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Letteratura scientifica selezionata sul tema "Syndrome MARCH"

Autore: Grafiati

Pubblicato: 25 gennaio 2025

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Articoli di riviste sul tema "Syndrome MARCH"

Freda, Benjamin J. "Cardiorenal syndrome, March 2018". Cleveland Clinic Journal of Medicine 85, n. 5 (maggio 2018): 360. http://dx.doi.org/10.3949/ccjm.85c.05001.

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BLASIER, DALE, RICHARD J. BARRY e TERRY WEAVER. "Forced March-Induced Peroneal Compartment Syndrome". Clinical Orthopaedics and Related Research &NA;, n. 284 (novembre 1992): 189???192. http://dx.doi.org/10.1097/00003086-199211000-00026.

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Author, No. "March Of Medicine". Journal of Nepal Medical Association 3, n. 3 (1 gennaio 2003): 240–42. http://dx.doi.org/10.31729/jnma.1061.

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1. Pigmentation due to Phenothiazines. Santove A. JAMA 1965, 191. 263.2. Paterson-Kelly Syndrome in Adolescence. Crawfurd M.D'A; Jacobs A; Murphy B; Peters D.K. Brit. med. J. 1965, 1, 693.3. Allergy to Penicillin. Van Arsdel P.P. JNMA 1965, 191. 2384. Gluten-sensitive Enteropathy and Eczema. Friedman M. Hare P.J. Lancet 1965, i, 5215. Jaundice wiht Oral Contraceptive. Culberg G., Lundstrom R, Stenram U. Brit. med. J. 1965, I, 695.

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Author, No. "March Of Medicine". Journal of Nepal Medical Association 3, n. 2 (1 gennaio 2003): 141–49. http://dx.doi.org/10.31729/jnma.962.

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1. Cutaneous Reactions to Antibiotics. Rees. B. Rees. JAMA, 1964, 189: 685.2. The 'Pill' and Thrombosis. Brit. med. J. 1964,2, 1089.3. Sensitivity to Cow's Milk. Heiner D.C., Wilson J.F.& Lahey M.E. JAMA 1964. 189; 563. 4. Deficiency State Associated with Isoniazid therapy. Aspinall D.L. Brit. med. J., 1964.2 1177.5 . Immunological Aspects of Cancer. Woodruff M.F.A. Lancet, 1964,2,265.6. Childhood Thyrotoxicosis. Saxena K.M. Craford J.M., & Talbot N.B. Brit. med. J., 1964,2,1153.7. Stevens-Johnson Syndrome from Long-Acting Sulphonamides. Leading Article, Brit. med. J., 1974,2,1410.

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Yasudo, Hiroki, Kiwako Yamamoto-Hanada, Limin Yang, Mayako Saito-Abe, Miori Sato, Yumiko Miyaji, Mami Shimada et al. "Pollen Food Allergy Syndrome in Allergic March". Nutrients 14, n. 13 (27 giugno 2022): 2658. http://dx.doi.org/10.3390/nu14132658.

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The association between pollen food allergy syndrome (PFAS) and allergic march remains unclear. In this prospective cohort study of the general population in Tokyo (T-Child Study), we found that sensitization to Cry j 1 and Fel d 1 at ages 5 and 9 years was associated with an increased risk of PFAS at 13 years old (at 5 years, Cry j 1: adjusted odds ratio aOR, 2.74; 95% confidence interval CI, 1.53–4.91; Fel d 1: aOR, 2.61; 95% CI, 1.31–5.19; at 9 years, Cry j 1: adjusted odds ratio aOR, 4.28; 95% confidence interval CI, 1.98–9.25; Fel d 1: aOR, 2.40; 95% CI, 1.33–4.32). In particular, sensitization to Bet v 1 at ages 5 and 9 years was associated with a strong risk of PFAS at the age of 13 years (at 5 years: aOR, 10.6; 95% CI, 2.64–42.5; at 9 years: aOR, 9.1; 95% CI, 4.71–17.6). PFAS risk by age 13 years was increased by any allergic symptom at 5 or 9 years, a combination of wheezing, eczema, and rhinitis, and Bet v 1 sensitization. Our findings suggest that PFAS may be associated with allergic march.

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Saha, Debasish Kumar, Madhurima Saha e Suraiya Nazneen. "Lateral Medullary Syndrome". Bangladesh Critical Care Journal 5, n. 1 (11 maggio 2017): 72–73. http://dx.doi.org/10.3329/bccj.v5i1.32548.

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Caporale, Christina M., Francesca Notturno, Massimo Caulo e Antonino Uncini. "Capsular warning syndrome mimicking a jacksonian sensory march". Journal of the Neurological Sciences 285, n. 1-2 (ottobre 2009): 262–64. http://dx.doi.org/10.1016/j.jns.2009.07.006.

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Alzahrani, Abdullah, Stephanie A. Kujawski, Glen R. Abedi, Safaa Tunkar, Holly M. Biggs, Nada Alghawi, Hani Jokhdar, Abdullah M. Assiri e John T. Watson. "Surveillance and Testing for Middle East Respiratory Syndrome Coronavirus, Saudi Arabia, March 2016–March 2019". Emerging Infectious Diseases 26, n. 7 (luglio 2020): 1571–74. http://dx.doi.org/10.3201/eid2607.200437.

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Li, Qing, Guo-Yan Yang e Jian-Ping Liu. "Syndrome Differentiation in Chinese Herbal Medicine for Irritable Bowel Syndrome: A Literature Review of Randomized Trials". Evidence-Based Complementary and Alternative Medicine 2013 (2013): 1–9. http://dx.doi.org/10.1155/2013/232147.

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Traditional Chinese medicine (TCM) has been commonly used for irritable bowel syndrome (IBS). Syndrome differentiation is one of the important characteristics of TCM. To assess the application and basic characteristics of syndrome differentiation in randomized controlled trials (RCTs) of Chinese herbal medicine for IBS, we performed this paper. We conducted electronic searches in main Chinese and English databases till March 2012. A total of 735 RCTs involving 67,784 IBS participants were included. 224 (30.5%) studies applied syndrome differentiation. The major syndromes of IBS patients were the syndrome of liver stagnation and spleen deficiency (56.8%), spleen-stomach weakness (49.4%), spleen-kidney yang deficiency (48.1%), and cold and heat in complexity (29.6%). Herbal formulas were prescribed based on syndrome differentiation in 202 studies. Chinese patent medicine was more commonly used in studies that only enrolled patients with a specific syndrome. 15 studies compared the therapeutic effect among different syndromes, of which 6 studies showed that there were significant differences among different syndromes. The low use of TCM syndrome differentiation in randomized trials of Chinese herbal medicine for IBS results in the poor pertinence of treatment. TCM syndrome differentiation should be used in further studies at the stage of recruitment, treatment, and data analyses.

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NUNNS, D., e I. M. SYMONDS. "Vulval Pain Syndrome Study Day, Derby, 5 March 1999". Journal of Obstetrics and Gynaecology 19, n. 5 (gennaio 1999): 566–68. http://dx.doi.org/10.1080/01443619964571.

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Più fonti

Tesi sul tema "Syndrome MARCH"

Leleu, Ambre. "Les organoïdes cérébraux : nouvelle plateforme pour la modélisation de pathologies neurodéveloppementales et neurodégénératives". Electronic Thesis or Diss., université Paris-Saclay, 2024. http://www.theses.fr/2024UPASL110.

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Les désordres du système nerveux central sont des pathologies qui peuvent se développer dès le neurodéveloppement du fœtus jusqu'à la mort de l'individu. La capacité d'évaluer la façon dont les cellules humaines reprogrammées (iPSCs) peuvent se développer et s'autoorganiser en trois dimensions en se transformant en organoïdes cérébraux a le potentiel de révolutionner notre approche à la fois de la recherche scientifique fondamentale et du traitement des maladies neurologiques humaines qui s'avèrent désormais comme la première cause de morbidité mondiale Nous avons exploré ce potentiel pour modéliser deux de ces pathologies : le syndrome MARCH, un syndrome neurodéveloppemental caractérisé par une hydranencéphalie, et la maladie d'Alzheimer, pathologie neurodégénérative. Nous décrivons la mise en place de ces modèles pathologiques ainsi que leurs avantages pour reproduire les symptômes physio-pathologiques associés, tout en soulignant les limites inhérentes à l'utilisation de cellules souches pluripotentes pour modéliser ces pathologies complexes
Central nervous system disorders are pathologies that can develop from fetal neurodevelopment through to individual death. The ability to assess how reprogrammed human cells (iPSCs) can develop and self-organize in three dimensions into brain organoids has the potential to revolutionize our approach to both basic scientific research and the treatment of human neurological diseases, now emerging as the leading cause of overall disease burden in the world. We have explored this potential to model two of these pathologies: MARCH syndrome, a neurodevelopmental syndrome characterized by hydranencephaly, and Alzheimer's disease, a neurodegenerative pathology. We describe the set-up of these pathological models and their advantages in reproducing the associated physio- pathological symptoms, while highlighting the inherent limitations of using pluripotent stem cells to model these complex pathologies

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Chauveau, Aurélie. "Identification des mutations à visée diagnostique et pronostique dans les néoplasies myéloprolifératives et impact sur l'épissage alternatif Sequential analysis of 18 genes in polycythemia vera and essential thrombocythemia reveals an association between mutational status and clinical outcome, in Genes chromosomes & cancer 56(5), May 2017 Benefits and pitfalls of pegylated interferon-α2a therapy in patients with myeloproliferative neoplasm-associated myelofibrosis: a French Intergroup of Myeloproliferative neoplasms (FIM) study, in Haematologica 103, March 2018". Thesis, Brest, 2019. http://www.theses.fr/2019BRES0042.

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Les néoplasies myéloprolifératives (NMP), non BCR-ABL1, regroupent principalement la polyglobulie de Vaquez (PV), la thrombocytémie essentielle (TE) et la myélofibrose primitive (MFP).Ces pathologies partagent, dans des proportions variables, une mutation commune, la mutation JAK2 V617F. La protéine JAK2 mutée a une activité tyrosine kinase constitutive, impliquée dans le développement de la maladie. Cette mutation, seule, n’explique pas l’hétérogénéité phénotypique au sein des NMP. L’avènement des techniques de séquençage haut débit a permis de mieux appréhender la physiopathologie. Notre travail avait pour objectif l’identification de mutations additionnelles au sein de deux cohortes suivies au long cours en lien avec un risque d’aggravation de la maladie, l’une regroupant des patients en phase chronique (TE et PV JAK2 V617F), la seconde regroupant des patients avec une myélofibrose traitée par interféron. A l’instar d’autres travaux récents, nous avons montré que le nombre de mutations et la présence de mutations additionnelles sont associés à l’évolution de la maladie, voire à la réponse au traitement.Parmi les mutations identifiées, certaines pourraient influencer l’épissage. La deuxième partie de ce travail a donc consisté à étudier l’épissage alternatif en fonction des mutations présentes, et en particulier la mutation JAK2 (V617F) et de manière globale dans les TE. Un saut de l’exon 14 de JAK2 a été décrit chez des patients NMP présentant, ou non, la mutation JAK2 V617F. Cette mutation du gène JAK2 est prédite pour altérer un site de fixation de la protéine SRSF6 régulatrice de l’épissage. Nous observons que le saut de l’exon 14 est un événement peu fréquent chez les patients, modulé en partie par l’expression des protéines SR. L’analyse transcriptomique montre une grande hétérogénéité entre les patients en termes d’expression et d’épissage, ce qui ne nous a pas permis de mettre en évidence de profil caractéristique. Ces résultats soulignent l’importance de l’identification des mutations additionnelles au diagnostic et au cours du suivi.Nous avons pu, en outre, identifier quelques transcrits alternatifs associés à la présence de ces mutations. Le rôle fonctionnel de ces variants reste à définir
Polycythemia vera (PV), essential thrombocythemia (ET) and primary myelofibrosis (PMF) are a group of Philadelphia-negative myeloproliferative neoplasm (MPN). These diseases share a common mutation, JAK2 V617F, in varying proportions. The mutated JAK2 protein has a constitutive tyrosine kinase activity, implicated in the physiopathology of MPN. This mutation alone does not explain the phenotypic heterogeneity within MPN.High throughput sequencing techniques helped understanding the physiopathology. This work aimed to identify additional mutations in two patient cohorts related to the aggravation risk of the disease. The first one consisted of patients in chronic phase (JAK2 V617F ET and PV), the second consisted in patients with myelofibrosis treated with interferon. Like other studies, we have shown that the number of mutations and the presence of additional mutations are associated with disease progression or with response to treatment. Some identified mutations could influence splicing. The second part of this work aimed at studying the putative impact of the JAK2 V617F mutation, on alternative splicing (AS).We also analyzed global AS profiles in ET. JAK2 exon 14 skipping has been described in NMP patients with or without the JAK2 V617F mutation.This mutation was predicted to alter the binding site of the SRSF6 splice-regulating protein. We observed that exon 14 skipping was an uncommon event in patients, in part related to SR protein expression. In addition, our transcriptomic-wide analysis showed a great heterogeneity between the patients with respect to both gene expression and splicing. This prevented us from identifying any characteristic profile. These results underscore the importance of identifying additional mutations at diagnosis and during follow-up. We have also been able to uncover some alternative transcripts associated with the presence of these mutations.The functional role of these variants remains to be defined

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Combes, Pierre. "Freezing a la marche et syndromes extra-pyramidaux". Toulouse 3, 1994. http://www.theses.fr/1994TOU31071.

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DENANCY, EDOUARD. "Syndrome de larsen : acquisition de la marche : une etape essentielle ; a propos d'un cas". Amiens, 1991. http://www.theses.fr/1991AMIEM037.

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Mui, Kin-cheong, e 梅堅祥. "Inactivation of DNA match repair proteins in premalignant lesions in Lynch syndrome". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B44659635.

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Herz, Damian Marc [Verfasser]. "Charakterisierung neuronaler oszillatorischer Aktivität bei Patienten mit idiopathischem Parkinson-Syndrom / Damian Marc Herz". Köln : Deutsche Zentralbibliothek für Medizin, 2011. http://d-nb.info/1012599272/34.

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Faim, Ferreira Samantha 1985. "Efeitos da exposição sub-aguda de manganês sobre a marcha em ratos". [s.n.], 2014. http://repositorio.unicamp.br/jspui/handle/REPOSIP/312460.

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Orientador: Alan Stewart Hazell
Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas
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Resumo: A neurotoxicidade por manganês (Manganismo) leva a uma disfunção neurológica caracterizada pelo desenvolvimento de ataxia, hipocinesia, rigidez e tremores. Evidências sugerem que os astrócitos desempenham um papel importante na disfunção cerebral nesta desordem, pois acumulam manganês e sequestram o metal na mitocôndria, o que inibe a oxidação fosforilativa. A exposição aguda ao manganês leva ao acúmulo focal do metal e perda neuronal no cerebelo. No entanto, a relação entre esta deposição localizada de manganês e as manifestações neurológicas não são claras. Neste estudo, foram caracterizados os efeitos do manganês na marcha e no equilíbrio após um tratamento subagudo em ratos adultos da linhagem Sprague-Dawley (CEMIB ¿ UNICAMP). Os animais foram divididos em três grupos: Controle; tratado com Mn e tratado com Mn + NAC (N - acetilcisteína), um anti-oxidante. Os ratos do grupo Mn receberam cloreto de manganês (II) (50 mg/kg de peso corporal, i.p.) diariamente durante 4 dias, enquanto os ratos do grupo Mn + NAC foram co-tratados diariamente com o cloreto de manganês (II) e NAC (163 mg/kg, i.p.). Na análise da marcha o grupo tratado com Mn demonstrou alteração na marcha, vizualizados pela diminuição da área da impressão plantar (comprimento x largura), do comprimento da passada e da base de suporte. Apesar das alterações observadas nesses parâmetros, os animais não apresentaram mudanças na pressão exercida pela pata durante a marcha. O grupo co-tratado com NAC não demonstrou essas alterações, apresentando-se semelhante ao controle. Nos estudos de imonohistoquímica, imunofluorescência, histoquímica e Western blotting o grupo tratado com Mn apresentou morte neuronal, aumento da reatividade astrocítica na camada granular, desarranjo na camada das células de Purkinje e aumento na expressão do transportador de glutamato GLT-1a. Estes resultados corroboram com as importantes alterações na função motora de animais tratados com Mn. O co-tratamento com o antioxidante NAC foi capaz de impedir parcialmente esses danos, exercendo uma ação protetora na área do cerebelo e na função motora. Em conclusão, demonstramos que a intoxicação por manganês gera alterações morfo-funcionais no cerebelo, as quais podem ser principalmente revertidos pelo uso do antioxidante NAC
Abstract: Manganese (Mn) neurotoxicity (Manganism) leads to a neurological disorder characterized by the development of ataxia, hypokinesia, rigidity and tremors. Evidence suggests that astrocytes play an important role in brain dysfunction in this disorder because accumulate manganese and sequester metal in the mitochondria, where it inhibits oxidative phosphorylation. Acute exposure to manganese leads to focal accumulation of the metal and neuronal loss in the cerebellum. However, the relationship between this localized deposition of manganese and neurological manifestations are unclear. In this study, we characterized the effects of manganese in gait and balance after a subacute treatment in Sprague - Dawley rats (CEMIB - UNICAMP). The animals were divided into three groups: control, treated with Mn and Mn + treated with NAC (N - acetylcysteine). The rats from manganese group were administered with manganese (II) chloride (50 mg / kg body weight , ip) daily for 4 days, whereas rats of the group Mn + NAC daily were co- treated with manganese chloride (II) and NAC (163 mg / kg , ip). In Catwalk test group treated with manganese showed changes in gait and balance, leading to a reduction of the area of the paw (length and width), the stride length and the base of support. Despite the changes observed in these parameters, the animals showed no changes in pressure exerted by the leg during gait. The group co-treated with NAC showed no such changes, keeping similar to the control group. In studies of immunohistochemistry, immunofluorescence, western blotting and histochemistry group treated with showed neuronal death, an increase in signal astrocytic reactivity in the granular layer, a derangement in the Purkinje cell layer and an increase in the expression of glutamate transporter GLT - 1a. These results suggest significant changes in motor function in animals treated with Mn. The co - treatment with the antioxidant NAC was able to partially prevent this damage, exerting a protective action in the area of the cerebellum and motor function. In conclusion, we demonstrated that manganese poisoning produces morphological and functional changes in the cerebellum, which can principally be reversed by the use of the antioxidant NAC
Mestrado
Fisiopatologia Médica
Mestra em Ciências

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Reis, Julia Guimarães. "Analise clinica e funcional da instabilidade patelofemoral objetiva". [s.n.], 2008. http://repositorio.unicamp.br/jspui/handle/REPOSIP/312040.

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Orientador: Sergio Rocha Piedade
Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas
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Resumo: Das anormalidades que envolvem a articulação do joelho, a disfunção do aparelho extensor é um dos problemas mais freqüentemente encontrados na prática ortopédica. Para abranger toda a complexidade de investigação da locomoção necessita-se de informações qualitativa e quantitativa da cinética (momentos e forças) e cinemática (ângulos). O objetivo do estudo foi identificar e analisar as alterações biomecânicas dos indivíduos com instabilidade patelofemoral objetiva durante a marcha. A amostra foi composta de 10 indivíduos com instabilidade patelofemoral (grupoI), média de idade de 25,6 (±7,6) anos, média de altura de 1,63 (±0,06) m e média de peso de 63,3 (±13,52) kg; e, 14 indivíduos sem história de lesão músculo-esquelética (grupo controle ou grupo II), com média de idade de 24,14 (±2,71) anos, média de altura de 1,63 (±0,05) m e média de peso de 59,43 (±10,02) kg. Ambos os grupos foram submetidos a uma análise cinemática e cinética, onde os mesmos caminharam em velocidade livre, numa passarela de 10 m de comprimento. As imagens foram filmadas por seis câmeras do sistema Qualysis, que capturou os sinais de marcadores reflexíveis posicionados no membro inferior das voluntárias. Paralelamente, aplicou-se no grupo I uma avaliação clínica do grau de funcionalidade dos joelhos lesados, onde a pontuação os classificou como funcionalmente ruins; e, um exame físico, onde ambos os membros apresentaram-se estatisticamente semelhantes. A análise dos dados cinemáticos e cinéticos foi realizada pelo programa Qgait que mostrou menor flexão de joelho, nas fases de apoio e balanço (p<0,0001); menor momento extensor de joelho, no apoio (p<0,0001); e, maior força de reação do solo (p=0,4094), no grupo de pacientes em relação ao controle. Foram avaliados também parâmetros espaços-temporais como velocidade (p=0,0053), cadência (p=0,0376) e comprimento da passada (p=0,0021), onde o grupo I apresentou valores inferiores comparado ao grupo controle. Já no período de apoio (p=0,1186), o grupo I superou o grupo II. Estes resultados sugerem que o grupo I utilizou várias estratégias durante a marcha, na tentativa de reduzir a dor e a pressão na articulação patelofemoral. Entretanto, a força de reação do solo não foi reduzida, o que poderá resultar em danos a outras articulações, em longo prazo, devido a cargas repetitivas na articulação tíbiofemoral
Abstract: Abnormalities involving knee joint, the dysfunction of the extensor apparatus, it is one of the problems most often found in orthopaedic practice. To cover the full complexity of of locomotion research, it is necessary qualitative and quantitative information of kinetic (moments and forces) and kinematics (angles). The objective of the study was to identify and analyse the biomechanical changes of individuals with objective patellofemoral instability during gait. The sample was composed of 10 individuals with patellofemoral instability (group I), mean age of 25.6 (± 7.6) years, the average height of 1.63 (± 0.06) m and mean weight of 63.3 (± 13 , 52) kg; and 14 individuals with no history of musculo-skeletal injury (control group or group II), with an average age of 24.14 (± 2.71) years, the average height of 1.63 (± 0.05) m and mean weight of 59.43 (± 10.02) kg. Both groups were subjected to an analysis kinematics and kinetics, where they walked naturally on a 10 m walkway. The images were filmed by six cameras Qualysis system, which captured the signs of reflective markers placed on the lower limb of the volunteers. Paralely, was applied in the group I a functional clinical assessment of the degree functionality in the knee injured that classified it as bad functionally; and, physical examination, where both limbs showed up statistically similar. The analysis of kinematic and kinetic data was performed by the Qgait program that showed less of knee flexion, in the stance and balance phase (p <0.0001), less knee extensor moment, in support (p <0.0001); and, greatest ground reaction force (p = 0.4094) in the group of patients with respect to control. They were also assessed spatiotemporal parameters such as speed (p = 0.0053), cadence (p = 0.0376) and stride length (p = 0.0021), where the group I showed lower values compared to control group. Already in the support period (p = 0.1186), group I overcame the group II. These results suggest that the group I used several strategies during gait, in an attempt to reduce the pain and pressure in patellofemoral joint. However, the ground reaction force was not reduced, which could result in damage to other joints, in the long term, due to repetitive loads in tibiofemoral joint
Mestrado
Pesquisa Experimental
Mestre em Cirurgia

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Baillieul, Sébastien. "Syndrome d'apnées du sommeil et cerveau : une relation bidirectionnelle Continuous positive airway pressure improves gait control in severe obstructive sleep apnoea: A prospective study Hypoxic conditioning and the central nervous system: A new therapeutic opportunity for brain and spinal cord injuries?" Thesis, Université Grenoble Alpes, 2020. https://thares.univ-grenoble-alpes.fr/2020GRALS025.pdf.

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Les contraintes physiologiques cérébrales rendent le cerveau humain vulnérable à l'hypoxie, qu’elle soit environnementale (haute altitude) ou en lien avec une pathologie hypoxémiante. Parmi ces pathologies, et en raison de sa forte prévalence dans la population générale, le syndrome d'apnées obstructives du sommeil (SAOS) est un modèle physiopathologique reconnu des effets délétères de l'hypoxie sur le cerveau. Les épisodes cycliques d'apnées et d'hypopnées survenant au cours du sommeil qui caractérisent le SAOS entraînent une hypoxie intermittente, une fragmentation du sommeil et des fluctuations de la pression intra-thoracique, tous trois facteurs déclenchant des mécanismes intermédiaires contribuant au développement de maladies cardio-métaboliques ainsi que des répercussions cérébrales (troubles cognitifs et accidents vasculaires cérébraux (AVC)). Ce travail de thèse explore la relation bidirectionnelle entre les syndromes d’apnées du sommeil (SAS) et le cerveau. Le premier axe se concentre sur les conséquences neurocognitives du SAOS au travers du contrôle de la marche. Les répercussions neurocognitives du SAOS sont à ce jour bien décrites et des troubles de la marche ont récemment été mis en évidence, avec une relation de type dose-réponse entre la gravité du SAOS et la sévérité des troubles de la marche. Il a ainsi été suggéré que la marche pouvait représenter un marqueur des répercussions cérébrales du SAOS. Les effets du traitement par pression positive continue (PPC) sur le contrôle de la marche ont été investigués au cours de ce travail de Thèse, avec des résultats contrastés. Dans une première étude prospective contrôlée, 8 semaines de traitement par PPC entraînaient une amélioration du contrôle de la marche chez des patients atteints de SAOS sévère (Baillieul et al., 2018, Plos One). Afin de valider ces résultats et d'étudier les corrélats neurophysiologiques du lien entre marche et SAOS, nous avons mené un essai contrôlé randomisé étudiant l'impact de 8 semaines de traitement par PPC comparativement à la Sham-PPC (Baillieul et al., 2020, Soumis). Contrairement à notre hypothèse, nous n'avons constaté aucune amélioration du contrôle de la marche dans le groupe PPC, résultat corroboré par l'absence d'impact de la PPC sur les déterminants du contrôle de la marche. Le deuxième axe est centré sur les répercussions cérébro-vasculaires des SAS. SAS et AVC sont deux pathologies graves et étroitement liées, le SAS pouvant être à la fois cause et conséquence potentielle des AVC. Le présent travail est axé sur l'identification des traits phénotypiques de SAS chez les patients post-AVC, afin d'en améliorer le diagnostic (Baillieul et al., en préparation). Le dépistage du SAS post-AVC est crucial en raison du risque élevé de morbi-mortalité et de conséquences fonctionnelles associées au SAS après AVC, mais il ne peut être effectif sans une identification plus précise des patients à risque de SAS. Le troisième axe a été conçu comme une perspective qui servira au développement du deuxième axe. Dans ce dernier axe, le potentiel de l'imagerie cérébrale et en particulier de l'imagerie par résonance magnétique pour développer des marqueurs de récupération et étudier les mécanismes physiopathologiques des déficiences liées aux AVC sont présentés, au travers de la marche et de son contrôle. Les corrélats neuronaux de la marche comme activité post-AVC sont mis en évidence, en utilisant une approche de type Voxel-based lesion-symptom mapping (Baillieul et al., 2019, Hum. Mov. Sci.). Les marqueurs d'imagerie basés sur l’utilisation du Diffusion tensor imaging pour prédire la récupération de la marche post-AVC sont également présentés (Soulard et al., 2019, Neurology). Ces travaux sur les marqueurs d'imagerie cérébrale de la récupération post-AVC serviront à développer des outils pour les recherches à venir sur les corrélats neuronaux des SAS post-AVC
The human brain is a perfect example of our dependence on oxygen. Brain physiological constraints render it vulnerable to hypoxia, such as encountered in environmental conditions (high altitude exposure) or pathological hypoxemic conditions. Among those pathological conditions, and due to its high prevalence in general population and the various levels of hypoxia resulting of the different degrees of severity of the pathology, obstructive sleep apnoea syndrome (OSAS) is a pathophysiological model of choice to investigate the detrimental effects of hypoxia on the brain. The cyclical, repeated episodes of apnoea and hypopnea during sleep that characterize OSAS result in intermittent hypoxia, sleep fragmentation and fluctuations in intrathoracic pressure, which are stressors that triggers mechanisms contributing to the initiation and progression of life-threatening cardiometabolic diseases, as well as several brain repercussions, such as cognitive impairment and stroke. This Thesis work explores the bidirectional relationship between sleep apnoea syndromes (SAS) and the brain. The first axis is focused on the neurocognitive consequences of OSAS through the lens of gait control. The neurocognitive signature of OSAS has been thoroughly investigated but recently, gait impairments have been highlighted in severe OSAS, with dose-response relationship between OSAS severity and the magnitude of gait impairments. As gait control relies at least partly on frontal lobe functions, it has been suggested that gait could represent a marker of OSAS brain repercussions. We investigated the effects of continuous positive airway pressure (CPAP) treatment on gait control, with contrasting results. In a first prospective controlled study, eight weeks of CPAP improved gait control in severe OSAS patients (Baillieul et al., 2018, Plos One). In order to validate those results and investigate the neurophysiological correlates of the link between gait control and OSAS, we conducted a randomized controlled trial which investigated the impact of an 8-week CPAP treatment compared to sham-CPAP on gait control in severe OSAS patients (Baillieul et al., 2020, Submitted). Contrary to our hypothesis, we found no improvement in gait control in the CPAP group and this result is substantiated by the absence of impact of CPAP on the determinants of gait control, further illustrating the complexity of the OSAS-neurocognitive relationship. The second axis is focused on the cerebrovascular repercussions of SAS. SAS and stroke are both severe intertwined conditions, SAS being both cause and potentially consequence of stroke. The present work is focused on the identification of phenotypic traits of SAS in post-stroke patients, to improve diagnosis of SAS following stroke (Baillieul et al., in preparation). Screening stroke patients for SAS is crucial due to the high risk of morbimortality and functional consequences associated to SAS following stroke but cannot be achieved without a more accurate identification of patients at risk to develop SAS following stroke. The third axis has been conceived as a perspective that will serve the development of the second axis. In this last axis, the potential of brain imagery and in particular magnetic resonance imagery to develop markers of stroke recovery as well as investigate the pathophysiological mechanisms underlying stroke-related deficiencies are presented, with a specific focus on gait and walking activity. The neural correlates of walking activity following stroke are highlighted, using a voxel-based lesion-symptom mapping approach (Baillieul et al., 2019, Hum. Mov. Sci.). Imagery markers of walking recovery following stroke using diffusion tensor imaging are also presented (Soulard et al. 2019, Neurology). This work on brain imagery markers of stroke recovery will further serve the development of investigations focused on the neural correlates of SAS following stroke

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Moura, Thayse de Lucena e. "Efeitos da inclina??o da esteira na marcha de crian?as com S?ndrome de Down". Universidade Federal do Rio Grande do Norte, 2009. http://repositorio.ufrn.br:8080/jspui/handle/123456789/16679.

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Made available in DSpace on 2014-12-17T15:16:07Z (GMT). No. of bitstreams: 1 ThayseLM.pdf: 2358410 bytes, checksum: 2b6dfd18cdd5c0c7624aef8c0dfc4278 (MD5) Previous issue date: 2009-11-09
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Background: Down syndrome (DS) is a genetic alteration characterized by being a nonprogressive congenital encephalopathy. Children with DS have hypotonia and developmental delays that interfere in the movement`s acquisition for these children. Objective: Analyze the effects of treadmill inclination on angle and spatiotemporal gait characteristics of these individuals. Methodology: We studied 23 subjects of both sexes, with ages ranged between 05 and 11 years, they presented ability to walk on level 5 classified according to the Functional Ambulation Category (FAC). Initially held a subjective evaluation of balance through a questionnaire (Berg Balance Scale-BBS) then the kinematic gait analysis was realized on a treadmill first, without inclination and then, with inclination of 10%, using the motion system analysis Qualisys System. Data analysis was done using BioStat 5.0 attributing significance level of 5%. Normality of data was verified using D'Agostino test and later was applied paired t-test to compare data in two experimental conditions. Results: There was a statistically significant difference in the spatiotemporal variables: reduction in the cadence (from 108.92 ? 39.07 to 99.11 ? 27.51, p <0.04), increase in cycle time (from 1.24 ? 0.27 to 1.36 ? 0.34, p = 0.03 ) and increase in time to take stock (from 0.77 ? 0.15 to 0.82 ? 0.18, p <0.001). Angular variables that showed statistically significant increasing were: the hip in the initial contact (12.23 ? 4.63 to 18.49 ? 5.17, p <0.0001) and max. flexion in balance (12.96?4:32 to 19.50 ? 4.51, p <0.0001 ), knee in the initial contact (15.59 to ? 6.71 to 21.63 ? 6.48, p <0.0001), the ankle in the initial contact (-2.79 ? 9.8 to 2.25 ? 8.79, p <0.0001), max dorsiflexion in stance (4.41 ? 10.07 to 7.13 ? 11.58, p <0.0009), maximum plantar flexion in the pre-assessment of the ankle joint (increase of -6.33 ? 8.77 to -2.69 ? 8.62, p <0.0004).Conclusions: The inclination acts in a positive way for angular and spatiotemporal features gait of children with Down syndrome, demonstrating possible benefit of using this surface in the gait rehabilitation of children with Down Syndrome
Contextualiza??o: A s?ndrome de Down (SD) ? uma altera??o gen?tica caracterizada por ser uma encefalopatia cong?nita n?o progressiva. As crian?as com SD apresentam hipotonia muscular e atraso no desenvolvimento neuropsicomotor que dificultam a aquisi??o da marcha para estas crian?as. Objetivo: Analisar os efeitos da inclina??o da esteira na marcha de crian?as com SD. Metodologia: Foram avaliados 23 sujeitos ( 9 do g?nero feminino e 14 do g?nero masculino), com m?dia de idade de 8,43 ?2,25 anos, com capacidade de deambular classificada em n?vel 5 de acordo com a Categoria de Deambula??o Funcional (FAC Functional Ambulatory Category). Inicialmente realizou-se avalia??o subjetiva de equil?brio atrav?s de question?rio (Escala de Equil?brio de Berg- BBS) em seguida, a an?lise cinem?tica da marcha em esteira el?trica sem inclina??o e com inclina??o de 10%, utilizando o sistema de an?lise do movimento Qualisys System. Para an?lise dos dados foi utilizado o programa Bioestat 5.0 atribuindo-se n?vel de signific?ncia de 5%. A normalidade dos dados foi verificada pelo teste D`Agostino e posteriormente foi aplicado o teste t-pareado para comparar os dados nas duas condi??es experimentais. Resultados: Observou-se diferen?a significante estatisticamente nas vari?veis espa?o-temporais: redu??o na cad?ncia ( de 108,92 ? 39,07 para 99,11 ? 27,51, p< 0,04) , aumento no tempo do ciclo (de 1,24 ? 0,27 para 1,36 ? 0,34, p=0,03) e aumento no tempo de balan?o (de 0,77 ? 0,15 para 0,82 ? 0,18, p< 0,001) . As vari?veis angulares que demonstraram aumento estatisticamente significante foram: quadril no contato inicial (de 12,23+4,63 para 18,49+ 5,17, p<0,0001) e m?x. flex?o no balan?o (de 12,96 ? 4,32 para 19,50 ? 4,51, p<0,0001); joelho no contato inicial (de 15,59 ? 6,71 para 21,63 ? 6,48, p< 0,0001); e tornozelo no contato inicial (de 2,79 ? 9,8 para 2,25 ? 8,79, p<0,0001), m?x. dorsiflex?o no apoio (de 4,41 ?10,07 para 7,13 ? 11,58, p<0,0009), m?x. flex?o plantar no pr?-balan?o (de 6,33 ? 8,77 para 2,69 ? 8,62, p<0,0004). Conclus?es: A inclina??o atua de forma positiva nas caracter?sticas angulares e espa?o-temporais da marcha de crian?as com S?ndrome de Down, demonstrando poss?vel benef?cio da utiliza??o deste tipo de superf?cie na reabilita??o da marcha desta popula??o

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Libri sul tema "Syndrome MARCH"

Springs, Ga ). March of Dimes International Conference on Post-Polio Syndrome (2000 Warm. March of Dimes International Conference on Post-Polio Syndrome: Identifying best practices in diagnosis & care. White Plains, NY: March of Dimes, 2001.

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Patman, Robert G. Strategic shortfall: The Somalia syndrome and the march to 9/11. Santa Barbara, Calif: Praeger Security International, 2010.

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Abrams, Estelle J. Acquired immunodeficiency syndrome (AIDS): Thirteenth update, January 1987 through March 1987 : 684 citations. [Bethesda, MD: National Library of Medicine, U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health], 1987.

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United States. National Technical Information Service., a cura di. Sick building syndrome: January 1990-March 1993 : citations from the NTIS bibliographic database. Springfield, Va: National Technical Information Service, 1993.

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International Turner Syndrome Symposium (5th 2000 Naples). Optimizing health care for Turner patients in the 21st century: Proceedings of the 5th International Turner Symposium held in Naples on 23-25 March 2000. A cura di Pasquino Anna Maria e Saenger Paul. Amsterdam: Elsevier Science, 2000.

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Angela, Harden Victoria, Risse Guenter B. 1932- e National Heart, Lung, and Blood Institute (U.S.), a cura di. AIDS and the historian: Proceedings of a conference at the National Institutes of Health, 20-21 March 1989. Bethesda, Maryland: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1991.

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Harden, Victoria Angela, e Guenter B. Risse. AIDS and the historian: Proceedings of a conference at the National Institutes of Health, 20-21 March 1989. [Bethesda, MD]: National Institutes of Health, Public Health Service, U.S. Department of Health and Human Services, 1991.

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Namibia. Ministry of Health and Social Services. Division: Expanded National HIV Response Coordination., a cura di. Progress report on the third medium plan on HIV/AIDS: April 2004-March 2006. Windhoek, Namibia: Directorate of Special Programmes, Division Expanded National HIV Response Coordination, 2006.

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1925-, Vaeth Jerome M., a cura di. Cancer and AIDS: 19th annual San Francisco Cancer Symposium, San Francisco Calif., March 2-4, 1984. Basel: Karger, 1985.

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Third World Symposium (1985 Grambling State University). Third World Symposium: Theme, Central America : epitomizing the Third World countries syndrome : March 12, 1985, Grambling State University, Grambling, Louisiana. Grambling, La: The Department, 1985.

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Zeitler, H. J., e B. Andondonskaja-Renz. "PTERIDINE LEVELS IN PATIENTS WITH AIDS (ACQUIRED IMMUNODEFICIENCY SYNDROME) AND WITH KAPOSI'S SARCOMA". In February 23–March 2, 1985, St. Christoph, Arlberg, Austria, a cura di Helmut Wachter, H. Ch Curtius e W. Pfleiderer, 593–606. Berlin, Boston: De Gruyter, 1985. http://dx.doi.org/10.1515/9783110860566-053.

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Cipolletta, Laura, Alessia Urbinati, Maria Vittoria Matassini, Marchesani Francesca, Stefano Gasparini e Alessandro Capucci. "Obstructive Sleep Apnoea Syndrome and Arrhythmia: Results of 10 Years’ Experience". In The First Outstanding 50 Years of “Università Politecnica delle Marche”, 247–64. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-33832-9_17.

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Dea, S., N. Sawyer, R. Alain e R. Athanassious. "Ultrastructural Characteristics and Morphogenesis of Porcine Reproductive and Respiratory Syndrome Virus Propagated in the Highly Permissive MARC-145 Cell Clone". In Advances in Experimental Medicine and Biology, 95–98. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1899-0_13.

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Shanmukhappa, Kumar, e Sanjay Kapil. "Cloning and Identification of MARC-145 Cell Proteins Binding to 3’ UTR and Partial Nucleoprotein Gene of Porcine Reproductive and Respiratory Syndrome Virus". In Advances in Experimental Medicine and Biology, 641–46. Boston, MA: Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-1325-4_95.

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"FIVE. The March Syndrome". In Between the Hills and the Sea, 323–76. Ithaca, NY: Cornell University Press, 2017. http://dx.doi.org/10.7591/9781501726798-006.

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Khan, Dr Imran, e Dr Balaji V. "A PROSPECTIVE CLINICAL OBSERVATION OF THROMBOTIC COMPLICATIONS IN COVID – 19". In Futuristic Trends in Medical Sciences Volume 3 Book 22, 145–52. Iterative International Publishers, Selfypage Developers Pvt Ltd, 2024. http://dx.doi.org/10.58532/v3bdms22p4ch1.

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In December of 2019, severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) began to infect humans in the city of Wuhan in the Hubei province of China. Later it rapidly spread to the rest of the world and declared as pandemic by the World Health Organization (WHO) in March of 2020.

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Zinkovsky, Daniel, e Michael R. Sood. "The Evaluation of Myocarditis in the Post-Covid-19 Era: Pearls and Perils for the Clinician". In Pericarditis - Diagnosis and Management Challenges [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.110395.

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Coronavirus disease 2019 (COVID-19), which is caused by the Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2), continues to remain a global threat since declared a pandemic by the World Health Organization in March 2020. While primarily a respiratory disease, its clinical manifestations vary widely ranging from asymptomatic infection to multi-organ failure and death. As more research becomes available, cardiovascular involvement including acute coronary syndrome, heart failure, arrhythmias, thromboembolism, myocarditis and pericarditis have been reported in both the acute infectious stage as well as the post-symptomatic period. Myocarditis is an inflammatory disease of the myocardium that can result from infectious or non-infectious causes including autoimmunity, drug and toxin exposures. This chapter discusses the incidence, pathology, diagnostic modalities, and the management of myocarditis with a special focus on the essential role of a comprehensive approach, while utilizing advanced cardiac imaging for the assessment of myocarditis in the post COVID-19 era.

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Parashar, Deeksha, e Dr P. R. Sodani. "DIGITAL CONTACT TRACING LANDSCAPE DURING COVID-19 PANDEMIC: ROLE OF MOBILE APPLICATIONS IN CONTACT TRACING". In NAVIGATING CHANGE IN HOSPITAL AND HEALTHCARE SETTING. KAAV PUBLICATIONS, 2023. http://dx.doi.org/10.52458/9789388996877.2023.eb.ch-05.

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Severe Acute Respiratory Syndrome (SARS) is a distinctive form of pneumonia recognized for its elevated transmission rate. It originated in Guangdong, China, in November 2002. A major SARS outbreak occurred in Singapore in mid-March 2003, with a connection to a traveler who had returned from Hong Kong. However, the subsequent spread through untraceable contacts ultimately led to its global dissemination, shaping the current Covid-19 pandemic [1]. Contact tracing plays a pivotal role in public health efforts to curb the spread and manage infectious diseases. By addressing the limitations of relying solely on symptomatic detection, contact tracing enhances sensitivity and community preparedness in responding to emerging epidemics.

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Wottrich, Joise, Eduardo Gonçalves, Carina Echer de Souza, Pauline Brendler Goettems Fiorin, Mirna Stela Ludwig, Thiago Gomes Heck e Matias Nunes Frizzo. "COVID-19: From Pathophysiology to Treatment". In COVID-19 Drug Development - Recent Advances, New Perspectives and Applications [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.107146.

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The new coronavirus first appeared in December 2019 in Wuhan, China, being officially named severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) by the International Committee on Taxonomy of Viruses (ICTV), as well as the name of the disease has been described as COVID-19 (coronavirus disease 2019). In March 2020, the disease was considered a global pandemic, with currently more than 514 million cases worldwide, with 6.4 million deaths. Severe cases of COVID-19 progress to acute respiratory distress syndrome (ARDS), on average about 8–9 days after the onset of symptoms. It is also worth mentioning that the severity of the disease in patients is not only due to the viral infection but also due to the host response. This phase, called a cytokine storm, reflects a state of systemic immune activation, with high levels of cytokines, such as IL-6, IL-1b, IL-2, IL-12, IL-18, TNF, and interferon gamma (IFN-γ). In this sense, the management of the disease largely depends on symptomatic and supportive treatments. For severely or critically ill patients with acute respiratory distress syndrome (ARDS) and sepsis, in addition to supplemental oxygen, mechanical ventilation, and ARDS-specific therapies, antiviral and antibiotic treatments should also be considered. Thus, the purpose of this chapter is to describe the pathophysiology and treatment of SARS-CoV-2 infection.

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Chakraborty, Stuti. "Neurocognitive, Neuropsychiatric, and Neurological Aspects of Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2, Covid-19)". In Infectious Diseases in Neurocognitive and Neuropsychiatric Medicine, 50–62. Oxford University PressOxford, 2024. http://dx.doi.org/10.1093/oso/9780192870414.003.0005.

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Abstract The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), also referred to as the novel coronavirus (Covid-19), belongs to the Sarbecovirus subgenus of the genus Betacoronavirus. In December 2019, unbiased sequencing in samples obtained from a cluster of patients symptomatic for pneumonia of unknown cause led to the discovery of Covid-19. The virus was highly transmittable and rapidly spreading, which led to the declaration of a global pandemic by the World Health Organization in March 2020. The symptoms of infection with Covid-19 replicated those seen in viral pneumonia and included cough, fever, and chest congestion and discomfort. More than two-thirds of patients hospitalized with the disease suffered from damage to the central nervous system in addition to respiratory symptoms. Along with neurological manifestations, evidence of psychiatric and cognitive involvement was also recorded in Covid-19 infection. Commonly reported psychiatric and neuropsychiatric presentations included depression, anxiety, post-traumatic stress disorder, delirium, altered consciousness, and dysexecutive syndrome. Cognitive decline because of Covid-19 infection was observed in functioning related to memory, executive functioning, and verbal fluency. This chapter discusses the multifold acute as well as persistent neuropsychiatric, cognitive, and neurological manifestations of Covid-19.

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Atti di convegni sul tema "Syndrome MARCH"

Torres-Avelar, Haidee, Diana Méndez-Nungaray e Isabel Martínez-Núñez. "P4 Budd-Chiari syndrome as initial presentation of antiphospholipid syndrome: case report". In 14th European Lupus Meeting, Bruges, Belgium, March 19–22, 2024. Lupus Foundation of America, 2024. http://dx.doi.org/10.1136/lupus-2024-el.58.

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Seisdedos, R., e P. Lopez. "5PSQ-034 Linezolid and serotonin syndrome". In 24th EAHP Congress, 27th–29th March 2019, Barcelona, Spain. British Medical Journal Publishing Group, 2019. http://dx.doi.org/10.1136/ejhpharm-2019-eahpconf.467.

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Tester, Andrew, Shubhangi Shewale e Steven Strachan. "6674 Improving thyroid surveillance in down’s syndrome". In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference, Birmingham, 25 March 2024 – 27 March 2024. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2024. http://dx.doi.org/10.1136/archdischild-2024-rcpch.409.

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Pearson, Madeline, e Jonathan Berg. "6374 Genotype: phenotype relationships in Myhre syndrome". In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference, Birmingham, 25 March 2024 – 27 March 2024. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2024. http://dx.doi.org/10.1136/archdischild-2024-rcpch.55.

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Cairns, Lauren, Callum Findlay, Kwamena Amonoo-Kuofi e Katherine Lachlan. "6802 Exploring tonsil pathology in PTEN hamartoma syndrome". In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference, Birmingham, 25 March 2024 – 27 March 2024. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2024. http://dx.doi.org/10.1136/archdischild-2024-rcpch.63.

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Méndez-Nungaray, Diana, Haidee Torres-Avelar, Alejandro Ortiz-Hernández e Luis Jara-Quezada. "P2 Catastrophic antiphospholipid syndrome during COVID-19 pandemic". In 14th European Lupus Meeting, Bruges, Belgium, March 19–22, 2024. Lupus Foundation of America, 2024. http://dx.doi.org/10.1136/lupus-2024-el.56.

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Calabuig, Pablo Martínez, Jorge Juan Fragío Gil, Roxana González Mazarío, Sara Moner Marín, Laura Salvador Maicas, Mireia Sanmartín Martínez, Amalia Rueda Cid, Juan José Lerma Garrido, Clara Molina Almela e Cristina Campos Fernández. "P157 Baricitinib for the treatment of Rhupus Syndrome". In 14th European Lupus Meeting, Bruges, Belgium, March 19–22, 2024. Lupus Foundation of America, 2024. http://dx.doi.org/10.1136/lupus-2024-el.211.

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Cosin Munilla, L., I. Ruiz-Jarabo, N. Ibañez-Heras, M. Gomez-Bermejo, C. Garzo-Bleda, A. Maraver-Villar e T. Molina-Garcia. "4CPS-101 Monitoring metabolic syndrome in olanzapine treated patients". In 28th EAHP Congress, Bordeaux, France, 20-21-22 March 2024. British Medical Journal Publishing Group, 2024. http://dx.doi.org/10.1136/ejhpharm-2024-eahp.205.

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Son, R., D. Hwang, AH Jung, JM Lim, SH Jung, SY Suh, HJ Hahn, YS Cho, EJ Park e HI Cheong. "5PSQ-082 Cyclophosphamide therapy in children with nephrotic syndrome". In Abstract Book, 23rd EAHP Congress, 21st–23rd March 2018, Gothenburg, Sweden. British Medical Journal Publishing Group, 2018. http://dx.doi.org/10.1136/ejhpharm-2018-eahpconf.436.

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Spencer, Matthew, Graham Shortland, Jennifer Evans, Jennifer Gardner, Zoe Morrison, Aung Min Saw, Stephen Jolles et al. "6727 The Syndrome Without A Name (SWAN) Clinic – Shortening the Diagnostic Odyssey". In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference, Birmingham, 25 March 2024 – 27 March 2024. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2024. http://dx.doi.org/10.1136/archdischild-2024-rcpch.636.

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Rapporti di organizzazioni sul tema "Syndrome MARCH"

Kalen, Nicholas. Bats of Colonial National Historical Park following white-nose syndrome. National Park Service, maggio 2023. http://dx.doi.org/10.36967/2299226.

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Abstract (sommario):

I conducted bat surveys at Colonial National Historical Park to assess the status of bat communities following potential impacts of white-nose syndrome (WNS) since its arrival in Virginia in 2009. This disease, caused by the fungus Pseudogymnoascus destructans, has severely reduced populations of several bat species in the eastern United States, threatening some with regional extirpation. In the East, most-affected species include the little brown bat (Myotis lucifugus), the federally-endangered northern long-eared bat (Myotis septentrionalis) and Indiana bat (Myotis sodalis) (USFWS 2007, USFWS 2022a), as well as the tricolored bat (Perimyotis subflavus), which has been proposed for endangered status (USFWS 2022b). I sampled sites in Yorktown and Jamestown Island with acoustic bat detectors from the spring of 2019 through the spring of 2021 and conducted capture surveys using mist nets in 2019 and 2021 to characterize seasonal occurrence of bat species with a focus on documenting WNS-imperiled species. Surveys also sought to document potential over-wintering of bats at COLO, especially northern long-eared bats, which occur year-round in the Coastal Plain of North Carolina. Acoustic results identified the presence of eleven bat species by echolocation calls: big brown bat (Eptesicus fuscus), eastern red bat (Lasiurus borealis), hoary bat (Lasiurus cinereus), silver-haired bat (Lasionycteris noctivagans), southeastern bat (Myotis austroriparius), little brown bat, northern long-eared bat, Indiana bat, evening bat (Nycticeius humeralis), tricolored bat, and Mexican free-tailed bat (Tadarida brasiliensis). Acoustic results included diagnostic echolocation calls of little brown, northern long-eared, and Indiana bats, however, presence should be interpreted with caution due to similarities of call structures among Myotis spp. bats. Capture surveys documented seven species: big brown, eastern red, hoary, silver-haired, southeastern, evening, and tricolored bats. To examine habitat associations of bat species, I used generalized linear mixed models of a selection of variable candidates: habitat type, distance to water, minimum nightly temperature, and nightly precipitation to predict summer activity by significant predictors. Activity of hoary, silver-haired, little brown, evening, tricolored, and Mexican free-tailed bats was highest in open habitats. Big brown bat and Indiana bat identifications were most associated with forest habitats. Eastern red bat activity was high in both forest and open sites. Southeastern bat activity was highest in wetland sites and was largely confined to these habitats. Northern long-eared bat activity was not significantly different among habitat types. To examine seasonality in bat species occurrence, I modeled acoustic activity in passes/night by Julian date using generalized additive models. Activity of big brown, eastern red, hoary, little brown, northern long-eared, tricolored, evening, and Mexican free-tailed bats was highest during summer. Silver-haired bat activity was highest in March indicative of seasonal migration. Hoary and Mexican free-tailed bat also exhibited high activity on several nights in the spring suggestive of migratory movement. Dormant season results suggest some winter occurrence for all identified bat species except Indiana bats. Very few characteristic calls of northern long-eared bats were observed from December through February, suggesting they winter locally in far lower abundances than in the Coastal Plain of North Carolina to the south.

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Jenkins, J. Lee, Edbert B. Hsu, Anna Russell, Allen Zhang, Lisa M. Wilson e Eric B. Bass. Infection Prevention and Control for the Emergency Medical Services and 911 Workforce. Agency for Healthcare Research and Quality (AHRQ), novembre 2022. http://dx.doi.org/10.23970/ahrqepctb42.

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Objectives. To summarize current evidence on exposures to infectious pathogens in the emergency medical services (EMS) and 911 workforce, and on practices for preventing, recognizing, and controlling occupationally acquired infectious diseases and related exposures in that workforce. Review methods. We obtained advice on how to answer four Guiding Questions by recruiting a panel of external experts on EMS clinicians, State-level EMS leadership, and programs relevant to EMS personnel, and by engaging representatives of professional societies in infectious diseases and emergency medicine. We searched PubMed®, Embase®, CINAHL®, and SCOPUS from January 2006 to March 2022 for relevant studies. We also searched for reports from State and Federal Government agencies or nongovernmental organizations interested in infection prevention and control in the EMS and 911 workforce. Results. Twenty-five observational studies reported on the epidemiology of infections in the EMS and 911 workforce. They did not report demographic differences except for a higher risk of hepatitis C in older workers and severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) in minorities. EMS clinicians certified/licensed in Advanced Life Support have a high risk for blood and fluid exposure, and EMS clinicians had a higher risk of hospitalization or death from SARS-CoV-2 than firefighters whose roles were not primarily related to medical care. Eleven observational studies reported on infection prevention and control practices (IPC), providing some evidence that hand hygiene, standard precautions, mandatory vaccine policies, and on-site vaccine clinics are effective. Research on IPC in EMS and 911 workers has increased significantly since the SARS-CoV-2 pandemic. Conclusions. Moderate evidence exists on the epidemiology of infections and effectiveness of IPC practices in EMS and 911 workers, including hand hygiene, standard precautions, mandatory vaccine policies, and vaccine clinics. Most evidence is observational, with widely varying methods, outcomes, and reporting. More research is needed on personal protective equipment effectiveness and vaccine acceptance, and better guidance is needed for research methods in the EMS and 911 worker setting.

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PCORI Biweekly COVID-19 Scan: Treatments for Patients With Acute Respiratory Distress Syndrome (March 18-31, 2021). Patient-Centered Outcomes Research Institute (PCORI), aprile 2021. http://dx.doi.org/10.25302/bcs23.2021.4.

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