Letteratura scientifica selezionata sul tema "Testicule – Foetus"
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Articoli di riviste sul tema "Testicule – Foetus":
Varma, Santosh K., e Eric Bloch. "Effects of prenatal administration of mestranol and two progestins on testosterone synthesis and reproductive tract development in male rats". Acta Endocrinologica 116, n. 2 (ottobre 1987): 193–99. http://dx.doi.org/10.1530/acta.0.1160193.
Grinspon, Romina P., e Rodolfo A. Rey. "Molecular Characterization of XX Maleness". International Journal of Molecular Sciences 20, n. 23 (3 dicembre 2019): 6089. http://dx.doi.org/10.3390/ijms20236089.
van der Schoot, P. "Foetal testes control the prenatal growth and differentiation of the gubernacular cones in rabbits--a tribute to the late Professor Alfred Jost". Development 118, n. 4 (1 agosto 1993): 1327–34. http://dx.doi.org/10.1242/dev.118.4.1327.
Rey, Rodolfo. "Anti-Müllerian hormone in disorders of sex determination and differentiation". Arquivos Brasileiros de Endocrinologia & Metabologia 49, n. 1 (febbraio 2005): 26–36. http://dx.doi.org/10.1590/s0004-27302005000100005.
Veeramachaneni, D. N. Rao, e Gary R. Klinefelter. "Phthalate-induced pathology in the foetal testis involves more than decreased testosterone production". REPRODUCTION 147, n. 4 (aprile 2014): 435–42. http://dx.doi.org/10.1530/rep-13-0441.
Bertoldo, Joselma da Silva Cavalcanti, Apolônio Gomes Ribeiro, Liana Mesquita Vilela, Paola Teles Soares, Maria Juliana Coelho Dias da Silva Teixeira e Júlio Cézar dos Santos Nascimento. "Male pseudohermaphroditism in a dog: case report". Acta Veterinaria Brasilica 17, n. 4 (31 dicembre 2023): 23–28. http://dx.doi.org/10.21708/avb.2023.17.4.11795.
Ngona, I. A., J. M. Beduin, A. B. F. Khang' Maté e C. Hanzen. "Etude descriptive des caractéristiques morphométriques et génitales de la chèvre de Lubumbashi en République démocratique du Congo". Revue d’élevage et de médecine vétérinaire des pays tropicaux 65, n. 3-4 (1 marzo 2012): 75. http://dx.doi.org/10.19182/remvt.10126.
Dehkordi, R. F., A. Parchami e P. Kheibari. "Studies on Foetal Testicular Development in Sheep". Asian Journal of Biological Sciences 1, n. 2 (15 giugno 2008): 100–102. http://dx.doi.org/10.3923/ajbs.2008.100.102.
Engels, Manon, Paul N. Span, Rod T. Mitchell, Joop J. T. M. Heuvel, Monica A. Marijnissen-van Zanten, Antonius E. van Herwaarden, Christina A. Hulsbergen-van de Kaa et al. "GATA transcription factors in testicular adrenal rest tumours". Endocrine Connections 6, n. 8 (novembre 2017): 866–75. http://dx.doi.org/10.1530/ec-17-0215.
Young, J. C., A. Jaiprakash, S. Mithraprabhu, C. Itman, S. Kitazawa e K. L. Loveland. "170. TGFβ SIGNALING IN AN IN VITRO SEMINOMA MODEL". Reproduction, Fertility and Development 21, n. 9 (2009): 88. http://dx.doi.org/10.1071/srb09abs170.
Tesi sul tema "Testicule – Foetus":
Eladak, Soria. "Effets du Bisphénol A et de ses Substituts sur le Testicule Foetal Humain et Murin". Sorbonne Paris Cité, 2015. http://www.theses.fr/2015USPCC203.
Since several decades, incidence of abnormalities in male reproductive function has been steadily increasing. Some of these alterations are thought to be the expression of a common syndrome, the testicular dysgenesis syndrome. It has been suggested that these pathologies could result from alterations affecting the testis during fetal life and their increasing incidence could be a consequence of an increasing exposure to endocrine disruptors (ED). Bisphenol A (BPA) is one of the most used and studied ED. The restriction on the use of BPA in some countries has led industrialists to use other molecules such as bisphenols S and F (BPS and BPF). Using the organotypic culture system named Fetal Testis Assay, we have shown that like BPA, 10-8 M of BPS and BPF decreases basal testosterone secretion by human fetal testis. Furthermore, human fetal testis seems more sensitive to BPS and BPF than mouse fetal testis. At 10-5 M, the three bisphenols also decrease Insl3 expression in mouse fetal testis. Moreover, we have developed a xenograft model of human fetal testis in the Nude mouse. The grafted testes present a well maintained architecture and a functional testosterone secretion. Addition of 10-5 M of BPA in the drinking water of the mice induces a decrease in density of germ cells in the grafted testes and modifies the expression of markers associated with the differentiation of these cells. Our results suggest that BPA decreases the number and accelerates the differentiation of germ cells in the human fetal testis. Finally, we have shown that ERRβ/γ receptors could be involved in the inhibition of testosterone production induced by BPA in human and mouse fetal testis
Lesné, Laurianne. "Antiépileptiques et antalgiques pendant la grossesse et homéostasie du testicule foetal humain". Electronic Thesis or Diss., Université de Rennes (2023-....), 2023. http://www.theses.fr/2023URENB075.
Fetal development is a period that is particularly vulnerable to imbalances in the environment in which it takes place. The fetal testis plays a crucial role in the masculinization of the reproductive organs via its endocrine function, and any alteration can give rise to a range of malformations that can have an impact on a man's future fertility. During development, the embryo and then the fetus are exposed to a complex chemical environment known as the maternal exposome. Among the families of substances that make up the exposome are the medicines that pregnant women take to treat a chronic pathology or acutely to treat a pregnancy-related symptom. We are interested in two classes of drugs: anti-epileptics (AEs) on the one hand, and analgesics on the other, because epidemiological studies have reported an increased risk of hypospadias in newborns exposed in utero to valproic acid, an AE, and of malformations of the reproductive system in those exposed to paracetamol and ibuprofen in particular. The occurrence of these alterations could be linked to disturbances in hormonal balance. We used an organotypic culture model of human fetal testicular explants to compare the disruption profiles generated by a short exposure - 3 days - to analgesics and a short and long exposure - 28 days - to AEs. To do this, we adapted and optimized our model to approximate the chronicity of treatment in vivo. These studies, using organotypic cultures of human fetal testes, showed that three AEs could, over a more or less long period, induce an alteration in the endocrine function of this organ, leaving open the possibility that with chronic treatment, the androgenic impregnation of the fetus could be affected, and that this could have consequences for the target organs. Although the NSAID analgesics studied appear to have less spectacular effects on the endocrine function of the developing human fetal testis, their use during pregnancy continues to present a risk for other organs whose development is sensitive to or dependent on prostaglandins
Charpentier, Gilles. "Etude d'un facteur testiculaire qui perturbe le developpement de l'ovaire du foetus de rat, in vitro". Paris 7, 1988. http://www.theses.fr/1988PA077032.
Muczynski, Vincent. "Polluants environnementaux et développement du testicule foetal humain : effets et mécanismes des phtalates". Phd thesis, Université Paris Sud - Paris XI, 2011. http://tel.archives-ouvertes.fr/tel-00631554.
Delon, Betty. "Hybridation in situ fluorescente en pathologie genetique humaine : preparation de sondes de peinture chromosomique humaine, cytogenetique interphasique de cellules germinales atypiques chez le foetus trisomique 21, validation technique sur tumeurs testiculaires invasives, incluses en paraffine". Clermont-Ferrand 1, 1997. http://www.theses.fr/1997CLF1MM11.
N'tumba-Byn, Thierry. "Mécanismes d’action des perturbateurs endocriniens bisphénol A et phtalates sur le développement du testicule fœtal". Thesis, Paris 11, 2013. http://www.theses.fr/2013PA11T006/document.
For several years, an increase in the incidence of pathologies connected to the male reproductive functions has been described in numerous studies. These anomalies are classified under the term “testicular dysgenesis syndrome”. This syndrome might find its origins in the deleterious effects of environmental pollutants on the testis development in fetal period. Among theses environmental pollutants, phthalates and bisphenol A (BPA) are the most produced plasticizers found in products of common use. Many studies were performed in order to determine their effects, and allowed to classify them as endocrine disruptors because of their reprotoxic effects. My thesis work is a study of the effects of these two endocrine disruptors on the fetal testis development.Our first study focuses on the effects of BPA on the fetal testis development. Using the organotypic culture model, we developed our study in three species: rat, mouse and human. We demonstrated that BPA decreases the testosterone secretion in the human fetal testis from a 10-8M concentration, while in rat and mouse the testosterone secretion is only affected by 10-5M BPA. We also demonstrated a decreased Insl-3 gene expression, in the same conditions. These results allowed us to evidence a difference of sensibility between species. To understand the mechanisms involved in the BPA toxic effect, we compared it with the effect of DES, another endocrine disruptor with estrogenic activity. Unlike BPA, DES decreases the fetal testosterone secretion in rodents and not in human. This result suggests the involvement of two different signalisation pathways for these two xenoestrogens. This hypothesis is reinforced by the study that we performed in mice invalidated for the estrogen receptor ERα. In those mice, the anti-androgenic effect of BPA is maintained, unlike DES effect.In parallel, we investigated the mechanisms of action of phtalates and particularly of their most prevalent active metabolite, the MEHP (mono-2-ethyl-hexyl phthalate). Following previous studies performed in our laboratory concerning the effects of MEHP, we intended to understand the mechanisms by which MEHP induces the apoptosis in male germ cells. We evidenced an increase in Stra8 gene expression in MEHP treated germ cells. This result suggests that MEHP might induce a wrong differentiation in male germ cells. Furthermore, we investigated the receptors and the signalisation pathway activated by MEHP. We observe that PPARα and PPARγ receptors agonists induce the same phenotypes as MEHP, namely an increase in the apoptosis and in Stra8 gene expression in germ cells
Habert, René. "Activité androgène du testicule chez le fœtus de rat in vivo et sa régulation". Paris 7, 1986. http://www.theses.fr/1986PA077086.
Boukari, Kahina. "Rôle des récepteurs stéroïdiens dans les fonctions testiculaires endocrine et exocrine au cours du développement chez l'homme". Paris 6, 2009. http://www.theses.fr/2009PA066138.
Gangnerau, Marie-Noelle. "Différenciation de la stéroïdogenèse testiculaire chez le rat : ontogénèse et régulation in vitro". Paris 7, 1985. http://www.theses.fr/1985PA077040.
Delbès, Géraldine. "Effets des oestrogènes sur le développement foetal et néonatal du testicule". Paris 11, 2005. http://www.theses.fr/2005PA11T015.