., e Joseph L. Schindler. "Epidemiology and Risk Factors". In Vascular Neurology Board Review, 49–55. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-52552-1_5.

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Amin, Hardik P., e Joseph L. Schindler. "Epidemiology and Risk Factors". In Vascular Neurology Board Review, 37–42. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-39605-7_5.

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Mikhailidis, D. P. "Cardiovascular Risk Factors and Peripheral Arterial Disease". In Vascular Surgery, 113–20. London: Springer London, 2003. http://dx.doi.org/10.1007/978-1-4471-3870-9_16.

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Daskalopoulou, Stella S., e Dimitri P. Mikhailidis. "Cardiovascular Risk Factors and Peripheral Arterial Disease". In Vascular Surgery, 165–72. London: Springer London, 2010. http://dx.doi.org/10.1007/978-1-84996-356-5_16.

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Daskalopoulou, Stella S., e Dimitri P. Mikhailidis. "Cardiovascular Risk Factors and Peripheral Arterial Disease". In Vascular Surgery, 133–40. London: Springer London, 2006. http://dx.doi.org/10.1007/1-84628-211-x_15.

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Daskalopoulou, Stella S., e Dimitri P. Mikhailidis. "Cardiovascular Risk Factors and Peripheral Arterial Disease". In Vascular Surgery, 189–200. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-65936-7_17.

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Hong, Keun-Sik. "Identification of Vascular Risk Factors". In Stroke Revisited: Diagnosis and Treatment of Ischemic Stroke, 247–53. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-1424-6_23.

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Haverkate, Frits. "Are C-Reactive Protein and Fibrinogen Risk Factors?" In Vascular Medicine, 13–21. Dordrecht: Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-009-0037-0_2.

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Zeitler, Eberhard. "Arteriosclerosis and Its Risk Factors". In Radiology of Peripheral Vascular Diseases, 19–28. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-56956-2_3.

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Shaper, A. G., S. G. Wannamethee e M. K. Walker. "Risk Factors and Cardiovascular Outcome". In Epidemiology of Peripheral Vascular Disease, 127–40. London: Springer London, 1991. http://dx.doi.org/10.1007/978-1-4471-1889-3_11.

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Atti di convegni sul tema "Vascular risk factors"

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Schramm, R., A. Zittermann, M. Morshuis, U. Fuchs, J. Fleischhauer, K. Hakim-Meibodi e J. Gummert. "Risk Stratification in Heart Transplantation According to Donor and Recipient Risk Factors". In 48th Annual Meeting German Society for Thoracic, Cardiac, and Vascular Surgery. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1679017.

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Kataeva, Nadezhda, Tatyana Zamoshchina, Mihail Svetlik, Elena Korotkova e Olesya Voronova. "COMPARATIVE CHARACTERISTICS OF NEUROMETABOLIC DRUGS IN PATIENTS WITH VASCULAR RISK FACTORS". In XV International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2019. http://dx.doi.org/10.29003/m412.sudak.ns2019-15/206-207.

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Khachatryan, K. V. "DUST LOAD AS PROBABLE CARDIOVASCULAR RISK FACTOR". In The 4th «OCCUPATION and HEALTH» International Youth Forum (OHIYF-2022). FSBSI «IRIOH», 2022. http://dx.doi.org/10.31089/978-5-6042929-6-9-2022-1-185-189.

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Abstract (sommario):
Introduction: The impact of harmful occupational factors has adverse effect to human body functional state due to regulatory mechanisms tension, which contributes to occupational diseases and premature aging development. The goal of the work: dust professions workers comparative assessment of cardiovascular risk indicators and elastic properties of vascular wall. Methods: First group included workers exposed to industrial aerosols during work (17 people), second group included workers working out of contact with industrial aerosol (15 people). The groups were comparable by age (53.0±1.43 and 54.7±2.41 years, respectively (p > 0.05). Vascular wall arterial stiffness were assessed by volumetric sphygmography, electrical impedance parameters, indicators of carbohydrate, lipid and nitrogen metabolism, cardiovascular risk assessment carried out by SCORE scale. Results: Working with dust factor, under lower levels of body mass index and lipid spectrum indices, higher indicators of vascular wall stiffness and pulse wave propagation velocity were noted compared to workers without contact with industrial aerosols. Stiffness indicators showed positive relationship with age, urea levels, high-density lipoproteins. Indicators of lipid metabolism (total cholesterol, triglycerides and low-density lipoproteins) showed positive relationship with fat mass level and negative relationship with musculoskeletal mass proportion and specific basal metabolism. Conclusions: There is reason to assume that dust load is probably significant cardiovascular risk factor and may be comparable in importance to traditional risk factors.
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Djordjevic, I., K. Eghbalzadeh, S. Heinen, G. Schlachtenberger, C. Weber, A. Sabashnikov, N. Mader, Y. H. Choi, O. Liakopoulos e T. Wahlers. "Risk Factors Associated with In-hospital Mortality for Patients with Acute Abdomen after Cardiac Surgery". In 48th Annual Meeting German Society for Thoracic, Cardiac, and Vascular Surgery. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1678849.

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Marinez Alberola, N., C. Fernandez-Carballido, R. Martin Domenech, T. Pedraz Penalva, G. Albert Espi e F. Sivera Mascaró. "AB0280 Gender-differences in cardio-vascular risk factors in patients with inflammatory rmds". In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.5722.

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Lisboa MendonÇa Fortunato, Paloma, M. Cristina de O. Regina e EVANDRO RODRIGUES CASTRO. "Income and Education : influence in cardio-vascular risk factors of MS and T2DM". In XXIV Congresso de Iniciação Científica da UNICAMP - 2016. Campinas - SP, Brazil: Galoa, 2016. http://dx.doi.org/10.19146/pibic-2016-50602.

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Martínez Zapico, A., AI Pérez Álvarez, L. Caminal Montero, B. Díaz López, L. Benavente Fernández, R. Gómez de la Torre, D. Colunga Argüelles, J. Rodríguez Carrio, P. López Suárez e A. Suárez Díaz. "PS8:171 Endothelial dysfunction and vascular risk factors in patients with systemic lupus erythematosus". In 11th European Lupus Meeting, Düsseldorf, Germany, 21–24 March 2018, Abstract presentations. Lupus Foundation of America, 2018. http://dx.doi.org/10.1136/lupus-2018-abstract.214.

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Panholzer, B., K. Pilarczyk, K. Huenges, G. Morun, A. M. Eide, R. Rusch, L. Balke et al. "Acute Kidney Injury in Patients with Severe Acute Respiratory Distress Syndrome Requiring Extracorporeal Membrane Oxygenation: Incidence, Prognostic Impact, and Risk Factors". In 48th Annual Meeting German Society for Thoracic, Cardiac, and Vascular Surgery. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1678929.

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Wall, Wolfgang A., Mahmoud Ismail, Andreas Maier e Michael W. Gee. "Adjoint Based Calibration of Coupled Simulation Approaches of Patient-Specific Vascular Models". In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80559.

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In recent years, computational methods have often been utilized for a better understanding of cardiovascular systems. They can be used to analyze vascular disease development [1], predict life threatening risk factors [2], and help improve vascular treatments.
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Gallo, D., G. Di Benedetto, L. Gaetano, D. Massai, L. Antiga, R. Ponzini, M. A. Deriu, A. Redaelli e U. Morbiducci. "Identification of Atheroprone Morphological Features in Wall Shear Stress Waveforms in Carotid Bifurcations: A CFD-Based Integrated Approach". In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19390.

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Vascular endothelial cells functions are modulated by shear stress at vessel wall. The presence of “abnormal” blood flow patterns can give rise to local variations from physiological wall shear stress (WSS) time histories. These variations might be risk factors that increase a vessel’s atherosusceptibility.
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Rapporti di organizzazioni sul tema "Vascular risk factors"

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Kanner, Joseph, Mark Richards, Ron Kohen e Reed Jess. Improvement of quality and nutritional value of muscle foods. United States Department of Agriculture, dicembre 2008. http://dx.doi.org/10.32747/2008.7591735.bard.

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Food is an essential to our existence but under certain conditions it could become the origin to the accumulative health damages. Technological processes as heating, chopping, mincing, grounding, promote the lipid oxidation process in muscle tissues and meat foodstuffs. Lipid oxidation occurred rapidly in turkey muscle, intermediate in duck, and slowest in chicken during frozen storage. Depletion of tocopherol during frozen storage was more rapid in turkey and duck compared to chicken. These processes developed from lipid peroxides produce many cytotoxic compounds including malondialdehyde (MDA). The muscle tissue is further oxidized in stomach conditions producing additional cytotoxic compounds. Oxidized lipids that are formed during digestion of a meal possess the potential to promote reactions that incur vascular diseases. A grape seed extract (1% of the meat weight) and butylated hydroxytoluene (0.2% of the lipid weight) were each effective at preventing formation of lipid oxidation products for 3 hours during co-incubation with cooked turkey meat in simulated gastric fluid (SGF). Polyphenols in the human diet, as an integral part of the meal prevent the generation and absorption of cytotoxic compounds and the destruction of essential nutrients, eg. antioxidants vitamins during the meal. Polyphenols act as antioxidants in the gastrointestinal tract; they scavenge free radicals and may interact with reactive carbonyls, enzymes and proteins. These all reactions results in decreasing the absorption of reactive carbonyls and possible other cytotoxic compounds into the plasma. Consumptions of diet high in fat and red meat are contributory risk factors partly due to an increase production of cytotoxic oxidized lipid products eg. MDA. However, the simultaneously consumption of polyphenols rich foods reduce these factors. Locating the biological site of action of polyphenols in the in the gastrointestinal tract may explain the paradox between the protective effect of a highly polyphenols rich diet and the low bioavailability of these molecules in human plasma. It may also explain the "French paradox" and the beneficial effect of Mediterranean and Japanese diets, in which food products with high antioxidants content such as polyphenols are consumed during the meal.
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Goodwin, Simon. IMPROVE-Stroke: IMproving the PRevention Of Vascular Events after Stroke or TIA – a randomised controlled pilot trial of nurse independent prescriber-led care pathway-based risk factor management. National Institute for Health and Care Research (NIHR), marzo 2022. http://dx.doi.org/10.3310/nihropenres.1115183.1.

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FEDOTKINA, S. A., O. V. MUZALEVA e E. V. KHUGAEVA. RETROSPECTIVE ANALYSIS OF THE USE OF TELEMEDICINE TECHNOLOGIES FOR THE PREVENTION, DIAGNOSIS AND TREATMENT OF HYPERTENSION. Science and Innovation Center Publishing House, 2021. http://dx.doi.org/10.12731/978-0-615-67320-2-4-22.

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Abstract (sommario):
Introduction. The economic losses associated with disability due to diseases of the circulatory system, as well as the costs of providing medical care to patients suffering from heart and vascular diseases, are increasing annually. The state preventive measures currently being carried out are of a delayed nature. The results of the medical examination of the population of the Russian Federation in recent years (2015-2019) indicate that the incidence of cardiovascular diseases, including hypertension, is at a fairly high level. In the middle of the last century, the Concept of risk factors for the development of chronic non-communicable diseases were formulated, in the structure of which cardiovascular diseases, including arterial hypertension, occupies one of the primary positions. The concept is based on the results of promising epidemiological studies, and, at present, is a methodological basis for planning and organizing primary prevention of cardiovascular diseases. The purpose of the study. Based on the analysis of literary sources (including foreign ones) containing experience in the use of telemedicine technologies, to assess their significance for the prevention, diagnosis and treatment of hypertension, as well as forecasting improvements in the quality of medical care when adapting to the use of clinical recommendations. Materials and methods. The article provides an analytical review of the use of modern telemedicine technologies in the prevention of hypertension. The results of the study and their discussion. The analysis of literary sources has shown that in the context of the progress of information and telecommunication technologies in the healthcare system, a fundamentally new direction has appeared in the organization and provision of medical care to the population - telemedicine, which will ensure the modern level of prevention, detection and treatment of chronic non-communicable diseases, and also determines positive medical, social and economic performance indicators. To date, updates in the legislative framework of the Russian Federation are aimed at ensuring that medical care with the use of telemedicine technologies is more widespread, taking into account the standards of medical care and clinical recommendations. Conclusion. Based on a review of literature sources, it has been established that the modern solution to the problem of improving the quality of medical care for patients, including those with hypertension, diseases is medical care using telemedicine technologies that prove their medical, social and economic effectiveness.
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