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Libri sul tema "Sporulation"

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Autore: Grafiati
Pubblicato: 4 giugno 2021
Ultima modifica: 6 settembre 2023

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1

Sykes, David. The growth and sporulation of Verticillium chlamydosporium. Manchester: University of Manchester, 1994.

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2

Pleurotus unter Stress: Ökophysiologische Untersuchungen zu Wasserhaushalt und Sporulation. Berlin: J. Cramer, 1992.

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3

Ebisuzaki, Lawrence Kentaro. Promoter analysis of the sporulation-specific gene SPS4 of the yeast Saccharomyces cerevisiae. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1993.

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4

Issar, Smith, Slepecky Ralph, Setlow Peter e International Spore Conference (10th : 1988 : Woods Hole, Mass.), a cura di. Regulation of procaryotic development: Structural and functional analysis of bacterial sporulation and germination. Washington, D.C: American Society for Microbiology, 1989.

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5

Issar, Smith, Slepecky Ralph, Setlow Peter e International Spore Conference, (10th : 1988 : Woods Hole, Mass.), a cura di. Regulation of procaryotic development: A structural and functional analysis of bacterial sporulation and germination. Washington, D.C: American Society for Microbiology, 1989.

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6

Molnár, Enikö. Role of Rim 101 in mitotic repression of the yeast sporulation-specific genes DIT1 and DIT2. Ottawa: National Library of Canada, 2001.

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7

Tanny, Jason Chaim. Functional analysis of NREDIT, an element that controls expression of mid-late sporulation-specific genes in Saccharomyces cervisiae. Ottawa: National Library of Canada, 1998.

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8

Garg, G. K. Final technical report (Jan. 1982 to Dec. 1986) on studies on factors influencing sporulation, dipicolinic acid synthesis and heat resistance in bacterial spores to obtain information for improving methods of preservation of food products. Pantnagar, U.P., India: Department of Biochemistry, College of Basic Sciences & Humanities, G.B. Pant University of Agriculture & Technology, 1986.

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9

Kurtz, Stephen E. Analysis of gene expression during sporulation in yeast. 1985.

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10

Rahn-Lee, Lilah Lillian. The Control of DNA Replication During Sporulation in Bacillus subtilis. 2010.

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11

Law, David To Sang. Temporal pattern of gene expression during sporulation in saccharomyces cerevisiae. 1993.

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12

Smith, Issar, e Ralph A. Slepeky. Regulation of Procaryotic Development: Structural and Functional Analysis of Bacterial Sporulation and Germination. Amer Society for Microbiology, 1989.

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13

Hural, Kirsten. Influence of atmospheric humidity on sporulation of Sphaerotheca pannosa (Wallr.) Lev. var. rosae Wor. 1986.

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14

Hepworth, Shelley Roanne. Regulation of SPS4, a gene that is expressed midway through sporulation in Saccharomyces cerevisiae. 1998.

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15

Percival-Smith, Anthony *. The isolation and characterization of DNA sequences preferentially expressed during sporulation in saccharomyces cerevisiae. 1987.

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16

Pak, Julia. Regulation of expression and activity of Ndt80, a key activator of sporulation-specific genes and a target of the meiotic recombination checkpoint in Saccharomyces cerevisiae. 2002.

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17

Smith, Robert M. Other bacterial diseasesErysipeloid. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0025.

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Abstract (sommario):
Erysipeloid is an acute bacterial infection usually causing acute localised cellulitis as a secondary infection of traumatised skin. It is caused by Erysipelothrix rhusiopathiae (insidiosa), a non-sporulating Gram-positive rod-shaped bacterium, ubiquitous in the environment. It is the cause of swine erysipelas and also a pathogen or commensal in a variety of wild and domestic birds, animal and marine species. Human infection primarily associated with occupational exposure to infected or contaminated animals or handling animal products and therefore is commoner in farmers, butchers and abattoir workers and fisherman.Risk factors for the rare human invasive E. rhusiopathiae infection include conditions that affect the host immune response, such as alcoholism, cancer and diabetes. Treatment is with penicillin.Erysipelas can affect animals of all ages but is recognised more frequently in juveniles. Swine exhibit similar stages to the disease in man. Clinical manifestations in swine vary from the classical rhomboid urticaria (diamond skin), the condition of greatest prevalence and economic importance, to sepsis, polyarthritis, pneumonia and death.Prevention is largely a matter of good hygiene, herd management and by raising awareness in those at risk (especially butchers, farmers and fishermen); ensuring that clinicians are aware of E. rhusiopathiae as a possible cause of occupational skin lesions and bacterial endocarditis is important.
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