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Libri sul tema "Syringomyelia"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 31 luglio 2024

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1

Klekamp, Jörg, e Madjid Samii. Syringomyelia. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-56023-1.

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2

Flint, Graham, e Clare Rusbridge, a cura di. Syringomyelia. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-13706-8.

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3

Tamaki, Norihiko, Ulrich Batzdorf e Tatsuya Nagashima, a cura di. Syringomyelia. Tokyo: Springer Japan, 2001. http://dx.doi.org/10.1007/978-4-431-67893-9.

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4

National Institutes of Health (U.S.) e National Institute of Neurological Disorders and Stroke (U.S.). Office of Scientific and Health Reports, a cura di. Syringomyelia. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1994.

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5

Anson, John A. Syringomyelia and the Chiari malformations. A cura di AANS Publications Committee. Park Ridge, Ill: American Association of Neurological Surgeons, 1997.

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6

Parker, James N., e Philip M. Parker. The official patient's sourcebook on syringomyelia. A cura di Icon Group International Inc. San Diego, Calif: Icon Health Publications, 2002.

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7

1928-, Batzdorf Ulrich, a cura di. Syringomyelia: Current concepts in diagnosis and treatment. Baltimore: Williams & Wilkins, 1991.

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8

Deutsche Gesellschaft für Neurochirurgie. Tagung. Stabilizing craniocervical operations: Calcium antagonists in SAH : current legal issues. Berlin: Springer-Verlag, 1990.

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9

Gutenberg-Universität, Johannes, a cura di. Die Änderung der operativen Therapieergebnisse der Syringomyelie nach Einführung der Kernspintomographie. [s.l.]: [s.n.], 1988.

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10

1850-, Hinsdale Guy. Syringomyelia. Franklin Classics, 2018.

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11

1858-1948, Hinsdale Guy. Syringomyelia. Franklin Classics Trade Press, 2018.

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12

1850-, Hinsdale Guy. Syringomyelia. Franklin Classics Trade Press, 2018.

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13

1850-, Hinsdale Guy. Syringomyelia. Franklin Classics, 2018.

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14

Barnett. Syringomyelia Mpn. W.B. Saunders Company, 1997.

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15

Syringomyelia: Diagnosis and Treatment. Springer, 2012.

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16

Syringomyelia And Hindbrain Hernia. Springer, 2012.

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17

Klekamp, Jörg, Madjid Samii e C. Matthies. Syringomyelia: Diagnosis and Treatment. Springer London, Limited, 2012.

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18

Klekamp, Jörg, Madjid Samii e C. Matthies. Syringomyelia: Diagnosis and Treatment. Springer London, Limited, 2012.

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19

Klekamp, Jörg, e Madjid Samii. Syringomyelia: Diagnosis and Treatment. Springer, 2001.

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20

Anson, John A. Syringomyelia and the Chiari Malformations (Neurosurgiacl Topic Vol 26). A cura di John A. Anson. American Association of Neurological Surgeons, 1997.

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21

Flint, Graham, e Clare Rusbridge. Syringomyelia: A Disorder of CSF Circulation. Springer Berlin / Heidelberg, 2016.

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22

Flint, Graham, e Clare Rusbridge. Syringomyelia: A Disorder of CSF Circulation. Springer, 2014.

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23

Flint, Graham, e Clare Rusbridge. Syringomyelia: A Disorder of CSF Circulation. Springer London, Limited, 2014.

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24

Tamaki, N. Syringomyelia: Current Concepts in Pathogenesis and Management. Springer, 2012.

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25

(Editor), N. Tamaki, U. Batzdorf (Editor) e T. Nagashima (Editor), a cura di. Syringomyelia: Current Concepts in Pathogenesis and Management. Springer, 2001.

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26

Tamaki, N., U. Batzdorf e T. Nagashima. Syringomyelia: Current Concepts in Pathogenesis and Management. Springer London, Limited, 2012.

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27

Syringomyelia: Current Concepts in Pathogenesis and Management. Springer, 2013.

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28

Chiari-like Malformation and Syringomyelia in the Cavalier King Charles Spaniel. Utrecht University, 2007.

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29

Batzdorf, Ulrich. Current Neurosurgical Practice: Syringomyelia Current Concepts in Diagnosis and Treatment (Current Neurosurgical Practice Series). Williams & Wilkins, 1991.

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30

Publications, ICON Health. The Official Patient's Sourcebook on Syringomyelia: A Revised and Updated Directory for the Internet Age. Icon Health Publications, 2002.

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31

News, PM Medical Health. 21st Century Complete Medical Guide to Syringomyelia, Authoritative Government Documents, Clinical References, and Practical Information for Patients and Physicians. Progressive Management, 2004.

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32

Boudreau, Eric, e Brian Egan. Anesthetic Management of Chiari Decompression. A cura di Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi e Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0028.

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Abstract (sommario):

Chiari malformation is a congenital neurodevelopment abnormality affecting the cerebellum and the brainstem. This chapter looks at the etiology, history, diagnostic criteria, physical exam findings, pathophysiology, and treatment for this disease process from the perspective of an anesthesiologist. The signs and symptoms stemming from Chiari malformations including syringomyelia, respiratory dysfunction, and vasomotor instability will be discussed. Preoperative considerations will be examined as well as ways to optimize these patients prior to surgery. Intraoperative anesthetic goals for patients undergoing Chiari decompression will be presented and postoperative objectives will be examined in detail. The ultimate purpose of this chapter is to review relevant literature and put forth a set of recommendations regarding the anesthetic care of complicated patients undergoing intracranial surgery.

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33

Mason, Peggy. Spinal Cord. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190237493.003.0004.

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The spinothalamic and lemniscal pathways carry somatosensory information from the periphery into the brain while the corticospinal pathway carries motor commands from the brain to motoneurons of the spinal cord. Following these pathways through the spinal cord allows the student to infer lesion location from symptoms. To exemplify the clinical importance of sympathetic outputs from thoracic segments, Horner syndrome is described. Similarly, the common problems caused by spinal cord injury on sacral parasympathetic functions are stressed. The contributions of specific spinal segments to breathing, hand and foot dexterity, and micturition are emphasized. Working through the logic of the symptoms caused by spinal hemisection (Brown-Séquard syndrome), pyramidal stroke, and syringomyelia provides the student with a clear framework for understanding spinal function in the clinical context.

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34

Brock, M., K. A. Bushe e M. Klinger. Stabilizing Craniocervical Operations: Calcium Antagonists in Sah : Current Legal Issues (Deutsche Gesellschaft Fur Neurochirurgie // Proceedings of the Annual Congress). Springer-Verlag, 1990.

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35

(Editor), K. A. Bushe, Mario Brock (Editor) e Margareta Klinger (Editor), a cura di. Stabilizing Craniocervical Operations. Calcium Antagonists in SAH. Current Legal Issues: Proceedings of the 40th Annual Meeting of the Deutsche Gesellschaft ... May 7-10, 1989 (Advances in Neurosurgery). Springer, 1990.

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36

Roser, F., e M. Tatagiba. Syringomyelie und Syringobulbie: H6 Therapie und Verlauf Neurologischer Erkrankungen. Kohlhammer Verlag, 2013.

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37

Roser, F., e M. Tatagiba. Syringomyelie und Syringobulbie: H6 Therapie und Verlauf Neurologischer Erkrankungen. Kohlhammer Verlag, 2013.

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38

Hydromyelie Syringomyelie und Gliose: Anatomische Untersuchungen über IHRE Histogenese Heft 72. Springer London, Limited, 2013.

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39

Scadding, John. Neuropathic pain. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0386.

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Abstract (sommario):

Pain signalled by a normal sensory system, nociceptive pain, serves a vital protective function. The peripheral and central nervous somatosensory systems permit rapid localization and identification of the nature of painful stimuli, prior to appropriate action to minimize or avoid potentially tissue damaging events. A reduction or absence of pain resulting from neurological disease emphasizes the importance of this normal protective function of pain. For example, tissue destruction occurs frequently in peripheral nerve diseases which cause severe sensory loss such as leprosy, and in central disorders such as syringomyelia. Neuropathic pain results from damage to somatosensory pathways and serves no protective function. This chapter provides an overview of neuropathic pain, considering its context, clinical features, pathophysiology, and treatment.In the peripheral nervous system, neuropathic pain is caused by conditions affecting small nerve fibres, and in the central nervous system by lesions of the spinothalamic tract and thalamus, and rarely by subcortical and cortical lesions. The clinical feature common to virtually all conditions leading to the development of neuropathic pain is the perception of pain in an area of sensory impairment, an apparently paradoxical situation. The exception is trigeminal neuralgia.Neuropathic pain is heterogeneous clinically, aetiologically, and pathophysiologically. Within a given diagnostic category, whether defined clinically or aetiologically, there are wide variations in reports of pain by patients. This heterogeneity poses one of the greatest challenges in understanding the mechanisms of neuropathic pain. Knowledge of the pathophysiology is an obvious pre-requisite to the development of effective treatments. The goal of a pathophysiologically based understanding of the symptoms and signs of neuropathic pain is, of course, just such a rational and specific approach to treatment. While this is not yet achievable, clinical-pathophysiological correlations have led to some recent advances in treatment.

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