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Libri sul tema "Reperfusion injury"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 31 luglio 2024

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1

A, Grace P., e Mathie Robert T, a cura di. Ischaemia reperfusion injury. Oxford: Blackwell Science, 1999.

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2

1946-, Das Dipak Kumar, a cura di. Pathophysiology of reperfusion injury. Boca Raton: CRC Press, 1993.

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3

1946-, Das Dipak Kumar, a cura di. Cellular, biochemical, and molecular aspects of reperfusion injury. New York: New York Academy of Sciences, 1994.

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4

M, Yellon Derek, Jennings Robert B. 1926- e Council on Cardiac Metabolism, a cura di. Myocardial protection: The pathophysiology of reperfusion and reperfusion injury. New York: Raven Press, 1992.

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5

Kaski, Juan Carlos, Derek J. Hausenloy, Bernard John Gersh e Derek M. Yellon, a cura di. Management of Myocardial Reperfusion Injury. London: Springer London, 2012. http://dx.doi.org/10.1007/978-1-84996-019-9.

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6

Kaski, Juan Carlos. Management of myocardial reperfusion injury. Dordrecht: Springer, 2012.

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7

Takenobu, Kamada, Shiga Takeshi 1931- e McCuskey Robert S, a cura di. Tissue perfusion and organ fuction: Ischemia/reperfusion injury. Amsterdam, the Netherlands: Elsevier, 1996.

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8

Bilenko, M. V. Ishemicheskie i reperfuzionnye povrezhdeniia organov: Molekuliarnye mekhanizmy, puti preduprezhdeniia i lecheniia. Moskva: Meditsina, 1989.

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9

Friedhelm, Beyersdorf, a cura di. Ischemia-reperfusion injury in cardiac surgery. Georgetown, Tx: Landes Bioscience, 2000.

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10

A, Fantini Gary, a cura di. Ischemia-reperfusion of skeletal muscle. Austin: R.G. Landes, 1994.

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11

Tósaki, Árpád. Reperfusion-induced arrhythmias: Control and mechanisms. Budapest: Scientia Pub., 1995.

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12

Michel, Piper Hans, e Preusse C. J, a cura di. Ischemia-reperfusion in cardiac surgery. Dordrecht: Kluwer Academic Publishers, 1993.

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13

Frithjof, Hammersen, e Messmer K, a cura di. Ischemia and reperfusion: Proceedings of the 7th Bodensee Symposium on Microcirculation, Konstanz/Bodensee, June 26-27, 1987. Basel: Karger, 1989.

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14

Chen, Timothy Han. Human Tissue Engineered Model of Myocardial Ischemia-Reperfusion Injury. [New York, N.Y.?]: [publisher not identified], 2018.

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15

Seibu, Mochizuki, a cura di. The ischemic heart. Boston: Kluwer Academic Publishers, 1998.

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16

Oreopoulos, George D. The use of hypertonic preconditioning in hepatic ischemia-reperfusion injury. Ottawa: National Library of Canada, 2000.

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17

Quadri, Syed Mohammed. Cell death associated with ischemia-reperfusion injury during lung transplantation. Ottawa: National Library of Canada, 2003.

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18

Wilson, Ian Clark. The role of leucocytes in neonatal myocardial ischaemia-reperfusion injury. Birmingham: University of Birmingham, 1994.

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19

Najjar, Samer. Effects of ischemia and reperfusion on mitochondrial phosphate uptake in rat renal proximal tubules. [New Haven, Conn: s.n.], 1993.

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20

Ostadal, Bohuslav. Cardiac ischemia: From injury to protection. Boston: Kluwer Academic Publishers, 1999.

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21

H, Opie Lionel, a cura di. Stunning, hibernation, and calcium in myocardial ischemia and reperfusion. Boston: Kluwer Academic, 1992.

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22

Perrot, Marc De. The role of cytokines and lymphocytes in ischemia-reperfusion injury after lung transplantation. Ottawa: National Library of Canada, 2002.

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23

Dorion, Dominique. Pathogenic mechanisms of ischemia-reperfusion injury in the porcine and human muscle flaps. Ottawa: National Library of Canada, 1993.

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24

Cardella, Jonathan A. A novel cell culture model to study ischemia-reperfusion injury in lung transplantation. Ottawa: National Library of Canada, 1999.

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25

Lai, Laura R. B. Protein oxidation occurs in cardiomyocytes exposed to an in vitro model of hypoxia/reperfusion injury. Ottawa: National Library of Canada, 1996.

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26

Steinau, Hans-Ulrich. Major limb replantation and postischemia syndrome: Investigation of acute ischemia-induced myopathy and reperfusion injury. Berlin: Springer-Verlag, 1987.

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27

Abdillahi, Mariane Lul. Molecular and Cellular Signaling Mechanisms Elucidating Aldose Reductase Mediated Ischemia-Reperfusion Injury in the Myocardium. [New York, N.Y.?]: [publisher not identified], 2012.

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28

A, Kloner Robert, e Przyklenk Karin 1956-, a cura di. Stunned myocardium: Properties, mechanisms, and clinical manifestations. New York: Dekker, 1993.

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29

Herman, Stanley Leon. Age differences in the susceptibility of the normal rabbit myocardium to injury following ischaemia and reperfusion. Ottawa: National Library of Canada, 1992.

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30

1940-, Tarr Merrill, e Samson Fred 1918-, a cura di. Oxygen free radicals in tissue damage. Boston: Birkhäuser, 1993.

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31

A, Salerno Tomas, e Ricci Marco, a cura di. Myocardial protection. Elmsford, N.Y: Blackwell Pub., 2004.

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32

V, Schaff Hartzell, a cura di. Vasoactive factors produced by the endothelium: Physiology and surgical implications. Austin: R.G. Landes, 1994.

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33

Yellon, Derek M. Myocardial Protection: The Pathophysiology of Reperfusion and Reperfusion Injury. Raven Pr, 1992.

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34

Yellon, Derek M., Derek J. Hausenloy, Juan Carlos Kaski e Bernard John Gersh. Management of Myocardial Reperfusion Injury. Springer, 2014.

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35

Hausenloy, Derek J., Juan Carlos Kaski e Bernard John Gersh. Management of Myocardial Reperfusion Injury. Springer, 2012.

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36

Melin, Jan. Renal Ischemia / Reperfusion Injury in Diabetes. Uppsala Universitet, 2002.

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37

Beyersdorf, Friedhelm. Ischemia-Reperfusion Injury in Cardiac Surgery. Taylor & Francis Group, 2000.

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38

(Editor), Takenobu Kamada, Takeshi Shiga (Editor) e Robert S. McCuskey (Editor), a cura di. Tissue Perfusion and Organ Function. Elsevier Science Pub Co, 1995.

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39

Fink, Mitchell P. Ischaemia-reperfusion injury in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0308.

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Abstract (sommario):

Ischaemia/reperfusion (I/R) injury contributes to the pathogenesis of many common clinical conditions, including stroke, myocardial damage after percutaneous intervention for acute coronary artery occlusion, primary graft dysfunction after solid organ transplantation. The mechanisms that are responsible for I/R injury remain incompletely understood, but damage caused by reactive oxygen species (ROS) and reactive nitrogen species clearly is important. A number of therapeutic approaches, such as administration of ROS scavengers, are effective in animal models of I/R injury, but for the most part, translation of these findings into strategies that can clearly benefit patients has yet to be achieved.

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40

Rubin, Barry Broidy. Mechanisms of prolonged skeletal muscle reperfusion injury. 1992.

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41

Hendrikx, M. Endogenous Myocardial Protection Against Ischemic/ Reperfusion Injury. Leuven University Press, 1994.

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42

Pluta, Ryszard. Ischemia-Reperfusion Pathways in Alzheimer's Disease. Nova Science Pub Inc, 2007.

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43

Ischemia-Reperfusion in Cardiac Surgery. Island Press, 1993.

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44

Jancso, Gabor. Ischaemic Postconditioning Reduces Reperfusion Injury After Aortic Revascularization Surgery. INTECH Open Access Publisher, 2012.

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45

Nagano, Makoto, Nobuakira Takeda, Naranjan S. Dhalla e Seibu Mochizuki. Ischemic Heart. Springer London, Limited, 2007.

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46

Cywes, Robert. The role of platelets in hepatic allograft preservation-reperfusion injury. 1995.

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47

Lindsay, Thomas F. Skeletal muscle ischemia-reperfusion injury: evidence for an oxygen derived free radical mechanism of injury. 1988.

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48

Reffelmann, Thorsten, e Robert Kloner. Adjunctive Reperfusion Therapy Post-AMI. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199544769.003.0009.

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Abstract (sommario):

• Reperfusion of the occluded coronary artery in an ST-segment-elevation myocardial infarction is the most effective approach for reducing infarct size, preserving left ventricular ejection fraction, lowering the incidence and severity of congestive heart failure and improving prognosis• Hence, several pharmacologic agents intended to improve target vessel patency as an adjunct to thrombolysis or primary percutaneous coronary intervention have been shown to be beneficial in patients with reperfusion therapy for acute myocardial infarction, namely antiplatelet and anticoagulation agents• Animal investigations have suggested that coronary reperfusion may also result in undesirable cardiac alterations, termed ‘reperfusion injury’, such as reversible contractile dysfunction (‘stunning’), microvascular obstruction (‘no-reflow’), and in several studies the progression of myocardial necrosis (‘lethal reperfusion injury’)• Clinical investigations of various pharmacologic interventions as an adjunctive therapy to reperfusion to reduce final infarct size, the amount of contractile dysfunction and to improve prognosis have been mostly inconsistent; only a few interventions, e.g. adenosine and atrial natriuretic peptide seem to show promise at least in certain subgroups.

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49

Pathophysiology of Ischemia Reperfusion Injury and Use of Fingolimod in Cardioprotection. Elsevier, 2019. http://dx.doi.org/10.1016/c2018-0-02692-4.

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50

Ahmed, Naseer, e Soban Sadiq. Pathophysiology of Ischemia Reperfusion Injury and Use of Fingolimod in Cardioprotection. Elsevier Science & Technology, 2019.

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