Letteratura scientifica selezionata sul tema "Trauma hemorrhagic shock"
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Articoli di riviste sul tema "Trauma hemorrhagic shock"
Horton, J. W. "Cardiac contractile effects of ethanolism and hemorrhagic shock". American Journal of Physiology-Heart and Circulatory Physiology 262, n. 4 (1 aprile 1992): H1096—H1103. http://dx.doi.org/10.1152/ajpheart.1992.262.4.h1096.
Testo completoD'Alessandro, Angelo, Hunter B. Moore, Ernest E. Moore, Matthew Wither, Travis Nemkov, Eduardo Gonzalez, Anne Slaughter et al. "Early hemorrhage triggers metabolic responses that build up during prolonged shock". American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 308, n. 12 (15 giugno 2015): R1034—R1044. http://dx.doi.org/10.1152/ajpregu.00030.2015.
Testo completoGODINHO, MAURICIO, PEDRO PADIM, PAULO ROBERTO B. EVORA e SANDRO SCARPELINI. "Curbing Inflammation in hemorrhagic trauma: a review". Revista do Colégio Brasileiro de Cirurgiões 42, n. 4 (agosto 2015): 273–78. http://dx.doi.org/10.1590/0100-69912015004013.
Testo completoNapolitano, Lena M. "Resuscitation following trauma and hemorrhagic shock". Critical Care Medicine 23, n. 5 (maggio 1995): 795–97. http://dx.doi.org/10.1097/00003246-199505000-00001.
Testo completoLaserna, Anna Karen Carrasco, Yiyang Lai, Guihua Fang, Rajaseger Ganapathy, Mohamed Shirhan Bin Mohamed Atan, Jia Lu, Jian Wu, Mahesh Uttamchandani, Shabbir M. Moochhala e Sam Fong Yau Li. "Metabolic Profiling of a Porcine Combat Trauma-Injury Model Using NMR and Multi-Mode LC-MS Metabolomics—A Preliminary Study". Metabolites 10, n. 9 (16 settembre 2020): 373. http://dx.doi.org/10.3390/metabo10090373.
Testo completoLiu, Fu-Chao, Chih-Wen Zheng e Huang-Ping Yu. "Maraviroc-Mediated Lung Protection following Trauma-Hemorrhagic Shock". BioMed Research International 2016 (2016): 1–8. http://dx.doi.org/10.1155/2016/5302069.
Testo completoChu, Xiaogang, Kumar Subramani, Marie Warren e Raghavan Pillai Raju. "Innate immune response in acute lung injury following hemorrhagic shock". Journal of Immunology 198, n. 1_Supplement (1 maggio 2017): 70.9. http://dx.doi.org/10.4049/jimmunol.198.supp.70.9.
Testo completoSchneider, Christian P., Martin G. Schwacha, T. S. Anantha Samy, Kirby I. Bland e Irshad H. Chaudry. "Androgen-mediated modulation of macrophage function after trauma-hemorrhage: central role of 5α-dihydrotestosterone". Journal of Applied Physiology 95, n. 1 (luglio 2003): 104–12. http://dx.doi.org/10.1152/japplphysiol.00182.2003.
Testo completoGauss, Tobias, Justin E. Richards, Costanza Tortù, François-Xavier Ageron, Sophie Hamada, Julie Josse, François Husson et al. "Association of Early Norepinephrine Administration With 24-Hour Mortality Among Patients With Blunt Trauma and Hemorrhagic Shock". JAMA Network Open 5, n. 10 (7 ottobre 2022): e2234258. http://dx.doi.org/10.1001/jamanetworkopen.2022.34258.
Testo completoRushing, G. D., R. C. Britt, J. N. Collins, F. J. Cole, L. J. Weireter e L. D. Britt. "Adrenal Insufficiency in Hemorrhagic Shock". American Surgeon 72, n. 6 (giugno 2006): 552–54. http://dx.doi.org/10.1177/000313480607200619.
Testo completoTesi sul tema "Trauma hemorrhagic shock"
Talving, Peep. "Aspects of hepatoduodenal trauma and fluid therapy in hemorrhagic shock /". Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-740-5/.
Testo completoAlexander, Geoffrey C. "The Effect of Adding Drag-Reducing Polymers to Resuscitation Fluid During Hemorrhagic Shock on Skeletal Muscle Microcirculation". VCU Scholars Compass, 2006. http://scholarscompass.vcu.edu/etd/1522.
Testo completoLabruto, Fausto. "Modifications of cardiovascular response to ischemia and trauma /". Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-379-5/.
Testo completoJi, Soo-Yeon. "COMPUTER-AIDED TRAUMA DECISION MAKING USING MACHINE LEARNING AND SIGNAL PROCESSING". VCU Scholars Compass, 2008. http://scholarscompass.vcu.edu/etd/1628.
Testo completoLundin, Sandra, e Jonas Molin. "Räddar liv eller slösar tid? : prehospital vätskebehandlings effekter på patienter i hemorragisk chock". Thesis, Sophiahemmet Högskola, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:shh:diva-3742.
Testo completoTrauma is the leading cause of death in Sweden for people between the age of 15 and 44 years and a large proportion of people die because of bleeding that occurs at the time of the injury. Bleeding also continues to be the leading cause of trauma-related death that could have been preventable both in a civilian and military setting. Trauma care is complex, often time-critical, and the ambulance nurses are often the first to care for these patients on the scene and therefore the first assessment and care of these patients is of great importance. Fluid resuscitation for critically injured trauma patients in hemorrhagic shock or threatening hemorrhagic shock is a debated topic and Sweden lacks national guidelines for trauma care and treatment prehospital. The regional guidelines sometimes for some manner differ between the counties in Sweden. The aim of this study was to determine the impact of fluid resuscitation for patients with hemorrhagic shock after trauma. As a method, a literature review was carried out, which included a total of 15 studies published between 2009 and 2019. The result revealed two main themes - the impact of fluid resuscitation on mortality and the impact of fluid resuscitation on coagulation. All four studies that examined how coagulation ability is affected by crystalloid hypertonic and/or isotonic fluid resuscitation in patients at risk of hemorrhagic shock after trauma, the severity seems to be dependent on the amount of fluid infused, the more fluid the more severe coagulation abnormalities. The result showed unclear evidence of the effect of fluid resuscitation in mortality for trauma patients in hemorrhagic shock. However, none of the studies showed it decreased in mortality. In conclusion, the majority of articles show that large amount of fluid given in prehospital care have no impact or did have a negative impact on survival of critically injured trauma patients in hemorrhagic shock. Many factors come into play and it is difficult to draw any conclusions based on the results and more research are needed.
Dufour-Gaume, Frédérique. "Enjeux, préparation et évaluation de produits sanguins labiles innovants adaptés aux blessés de guerre". Electronic Thesis or Diss., université Paris-Saclay, 2023. http://www.theses.fr/2023UPASQ076.
Testo completoWar casualties associate multiple injuries with shock hemorrhage. Despite the therapeutic progress of recent years, hemorrhage is the leading cause of preventable deaths and secondary multiple organ failure can lead to vital and functional prognosis. Management of war-injured patients based on damage control resuscitation and massive transfusion of whole blood reduced considerably the number of deaths. Nevertheless, during foreign operations whole blood is sometimes lacking because of logistic limitations and massive casualties. Modified blood products that are free from constraints could help war-injured patients to survive. To achieve this objective, we developed two French hyper-concentrated lyophilized plasmas with (FLYP-H/LP) or not (FLYP-H) lyophilized platelets. The production of these products is of a high quality. FLYP-H and FLYP-H/LP are high protein products, especially albumin, which confer them a hyperosmolarity twice that of plasma. In FLYP-H/LP, platelets were lysed during the manufacturing process and liberated high quantities of coagulation factors, such as fibrinogen. The therapeutic effects of FLYP-H and FLYP-H/LP were evaluated thanks to our war-injured porcin model. Statistical analysis highlighted the beneficial effects of FLYP-H and FLYP-H/LP on cardiovascular function and hemostasis. These results open the door to more analysis but on human FLYP-H and FLYP-H/LP
Prunet, Bertrand. "Contusion pulmonaire : aspects physiopathologiques et conséquences thérapeutiques". Thesis, Aix-Marseille, 2015. http://www.theses.fr/2015AIXM5001.
Testo completoPulmonary contusion is often associated with hemorrhagic shock, constituting a challenge in trauma care. For patients who have sustained lung contusions, fluid resuscitation should be carefully performed, because injured lungs are particularly vulnerable to massive fluid infusions with an increased risk of pulmonary edema and compliance impairment. Fluid administration should be included in an optimized and goal directed resuscitation, based on blood pressure objectives and hemodynamical monitoring. The use of fluids with high volume-expanding capacities (hypertonic saline, colloids) is probably interesting, as well as early introduction of vasopressors. Hemodynamic monitoring will allow to conduct resuscitation on blood pressure objectives, on preload parameters and on extravascular lung water measurement.Our work, based on experimental and clinical studies, objective to characterize the current modalities of ventilatory and hemodynamical aspect of pulmonary contusion care
Morrison, C. Anne Horwitz Irwin Hwang Lu-Yu. "Hypotensive resuscitation versus standard fluid resuscitation for the management of trauma patients in hemorrhagic shock : the safety phase of a randomized controlled trial". 2009. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:1465583.
Testo completoFraga, Caroline Anne Pires. "The use of Tranexamic acid in trauma patients with hemorrhagic shock - a retrospective study in a tertiary care hospital". Master's thesis, 2020. https://hdl.handle.net/10216/128787.
Testo completoIntroduction - Hemorrhagic shock is the major complication and cause of morbi-mortality in trauma patients. We aimed to study the impact of tranexamic acid in trauma patients with hemorrhagic shock in a tertiary hospital. Material and methods - Our study, performed at Centro Hospitalar e Universitário São João, included adult trauma patients with hemorrhagic shock admitted to the Intensive Care Department. We defined hemorrhagic shock as Systolic Blood Pressure ≤90 mmHg and/or Heat Rate ≥110 bpm. In the absence of these, the clinical judgment of the attending physician prevailed. We did patients cohort with and without tranexamic acid use: we assessed global mortality, invasive mechanical ventilation duration, Intensive Care Unit and hospital length of stay, red blood cells units transfused and vascular occlusive events. Results - 105 patients were included from the 1045 admitted for trauma between January 2017 and December 2018: of those, 38 received tranexamic acid (36,2%) versus 67 without it (63,8%). Tranexamic acid group registered a higher severity (APACHE II p=0,038) and mortality in the Intensive Care Unit either in the global population (p=0,003) or in the stratified Traumatic Brain Injury subgroup (p=0,037). We found no statistically significant differences in the secondary outcomes. Discussion - Trauma patients with hemorrhagic shock admitted in the Intensive Care Department and treated with tranexamic acid had higher mortality, probably explained by the higher severity observed. We didnt find increased vascular occlusive events in tranexamic acid group. Conclusions - It is necessary to complement these findings with more studies and larger cohorts to assess the real benefits of tranexamic acid use in these patients.
Fraga, Caroline Anne Pires. "The use of Tranexamic acid in trauma patients with hemorrhagic shock - a retrospective study in a tertiary care hospital". Dissertação, 2020. https://hdl.handle.net/10216/128787.
Testo completoIntroduction - Hemorrhagic shock is the major complication and cause of morbi-mortality in trauma patients. We aimed to study the impact of tranexamic acid in trauma patients with hemorrhagic shock in a tertiary hospital. Material and methods - Our study, performed at Centro Hospitalar e Universitário São João, included adult trauma patients with hemorrhagic shock admitted to the Intensive Care Department. We defined hemorrhagic shock as Systolic Blood Pressure ≤90 mmHg and/or Heat Rate ≥110 bpm. In the absence of these, the clinical judgment of the attending physician prevailed. We did patients cohort with and without tranexamic acid use: we assessed global mortality, invasive mechanical ventilation duration, Intensive Care Unit and hospital length of stay, red blood cells units transfused and vascular occlusive events. Results - 105 patients were included from the 1045 admitted for trauma between January 2017 and December 2018: of those, 38 received tranexamic acid (36,2%) versus 67 without it (63,8%). Tranexamic acid group registered a higher severity (APACHE II p=0,038) and mortality in the Intensive Care Unit either in the global population (p=0,003) or in the stratified Traumatic Brain Injury subgroup (p=0,037). We found no statistically significant differences in the secondary outcomes. Discussion - Trauma patients with hemorrhagic shock admitted in the Intensive Care Department and treated with tranexamic acid had higher mortality, probably explained by the higher severity observed. We didnt find increased vascular occlusive events in tranexamic acid group. Conclusions - It is necessary to complement these findings with more studies and larger cohorts to assess the real benefits of tranexamic acid use in these patients.
Libri sul tema "Trauma hemorrhagic shock"
J, Sugerman Harvey, e DeMaria Eric J. 1959-, a cura di. Cytokines in trauma and hemorrhage. New York: Chapman & Hall, 1997.
Cerca il testo completo1950-, Pope Andrew MacPherson, French Geoffrey, Longnecker David E. 1939- e Institute of Medicine (U.S.). Committee on Fluid Resuscitation for Combat Casualties, a cura di. Fluid resuscitation: State of the science for treating combat casualties and civilian injuries. Washington, D.C: National Academy Press, 1999.
Cerca il testo completoCytokines In Trauma & Hemorrhage. Thomson Publishing Group, 1997.
Cerca il testo completoSabato, Stefan. Massive Transfusion in a Child. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0024.
Testo completoFluid Resuscitation: State of the Science for Treating Combat Casualties and Civilian Injuries. National Academies Press, 1999.
Cerca il testo completoMedicine, Institute of, e Committee on Fluid Resuscitation for Combat Casualties. Fluid Resuscitation: State of the Science for Treating Combat Casualties and Civilian Injuries. National Academies Press, 1999.
Cerca il testo completoMedicine, Institute of, e Committee on Fluid Resuscitation for Combat Casualties. Fluid Resuscitation: State of the Science for Treating Combat Casualties and Civilian Injuries. National Academies Press, 1999.
Cerca il testo completoMedicine, Institute of, e Committee on Fluid Resuscitation for Combat Casualties. Fluid Resuscitation: State of the Science for Treating Combat Casualties and Civilian Injuries. National Academies Press, 1999.
Cerca il testo completoWeyker, Paul David, Christopher Allen-John Webb e Tricia E. Brentjens. Hypovolemic Shock. A cura di Matthew D. McEvoy e Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0097.
Testo completoFinn, Patrick C., e Michael C. Reade. Bleeding Emergencies (DRAFT). A cura di Raghavan Murugan e Joseph M. Darby. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190612474.003.0010.
Testo completoCapitoli di libri sul tema "Trauma hemorrhagic shock"
Zhao, Zi-Gang, Yu-Ping Zhang, Li-Min Zhang e Ya-Xiong Guo. "Lymph Formation and Transport: Role in Trauma-Hemorrhagic Shock". In Severe Trauma and Sepsis, 67–95. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-3353-8_5.
Testo completoLivingston, D. H., J. Hseih, T. Murphy e B. F. Rush. "Hemorrhagic Shock Inhibits Myelopoiesis Independent of Bacterial Translocation". In Host Defense Dysfunction in Trauma, Shock and Sepsis, 339–48. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_36.
Testo completoOchoa, J. B., T. R. Billiar e A. B. Peitzman. "The Role of Nitric Oxide in Hemorrhagic Shock and Trauma". In Shock, Sepsis, and Organ Failure — Nitric Oxide, 84–101. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79343-1_4.
Testo completoLivingston, D. H., e M. A. Malangoni. "Effect of Therapeutic Gamma-Interferon Administration on Wound Infection After Resuscitated Hemorrhagic Shock". In Immune Consequences of Trauma, Shock, and Sepsis, 551–55. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-73468-7_66.
Testo completoErtel, W., E. Faist e I. H. Chaudry. "The Biological Characteristics of Cytokines and Their Interactions with Prostaglandins Following Hemorrhagic Shock". In Host Defense Dysfunction in Trauma, Shock and Sepsis, 655–64. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_83.
Testo completoTolentino, Rafael, Timothy J. Holleran e Laura S. Johnson. "Futility of Care in Hemorrhagic Shock: When Prolonging the Massive Transfusion Protocol Is of No Benefit". In Difficult Decisions in Trauma Surgery, 35–44. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-81667-4_4.
Testo completoStadlbauer, K. H., H. G. Wagner-Berger, C. Raedler, W. Voelckel, V. Wenzel, A. C. Krismer, K. Rheinberger, K. H. Lindner e A. Koenigsrainer. "Survival with Full Vital Organ Function Recovery after Uncontrolled Hemorrhagic Shock in a Penetrating Liver Trauma Model with Vasopressin in Pigs". In Zurück in die Zukunft, 638. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-55611-1_443.
Testo completoChaudry, I. H., R. N. Stephan, J. M. Harkema e R. E. Dean. "Immunological Alterations Following Simple Hemorrhage". In Immune Consequences of Trauma, Shock, and Sepsis, 363–73. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-73468-7_47.
Testo completoChaudry, I. H., A. Ayala, D. Meldrum e W. Ertel. "Hemorrhage-Induced Alterations in Cell-Mediated Immune Function". In Host Defense Dysfunction in Trauma, Shock and Sepsis, 149–59. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_16.
Testo completoClemens, M. G. "Hepatic Parenchymal and Nonparenchymal Cells in Hemorrhage and Ischemia". In Host Defense Dysfunction in Trauma, Shock and Sepsis, 127–37. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_14.
Testo completoAtti di convegni sul tema "Trauma hemorrhagic shock"
Rahbar, Elaheh, Bryan A. Cotton, John B. Holcomb e Charles E. Wade. "Reduced Plasma Oncotic Pressure is Indicative of Injury Severity in Trauma Patients". In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14539.
Testo completoThau, M. R., T. Liu, N. A. Sathe, G. O'Keefe, C. E. Wade, E. E. Fox, J. B. Holcomb et al. "Latent Class Analysis in a Trauma Cohort with Hemorrhagic Shock Identifies Two Distinct Sub-Phenotypes with a Differential Treatment Response to Blood Transfusion Ratios". In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a5038.
Testo completoSotudeh-Chafi, M., N. Abolfathi, A. Nick, V. Dirisala, G. Karami e M. Ziejewski. "A Multi-Scale Finite Element Model for Shock Wave-Induced Axonal Brain Injury". In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192342.
Testo completoNemeth, Christopher, Adam Amos-Binks, Gregory Rule, Dawn Laufersweiler, Natalie Keeney, Yuliya Pinevich e Vitaly Herasevich. "Real Time Battlefield Casualty Care Decision Support". In 13th International Conference on Applied Human Factors and Ergonomics (AHFE 2022). AHFE International, 2022. http://dx.doi.org/10.54941/ahfe1002112.
Testo completoGuzas, Emily L., Stephen E. Turner, Matthew Babina, Brandon Casper, Thomas N. Fetherston e Joseph M. Ambrico. "Validation of a Surrogate Model for Marine Mammal Lung Dynamics Under Underwater Explosive Impulse". In ASME 2019 Verification and Validation Symposium. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/vvs2019-5143.
Testo completoRapporti di organizzazioni sul tema "Trauma hemorrhagic shock"
Tweardy, David J. Prevention of Trauma/Hemorrhagic Shock-Induced Mortality,Apoptosis, Inflammation and Mitochondrial Dysfunction. Fort Belvoir, VA: Defense Technical Information Center, dicembre 2013. http://dx.doi.org/10.21236/ada612817.
Testo completoTweardy, David J. Prevention of Trauma/Hemorrhagic Shock-Induced Mortality, Apoptosis, Inflammation and Mitochondrial Dysfunction. Fort Belvoir, VA: Defense Technical Information Center, dicembre 2012. http://dx.doi.org/10.21236/ada612818.
Testo completoTweardy, David J. Prevention of Trauma/Hemorrhagic Shock-Induced Mortality, Apoptosis, Inflammation and Mitochondrial Dysfunction Using IL-6 as a Resuscitation Adjuvant. Fort Belvoir, VA: Defense Technical Information Center, dicembre 2011. http://dx.doi.org/10.21236/ada612819.
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