Letteratura scientifica selezionata sul tema "Récepteurs glucocorticoïdes"
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Articoli di riviste sul tema "Récepteurs glucocorticoïdes"
Riguen, M., H. Chaker, N. Dammak, S. Toumi, A. Kammoun, H. Makni, T. Boudawara, S. Yaich, K. Kammoun e M. Ben Hmida. "Apport de l’expression rénale des récepteurs aux glucocorticoïdes en immunohistochimie au cours du syndrome néphrotique idiopathique". Néphrologie & Thérapeutique 17, n. 5 (settembre 2021): 330. http://dx.doi.org/10.1016/j.nephro.2021.07.212.
Testo completoBen Rhouma, K., S. Schimchowitsch, M. E. Stoeckel, J. M. Felix e M. Sakly. "Implication des Récepteurs Glucocorticoïdes de Type II dans l'Action Apoptotique de l'Aldostérone sur les Thymocytes de Rat". Archives of Physiology and Biochemistry 105, n. 2 (gennaio 1997): 216–24. http://dx.doi.org/10.1076/apab.105.2.216.12919.
Testo completoLaulhé, M., E. Pussard, J. Perrot, P. Kamenicky, M. Lombes, S. Viengchareun e L. Martinerie. "Polymorphisme du gène du récepteur glucocorticoïde (nr3c1) et hypersensibilité aux glucocorticoïdes : mécanismes moléculaires de l’insuffisance corticotrope glucocorticoïde-induite". Annales d'Endocrinologie 82, n. 5 (ottobre 2021): 261. http://dx.doi.org/10.1016/j.ando.2021.08.026.
Testo completoPrin, L., P. Lefebvre, V. Gruart, M. Capron, L. Storme, P. Formstecher, S. Loiseau e A. Capron. "Polynucléaire éosinophile et récepteur glucocorticoïde". Revue Française d'Allergologie et d'Immunologie Clinique 30, n. 2 (aprile 1990): 83–85. http://dx.doi.org/10.1016/s0335-7457(05)80216-3.
Testo completoBoullu, S., JG Velut, C. Oliver e M. Grino. "L'isoforme β du récepteur des glucocorticoïdes : un facteur de résistance aux glucocorticoïdes ?" médecine/sciences 14, n. 6-7 (1998): 812. http://dx.doi.org/10.4267/10608/1147.
Testo completoRoumestan, C., C. Gougat, D. Jaffuel e M. Mathieu. "Les glucocorticoïdes et leur récepteur : mécanismes d'action et conséquences cliniques". La Revue de Médecine Interne 25, n. 9 (settembre 2004): 636–47. http://dx.doi.org/10.1016/j.revmed.2004.01.012.
Testo completoVilarem, MJ. "Un antituberculeux, la rifampicine : ligand et activateur du récepteur des glucocorticoïdes." médecine/sciences 14, n. 4 (1998): 451. http://dx.doi.org/10.4267/10608/1061.
Testo completoKahn, A., e JC Dreyfus. "Le récepteur des glucocorticoïdes... et son homologie avec l'oncogène v-erb-A". médecine/sciences 2, n. 3 (1986): 151. http://dx.doi.org/10.4267/10608/3461.
Testo completoVitellius, G., J. Bouligand, J. Fagart, F. Castinetti, A. Guiochon Mantel, B. Delemer, S. Trabado e M. Lombès. "Trois mutations originales du récepteur des glucocorticoïdes révélées par un incidentalome surrénalien". Annales d'Endocrinologie 76, n. 4 (settembre 2015): 320. http://dx.doi.org/10.1016/j.ando.2015.07.090.
Testo completoDalle, H., M. Garcia, T. Ledent, T. T. H. Do, M. Buyse, R. Denis, S. Luquet, B. Fève e M. Moldes. "Rôle du récepteur des glucocorticoïdes adipocytaire dans la lipodystrophie induite par la corticostérone". Annales d'Endocrinologie 77, n. 4 (settembre 2016): 294. http://dx.doi.org/10.1016/j.ando.2016.07.907.
Testo completoTesi sul tema "Récepteurs glucocorticoïdes"
Sablonnière, Bernard. "Organisation hétéro-oligomérique du récepteur des glucocorticoi͏̈des". Lille 1, 1988. http://www.theses.fr/1988LIL10117.
Testo completoAmbroggi, Frédéric. "Identification de la cible cellulaire des effets des glucocorticoïdes". Bordeaux 2, 2005. http://www.theses.fr/2005BOR21303.
Testo completoLustenberger, Patrick. "La chromatographie d'affinité des récepteurs des hormones stéroïdes : application à la purification du récepteur des glucocorticoi͏̈des du foie de lapin". Lille 1, 1986. http://www.theses.fr/1986LIL10137.
Testo completoBelahsen, Youness. "Purification et caractérisation du récepteur des glucocorticoïdes sous forme transformée". Lille 1, 1987. http://www.theses.fr/1987LIL10045.
Testo completoCarillo, Conesa María-Ángeles. "Le rôle des glucorticoïdes et du récepteur des glucorticoïdes dans les processus de neurodegenerescence et d'inflammation dans le Système Nerveux Central". Paris 6, 2011. http://www.theses.fr/2011PA066688.
Testo completoKootar, Scherazad. "L’importance des récepteurs aux glucocorticoïdes dans la physiopathologie de la maladie d’Alzheimer". Thesis, Université Côte d'Azur (ComUE), 2017. http://www.theses.fr/2017AZUR4015/document.
Testo completoStrong evidence shows that oligomeric forms of the amyloid-ß peptide (oAß) cause synapse dysfunction promoting loss of hippocampus-dependent memories in the early phase of Alzheimer’s disease (AD). AD is also associated with Hypothalamus-Pituitary-Adrenal (HPA) axis dysfunction which results in an increase of glucocorticoids (CORT) activating glucocorticoid receptors (GRs). We showed that subchronic GR antagonist in 4 month Tg2576 (Tg+) mice could rescue the synaptic deficit and memory impairment (Lanté et al., 2015).In this context, we studied the contribution of GRs to AD physiopathology. Dysregulated HPA axis was characterized by increased CORT levels at 4 and 6 months of age and by loss of CORT feedback inhibition in the Tg+ mice. We further crossed the Tg+ with GRlox/lox to produce GRlox/loxTg+ mice. These mice innately exhibited high CORT levels from weaning period and due to other several unforeseen reasons, we discontinued using this new mouse model. Instead, to identify the functional relationship between the GRs and oAß at synapses, we shifted to acute oAß treatment in neurons in vitro and ex-vivo hippocampus slices. In neuron cultures, GR levels increased in the post synaptic density upon acute oAß treatment. Further, treatment of oAß on ex-vivo hippocampus slices after either pharmacological blocking of GR or genetic ablation, prevented the oAβ-dependent LTP impairment. To conclude, our results with the Tg+ mice suggest that a neuroendocrine dysregulation occurs during the onset of AD pathology. Additionally, we have evidence for a functional relationship between oAß and GRs with GRs at the synapse playing an important role in acute Aß-induced synapto-toxicity
Kootar, Scherazad. "L’importance des récepteurs aux glucocorticoïdes dans la physiopathologie de la maladie d’Alzheimer". Electronic Thesis or Diss., Université Côte d'Azur (ComUE), 2017. http://www.theses.fr/2017AZUR4015.
Testo completoStrong evidence shows that oligomeric forms of the amyloid-ß peptide (oAß) cause synapse dysfunction promoting loss of hippocampus-dependent memories in the early phase of Alzheimer’s disease (AD). AD is also associated with Hypothalamus-Pituitary-Adrenal (HPA) axis dysfunction which results in an increase of glucocorticoids (CORT) activating glucocorticoid receptors (GRs). We showed that subchronic GR antagonist in 4 month Tg2576 (Tg+) mice could rescue the synaptic deficit and memory impairment (Lanté et al., 2015).In this context, we studied the contribution of GRs to AD physiopathology. Dysregulated HPA axis was characterized by increased CORT levels at 4 and 6 months of age and by loss of CORT feedback inhibition in the Tg+ mice. We further crossed the Tg+ with GRlox/lox to produce GRlox/loxTg+ mice. These mice innately exhibited high CORT levels from weaning period and due to other several unforeseen reasons, we discontinued using this new mouse model. Instead, to identify the functional relationship between the GRs and oAß at synapses, we shifted to acute oAß treatment in neurons in vitro and ex-vivo hippocampus slices. In neuron cultures, GR levels increased in the post synaptic density upon acute oAß treatment. Further, treatment of oAß on ex-vivo hippocampus slices after either pharmacological blocking of GR or genetic ablation, prevented the oAβ-dependent LTP impairment. To conclude, our results with the Tg+ mice suggest that a neuroendocrine dysregulation occurs during the onset of AD pathology. Additionally, we have evidence for a functional relationship between oAß and GRs with GRs at the synapse playing an important role in acute Aß-induced synapto-toxicity
Burollaud, Thierry. "Etude du site actif du récepteur des glucocorticoi͏̈des : modalités comparées de son interaction avec les stéroi͏̈des agonistes et antagonistes". Lille 1, 1993. http://www.theses.fr/1993LIL10091.
Testo completoFormstecher, Pierre. "Le site de liaison aux stéroi͏̈des du récepteur des glucocorticoïdes : caractérisation à l'aide de sondes moléculaires diverses". Lille 1, 1986. http://www.theses.fr/1986LIL10136.
Testo completoMuller, Caroline. "Etude du mécanisme d'action anti-glucocorticoïde des dérivés 7-hydroxylés de la déhydroépiandrostérone". Paris, CNAM, 2006. http://www.theses.fr/2006CNAM0554.
Testo completoExperiments with a confocal microscope with a COS cell line transfected with the human glucocorticoid receptor (hGR) showed that dehydroepiandrosterone (DHEA) and its 7-hydroxylated derivatives do not modify the location of the receptor and do not prevent the nuclear transfer of hGR. These steroids had no effect on the transactivation activity of hGR. Thus, these neurosteroids were inactive on this model and their genomic action through the GR could be excluded. The human 11β-hydroxysteroid dehydrogenase type 1 (h11β-HSD1) was expressed in the yeast and each of the enzyme-catalyzed reactions were analyzed. The inter-conversion of the 7-hydroxylated DHEA metabolites was demonstrated as well as the oxidoreduction of cortisol and cortisone. A competition for the binding with the h11β-HSD1 may take place between these steroids and the glucocorticoid activation process, the former triggering immunity and the latter suppressing it