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Bibliografie tematiche / Strophanthinum

Letteratura scientifica selezionata sul tema "Strophanthinum"

Autore: Grafiati

Pubblicato: 25 luglio 2024

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Articoli di riviste sul tema "Strophanthinum"

1

Shah, Yatin. "Strophanthin – ein besonderes Herzglykosid". Zeitschrift für Komplementärmedizin 3, n. 02 (aprile 2011): 48–51. http://dx.doi.org/10.1055/s-0030-1270928.

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2

Schmidramsi, H., B. Ostermayr e J. v. Arnim. "Proving of g-strophanthin". British Homoeopathic journal 82, n. 4 (ottobre 1993): 269. http://dx.doi.org/10.1016/s0007-0785(05)80679-9.

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3

Tsyvunin, V. V., S. Yu Shtrygol, D. V. Shtrygol e D. P. Kavraiskyi. "Anticonvulsive potential of cardiac glycosides under conditions of pentilentetrazole-induced seizures in mice: comparative study". Acta Medica Leopoliensia 27, n. 1-2 (giugno 2021): 63–69. http://dx.doi.org/10.25040/aml2021.01-02.063.

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Abstract (sommario):

Aim. The research provides a comparative analysis of the possible anticonvulsant action of different cardiac glycosides, namely: digoxin, lanatoside C, strophanthin G and corglycone. In addition, it detrmines the leading medication among the abovementioned ones by dose-dependence of its anticonvulsant action. Material and Methods. The research was performed on 66 random-bred albino male mice. The anticonvulsant effect of cardiac glycosides was studied in a baseline model of pentylenetetrazole-induced seizures. The first series of experiment evaluated the effect of cardiac glycosides on the course of model seizures in comparable doses of approximately 1/10 LD50 for the corresponding drug: digoxin, lanatoside C and strophanthin G - at a dose of 0.8 mg/kg; corglycone - at a dose of 1 mg/kg. The second series of experiments used the drug-leader, which was digoxin, in a wide dose range from 0.2 to 1.6 mg/kg. Digoxin, strophanthin G and corglycone were administered subcutaneously for 15 minutes, lanatoside C - intragastrically for 30 minutes before the induction of experimental seizures. Convulsive agent - pentylenetetrazole in the form of an aqueous solution was administered to animals subcutaneously at a dose of 80 mg/kg. Results and Discussion. Digoxin at a dose of 0.8 mg/kg under conditions of pentylenetetrazole-induced seizures shows a pronounced anticonvulsant activity: it is the only one among the studied cardiac glycosides that probably reduces lethality. In addition, digoxin prolongs the latency period of the first attacks, and reduces the number of clonic-tonic paroxysms in 1 mouse. Moderate anticonvulsant properties of lanatoside C were found both by a statistically significant decrease in the number of clonic-tonic seizures in 1 mouse, and by a significant reduction in the duration of the convulsive period. Although strophanthin G is unlikely to affect lethality, it moderately reduces the severity of pentylenetetrazole-induced seizures in mice, as evidenced by a statistically significant prolongation of the latency period of the first seizures, as well as a decrease in the number of clonic-tonic seizures in 1 mouse and the duration of seizures. Prophylactic administration of corglycone only prolongs the latency period of seizures and significantly reduces the number of clonic-tonic seizures in 1 mouse. The results of the dose-dependence study of digoxin anticonvulsant action show a clear anticonvulsant potential of this cardiac glycoside in a wide range of doses - from 0.2 to 1.6 mg/kg - with a maximum effect at a dose of 0.8 mg/kg. Conclusions. It was found that cardiac glycosides have a different severity of anticonvulsant effect: the most powerful anticonvulsant effect is due to digoxin, lanatoside C and strophanthin G have moderate properties, and the least pronounced effect is characteristic to corglycone. In addition, it was determined that digoxin exhibits anticonvulsant properties in a wide range of doses, and has the most pronounced anticonvulsant effect at a dose of 0.8 mg/kg. The obtained results substantiate the expediency of further in-depth study of digoxin as an anticonvulsant medicine.

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4

Vilensky, L. "Strofantin and novazurol for heart disease". Kazan medical journal 20, n. 9 (11 agosto 2021): 990–91. http://dx.doi.org/10.17816/kazmj77072.

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Abstract (sommario):

Weiss (Deut, med Woch., 1924 No. 33) reports on his observations on the treatment of severe cardiac patients with strophanthin and novosurol. In chronic heart diseases in a state of sharp decompensation, sometimes digitalis has no effect, especially when the body, due to the prolonged use of small doses of digitalis, has become accustomed to this remedy.

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5

McKenzie, A. G. "The rise and fall of strophanthin". International Congress Series 1242 (dicembre 2002): 95–100. http://dx.doi.org/10.1016/s0531-5131(02)00729-x.

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6

Dadashyan, A. M. "Experience in the use of convallotoxin in circulatory failure". Kazan medical journal 43, n. 5 (16 novembre 2021): 52–53. http://dx.doi.org/10.17816/kazmj87787.

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In the domestic literature, there are very few works on the action of convallotoxin, but they testify to the great effectiveness of the drug in patients with cardiovascular insufficiency. KA Khasanov believes that the therapeutic effect of konvallotoxin is not inferior to strophanthin, has a pronounced vascular and sedentary effect. LI Zhukovsky and BM Klebanov, noting the positive effect of konvallotoxin, pay attention to its side effects (the occurrence of pain and a feeling of compression in the heart, palpitations, shortness of breath, headaches, etc.).

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7

Frost, Jørgen, e Erik Warburg. "Clinical experiences concerning the effect of strophanthin". Acta Medica Scandinavica 130, S206 (24 aprile 2009): 132–44. http://dx.doi.org/10.1111/j.0954-6820.1948.tb12030.x.

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8

Ionova, E. O., I. A. Miroshkina, A. V. Sorokina e S. A. Kryzhanovskii. "Comparative evaluation of echocardiographic and morphometric characteristics of the rat heart left ventricle". Pharmacokinetics and Pharmacodynamics, n. 1 (10 maggio 2023): 41–44. http://dx.doi.org/10.37489/2587-7836-2023-1-41-44.

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Abstract (sommario):

Purpose of the study. Comparative evaluation of echocardiographic and morphometric dimensions of the rat heart left ventricle. Materials and methods. The study included 10 outbred male rats weighing 160–180 g. The size of the heart left ventricle was assessed using echocardiography and morphometry. The measurements were carried out according to standard protocols. To perform morphometric measurements, hearts were stopped in systole with a lethal dose (1.0 mg/kg) of 0.025 % strophanthin K solution, and in diastole, by immersing the hearts in a chilled physiological solution (calcium-free medium). Results. It was shown that the systolic size of the left ventricle of the heart according to echocardiography is 1.79Ѓ}0.10 mm, and morphometry 1.64Ѓ}0.09 mm (p = 0,302); diastolic size of the left ventricle, respectively, 3.42Ѓ}0.16 mm and 3.66Ѓ}0.17 mm (р = 0,318). The detected discrepancies do not exceed 10 % and, apparently, are due to the fact that the physiological dimensions of the heart left ventricle are measured by a non-invasive echocardiographic method, and for morphometric measurements of the dimensions in the systole, the heart was stopped by injection of strophanthin K, which entails an overload of heart cells with Ca2+ ions and as a result, contracture of cardiomyocytes; post-mortem cardiac arrest in diastole, in a calcium-free environment, is accompanied by a loss of cardiac muscle tone and, therefore, dilatation of the left ventricle will naturally be greater than physiological. Conclusion. Noninvasive echocardiographic measurement of the size of the heart left ventricle fully reflects the actual size of the left ventricle of the rat heart (the difference does not exceed 10 %), i. e. echocardiographic measurements are valid.

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9

Grosa, Giorgio, Gianna Allegrone e Erika Del Grosso. "LC–ESI-MS/MS characterization of strophanthin-K". Journal of Pharmaceutical and Biomedical Analysis 38, n. 1 (giugno 2005): 79–86. http://dx.doi.org/10.1016/j.jpba.2004.12.008.

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10

Olesin, A. I., I. V. Konstantinova, N. N. Tyuteleva e V. S. Ivanov. "Clinical and prognostic significance of left ventricular dysfunction predictors in patients with ventricular ectopy and without structural heart disease". Fundamental and Clinical Medicine 8, n. 3 (29 settembre 2023): 53–67. http://dx.doi.org/10.23946/2500-0764-2023-8-3-53-67.

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Aim. To determine the predictors of left ventricular dysfunction in patients with ventricular ectopic beats without structural heart disease.Material and Methods. We modeled ventricular ectopy in rats through early afterdepolarization (aconitine-induced arrhythmia) and delayed afterdepolarization (adrenaline arrhythmia). In addition, we modeled ventricular ectopy in rabbits and cats by delayed afterdepolarization (barium chloride-induced and strophanthin arrhythmias, respectively) and also modeled ventricular ectopy in dogs by re-entry hydrogen peroxide-induced arrhythmia. In addition to conventional electrocardiography parameters, we analyzed pre-ectopic interval, its variability, and the internal deviation index. Further, the study included 514 patients aged 16 to 34 years (mean 21.2 ± 0.2 years), and the number of premature ventricular contractions (PVCs) per day of observation ranged from 6,157 to 37,254 (mean 19,706 ± 656 PVCs). We registered the same parameters as in experimental arrhythmias but calculated them separately for mono- and polymorphic, left and right ventricular out-flow tract arrhythmias. The duration of follow-up of patients was up to 10 years. The endpoint was the detection or absence of cardiovascular and/or extracardiac pathology.Results. We recorded polymorphic PVCs and early monomorphic PVCs when modeling ventricular arrhythmias by the mechanism of delayed post-depolarization and early post-depolarization, respectively. Both early and late monomorphic PVCs were documented when inducing ventricular arrhythmias by re-entry. When modeling hydrogen peroxide-induced and strophanthin arrhythmias, we observed significantly higher values of PVC-QRS complex and ventricular arrhythmia internal deviation index in comparison with aconitine-induced arrhythmia. Favourable outcome was registered in 50.97% of patients, whilst coronary artery disease, arterial hypertension, and mitral valve prolapse were documented in 7.98%, 16.73% and 2.92% patients. The rest of the patients had gastrointestinal diseases. In patients with favourable outcome, the signs of monomorphic PVCs correlated with those revealed during the modeling of ventricular ectopy by early afterdepolarization (r = 0.92), whereas those signs of polymorphic PVCs correlated with those observed at barium chloride-induced delayed afterdepolarization (r = 0.94). In patients with CAD, signs of PVCs correlated with those registered during re-entry hydrogen peroxide-induced arrhythmia (r = 0.93), Finally, in patients with arterial hypertension and mitral valve prolapse signs of PVCs correlated with those documented at strophanthin-(r = 0.92) and adrenaline-induced delayed afterdepolarization (r = 0.89). In these patients, the values for both monomorphic and polymorphic PVCs, ventricular arrhythmia internal deviation index, duration of PVC-QRS complex and PVC-QRS/QRSaverage did not exceed 0.42 units, 149 ms and 1,44 units, respectively. The development of coronary artery disease and arterial hypertension well correlated with an increase in ventricular arrhythmia internal deviation index ≥ 0.56 units and QRS complex duration ≥ 157 ms. Mitral valve prolapse was associated with the duration of the QRS complex ≥ 159 ms of polymorphic PVCs.Conclusion. In patients with ventricular ectopy but without structural heart disease, an increase in the values of ventricular arrhythmia internal deviation index and the duration of PVC-QRS complex was ≥ 0.48 units and 149 ms, respectively, associated with the development of cardiovascular pathology. Development of coronary artery disease and hypertension correlated with ventricular arrhythmia internal deviation index ≥ 0.56 units, and QRS complex duration ≥ 157 ms in monomorphic and polymorphic PVCs, whereas development of mitral valve prolapse correlated QRS complex duration ≥ 159 ms in polymorphic PVCs.

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Più fonti

Tesi sul tema "Strophanthinum"

1

Weitkamp, Christine. "Warum zeigen die Herzglykoside Ouabain und Digoxin unterschiedliche Kreislaufwirkung? Charakterisierung eines mutmasslichen Herzglykosid-Bindungsproteins im Serum und Analyse der Wirkung beider Steroide auf die Endothelin-1- und NO-Freisetzung aus arteriellen Endothelzellen /". Wettenberg : VVB Laufersweiler, 2005. http://deposit.ddb.de/cgi-bin/dokserv?idn=976405792.

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2

Dhein, Stefan. "Elektrophysiologisches Wirkprofil von Triamteren, Triamterenester und Strophanthin sowie deren Kombination am isolierten Meerschweinchen-Papillarmuskel". 1987. http://catalog.hathitrust.org/api/volumes/oclc/17596333.html.

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3

Büchner, Frank H. "Untersuchungen zum Einfluß von Dichlormethan auf das Herz-Kreislauf-System am Strophanthin-Arrhythmie-Modell der Ratte /". 1997. http://www.gbv.de/dms/bs/toc/244065012.pdf.

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Libri sul tema "Strophanthinum"

1

Petry, Rolf-Jürgen. Strophanthin. Der mögliche Sieg über den Herzinfarkt. Die Fehlbeurteilung eines außergewöhnlichen Medikaments. Florilegium Verlag, 2003.

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Capitoli di libri sul tema "Strophanthinum"

1

Bonah, Christian. "‘We need for digitalis preparations what the state has established for serumtherapy …’ From Collecting Plants to International Standardization: The Case of Strophanthin, 1900–38". In Evaluating and Standardizing Therapeutic Agents, 1890–1950, 202–28. London: Palgrave Macmillan UK, 2010. http://dx.doi.org/10.1057/9780230285590_12.

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2

"Strophanthin, k-Strophanthin-β". In Spectroscopic Data of Steroid Glycosides: Cardenolides and Pregnanes, 2413–14. Boston, MA: Springer US, 2006. http://dx.doi.org/10.1007/978-0-387-39576-0_360.

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3

"strophanthin, n." In Oxford English Dictionary. 3^a ed. Oxford University Press, 2023. http://dx.doi.org/10.1093/oed/2815914232.

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4

"Emicymarin, e-Strophanthin". In Spectroscopic Data of Steroid Glycosides: Cardenolides and Pregnanes, 2170. Boston, MA: Springer US, 2006. http://dx.doi.org/10.1007/978-0-387-39576-0_90.

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5

"k-Strophanthoside, k-Strophanthin-γ". In Spectroscopic Data of Steroid Glycosides: Cardenolides and Pregnanes, 2584–85. Boston, MA: Springer US, 2006. http://dx.doi.org/10.1007/978-0-387-39576-0_535.

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6

"Cymarin, Cimarin, k-Strophanthin-α". In Spectroscopic Data of Steroid Glycosides: Cardenolides and Pregnanes, 2149–51. Boston, MA: Springer US, 2006. http://dx.doi.org/10.1007/978-0-387-39576-0_67.

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7

"Acocantherin, Ouabain, g-Strophanthin, g-Strophanthoside". In Spectroscopic Data of Steroid Glycosides: Cardenolides and Pregnanes, 2279–80. Boston, MA: Springer US, 2006. http://dx.doi.org/10.1007/978-0-387-39576-0_216.

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