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Libri sul tema "Reperfusion injury Pathophysiology"

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Autore: Grafiati
Pubblicato: 4 giugno 2021
Ultima modifica: 29 gennaio 2023

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1

1946-, Das Dipak Kumar, a cura di. Pathophysiology of reperfusion injury. Boca Raton: CRC Press, 1993.

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2

A, Grace P., e Mathie Robert T, a cura di. Ischaemia reperfusion injury. Oxford: Blackwell Science, 1999.

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3

Michael, Piper Hans, a cura di. Pathophysiology of severe ischemic myocardial injury. Dordrecht: Kluwer Academic Publishers, 1990.

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4

Bodensee, Symposium on Microcirculation (7th 1987 Konstanz Germany). Ischemia and reperfusion: Proceedings of the 7th Bodensee Symposium on Microcirculation, Konstanz/Bodensee, June 26-27, 1987. Basel: Karger, 1989.

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5

Najjar, Samer. Effects of ischemia and reperfusion on mitochondrial phosphate uptake in rat renal proximal tubules. [New Haven, Conn: s.n.], 1993.

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6

Ostadal, Bohuslav. Cardiac ischemia: From injury to protection. Boston: Kluwer Academic Publishers, 1999.

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7

Ostadal, Bohuslav. Cardiac ischemia: From injury to protection. Boston: Kluwer Academic Publishers, 1999.

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8

H, Opie Lionel, a cura di. Stunning, hibernation, and calcium in myocardial ischemia and reperfusion. Boston: Kluwer Academic, 1992.

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9

Kukreja, Rakesh C. Free radicals, cardiovascular dysfunction, and protection strategies. Austin: R.G. Landes Co., 1994.

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10

S, Dhalla Naranjan, a cura di. Myocardial ischemia and preconditioning. Boston: Kluwer Academic, 2003.

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11

Seccombe, John F. Vasoactive factors produced by the endothelium: Physiology and surgical implications. Austin: R.G. Landes, 1994.

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12

M, Yellon Derek, Jennings Robert B. 1926- e Council on Cardiac Metabolism, a cura di. Myocardial protection: The pathophysiology of reperfusion and reperfusion injury. New York: Raven Press, 1992.

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13

Yellon, Derek M. Myocardial Protection: The Pathophysiology of Reperfusion and Reperfusion Injury. Raven Pr, 1992.

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14

A, Fantini Gary, a cura di. Ischemia-reperfusion of skeletal muscle. Austin: R.G. Landes, 1994.

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15

Pathophysiology of Ischemia Reperfusion Injury and Use of Fingolimod in Cardioprotection. Elsevier, 2019. http://dx.doi.org/10.1016/c2018-0-02692-4.

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16

Ahmed, Naseer. Pathophysiology of Ischemia Reperfusion Injury and Use of Fingolimod in Cardioprotection. Elsevier Science & Technology Books, 2019.

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17

Ahmed, Naseer, e Soban Sadiq. Pathophysiology of Ischemia Reperfusion Injury and Use of Fingolimod in Cardioprotection. Elsevier Science & Technology, 2019.

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18

O'Farrell, Dermot Anthony. Aspects of the pathophysiology and prevention of reperfusion injury in skeletal muscle. 1993.

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19

Bilenko, M. V. Ischemia and Reperfusion of Various Organs: Injury Mechanisms, Methods of Prevention and Treatment. Nova Science Publishers, 2000.

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20

H, Opie Lionel. Stunning, Hibernation, and Calcium in Myocardial Ischemia and Reperfusion. Springer London, Limited, 2012.

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21

(Editor), Bohuslav Ost'ádal, e Frantisek Kolár (Editor), a cura di. Cardiac Ischemia: - From Injury to Protection (Basic Science for the Cardiologist). Springer, 1999.

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22

Neil, Granger D., e Schmid-Schönbein G. W, a cura di. Physiology and pathophysiology of leukocyte adhesion. New York: Oxford University Press, 1995.

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23

Bouchama, Abderrezak. Pathophysiology and management of hyperthermia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0353.

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Abstract (sommario):
Hyperthermia is a state of elevated core temperature that rises rapidly above 40°C, secondary to failure of thermoregulation. Hyperthermia has many causes, but it is the hallmark of three conditions—heatstroke, malignant hyperthermia, and neuroleptic malignant syndrome. The clinical and metabolic alterations of hyperthermia, if left untreated, can culminate in multiple organ system failure and death. High temperature causes direct cellular death and tissue damage. The extent of tissue injury is a function of the degree and duration of hyperthermia. Heat-induced ischaemia-reperfusion injury, and exacerbated activation of inflammation and coagulation are also contributory. Hyperthermia is a true medical emergency with rapid progression to multiple organ system failure and death. The primary therapeutic goal is to reduce body temperature as quickly as possible using physical cooling methods, and if indicated, the use of pharmacological treatment to accelerate cooling. There is no evidence of the superiority of one cooling technique over another. Non-invasive techniques that are easy to use and well-tolerated are preferred. Pharmacological cooling with Dantrolene sodium is crucial in the treatment of malignant hyperthermia.
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24

Oxygen free radicals in tissue damage. Boston: Birkhäuser, 1993.

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25

Hausenloy, Derek, e Derek Yellon, a cura di. An Introduction to Cardioprotection. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199544769.003.0001.

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Abstract (sommario):
• In its broadest sense, the term ‘cardioprotection’ encompasses ‘all mechanisms and means that contribute to the preservation of the heart by reducing or even preventing myocardial damage’• However, for the purposes of this book, the term ‘cardioprotection’ will refer to the endogenous mechanisms and therapeutic strategies that reduce or prevent myocardial damage induced by acute ischaemia-reperfusion injury• In this context, cardioprotection begins with the primary prevention of coronary heart disease and includes the reduction of myocardial injury sustained during coronary artery bypass graft surgery, and an acute myocardial infarction, conditions with considerable morbidity and mortality• An understanding of the pathophysiology of acute myocardial ischaemia-reperfusion injury is essential when designing new cardioprotective strategies• Several methods exist for both quantifying myocardial damage induced by acute ischaemia-reperfusion injury and for assessing myocardial salvage following the application of cardioprotective strategies• Importantly, novel cardioprotective strategies must be capable of preventing and reducing myocardial damage over and above that provided by current optimal therapy.
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26

1945-, Hori M., Maruyama Yukio 1941- e Reneman R. S, a cura di. Cardiac adaptation and failure. [Tokyo: Springer, 1994.

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27

Takeda, Nobuakira, Naranjan S. Dhalla, Manjeet Singh e Anton Lukas. Myocardial Ischemia and Preconditioning. Springer, 2012.

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28

(Editor), Naranjan S. Dhalla, Nobuakira Takeda (Editor), Manjeet Singh (Editor) e Anton Lukas (Editor), a cura di. Myocardial Ischemia and Preconditioning (Progress in Experimental Cardiology). Springer, 2002.

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29

Hori, Masatsugu, Robert S. Reneman e Yukio Maruyama. Cardiac Adaptation and Failure. Springer London, Limited, 2013.

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30

Hori, Masatsugu, Robert S. Reneman e Yukio Maruyama. Cardiac Adaptation and Failure. Springer, 2013.

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